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Tytuł:
Lipidomic QTL in Diversity Outbred mice identifies a novel function for α/β hydrolase domain 2 (Abhd2) as an enzyme that metabolizes phosphatidylcholine and cardiolipin.
Autorzy:
Price TR; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Stapleton DS; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Schueler KL; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Norris MK; Department of Nutrition and Integrative Physiology, University of Utah, Salt Lake City, Utah, United States of America.
Parks BW; Department of Nutritional Sciences, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Yandell BS; Department of Statistics, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Churchill GA; The Jackson Laboratory, Bar Harbor, Maine, United States of America.
Holland WL; Department of Nutrition and Integrative Physiology, University of Utah, Salt Lake City, Utah, United States of America.
Keller MP; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Attie AD; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
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Źródło:
PLoS genetics [PLoS Genet] 2023 Jul 31; Vol. 19 (7), pp. e1010713. Date of Electronic Publication: 2023 Jul 31 (Print Publication: 2023).
Typ publikacji:
Journal Article
MeSH Terms:
Cardiolipins*/genetics
Cardiolipins*/metabolism
Hydrolases*/genetics
Hydrolases*/metabolism
Animals ; Male ; Mice ; Collaborative Cross Mice/metabolism ; Lipidomics ; Phosphatidylcholines/genetics ; Phospholipids/genetics ; Phospholipids/metabolism
Czasopismo naukowe
Tytuł:
Identification of sample mix-ups and mixtures in microbiome data in Diversity Outbred mice.
Autorzy:
Lobo AK; Department of Biostatistics and Medical Informatics, University of Wisconsin-Madison, Madison, WI 53706, USA.
Traeger LL; Department of Bacteriology, University of Wisconsin-Madison, Madison, WI 53706, USA.
Keller MP; Department of Biochemistry, University of Wisconsin-Madison, Madison, WI 53706, USA.
Attie AD; Department of Biochemistry, University of Wisconsin-Madison, Madison, WI 53706, USA.
Rey FE; Department of Bacteriology, University of Wisconsin-Madison, Madison, WI 53706, USA.
Broman KW; Department of Biostatistics and Medical Informatics, University of Wisconsin-Madison, Madison, WI 53706, USA.
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Źródło:
G3 (Bethesda, Md.) [G3 (Bethesda)] 2021 Oct 19; Vol. 11 (11).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Collaborative Cross Mice*
Microbiota*/genetics
Animals ; Genotype ; Mice ; Quantitative Trait Loci
Czasopismo naukowe
Tytuł:
Genetic determinants of gut microbiota composition and bile acid profiles in mice.
Autorzy:
Kemis JH; Department of Bacteriology, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Linke V; Department of Chemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Barrett KL; Department of Bacteriology, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Boehm FJ; Department of Statistics, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Traeger LL; Department of Bacteriology, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Keller MP; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Rabaglia ME; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Schueler KL; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Stapleton DS; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Gatti DM; Jackson Laboratory, Bar Harbor, Maine, United States of America.
Churchill GA; Jackson Laboratory, Bar Harbor, Maine, United States of America.
Amador-Noguez D; Department of Bacteriology, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Russell JD; Department of Chemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Yandell BS; Department of Statistics, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Broman KW; Department of Biostatistics and Medical Informatics, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Coon JJ; Department of Chemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.; Morgridge Institute for Research, Madison, Wisconsin, United States of America.; Department of Biomolecular Chemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Attie AD; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Rey FE; Department of Bacteriology, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
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Źródło:
PLoS genetics [PLoS Genet] 2019 Aug 29; Vol. 15 (8), pp. e1008073. Date of Electronic Publication: 2019 Aug 29 (Print Publication: 2019).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Bile Acids and Salts/*metabolism
Biological Variation, Population/*genetics
Gastrointestinal Microbiome/*physiology
Organic Anion Transporters, Sodium-Dependent/*genetics
Quantitative Trait Loci/*genetics
Symporters/*genetics
Akkermansia ; Animals ; Bile Acids and Salts/blood ; Collaborative Cross Mice ; Female ; Firmicutes/growth & development ; Male ; Metabolic Networks and Pathways/genetics ; Mice ; Models, Animal ; Organic Anion Transporters, Sodium-Dependent/metabolism ; Symporters/metabolism ; Verrucomicrobia/growth & development
SCR Organism:
Akkermansia muciniphila; Turicibacter sanguinis
Czasopismo naukowe
Tytuł:
Increased transport of acetyl-CoA into the endoplasmic reticulum causes a progeria-like phenotype.
