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Tytuł :
Compartment-specific transcriptomics of ozone-exposed murine lungs reveals sex- and cell type-associated perturbations relevant to mucoinflammatory lung diseases.
Autorzy :
Choudhary I; Department of Comparative Biomedical Sciences, School of Veterinary Medicine, Louisiana State University, Baton Rouge, Louisiana.
Vo T; Department of Comparative Biomedical Sciences, School of Veterinary Medicine, Louisiana State University, Baton Rouge, Louisiana.
Paudel K; Department of Comparative Biomedical Sciences, School of Veterinary Medicine, Louisiana State University, Baton Rouge, Louisiana.
Patial S; Department of Comparative Biomedical Sciences, School of Veterinary Medicine, Louisiana State University, Baton Rouge, Louisiana.
Saini Y; Department of Comparative Biomedical Sciences, School of Veterinary Medicine, Louisiana State University, Baton Rouge, Louisiana.
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Źródło :
American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2021 Jan 01; Vol. 320 (1), pp. L99-L125. Date of Electronic Publication: 2020 Oct 07.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Epithelial Cells/*metabolism
Lung Diseases/*genetics
Macrophages, Alveolar/*metabolism
Mucociliary Clearance/*genetics
Ozone/*toxicity
Pneumonia/*genetics
Transcriptome/*drug effects
Animals ; Epithelial Cells/drug effects ; Epithelial Cells/pathology ; Female ; Lung Diseases/chemically induced ; Lung Diseases/pathology ; Macrophages, Alveolar/drug effects ; Macrophages, Alveolar/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Mucociliary Clearance/drug effects ; Pneumonia/chemically induced ; Pneumonia/pathology
Czasopismo naukowe
Tytuł :
Human coronaviruses 229E and OC43 replicate and induce distinct antiviral responses in differentiated primary human bronchial epithelial cells.
Autorzy :
Loo SL; Viral Immunology and Respiratory Disease group, University of Newcastle, Newcastle, New South Wales, Australia.; Priority Research Centre for Healthy Lungs, University of Newcastle and Hunter Medical Research Institute, Newcastle, New South Wales, Australia.
Wark PAB; Priority Research Centre for Healthy Lungs, University of Newcastle and Hunter Medical Research Institute, Newcastle, New South Wales, Australia.; Department of Respiratory and Sleep Medicine, John Hunter Hospital, Newcastle, New South Wales, Australia.
Esneau C; Viral Immunology and Respiratory Disease group, University of Newcastle, Newcastle, New South Wales, Australia.; Priority Research Centre for Healthy Lungs, University of Newcastle and Hunter Medical Research Institute, Newcastle, New South Wales, Australia.
Nichol KS; Priority Research Centre for Healthy Lungs, University of Newcastle and Hunter Medical Research Institute, Newcastle, New South Wales, Australia.
Hsu AC; Priority Research Centre for Healthy Lungs, University of Newcastle and Hunter Medical Research Institute, Newcastle, New South Wales, Australia.
Bartlett NW; Viral Immunology and Respiratory Disease group, University of Newcastle, Newcastle, New South Wales, Australia.; Priority Research Centre for Healthy Lungs, University of Newcastle and Hunter Medical Research Institute, Newcastle, New South Wales, Australia.
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Źródło :
American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2020 Dec 01; Vol. 319 (6), pp. L926-L931. Date of Electronic Publication: 2020 Sep 09.
Typ publikacji :
Journal Article
MeSH Terms :
Antiviral Agents/*pharmacology
Bronchi/*immunology
Coronavirus 229E, Human/*immunology
Coronavirus Infections/*immunology
Epithelial Cells/*immunology
Virus Replication/*drug effects
Bronchi/drug effects ; Bronchi/virology ; Cells, Cultured ; Coronavirus 229E, Human/drug effects ; Coronavirus Infections/drug therapy ; Coronavirus Infections/virology ; Epithelial Cells/drug effects ; Epithelial Cells/virology ; Humans ; Interferons/metabolism
Czasopismo naukowe
Tytuł :
E-cigarette constituents propylene glycol and vegetable glycerin decrease glucose uptake and its metabolism in airway epithelial cells in vitro.
Autorzy :
Woodall M; Institute for Infection and Immunity, St George's, University of London, Tooting, London, United Kingdom.
Jacob J; Institute for Infection and Immunity, St George's, University of London, Tooting, London, United Kingdom.
