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Wyszukujesz frazę ""Autocrine Communication"" wg kryterium: Temat


Tytuł:
Macrophage Migration Inhibitor Factor Upregulates MCP-1 Expression in an Autocrine Manner in Hepatocytes during Acute Mouse Liver Injury.
Autorzy:
Xie J; Department of Cell Biology, Municipal Laboratory for Liver Protection and Regulation of Regeneration, Capital Medical University, Beijing 100069, China.
Yang L; Department of Cell Biology, Municipal Laboratory for Liver Protection and Regulation of Regeneration, Capital Medical University, Beijing 100069, China.
Tian L; Department of Cell Biology, Municipal Laboratory for Liver Protection and Regulation of Regeneration, Capital Medical University, Beijing 100069, China.
Li W; Department of Cell Biology, Municipal Laboratory for Liver Protection and Regulation of Regeneration, Capital Medical University, Beijing 100069, China.
Yang L; Department of Cell Biology, Municipal Laboratory for Liver Protection and Regulation of Regeneration, Capital Medical University, Beijing 100069, China.
Li L; Department of Cell Biology, Municipal Laboratory for Liver Protection and Regulation of Regeneration, Capital Medical University, Beijing 100069, China.
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Źródło:
Scientific reports [Sci Rep] 2016 Jun 08; Vol. 6, pp. 27665. Date of Electronic Publication: 2016 Jun 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*
Gene Expression Regulation*
Chemokine CCL2/*genetics
Hepatocytes/*metabolism
Liver Diseases/*genetics
Liver Diseases/*metabolism
Macrophage Migration-Inhibitory Factors/*metabolism
Animals ; Biomarkers ; Carbon Tetrachloride/adverse effects ; Chemical and Drug Induced Liver Injury/genetics ; Chemical and Drug Induced Liver Injury/metabolism ; Chemokine CCL2/metabolism ; Disease Models, Animal ; Liver Diseases/pathology ; Mice ; Phosphorylation ; Protein Kinase Inhibitors/pharmacology ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Signal Transduction/drug effects ; p38 Mitogen-Activated Protein Kinases/metabolism
Czasopismo naukowe
Tytuł:
Vasoactive Intestinal Peptide modulates trophoblast-derived cell line function and interaction with phagocytic cells through autocrine pathways.
Autorzy:
Vota D; Laboratory of Immunopharmacology, Department of Biological Chemistry, School of Sciences, University of Buenos Aires, IQUIBICEN-CONICET, Ciudad Universitaria, Pab. 2, (1428) Buenos Aires, Argentina.
Paparini D; Laboratory of Immunopharmacology, Department of Biological Chemistry, School of Sciences, University of Buenos Aires, IQUIBICEN-CONICET, Ciudad Universitaria, Pab. 2, (1428) Buenos Aires, Argentina.
Hauk V; Laboratory of Immunopharmacology, Department of Biological Chemistry, School of Sciences, University of Buenos Aires, IQUIBICEN-CONICET, Ciudad Universitaria, Pab. 2, (1428) Buenos Aires, Argentina.
Toro A; Laboratory of Placental Molecular Physiology, Department of Biological Chemistry, School of Sciences, University of Buenos Aires, IQUIBICEN-CONICET, Ciudad Universitaria, Pab. 2, (1428) Buenos Aires, Argentina.
Merech F; Laboratory of Immunopharmacology, Department of Biological Chemistry, School of Sciences, University of Buenos Aires, IQUIBICEN-CONICET, Ciudad Universitaria, Pab. 2, (1428) Buenos Aires, Argentina.
Varone C; Laboratory of Placental Molecular Physiology, Department of Biological Chemistry, School of Sciences, University of Buenos Aires, IQUIBICEN-CONICET, Ciudad Universitaria, Pab. 2, (1428) Buenos Aires, Argentina.
Ramhorst R; Laboratory of Immunopharmacology, Department of Biological Chemistry, School of Sciences, University of Buenos Aires, IQUIBICEN-CONICET, Ciudad Universitaria, Pab. 2, (1428) Buenos Aires, Argentina.
Pérez Leirós C; Laboratory of Immunopharmacology, Department of Biological Chemistry, School of Sciences, University of Buenos Aires, IQUIBICEN-CONICET, Ciudad Universitaria, Pab. 2, (1428) Buenos Aires, Argentina.
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Źródło:
Scientific reports [Sci Rep] 2016 May 23; Vol. 6, pp. 26364. Date of Electronic Publication: 2016 May 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*
Phagocytes/*cytology
Trophoblasts/*cytology
Vasoactive Intestinal Peptide/*metabolism
Cell Line ; Cell Movement ; Female ; Gene Knockdown Techniques ; Humans ; Pregnancy ; Pregnancy Trimester, First ; Protein Kinases/metabolism ; Receptors, Pituitary Adenylate Cyclase-Activating Polypeptide/metabolism ; Signal Transduction ; Vasoactive Intestinal Peptide/genetics
Czasopismo naukowe
Tytuł:
Baicalein inhibits FURIN-MT1-MMP-mediated invasion of ectopic endometrial stromal cells in endometriosis possibly by reducing the secretion of TGFB1.
Autorzy:
Ke JY; Department of Obstetrics and Gynecology, Shanghai Medical College of Fudan University, Shanghai, China.; Institute of Obstetrics and Gynecology, Hospital of Obstetrics and Gynecology, Fudan University, Shanghai, China.; Shanghai Key Laboratory of Female Reproductive Endocrine Related Disease, Shanghai, China.
Yang J; Department of Obstetrics and Gynecology, Shanghai Medical College of Fudan University, Shanghai, China.; Institute of Obstetrics and Gynecology, Hospital of Obstetrics and Gynecology, Fudan University, Shanghai, China.; Shanghai Key Laboratory of Female Reproductive Endocrine Related Disease, Shanghai, China.
