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    Wyświetlanie 1-18 z 18
    Tytuł:
    The Exposure to Lead (Pb) Exacerbates Immunological Abnormalities in BTBR T /J Mice through the Regulation of Signaling Pathways Relevant to T Cells.
    Autorzy:
    Assiri MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Albekairi TH; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ansari MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Shahid M; Department of Pharmaceutics, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Aldossari AA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Almutairi MM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Almanaa TN; Department of Botany and Microbiology, College of Science, King Saud University, Riyadh 11451, Saudi Arabia.
    Alwetaid MY; Department of Botany and Microbiology, College of Science, King Saud University, Riyadh 11451, Saudi Arabia.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
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    Źródło:
    International journal of molecular sciences [Int J Mol Sci] 2023 Nov 11; Vol. 24 (22). Date of Electronic Publication: 2023 Nov 11.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Interleukin-10*/pharmacology
    Autism Spectrum Disorder*/chemically induced
    Humans ; Mice ; Animals ; Lead/toxicity ; Interleukin-9/pharmacology ; Signal Transduction ; Forkhead Transcription Factors/genetics ; Forkhead Transcription Factors/metabolism ; RNA, Messenger ; Mice, Inbred C57BL ; Disease Models, Animal
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Aflatoxin B 1 Exposure Aggravates Neurobehavioral Deficits and Immune Dysfunctions of Th1, Th9, Th17, Th22, and T Regulatory Cell-Related Transcription Factor Signaling in the BTBR T /J Mouse Model of Autism.
    Autorzy:
    Alwetaid MY; Department of Botany and Microbiology, College of Science, King Saud University, Riyadh 11451, Saudi Arabia.
    Almanaa TN; Department of Botany and Microbiology, College of Science, King Saud University, Riyadh 11451, Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ansari MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Hussein MH; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
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    Źródło:
    Brain sciences [Brain Sci] 2023 Oct 27; Vol. 13 (11). Date of Electronic Publication: 2023 Oct 27.
    Typ publikacji:
    Journal Article
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Histamine H4 Receptor Antagonist Ameliorates the Progression of Experimental Autoimmune Encephalomyelitis via Regulation of T-Cell Imbalance.
    Autorzy:
    Aldossari AA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Assiri MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ansari MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Albekairi TH; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Alomar HA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Al-Mazroua HA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Almanaa TN; Department of Botany and Microbiology, College of Science, King Saud University, Riyadh 11451, Saudi Arabia.
    Al-Hamamah MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Alwetaid MY; Department of Botany and Microbiology, College of Science, King Saud University, Riyadh 11451, Saudi Arabia.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
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    Źródło:
    International journal of molecular sciences [Int J Mol Sci] 2023 Oct 17; Vol. 24 (20). Date of Electronic Publication: 2023 Oct 17.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Encephalomyelitis, Autoimmune, Experimental*/drug therapy
    Multiple Sclerosis*/drug therapy
    Mice ; Animals ; Receptors, Histamine H4 ; Transforming Growth Factor beta1 ; Nuclear Receptor Subfamily 1, Group F, Member 3/metabolism ; Interleukin-17/metabolism ; Interleukin-9 ; Histamine Antagonists/pharmacology ; Histamine Antagonists/therapeutic use ; Forkhead Transcription Factors/genetics ; Mice, Inbred C57BL
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Histamine H4 Receptor Agonist, 4-Methylhistamine, Aggravates Disease Progression and Promotes Pro-Inflammatory Signaling in B Cells in an Experimental Autoimmune Encephalomyelitis Mouse Model.
    Autorzy:
    Alsaad AMS; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ansari MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Alomar HA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
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    Źródło:
    International journal of molecular sciences [Int J Mol Sci] 2023 Aug 20; Vol. 24 (16). Date of Electronic Publication: 2023 Aug 20.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Encephalomyelitis, Autoimmune, Experimental*/drug therapy
    Neurodegenerative Diseases*
    Animals ; Mice ; Granulocyte-Macrophage Colony-Stimulating Factor ; Interleukin-6 ; Receptors, Histamine H4 ; Tumor Necrosis Factor-alpha ; NF-kappa B ; Adaptor Proteins, Signal Transducing ; Inflammation/drug therapy ; Antigens, CD19 ; Disease Progression
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Upregulation of Inflammatory Mediators in Peripheral Blood CD40 Cells in Children with Autism Spectrum Disorder.