Kalsi KK; Institute for Infection and Immunity, St George's, University of London, Tooting, London, United Kingdom.
Schroeder V; Immunology and Respiratory Diseases Research, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
Davis E; Marsico Lung Institute and Department of Cell Biology and Physiology, University of North Carolina, Chapel Hill, North Carolina.
Kenyon B; Marsico Lung Institute and Department of Cell Biology and Physiology, University of North Carolina, Chapel Hill, North Carolina.
Khan I; Institute for Infection and Immunity, St George's, University of London, Tooting, London, United Kingdom.
Garnett JP; Immunology and Respiratory Diseases Research, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
Tarran R; Marsico Lung Institute and Department of Cell Biology and Physiology, University of North Carolina, Chapel Hill, North Carolina.
Baines DL; Institute for Infection and Immunity, St George's, University of London, Tooting, London, United Kingdom.
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Źródło :
American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2020 Dec 01; Vol. 319 (6), pp. L957-L967. Date of Electronic Publication: 2020 Sep 30.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Electronic Nicotine Delivery Systems*
Epithelial Cells/*drug effects
Glucose/*metabolism
Respiratory System/*drug effects
Biological Transport/drug effects ; Biological Transport/physiology ; Glycerol/pharmacology ; Humans ; Propylene Glycol/pharmacology
Czasopismo naukowe
Tytuł :
AMP-activated protein kinase contributes to cisplatin-induced renal epithelial cell apoptosis and acute kidney injury.
Autorzy :
Jin X; Section of Nephrology, Department of Medicine, Baylor College of Medicine, Houston, Texas.; Department of Anesthesiology, The Affiliated Hospital of Guilin Medical University, Guilin, Guangxi, China.
An C; Section of Nephrology, Department of Medicine, Baylor College of Medicine, Houston, Texas.; Division of Nephrology, Department of Medicine, University of Connecticut School of Medicine, Farmington, Connecticut.
Jiao B; Division of Nephrology, Department of Medicine, University of Connecticut School of Medicine, Farmington, Connecticut.
Safirstein RL; Renal Section, Veterans Affairs Connecticut Healthcare System, West Haven, Connecticut.
Wang Y; Section of Nephrology, Department of Medicine, Baylor College of Medicine, Houston, Texas.; Division of Nephrology, Department of Medicine, University of Connecticut School of Medicine, Farmington, Connecticut.; Renal Section, Veterans Affairs Connecticut Healthcare System, West Haven, Connecticut.; Department of Cell Biology, University of Connecticut School of Medicine, Farmington, Connecticut.; Institute for Systems Genomics, University of Connecticut School of Medicine, Farmington, Connecticut.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Dec 01; Vol. 319 (6), pp. F1073-F1080. Date of Electronic Publication: 2020 Oct 26.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
AMP-Activated Protein Kinases/*metabolism
Acute Kidney Injury/*chemically induced
Apoptosis/*drug effects
Cisplatin/*toxicity
Epithelial Cells/*drug effects
Kidney Tubules/*drug effects
AMP-Activated Protein Kinases/genetics ; Acute Kidney Injury/enzymology ; Acute Kidney Injury/pathology ; Animals ; Caspase 3/metabolism ; Cell Line ; Epithelial Cells/enzymology ; Epithelial Cells/pathology ; Kidney Tubules/enzymology ; Kidney Tubules/pathology ; Mice ; Phosphorylation ; Signal Transduction ; Tumor Suppressor Protein p53/metabolism ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
Silencing of the lncRNA TUG1 attenuates the epithelial-mesenchymal transition of renal tubular epithelial cells by sponging miR-141-3p via regulating β-catenin.
Autorzy :
Zhang B; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.
Zhao C; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.
Hou L; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.
Wu Y; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Dec 01; Vol. 319 (6), pp. F1125-F1134. Date of Electronic Publication: 2020 Nov 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Epithelial-Mesenchymal Transition*
Epithelial Cells/*metabolism
Kidney Diseases/*metabolism
Kidney Tubules/*metabolism
MicroRNAs/*metabolism
RNA, Long Noncoding/*metabolism
beta Catenin/*metabolism
Animals ; Cell Line ; Cell Movement ; Cell Proliferation ; Disease Models, Animal ; Epithelial Cells/pathology ; Fibrosis ; Humans ; Kidney Diseases/etiology ; Kidney Diseases/genetics ; Kidney Diseases/pathology ; Kidney Tubules/pathology ; Mice, Inbred BALB C ; MicroRNAs/genetics ; RNA Interference ; RNA, Long Noncoding/genetics ; Signal Transduction ; Ureteral Obstruction/complications ; beta Catenin/genetics
Czasopismo naukowe
Tytuł :
EpCAM mark epithelial progenitor cells in postnatal human lung and are associated with pathogenesis of pulmonary disease including lung adenocarcinoma.