Li J; Department of Obstetrics and Gynecology, Shanghai Medical College of Fudan University, Shanghai, China.; Department of Integrated Traditional & Western Medicine, Obstetrics and Gynecology Hospital of Fudan University, Shanghai, China.
Xu Z; Department of Obstetrics and Gynecology, Shanghai Medical College of Fudan University, Shanghai, China.; Department of Integrated Traditional & Western Medicine, Obstetrics and Gynecology Hospital of Fudan University, Shanghai, China.
Li MQ; Department of Obstetrics and Gynecology, Shanghai Medical College of Fudan University, Shanghai, China.; Institute of Obstetrics and Gynecology, Hospital of Obstetrics and Gynecology, Fudan University, Shanghai, China.; Shanghai Key Laboratory of Female Reproductive Endocrine Related Disease, Shanghai, China.
Zhu ZL; Department of Obstetrics and Gynecology, Shanghai Medical College of Fudan University, Shanghai, China.; Department of Integrated Traditional & Western Medicine, Obstetrics and Gynecology Hospital of Fudan University, Shanghai, China.
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Źródło:
American journal of reproductive immunology (New York, N.Y. : 1989) [Am J Reprod Immunol] 2021 Mar; Vol. 85 (3), pp. e13344. Date of Electronic Publication: 2020 Sep 26.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Choristoma/*pathology
Endometrium/*pathology
Furin/*metabolism
Matrix Metalloproteinase 14/*metabolism
Transforming Growth Factor beta1/*metabolism
Adult ; Autocrine Communication ; Cell Growth Processes ; Cells, Cultured ; Choristoma/metabolism ; Down-Regulation ; Endometriosis ; Endometrium/metabolism ; Female ; Humans ; Matrix Metalloproteinases/metabolism ; Middle Aged
Czasopismo naukowe
Tytuł:
Cooperative Role of Thrombopoietin and Vascular Endothelial Growth Factor-A in the Progression of Liver Cirrhosis to Hepatocellular Carcinoma.
Autorzy:
Vizio B; Department of Medical Sciences, University of Turin, Via Genova 3, 10126 Turin, Italy.
Bosco O; Department of Medical Sciences, University of Turin, Via Genova 3, 10126 Turin, Italy.
David E; Pathology Unit, AOU Città della Salute e della Scienza di Torino, University of Turin, Via Santena 7, 10126 Turin, Italy.
Caviglia GP; Department of Medical Sciences, University of Turin, Via Cavour 31, 10123 Turin, Italy.
Abate ML; Department of Medical Sciences, University of Turin, Via Cavour 31, 10123 Turin, Italy.
Schiavello M; Department of Medical Sciences, University of Turin, Via Genova 3, 10126 Turin, Italy.
Pucci A; Department of Histopathology, Pisa University Hospital, Via Roma 57, 56126 Pisa, Italy.
Smedile A; Department of Medical Sciences, University of Turin, Via Cavour 31, 10123 Turin, Italy.
Paraluppi G; General Surgery 2U and Liver Transplantation Center, AOU Città della Salute e della Scienza di Torino, University of Turin, Corso Bramante 88, 10126 Turin, Italy.
Romagnoli R; General Surgery 2U and Liver Transplantation Center, AOU Città della Salute e della Scienza di Torino, University of Turin, Corso Bramante 88, 10126 Turin, Italy.
Lupia E; Department of Medical Sciences, University of Turin, Via Genova 3, 10126 Turin, Italy.
Bellone G; Department of Medical Sciences, University of Turin, Via Genova 3, 10126 Turin, Italy.
Montrucchio G; Department of Medical Sciences, University of Turin, Via Genova 3, 10126 Turin, Italy.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Feb 12; Vol. 22 (4). Date of Electronic Publication: 2021 Feb 12.
Typ publikacji:
Journal Article
MeSH Terms:
Carcinoma, Hepatocellular/*metabolism
Liver Cirrhosis/*metabolism
Liver Neoplasms/*metabolism
Neoplasm Proteins/*biosynthesis
Thrombopoietin/*biosynthesis
Vascular Endothelial Growth Factor A/*biosynthesis
Aged ; Autocrine Communication ; Carcinoma, Hepatocellular/etiology ; Carcinoma, Hepatocellular/pathology ; Female ; Gene Expression Regulation, Neoplastic ; Hep G2 Cells ; Humans ; Liver Cirrhosis/complications ; Liver Cirrhosis/pathology ; Liver Neoplasms/etiology ; Liver Neoplasms/pathology ; Male ; Middle Aged ; Paracrine Communication
Czasopismo naukowe
Tytuł:
Senescence-Associated Secretory Phenotype Suppression Mediated by Small-Sized Mesenchymal Stem Cells Delays Cellular Senescence through TLR2 and TLR5 Signaling.
Autorzy:
Kwon JH; Biomedical Research Institute, MEDIPOST Co., Ltd., Seongnam 13494, Korea.
Kim M; Biomedical Research Institute, MEDIPOST Co., Ltd., Seongnam 13494, Korea.
Um S; Biomedical Research Institute, MEDIPOST Co., Ltd., Seongnam 13494, Korea.
Lee HJ; Biomedical Research Institute, MEDIPOST Co., Ltd., Seongnam 13494, Korea.
Bae YK; Biomedical Research Institute, MEDIPOST Co., Ltd., Seongnam 13494, Korea.
Choi SJ; Biomedical Research Institute, MEDIPOST Co., Ltd., Seongnam 13494, Korea.
Hwang HH; King Abdullah University of Science and Technology, Thuwal 47000, Makkah Province, Saudi Arabia.
Oh W; Biomedical Research Institute, MEDIPOST Co., Ltd., Seongnam 13494, Korea.
