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Tytuł:
Growth differentiation factor 7 autocrine signaling promotes hepatic progenitor cell expansion in liver fibrosis.
Autorzy:
Kong D; Department of Gastroenterology and Hepatology, University of Groningen, University Medical Center Groningen, Hanzeplein 1, 9713 GZ, Groningen, The Netherlands.
Mourtzinos A; Department of Gastroenterology and Hepatology, University of Groningen, University Medical Center Groningen, Hanzeplein 1, 9713 GZ, Groningen, The Netherlands.
Heegsma J; Department of Gastroenterology and Hepatology, University of Groningen, University Medical Center Groningen, Hanzeplein 1, 9713 GZ, Groningen, The Netherlands.
Blokzijl H; Department of Gastroenterology and Hepatology, University of Groningen, University Medical Center Groningen, Hanzeplein 1, 9713 GZ, Groningen, The Netherlands.
de Meijer VE; Department of Surgery, Division of Hepato-Pancreato-Biliary Surgery and Liver Transplantation, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Faber KN; Department of Gastroenterology and Hepatology, University of Groningen, University Medical Center Groningen, Hanzeplein 1, 9713 GZ, Groningen, The Netherlands. .
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Źródło:
Stem cell research & therapy [Stem Cell Res Ther] 2023 Oct 05; Vol. 14 (1), pp. 288. Date of Electronic Publication: 2023 Oct 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*
Liver Diseases*/pathology
Humans ; Hepatic Stellate Cells/metabolism ; Liver/metabolism ; Liver Cirrhosis/pathology ; Stem Cells/metabolism ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Droplet-based forward genetic screening of astrocyte-microglia cross-talk.
Autorzy:
Wheeler MA; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA.
Clark IC; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Department of Bioengineering, College of Engineering, California Institute for Quantitative Biosciences, University of California Berkeley, Berkeley, CA 94720, USA.
Lee HG; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Li Z; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA.
Linnerbauer M; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Rone JM; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Blain M; Neuroimmunology Unit, Montreal Neurological Institute, Department of Neurology and Neurosurgery, McGill University, Montreal, QC H3A 2B4, Canada.
Akl CF; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Piester G; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642, USA.
Giovannoni F; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Charabati M; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Lee JH; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Kye YC; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Evergrande Center for Immunologic Diseases, Harvard Medical School and Brigham and Women's Hospital, Boston, MA 02115, USA.
Choi J; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Evergrande Center for Immunologic Diseases, Harvard Medical School and Brigham and Women's Hospital, Boston, MA 02115, USA.
Sanmarco LM; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Srun L; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Chung EN; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Flausino LE; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Andersen BM; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Department of Neurology, Jamaica Plain Veterans Affairs Hospital, Harvard Medical School, Boston, MA 02130, USA.
Rothhammer V; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Department of Neurology, University Hospital Erlangen, Friedrich-Alexander University Erlangen-Nuernberg, Erlangen, Germany.
Yano H; Jill Roberts Institute for Research in Inflammatory Bowel Disease, Joan and Sanford I. Weill Department of Medicine, Department of Microbiology and Immunology, Weill Cornell Medicine, Cornell University, New York, NY 10021, USA.
Illouz T; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.
Zandee SEJ; Neuroimmunology Research Lab, Centre de Recherche du Centre Hospitalier de l'Université de Montréal, Montreal, QC H2X 0A9, Canada.
Daniel C; Type 1 Diabetes Immunology, Helmholtz Diabetes Center at Helmholtz Zentrum München, 80939 Munich, Germany.; Deutsches Zentrum für Diabetesforschung, 85764 Munich-Neuherberg, Germany.; Division of Clinical Pharmacology, Department of Medicine IV, Ludwig-Maximilians-Universität München, 80337 Munich, Germany.
Artis D; Jill Roberts Institute for Research in Inflammatory Bowel Disease, Joan and Sanford I. Weill Department of Medicine, Department of Microbiology and Immunology, Weill Cornell Medicine, Cornell University, New York, NY 10021, USA.; Friedman Center for Nutrition and Inflammation, Joan and Sanford I. Weill Department of Medicine, Department of Microbiology and Immunology, Weill Cornell Medicine, Cornell University, New York, NY 10021, USA.
Prinz M; Institute of Neuropathology, University of Freiburg, D-79106 Freiburg, Germany.; Signaling Research Centres BIOSS and CIBSS, University of Freiburg, D-79106 Freiburg, Germany.; Center for Basics in NeuroModulation, Faculty of Medicine, University of Freiburg, D-79106 Freiburg, Germany.
Abate AR; Department of Bioengineering and Therapeutic Sciences, University of California, San Francisco, California Institute for Quantitative Biosciences, San Francisco, CA 94158, USA.; Chan Zuckerberg Biohub, San Francisco, CA, USA.
Kuchroo VK; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA.; Evergrande Center for Immunologic Diseases, Harvard Medical School and Brigham and Women's Hospital, Boston, MA 02115, USA.
Antel JP; Neuroimmunology Unit, Montreal Neurological Institute, Department of Neurology and Neurosurgery, McGill University, Montreal, QC H3A 2B4, Canada.