Autorzy :
Wang L; Division of General Thoracic Surgery, Inselspital, Bern University Hospital, Bern, Switzerland.; Department of BioMedical Research, University of Bern, Switzerland.
Dorn P; Division of General Thoracic Surgery, Inselspital, Bern University Hospital, Bern, Switzerland.
Simillion C; Interfaculty Bioinformatics, University of Bern, Switzerland.
Froment L; Division of General Thoracic Surgery, Inselspital, Bern University Hospital, Bern, Switzerland.; Department of BioMedical Research, University of Bern, Switzerland.
Berezowska S; Institute of Pathology, University of Bern, Bern, Switzerland.
Tschanz SA; Institute of Anatomy, University of Bern, Switzerland.
Haenni B; Institute of Anatomy, University of Bern, Switzerland.
Blank F; Department of BioMedical Research, University of Bern, Switzerland.; DCR Live Imaging Core, University of Bern, Switzerland.
Wotzkow C; DCR Live Imaging Core, University of Bern, Switzerland.
Peng RW; Division of General Thoracic Surgery, Inselspital, Bern University Hospital, Bern, Switzerland.; Department of BioMedical Research, University of Bern, Switzerland.
Marti TM; Division of General Thoracic Surgery, Inselspital, Bern University Hospital, Bern, Switzerland.; Department of BioMedical Research, University of Bern, Switzerland.
Bode PK; Department of Pathology and Molecular Pathology, University Hospital Zurich, Switzerland.
Moehrlen U; Department of Pediatric Surgery, University Children's Hospital, Zurich, Switzerland.
Schmid RA; Division of General Thoracic Surgery, Inselspital, Bern University Hospital, Bern, Switzerland.; Department of BioMedical Research, University of Bern, Switzerland.
Hall SRR; Division of General Thoracic Surgery, Inselspital, Bern University Hospital, Bern, Switzerland.; Department of BioMedical Research, University of Bern, Switzerland.
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Źródło :
American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2020 Nov 01; Vol. 319 (5), pp. L794-L809. Date of Electronic Publication: 2020 Jul 29.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
5'-Nucleotidase/*metabolism
Carcinoma, Non-Small-Cell Lung/*metabolism
Epithelial Cells/*metabolism
Stem Cells/*cytology
Animals ; Cell Differentiation/physiology ; Epithelial Cell Adhesion Molecule/metabolism ; Humans ; Lung/pathology ; Lung Neoplasms/metabolism ; Mesenchymal Stem Cells/metabolism ; Mice
Czasopismo naukowe
Tytuł :
Sprr2f protects against renal injury by decreasing the level of reactive oxygen species in female mice.
Autorzy :
Huynh KM; Department of Urology, School of Medicine, Stanford University, Stanford, California.
Wong AC; Department of Urology, School of Medicine, Stanford University, Stanford, California.
Wu B; Department of Urology, School of Medicine, Stanford University, Stanford, California.
Horschman M; Department of Urology, School of Medicine, Stanford University, Stanford, California.
Zhao H; Department of Urology, School of Medicine, Stanford University, Stanford, California.
Brooks JD; Department of Urology, School of Medicine, Stanford University, Stanford, California.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Nov 01; Vol. 319 (5), pp. F876-F884. Date of Electronic Publication: 2020 Oct 05.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Cornified Envelope Proline-Rich Proteins/*metabolism
Epithelial Cells/*metabolism
Kidney/*metabolism
Reactive Oxygen Species/*metabolism
Animals ; Cornified Envelope Proline-Rich Proteins/genetics ; Disease Models, Animal ; Female ; Kidney Tubules/metabolism ; Mice, Knockout ; Renal Insufficiency, Chronic/metabolism ; Renal Insufficiency, Chronic/pathology ; Reperfusion Injury/metabolism ; Reperfusion Injury/pathology ; Ureteral Obstruction/pathology
Czasopismo naukowe
Tytuł :
Retromer is involved in epithelial Na channel trafficking.
