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    Tytuł:
    Cis-regulatory atlas of primary human CD4+ T cells.
    Autorzy:
    Stefan K; Division of Allergy & Immunology, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, MLC 7028, Cincinnati, OH, 45229-3026, USA.; Medical Scientist Training Program (MSTP), University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA.
    Barski A; Division of Allergy & Immunology, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, MLC 7028, Cincinnati, OH, 45229-3026, USA. Artem.Barski@cchmc.org.; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, 45229-3026, USA. Artem.Barski@cchmc.org.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA. Artem.Barski@cchmc.org.
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    Źródło:
    BMC genomics [BMC Genomics] 2023 May 11; Vol. 24 (1), pp. 253. Date of Electronic Publication: 2023 May 11.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    CD4-Positive T-Lymphocytes*
    Enhancer Elements, Genetic*
    Nucleosomes*
    Humans ; Cell Differentiation ; Cell Line
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    maxATAC: Genome-scale transcription-factor binding prediction from ATAC-seq with deep neural networks.
    Autorzy:
    Cazares TA; Immunology Graduate Program, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Rizvi FW; Systems Biology and Physiology Graduate Program, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Iyer B; Division of Biomedical Informatics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Electrical Engineering and Computer Science, University of Cincinnati, Cincinnati, Ohio, United States of America.
    Chen X; The Center for Autoimmune Genetics and Etiology (CAGE), Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Kotliar M; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Bejjani AT; Molecular and Developmental Biology Graduate Program, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Wayman JA; Division of Immunobiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Donmez O; The Center for Autoimmune Genetics and Etiology (CAGE), Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Wronowski B; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Parameswaran S; The Center for Autoimmune Genetics and Etiology (CAGE), Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Kottyan LC; The Center for Autoimmune Genetics and Etiology (CAGE), Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Barski A; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Weirauch MT; Division of Biomedical Informatics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; The Center for Autoimmune Genetics and Etiology (CAGE), Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Division of Developmental Biology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Prasath VBS; Division of Biomedical Informatics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Electrical Engineering and Computer Science, University of Cincinnati, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Miraldi ER; Division of Biomedical Informatics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Electrical Engineering and Computer Science, University of Cincinnati, Cincinnati, Ohio, United States of America.; Division of Immunobiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
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    Źródło:
    PLoS computational biology [PLoS Comput Biol] 2023 Jan 31; Vol. 19 (1), pp. e1010863. Date of Electronic Publication: 2023 Jan 31 (Print Publication: 2023).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Chromatin Immunoprecipitation Sequencing*
    High-Throughput Nucleotide Sequencing*/methods
    Nerve Net*
    Humans ; Chromatin/genetics ; Deoxyribonucleases/genetics ; Sequence Analysis, DNA/methods
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    KMT2C/D COMPASS complex-associated diseases [K CD COM-ADs]: an emerging class of congenital regulopathies.
    Autorzy:
    Lavery WJ; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center (CCHMC), 3333 Burnet Avenue, Cincinnati, OH, 45229-3026, USA.
    Barski A; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center (CCHMC), 3333 Burnet Avenue, Cincinnati, OH, 45229-3026, USA.; Division of Human Genetics, CCHMC, Cincinnati, OH, USA.
    Wiley S; Division of Developmental and Behavioral Pediatrics, CCHMC, Cincinnati, OH, USA.
    Schorry EK; Division of Human Genetics, CCHMC, Cincinnati, OH, USA.
    Lindsley AW; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center (CCHMC), 3333 Burnet Avenue, Cincinnati, OH, 45229-3026, USA. .
