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Tytuł:
Deletion of Slc9a1 in Cx3cr1 cells stimulated microglial subcluster CREB1 signaling and microglia-oligodendrocyte crosstalk.
Autorzy:
Song S; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA. .; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA. .; Veterans Affairs Pittsburgh Health Care System, Pittsburgh, PA, 15213, USA. .
Oft H; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Metwally S; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Paruchuri S; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Bielanin J; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Fiesler V; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA.; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA.
Sneiderman C; Department of Neurological Surgery, University of Pittsburgh, Pittsburgh, PA, USA.
Kohanbash G; Department of Neurological Surgery, University of Pittsburgh, Pittsburgh, PA, USA.
Sun D; Department of Neurology, University of Pittsburgh, 3501 Fifth Avenue, Pittsburgh, PA, 15213, USA. .; Pittsburgh Institute for Neurodegenerative Disorders, University of Pittsburgh, Pittsburgh, PA, USA. .; Veterans Affairs Pittsburgh Health Care System, Pittsburgh, PA, 15213, USA. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 20; Vol. 21 (1), pp. 69. Date of Electronic Publication: 2024 Mar 20.
Typ publikacji:
Journal Article
MeSH Terms:
Brain*/metabolism
Microglia*/metabolism
Sodium-Hydrogen Exchanger 1*/metabolism
Animals ; Mice ; CX3C Chemokine Receptor 1/metabolism ; Macrophages/metabolism ; Oligodendroglia/metabolism ; Signal Transduction/genetics
Czasopismo naukowe
Tytuł:
Border-associated macrophages in the central nervous system.
Autorzy:
Sun R; Department of Neurological Surgery, Washington University School of Medicine in St. Louis, 660 S. Euclid Ave., Box 8057, St. Louis, MO, 63110, USA. .
Jiang H; Department of Anesthesiology, Washington University Pain Center, Washington University School of Medicine in St. Louis, 660 S. Euclid Ave., CB 8054, St. Louis, MO, 63110, USA. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 13; Vol. 21 (1), pp. 67. Date of Electronic Publication: 2024 Mar 13.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Central Nervous System*/pathology
Central Nervous System Diseases*
Humans ; Macrophages/pathology ; Microglia/pathology ; Brain/pathology
Czasopismo naukowe
Tytuł:
TYROBP/DAP12 knockout in Huntington's disease Q175 mice cell-autonomously decreases microglial expression of disease-associated genes and non-cell-autonomously mitigates astrogliosis and motor deterioration.
Autorzy:
Creus-Muncunill J; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, USA.
Haure-Mirande JV; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, USA.
Mattei D; Nash Family Department of Neuroscience and Friedman Brain Institute, Icahn School of Medicine at Mount Sinai, New York, USA.
Bons J; Buck Institute for Research on Aging, Novato, CA, USA.
Ramirez AV; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, USA.
Hamilton BW; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, USA.
Corwin C; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, USA.
Chowdhury S; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, USA.
Schilling B; Buck Institute for Research on Aging, Novato, CA, USA.
Ellerby LM; Buck Institute for Research on Aging, Novato, CA, USA.
Ehrlich ME; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, USA. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 08; Vol. 21 (1), pp. 66. Date of Electronic Publication: 2024 Mar 08.
Typ publikacji:
Journal Article
MeSH Terms:
Huntington Disease*/metabolism
Mice ; Animals ; Humans ; Microglia/metabolism ; Gliosis/genetics ; Gliosis/metabolism ; Proteomics ; Corpus Striatum/metabolism ; Disease Models, Animal ; Mice, Transgenic ; Membrane Proteins/metabolism ; Adaptor Proteins, Signal Transducing/metabolism
Czasopismo naukowe
Tytuł:
Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions.
Autorzy:
Shahror RA; Department of Pharmacology and Toxicology, College of Medicine, University of Arkansas for Medical Sciences (UAMS), 4301 West Markham Street, Slot 611, BIOMED-1, B306, Little Rock, AR, 72205, USA.
Morris CA; Department of Pharmacology and Toxicology, College of Medicine, University of Arkansas for Medical Sciences (UAMS), 4301 West Markham Street, Slot 611, BIOMED-1, B306, Little Rock, AR, 72205, USA.
Mohammed AA; Department of Pharmacology and Toxicology, College of Medicine, University of Arkansas for Medical Sciences (UAMS), 4301 West Markham Street, Slot 611, BIOMED-1, B306, Little Rock, AR, 72205, USA.
Wild M; Department of Pharmacology and Toxicology, College of Medicine, University of Arkansas for Medical Sciences (UAMS), 4301 West Markham Street, Slot 611, BIOMED-1, B306, Little Rock, AR, 72205, USA.
Zaman B; Department of Pharmacology and Toxicology, College of Medicine, University of Arkansas for Medical Sciences (UAMS), 4301 West Markham Street, Slot 611, BIOMED-1, B306, Little Rock, AR, 72205, USA.
Mitchell CD; Department of Pharmacology and Toxicology, College of Medicine, University of Arkansas for Medical Sciences (UAMS), 4301 West Markham Street, Slot 611, BIOMED-1, B306, Little Rock, AR, 72205, USA.
Phillips PH; Department of Ophthalmology, Harvey & Bernice Jones Eye Institute, University of Arkansas for Medical Sciences, Little Rock, AR, USA.
Rusch NJ; Department of Pharmacology and Toxicology, College of Medicine, University of Arkansas for Medical Sciences (UAMS), 4301 West Markham Street, Slot 611, BIOMED-1, B306, Little Rock, AR, 72205, USA.