Prat A; Neuroimmunology Research Lab, Centre de Recherche du Centre Hospitalier de l'Université de Montréal, Montreal, QC H2X 0A9, Canada.
Quintana FJ; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA.; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA.
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Źródło:
Science (New York, N.Y.) [Science] 2023 Mar 10; Vol. 379 (6636), pp. 1023-1030. Date of Electronic Publication: 2023 Mar 09.
Typ publikacji:
Journal Article
MeSH Terms:
Astrocytes*/physiology
Genetic Testing*/methods
Microfluidic Analytical Techniques*/methods
Microglia*/physiology
Amphiregulin*/genetics
Autocrine Communication*/genetics
High-Throughput Screening Assays ; Gene Expression ; Humans
Czasopismo naukowe
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Tytuł:
Autocrine signaling can explain the emergence of Allee effects in cancer cell populations.
Autorzy:
Gerlee P; Mathematical Sciences, Chalmers University of Technology, Gothenburg, Sweden.; Mathematical Sciences, University of Gothenburg, Gothenburg, Sweden.
Altrock PM; Department of Integrated Mathematical Oncology, Moffitt Cancer Center, Tampa, Florida, United States of America.; Department of Evolutionary Theory, Max Planck Institute for Evolutionary Biology, Plön, Germany.
Malik A; Department of Immunology, Genetics and Pathology, Uppsala University, Uppsala, Sweden.
Krona C; Department of Immunology, Genetics and Pathology, Uppsala University, Uppsala, Sweden.
Nelander S; Department of Immunology, Genetics and Pathology, Uppsala University, Uppsala, Sweden.
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Źródło:
PLoS computational biology [PLoS Comput Biol] 2022 Mar 03; Vol. 18 (3), pp. e1009844. Date of Electronic Publication: 2022 Mar 03 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*
Neoplasms*
Ecology ; Feedback ; Humans ; Models, Biological ; Population Density ; Population Dynamics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Doxorubicin-Resistant TNBC Cells Exhibit Rapid Growth with Cancer Stem Cell-like Properties and EMT Phenotype, Which Can Be Transferred to Parental Cells through Autocrine Signaling.
Autorzy:
Paramanantham A; Departments of Internal Medicine, Institute of Health Sciences, Gyeongsang National University Hospital, Gyeongsang National University College of Medicine, 90 Chilam-dong, Jinju 660-702, Korea.; School of Veterinary and Institute of Life Science, Gyeongsang National University, 900 Gajwadong, Jinju 660-701, Korea.
Jung EJ; Departments of Internal Medicine, Institute of Health Sciences, Gyeongsang National University Hospital, Gyeongsang National University College of Medicine, 90 Chilam-dong, Jinju 660-702, Korea.
Kim HJ; Departments of Pharmacology, Institute of Health Sciences, Gyeongsang National University Hospital, Gyeongsang National University School of Medicine, Jinju 660-702, Korea.
Jeong BK; Departments of Radiation Oncology, Institute of Health Sciences, Gyeongsang National University Hospital, Gyeongsang National University College of Medicine, 90 Chilam-dong, Jinju 660-702, Korea.
Jung JM; Departments of Neurosurgery, Institute of Health Sciences, Gyeongsang National University Hospital, Gyeongsang National University College of Medicine, 90 Chilam-dong, Jinju 660-702, Korea.
Kim GS; School of Veterinary and Institute of Life Science, Gyeongsang National University, 900 Gajwadong, Jinju 660-701, Korea.
Chan HS; Department of Surgery, Institute of Health Sciences, Gyeongsang National University Hospital, Gyeongsang National University School of Medicine, Jinju 660-702, Korea.
Lee WS; Departments of Internal Medicine, Institute of Health Sciences, Gyeongsang National University Hospital, Gyeongsang National University College of Medicine, 90 Chilam-dong, Jinju 660-702, Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Nov 18; Vol. 22 (22). Date of Electronic Publication: 2021 Nov 18.
Typ publikacji:
Journal Article
MeSH Terms:
Antibiotics, Antineoplastic/*pharmacology
Autocrine Communication/*drug effects
Doxorubicin/*pharmacology
Drug Resistance, Neoplasm/*genetics
Epithelial-Mesenchymal Transition/*drug effects
Neoplastic Stem Cells/*drug effects
Antigens, CD/genetics ; Antigens, CD/metabolism ; Autocrine Communication/genetics ; Cadherins/genetics ; Cadherins/metabolism ; Cell Line, Tumor ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Cyclin D1/genetics ; Cyclin D1/metabolism ; Epithelial Cells/drug effects ; Epithelial Cells/metabolism ; Epithelial Cells/pathology ; Epithelial-Mesenchymal Transition/genetics ; ErbB Receptors/genetics ; ErbB Receptors/metabolism ; Female ; Gene Expression Regulation, Neoplastic ; Humans ; Intercellular Adhesion Molecule-1/genetics ; Intercellular Adhesion Molecule-1/metabolism ; Matrix Metalloproteinase 2/genetics ; Matrix Metalloproteinase 2/metabolism ; Matrix Metalloproteinase 9/genetics ; Matrix Metalloproteinase 9/metabolism ; Mitogen-Activated Protein Kinase 1/genetics ; Mitogen-Activated Protein Kinase 1/metabolism ; Mitogen-Activated Protein Kinase 3/genetics ; Mitogen-Activated Protein Kinase 3/metabolism ; Neoplastic Stem Cells/metabolism ; Neoplastic Stem Cells/pathology ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction ; Triple Negative Breast Neoplasms/genetics ; Triple Negative Breast Neoplasms/metabolism ; Triple Negative Breast Neoplasms/pathology ; beta Catenin/genetics ; beta Catenin/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Role of the α Cell in the Pathogenesis of Diabetes: A World beyond the Mirror.
