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Wyświetlanie 1-20 z 28
Tytuł:
EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.
Autorzy:
Styles CT; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Bazot Q; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Parker GA; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
White RE; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Paschos K; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Allday MJ; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
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Źródło:
PLoS biology [PLoS Biol] 2017 Aug 03; Vol. 15 (8), pp. e2001992. Date of Electronic Publication: 2017 Aug 03 (Print Publication: 2017).
Typ publikacji:
Journal Article
MeSH Terms:
Virus Latency*
B-Lymphocytes/*virology
Epstein-Barr Virus Infections/*immunology
Herpesvirus 4, Human/*physiology
Viral Proteins/*physiology
B-Lymphocytes/physiology ; Biomarkers/metabolism ; Cell Differentiation ; Cell Line, Tumor ; Cyclin-Dependent Kinase Inhibitor p18/metabolism ; Histone Code ; Humans ; Immunoglobulins/metabolism ; Plasma Cells/metabolism ; Positive Regulatory Domain I-Binding Factor 1 ; Repressor Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.
Autorzy:
Kalchschmidt JS; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Gillman AC; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Paschos K; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Bazot Q; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Kempkes B; Department of Gene Vectors, Hematologikum, Helmholtz Zentrum München, National Research Center for Environmental Health, München, Germany.
Allday MJ; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
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Źródło:
PLoS pathogens [PLoS Pathog] 2016 Jan 11; Vol. 12 (1), pp. e1005383. Date of Electronic Publication: 2016 Jan 11 (Print Publication: 2016).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
B-Lymphocytes/*virology
Epstein-Barr Virus Infections/*genetics
Epstein-Barr Virus Nuclear Antigens/*genetics
Gene Expression Regulation, Viral/*genetics
Immunoglobulin J Recombination Signal Sequence-Binding Protein/*genetics
Cells, Cultured ; Chromatin/genetics ; Chromatin Immunoprecipitation ; Host-Parasite Interactions/genetics ; Humans ; Reverse Transcriptase Polymerase Chain Reaction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.
Autorzy:
Bazot Q; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Paschos K; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Skalska L; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Kalchschmidt JS; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Parker GA; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
Allday MJ; Molecular Virology, Department of Medicine, Imperial College London, London, United Kingdom.
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Źródło:
PLoS pathogens [PLoS Pathog] 2015 Jul 08; Vol. 11 (7), pp. e1005031. Date of Electronic Publication: 2015 Jul 08 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Transformation, Neoplastic/*genetics
Epstein-Barr Virus Nuclear Antigens/*genetics
MicroRNAs/*genetics
B-Lymphocytes/virology ; Blotting, Western ; Chromatin Immunoprecipitation ; Cyclin-Dependent Kinase Inhibitor p57/biosynthesis ; Epstein-Barr Virus Nuclear Antigens/metabolism ; Flow Cytometry ; Gene Expression Regulation, Neoplastic/genetics ; Humans ; Immunoprecipitation ; MicroRNAs/biosynthesis ; Oncogenes ; Real-Time Polymerase Chain Reaction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.
Autorzy:
Skalska L; Section of Virology, Department of Medicine, Imperial College London, St Mary's Campus, London, United Kingdom.
White RE
Parker GA
Turro E; Department of Oncology, University of Cambridge, Hills Road, Cambridge CB2 0XZ, United Kingdom and Cancer Research UK, Cambridge Research Institute, Robinson Way, Cambridge CB2 0RE, United Kingdom
Sinclair AJ
Paschos K
Allday MJ
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Źródło:
PLoS pathogens [PLoS Pathog] 2013 Feb; Vol. 9 (2), pp. e1003187. Date of Electronic Publication: 2013 Feb 21.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Lymphocyte Activation*
B-Lymphocytes/*virology
Cyclin-Dependent Kinase Inhibitor p16/*genetics
Epigenetic Repression/*genetics
Herpesvirus 4, Human/*physiology
Antigens, Viral/genetics ; Antigens, Viral/metabolism ; Cell Line ; Cell Proliferation ; Cell Survival ; Cyclin-Dependent Kinase Inhibitor p16/metabolism ; Epstein-Barr Virus Infections/immunology ; Epstein-Barr Virus Infections/virology ; Epstein-Barr Virus Nuclear Antigens/genetics ; Epstein-Barr Virus Nuclear Antigens/metabolism ; Genetic Loci ; Herpesvirus 4, Human/immunology ; Humans ; Oligonucleotide Array Sequence Analysis ; Phosphorylation ; Primary Cell Culture ; Virus Latency
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Latent Epstein-Barr virus can inhibit apoptosis in B cells by blocking the induction of NOXA expression.