Jin HJ; Biomedical Research Institute, MEDIPOST Co., Ltd., Seongnam 13494, Korea.
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Źródło:
Cells [Cells] 2021 Jan 03; Vol. 10 (1). Date of Electronic Publication: 2021 Jan 03.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Size*
Cellular Senescence*
Signal Transduction*
Mesenchymal Stem Cells/*cytology
Mesenchymal Stem Cells/*metabolism
Toll-Like Receptor 2/*metabolism
Toll-Like Receptor 5/*metabolism
Autocrine Communication ; Chemokine CXCL1/metabolism ; Fetal Blood/cytology ; Humans ; Infant, Newborn ; Inflammation Mediators/metabolism ; Interleukin-8/metabolism ; Phenotype ; Receptors, Interleukin-8B/metabolism
Czasopismo naukowe
Tytuł:
Control of myeloid cell density in barrier tissues.
Autorzy:
Lohrmann F; Department of Pediatrics and Adolescent Medicine, Faculty of Medicine, Medical Center - University of Freiburg, Germany.; Institute for Immunodeficiency (IFI), Faculty of Medicine, Center for Chronic Immunodeficiency, Medical Center, University of Freiburg, Germany.; Spemann Graduate School for Biology and Medicine, University of Freiburg, Germany.; IMM-PACT Clinician Scientist Program, Faculty of Medicine, University of Freiburg, Germany.
Forde AJ; Institute for Immunodeficiency (IFI), Faculty of Medicine, Center for Chronic Immunodeficiency, Medical Center, University of Freiburg, Germany.; Faculty of Biology, university of Freiburg, Germany.
Merck P; Institute for Immunodeficiency (IFI), Faculty of Medicine, Center for Chronic Immunodeficiency, Medical Center, University of Freiburg, Germany.
Henneke P; Department of Pediatrics and Adolescent Medicine, Faculty of Medicine, Medical Center - University of Freiburg, Germany.; Institute for Immunodeficiency (IFI), Faculty of Medicine, Center for Chronic Immunodeficiency, Medical Center, University of Freiburg, Germany.
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Źródło:
The FEBS journal [FEBS J] 2021 Jan; Vol. 288 (2), pp. 405-426. Date of Electronic Publication: 2020 Jun 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Models, Immunological*
Dendritic Cells/*immunology
Granulocytes/*microbiology
Macrophages/*immunology
Microbiota/*physiology
Symbiosis/*immunology
Animals ; Autocrine Communication/immunology ; Cell Count ; Cell Differentiation ; Cell Proliferation ; Cytokines/genetics ; Cytokines/immunology ; Dendritic Cells/microbiology ; Granulocytes/immunology ; Homeostasis/immunology ; Humans ; Immunity, Innate ; Intestinal Mucosa/immunology ; Intestinal Mucosa/microbiology ; Macrophages/microbiology ; Respiratory Mucosa/immunology ; Respiratory Mucosa/microbiology ; Skin/immunology ; Skin/microbiology
Czasopismo naukowe
Tytuł:
TNF- α Autocrine Feedback Loops in Human Monocytes: The Pro- and Anti-Inflammatory Roles of the TNF- α Receptors Support the Concept of Selective TNFR1 Blockade In Vivo .
Autorzy:
Gane JM; Department of Clinical and Experimental Medicine, University of Birmingham, Edgbaston, Birmingham, UK.
Stockley RA; Lung Investigation Unit, University Hospital Birmingham NHS Foundation Trust, Edgbaston, Birmingham, UK.
Sapey E; Department of Clinical and Experimental Medicine, University of Birmingham, Edgbaston, Birmingham, UK.
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Źródło:
Journal of immunology research [J Immunol Res] 2016; Vol. 2016, pp. 1079851. Date of Electronic Publication: 2016 Sep 22.
Typ publikacji:
Journal Article
MeSH Terms:
Autocrine Communication*
Monocytes/*metabolism
Tumor Necrosis Factor-alpha/*metabolism
Adult ; Cell Membrane/metabolism ; Cells, Cultured ; Cytokines/metabolism ; Female ; Gene Expression ; Humans ; Inflammation/immunology ; Inflammation/metabolism ; Inflammation Mediators ; Male ; Middle Aged ; Monocytes/drug effects ; Monocytes/immunology ; Receptors, Tumor Necrosis Factor/genetics ; Receptors, Tumor Necrosis Factor/metabolism ; Receptors, Tumor Necrosis Factor, Type I/antagonists & inhibitors ; Receptors, Tumor Necrosis Factor, Type I/genetics ; Receptors, Tumor Necrosis Factor, Type I/metabolism ; Receptors, Tumor Necrosis Factor, Type II/genetics ; Receptors, Tumor Necrosis Factor, Type II/metabolism ; Young Adult
Czasopismo naukowe
Tytuł:
A murine model of acute myeloid leukemia with Evi1 overexpression and autocrine stimulation by an intracellular form of GM-CSF in DA-3 cells.
Autorzy:
Cardona ME; a Department of Laboratory Medicine , Clinical Research Center, Karolinska Institutet , Huddinge , Sweden.
Simonson OE; a Department of Laboratory Medicine , Clinical Research Center, Karolinska Institutet , Huddinge , Sweden.
Oprea II; a Department of Laboratory Medicine , Clinical Research Center, Karolinska Institutet , Huddinge , Sweden.
Moreno PM; a Department of Laboratory Medicine , Clinical Research Center, Karolinska Institutet , Huddinge , Sweden.
Silva-Lara MF; b Departament of Basic Science, Medical School, Universidad Industrial de Santander , Colombia.
Mohamed AJ; a Department of Laboratory Medicine , Clinical Research Center, Karolinska Institutet , Huddinge , Sweden.; e Environmental and Life Sciences, Faculty of Science, Universiti Brunei Darussalam , Negara Brunei Darussalam , Brunei.