Autorzy :
Cheung TT; Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand.
Geda AC; Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand.
Ware AW; Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand.
Rasulov SR; Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand.
Tenci P; Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand.
Hamilton KL; Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand.
McDonald FJ; Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, New Zealand.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Nov 01; Vol. 319 (5), pp. F895-F907. Date of Electronic Publication: 2020 Oct 05.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Endosomes/*metabolism
Epithelial Cells/*metabolism
Epithelial Sodium Channels/*metabolism
Protein Transport/*physiology
Cell Membrane/metabolism ; Cell Movement/physiology ; Endocytosis/physiology ; Epithelial Cells/physiology ; Humans ; Sodium/metabolism
Czasopismo naukowe
Tytuł :
Involvement of the CDKL5-SOX9 signaling axis in rhabdomyolysis-associated acute kidney injury.
Autorzy :
Kim JY; Division of Pharmaceutics and Pharmacology, College of Pharmacy and Comprehensive Cancer Center, The Ohio State University, Columbus, Ohio.
Bai Y; Division of Pharmaceutics and Pharmacology, College of Pharmacy and Comprehensive Cancer Center, The Ohio State University, Columbus, Ohio.
Jayne LA; Division of Pharmaceutics and Pharmacology, College of Pharmacy and Comprehensive Cancer Center, The Ohio State University, Columbus, Ohio.
Cianciolo RE; Department of Veterinary Biosciences, College of Veterinary Medicine, The Ohio State University, Columbus, Ohio.
Bajwa A; Transplant Research Institute, James D. Eason Transplant Institute, Department of Surgery, College of Medicine, The University of Tennessee Health Science Center, Memphis, Tennessee.
Pabla NS; Division of Pharmaceutics and Pharmacology, College of Pharmacy and Comprehensive Cancer Center, The Ohio State University, Columbus, Ohio.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Nov 01; Vol. 319 (5), pp. F920-F929. Date of Electronic Publication: 2020 Oct 12.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Acute Kidney Injury/*metabolism
Epithelial Cells/*metabolism
Kidney Tubules/*metabolism
Protein-Serine-Threonine Kinases/*metabolism
SOX9 Transcription Factor/*metabolism
Acute Kidney Injury/pathology ; Cell Death/physiology ; Humans ; Kidney/metabolism ; Phosphorylation ; Rhabdomyolysis/chemically induced ; Signal Transduction/physiology
Czasopismo naukowe
Tytuł :
Soluble (pro)renin receptor promotes the fibrotic response in renal proximal tubule epithelial cells in vitro via the Akt/β-catenin/Snail signaling pathway.
Autorzy :
Xie S; Institute of Hypertension, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.; Department of Internal Medicine, University of Utah and Veterans Affairs Medical Center, Salt Lake City, Utah.
Su J; Institute of Hypertension, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Lu A; Institute of Hypertension, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Lai Y; Institute of Hypertension, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Mo S; Institute of Hypertension, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Pu M; Institute of Hypertension, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Yang T; Institute of Hypertension, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.; Department of Internal Medicine, University of Utah and Veterans Affairs Medical Center, Salt Lake City, Utah.; The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Nov 01; Vol. 319 (5), pp. F941-F953. Date of Electronic Publication: 2020 Aug 31.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Epithelial Cells/*metabolism
Kidney Tubules, Proximal/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
beta Catenin/*metabolism
Humans ; Kidney/metabolism ; Receptors, Cell Surface/metabolism ; Renal Insufficiency, Chronic/metabolism ; Renal Insufficiency, Chronic/pathology ; Signal Transduction/physiology ; Up-Regulation/physiology
Czasopismo naukowe
Tytuł :
Noncanonical role for Ku70/80 in the prevention of allergic airway inflammation via maintenance of airway epithelial cell organelle homeostasis.
Autorzy :
Rehman R; Molecular Pathobiology of Respiratory Diseases, Council of Scientific and Industrial Research-Institute of Genomics and Integrative Biology, Delhi, India.; Academy of Scientific and Innovative Research, Ghaziabad, India.; Division of Pulmonary and Critical Care Medicine, Department of Medicine, Brigham and Women's Hospital, Boston, Massachusetts.
Vijayakumar VE; Molecular Pathobiology of Respiratory Diseases, Council of Scientific and Industrial Research-Institute of Genomics and Integrative Biology, Delhi, India.