Shosha E; Department of Pharmacology and Toxicology, College of Medicine, University of Arkansas for Medical Sciences (UAMS), 4301 West Markham Street, Slot 611, BIOMED-1, B306, Little Rock, AR, 72205, USA.; Clinical Pharmacy Department, Cairo University, Cairo, Egypt.
Fouda AY; Department of Pharmacology and Toxicology, College of Medicine, University of Arkansas for Medical Sciences (UAMS), 4301 West Markham Street, Slot 611, BIOMED-1, B306, Little Rock, AR, 72205, USA. .; Clinical Pharmacy Department, Cairo University, Cairo, Egypt. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 07; Vol. 21 (1), pp. 65. Date of Electronic Publication: 2024 Mar 07.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Diabetic Retinopathy*
Retinal Diseases*/pathology
Humans ; Retina/pathology ; Macrophages/pathology ; Ischemia/pathology
Czasopismo naukowe
Tytuł:
The contribution of pattern recognition receptor signalling in the development of age related macular degeneration: the role of toll-like-receptors and the NLRP3-inflammasome.
Autorzy:
Brandli A; Department of Anatomy and Physiology, The University of Melbourne, Grattan St, Parkville, Victoria, 3010, Australia.; Roche Pharma Research and Early Development, F. Hoffmann-La Roche Ltd, Basel, Switzerland.
Vessey KA; Department of Anatomy and Physiology, The University of Melbourne, Grattan St, Parkville, Victoria, 3010, Australia.
Fletcher EL; Department of Anatomy and Physiology, The University of Melbourne, Grattan St, Parkville, Victoria, 3010, Australia. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 05; Vol. 21 (1), pp. 64. Date of Electronic Publication: 2024 Mar 05.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Inflammasomes*
Macular Degeneration*
Animals ; NLR Family, Pyrin Domain-Containing 3 Protein ; Toll-Like Receptors ; Inflammation
Czasopismo naukowe
Tytuł:
Single-cell RNA sequencing reveals peripheral blood leukocyte responses to spinal cord injury in mice with humanised immune systems.
Autorzy:
Gillespie ER; School of Biomedical Sciences, Faculty of Medicine, The University of Queensland, Brisbane, QLD, 4072, Australia.
Grice LF; School of Biomedical Sciences, Faculty of Medicine, The University of Queensland, Brisbane, QLD, 4072, Australia.; Institute for Molecular Bioscience, The University of Queensland, Brisbane, Australia.
Courtney IG; School of Biomedical Sciences, Faculty of Medicine, The University of Queensland, Brisbane, QLD, 4072, Australia.
Lao HW; School of Biomedical Sciences, Faculty of Medicine, The University of Queensland, Brisbane, QLD, 4072, Australia.
Jung W; School of Biomedical Sciences, Faculty of Medicine, The University of Queensland, Brisbane, QLD, 4072, Australia.
Ramkomuth S; Institute for Molecular Bioscience, The University of Queensland, Brisbane, Australia.
Xie J; School of Biomedical Sciences, Faculty of Medicine, The University of Queensland, Brisbane, QLD, 4072, Australia.; Institute for Molecular Bioscience, The University of Queensland, Brisbane, Australia.
Brown DA; Neuroinflammation Research Group, Centre for Immunology and Allergy Research, Westmead Institute for Medical Research, Sydney, Australia.; Centre for Immunology and Allergy Research, Westmead Institute for Medical Research, The University of Sydney, Sydney, Australia.; Institute for Clinical Pathology, New South Wales Health Pathology, Sydney, Australia.
Walsham J; Intensive Care Unit, Princess Alexandra Hospital, Brisbane, Australia.; Medical School, Faculty of Medicine, The University of Queensland, Brisbane, Australia.
Radford KJ; Mater Research Institute, The University of Queensland, Translational Research Institute, Brisbane, Australia.
Nguyen QH; Institute for Molecular Bioscience, The University of Queensland, Brisbane, Australia.; QIMR Berghofer Medical Research Institute, Brisbane, Australia.
Ruitenberg MJ; School of Biomedical Sciences, Faculty of Medicine, The University of Queensland, Brisbane, QLD, 4072, Australia. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 01; Vol. 21 (1), pp. 63. Date of Electronic Publication: 2024 Mar 01.
Typ publikacji:
Journal Article
MeSH Terms:
Spinal Cord Injuries*/metabolism
Spinal Cord Diseases*
Mice ; Humans ; Animals ; Leukocytes/pathology ; Gene Expression ; Sequence Analysis, RNA
Czasopismo naukowe
Tytuł:
Inhibition of MMP8 effectively alleviates manic-like behavior and reduces neuroinflammation by modulating astrocytic CEBPD.
Autorzy:
Wang TY; Department of Psychiatry, National Cheng Kung University Hospital, College of Medicine, National Cheng Kung University, Tainan, Taiwan.
Weng EF; Neuroscience and Brain Disease Center, China Medical University, Taichung, Taiwan.; Graduate Institute of Biomedical Sciences, College of Medicine, China Medical University, Taichung, 404333, Taiwan.
Hsu YC; Neuroscience and Brain Disease Center, China Medical University, Taichung, Taiwan.; School of Medicine, College of Medicine, China Medical University, Taichung, Taiwan.
Shiu LP; Neuroscience and Brain Disease Center, China Medical University, Taichung, Taiwan.
Huang TW; Neuroscience and Brain Disease Center, China Medical University, Taichung, Taiwan.; Graduate Institute of Biomedical Sciences, College of Medicine, China Medical University, Taichung, 404333, Taiwan.; Ph.D. Program for Aging, China Medical University, Taichung, Taiwan.