Christensson B; c Department of Laboratory Medicine , Division of Pathology, Karolinska University Hospital , Huddinge , Sweden.
Gahrton G; d Department of Medicine , Division of Hematology, Karolinska University Hospital , Huddinge , Sweden.
Dilber MS; d Department of Medicine , Division of Hematology, Karolinska University Hospital , Huddinge , Sweden.
Smith CI; a Department of Laboratory Medicine , Clinical Research Center, Karolinska Institutet , Huddinge , Sweden.
Arteaga HJ; a Department of Laboratory Medicine , Clinical Research Center, Karolinska Institutet , Huddinge , Sweden.; b Departament of Basic Science, Medical School, Universidad Industrial de Santander , Colombia.
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Źródło:
Leukemia & lymphoma [Leuk Lymphoma] 2016; Vol. 57 (1), pp. 183-92. Date of Electronic Publication: 2015 Jun 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*
Gene Expression*
DNA-Binding Proteins/*genetics
Granulocyte-Macrophage Colony-Stimulating Factor/*metabolism
Leukemia, Myeloid, Acute/*genetics
Leukemia, Myeloid, Acute/*metabolism
Proto-Oncogenes/*genetics
Transcription Factors/*genetics
Animals ; Biomarkers ; Biopsy ; Cell Line, Tumor ; Cell Transformation, Neoplastic/genetics ; Cell Transformation, Neoplastic/metabolism ; Disease Models, Animal ; Isografts ; Leukemia, Myeloid, Acute/pathology ; MDS1 and EVI1 Complex Locus Protein ; Mice ; Neoplasm Metastasis ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Tumor Burden
Czasopismo naukowe
Tytuł:
Neuropeptide Y upregulates Runx2 and osterix and enhances osteogenesis in mouse MC3T3‑E1 cells via an autocrine mechanism.
Autorzy:
Zhang B; Department of Joint Surgery, The Second Hospital, Cheeloo College of Medicine, Shandong University, Jinan, Shandong 250033, P.R. China.
Zhang X; Department of Obstetrics and Gynecology, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, Shandong 250012, P.R. China.
Xiao J; Department of Evidence‑Based Medicine, Institute of Medical Sciences, The Second Hospital, Cheeloo College of Medicine, Shandong University, Jinan, Shandong 250033, P.R. China.
Zhou X; Department of Joint Surgery, The Second Hospital, Cheeloo College of Medicine, Shandong University, Jinan, Shandong 250033, P.R. China.
Chen Y; Departments of Central Research Lab, The Second Hospital, Cheeloo College of Medicine, Shandong University, Jinan, Shandong 250033, P.R. China.
Gao C; Departments of Spinal Surgery, The Second Hospital, Cheeloo College of Medicine, Shandong University, Jinan, Shandong 250033, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2020 Nov; Vol. 22 (5), pp. 4376-4382. Date of Electronic Publication: 2020 Sep 14.
Typ publikacji:
Journal Article
MeSH Terms:
Osteogenesis*/drug effects
Up-Regulation*
Core Binding Factor Alpha 1 Subunit/*metabolism
Neuropeptide Y/*metabolism
Sp7 Transcription Factor/*metabolism
Alkaline Phosphatase/metabolism ; Animals ; Autocrine Communication ; Cell Differentiation ; Cell Line ; Mice ; Neuropeptide Y/genetics ; Osteoblasts/cytology ; Osteoblasts/drug effects ; Osteoblasts/metabolism ; Osteocalcin/metabolism ; RNA, Small Interfering/pharmacology
Czasopismo naukowe
Tytuł:
Heregulin Drives Endocrine Resistance by Altering IL-8 Expression in ER-Positive Breast Cancer.
Autorzy:
Papadimitropoulou A; Center of Basic Research, Biomedical Research Foundation of the Academy of Athens, 115 27 Athens, Greece.
Vellon L; Stem Cells Laboratory, Institute of Biology and Experimental Medicine (IBYME-CONICET), Buenos Aires C1428ADN, Argentina.
Atlas E; Environmental Health Science and Research Bureau, Health Canada, Ottawa, ON K1A 0K9, Canada.; Department of Biochemistry, Microbiology, and Immunology, University of Ottawa, Ottawa, ON K1N 6N5, Canada.
Steen TV; Division of Experimental Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA.
Cuyàs E; Program Against Cancer Therapeutic Resistance (ProCURE), Metabolism and Cancer Group, Catalan Institute of Oncology, 17007 Girona, Spain.; Girona Biomedical Research Institute (IDIBGI), 17190 Salt, Girona, Spain.
Verdura S; Program Against Cancer Therapeutic Resistance (ProCURE), Metabolism and Cancer Group, Catalan Institute of Oncology, 17007 Girona, Spain.; Girona Biomedical Research Institute (IDIBGI), 17190 Salt, Girona, Spain.
Espinoza I; Department of Preventive Medicine, John D. Bower School of Population Health, University of Mississippi Medical Center, Jackson, MS 39216, USA.; Cancer Institute, School of Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA.
Menendez JA; Program Against Cancer Therapeutic Resistance (ProCURE), Metabolism and Cancer Group, Catalan Institute of Oncology, 17007 Girona, Spain.; Girona Biomedical Research Institute (IDIBGI), 17190 Salt, Girona, Spain.
Lupu R; Department of Biochemistry, Microbiology, and Immunology, University of Ottawa, Ottawa, ON K1N 6N5, Canada.; Department of Biochemistry and Molecular Biology Laboratory, Mayo Clinic Minnesota, Rochester, MN 55905, USA.; Mayo Clinic Cancer Center, Rochester, MN 55905, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Oct 19; Vol. 21 (20). Date of Electronic Publication: 2020 Oct 19.