Autorzy:
Martínez MS; MedStar Health Internal Medicine, Georgetown University Affiliated, Baltimore, MD 21218-2829, USA.
Manzano A; Endocrine and Metabolic Diseases Research Center, School of Medicine, Universidad del Zulia, Maracaibo 4002, Venezuela.
Olivar LC; Endocrine and Metabolic Diseases Research Center, School of Medicine, Universidad del Zulia, Maracaibo 4002, Venezuela.
Nava M; Endocrine and Metabolic Diseases Research Center, School of Medicine, Universidad del Zulia, Maracaibo 4002, Venezuela.
Salazar J; Endocrine and Metabolic Diseases Research Center, School of Medicine, Universidad del Zulia, Maracaibo 4002, Venezuela.
D'Marco L; Department of Nephrology, Hospital Clinico Universitario de Valencia, INCLIVA, University of Valencia, 46010 Valencia, Spain.
Ortiz R; Facultad de Medicina, Universidad Católica de Cuenca, Ciudad de Cuenca, Azuay 010105, Ecuador.
Chacín M; Facultad de Ciencias de la Salud, Universidad Simón Bolívar, Barranquilla 080022, Colombia.
Guerrero-Wyss M; Escuela de Nutrición y Dietética, Facultad de Ciencias Para el Cuidado de la Salud, Universidad San Sebastián, Valdivia 5090000, Chile.
Cabrera de Bravo M; City of Houston Health Department, Houston, TX 77054, USA.
Cano C; Endocrine and Metabolic Diseases Research Center, School of Medicine, Universidad del Zulia, Maracaibo 4002, Venezuela.
Bermúdez V; Facultad de Ciencias de la Salud, Universidad Simón Bolívar, Barranquilla 080022, Colombia.
Angarita L; Escuela de Nutrición y Dietética, Facultad de Medicina, Universidad Andres Bello, Sede Concepción 4260000, Chile.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Sep 01; Vol. 22 (17). Date of Electronic Publication: 2021 Sep 01.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Autocrine Communication*
Paracrine Communication*
Diabetes Mellitus, Type 1/*metabolism
Diabetes Mellitus, Type 2/*metabolism
Glucagon-Secreting Cells/*metabolism
Insulin-Secreting Cells/*metabolism
Animals ; Diabetes Mellitus, Type 1/drug therapy ; Diabetes Mellitus, Type 1/pathology ; Diabetes Mellitus, Type 2/drug therapy ; Diabetes Mellitus, Type 2/pathology ; Dipeptidyl-Peptidase IV Inhibitors/therapeutic use ; Glucagon/metabolism ; Glucagon-Like Peptide 1/antagonists & inhibitors ; Glucagon-Like Peptide 1/metabolism ; Glucagon-Secreting Cells/pathology ; Humans ; Hypoglycemic Agents/therapeutic use ; Insulin-Secreting Cells/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Oxytocin and Bone: Review and Perspectives.
Autorzy:
Breuil V; Faculté de Médecine Nice, UMR E-4320 TIRO-MATOs CEA/DRF/Institut Joliot, Université Côte d'Azur, CEDEX 2, 06107 Nice, France.
Trojani MC; Faculté de Médecine Nice, UMR E-4320 TIRO-MATOs CEA/DRF/Institut Joliot, Université Côte d'Azur, CEDEX 2, 06107 Nice, France.
Ez-Zoubir A; French National Centre for Scientific Research (CNRS), Inserm, iBV, Université Côte d'Azur, CEDEX 2, 06107 Nice, France.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Aug 09; Vol. 22 (16). Date of Electronic Publication: 2021 Aug 09.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Autocrine Communication*
Bone Density*
Paracrine Communication*
Sex Characteristics*
Bone and Bones/*metabolism
Oxytocin/*metabolism
Amenorrhea/metabolism ; Animals ; Anorexia Nervosa/metabolism ; Bone and Bones/pathology ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; Cardiovascular Diseases/metabolism ; Cardiovascular Diseases/pathology ; Cognitive Dysfunction/metabolism ; Cognitive Dysfunction/pathology ; Diabetes Mellitus/metabolism ; Diabetes Mellitus/pathology ; Estrogens/deficiency ; Estrogens/metabolism ; Female ; Humans ; Male ; Osteoporosis, Postmenopausal/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Autocrine signaling by receptor tyrosine kinases in urothelial carcinoma of the bladder.
Autorzy:
Lee YH; Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
Lee MM; Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
De Silva DM; Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
Roy A; Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
Wright CE; Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
Wong TK; Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
Costello R; Genitourinary Malignancies Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
Olaku O; Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
Grubb RL 3rd; Department of Urology, Medical University of South Carolina, Charleston, South Carolina, United States of America.