Wu HC; Neuroscience and Brain Disease Center, China Medical University, Taichung, Taiwan.; Graduate Institute of Biomedical Sciences, College of Medicine, China Medical University, Taichung, 404333, Taiwan.
Hong JS; Neurobiology Laboratory, National Institute of Environmental Health Sciences, National Institutes of Health, 111 T.W. Alexander Dr., Research Triangle Park, Durham, NC, 27709, USA.
Wang SM; Neuroscience and Brain Disease Center, China Medical University, Taichung, Taiwan. .; Graduate Institute of Biomedical Sciences, College of Medicine, China Medical University, Taichung, 404333, Taiwan. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 28; Vol. 21 (1), pp. 61. Date of Electronic Publication: 2024 Feb 28.
Typ publikacji:
Journal Article
MeSH Terms:
Neuroinflammatory Diseases*
Ouabain*/toxicity
Animals ; Humans ; Mice ; Astrocytes/metabolism ; CCAAT-Enhancer-Binding Protein-delta/metabolism ; Matrix Metalloproteinase 8/metabolism
Czasopismo naukowe
Tytuł:
Elevated SLC7A2 expression is associated with an abnormal neuroinflammatory response and nitrosative stress in Huntington's disease.
Autorzy:
Gaudet ID; Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton, FL, 33431, USA.
Xu H; Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton, FL, 33431, USA.
Gordon E; Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton, FL, 33431, USA.
Cannestro GA; Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton, FL, 33431, USA.
Lu ML; Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton, FL, 33431, USA.
Wei J; Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton, FL, 33431, USA. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 28; Vol. 21 (1), pp. 59. Date of Electronic Publication: 2024 Feb 28.
Typ publikacji:
Journal Article
MeSH Terms:
Huntington Disease*/genetics
Nitrosative Stress*
Animals ; Humans ; Mice ; Amino Acid Transport Systems, Basic/metabolism ; Arginine ; Cell Line ; Inflammation ; Neuroinflammatory Diseases
Czasopismo naukowe
Tytuł:
Differential contribution of THIK-1 K channels and P2X7 receptors to ATP-mediated neuroinflammation by human microglia.
Autorzy:
Rifat A; Institute of Neurophysiology, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117, Berlin, Germany.; Berlin Institute of Health at Charité - Universitätsmedizin Berlin, Charitéplatz 1, 10117, Berlin, Germany.
Ossola B; Cerevance Ltd, 418 Cambridge Science Park, Milton Road, Cambridge, CB4 0PZ, UK.
Bürli RW; Cerevance Ltd, 418 Cambridge Science Park, Milton Road, Cambridge, CB4 0PZ, UK.
Dawson LA; Cerevance Ltd, 418 Cambridge Science Park, Milton Road, Cambridge, CB4 0PZ, UK.
Brice NL; Cerevance Ltd, 418 Cambridge Science Park, Milton Road, Cambridge, CB4 0PZ, UK.
Rowland A; Cerevance Ltd, 418 Cambridge Science Park, Milton Road, Cambridge, CB4 0PZ, UK.
Lizio M; Cerevance Ltd, 418 Cambridge Science Park, Milton Road, Cambridge, CB4 0PZ, UK.
Xu X; Cerevance Ltd, 418 Cambridge Science Park, Milton Road, Cambridge, CB4 0PZ, UK.
Page K; Cerevance Ltd, 418 Cambridge Science Park, Milton Road, Cambridge, CB4 0PZ, UK.
Fidzinski P; Berlin Institute of Health at Charité - Universitätsmedizin Berlin, Charitéplatz 1, 10117, Berlin, Germany.; Department of Neurology, Epilepsy-Center Berlin-Brandenburg, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117, Berlin, Germany.; Neurocure Cluster of Excellence, Neuroscience Clinical Research Center, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117, Berlin, Germany.
Onken J; Berlin Institute of Health at Charité - Universitätsmedizin Berlin, Charitéplatz 1, 10117, Berlin, Germany.; Department of Neurosurgery, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117, Berlin, Germany.
Holtkamp M; Department of Neurology, Epilepsy-Center Berlin-Brandenburg, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117, Berlin, Germany.
Heppner FL; Neurocure Cluster of Excellence, Neuroscience Clinical Research Center, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117, Berlin, Germany.; Department of Neuropathology, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117, Berlin, Germany.; German Center for Neurodegenerative Diseases (DZNE) Berlin, 10117, Berlin, Germany.
Geiger JRP; Institute of Neurophysiology, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117, Berlin, Germany.
Madry C; Institute of Neurophysiology, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117, Berlin, Germany. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 26; Vol. 21 (1), pp. 58. Date of Electronic Publication: 2024 Feb 26.
Typ publikacji:
Journal Article
MeSH Terms:
Microglia*/metabolism
NLR Family, Pyrin Domain-Containing 3 Protein*/metabolism
Humans ; Neuroinflammatory Diseases ; Ion Channels/metabolism ; Adenosine Triphosphate/pharmacology ; Adenosine Triphosphate/metabolism ; Receptors, Purinergic P2X7/metabolism
Czasopismo naukowe
Tytuł:
SPOCK2 modulates neuropathic pain by interacting with MT1-MMP to regulate astrocytic MMP-2 activation in rats with chronic constriction injury.
Autorzy:
Wang C; Department of Pain, The First Hospital of China Medical University, Shenyang, 110001, China.
Xu Y; Department of Pathology, The First Hospital of China Medical University, Shenyang, 110001, China.
Xu M; Department of Pain, The First Hospital of China Medical University, Shenyang, 110001, China.
Sun C; Department of Pain, The First Hospital of China Medical University, Shenyang, 110001, China.
Zhang X; Department of Pain, The First Hospital of China Medical University, Shenyang, 110001, China.