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    Źródło:
    Clinical epigenetics [Clin Epigenetics] 2020 Jan 10; Vol. 12 (1), pp. 10. Date of Electronic Publication: 2020 Jan 10.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
    MeSH Terms:
    Abnormalities, Multiple/*genetics
    Craniofacial Abnormalities/*genetics
    DNA-Binding Proteins/*genetics
    Face/*abnormalities
    Heart Defects, Congenital/*genetics
    Hematologic Diseases/*genetics
    Intellectual Disability/*genetics
    Neoplasm Proteins/*genetics
    Rubinstein-Taybi Syndrome/*genetics
    Vestibular Diseases/*genetics
    Abnormalities, Multiple/diagnosis ; Chromosome Deletion ; Chromosomes, Human, Pair 9/genetics ; Craniofacial Abnormalities/diagnosis ; DNA-Binding Proteins/metabolism ; Female ; Heart Defects, Congenital/diagnosis ; Hematologic Diseases/diagnosis ; Humans ; Intellectual Disability/diagnosis ; Male ; Neoplasm Proteins/metabolism ; Phenotype ; Rubinstein-Taybi Syndrome/diagnosis ; Vestibular Diseases/diagnosis
    SCR Disease Name:
    Kabuki syndrome; Kleefstra Syndrome
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    CWL-Airflow: a lightweight pipeline manager supporting Common Workflow Language.
    Autorzy:
    Kotliar M; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
    Kartashov AV; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
    Barski A; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.; Division of Human Genetics, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
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    Źródło:
    GigaScience [Gigascience] 2019 Jul 01; Vol. 8 (7).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural
    MeSH Terms:
    Software*
    Workflow*
    Chromatin Immunoprecipitation Sequencing/*methods
    Genomics/*methods
    Animals ; Big Data ; Humans
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Genetic, Inflammatory, and Epithelial Cell Differentiation Factors Control Expression of Human Calpain-14.
    Autorzy:
    Miller DE; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Forney C; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Rochman M; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Cranert S; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Habel J; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Rymer J; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Lynch A; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Schroeder C; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Lee J; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Sauder A; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Smith Q; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Chawla M; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Trimarchi MP; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Lu X; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Fjellman E; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Brusilovsky M; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Barski A; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.; Department of Pediatrics, University of Cincinnati, College of Medicine, Ohio 45229.
    Waggoner S; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.; Department of Pediatrics, University of Cincinnati, College of Medicine, Ohio 45229.
    Weirauch MT; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.; Department of Pediatrics, University of Cincinnati, College of Medicine, Ohio 45229.; Divisions of Biomedical Informatics and Developmental Biology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.
    Rothenberg ME; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.; Department of Pediatrics, University of Cincinnati, College of Medicine, Ohio 45229.
    Kottyan LC; Center for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229 .; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229.; Department of Pediatrics, University of Cincinnati, College of Medicine, Ohio 45229.
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    Źródło:
    G3 (Bethesda, Md.) [G3 (Bethesda)] 2019 Mar 07; Vol. 9 (3), pp. 729-736. Date of Electronic Publication: 2019 Mar 07.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Gene Expression Regulation*
    Polymorphism, Single Nucleotide*
    Calpain/*genetics
    Eosinophilic Esophagitis/*genetics
    Epithelial Cells/*enzymology
    Interleukin-13/*metabolism
    STAT6 Transcription Factor/*metabolism
    Cell Line ; Eosinophilic Esophagitis/metabolism ; Genetic Predisposition to Disease ; Humans ; Inflammation ; Interleukin-4/metabolism ; Promoter Regions, Genetic
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    RNF8 and SCML2 cooperate to regulate ubiquitination and H3K27 acetylation for escape gene activation on the sex chromosomes.
    Autorzy:
    Adams SR; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.; Department of Obstetrics and Gynecology, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Maezawa S; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Alavattam KG; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Abe H; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Sakashita A; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Shroder M; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Broering TJ; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Sroga Rios J; Department of Obstetrics and Gynecology, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Thomas MA; Department of Obstetrics and Gynecology, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Lin X; State Key Laboratory of Genetic Engineering, Institute of Genetics, Collaborative Innovation Center of Genetics and Development, School of Life Sciences, Fudan University, Shanghai, China.
    Price CM; Department of Cancer Biology, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Barski A; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.; Division of Allergy and Immunology, Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Andreassen PR; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.; Division of Experimental Hematology and Cancer Biology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Namekawa SH; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
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    Źródło:
    PLoS genetics [PLoS Genet] 2018 Feb 20; Vol. 14 (2), pp. e1007233. Date of Electronic Publication: 2018 Feb 20 (Print Publication: 2018).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Histones/*metabolism
    Polycomb-Group Proteins/*physiology
    Sex Chromosomes/*genetics
    Transcriptional Activation/*genetics
    Ubiquitin-Protein Ligases/*physiology
    Ubiquitination/*genetics
    Acetylation ; Animals ; Female ; Male ; Meiosis/genetics ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Sex Chromosomes/metabolism ; Spermatids/physiology ; Spermatogenesis/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression.
