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Wyszukujesz frazę ""American Physiological Society"" wg kryterium: PB


Tytuł:
Suppressing kidney angiotensinogen in blood pressure regulation.
Autorzy:
Saigusa T; Division of Nephrology, Medical University of South Carolina, Charleston, South Carolina.
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Źródło:
Physiological reports [Physiol Rep] 2016 Feb; Vol. 4 (3).
Typ publikacji:
Editorial; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Angiotensinogen/*metabolism
Blood Pressure/*physiology
Kidney/*physiology
Renin-Angiotensin System/*physiology
Animals ; Humans ; Mice
Raport
Tytuł:
Effect of increased protein intake on renal acid load and renal hemodynamic responses.
Autorzy:
Teunissen-Beekman KF; Top Institute Food and Nutrition, Wageningen, The Netherlands Department of Human Biology, NUTRIM School for Nutrition, Toxicology and Metabolism Maastricht University, Maastricht, The Netherlands.
Dopheide J; Top Institute Food and Nutrition, Wageningen, The Netherlands Department of Human Biology, NUTRIM School for Nutrition, Toxicology and Metabolism Maastricht University, Maastricht, The Netherlands.
Geleijnse JM; Top Institute Food and Nutrition, Wageningen, The Netherlands Division of Human Nutrition, Wageningen University, Wageningen, The Netherlands.
Bakker SJ; Top Institute Food and Nutrition, Wageningen, The Netherlands Department of Medicine, University Medical Center Groningen and University of Groningen, Groningen, The Netherlands.
Brink EJ; Top Institute Food and Nutrition, Wageningen, The Netherlands.
de Leeuw PW; Department of Medicine, Maastricht University Medical Center and Cardiovascular Research Institute Maastricht (CARIM), Maastricht, The Netherlands.
van Baak MA; Top Institute Food and Nutrition, Wageningen, The Netherlands Department of Human Biology, NUTRIM School for Nutrition, Toxicology and Metabolism Maastricht University, Maastricht, The Netherlands .
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Źródło:
Physiological reports [Physiol Rep] 2016 Mar; Vol. 4 (5).
Typ publikacji:
Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
MeSH Terms:
Acid-Base Equilibrium/*physiology
Dietary Proteins/*administration & dosage
Dietary Proteins/*metabolism
Hemodynamics/*physiology
Kidney/*metabolism
Renal Circulation/*physiology
Blood Pressure/physiology ; Double-Blind Method ; Female ; Glomerular Filtration Rate/physiology ; Humans ; Male ; Middle Aged ; Obesity/diet therapy ; Obesity/metabolism ; Polysaccharides/administration & dosage
Czasopismo naukowe
Tytuł:
Measurement of glomerular filtration rate by dynamic contrast-enhanced magnetic resonance imaging using a subject-specific two-compartment model.
Autorzy:
Tipirneni-Sajja A; Department of Diagnostic Imaging, St. Jude Children's Research Hospital, Memphis, Tennessee Department of Biomedical Engineering, University of Memphis, Memphis, Tennessee.
Loeffler RB; Department of Diagnostic Imaging, St. Jude Children's Research Hospital, Memphis, Tennessee.
Oesingmann N; Siemens Medical Solutions, New York, New York.
Bissler J; Division of Nephrology, St. Jude Children's Research Hospital, Memphis, Tennessee Department of Pediatric Nephrology, Le Bonheur Children's Hospital, Memphis, Tennessee.
Song R; Department of Diagnostic Imaging, St. Jude Children's Research Hospital, Memphis, Tennessee.
McCarville B; Department of Diagnostic Imaging, St. Jude Children's Research Hospital, Memphis, Tennessee.
Jones DP; Department of Pediatrics, Vanderbilt University School of Medicine, Nashville, Tennessee.
Hudson M; Department of Oncology, St. Jude Children's Research Hospital, Memphis, Tennessee.