Tao X; Department of Pain, The First Hospital of China Medical University, Shenyang, 110001, China.
Song T; Department of Pain, The First Hospital of China Medical University, Shenyang, 110001, China. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 22; Vol. 21 (1), pp. 57. Date of Electronic Publication: 2024 Feb 22.
Typ publikacji:
Journal Article
MeSH Terms:
Astrocytes*/metabolism
Neuralgia*/etiology
Neuralgia*/metabolism
Animals ; Rats ; Constriction ; Matrix Metalloproteinase 14 ; Matrix Metalloproteinase 2 ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł:
Gliosis-dependent expression of complement factor H truncated variants attenuates retinal neurodegeneration following ischemic injury.
Autorzy:
Biber J; Department of Physiological Genomics, Ludwig-Maximilians-Universität München, Planegg-Martinsried, Germany.
Jabri Y; Department of Ophthalmology, University Hospital Regensburg, Regensburg, Germany.
Glänzer S; Department of Physiological Genomics, Ludwig-Maximilians-Universität München, Planegg-Martinsried, Germany.
Dort A; Experimental Ophthalmology, University of Marburg, Marburg, Germany.
Hoffelner P; Department of Physiological Genomics, Ludwig-Maximilians-Universität München, Planegg-Martinsried, Germany.
Schmidt CQ; Institute of Experimental and Clinical Pharmacology, Toxicology and Pharmacology of Natural Products, University of Ulm Medical Center, Ulm, Germany.; Institute of Pharmacy, Biochemical Pharmacy Group, Martin Luther University Halle-Wittenberg, Halle, Germany.
Bludau O; Department of Physiological Genomics, Ludwig-Maximilians-Universität München, Planegg-Martinsried, Germany.
Pauly D; Experimental Ophthalmology, University of Marburg, Marburg, Germany. .
Grosche A; Department of Physiological Genomics, Ludwig-Maximilians-Universität München, Planegg-Martinsried, Germany.
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 22; Vol. 21 (1), pp. 56. Date of Electronic Publication: 2024 Feb 22.
Typ publikacji:
Journal Article
Czasopismo naukowe
Tytuł:
Dimethyl fumarate improves cognitive impairment and neuroinflammation in mice with Alzheimer's disease.
Autorzy:
Wang T; Department of Neuroscience and Pathobiology, Research Institute of Environmental Medicine, Nagoya University, Aichi, 464-8601, Japan.; Department of Neuroscience and Pathobiology, Nagoya University Graduate School of Medicine, Aichi, Japan.
Sobue A; Department of Neuroscience and Pathobiology, Research Institute of Environmental Medicine, Nagoya University, Aichi, 464-8601, Japan. .; Medical Interactive Research and Academia Industry Collaboration Center, Research Institute of Environmental Medicine, Nagoya University, Aichi, Japan. .
Watanabe S; Department of Neuroscience and Pathobiology, Research Institute of Environmental Medicine, Nagoya University, Aichi, 464-8601, Japan.
Komine O; Department of Neuroscience and Pathobiology, Research Institute of Environmental Medicine, Nagoya University, Aichi, 464-8601, Japan.
Saido TC; Laboratory for Proteolytic Neuroscience, RIKEN Center for Brain Science, Saitama, Japan.
Saito T; Department of Neuroscience and Pathobiology, Research Institute of Environmental Medicine, Nagoya University, Aichi, 464-8601, Japan.; Department of Neurocognitive Science, Institute of Brain Science, Nagoya City University Graduate School of Medical Sciences, Aichi, Japan.
Yamanaka K; Department of Neuroscience and Pathobiology, Research Institute of Environmental Medicine, Nagoya University, Aichi, 464-8601, Japan. .; Department of Neuroscience and Pathobiology, Nagoya University Graduate School of Medicine, Aichi, Japan. .; Institute for Glyco-Core Research (iGCORE), Nagoya University, Aichi, Japan. .; Center for One Medicine Innovative Translational Research (COMIT), Nagoya University, Nagoya, Aichi, Japan. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 21; Vol. 21 (1), pp. 55. Date of Electronic Publication: 2024 Feb 21.
Typ publikacji:
Journal Article
MeSH Terms:
Alzheimer Disease*/complications
Alzheimer Disease*/drug therapy
Alzheimer Disease*/genetics
Cognitive Dysfunction*/drug therapy
Cognitive Dysfunction*/etiology
Mice ; Animals ; Dimethyl Fumarate/pharmacology ; Dimethyl Fumarate/therapeutic use ; Mice, Transgenic ; Neuroinflammatory Diseases ; NF-E2-Related Factor 2/metabolism ; Disease Models, Animal
Czasopismo naukowe
Tytuł:
Integration of National Health Insurance claims data and animal models reveals fexofenadine as a promising repurposed drug for Parkinson's disease.
Autorzy:
Kim JB; Department of Pharmacology, Ajou University School of Medicine, 164, Worldcup-Ro, Yeongtong-Gu, Suwon, 16499, Korea.; Center for Convergence Research of Neurological Disorders, Ajou University School of Medicine, Suwon, Korea.; Neuroscience Graduate Program, Department of Biomedical Sciences, Ajou University School of Medicine, Suwon, Korea.
Kim Y; Department of Biomedical Systems Informatics, Yonsei University College of Medicine, Seoul, Korea.
Kim SJ; Center for Convergence Research of Neurological Disorders, Ajou University School of Medicine, Suwon, Korea.
Ha TY; Department of Pharmacology, Ajou University School of Medicine, 164, Worldcup-Ro, Yeongtong-Gu, Suwon, 16499, Korea.; Center for Convergence Research of Neurological Disorders, Ajou University School of Medicine, Suwon, Korea.; Neuroscience Research Institute, Gachon University, Incheon, Korea.