    Autorzy:
    Aldossari AA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ansari MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Al-Ayadhi LY; Department of Physiology, College of Medicine, King Saud University, Riyadh 11451, Saudi Arabia.
    Alanazi MM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Shahid M; Department of Pharmaceutics, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Alwetaid MY; Department of Botany and Microbiology, College of Science, King Saud University, Riyadh 11451, Saudi Arabia.
    Hussein MH; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
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    Źródło:
    International journal of molecular sciences [Int J Mol Sci] 2023 Apr 19; Vol. 24 (8). Date of Electronic Publication: 2023 Apr 19.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Autism Spectrum Disorder*
    Humans ; Child ; Child, Preschool ; Interleukin-17/metabolism ; Up-Regulation ; Nuclear Receptor Subfamily 1, Group F, Member 3/metabolism ; Tumor Necrosis Factor-alpha/genetics ; Tumor Necrosis Factor-alpha/metabolism ; Cytokines/metabolism ; Receptors, Chemokine/metabolism ; Transcription Factors/metabolism ; CD40 Antigens/genetics ; CD40 Antigens/metabolism ; RNA, Messenger/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Cadmium Exposure Is Associated with Behavioral Deficits and Neuroimmune Dysfunction in BTBR T Itpr3tf/J Mice.
    Autorzy:
    Alanazi MM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ansari MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Al-Mazroua HA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Aldossari AA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Almutairi MM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Albekairi TH; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Hussein MH; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Al-Hamamah MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
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    Źródło:
    International journal of molecular sciences [Int J Mol Sci] 2023 Mar 31; Vol. 24 (7). Date of Electronic Publication: 2023 Mar 31.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Interleukin-17*/metabolism
    Autism Spectrum Disorder*/metabolism
    Mice ; Animals ; Cadmium/toxicity ; Nuclear Receptor Subfamily 1, Group F, Member 3/genetics ; Nuclear Receptor Subfamily 1, Group F, Member 3/metabolism ; Tumor Necrosis Factor-alpha/genetics ; Mice, Inbred C57BL ; Mice, Inbred Strains ; RNA, Messenger/metabolism ; Disease Models, Animal
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Pharmacological Inhibition of STAT3 by Stattic Ameliorates Clinical Symptoms and Reduces Autoinflammation in Myeloid, Lymphoid, and Neuronal Tissue Compartments in Relapsing-Remitting Model of Experimental Autoimmune Encephalomyelitis in SJL/J Mice.
    Autorzy:
    Alhazzani K; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Al-Harbi NO; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Sarawi W; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Alqarni SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Algahtani M; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
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    Źródło:
    Pharmaceutics [Pharmaceutics] 2021 Jun 22; Vol. 13 (7). Date of Electronic Publication: 2021 Jun 22.
    Typ publikacji:
    Journal Article
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Chemokine Receptor 5 Antagonism Causes Reduction in Joint Inflammation in a Collagen-Induced Arthritis Mouse Model.
    Autorzy:
    Ansari MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.; Department of Pharmacology and Toxicology, Faculty of Pharmacy, Al-Azhar University, Nasr City, Cairo 11884, Egypt.
    Shahid M; Department of Pharmaceutics, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Alyousef FS; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Alswailem MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Alqinyah M; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
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    Źródło:
    Molecules (Basel, Switzerland) [Molecules] 2021 Mar 25; Vol. 26 (7). Date of Electronic Publication: 2021 Mar 25.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Arthritis, Experimental/*drug therapy
    Maraviroc/*pharmacology
    Receptors, CCR5/*immunology
    Animals ; Arthritis, Experimental/immunology ; Arthritis, Experimental/pathology ; CD8-Positive T-Lymphocytes/immunology ; CD8-Positive T-Lymphocytes/pathology ; Cytokines/immunology ; GATA3 Transcription Factor/immunology ; Male ; Mice ; Nuclear Receptor Subfamily 1, Group F, Member 3/immunology ; Th17 Cells/immunology ; Th17 Cells/pathology ; Toll-Like Receptor 9/immunology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    5-Aminoisoquinolinone, a PARP-1 Inhibitor, Ameliorates Immune Abnormalities through Upregulation of Anti-Inflammatory and Downregulation of Inflammatory Parameters in T Cells of BTBR Mouse Model of Autism.