Jaiswal A; Molecular Pathobiology of Respiratory Diseases, Council of Scientific and Industrial Research-Institute of Genomics and Integrative Biology, Delhi, India.; Academy of Scientific and Innovative Research, Ghaziabad, India.; Molecular Pathobiology of Respiratory Diseases, Cell Biology and Physiology Division, Council of Scientific and Industrial Research-Indian Institute of Chemical Biology, Kolkata, India.
Jain V; Molecular Pathobiology of Respiratory Diseases, Council of Scientific and Industrial Research-Institute of Genomics and Integrative Biology, Delhi, India.; Academy of Scientific and Innovative Research, Ghaziabad, India.
Mukherjee S; Molecular Pathobiology of Respiratory Diseases, Council of Scientific and Industrial Research-Institute of Genomics and Integrative Biology, Delhi, India.; Academy of Scientific and Innovative Research, Ghaziabad, India.
Vellarikkal SK; Academy of Scientific and Innovative Research, Ghaziabad, India.; Broad Institute of the Massachusetts Institute of Technology and Harvard University, Cambridge, Massachusetts.
Dieffenbach PB; Division of Pulmonary and Critical Care Medicine, Department of Medicine, Brigham and Women's Hospital, Boston, Massachusetts.
Fredenburgh LE; Division of Pulmonary and Critical Care Medicine, Department of Medicine, Brigham and Women's Hospital, Boston, Massachusetts.
Prakash YS; Departments of Anesthesiology and Physiology and Biomedical Engineering, Mayo Clinic, Rochester, Minnesota.
Ghosh B; Molecular Pathobiology of Respiratory Diseases, Council of Scientific and Industrial Research-Institute of Genomics and Integrative Biology, Delhi, India.; Academy of Scientific and Innovative Research, Ghaziabad, India.
Agrawal A; Molecular Pathobiology of Respiratory Diseases, Council of Scientific and Industrial Research-Institute of Genomics and Integrative Biology, Delhi, India.; Academy of Scientific and Innovative Research, Ghaziabad, India.
Mabalirajan U; Molecular Pathobiology of Respiratory Diseases, Council of Scientific and Industrial Research-Institute of Genomics and Integrative Biology, Delhi, India.; Academy of Scientific and Innovative Research, Ghaziabad, India.; Molecular Pathobiology of Respiratory Diseases, Cell Biology and Physiology Division, Council of Scientific and Industrial Research-Indian Institute of Chemical Biology, Kolkata, India.
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Źródło :
American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2020 Oct 01; Vol. 319 (4), pp. L728-L741. Date of Electronic Publication: 2020 Sep 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Epithelial Cells/*metabolism
Homeostasis/*physiology
Inflammation/*prevention & control
Ku Autoantigen/*metabolism
Animals ; Asthma/pathology ; Asthma/prevention & control ; Endoplasmic Reticulum Stress/physiology ; Epithelial Cells/pathology ; Humans ; Inflammation/metabolism ; Lung/metabolism ; Lung/pathology ; Mice ; Oxidative Stress/physiology ; Respiratory Hypersensitivity/pathology
Czasopismo naukowe
Tytuł :
Prenatal smoke exposure dysregulates lung epithelial cell differentiation in mouse offspring: role for AREG-induced EGFR signaling.
Autorzy :
Lkhagvadorj K; Department of Pathology and Medical Biology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Groningen Research Institute for Asthma and COPD, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Department of Pulmonology and Allergology, Mongolian National University of Medical Sciences, Ulaanbaatar, Mongolia.
Zeng Z; Department of Pathology and Medical Biology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Groningen Research Institute for Asthma and COPD, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Song J; Department of Pathology and Medical Biology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Groningen Research Institute for Asthma and COPD, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Reinders-Luinge M; Department of Pathology and Medical Biology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Groningen Research Institute for Asthma and COPD, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Kooistra W; Department of Pathology and Medical Biology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Groningen Research Institute for Asthma and COPD, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Song S; Groningen Research Institute for Asthma and COPD, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Department of Molecular Pharmacology, Groningen Research Institute for Pharmacy, University of Groningen, Groningen, The Netherlands.
Krauss-Etschmann S; Research Center Borstel, Leibniz Lung Center, Christian-Albrechts-University, Kiel, Germany.
Melgert BN; Groningen Research Institute for Asthma and COPD, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Department of Molecular Pharmacology, Groningen Research Institute for Pharmacy, University of Groningen, Groningen, The Netherlands.