Kim DK; Center for Convergence Research of Neurological Disorders, Ajou University School of Medicine, Suwon, Korea.
Kim DW; Department of Biomedical Systems Informatics, Yonsei University College of Medicine, Seoul, Korea.
So M; Standigm Inc., Seoul, Korea.
Kim SH; Center for Convergence Research of Neurological Disorders, Ajou University School of Medicine, Suwon, Korea.; Department of Physiology, Ajou University School of Medicine, Suwon, Korea.
Woo HG; Center for Convergence Research of Neurological Disorders, Ajou University School of Medicine, Suwon, Korea.; Department of Physiology, Ajou University School of Medicine, Suwon, Korea.
Yoon D; Department of Biomedical Systems Informatics, Yonsei University College of Medicine, Seoul, Korea. .
Park SM; Department of Pharmacology, Ajou University School of Medicine, 164, Worldcup-Ro, Yeongtong-Gu, Suwon, 16499, Korea. .; Center for Convergence Research of Neurological Disorders, Ajou University School of Medicine, Suwon, Korea. .; Neuroscience Graduate Program, Department of Biomedical Sciences, Ajou University School of Medicine, Suwon, Korea. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 21; Vol. 21 (1), pp. 53. Date of Electronic Publication: 2024 Feb 21.
Typ publikacji:
Journal Article
MeSH Terms:
Parkinson Disease*/pathology
Neurodegenerative Diseases*/metabolism
Terfenadine/*analogs & derivatives
Animals ; Caenorhabditis elegans/metabolism ; Oxidopamine ; Disease Models, Animal ; alpha-Synuclein/metabolism ; Dopaminergic Neurons
Czasopismo naukowe
Tytuł:
Parkinson's disease-derived α-synuclein assemblies combined with chronic-type inflammatory cues promote a neurotoxic microglial phenotype.
Autorzy:
Yildirim-Balatan C; Sorbonne Université, Paris, France.; Institut du Cerveau - Paris Brain Institute - ICM, Hôpital de la Pitié-Salpêtrière, 91 Bd de l'Hôpital, 75013, Paris, France.; Inserm UMRS 1127, Paris, France.; CNRS UMR 7225, Paris, France.
Fenyi A; CEA and Laboratory of Neurodegenerative Diseases, CNRS, Institut François Jacob, MIRCen, 92265, Fontenay-aux-Roses, France.
Besnault P; Sorbonne Université, Paris, France.; Institut du Cerveau - Paris Brain Institute - ICM, Hôpital de la Pitié-Salpêtrière, 91 Bd de l'Hôpital, 75013, Paris, France.; Inserm UMRS 1127, Paris, France.; CNRS UMR 7225, Paris, France.
Gomez L; Sorbonne Université, Paris, France.; Institut du Cerveau - Paris Brain Institute - ICM, Hôpital de la Pitié-Salpêtrière, 91 Bd de l'Hôpital, 75013, Paris, France.; Inserm UMRS 1127, Paris, France.; CNRS UMR 7225, Paris, France.
Sepulveda-Diaz JE; Sorbonne Université, Paris, France.; Institut du Cerveau - Paris Brain Institute - ICM, Hôpital de la Pitié-Salpêtrière, 91 Bd de l'Hôpital, 75013, Paris, France.; Inserm UMRS 1127, Paris, France.; CNRS UMR 7225, Paris, France.
Michel PP; Sorbonne Université, Paris, France.; Institut du Cerveau - Paris Brain Institute - ICM, Hôpital de la Pitié-Salpêtrière, 91 Bd de l'Hôpital, 75013, Paris, France.; Inserm UMRS 1127, Paris, France.; CNRS UMR 7225, Paris, France.
Melki R; CEA and Laboratory of Neurodegenerative Diseases, CNRS, Institut François Jacob, MIRCen, 92265, Fontenay-aux-Roses, France.
Hunot S; Sorbonne Université, Paris, France. .; Institut du Cerveau - Paris Brain Institute - ICM, Hôpital de la Pitié-Salpêtrière, 91 Bd de l'Hôpital, 75013, Paris, France. .; Inserm UMRS 1127, Paris, France. .; CNRS UMR 7225, Paris, France. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 21; Vol. 21 (1), pp. 54. Date of Electronic Publication: 2024 Feb 21.
Typ publikacji:
Journal Article
MeSH Terms:
Parkinson Disease*/pathology
Neurotoxicity Syndromes*/metabolism
Humans ; alpha-Synuclein/genetics ; alpha-Synuclein/metabolism ; Microglia/metabolism ; Tumor Necrosis Factor-alpha/metabolism ; Cues ; Inflammation/metabolism ; Dopaminergic Neurons/pathology ; Glutamates/metabolism ; Iron/metabolism
Czasopismo naukowe
Tytuł:
The insular cortex is not insular in thyroid eye disease: neuroimaging revelations of central-peripheral system interaction.
Autorzy:
Zhang H; Department of Ophthalmology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Orbital Diseases and Ocular Oncology, Shanghai, China.
Liu Y; Department of Ophthalmology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Orbital Diseases and Ocular Oncology, Shanghai, China.
Xia D; School of Health Science and Engineering, University of Shanghai for Science and Technology, Shanghai, China.
Jiang M; Department of Radiology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Li Y; Department of Ophthalmology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Orbital Diseases and Ocular Oncology, Shanghai, China.
Sun J; Department of Ophthalmology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Orbital Diseases and Ocular Oncology, Shanghai, China.
Guan H; Department of Endocrinology, Guangdong Provincial People's Hospital (Guangdong Academy of Medical Sciences), Southern Medical University, Guangzhou, China.