Typ publikacji:
Journal Article
MeSH Terms:
Breast Neoplasms/*metabolism
Endocrine System/*metabolism
Interleukin-8/*metabolism
Neuregulin-1/*metabolism
Receptors, Estrogen/*metabolism
Autocrine Communication ; Breast Neoplasms/pathology ; Chemokines/metabolism ; Female ; Humans ; MCF-7 Cells ; Models, Biological ; Receptor, ErbB-2/metabolism ; Transcription, Genetic ; Up-Regulation
Czasopismo naukowe
Tytuł:
PKCδ maintains phenotypes of tumor initiating cells through cytokine-mediated autocrine loop with positive feedback.
Autorzy:
Kim RK; Department of Life Science, Laboratory of Molecular Biochemistry, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea.
Suh Y; Department of Life Science, Laboratory of Molecular Biochemistry, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea.
Hwang E; Department of Life Science, Laboratory of Molecular Biochemistry, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea.
Yoo KC; Department of Life Science, Laboratory of Molecular Biochemistry, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea.
Choi KS; Molecular and Cellular Imaging Center, Samsung Biomedical Research Institute, Sungkyunkwan University School of Medicine, Seoul, Korea.
An S; Functional Genoproteome Research Centre, Konkuk University, Seoul, Korea.
Hwang SG; Division of Radiation Cancer Biology, Korea Institute of Radiological and Medical Sciences, Seoul, Korea.
Kim IG; Department of Radiation Biology, Environmental Radiation Research Group, Korea Atomic Energy Research Institute, Daejeon, Korea.
Kim MJ; Department of Life Science, Laboratory of Molecular Biochemistry, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea.; Laboratory of Radiation Exposure and Therapeutics, National Radiation Emergency Medical Center, Korea Institute of Radiological and Medical Sciences, Seoul, Korea.
Lee HJ; Division of Radiation Effect, Korea Institute of Radiological and Medical Sciences, Seoul, Korea.
Lee SJ; Department of Life Science, Laboratory of Molecular Biochemistry, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea.
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Źródło:
Oncogene [Oncogene] 2015 Nov 12; Vol. 34 (46), pp. 5749-59. Date of Electronic Publication: 2015 Mar 09.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*
Glioblastoma/*metabolism
Glioblastoma/*pathology
Neoplastic Stem Cells/*pathology
Protein Kinase C-delta/*metabolism
Animals ; Cell Line, Tumor ; Humans ; Mice ; Neoplastic Stem Cells/metabolism ; Phosphorylation ; Proto-Oncogene Proteins c-akt/metabolism ; Serine/metabolism ; Signal Transduction
Czasopismo naukowe
Tytuł:
Autocrine amplification of immature myeloid cells by IL-6 in multiple myeloma-infiltrated bone marrow.
Autorzy:
Matthes T; Division of Hematology, University Hospitals, Geneva, Switzerland.
Manfroi B; Analytical Immunology for chronic pathologies, Albert Bonniot Institute, INSERM/University Joseph Fourier, Grenoble, France.
Zeller A; Department of Pathology and Immunology, Medicine Faculty, Geneva, Switzerland.
Dunand-Sauthier I; Department of Pathology and Immunology, Medicine Faculty, Geneva, Switzerland.
Bogen B; Institute for Immunology, University Hospital, Oslo, Norway.
Huard B; Division of Hematology, University Hospitals, Geneva, Switzerland.; Analytical Immunology for chronic pathologies, Albert Bonniot Institute, INSERM/University Joseph Fourier, Grenoble, France.; Department of Pathology and Immunology, Medicine Faculty, Geneva, Switzerland.
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Źródło:
Leukemia [Leukemia] 2015 Sep; Vol. 29 (9), pp. 1882-90. Date of Electronic Publication: 2015 Jun 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*
Bone Marrow/*pathology
Interleukin-6/*metabolism
Multiple Myeloma/*metabolism
Multiple Myeloma/*pathology
Myeloid Cells/*metabolism
Myeloid Cells/*pathology
Animals ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Female ; Immunophenotyping ; Interleukin-6/pharmacology ; Mice ; Models, Biological ; Myeloid Cells/drug effects ; Myeloid Progenitor Cells/drug effects ; Myeloid Progenitor Cells/metabolism ; Myeloid Progenitor Cells/pathology ; RANK Ligand/metabolism ; Signal Transduction ; Tumor Necrosis Factor Ligand Superfamily Member 13/metabolism
Czasopismo naukowe
Tytuł:
Type I Interferons Function as Autocrine and Paracrine Factors to Induce Autotaxin in Response to TLR Activation.
Autorzy:
Song J; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
Guan M; Key Laboratory of Analytical Chemistry for Living Biosystems, Institute of Chemistry Chinese Academy of Sciences, Beijing, China.
Zhao Z; Key Laboratory of Analytical Chemistry for Living Biosystems, Institute of Chemistry Chinese Academy of Sciences, Beijing, China.
Zhang J; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
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Źródło:
PloS one [PLoS One] 2015 Aug 27; Vol. 10 (8), pp. e0136629. Date of Electronic Publication: 2015 Aug 27 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication/*physiology
Interferon-alpha/*biosynthesis
Interferon-beta/*biosynthesis
Paracrine Communication/*physiology
Phosphoric Diester Hydrolases/*biosynthesis
Toll-Like Receptors/*metabolism
Autocrine Communication/drug effects ; Cell Line, Tumor ; Dendritic Cells/cytology ; Dendritic Cells/metabolism ; Enzyme Induction/drug effects ; Enzyme Induction/genetics ; Gene Knockdown Techniques ; Humans ; Interferon-alpha/genetics ; Interferon-beta/genetics ; Lipopolysaccharides/pharmacology ; Lysophospholipids/biosynthesis ; Lysophospholipids/genetics ; Monocytes/cytology ; Monocytes/metabolism ; Oligodeoxyribonucleotides/pharmacology ; Paracrine Communication/drug effects ; Phosphoric Diester Hydrolases/genetics ; Poly I-C/pharmacology ; Receptor, Interferon alpha-beta/genetics ; Receptor, Interferon alpha-beta/metabolism ; Toll-Like Receptors/agonists ; Toll-Like Receptors/genetics
Czasopismo naukowe
Tytuł:
TNF-α Induced by Hepatitis C Virus via TLR7 and TLR8 in Hepatocytes Supports Interferon Signaling via an Autocrine Mechanism.