Spunt SL; Department of Pediatrics, Stanford University School of Medicine, Stanford, California.
Hillenbrand CM; Department of Diagnostic Imaging, St. Jude Children's Research Hospital, Memphis, Tennessee .
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Źródło:
Physiological reports [Physiol Rep] 2016 Apr; Vol. 4 (7). Date of Electronic Publication: 2016 Apr 13.
Typ publikacji:
Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Glomerular Filtration Rate*
Magnetic Resonance Imaging*
Models, Biological*
Contrast Media/*administration & dosage
Gadolinium DTPA/*administration & dosage
Kidney/*physiopathology
Wilms Tumor/*diagnosis
Adult ; Biomarkers/blood ; Chemoradiotherapy, Adjuvant ; Chemotherapy, Adjuvant ; Creatinine/blood ; Female ; Hematocrit ; Humans ; Kidney/diagnostic imaging ; Linear Models ; Male ; Nephrectomy ; Pilot Projects ; Predictive Value of Tests ; Radionuclide Imaging ; Radiopharmaceuticals/administration & dosage ; Radiopharmaceuticals/blood ; Technetium Tc 99m Pentetate/administration & dosage ; Technetium Tc 99m Pentetate/blood ; Time Factors ; Treatment Outcome ; Wilms Tumor/blood ; Wilms Tumor/diagnostic imaging ; Wilms Tumor/physiopathology ; Wilms Tumor/therapy ; Young Adult
Czasopismo naukowe
Tytuł:
Maternal corticosterone exposure in the mouse programs sex-specific renal adaptations in the renin-angiotensin-aldosterone system in 6-month offspring.
Autorzy:
Cuffe JS; School of Biomedical Science, The University of Queensland, St Lucia, Australia.
Burgess DJ; School of Biomedical Science, The University of Queensland, St Lucia, Australia.
O'Sullivan L; School of Biomedical Science, The University of Queensland, St Lucia, Australia.
Singh RR; School of Biomedical Science, The University of Queensland, St Lucia, Australia.
Moritz KM; School of Biomedical Science, The University of Queensland, St Lucia, Australia .
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Źródło:
Physiological reports [Physiol Rep] 2016 Apr; Vol. 4 (8).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Sex Characteristics*
Corticosterone/*toxicity
Kidney/*drug effects
Prenatal Exposure Delayed Effects/*metabolism
Renin-Angiotensin System/*drug effects
Angiotensin II/biosynthesis ; Animals ; Blotting, Western ; Disease Models, Animal ; Female ; Kidney/metabolism ; Kidney/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Pregnancy ; Proto-Oncogene Mas ; RNA, Messenger/analysis ; Renin/biosynthesis ; Renin-Angiotensin System/physiology ; Reverse Transcriptase Polymerase Chain Reaction
Czasopismo naukowe
Tytuł:
Effect of NBCe1 deletion on renal citrate and 2-oxoglutarate handling.
Autorzy:
Osis G; Division of Nephrology, Hypertension and Renal Transplantation, University of Florida College of Medicine, Gainesville, Florida.
Handlogten ME; Division of Nephrology, Hypertension and Renal Transplantation, University of Florida College of Medicine, Gainesville, Florida.
Lee HW; Division of Nephrology, Hypertension and Renal Transplantation, University of Florida College of Medicine, Gainesville, Florida.
Hering-Smith KS; Renal Division, Tulane University College of Medicine, New Orleans, Louisiana.
Huang W; Renal Division, Tulane University College of Medicine, New Orleans, Louisiana.
Romero MF; Department of Physiology & Biomedical Engineering and Nephrology & Hypertension, Mayo Clinic College Of Medicine, Rochester, Minnesota.
Verlander JW; Division of Nephrology, Hypertension and Renal Transplantation, University of Florida College of Medicine, Gainesville, Florida.