Zhu L; Department of Radiology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Song X; Department of Ophthalmology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Orbital Diseases and Ocular Oncology, Shanghai, China.
Wang J; Institute of Sports Medicine and Health, Chengdu Sport University, Chengdu, China. .
Fan X; Department of Ophthalmology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China. .; Shanghai Key Laboratory of Orbital Diseases and Ocular Oncology, Shanghai, China. .
Zhou H; Department of Ophthalmology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China. .; Shanghai Key Laboratory of Orbital Diseases and Ocular Oncology, Shanghai, China. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 17; Vol. 21 (1), pp. 51. Date of Electronic Publication: 2024 Feb 17.
Typ publikacji:
Journal Article
MeSH Terms:
Insular Cortex*
Graves Ophthalmopathy*
Humans ; Animals ; Mice ; Magnetic Resonance Imaging/methods ; Neuroimaging ; Brain ; Brain Mapping/methods
Czasopismo naukowe
Tytuł:
Combination protein biomarkers predict multiple sclerosis diagnosis and outcomes.
Autorzy:
Kodosaki E; UK Dementia Research Institute at University College London, London, WC1E6BT, UK.; Department of Neurodegenerative Disease, UCL Institute of Neurology, Queen Square, London, WC1N3BG, UK.; Division of Psychological Medicine and Clinical Neurosciences, School of Medicine, Cardiff University, Cardiff, CF14 4XW, UK.
Watkins WJ; Division of Infection and Immunity, School of Medicine, Cardiff University, Cardiff, UK.
Loveless S; Division of Psychological Medicine and Clinical Neurosciences, School of Medicine, Cardiff University, Cardiff, CF14 4XW, UK.
Kreft KL; Department of Neurology, University Hospital of Wales, Cardiff, UK.
Richards A; Division of Psychological Medicine and Clinical Neurosciences, School of Medicine, Cardiff University, Cardiff, CF14 4XW, UK.
Anderson V; Division of Psychological Medicine and Clinical Neurosciences, School of Medicine, Cardiff University, Cardiff, CF14 4XW, UK.
Hurler L; Department of Internal Medicine and Haematology, Semmelweis University, Budapest, 1085, Hungary.
Robertson NP; Division of Psychological Medicine and Clinical Neurosciences, School of Medicine, Cardiff University, Cardiff, CF14 4XW, UK.; Department of Neurology, University Hospital of Wales, Cardiff, UK.
Zelek WM; Division of Infection and Immunity, School of Medicine, Cardiff University, Cardiff, UK.
Tallantyre EC; Division of Psychological Medicine and Clinical Neurosciences, School of Medicine, Cardiff University, Cardiff, CF14 4XW, UK. .; Department of Neurology, University Hospital of Wales, Cardiff, UK. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 17; Vol. 21 (1), pp. 52. Date of Electronic Publication: 2024 Feb 17.
Typ publikacji:
Journal Article
MeSH Terms:
Multiple Sclerosis*/diagnosis
Multiple Sclerosis*/cerebrospinal fluid
Chitinases*
Humans ; Osteopontin ; Vitamin D-Binding Protein ; Biomarkers/cerebrospinal fluid ; Recurrence
Czasopismo naukowe
Tytuł:
Advanced patient-specific microglia cell models for pre-clinical studies in Alzheimer's disease.
Autorzy:
Cuní-López C; Mental Health and Neuroscience Department, QIMR Berghofer Medical Research Institute, Herston, QLD, 4006, Australia.; Faculty of Medicine, The University of Queensland, Herston, QLD, 4006, Australia.
Stewart R; Mental Health and Neuroscience Department, QIMR Berghofer Medical Research Institute, Herston, QLD, 4006, Australia.; UQ Centre for Clinical Research, The University of Queensland, Brisbane City, QLD, 4029, Australia.
Oikari LE; Mental Health and Neuroscience Department, QIMR Berghofer Medical Research Institute, Herston, QLD, 4006, Australia.; School of Biomedical Sciences, Queensland University of Technology, Brisbane City, QLD, 4000, Australia.
Nguyen TH; Scientific Services, QIMR Berghofer Medical Research Institute, Herston, QLD, 4006, Australia.
Roberts TL; UQ Centre for Clinical Research, The University of Queensland, Brisbane City, QLD, 4029, Australia.; Ingham Institute for Applied Medical Research and School of Medicine, Western Sydney University, Liverpool, NSW, 2170, Australia.
Sun Y; Mental Health and Neuroscience Department, QIMR Berghofer Medical Research Institute, Herston, QLD, 4006, Australia.
Guo CC; Mental Health and Neuroscience Department, QIMR Berghofer Medical Research Institute, Herston, QLD, 4006, Australia.; ActiGraph LLC, Pensacola, FL, 32502, USA.
Lupton MK; Mental Health and Neuroscience Department, QIMR Berghofer Medical Research Institute, Herston, QLD, 4006, Australia.; Faculty of Medicine, The University of Queensland, Herston, QLD, 4006, Australia.; School of Biomedical Sciences, Queensland University of Technology, Brisbane City, QLD, 4000, Australia.
White AR; Mental Health and Neuroscience Department, QIMR Berghofer Medical Research Institute, Herston, QLD, 4006, Australia. .; Faculty of Medicine, The University of Queensland, Herston, QLD, 4006, Australia. .
Quek H; Mental Health and Neuroscience Department, QIMR Berghofer Medical Research Institute, Herston, QLD, 4006, Australia. .; School of Biomedical Sciences, The University of Queensland, Lucia, QLD, 4072, Australia. .; School of Biomedical Sciences, Queensland University of Technology, Brisbane City, QLD, 4000, Australia. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 15; Vol. 21 (1), pp. 50. Date of Electronic Publication: 2024 Feb 15.