Autorzy:
Yee J; Section of Virology, Division of Infectious Diseases, Faculty of Medicine, Imperial College London, London, United Kingdom.
White RE
Anderton E
Allday MJ
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Źródło:
PloS one [PLoS One] 2011; Vol. 6 (12), pp. e28506. Date of Electronic Publication: 2011 Dec 09.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/drug effects
Virus Latency*
B-Lymphocytes/*pathology
B-Lymphocytes/*virology
Herpesvirus 4, Human/*physiology
Proto-Oncogene Proteins c-bcl-2/*metabolism
B-Lymphocytes/drug effects ; Cell Line ; Drug Resistance, Viral/drug effects ; Etoposide/pharmacology ; Gene Deletion ; Gene Expression Regulation, Viral/drug effects ; Gene Knockout Techniques ; Genetic Loci/genetics ; Herpesvirus 4, Human/drug effects ; Herpesvirus 4, Human/genetics ; Humans ; Ionomycin/pharmacology ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; RNA, Small Interfering/metabolism ; Reproducibility of Results ; Signal Transduction/drug effects ; Staurosporine/pharmacology ; Viral Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification.
Autorzy:
White RE; Section of Virology, Imperial College London, London, United Kingdom.
Groves IJ
Turro E
Yee J
Kremmer E
Allday MJ
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Źródło:
PloS one [PLoS One] 2010 Nov 15; Vol. 5 (11), pp. e13979. Date of Electronic Publication: 2010 Nov 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Profiling*
Antigens, Viral/*genetics
Chromatin/*metabolism
Antigens, Viral/physiology ; Cell Line, Tumor ; Cluster Analysis ; Epigenomics ; Epstein-Barr Virus Nuclear Antigens/genetics ; Epstein-Barr Virus Nuclear Antigens/physiology ; Gene Knockout Techniques ; HEK293 Cells ; Herpesvirus 4, Human/genetics ; Herpesvirus 4, Human/physiology ; Host-Pathogen Interactions/genetics ; Humans ; Oligonucleotide Array Sequence Analysis ; Reverse Transcriptase Polymerase Chain Reaction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP.
Autorzy:
Skalska L; Section of Virology, Division of Infectious Diseases, Faculty of Medicine, Imperial College London, London, United Kingdom.
White RE
Franz M
Ruhmann M
Allday MJ
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Źródło:
PLoS pathogens [PLoS Pathog] 2010 Jun 10; Vol. 6 (6), pp. e1000951. Date of Electronic Publication: 2010 Jun 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epigenesis, Genetic*
Alcohol Oxidoreductases/*metabolism
Antigens, Viral/*metabolism
Cyclin-Dependent Kinase Inhibitor p16/*genetics
DNA-Binding Proteins/*metabolism
Epstein-Barr Virus Infections/*metabolism
Epstein-Barr Virus Nuclear Antigens/*metabolism
Herpesvirus 4, Human/*metabolism
Alcohol Oxidoreductases/genetics ; Antigens, Viral/genetics ; B-Lymphocytes/metabolism ; B-Lymphocytes/pathology ; Blotting, Western ; Cell Proliferation ; Chromatin Assembly and Disassembly ; Chromatin Immunoprecipitation ; Cyclin-Dependent Kinase Inhibitor p16/metabolism ; DNA Methylation ; DNA-Binding Proteins/genetics ; Epstein-Barr Virus Infections/genetics ; Epstein-Barr Virus Nuclear Antigens/genetics ; Flow Cytometry ; Gene Expression Regulation ; Herpesvirus 4, Human/genetics ; Humans ; RNA, Messenger/genetics ; Reverse Transcriptase Polymerase Chain Reaction ; Transcriptional Activation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Epstein-barr virus latency in B cells leads to epigenetic repression and CpG methylation of the tumour suppressor gene Bim.