Weiner ID; Division of Nephrology, Hypertension and Renal Transplantation, University of Florida College of Medicine, Gainesville, Florida Nephrology and Hypertension Section, North Florida/South Georgia Veterans Health System, Gainesville, Florida .
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Źródło:
Physiological reports [Physiol Rep] 2016 Apr; Vol. 4 (8).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:
Citric Acid/*metabolism
Ketoglutaric Acids/*metabolism
Kidney/*metabolism
Sodium-Bicarbonate Symporters/*metabolism
Acidosis/genetics ; Acidosis/metabolism ; Animals ; Immunoblotting ; Immunohistochemistry ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Real-Time Polymerase Chain Reaction ; Sodium-Bicarbonate Symporters/genetics
Czasopismo naukowe
Tytuł:
Dietary salt regulates uroguanylin expression and signaling activity in the kidney, but not in the intestine.
Autorzy:
Fellner RC; Department of Cell Biology and Physiology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina.
Moss NG; Department of Cell Biology and Physiology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina.
Goy MF; Department of Cell Biology and Physiology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina .
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Źródło:
Physiological reports [Physiol Rep] 2016 May; Vol. 4 (9). Date of Electronic Publication: 2016 May 15.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Intestinal Mucosa/*metabolism
Kidney/*metabolism
Natriuretic Peptides/*biosynthesis
Signal Transduction/*physiology
Sodium, Dietary/*administration & dosage
Animals ; Biomarkers/blood ; Biomarkers/urine ; Gene Expression Regulation ; Intestines/drug effects ; Kidney/drug effects ; Male ; Mice ; Mice, Inbred C57BL ; Natriuretic Peptides/blood ; Natriuretic Peptides/urine ; Rats ; Rats, Sprague-Dawley ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł:
Extracorporeal shock wave therapy does not improve hypertensive nephropathy.
Autorzy:
Caron J; INSERM UNIT 1155, Paris, F-75020, France.
Michel PA; INSERM UNIT 1155, Paris, F-75020, France Université Pierre et Marie Curie-Paris 6 UMR S 1155, Paris, France Department of Nephrology, AP-HP Hôpital Tenon, Paris, F-75020, France.
Dussaule JC; INSERM UNIT 1155, Paris, F-75020, France Université Pierre et Marie Curie-Paris 6 UMR S 1155, Paris, France Department of physiology, AP-HP Hôpital Saint-Antoine, Paris, F-75012, France.
Chatziantoniou C; INSERM UNIT 1155, Paris, F-75020, France Université Pierre et Marie Curie-Paris 6 UMR S 1155, Paris, France.
Ronco P; INSERM UNIT 1155, Paris, F-75020, France Université Pierre et Marie Curie-Paris 6 UMR S 1155, Paris, France Department of Nephrology, AP-HP Hôpital Tenon, Paris, F-75020, France.
Boffa JJ; INSERM UNIT 1155, Paris, F-75020, France Université Pierre et Marie Curie-Paris 6 UMR S 1155, Paris, France Department of Nephrology, AP-HP Hôpital Tenon, Paris, F-75020, France .
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Źródło:
Physiological reports [Physiol Rep] 2016 Jun; Vol. 4 (11).
Typ publikacji:
Journal Article
MeSH Terms:
Lithotripsy*
Hypertension, Renal/*therapy
Kidney/*pathology
Nephritis/*therapy
Animals ; Disease Models, Animal ; Hypertension, Renal/chemically induced ; Hypertension, Renal/pathology ; Male ; NG-Nitroarginine Methyl Ester ; Nephritis/chemically induced ; Nephritis/pathology ; Rats ; Treatment Outcome
SCR Disease Name:
Hypertensive Nephropathy
Czasopismo naukowe
Tytuł:
Vitamin D deficiency contributes to vascular damage in sustained ischemic acute kidney injury.
Autorzy:
de Bragança AC; Division of Nephrology, Laboratory of Basic Science LIM-12, University of São Paulo School of Medicine, São Paulo, Brazil .