Typ publikacji:
Journal Article
MeSH Terms:
Alzheimer Disease*/pathology
Humans ; Microglia/metabolism ; Neuroglia/metabolism ; Amyloid beta-Peptides/metabolism ; Cytokines/metabolism
Czasopismo naukowe
Tytuł:
Intracerebellar injection of monocytic immature myeloid cells prevents the adverse effects caused by stereotactic surgery in a model of cerebellar neurodegeneration.
Autorzy:
Del Pilar C; Institute for Neuroscience of Castile and Leon, INCyL, Universidad de Salamanca, C/Pintor Fernando Gallego 1, 37007, Salamanca, Spain.; Institute of Biomedical Research of Salamanca, IBSAL, Salamanca, Spain.
Garrido-Matilla L; Institute for Neuroscience of Castile and Leon, INCyL, Universidad de Salamanca, C/Pintor Fernando Gallego 1, 37007, Salamanca, Spain.; Departamento de Psicobiología, Facultad de Psicología, Universidad Nacional de Educación a Distancia (UNED), Madrid, Spain.
Del Pozo-Filíu L; Institute for Neuroscience of Castile and Leon, INCyL, Universidad de Salamanca, C/Pintor Fernando Gallego 1, 37007, Salamanca, Spain.; Translational Stroke Laboratory (TREAT), Clinical Neurosciences Research Laboratory (LINC), Health Research Institute of Santiago de Compostela (IDIS), Santiago de Compostela, Spain.
Lebrón-Galán R; Neuroimmuno-Repair Group, Hospital Nacional de Parapléjicos-SESCAM, Finca La Peraleda s/n, 45004, Toledo, Spain.; Hospital Universitario de Toledo, Avd. Río Guadiana, s/n, 45007, Toledo, Spain.
Arias RF; Institute for Neuroscience of Castile and Leon, INCyL, Universidad de Salamanca, C/Pintor Fernando Gallego 1, 37007, Salamanca, Spain.; Institute of Biomedical Research of Salamanca, IBSAL, Salamanca, Spain.
Clemente D; Neuroimmuno-Repair Group, Hospital Nacional de Parapléjicos-SESCAM, Finca La Peraleda s/n, 45004, Toledo, Spain.; Centro de Investigación Biomédica en Red de Enfermedades Neurodegenerativas (CIBERNED), Carlos III Health Institute, Av. Monforte de Lemos, 3-5, 28029, Madrid, Spain.
Alonso JR; Institute for Neuroscience of Castile and Leon, INCyL, Universidad de Salamanca, C/Pintor Fernando Gallego 1, 37007, Salamanca, Spain.; Institute of Biomedical Research of Salamanca, IBSAL, Salamanca, Spain.
Weruaga E; Institute for Neuroscience of Castile and Leon, INCyL, Universidad de Salamanca, C/Pintor Fernando Gallego 1, 37007, Salamanca, Spain. .; Institute of Biomedical Research of Salamanca, IBSAL, Salamanca, Spain. .
Díaz D; Institute for Neuroscience of Castile and Leon, INCyL, Universidad de Salamanca, C/Pintor Fernando Gallego 1, 37007, Salamanca, Spain. .; Institute of Biomedical Research of Salamanca, IBSAL, Salamanca, Spain. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 14; Vol. 21 (1), pp. 49. Date of Electronic Publication: 2024 Feb 14.
Typ publikacji:
Journal Article
MeSH Terms:
Myeloid Cells*/metabolism
Cerebellum*
Mice ; Humans ; Animals ; Purkinje Cells/pathology ; Monocytes ; Immunosuppressive Agents
Czasopismo naukowe
Tytuł:
The role of CD56 NK cells in neurodegenerative disorders.
Autorzy:
Rodriguez-Mogeda C; Department of Molecular Cell Biology and Immunology, Amsterdam UMC Location Vrije Universiteit Amsterdam, Amsterdam, The Netherlands.; Amsterdam Neuroscience, Amsterdam, The Netherlands.; MS Center Amsterdam, Amsterdam UMC Location Vrije Universiteit, Amsterdam, The Netherlands.
van Ansenwoude CMJ; Department of Molecular Cell Biology and Immunology, Amsterdam UMC Location Vrije Universiteit Amsterdam, Amsterdam, The Netherlands.; Amsterdam Neuroscience, Amsterdam, The Netherlands.; MS Center Amsterdam, Amsterdam UMC Location Vrije Universiteit, Amsterdam, The Netherlands.
van der Molen L; IQ Health Science Department, Radboud University Medical Center, Nijmegen, The Netherlands.
Strijbis EMM; Amsterdam Neuroscience, Amsterdam, The Netherlands.; MS Center Amsterdam, Amsterdam UMC Location Vrije Universiteit, Amsterdam, The Netherlands.; Department of Neurology, Amsterdam UMC Location Vrije Universiteit, Amsterdam, The Netherlands.
Mebius RE; Department of Molecular Cell Biology and Immunology, Amsterdam UMC Location Vrije Universiteit Amsterdam, Amsterdam, The Netherlands.; Amsterdam Infection and Immunity Institute, Amsterdam, The Netherlands.
de Vries HE; Department of Molecular Cell Biology and Immunology, Amsterdam UMC Location Vrije Universiteit Amsterdam, Amsterdam, The Netherlands. .; Amsterdam Neuroscience, Amsterdam, The Netherlands. .; MS Center Amsterdam, Amsterdam UMC Location Vrije Universiteit, Amsterdam, The Netherlands. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 13; Vol. 21 (1), pp. 48. Date of Electronic Publication: 2024 Feb 13.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Antineoplastic Agents*
Neurodegenerative Diseases*
Humans ; Killer Cells, Natural ; Cytokines ; Cell Differentiation
Czasopismo naukowe
Tytuł:
Inflammatory biomarkers for neurobehavioral dysregulation in former American football players: findings from the DIAGNOSE CTE Research Project.