Agarwal PK; Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
Apolo AB; Genitourinary Malignancies Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
Bottaro DP; Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, United States of America.
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Źródło:
PloS one [PLoS One] 2021 Jul 22; Vol. 16 (7), pp. e0241766. Date of Electronic Publication: 2021 Jul 22 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, N.I.H., Intramural
MeSH Terms:
Autocrine Communication*
Receptor Protein-Tyrosine Kinases/*metabolism
Urinary Bladder Neoplasms/*pathology
Animals ; Cell Line, Tumor ; Databases, Factual ; Epithelial-Mesenchymal Transition/genetics ; Gene Expression Regulation, Neoplastic ; Humans ; Intercellular Signaling Peptides and Proteins/genetics ; Intercellular Signaling Peptides and Proteins/metabolism ; Kaplan-Meier Estimate ; Mice ; Neoplasm Invasiveness ; Protein-Tyrosine Kinases/genetics ; Protein-Tyrosine Kinases/metabolism ; Proto-Oncogene Proteins/antagonists & inhibitors ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins/metabolism ; RNA Interference ; RNA, Small Interfering/metabolism ; Receptor Protein-Tyrosine Kinases/antagonists & inhibitors ; Receptor Protein-Tyrosine Kinases/genetics ; Receptors, Granulocyte-Macrophage Colony-Stimulating Factor/genetics ; Receptors, Granulocyte-Macrophage Colony-Stimulating Factor/metabolism ; Transplantation, Heterologous ; Urinary Bladder Neoplasms/metabolism ; Urinary Bladder Neoplasms/mortality ; Axl Receptor Tyrosine Kinase
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Roles of Various Prostaglandins in Fibrosis: A Review.
Autorzy:
Li K; Department of Ophthalmology, The Second Hospital of Jilin University, Changchun 130000, China.
Zhao J; Department of Ophthalmology, The Second Hospital of Jilin University, Changchun 130000, China.
Wang M; Department of Ophthalmology, The Second Hospital of Jilin University, Changchun 130000, China.
Niu L; Department of Ophthalmology, The Second Hospital of Jilin University, Changchun 130000, China.
Wang Y; Department of Ophthalmology, The Second Hospital of Jilin University, Changchun 130000, China.
Li Y; Department of Ophthalmology, The Second Hospital of Jilin University, Changchun 130000, China.
Zheng Y; Department of Ophthalmology, The Second Hospital of Jilin University, Changchun 130000, China.
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Źródło:
Biomolecules [Biomolecules] 2021 May 24; Vol. 11 (6). Date of Electronic Publication: 2021 May 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Autocrine Communication*
Paracrine Communication*
Prostaglandin-Endoperoxide Synthases*
Prostaglandins/*metabolism
Animals ; Chronic Disease ; Fibrosis ; Humans
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Autocrine TGF-β in Cancer: Review of the Literature and Caveats in Experimental Analysis.
Autorzy:
Ungefroren H; First Department of Medicine, University Hospital Schleswig-Holstein, Campus Lübeck, D-23538 Lübeck, Germany.; Clinic for General Surgery, Visceral, Thoracic, Transplantation and Pediatric Surgery, University Hospital Schleswig-Holstein, Campus Kiel, D-24105 Kiel, Germany.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Jan 19; Vol. 22 (2). Date of Electronic Publication: 2021 Jan 19.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Autocrine Communication*/drug effects
Autocrine Communication*/physiology
Neoplasms/*metabolism
Transforming Growth Factor beta/*physiology
Animals ; Biomedical Research/methods ; Biomedical Research/standards ; Feedback, Physiological/drug effects ; Feedback, Physiological/physiology ; Gene Expression Regulation, Neoplastic ; Humans ; Neoplasms/genetics ; Neoplasms/pathology ; Receptors, Transforming Growth Factor beta/genetics ; Receptors, Transforming Growth Factor beta/metabolism ; Research Design ; Transforming Growth Factor beta/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Autocrine and Paracrine Effects of Vascular Endothelial Cells Promote Cutaneous Wound Healing.
Autorzy:
Lu Y; Department of Plastic Surgery, The First Affiliated Hospital of Jinan University, Key Laboratory of Regenerative Medicine, Ministry of Education, Guangzhou, Guangdong Province 510630, China.
Yang Y; Department of Orthopedics, The First Affiliated Hospital of Jinan University, No. 613, Whampoa Avenue West, Guangzhou, Guangdong Province, China.
Xiao L; Department of Plastic Surgery, The First Affiliated Hospital of Jinan University, Key Laboratory of Regenerative Medicine, Ministry of Education, Guangzhou, Guangdong Province 510630, China.
Li S; Department of Plastic Surgery, The First Affiliated Hospital of Jinan University, Key Laboratory of Regenerative Medicine, Ministry of Education, Guangzhou, Guangdong Province 510630, China.
Liao X; Department of Plastic Surgery, The First Affiliated Hospital of Jinan University, Key Laboratory of Regenerative Medicine, Ministry of Education, Guangzhou, Guangdong Province 510630, China.
Liu H; Department of Plastic Surgery, The First Affiliated Hospital of Jinan University, Key Laboratory of Regenerative Medicine, Ministry of Education, Guangzhou, Guangdong Province 510630, China.