Cao J; Laboratory of Environmental Medicine and Developmental Toxicology, Shantou University Medical College, Shantou, China.
Hylkema MN; Department of Pathology and Medical Biology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Groningen Research Institute for Asthma and COPD, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
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Źródło :
American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2020 Oct 01; Vol. 319 (4), pp. L742-L751. Date of Electronic Publication: 2020 Aug 12.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Amphiregulin/*metabolism
Amphiregulin/*pharmacology
Epithelial Cells/*drug effects
ErbB Receptors/*metabolism
Smoke/*adverse effects
Animals ; Animals, Newborn ; Cell Differentiation/drug effects ; Cell Differentiation/physiology ; Epidermal Growth Factor/metabolism ; Epithelial Cells/metabolism ; Mice ; Signal Transduction/drug effects ; Signal Transduction/physiology ; Tobacco/adverse effects
Czasopismo naukowe
Tytuł :
Intratubular epithelial-mesenchymal transition and tubular atrophy after kidney injury in mice.
Autorzy :
Yamashita N; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Kusaba T; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Nakata T; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Tomita A; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Ida T; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Watanabe-Uehara N; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Ikeda K; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Kitani T; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Uehara M; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Kirita Y; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Matoba S; Department of Cardiovascular Medicine, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Humphreys BD; Division of Nephrology, Washington University School of Medicine in St. Louis, St. Louis, Missouri.
Tamagaki K; Department of Nephrology, Graduate School of Medical Science, Kyoto Prefectural University of Medicine, Kyoto, Japan.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Oct 01; Vol. 319 (4), pp. F579-F591. Date of Electronic Publication: 2020 Aug 17.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Epithelial-Mesenchymal Transition*/drug effects
Acute Kidney Injury/*pathology
Epithelial Cells/*pathology
Kidney Tubules, Proximal/*pathology
Acute Kidney Injury/drug therapy ; Acute Kidney Injury/metabolism ; Animals ; Atrophy ; Cell Line ; Cell Proliferation ; Disease Models, Animal ; Enzyme Inhibitors/pharmacology ; Epithelial Cells/drug effects ; Epithelial Cells/metabolism ; Fibrosis ; Focal Adhesion Kinase 1/antagonists & inhibitors ; Focal Adhesion Kinase 1/metabolism ; Kidney Tubules, Proximal/drug effects ; Kidney Tubules, Proximal/metabolism ; Male ; Mice, Transgenic ; Phenotype ; Phosphorylation ; Rats ; Sodium-Phosphate Cotransporter Proteins, Type IIa/genetics ; Sodium-Phosphate Cotransporter Proteins, Type IIa/metabolism
Czasopismo naukowe
Tytuł :
Expression of Acsm2 , a kidney-specific gene, parallels the function and maturation of proximal tubular cells.
Autorzy :
Watanabe H; Department of Pediatrics, Child Health Research Center, University of Virginia School of Medicine, Charlottesville, Virginia.
Paxton RL; Department of Pediatrics, Child Health Research Center, University of Virginia School of Medicine, Charlottesville, Virginia.
Tolerico MR; Department of Biology, University of Virginia, Charlottesville, Virginia.
Nagalakshmi VK; Department of Pediatrics, Child Health Research Center, University of Virginia School of Medicine, Charlottesville, Virginia.
Tanaka S; Division of Nephrology and Center for Immunity, Inflammation, and Regenerative Medicine, University of Virginia, Charlottesville, Virginia.
Okusa MD; Division of Nephrology and Center for Immunity, Inflammation, and Regenerative Medicine, University of Virginia, Charlottesville, Virginia.
Goto S; Division of Clinical Nephrology and Rheumatology, Kidney Research Center, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.
Narita I; Division of Clinical Nephrology and Rheumatology, Kidney Research Center, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.
Watanabe S; Department of Pediatrics, Izu Medical and Welfare Center, Shizuoka, Japan.
Sequeira-Lοpez MLS; Department of Pediatrics, Child Health Research Center, University of Virginia School of Medicine, Charlottesville, Virginia.