    Autorzy:
    Alhosaini K; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ansari MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Alhazzani K; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Albekairi TH; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Al-Mazroua HA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Mahmood HM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh 11451, Saudi Arabia.
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    Źródło:
    Brain sciences [Brain Sci] 2021 Feb 17; Vol. 11 (2). Date of Electronic Publication: 2021 Feb 17.
    Typ publikacji:
    Journal Article
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    The role of NF-κB and AhR transcription factors in lead-induced lung toxicity in human lung cancer A549 cells.
    Autorzy:
    Attafi IM; Department of Pharmacology & Toxicology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.; Poison Control & Medical Forensic Chemistry Center, Jazan Health Affairs, Jazan, Saudi Arabia.
    Bakheet SA; Department of Pharmacology & Toxicology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.
    Korashy HM; Department of Pharmaceutical Sciences, College of Pharmacy, Qatar University, Doha, Qatar.
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    Źródło:
    Toxicology mechanisms and methods [Toxicol Mech Methods] 2020 Mar; Vol. 30 (3), pp. 197-207. Date of Electronic Publication: 2019 Nov 20.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Basic Helix-Loop-Helix Transcription Factors/*physiology
    Lead/*toxicity
    Lung/*drug effects
    NF-kappa B/*physiology
    Receptors, Aryl Hydrocarbon/*physiology
    A549 Cells ; Apoptosis/drug effects ; Basic Helix-Loop-Helix Transcription Factors/antagonists & inhibitors ; Humans ; Lung/metabolism ; Lung/pathology ; NF-kappa B/antagonists & inhibitors ; Oxidative Stress/drug effects ; Receptors, Aryl Hydrocarbon/antagonists & inhibitors ; Resveratrol/pharmacology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Elevated IL-16 expression is associated with development of immune dysfunction in children with autism.
    Autorzy:
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Kingdom of Saudi Arabia. s_.; College of Pharmacy, King Saud University, 2457, Riyadh, 11451, Saudi Arabia. s_.
    Ansari MA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Kingdom of Saudi Arabia.
    Nadeem A; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Kingdom of Saudi Arabia.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Kingdom of Saudi Arabia.
    Al-Ayadhi LY; Autism Research and Treatment Center, AL-Amodi Autism Research Chair, Department of Physiology, College of Medicine, King Saud University, Riyadh, Kingdom of Saudi Arabia.
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Kingdom of Saudi Arabia.; Department of Pharmacology and Toxicology, College of Pharmacy, Al-Azhar University, Cairo, Egypt.
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    Źródło:
    Psychopharmacology [Psychopharmacology (Berl)] 2019 Feb; Vol. 236 (2), pp. 831-838. Date of Electronic Publication: 2018 Nov 19.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Autism Spectrum Disorder/*blood
    Autism Spectrum Disorder/*immunology
    Interleukin-16/*blood
    Autism Spectrum Disorder/diagnosis ; Biomarkers/blood ; Blotting, Western/methods ; Child ; Child, Preschool ; Female ; Flow Cytometry/methods ; Gene Expression ; Humans ; Interleukin-16/biosynthesis ; Interleukin-16/genetics ; Leukocytes, Mononuclear/metabolism ; Male
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Utility of Dexrazoxane for the Attenuation of Epirubicin-Induced Genetic Alterations in Mouse Germ Cells.
    Autorzy:
    Attia SM; Pharmacology and Toxicology Department, Faculty of Pharmacy, King Saud University, 11451, Riyadh, Saudi Arabia.; Pharmacology and Toxicology Department, Faculty of Pharmacy, Al-Azhar University, Nasr City, Cairo, Egypt.
    Ahmad SF; Pharmacology and Toxicology Department, Faculty of Pharmacy, King Saud University, 11451, Riyadh, Saudi Arabia.
    Ansaria MA; Pharmacology and Toxicology Department, Faculty of Pharmacy, King Saud University, 11451, Riyadh, Saudi Arabia.
    Nadeem A; Pharmacology and Toxicology Department, Faculty of Pharmacy, King Saud University, 11451, Riyadh, Saudi Arabia.