Autorzy:
Lee J; Department of Molecular Microbiology and Immunology, University of Southern California, Keck School of Medicine, Los Angeles, California, United States of America.
Tian Y; Department of Molecular Microbiology and Immunology, University of Southern California, Keck School of Medicine, Los Angeles, California, United States of America.
Chan ST; Department of Molecular Microbiology and Immunology, University of Southern California, Keck School of Medicine, Los Angeles, California, United States of America.
Kim JY; Department of Molecular Microbiology and Immunology, University of Southern California, Keck School of Medicine, Los Angeles, California, United States of America.
Cho C; Department of Molecular Microbiology and Immunology, University of Southern California, Keck School of Medicine, Los Angeles, California, United States of America.
Ou JH; Department of Molecular Microbiology and Immunology, University of Southern California, Keck School of Medicine, Los Angeles, California, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2015 May 29; Vol. 11 (5), pp. e1004937. Date of Electronic Publication: 2015 May 29 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Autocrine Communication*
Carcinoma, Hepatocellular/*immunology
Hepatitis C/*immunology
Interferons/*metabolism
Toll-Like Receptor 7/*metabolism
Toll-Like Receptor 8/*metabolism
Tumor Necrosis Factor-alpha/*pharmacology
Apoptosis ; Blotting, Western ; Carcinoma, Hepatocellular/drug therapy ; Carcinoma, Hepatocellular/metabolism ; Carcinoma, Hepatocellular/virology ; Cell Proliferation ; Cells, Cultured ; Chromatin Immunoprecipitation ; Cytokines/genetics ; Cytokines/metabolism ; Hepacivirus/physiology ; Hepatitis C/drug therapy ; Hepatitis C/metabolism ; Hepatitis C/virology ; Hepatocytes/drug effects ; Hepatocytes/immunology ; Hepatocytes/virology ; Humans ; Interferons/genetics ; Liver Neoplasms/drug therapy ; Liver Neoplasms/immunology ; Liver Neoplasms/metabolism ; Liver Neoplasms/virology ; NF-kappa B/genetics ; NF-kappa B/metabolism ; RNA, Messenger/genetics ; Real-Time Polymerase Chain Reaction ; Reverse Transcriptase Polymerase Chain Reaction ; Signal Transduction ; Toll-Like Receptor 7/genetics ; Toll-Like Receptor 8/genetics
Czasopismo naukowe
Tytuł:
4-1BB Signaling Enhances Primary and Secondary Population Expansion of CD8+ T Cells by Maximizing Autocrine IL-2/IL-2 Receptor Signaling.
Autorzy:
Oh HS; Cancer Immunology Branch, Division of Cancer Biology, National Cancer Center, Ilsan, Goyang, Gyeonggi, Korea.
Choi BK; Cancer Immunology Branch, Division of Cancer Biology, National Cancer Center, Ilsan, Goyang, Gyeonggi, Korea.
Kim YH; Immune Cell Production Unit, Program for Immunotherapeutic Research, National Cancer Center, Ilsan, Goyang, Gyeonggi, Korea.
Lee DG; Cancer Immunology Branch, Division of Cancer Biology, National Cancer Center, Ilsan, Goyang, Gyeonggi, Korea.
Hwang S; Cancer Immunology Branch, Division of Cancer Biology, National Cancer Center, Ilsan, Goyang, Gyeonggi, Korea.
Lee MJ; Cancer Immunology Branch, Division of Cancer Biology, National Cancer Center, Ilsan, Goyang, Gyeonggi, Korea.
Park SH; Cancer Immunology Branch, Division of Cancer Biology, National Cancer Center, Ilsan, Goyang, Gyeonggi, Korea.
Bae YS; Department of Biological Sciences, Sungkyunkwan University, Suwon, Gyeonggi, Korea.
Kwon BS; Cancer Immunology Branch, Division of Cancer Biology, National Cancer Center, Ilsan, Goyang, Gyeonggi, Korea; Section of Clinical Immunology, Allergy, and Rheumatology, Department of Medicine, Tulane University Health Sciences Center, New Orleans, Louisiana, United States of America.
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Źródło:
PloS one [PLoS One] 2015 May 11; Vol. 10 (5), pp. e0126765. Date of Electronic Publication: 2015 May 11 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*
Signal Transduction*/drug effects
CD8-Positive T-Lymphocytes/*metabolism
Interleukin-2/*metabolism
Receptors, Interleukin-2/*metabolism
Tumor Necrosis Factor Receptor Superfamily, Member 9/*metabolism
Animals ; CD4-Positive T-Lymphocytes/immunology ; CD4-Positive T-Lymphocytes/metabolism ; CD8-Positive T-Lymphocytes/immunology ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Gene Expression ; Lymphocyte Activation ; Mice ; Mice, Knockout ; Phosphatidylinositol 3-Kinases/metabolism ; Protein Kinase Inhibitors/pharmacology ; Proto-Oncogene Proteins c-akt/metabolism ; Receptors, Interleukin-2/genetics
Czasopismo naukowe
Tytuł:
Pain: Turning down the heat in pain.