    Autorzy:
    Yue M; Division of Reproductive Sciences, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Ogawa A; Division of Reproductive Sciences, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Yamada N; Division of Reproductive Sciences, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Charles Richard JL; Division of Reproductive Sciences, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
    Barski A; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.; Division of Allergy & Immunology and Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.
    Ogawa Y; Division of Reproductive Sciences, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, United States of America.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America.
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    Źródło:
    PLoS genetics [PLoS Genet] 2017 Jul 07; Vol. 13 (7), pp. e1006890. Date of Electronic Publication: 2017 Jul 07 (Print Publication: 2017).
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Transcription, Genetic*
    DNA-Binding Proteins/*genetics
    Nuclear Proteins/*genetics
    RNA, Long Noncoding/*genetics
    Transcription Factors/*genetics
    X Chromosome Inactivation/*genetics
    Animals ; Cell Differentiation/genetics ; DNA-Binding Proteins/biosynthesis ; Embryonic Stem Cells/metabolism ; Exons/genetics ; Gene Deletion ; Gene Expression Regulation ; Gene Silencing ; Histone Code/genetics ; Mice ; Nuclear Proteins/biosynthesis ; Transcription Factors/biosynthesis ; X Chromosome/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Rapid Recall Ability of Memory T cells is Encoded in their Epigenome.
    Autorzy:
    Barski A; Divisions of Allergy &Immunology and Human Genetics, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, 45229, USA.
    Cuddapah S; Department of Environmental Medicine, New York University School of Medicine, NY, 10987, USA.
    Kartashov AV; Divisions of Allergy &Immunology and Human Genetics, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, 45229, USA.
    Liu C; Divisions of Allergy &Immunology and Human Genetics, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, 45229, USA.
    Imamichi H; Clinical and Molecular Retrovirology Section, Laboratory of Immunoregulation, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, 20892, USA.
    Yang W; Department of Physics, The George Washington University, D.C., 20052, USA.
    Peng W; Department of Physics, The George Washington University, D.C., 20052, USA.
    Lane HC; Clinical and Molecular Retrovirology Section, Laboratory of Immunoregulation, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, 20892, USA.
    Zhao K; Systems Biology Center, National Heart, Lung and Blood Institute, National Institutes of Health, Bethesda, MD 20892, USA.
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    Źródło:
    Scientific reports [Sci Rep] 2017 Jan 05; Vol. 7, pp. 39785. Date of Electronic Publication: 2017 Jan 05.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural
    MeSH Terms:
    Epigenesis, Genetic*
    Immunologic Memory*
    T-Lymphocytes/*immunology
    Cell Differentiation ; Cytokines/genetics ; Cytokines/metabolism ; Histone Code ; Humans
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Polycomb repressive complex 1 controls uterine decidualization.
    Autorzy:
    Bian F; Division of Reproductive Sciences, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH 49267, USA.; Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.
    Gao F; Division of Reproductive Sciences, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH 49267, USA.; Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.
    Kartashov AV; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH 49267, USA.; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.
    Jegga AG; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH 49267, USA.; Biomedical Informatics, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.
    Barski A; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH 49267, USA.; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.
    Das SK; Division of Reproductive Sciences, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH 49267, USA.; Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.
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    Źródło:
    Scientific reports [Sci Rep] 2016 May 16; Vol. 6, pp. 26061. Date of Electronic Publication: 2016 May 16.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Decidua/*pathology
    Ligases/*metabolism
    Polycomb Repressive Complex 1/*metabolism
    Polycomb-Group Proteins/*metabolism
    Repressor Proteins/*metabolism
    Stromal Cells/*physiology
    Ubiquitin-Protein Ligases/*metabolism
    Uterus/*pathology
    Animals ; Bone Morphogenetic Protein 2/genetics ; Cells, Cultured ; Epigenetic Repression ; Female ; Gene Expression Regulation ; Histones/metabolism ; Ligases/genetics ; Male ; Mice ; Mice, Knockout ; Polycomb Repressive Complex 1/genetics ; Polyploidy ; RNA, Small Interfering/genetics ; Repressor Proteins/genetics ; Sequence Analysis, RNA ; Ubiquitin-Protein Ligases/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Poised chromatin and bivalent domains facilitate the mitosis-to-meiosis transition in the male germline.