Autorzy:
Peng Y; Department of Medicine, University of Wisconsin-Madison, Madison, Wisconsin.; Waisman Center, University of Wisconsin-Madison, Madison, Wisconsin.
Shapiro SL; Department of Medicine, University of Wisconsin-Madison, Madison, Wisconsin.; Waisman Center, University of Wisconsin-Madison, Madison, Wisconsin.
Banduseela VC; Department of Medicine, University of Wisconsin-Madison, Madison, Wisconsin.; Waisman Center, University of Wisconsin-Madison, Madison, Wisconsin.
Dieterich IA; Department of Medicine, University of Wisconsin-Madison, Madison, Wisconsin.; Waisman Center, University of Wisconsin-Madison, Madison, Wisconsin.; Neuroscience Training Program, University of Wisconsin-Madison, Madison, Wisconsin.
Hewitt KJ; Department of Cell and Regenerative Biology, University of Wisconsin-Madison, Madison, Wisconsin.
Bresnick EH; Department of Cell and Regenerative Biology, University of Wisconsin-Madison, Madison, Wisconsin.
Kong G; Department of Cell and Regenerative Biology, University of Wisconsin-Madison, Madison, Wisconsin.
Zhang J; Department of Cell and Regenerative Biology, University of Wisconsin-Madison, Madison, Wisconsin.
Schueler KL; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin.
Keller MP; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin.
Attie AD; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin.
Hacker TA; Cardiovascular Research Center, University of Wisconsin-Madison, Madison, Wisconsin.
Sullivan R; Department of Comparative Biosciences, University of Wisconsin-Madison, Madison, Wisconsin.
Kielar-Grevstad E; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin.
Arriola Apelo SI; Department of Medicine, University of Wisconsin-Madison, Madison, Wisconsin.
Lamming DW; Department of Medicine, University of Wisconsin-Madison, Madison, Wisconsin.
Anderson RM; Department of Medicine, University of Wisconsin-Madison, Madison, Wisconsin.; Geriatric Research Education Clinical Center, Veterans Affairs Medical Center, Madison, Wisconsin.
Puglielli L; Department of Medicine, University of Wisconsin-Madison, Madison, Wisconsin.; Waisman Center, University of Wisconsin-Madison, Madison, Wisconsin.; Geriatric Research Education Clinical Center, Veterans Affairs Medical Center, Madison, Wisconsin.; Department of Neuroscience, University of Wisconsin-Madison, Madison, Wisconsin.
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Źródło:
Aging cell [Aging Cell] 2018 Oct; Vol. 17 (5), pp. e12820. Date of Electronic Publication: 2018 Jul 27.
Typ publikacji:
Journal Article
MeSH Terms:
Acetyl Coenzyme A/*metabolism
Endoplasmic Reticulum/*metabolism
Progeria/*metabolism
Progeria/*pathology
Animals ; Autophagy ; Biological Transport ; Blood Glucose/metabolism ; Cholesterol/blood ; Female ; Hematopoiesis ; Inflammation/pathology ; Insulin/blood ; Male ; Membrane Transport Proteins/metabolism ; Mice, Transgenic ; Phenotype ; Progeria/blood ; Signal Transduction
Czasopismo naukowe
Tytuł:
The Transcription Factor Nfatc2 Regulates β-Cell Proliferation and Genes Associated with Type 2 Diabetes in Mouse and Human Islets.
Autorzy:
Keller MP; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Paul PK; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Rabaglia ME; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Stapleton DS; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Schueler KL; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Broman AT; Department of Biostatistics & Medical Informatics, University of Wisconsin, Madison, Wisconsin, United States of America.
Ye SI; Department of Statistics, University of Wisconsin, Madison, Wisconsin, United States of America.
Leng N; Department of Biostatistics & Medical Informatics, University of Wisconsin, Madison, Wisconsin, United States of America.
Brandon CJ; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Neto EC; Sage Bionetworks, Seattle, Washington.
Plaisier CL; Institute for Systems Biology, Seattle, Washington.
Simonett SP; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Kebede MA; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Sheynkman GM; Department of Chemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Klein MA; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Baliga NS; Institute for Systems Biology, Seattle, Washington.
Smith LM; Department of Chemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
Broman KW; Department of Biostatistics & Medical Informatics, University of Wisconsin, Madison, Wisconsin, United States of America.
Yandell BS; Department of Statistics, University of Wisconsin, Madison, Wisconsin, United States of America.