Gomez RA; Department of Pediatrics, Child Health Research Center, University of Virginia School of Medicine, Charlottesville, Virginia.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Oct 01; Vol. 319 (4), pp. F603-F611. Date of Electronic Publication: 2020 Aug 24.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Coenzyme A Ligases/*metabolism
Epithelial Cells/*metabolism
Kidney Tubules, Proximal/*metabolism
Mitochondrial Proteins/*metabolism
Acute Kidney Injury/enzymology ; Acute Kidney Injury/genetics ; Acute Kidney Injury/pathology ; Animals ; Coenzyme A Ligases/genetics ; Disease Models, Animal ; Epithelial Cells/pathology ; Fibrosis ; Gene Expression Regulation, Developmental ; Gene Expression Regulation, Enzymologic ; Humans ; Integrin beta1/genetics ; Integrin beta1/metabolism ; Kidney Tubules, Proximal/pathology ; Male ; Mice, Inbred C57BL ; Mice, Knockout ; Mitochondrial Proteins/genetics ; Renal Insufficiency, Chronic/enzymology ; Renal Insufficiency, Chronic/genetics ; Renal Insufficiency, Chronic/pathology ; Renin/genetics ; Renin/metabolism ; Reperfusion Injury/enzymology ; Reperfusion Injury/genetics ; Reperfusion Injury/pathology
Czasopismo naukowe
Tytuł :
MAD2B contributes to parietal epithelial cell activation and crescentic glomerulonephritis via Skp2.
Autorzy :
Ye C; Department of Nephrology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Xiong W; Department of Nephrology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Lei CT; Department of Nephrology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Tang H; Department of Nephrology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Su H; Department of Nephrology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Yi F; Department of Pharmacology, School of Basic Medical Science, Shandong University, Jinan, China.
Zhang C; Department of Nephrology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Oct 01; Vol. 319 (4), pp. F636-F646. Date of Electronic Publication: 2020 Aug 24.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Proliferation*/drug effects
Epithelial Cells/*metabolism
Glomerulonephritis/*enzymology
Kidney Glomerulus/*metabolism
Mad2 Proteins/*metabolism
S-Phase Kinase-Associated Proteins/*metabolism
Animals ; Epithelial Cells/drug effects ; Epithelial Cells/pathology ; Etanercept/pharmacology ; Extracellular Matrix/metabolism ; Extracellular Matrix/pathology ; Gene Expression Regulation ; Glomerulonephritis/drug therapy ; Glomerulonephritis/genetics ; Glomerulonephritis/pathology ; Glucocorticoids/pharmacology ; Humans ; Kidney Glomerulus/drug effects ; Kidney Glomerulus/pathology ; Mad2 Proteins/genetics ; Male ; Mice ; Prednisolone/analogs & derivatives ; Prednisolone/pharmacology ; RAW 264.7 Cells ; Rats, Inbred WKY ; S-Phase Kinase-Associated Proteins/genetics ; Signal Transduction
Czasopismo naukowe
Tytuł :
Protein disulfide isomerase inhibition impairs Keap1/Nrf2 signaling and mitochondrial function and induces apoptosis in renal proximal tubular cells.
Autorzy :
Pokkunuri ID; Heart and Kidney Institute, College of Pharmacy, University of Houston, Houston, Texas.
Lokhandwala MF; Heart and Kidney Institute, College of Pharmacy, University of Houston, Houston, Texas.
Banday AA; Heart and Kidney Institute, College of Pharmacy, University of Houston, Houston, Texas.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Oct 01; Vol. 319 (4), pp. F686-F696. Date of Electronic Publication: 2020 Aug 24.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Apoptosis*/drug effects
Epithelial Cells/*enzymology
Kelch-Like ECH-Associated Protein 1/*metabolism
Kidney Tubules, Proximal/*enzymology
Mitochondria/*enzymology
NF-E2-Related Factor 2/*metabolism
Protein Disulfide-Isomerases/*metabolism
Active Transport, Cell Nucleus ; Antioxidants/pharmacology ; Cell Line ; Cyclic N-Oxides/pharmacology ; Energy Metabolism ; Epithelial Cells/drug effects ; Epithelial Cells/pathology ; Humans ; Kelch-Like ECH-Associated Protein 1/genetics ; Kidney Tubules, Proximal/drug effects ; Kidney Tubules, Proximal/pathology ; Mitochondria/drug effects ; Mitochondria/genetics ; Mitochondria/pathology ; NF-E2-Related Factor 2/genetics ; Oxidative Stress ; Protein Disulfide-Isomerases/genetics ; RNA Interference ; Signal Transduction ; Spin Labels
Czasopismo naukowe

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