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Źródło:
BioMed research international [Biomed Res Int] 2021 Apr 10; Vol. 2021, pp. 6695663. Date of Electronic Publication: 2021 Apr 10 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Autocrine Communication*/drug effects
Paracrine Communication*/drug effects
Wound Healing*/drug effects
Human Umbilical Vein Endothelial Cells/*metabolism
Skin/*pathology
Antigens, CD/metabolism ; Biomarkers/metabolism ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Culture Media, Conditioned/pharmacology ; Cytokines/metabolism ; Fibroblasts/cytology ; Fibroblasts/drug effects ; Human Embryonic Stem Cells/cytology ; Human Embryonic Stem Cells/drug effects ; Human Umbilical Vein Endothelial Cells/drug effects ; Humans ; Mesenchymal Stem Cells/cytology ; Mesenchymal Stem Cells/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Autocrine insulin pathway signaling regulates actin dynamics in cell wound repair.
Autorzy:
Nakamura M; Basic Sciences Division, Fred Hutchinson Cancer Research Center, Seattle, WA, United States of America.
Verboon JM; Basic Sciences Division, Fred Hutchinson Cancer Research Center, Seattle, WA, United States of America.
Allen TE; Basic Sciences Division, Fred Hutchinson Cancer Research Center, Seattle, WA, United States of America.
Abreu-Blanco MT; Basic Sciences Division, Fred Hutchinson Cancer Research Center, Seattle, WA, United States of America.
Liu R; Basic Sciences Division, Fred Hutchinson Cancer Research Center, Seattle, WA, United States of America.
Dominguez ANM; Basic Sciences Division, Fred Hutchinson Cancer Research Center, Seattle, WA, United States of America.
Delrow JJ; Genomics Shared Resource, Fred Hutchinson Cancer Research Center, Seattle, WA, United States of America.
Parkhurst SM; Basic Sciences Division, Fred Hutchinson Cancer Research Center, Seattle, WA, United States of America.
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Źródło:
PLoS genetics [PLoS Genet] 2020 Dec 11; Vol. 16 (12), pp. e1009186. Date of Electronic Publication: 2020 Dec 11 (Print Publication: 2020).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Autocrine Communication*
Signal Transduction*
Wound Healing*
Actins/*metabolism
Insulin/*metabolism
Animals ; Drosophila Proteins/genetics ; Drosophila Proteins/metabolism ; Drosophila melanogaster ; Intracellular Signaling Peptides and Proteins/genetics ; Intracellular Signaling Peptides and Proteins/metabolism ; Profilins/genetics ; Profilins/metabolism ; Transcriptome
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The COX-2-derived PGE 2 autocrine contributes to bradykinin-induced matrix metalloproteinase-9 expression and astrocytic migration via STAT3 signaling.
Autorzy:
Lee TH; Stroke Center and Stroke Section, Department of Neurology, Chang Gung Memorial Hospital, Linkou Medical Center, College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Liu PS; Department of Microbiology, Soochow University, Taipei, Taiwan.
Tsai MM; Department of Nursing, Division of Basic Medical Sciences, Research Center for Chinese Herbal Medicine, Graduate Institute of Health Industry Technology, Chang Gung University of Science and Technology, 261 Wenhua 1st Road, Guishan, Taoyuan, Taiwan.; Department of General Surgery, Chang Gung Memorial Hospital, Chiayi, Taiwan.
Chen JL; Division of Chinese Internal Medicine, Center for Traditional Chinese Medicine, Chang Gung Memorial Hospital, School of Traditional Chinese Medicine, College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Wang SJ; School of Medicine, Fu Jen Catholic University, New Taipei City, Taiwan.
Hsieh HL; Department of Nursing, Division of Basic Medical Sciences, Research Center for Chinese Herbal Medicine, Graduate Institute of Health Industry Technology, Chang Gung University of Science and Technology, 261 Wenhua 1st Road, Guishan, Taoyuan, Taiwan. .; Department of Neurology, Chang Gung Memorial Hospital, Taoyuan, Taiwan. .
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2020 Nov 23; Vol. 18 (1), pp. 185. Date of Electronic Publication: 2020 Nov 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*/drug effects
Astrocytes/*cytology
Astrocytes/*enzymology
Bradykinin/*pharmacology
Cell Movement/*drug effects
Dinoprostone/*metabolism
Matrix Metalloproteinase 9/*metabolism
STAT3 Transcription Factor/*metabolism
Animals ; Astrocytes/drug effects ; Celecoxib/pharmacology ; Cell Line ; Janus Kinase 2/metabolism ; MAP Kinase Signaling System/drug effects ; Rats ; Receptors, Prostaglandin E/metabolism ; Signal Transduction/drug effects ; Up-Regulation/drug effects ; src-Family Kinases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin-34, a Novel Paracrine/Autocrine Factor in Mouse Testis, and Its Possible Role in the Development of Spermatogonial Cells In Vitro.
Autorzy:
Sawaied A; The Shraga Segal Department of Microbiology, Immunology, and Genetics, Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer Sheva 84105, Israel.; The Center of Advanced Research and Education in Reproduction (CARER), Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer Sheva 84105, Israel.