Kendziorski C; Department of Biostatistics & Medical Informatics, University of Wisconsin, Madison, Wisconsin, United States of America.
Attie AD; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin, United States of America.
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Źródło:
PLoS genetics [PLoS Genet] 2016 Dec 09; Vol. 12 (12), pp. e1006466. Date of Electronic Publication: 2016 Dec 09 (Print Publication: 2016).
Typ publikacji:
Journal Article
MeSH Terms:
Diabetes Mellitus, Type 2/*genetics
Insulin/*genetics
NFATC Transcription Factors/*genetics
Animals ; Cell Proliferation/genetics ; Chromosome Mapping ; Diabetes Mellitus, Type 2/metabolism ; Diabetes Mellitus, Type 2/pathology ; Gene Expression Regulation ; Genetic Linkage ; Genome ; Genome-Wide Association Study ; Humans ; Insulin-Secreting Cells/metabolism ; Insulin-Secreting Cells/pathology ; Islets of Langerhans/metabolism ; Islets of Langerhans/pathology ; Mice ; Mice, Obese ; NFATC Transcription Factors/biosynthesis ; Promoter Regions, Genetic
Czasopismo naukowe
Tytuł:
Unexpected partial correction of metabolic and behavioral phenotypes of Alzheimer's APP/PSEN1 mice by gene targeting of diabetes/Alzheimer's-related Sorcs1.
Autorzy:
Knight EM; Departments of Neurology and Psychiatry, and Alzheimer's Disease Research Center, Icahn School of Medicine at Mount Sinai, New York, 10029, USA. .
Ruiz HH; Diabetes, Obesity, and Metabolism Institute, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.
Kim SH; Departments of Neurology and Psychiatry, and Alzheimer's Disease Research Center, Icahn School of Medicine at Mount Sinai, New York, 10029, USA.
Harte JC; Departments of Neurology and Psychiatry, and Alzheimer's Disease Research Center, Icahn School of Medicine at Mount Sinai, New York, 10029, USA.; Departments of Neurology and Pediatrics, Icahn School of Medicine at Mount Sinai, New York, 10029, USA.
Hsieh W; Diabetes, Obesity, and Metabolism Institute, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.
Glabe C; Universty of California, Irvine, CA, 92697, USA.; Biochemistry Department, Faculty of Science and Experimental Biochemistry Unit, King Fahd Medical Research Center, King Abdulaziz University, Jeddah, Saudi Arabia.
Klein WL; Department of Neurobiology, Northwestern University, Evanston, IL, 60208, USA.
Attie AD; Department of Biochemistry, University of Wisconsin, Madison, WI, 53706, USA.
Buettner C; Diabetes, Obesity, and Metabolism Institute, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.
Ehrlich ME; Departments of Neurology and Pediatrics, Icahn School of Medicine at Mount Sinai, New York, 10029, USA.
Gandy S; Departments of Neurology and Psychiatry, and Alzheimer's Disease Research Center, Icahn School of Medicine at Mount Sinai, New York, 10029, USA. .; James J. Peters Veterans Affairs Medical Center, Bronx, New York, 10468, USA. .
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Źródło:
Acta neuropathologica communications [Acta Neuropathol Commun] 2016 Feb 25; Vol. 4, pp. 16. Date of Electronic Publication: 2016 Feb 25.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Alzheimer Disease/*complications
Alzheimer Disease/*genetics
Diabetes Mellitus, Type 2/*physiopathology
Receptors, Cell Surface/*deficiency
Amino Acids, Branched-Chain/blood ; Amyloid beta-Peptides/blood ; Amyloid beta-Protein Precursor/genetics ; Animals ; Body Composition/genetics ; Diabetes Mellitus, Type 2/etiology ; Diabetes Mellitus, Type 2/genetics ; Diet, High-Fat/adverse effects ; Disease Models, Animal ; Exploratory Behavior/physiology ; Female ; Glucose Tolerance Test ; Humans ; Insulin/blood ; Insulin Resistance ; Lipids/blood ; Male ; Maze Learning/physiology ; Mice ; Mice, Inbred C57BL ; Mice, Transgenic ; Mutation/genetics ; Phenotype ; Presenilin-1/genetics ; Receptors, Cell Surface/genetics ; Sex Factors
Czasopismo naukowe
Tytuł:
The Mouse Universal Genotyping Array: From Substrains to Subspecies.
Autorzy:
Morgan AP; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Fu CP; Department of Computer Science, University of North Carolina, Chapel Hill, North Carolina 27599.