Autorzy:
van Amerongen S; Boston University CTE Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Alzheimer Center Amsterdam, Neurology, Vrije Universiteit Amsterdam, Amsterdam UMC Location VUmc, Amsterdam, The Netherlands.; Amsterdam Neuroscience, Neurodegeneration, Amsterdam, The Netherlands.
Pulukuri SV; Boston University CTE Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.
Tuz-Zahra F; Department of Biostatistics, Boston University School of Public Health, Boston, MA, USA.
Tripodis Y; Boston University CTE Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Department of Biostatistics, Boston University School of Public Health, Boston, MA, USA.; Boston University Alzheimer's Disease Research Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.
Cherry JD; Boston University CTE Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Boston University Alzheimer's Disease Research Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; VA Boston Healthcare System, U.S. Department of Veteran Affairs, Boston, MA, USA.; Department of Veterans Affairs Medical Center, Bedford, MA, USA.; Department of Pathology and Laboratory Medicine, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.
Bernick C; Cleveland Clinic Lou Ruvo Center for Brain Health, Las Vegas, NV, USA.
Geda YE; Department of Neurology and the Franke Global Neuroscience Education Center, Barrow Neurological Institute, Phoenix, AZ, USA.
Wethe JV; Department of Psychiatry and Psychology, Mayo Clinic School of Medicine, Mayo Clinic Arizona, Scottsdale, AZ, USA.
Katz DI; Boston University CTE Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Department of Neurology, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Brain Injury Program, Encompass Health Braintree Rehabilitation Hospital, Braintree, MA, USA.
Alosco ML; Boston University CTE Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Boston University Alzheimer's Disease Research Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Department of Neurology, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.
Adler CH; Department of Neurology, Mayo Clinic College of Medicine, Mayo Clinic Arizona, Scottsdale, AZ, USA.
Balcer LJ; Departments of Neurology, Population Health and Ophthalmology, NYU Grossman School of Medicine, New York, NY, USA.
Ashton NJ; Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy at the University of Gothenburg, Mölndal, Sweden.; Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.; Institute of Psychiatry, Psychology and Neuroscience, King's College London, Maurice Wohl Institute Clinical Neuroscience Institute, London, UK.
Blennow K; Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy at the University of Gothenburg, Mölndal, Sweden.; Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.
Zetterberg H; Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy at the University of Gothenburg, Mölndal, Sweden.; Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.; Institute of Psychiatry, Psychology and Neuroscience, King's College London, Maurice Wohl Institute Clinical Neuroscience Institute, London, UK.; Department of Neurodegenerative Disease, UCL Institute of Neurology, Queen Square, London, UK.; UK Dementia Research Institute at UCL, London, UK.; Hong Kong Center for Neurodegenerative Diseases, Clear Water Bay, Hong Kong, China.; Wisconsin Alzheimer's Disease Research Center, University of Wisconsin School of Medicine and Public Health, University of Wisconsin-Madison, Madison, WI, 53792, USA.
Daneshvar DH; Department of Physical Medicine and Rehabilitation, Harvard Medical School, Boston, MA, USA.
Colasurdo EA; Veterans Affairs Northwest Mental Illness Research, Education, and Clinical Center, Seattle, WA, USA.
Iliff JJ; Veterans Affairs Northwest Mental Illness Research, Education, and Clinical Center, Seattle, WA, USA.; Department of Psychiatry and Behavioral Sciences, University of Washington School of Medicine, Seattle, WA, USA.
Li G; Veterans Affairs Northwest Mental Illness Research, Education, and Clinical Center, Seattle, WA, USA.; Department of Psychiatry and Behavioral Sciences, University of Washington School of Medicine, Seattle, WA, USA.; Education, and Clinical Center, Veterans Affairs Puget Sound Health Care System Geriatric Research, Seattle, WA, USA.
Peskind ER; Veterans Affairs Northwest Mental Illness Research, Education, and Clinical Center, Seattle, WA, USA.; Department of Psychiatry and Behavioral Sciences, University of Washington School of Medicine, Seattle, WA, USA.
Shenton ME; Psychiatry Neuroimaging Laboratory, Harvard Medical School, Departments of Psychiatry and Radiology, Brigham and Women's Hospital, Boston, MA, USA.
Reiman EM; Banner Alzheimer's Institute, University of Arizona, Arizona State University, Translational Genomics Research Institute, and Arizona Alzheimer's Consortium, Phoenix, AZ, USA.
Cummings JL; Chambers-Grundy Center for Transformative Neuroscience, Department of Brain Health, School of Integrated Health Sciences, University of Nevada Las Vegas, Las Vegas, NV, USA.
Stern RA; Boston University CTE Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA. .; Boston University Alzheimer's Disease Research Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA. .; Department of Neurology, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA. .; Departments of Neurosurgery, and Anatomy and Neurobiology, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA. .
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Corporate Authors:
DIAGNOSE CTE Research Project Investigators
Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 09; Vol. 21 (1), pp. 46. Date of Electronic Publication: 2024 Feb 09.
Typ publikacji:
Journal Article
MeSH Terms:
Chronic Traumatic Encephalopathy*/pathology
Football*
Brain Injuries, Traumatic*
Humans ; Aged ; Middle Aged ; Interleukin-6 ; Biomarkers
Czasopismo naukowe

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