Lunenfeld E; The Center of Advanced Research and Education in Reproduction (CARER), Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer Sheva 84105, Israel.; Department of OB/GYN, Soroka Medical Center, Beer Sheva 8410501, Israel.
Huleihel M; The Shraga Segal Department of Microbiology, Immunology, and Genetics, Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer Sheva 84105, Israel.; The Center of Advanced Research and Education in Reproduction (CARER), Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer Sheva 84105, Israel.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Oct 30; Vol. 21 (21). Date of Electronic Publication: 2020 Oct 30.
Typ publikacji:
Journal Article
MeSH Terms:
Autocrine Communication*
Cell Differentiation*
Paracrine Communication*
Interleukins/*metabolism
Spermatogonia/*cytology
Testis/*cytology
Animals ; Cells, Cultured ; In Vitro Techniques ; Male ; Mice ; Mice, Inbred ICR ; Spermatogonia/metabolism ; Testis/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Schwann Cell Autocrine and Paracrine Regulatory Mechanisms, Mediated by Allopregnanolone and BDNF, Modulate PKCε in Peripheral Sensory Neurons.
Autorzy:
Bonalume V; Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, 20133 Milan, Italy.
Caffino L; Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, 20133 Milan, Italy.
Castelnovo LF; Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, 20133 Milan, Italy.; Marine Science Institute, The University of Texas at Austin, 750 Channel View Drive, Port Aransas, TX 78373, USA.
Faroni A; Blond McIndoe Laboratories, Division of Cell Matrix Biology and Regenerative Medicine, Faculty of Biology, Medicine and Health, The University of Manchester, Manchester M13 9PL, UK.
Giavarini F; Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, 20133 Milan, Italy.
Liu S; Institute of Pharmacology, Heidelberg University, 68167 Mannheim, Germany.
Caruso D; Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, 20133 Milan, Italy.
Schmelz M; Experimental Pain Research, Medical Faculty Mannheim, Heidelberg University, 68167 Mannheim, Germany.
Fumagalli F; Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, 20133 Milan, Italy.
Carr RW; Experimental Pain Research, Medical Faculty Mannheim, Heidelberg University, 68167 Mannheim, Germany.
Magnaghi V; Department of Pharmacological and Biomolecular Sciences, Università degli Studi di Milano, 20133 Milan, Italy.
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Źródło:
Cells [Cells] 2020 Aug 11; Vol. 9 (8). Date of Electronic Publication: 2020 Aug 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication/*physiology
Brain-Derived Neurotrophic Factor/*metabolism
Neuralgia/*metabolism
Paracrine Communication/*physiology
Pregnanolone/*metabolism
Schwann Cells/*metabolism
Animals ; Autocrine Communication/genetics ; Blotting, Western ; Cells, Cultured ; Chromatography, Liquid ; Enzyme-Linked Immunosorbent Assay ; Fluorescent Antibody Technique ; Ganglia, Spinal/metabolism ; Humans ; Hyperalgesia/metabolism ; Paracrine Communication/genetics ; Rats, Sprague-Dawley ; Sensory Receptor Cells/metabolism ; Tandem Mass Spectrometry
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Oxidative Stress Induces a VEGF Autocrine Loop in the Retina: Relevance for Diabetic Retinopathy.
Autorzy:
Rossino MG; Department of Biology, University of Pisa, 56126 Pisa, Italy.
Lulli M; Department of Experimental and Clinical Biomedical Sciences 'Mario Serio', University of Florence, 50134 Florence, Italy.
Amato R; Department of Biology, University of Pisa, 56126 Pisa, Italy.
Cammalleri M; Department of Biology, University of Pisa, 56126 Pisa, Italy.; Interdepartmental Research Center Nutrafood 'Nutraceuticals and Food for Health', University of Pisa, 56124 Pisa, Italy.
Monte MD; Department of Biology, University of Pisa, 56126 Pisa, Italy.; Interdepartmental Research Center Nutrafood 'Nutraceuticals and Food for Health', University of Pisa, 56124 Pisa, Italy.
Casini G; Department of Biology, University of Pisa, 56126 Pisa, Italy.; Interdepartmental Research Center Nutrafood 'Nutraceuticals and Food for Health', University of Pisa, 56124 Pisa, Italy.
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Źródło:
Cells [Cells] 2020 Jun 11; Vol. 9 (6). Date of Electronic Publication: 2020 Jun 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*/drug effects
Oxidative Stress*/drug effects
Diabetic Retinopathy/*metabolism
Diabetic Retinopathy/*pathology
Retina/*metabolism
Retina/*pathology
Vascular Endothelial Growth Factor A/*metabolism
Animals ; Cell Line ; Cell Nucleus/drug effects ; Cell Nucleus/metabolism ; Culture Media, Conditioned/pharmacology ; Diabetic Retinopathy/genetics ; Humans ; Hypoxia-Inducible Factor 1, alpha Subunit/metabolism ; Mice, Inbred C57BL ; Models, Biological ; NF-E2-Related Factor 2/metabolism ; Protein Transport/drug effects ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Vascular Endothelial Growth Factor A/genetics ; Vascular Endothelial Growth Factor Receptor-2/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Concerted EP2 and EP4 Receptor Signaling Stimulates Autocrine Prostaglandin E 2 Activation in Human Podocytes.