Volpini RA; Division of Nephrology, Laboratory of Basic Science LIM-12, University of São Paulo School of Medicine, São Paulo, Brazil.
Mehrotra P; Department of Cellular and Integrative Physiology, Indiana University School of Medicine, Indianapolis, Indiana.
Andrade L; Division of Nephrology, Laboratory of Basic Science LIM-12, University of São Paulo School of Medicine, São Paulo, Brazil.
Basile DP; Department of Cellular and Integrative Physiology, Indiana University School of Medicine, Indianapolis, Indiana.
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Źródło:
Physiological reports [Physiol Rep] 2016 Jul; Vol. 4 (13).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Acute Kidney Injury/*pathology
Capillaries/*pathology
Kidney/*blood supply
Reperfusion Injury/*pathology
Vitamin D Deficiency/*pathology
Actins/metabolism ; Acute Kidney Injury/complications ; Acute Kidney Injury/immunology ; Acute Kidney Injury/metabolism ; Animals ; CD8-Positive T-Lymphocytes/immunology ; CD8-Positive T-Lymphocytes/metabolism ; Calcium-Binding Proteins/metabolism ; Disease Models, Animal ; Disease Progression ; Fibrosis ; Interferon-gamma/metabolism ; Interleukin-17/metabolism ; Kidney/immunology ; Kidney/metabolism ; Kidney/pathology ; Male ; Nephritis/etiology ; Nephritis/pathology ; Rats, Wistar ; Renal Insufficiency, Chronic/etiology ; Renal Insufficiency, Chronic/pathology ; Reperfusion Injury/complications ; Reperfusion Injury/immunology ; Reperfusion Injury/metabolism ; S100 Calcium-Binding Protein A4 ; S100 Proteins/metabolism ; T-Lymphocytes, Regulatory/immunology ; T-Lymphocytes, Regulatory/metabolism ; Th17 Cells/immunology ; Th17 Cells/metabolism ; Time Factors ; Vitamin D Deficiency/complications ; Vitamin D Deficiency/immunology ; Vitamin D Deficiency/metabolism
Czasopismo naukowe
Tytuł:
ROMK expression remains unaltered in a mouse model of familial hyperkalemic hypertension caused by the CUL3Δ403-459 mutation.
Autorzy:
Murthy M; Division of Experimental Medicine and Immunotherapeutics, Department of Medicine, University of Cambridge, Cambridge, CB2 2QQ, United Kingdom.
Kurz T; MRC Protein Phosphorylation and Ubiquitylation Unit, College of Life Sciences University of Dundee, Dow Street, Dundee DD15EH, Scotland, United Kingdom.
O'Shaughnessy KM; Division of Experimental Medicine and Immunotherapeutics, Department of Medicine, University of Cambridge, Cambridge, CB2 2QQ, United Kingdom .
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Źródło:
Physiological reports [Physiol Rep] 2016 Jul; Vol. 4 (13).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Mutation*
Cullin Proteins/*genetics
Kidney/*enzymology
Potassium Channels, Inwardly Rectifying/*metabolism
Pseudohypoaldosteronism/*enzymology
Adaptor Proteins, Signal Transducing ; Animals ; Biomarkers/blood ; Cell Line ; Gene Knock-In Techniques ; Genetic Predisposition to Disease ; Membrane Potentials ; Mice, Transgenic ; Microfilament Proteins/genetics ; Microfilament Proteins/metabolism ; Oocytes ; Phenotype ; Potassium/blood ; Potassium Channels, Inwardly Rectifying/genetics ; Protein Processing, Post-Translational ; Protein Serine-Threonine Kinases/genetics ; Protein Serine-Threonine Kinases/metabolism ; Pseudohypoaldosteronism/blood ; Pseudohypoaldosteronism/genetics ; Transfection ; Xenopus laevis
Czasopismo naukowe

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