Kao CY; Department of Computer Science, University of North Carolina, Chapel Hill, North Carolina 27599.
Welsh CE; Department of Mathematics and Computer Science, Rhodes College, Memphis, Tennessee 38112.
Didion JP; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Yadgary L; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Hyacinth L; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Ferris MT; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Bell TA; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Miller DR; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Giusti-Rodriguez P; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Nonneman RJ; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Cook KD; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Whitmire JK; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Gralinski LE; Department of Epidemiology, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599.
Keller M; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706.
Attie AD; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706.
Churchill GA; The Jackson Laboratory, Bar Harbor, Maine 04609.
Petkov P; The Jackson Laboratory, Bar Harbor, Maine 04609.
Sullivan PF; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599 Department of Psychiatry, University of North Carolina, Chapel Hill, North Carolina 27599.
Brennan JR; Mutant Mouse Resource and Research Center, University of North Carolina, Chapel Hill, North Carolina 27599.
McMillan L; Department of Computer Science, University of North Carolina, Chapel Hill, North Carolina 27599.
Pardo-Manuel de Villena F; Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27599 Lineberger Comprehensive Cancer Center and Carolina Center for Genome Sciences, University of North Carolina, Chapel Hill, North Carolina 27599 .
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Źródło:
G3 (Bethesda, Md.) [G3 (Bethesda)] 2015 Dec 18; Vol. 6 (2), pp. 263-79. Date of Electronic Publication: 2015 Dec 18.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Chromosome Mapping*/methods
Genome*
Genomics*/methods
Genotype*
Alleles ; Animals ; Computational Biology/methods ; Gene Dosage ; Genetics, Population ; Mice ; Mice, Inbred Strains ; Oligonucleotide Array Sequence Analysis ; Phylogeny ; Polymorphism, Single Nucleotide
Czasopismo naukowe
Tytuł:
Identification and Correction of Sample Mix-Ups in Expression Genetic Data: A Case Study.
Autorzy:
Broman KW; Department of Biostatistics and Medical Informatics, University of Wisconsin, Madison, Wisconsin 53706 .
Keller MP; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706.
Broman AT; Department of Biostatistics and Medical Informatics, University of Wisconsin, Madison, Wisconsin 53706.
Kendziorski C; Department of Biostatistics and Medical Informatics, University of Wisconsin, Madison, Wisconsin 53706.
Yandell BS; Department of Statistics, University of Wisconsin, Madison, Wisconsin 53706 Department of Horticulture, University of Wisconsin, Madison, Wisconsin 53706.
Sen Ś; Department of Epidemiology and Biostatistics, University of California, San Francisco, California 94107.
Attie AD; Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706.
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Źródło:
G3 (Bethesda, Md.) [G3 (Bethesda)] 2015 Aug 19; Vol. 5 (10), pp. 2177-86. Date of Electronic Publication: 2015 Aug 19.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Gene Expression*
Animals ; Chromosome Mapping ; Computational Biology/methods ; Gene Expression Profiling ; Genome-Wide Association Study ; Genomics/methods ; Genotype ; Lod Score ; Mice ; Phenotype ; Quantitative Trait Loci ; Transcriptome
Czasopismo naukowe
Tytuł:
Positional cloning of a type 2 diabetes quantitative trait locus; tomosyn-2, a negative regulator of insulin secretion.
Autorzy:
Bhatnagar S; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, United States of America.
Oler AT
Rabaglia ME
Stapleton DS
Schueler KL
Truchan NA
Worzella SL
Stoehr JP
Clee SM
Yandell BS
Keller MP
Thurmond DC
Attie AD
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Źródło:
PLoS genetics [PLoS Genet] 2011 Oct; Vol. 7 (10), pp. e1002323. Date of Electronic Publication: 2011 Oct 06.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Diabetes Mellitus, Type 2/*genetics
Insulin/*metabolism
R-SNARE Proteins/*genetics
R-SNARE Proteins/*metabolism
8-Bromo Cyclic Adenosine Monophosphate/pharmacology ; Adaptor Proteins, Vesicular Transport ; Animals ; Chromosome Mapping ; Cloning, Molecular ; Disease Models, Animal ; Genetic Predisposition to Disease ; Glucose/analysis ; HEK293 Cells ; Humans ; Hypoglycemia/genetics ; Insulin Secretion ; Islets of Langerhans/metabolism ; Islets of Langerhans/pathology ; Leptin/genetics ; Leptin/metabolism ; Mice ; Mice, Inbred C57BL ; Polymorphism, Single Nucleotide ; Qa-SNARE Proteins/genetics ; Qa-SNARE Proteins/metabolism ; Quantitative Trait Loci/genetics ; Rats ; SNARE Proteins/metabolism ; Syntaxin 1/genetics ; Syntaxin 1/metabolism
Czasopismo naukowe
Tytuł:
Gastrointestinal hormones and the regulation of β-cell mass.