Autorzy:
Bray N
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Źródło:
Nature reviews. Neuroscience [Nat Rev Neurosci] 2015 Apr; Vol. 16 (4), pp. 187. Date of Electronic Publication: 2015 Mar 04.
Typ publikacji:
Journal Article; Comment
MeSH Terms:
Autocrine Communication*
Neurons/*metabolism
Pain/*metabolism
Receptors, GABA-B/*metabolism
TRPV Cation Channels/*metabolism
gamma-Aminobutyric Acid/*metabolism
Animals ; Female ; Male
Czasopismo naukowe
Tytuł:
Semaphorin4A-Plexin D1 Axis Induces Th2 and Th17 While Represses Th1 Skewing in an Autocrine Manner.
Autorzy:
Carvalheiro T; Department of Rheumatology & Clinical Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.; Center for Translational Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.
Rafael-Vidal C; Rheumatology & Immuno-mediated Diseases Research Group (IRIDIS), Galicia Sur Health Research Institute (IIS Galicia Sur), SERGAS-UVIGO, 36312 Vigo, Spain.; Rheumatology Department, University Hospital Complex of Vigo, 36312 Vigo, Spain.
Malvar-Fernandez B; Department of Rheumatology & Clinical Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.; Center for Translational Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.
Lopes AP; Department of Rheumatology & Clinical Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.; Center for Translational Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.
Pego-Reigosa JM; Rheumatology & Immuno-mediated Diseases Research Group (IRIDIS), Galicia Sur Health Research Institute (IIS Galicia Sur), SERGAS-UVIGO, 36312 Vigo, Spain.; Rheumatology Department, University Hospital Complex of Vigo, 36312 Vigo, Spain.
Radstake TRDJ; Department of Rheumatology & Clinical Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.; Center for Translational Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.
Garcia S; Department of Rheumatology & Clinical Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.; Center for Translational Immunology, University Medical Center Utrecht, University of Utrecht, 3508 GA Utrecht, The Netherlands.; Rheumatology & Immuno-mediated Diseases Research Group (IRIDIS), Galicia Sur Health Research Institute (IIS Galicia Sur), SERGAS-UVIGO, 36312 Vigo, Spain.; Rheumatology Department, University Hospital Complex of Vigo, 36312 Vigo, Spain.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Sep 22; Vol. 21 (18). Date of Electronic Publication: 2020 Sep 22.
Typ publikacji:
Journal Article
MeSH Terms:
Autocrine Communication/*immunology
Intracellular Signaling Peptides and Proteins/*immunology
Membrane Glycoproteins/*immunology
Semaphorins/*immunology
Th1 Cells/*immunology
Th17 Cells/*immunology
Th2 Cells/*immunology
Cell Differentiation/immunology ; Cytokines/immunology ; Gene Expression Regulation/immunology ; Humans ; Scleroderma, Systemic/immunology ; Scleroderma, Systemic/pathology ; Th1 Cells/pathology ; Th17 Cells/pathology ; Th2 Cells/pathology
Czasopismo naukowe
Tytuł:
VEGF is an autocrine/paracrine neuroprotective factor for injured retinal ganglion neurons.
Autorzy:
Froger N; Sorbonne Université, INSERM, CNRS, Institut de La Vision, 17 rue Moreau, 75012, Paris, France. .
Matonti F; Sorbonne Université, INSERM, CNRS, Institut de La Vision, 17 rue Moreau, 75012, Paris, France.; Centre Monticelli Paradis, 433 bis rue Paradis, 13008, Marseille, France.; Aix Marseille Univ, CNRS, INT, Inst Neurosci Timone, 13005, Marseille, France.
Roubeix C; Sorbonne Université, INSERM, CNRS, Institut de La Vision, 17 rue Moreau, 75012, Paris, France.
Forster V; Sorbonne Université, INSERM, CNRS, Institut de La Vision, 17 rue Moreau, 75012, Paris, France.
Ivkovic I; Sorbonne Université, INSERM, CNRS, Institut de La Vision, 17 rue Moreau, 75012, Paris, France.
Brunel N; UMS 29 INSERM Plateforme FluExGen UPMC, 75012, Paris, France.
Baudouin C; Sorbonne Université, INSERM, CNRS, Institut de La Vision, 17 rue Moreau, 75012, Paris, France.; CHNO Des Quinze-Vingts, DHU Sight Restore, INSERM-DGOS CIC 1423, 75012, Paris, France.
Sahel JA; Sorbonne Université, INSERM, CNRS, Institut de La Vision, 17 rue Moreau, 75012, Paris, France.; CHNO Des Quinze-Vingts, DHU Sight Restore, INSERM-DGOS CIC 1423, 75012, Paris, France.; Fondation Ophtalmologique Adolphe de Rothschild, 75020, Paris, France.; Department of Ophthalmology, The University of Pittsburgh School of Medicine, Pittsburgh, PA, 15213, USA.
Picaud S; Sorbonne Université, INSERM, CNRS, Institut de La Vision, 17 rue Moreau, 75012, Paris, France. .; Fondation Ophtalmologique Adolphe de Rothschild, 75020, Paris, France. .