    Autorzy:
    Sin HS; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, 45229, USA.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, 49229, USA.; Present address: Department of Developmental Biology, Department of Genetics, Stanford University School of Medicine, Stanford, CA, 94305, USA.
    Kartashov AV; Division of Allergy and Immunology, Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, 45229, USA.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, 49229, USA.
    Hasegawa K; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, 45229, USA.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, 49229, USA.; Present address: Department of Medicine, Stanford University School of Medicine, Stanford, CA, 94305, USA.
    Barski A; Division of Allergy and Immunology, Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, 45229, USA. artem.barski@cchmc.org.; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, 49229, USA. artem.barski@cchmc.org.
    Namekawa SH; Division of Reproductive Sciences, Division of Developmental Biology, Perinatal Institute, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, 45229, USA. .; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, 49229, USA. .
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    Źródło:
    BMC biology [BMC Biol] 2015 Jul 22; Vol. 13, pp. 53. Date of Electronic Publication: 2015 Jul 22.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Epigenesis, Genetic*
    Meiosis*
    Mitosis*
    Spermatogenesis*
    Chromatin/*genetics
    Germ Cells/*cytology
    Sex Chromosomes/*genetics
    Animals ; Cells, Cultured ; Chromatin/chemistry ; Gene Expression Regulation, Developmental ; Genes, X-Linked ; Germ Cells/metabolism ; Histones/chemistry ; Histones/genetics ; Male ; Mice, Inbred C57BL ; Sex Chromosomes/chemistry ; Transcriptional Activation ; Transcriptome ; X Chromosome Inactivation
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Functional characterization of human T cell hyporesponsiveness induced by CTLA4-Ig.
    Autorzy:
    Rochman Y; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, Ohio, United States of America; Division of Immunobiology, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, Ohio, United States of America.
    Yukawa M; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, Ohio, United States of America.
    Kartashov AV; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, Ohio, United States of America.
    Barski A; Division of Allergy and Immunology, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, Ohio, United States of America; Division of Human Genetics, Cincinnati Children's Hospital Medical Center and Department of Pediatrics, College of Medicine, University of Cincinnati, Cincinnati, Ohio, United States of America.
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    Źródło:
    PloS one [PLoS One] 2015 Apr 10; Vol. 10 (4), pp. e0122198. Date of Electronic Publication: 2015 Apr 10 (Print Publication: 2015).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural
    MeSH Terms:
    CTLA-4 Antigen/*immunology
    Immunoglobulins/*immunology
    Recombinant Fusion Proteins/*immunology
    T-Lymphocytes/*immunology
    T-Lymphocytes/*metabolism
    Antigens, Surface/genetics ; Antigens, Surface/metabolism ; CD4-Positive T-Lymphocytes/immunology ; CD4-Positive T-Lymphocytes/metabolism ; CTLA-4 Antigen/genetics ; Clonal Anergy ; Cyclin-Dependent Kinase Inhibitor p27/metabolism ; Cytokines/biosynthesis ; Gene Expression ; Gene Expression Profiling ; Humans ; Immunoglobulins/genetics ; Immunologic Memory ; Immunophenotyping ; Lymphocyte Activation/drug effects ; Lymphocyte Activation/immunology ; Phenotype ; Receptors, Antigen, T-Cell/metabolism ; Recombinant Fusion Proteins/pharmacology ; Signal Transduction ; T-Lymphocyte Subsets/drug effects ; T-Lymphocyte Subsets/immunology ; T-Lymphocyte Subsets/metabolism ; T-Lymphocytes/drug effects ; T-Lymphocytes, Regulatory/immunology ; T-Lymphocytes, Regulatory/metabolism ; Transcriptome ; Ubiquitin-Protein Ligases/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Pol II and its associated epigenetic marks are present at Pol III-transcribed noncoding RNA genes.
    Autorzy:
    Barski A; Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Maryland, USA.