Autorzy:
Lavine JA; Department of Biochemistry, University of Wisconsin-Madison, Madison, WI 53706, USA.
Attie AD
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Źródło:
Annals of the New York Academy of Sciences [Ann N Y Acad Sci] 2010 Nov; Vol. 1212, pp. 41-58. Date of Electronic Publication: 2010 Oct 29.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Gastrointestinal Hormones/*physiology
Insulin-Secreting Cells/*cytology
Islets of Langerhans/*anatomy & histology
Animals ; Cell Count ; Cell Proliferation/drug effects ; Gastrointestinal Hormones/metabolism ; Gastrointestinal Hormones/pharmacology ; Humans ; Insulin-Secreting Cells/drug effects ; Insulin-Secreting Cells/physiology ; Islets of Langerhans/cytology ; Islets of Langerhans/drug effects ; Islets of Langerhans/physiology ; Models, Biological ; Organ Size/drug effects ; Regeneration/drug effects ; Regeneration/physiology
Czasopismo naukowe
Tytuł:
Genetic validation of whole-transcriptome sequencing for mapping expression affected by cis-regulatory variation.
Autorzy:
Babak T; Rosetta Inpharmatics, LLC, 401 Terry Ave N, Seattle, WA 98109, USA.
Garrett-Engele P
Armour CD
Raymond CK
Keller MP
Chen R
Rohl CA
Johnson JM
Attie AD
Fraser HB
Schadt EE
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Źródło:
BMC genomics [BMC Genomics] 2010 Aug 13; Vol. 11, pp. 473. Date of Electronic Publication: 2010 Aug 13.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression*
Genetic Variation*
Quantitative Trait Loci*
Gene Expression Profiling/*methods
Sequence Analysis, DNA/*methods
Alleles ; Alternative Splicing ; Animals ; Antisense Elements (Genetics)/genetics ; Mice ; Oligonucleotide Array Sequence Analysis ; RNA, Untranslated/genetics ; Transcription, Genetic
Czasopismo naukowe
Tytuł:
Liver and adipose expression associated SNPs are enriched for association to type 2 diabetes.
Autorzy:
Zhong H; Department of Genetics, Rosetta Inpharmatics, Seattle, Washington, United States of America.
Beaulaurier J
Lum PY
Molony C
Yang X
Macneil DJ
Weingarth DT
Zhang B
Greenawalt D
Dobrin R
Hao K
Woo S
Fabre-Suver C
Qian S
Tota MR
Keller MP
Kendziorski CM
Yandell BS
Castro V
Attie AD
Kaplan LM
Schadt EE
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Źródło:
PLoS genetics [PLoS Genet] 2010 May 06; Vol. 6 (5), pp. e1000932. Date of Electronic Publication: 2010 May 06.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression*
Genome-Wide Association Study*
Polymorphism, Single Nucleotide*
Adipose Tissue/*metabolism
Diabetes Mellitus, Type 2/*genetics
Liver/*metabolism
Animals ; Cohort Studies ; DNA-Binding Proteins/genetics ; DNA-Binding Proteins/metabolism ; Diabetes Mellitus, Type 2/metabolism ; Female ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Obese ; Transcription Factors/genetics ; Transcription Factors/metabolism
Czasopismo naukowe
Tytuł:
Getting biological about the genetics of diabetes.
Autorzy:
Newgard CB; Sarah W. Stedman Nutrition and Metabolism Center and Department of Pharmacology and Cancer Biology, Duke University Medical Center, Durham, North Carolina, USA. />Attie AD
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Źródło:
Nature medicine [Nat Med] 2010 Apr; Vol. 16 (4), pp. 388-91.
Typ publikacji:
Journal Article
MeSH Terms:
Diabetes Mellitus, Type 2/*genetics
Animals ; Disease Models, Animal ; Genetic Predisposition to Disease/genetics ; Genome-Wide Association Study ; Genotype ; Humans ; Metabolomics ; Mice ; Oligonucleotide Array Sequence Analysis ; Phenotype
Czasopismo naukowe

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