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Źródło:
Scientific reports [Sci Rep] 2020 Jul 24; Vol. 10 (1), pp. 12409. Date of Electronic Publication: 2020 Jul 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Glaucoma/*drug therapy
Retinal Ganglion Cells/*metabolism
Vascular Endothelial Growth Factor A/*metabolism
Aged ; Aged, 80 and over ; Animals ; Autocrine Communication/drug effects ; Cell Survival/drug effects ; Cells, Cultured ; Diabetic Retinopathy/complications ; Diabetic Retinopathy/drug therapy ; Female ; Glaucoma/etiology ; Glaucoma/pathology ; Humans ; Intravitreal Injections ; Macular Degeneration/complications ; Macular Degeneration/drug therapy ; Macular Edema/complications ; Macular Edema/drug therapy ; Male ; Mesenchymal Stem Cells/metabolism ; Middle Aged ; Paracrine Communication/drug effects ; Primary Cell Culture ; Prospective Studies ; Ranibizumab/administration & dosage ; Rats ; Receptors, Vascular Endothelial Growth Factor/administration & dosage ; Recombinant Fusion Proteins/administration & dosage ; Recombinant Proteins/genetics ; Recombinant Proteins/metabolism ; Retinal Ganglion Cells/drug effects ; Retinal Ganglion Cells/pathology ; Vascular Endothelial Growth Factor A/antagonists & inhibitors ; Vascular Endothelial Growth Factor B/genetics ; Vascular Endothelial Growth Factor B/metabolism ; Vascular Endothelial Growth Factor Receptor-1/antagonists & inhibitors ; Vascular Endothelial Growth Factor Receptor-1/metabolism
Czasopismo naukowe
Tytuł:
Adrenomedullin has a cytoprotective role against endoplasmic reticulum stress for pancreatic β-cells in autocrine and paracrine manners.
Autorzy:
Suetomi R; Division of Endocrinology, Metabolism, Hematological Science and Therapeutics, Department of Bio-Signal Analysis, Graduate School of Medicine, Yamaguchi University, Ube, Japan.
Ohta Y; Division of Endocrinology, Metabolism, Hematological Science and Therapeutics, Department of Bio-Signal Analysis, Graduate School of Medicine, Yamaguchi University, Ube, Japan.; Department of Diabetes Research, School of Medicine, Yamaguchi University, Ube, Japan.
Akiyama M; Division of Endocrinology, Metabolism, Hematological Science and Therapeutics, Department of Bio-Signal Analysis, Graduate School of Medicine, Yamaguchi University, Ube, Japan.
Matsumura T; Division of Endocrinology, Metabolism, Hematological Science and Therapeutics, Department of Bio-Signal Analysis, Graduate School of Medicine, Yamaguchi University, Ube, Japan.
Taguchi A; Division of Endocrinology, Metabolism, Hematological Science and Therapeutics, Department of Bio-Signal Analysis, Graduate School of Medicine, Yamaguchi University, Ube, Japan.
Yamamoto K; Division of Endocrinology, Metabolism, Hematological Science and Therapeutics, Department of Bio-Signal Analysis, Graduate School of Medicine, Yamaguchi University, Ube, Japan.
Kamatani T; Division of Endocrinology, Metabolism, Hematological Science and Therapeutics, Department of Bio-Signal Analysis, Graduate School of Medicine, Yamaguchi University, Ube, Japan.
Tanizawa Y; Division of Endocrinology, Metabolism, Hematological Science and Therapeutics, Department of Bio-Signal Analysis, Graduate School of Medicine, Yamaguchi University, Ube, Japan.
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Źródło:
Journal of diabetes investigation [J Diabetes Investig] 2020 Jul; Vol. 11 (4), pp. 823-833. Date of Electronic Publication: 2020 Mar 10.
Typ publikacji:
Journal Article
MeSH Terms:
Adrenomedullin/*metabolism
Endoplasmic Reticulum Stress/*drug effects
Insulin-Secreting Cells/*physiology
Protective Agents/*metabolism
Animals ; Apoptosis/drug effects ; Autocrine Communication/drug effects ; Cell Line ; Humans ; Mice ; PPAR gamma/metabolism ; Paracrine Communication/drug effects ; Pioglitazone/pharmacology ; Receptors, Adrenomedullin/metabolism ; Signal Transduction/drug effects ; Thapsigargin/pharmacology
Czasopismo naukowe
Tytuł:
A generalizable data-driven multicellular model of pancreatic ductal adenocarcinoma.
Autorzy:
Aguilar B; Institute for Systems Biology, 401 Terry Avenue North, Seattle, WA 98109, USA.
Gibbs DL; Institute for Systems Biology, 401 Terry Avenue North, Seattle, WA 98109, USA.
Reiss DJ; Bristol-Myers Squibb, 400 Dexter Avenue North, Suite 1200, Seattle, WA 98109, USA.
McConnell M; Bristol-Myers Squibb, 400 Dexter Avenue North, Suite 1200, Seattle, WA 98109, USA.
Danziger SA; Bristol-Myers Squibb, 400 Dexter Avenue North, Suite 1200, Seattle, WA 98109, USA.
Dervan A; Bristol-Myers Squibb, 400 Dexter Avenue North, Suite 1200, Seattle, WA 98109, USA.
Trotter M; BMS Center for Innovation and Translational Research Europe (CITRE), Pabellon de Italia, Calle Isaac Newton 4, Sevilla 41092, Spain.
Bassett D; Bristol-Myers Squibb, 400 Dexter Avenue North, Suite 1200, Seattle, WA 98109, USA.
Hershberg R; Formerly Celgene Corporation, 400 Dexter Avenue North, Suite 1200, Seattle, WA 98109, USA.
Ratushny AV; Bristol-Myers Squibb, 400 Dexter Avenue North, Suite 1200, Seattle, WA 98109, USA.
Shmulevich I; Institute for Systems Biology, 401 Terry Avenue North, Seattle, WA 98109, USA.
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Źródło:
GigaScience [Gigascience] 2020 Jul 01; Vol. 9 (7).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Algorithms*
Disease Susceptibility*
Models, Biological*
Carcinoma, Pancreatic Ductal/*etiology
Carcinoma, Pancreatic Ductal/*pathology
Autocrine Communication ; Carcinoma, Pancreatic Ductal/metabolism ; Cell Communication/genetics ; Cytokines/metabolism ; Gene Expression Regulation, Neoplastic ; Humans ; Organ Specificity ; Paracrine Communication ; Phenotype
Czasopismo naukowe

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