    Chepelev I
    Liko D
    Cuddapah S
    Fleming AB
    Birch J
    Cui K
    White RJ
    Zhao K
    Pokaż więcej
    Źródło:
    Nature structural & molecular biology [Nat Struct Mol Biol] 2010 May; Vol. 17 (5), pp. 629-34. Date of Electronic Publication: 2010 Apr 25.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Epigenesis, Genetic*
    Genome, Human*
    DNA Polymerase II/*genetics
    RNA Polymerase III/*genetics
    Binding Sites ; Cell Line ; Chromatin/genetics ; DNA Polymerase II/metabolism ; Gene Expression ; Genes ; Genetic Loci ; Humans ; RNA Polymerase III/analysis ; RNA Polymerase III/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Combinatorial patterns of histone acetylations and methylations in the human genome.
    Autorzy:
    Wang Z; Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, US National Institutes of Health, Bethesda, Maryland 20892, USA.
    Zang C
    Rosenfeld JA
    Schones DE
    Barski A
    Cuddapah S
    Cui K
    Roh TY
    Peng W
    Zhang MQ
    Zhao K
    Pokaż więcej
    Źródło:
    Nature genetics [Nat Genet] 2008 Jul; Vol. 40 (7), pp. 897-903. Date of Electronic Publication: 2008 Jun 15.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Genome, Human*/physiology
    Histone Acetyltransferases/*metabolism
    Histone-Lysine N-Methyltransferase/*metabolism
    Histones/*metabolism
    Acetylation ; Cells, Cultured ; Chromosome Mapping ; Cluster Analysis ; Enhancer Elements, Genetic ; Histone Methyltransferases ; Humans ; Methylation ; Promoter Regions, Genetic ; Protein Binding ; Protein Methyltransferases
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Identification of novel androgen receptor target genes in prostate cancer.
    Autorzy:
    Jariwala U; Department of Biochemistry and Molecular Biology, Keck School of Medicine, University of Southern California, Los Angeles, USA. />Prescott J
    Jia L
    Barski A
    Pregizer S
    Cogan JP
    Arasheben A
    Tilley WD
    Scher HI
    Gerald WL
    Buchanan G
    Coetzee GA
    Frenkel B
    Pokaż więcej
    Źródło:
    Molecular cancer [Mol Cancer] 2007 Jun 06; Vol. 6, pp. 39. Date of Electronic Publication: 2007 Jun 06.
    Typ publikacji:
    Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Androgens*
    Gene Expression Regulation, Neoplastic*/drug effects
    Adenocarcinoma/*genetics
    Neoplasm Proteins/*genetics
    Prostatic Neoplasms/*genetics
    Receptors, Androgen/*physiology
    Adenocarcinoma/metabolism ; Binding Sites ; Cell Adhesion Molecules/biosynthesis ; Cell Adhesion Molecules/genetics ; Cell Line, Tumor/drug effects ; Cell Line, Tumor/metabolism ; Chromosomes, Human/drug effects ; Chromosomes, Human/metabolism ; Dihydrotestosterone/pharmacology ; Extracellular Matrix Proteins/biosynthesis ; Extracellular Matrix Proteins/genetics ; Gene Expression Profiling ; Glutathione Transferase/biosynthesis ; Glutathione Transferase/genetics ; Humans ; Intracellular Signaling Peptides and Proteins/genetics ; Male ; Mucin-6 ; Mucins/biosynthesis ; Mucins/genetics ; Neoplasm Proteins/biosynthesis ; Neoplasms, Hormone-Dependent/genetics ; Neoplasms, Hormone-Dependent/metabolism ; Nuclear Proteins/biosynthesis ; Nuclear Proteins/genetics ; Oligonucleotide Array Sequence Analysis ; Ornithine-Oxo-Acid Transaminase/biosynthesis ; Ornithine-Oxo-Acid Transaminase/genetics ; Prostatic Neoplasms/metabolism ; Protein Kinase C-delta/biosynthesis ; Protein Kinase C-delta/genetics ; Pyrroline Carboxylate Reductases/biosynthesis ; Pyrroline Carboxylate Reductases/genetics ; Receptors, Androgen/genetics ; TRPV Cation Channels/biosynthesis ; TRPV Cation Channels/genetics ; Transcription, Genetic ; delta-1-Pyrroline-5-Carboxylate Reductase
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-14 z 14

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