    Al-Shabanah OA; Pharmacology and Toxicology Department, Faculty of Pharmacy, King Saud University, 11451, Riyadh, Saudi Arabia.
    Al-Harbi MM; Pharmacology and Toxicology Department, Faculty of Pharmacy, King Saud University, 11451, Riyadh, Saudi Arabia.
    Bakheet SA; Pharmacology and Toxicology Department, Faculty of Pharmacy, King Saud University, 11451, Riyadh, Saudi Arabia.
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    Źródło:
    PloS one [PLoS One] 2016 Sep 30; Vol. 11 (9), pp. e0163703. Date of Electronic Publication: 2016 Sep 30 (Print Publication: 2016).
    Typ publikacji:
    Journal Article
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Aneugenic effects of epirubicin in somatic and germinal cells of male mice.
    Autorzy:
    Attia SM; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia; Laboratory of Chemical and Clinical Pathology, Ministry of Health, Nasr City, Cairo, Egypt.
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.
    Okash RM; Department of Pharmacology and Toxicology, College of Pharmacy, Al-Azhar University, Cairo, Egypt.
    Bakheet SA; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.
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    Źródło:
    PloS one [PLoS One] 2014 Oct 10; Vol. 9 (10), pp. e109942. Date of Electronic Publication: 2014 Oct 10 (Print Publication: 2014).
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Aneugens/*pharmacology
    Bone Marrow Cells/*drug effects
    Epirubicin/*pharmacology
    Meiosis/*drug effects
    Spermatozoa/*drug effects
    Animals ; In Situ Hybridization, Fluorescence ; Male ; Mice
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Anti-inflammatory effect of Euphorbia hirta in an adjuvant-induced arthritic murine model.
    Autorzy:
    Ahmad SF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University , PO Box 11451, Riyadh , Saudi Arabia.
    Attia SM
    Bakheet SA
    Ashour AE
    Zoheir KM
    Abd-Allah AR
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    Źródło:
    Immunological investigations [Immunol Invest] 2014; Vol. 43 (3), pp. 197-211. Date of Electronic Publication: 2014 Jan 06.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Euphorbia*
    Arthritis, Experimental/*drug therapy
    Mycobacterium tuberculosis/*immunology
    Phytotherapy/*methods
    T-Lymphocytes/*immunology
    Animals ; Anti-Inflammatory Agents/administration & dosage ; Antigens, Bacterial/immunology ; Antigens, CD/metabolism ; Arthritis, Experimental/immunology ; Cells, Cultured ; Cytokines/metabolism ; Disease Models, Animal ; Female ; Humans ; Inflammation Mediators/metabolism ; Lymphocyte Activation/drug effects ; Mice ; Mice, Inbred BALB C ; Oils/administration & dosage ; Paraffin/administration & dosage ; Plant Extracts/administration & dosage
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Reversal of cisplatin-induced carnitine deficiency and energy starvation by propionyl-L-carnitine in rat kidney tissues.
    Autorzy:
    Aleisa AM; Department of Pharmacology, College of Pharmacy, King Saud University, Riyadh, Kingdom of Saudi Arabia.
    Al-Majed AA
    Al-Yahya AA
    Al-Rejaie SS
    Bakheet SA
    Al-Shabanah OA
    Sayed-Ahmed MM
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    Źródło:
    Clinical and experimental pharmacology & physiology [Clin Exp Pharmacol Physiol] 2007 Dec; Vol. 34 (12), pp. 1252-9.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Antineoplastic Agents*
    Cisplatin*
    Carnitine/*analogs & derivatives
    Carnitine/*deficiency
    Kidney Diseases/*chemically induced
    Kidney Diseases/*drug therapy
    Adenosine Diphosphate/metabolism ; Adenosine Triphosphate/metabolism ; Animals ; Blood Urea Nitrogen ; Carnitine/blood ; Carnitine/metabolism ; Carnitine/therapeutic use ; Creatinine/blood ; Glutathione/metabolism ; Kidney Diseases/metabolism ; Kidney Diseases/pathology ; Male ; Nitrates/metabolism ; Nitrites/metabolism ; Protective Agents/therapeutic use ; Rats ; Rats, Wistar ; Thiobarbituric Acid Reactive Substances/metabolism
    Czasopismo naukowe
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