Autorzy:
Paschos K; Department of Virology, Faculty of Medicine, Imperial College London, London, United Kingdom.
Smith P
Anderton E
Middeldorp JM
White RE
Allday MJ
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Źródło:
PLoS pathogens [PLoS Pathog] 2009 Jun; Vol. 5 (6), pp. e1000492. Date of Electronic Publication: 2009 Jun 26.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
DNA Methylation*
Apoptosis Regulatory Proteins/*genetics
B-Lymphocytes/*physiology
B-Lymphocytes/*virology
Herpesvirus 4, Human/*physiology
Membrane Proteins/*genetics
Proto-Oncogene Proteins/*genetics
Virus Latency/*physiology
Acetylation ; Apoptosis Regulatory Proteins/biosynthesis ; Apoptosis Regulatory Proteins/metabolism ; B-Lymphocytes/metabolism ; Bcl-2-Like Protein 11 ; Cell Line, Tumor ; CpG Islands ; DNA (Cytosine-5-)-Methyltransferases/metabolism ; Epigenesis, Genetic ; Gene Expression ; Genes, Tumor Suppressor ; Histone Deacetylases/metabolism ; Histones/metabolism ; Humans ; Membrane Proteins/biosynthesis ; Membrane Proteins/metabolism ; Polymerase Chain Reaction ; Promoter Regions, Genetic ; Proteasome Endopeptidase Complex/metabolism ; Proto-Oncogene Proteins/biosynthesis ; Proto-Oncogene Proteins/metabolism ; RNA Stability ; Sequence Analysis, DNA ; Tumor Cells, Cultured
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
TGF-beta induces apoptosis in human B cells by transcriptional regulation of BIK and BCL-XL.
Autorzy:
Spender LC; Growth Factor Signalling Laboratory, The Beatson Institute for Cancer Research, Garscube Estate, Switchback Road, Bearsden, Glasgow, UK. />O'Brien DI
Simpson D
Dutt D
Gregory CD
Allday MJ
Clark LJ
Inman GJ
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Źródło:
Cell death and differentiation [Cell Death Differ] 2009 Apr; Vol. 16 (4), pp. 593-602. Date of Electronic Publication: 2009 Jan 09.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Apoptosis Regulatory Proteins/*metabolism
B-Lymphocytes/*drug effects
Gene Expression Regulation/*drug effects
Membrane Proteins/*metabolism
Transforming Growth Factor beta/*pharmacology
bcl-X Protein/*metabolism
Apoptosis Regulatory Proteins/genetics ; B-Lymphocytes/cytology ; B-Lymphocytes/metabolism ; Cell Line, Tumor ; Cells, Cultured ; Electrophoretic Mobility Shift Assay ; Flow Cytometry ; Humans ; Immunoblotting ; Immunohistochemistry ; In Vitro Techniques ; Membrane Proteins/genetics ; Mitochondrial Proteins ; Reverse Transcriptase Polymerase Chain Reaction ; bcl-X Protein/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The curious case of the tumour virus: 50 years of Burkitt's lymphoma.
Autorzy:
Thorley-Lawson DA; Department of Pathology, Jaharis Building, Tufts University School of Medicine, 150 Harrison Avenue, Boston, Massachusetts 02111, USA. />Allday MJ
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Źródło:
Nature reviews. Microbiology [Nat Rev Microbiol] 2008 Dec; Vol. 6 (12), pp. 913-24.
Typ publikacji:
Historical Article; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Burkitt Lymphoma/*virology
Africa ; Apoptosis ; Burkitt Lymphoma/etiology ; Burkitt Lymphoma/history ; Epstein-Barr Virus Nuclear Antigens/genetics ; Genes, myc ; Herpesvirus 4, Human/genetics ; Herpesvirus 4, Human/isolation & purification ; Herpesvirus 4, Human/pathogenicity ; History, 20th Century ; History, 21st Century ; Humans ; Malaria/complications ; Malaria/history ; Malaria/immunology ; Models, Biological ; Receptors, Notch/physiology ; Signal Transduction ; Translocation, Genetic
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Wyświetlanie 1-20 z 28

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