Autorzy:
Mangelsen E; Charité - Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Institute of Clinical Pharmacology and Toxicology, Charitéplatz 1, 10117 Berlin, Germany.
Rothe M; Lipidomix GmbH, Robert-Rössle-Str. 10, B55, 13125 Berlin, Germany.
Schulz A; Charité - Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Institute of Clinical Pharmacology and Toxicology, Charitéplatz 1, 10117 Berlin, Germany.
Kourpa A; Max Delbrück Center for Molecular Medicine in the Helmholtz Association, Electrochemical Signaling in Development and Disease, Robert-Rössle-Str. 10, 13125 Berlin, Germany.
Panáková D; Max Delbrück Center for Molecular Medicine in the Helmholtz Association, Electrochemical Signaling in Development and Disease, Robert-Rössle-Str. 10, 13125 Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), Partner Site Berlin, Potsdamer Straße 58, 10785 Berlin, Germany.
Kreutz R; Charité - Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Institute of Clinical Pharmacology and Toxicology, Charitéplatz 1, 10117 Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), Partner Site Berlin, Potsdamer Straße 58, 10785 Berlin, Germany.
Bolbrinker J; Charité - Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Institute of Clinical Pharmacology and Toxicology, Charitéplatz 1, 10117 Berlin, Germany.
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Źródło:
Cells [Cells] 2020 May 19; Vol. 9 (5). Date of Electronic Publication: 2020 May 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autocrine Communication*
Signal Transduction*
Dinoprostone/*analogs & derivatives
Podocytes/*metabolism
Receptors, Prostaglandin E, EP2 Subtype/*metabolism
Receptors, Prostaglandin E, EP4 Subtype/*metabolism
Animals ; Cell Differentiation ; Cyclic AMP/metabolism ; Cyclooxygenase 2/genetics ; Cyclooxygenase 2/metabolism ; Dinoprostone/metabolism ; Disease Models, Animal ; Gene Expression Regulation ; Humans ; Metabolome ; Podocytes/cytology ; Rats, Wistar ; Receptors, Prostaglandin E, EP2 Subtype/antagonists & inhibitors ; Receptors, Prostaglandin E, EP2 Subtype/genetics ; Receptors, Prostaglandin E, EP4 Subtype/antagonists & inhibitors ; Receptors, Prostaglandin E, EP4 Subtype/genetics ; Renal Insufficiency, Chronic/blood ; Renal Insufficiency, Chronic/genetics ; Renal Insufficiency, Chronic/metabolism ; Shear Strength ; Stress, Mechanical
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Role of Sonic Hedgehog Signaling in the Tumor Microenvironment of Oral Squamous Cell Carcinoma.
Autorzy:
Takabatake K; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.
Shimo T; Division of Reconstructive Surgery for Oral and Maxillofacial Region, Department of Human Biology and Pathophysiology, School of Dentistry, Health Sciences University of Hokkaido, Hokkaido 0610293, Japan.
Murakami J; Department of Oral and Maxillofacial Radiology, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.
Anqi C; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.; Department of Anatomy, Basic Medical Science College, Harbin Medical University, Harbin 150081, China.
Kawai H; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.
Yoshida S; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.
Wathone Oo M; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.
Haruka O; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.
Sukegawa S; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.; Department of Oral and Maxillofacial Surgery, Kagawa Prefectural Central Hospital, Kagawa 7608557, Japan.
Tsujigiwa H; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.; Department of Life Science, Faculty of Science, Okayama University of Science, Okayama 7000005, Japan.
Nakano K; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.
Nagatsuka H; Department of Oral Pathology and Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama 7008525, Japan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Nov 17; Vol. 20 (22). Date of Electronic Publication: 2019 Nov 17.
Typ publikacji:
Journal Article
MeSH Terms:
Autocrine Communication*
Paracrine Communication*
Signal Transduction*
Carcinoma, Squamous Cell/*pathology
Hedgehog Proteins/*metabolism
Mouth Neoplasms/*pathology
Cancer-Associated Fibroblasts/metabolism ; Cancer-Associated Fibroblasts/pathology ; Carcinoma, Squamous Cell/metabolism ; Hedgehog Proteins/genetics ; Humans ; Immunohistochemistry ; Macrophages/metabolism ; Macrophages/pathology ; Mouth Neoplasms/metabolism ; Neoplasm Invasiveness ; Patched-1 Receptor/genetics ; Patched-1 Receptor/metabolism ; Tumor Microenvironment
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Mechanistic insights into autocrine and paracrine roles of endothelial GABA signaling in the embryonic forebrain.
Autorzy:
Choi YK; Department of Psychiatry, Harvard Medical School, Boston, MA, 02215, USA. .; Angiogenesis and Brain Development Laboratory, Division of Basic Neuroscience, McLean Hospital, 115 Mill Street, Belmont, MA, 02478, USA. .
Vasudevan A; Department of Psychiatry, Harvard Medical School, Boston, MA, 02215, USA. .; Angiogenesis and Brain Development Laboratory, Division of Basic Neuroscience, McLean Hospital, 115 Mill Street, Belmont, MA, 02478, USA. .
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Źródło:
Scientific reports [Sci Rep] 2019 Nov 07; Vol. 9 (1), pp. 16256. Date of Electronic Publication: 2019 Nov 07.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Autocrine Communication*
Paracrine Communication*
Signal Transduction*
Endothelial Cells/*metabolism
Prosencephalon/*metabolism
Receptors, GABA/*metabolism
gamma-Aminobutyric Acid/*metabolism
Animals ; Cell Differentiation ; Computational Biology/methods ; Embryonic Development ; Gene Expression Profiling ; Gene Expression Regulation, Developmental ; Male ; Mice ; Mice, Knockout ; Models, Biological ; Neurogenesis ; Prosencephalon/embryology ; Transcriptome
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Tumour-Secreted Hsp90α on External Surface of Exosomes Mediates Tumour - Stromal Cell Communication via Autocrine and Paracrine Mechanisms.
Autorzy:
Tang X; Department of Dermatology and the USC-Norris Comprehensive Cancer Center, University of Southern California Keck Medical Centre, Los Angeles, CA, 90033, USA.
Chang C; Department of Dermatology and the USC-Norris Comprehensive Cancer Center, University of Southern California Keck Medical Centre, Los Angeles, CA, 90033, USA.
Guo J; Department of Dermatology and the USC-Norris Comprehensive Cancer Center, University of Southern California Keck Medical Centre, Los Angeles, CA, 90033, USA.
Lincoln V; Department of Dermatology and the USC-Norris Comprehensive Cancer Center, University of Southern California Keck Medical Centre, Los Angeles, CA, 90033, USA.
Liang C; Department of Molecular Microbiology & Immunology, the USC-Norris Comprehensive Cancer Centre, University of Southern California Keck Medical Centre, Los Angeles, CA, 90033, USA.
Chen M; Department of Dermatology and the USC-Norris Comprehensive Cancer Center, University of Southern California Keck Medical Centre, Los Angeles, CA, 90033, USA.
Woodley DT; Department of Dermatology and the USC-Norris Comprehensive Cancer Center, University of Southern California Keck Medical Centre, Los Angeles, CA, 90033, USA.
Li W; Department of Dermatology and the USC-Norris Comprehensive Cancer Center, University of Southern California Keck Medical Centre, Los Angeles, CA, 90033, USA. .
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Źródło:
Scientific reports [Sci Rep] 2019 Oct 22; Vol. 9 (1), pp. 15108. Date of Electronic Publication: 2019 Oct 22.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Autocrine Communication*/drug effects
Cell Communication*/drug effects
Paracrine Communication*/drug effects
Exosomes/*metabolism
HSP90 Heat-Shock Proteins/*metabolism
Neoplasms/*metabolism
Neoplasms/*pathology
Antibodies, Monoclonal/pharmacology ; Biomarkers, Tumor/metabolism ; Cell Line, Tumor ; Exosomes/drug effects ; Humans ; Models, Biological ; Stromal Cells/drug effects ; Stromal Cells/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Autocrine Production of PDGF Stimulated by the Tenascin-C-Derived Peptide TNIIIA2 Induces Hyper-Proliferation in Glioblastoma Cells.
Autorzy:
Fujita M; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda, Chiba 278-8510, Japan.
Yamamoto T; Department of Neurosurgery, Graduate School of Medicine, Yokohama City University, 3-9 Fukuura, Kanazawa-ku, Yokohama, Kanagawa 236-0004, Japan.
Iyoda T; Department of Pharmacy, Faculty of Pharmaceutical Sciences, Sanyo-Onoda City University, 1-1-1 Daigaku-Doori, Sanyo-Onoda, Yamaguchi 756-0884, Japan.
Fujisawa T; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda, Chiba 278-8510, Japan.
Nagai R; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda, Chiba 278-8510, Japan.
Kudo C; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda, Chiba 278-8510, Japan.
Sasada M; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda, Chiba 278-8510, Japan.
Kodama H; Faculty of Science and Engineering, Saga University, 1 Honjo-machi, Saga-city, Saga 840-8502, Japan.
Fukai F; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, 2641 Yamazaki, Noda, Chiba 278-8510, Japan. .; Translational Research Center, Research Institutes for Science and Technology, Tokyo University of Science, 2641 Yamazaki, Noda, Chiba 278-8510, Japan. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Jun 28; Vol. 20 (13). Date of Electronic Publication: 2019 Jun 28.
Typ publikacji:
Journal Article
MeSH Terms:
Autocrine Communication*
Cell Proliferation*
Brain Neoplasms/*metabolism
Glioblastoma/*metabolism
Platelet-Derived Growth Factor/*metabolism
Tenascin/*metabolism
Animals ; Brain Neoplasms/pathology ; Cell Line, Tumor ; Cell Movement ; Cells, Cultured ; Fibroblasts/metabolism ; Fibroblasts/physiology ; Glioblastoma/pathology ; Humans ; Male ; Matrix Metalloproteinase 2/metabolism ; Mice ; Neurons/drug effects ; Neurons/metabolism ; Neurons/physiology ; Peptide Fragments/chemistry ; Peptide Fragments/pharmacology ; Rats ; Rats, Wistar ; Receptors, Platelet-Derived Growth Factor/metabolism ; Tenascin/chemistry
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 1-20 z 477

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