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Wyszukujesz frazę ""CCR8"" wg kryterium: Temat


Tytuł :
ARNT-dependent CCR8 reprogrammed LDH isoform expression correlates with poor clinical outcomes of prostate cancer.
Autorzy :
Chen G; Department of Urology, The First Affiliated Hospital of Jinan University, Guangzhou, Guangdong, China.
Cai ZD; Guangdong Provincial Institute of Nephrology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Lin ZY; Department of Urology, The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, Guangdong, China.
Wang C; School of pharmaceutical sciences, Wenzhou Medical University, Wenzhou, China.
Liang YX; Guangdong Key Laboratory of Clinical Molecular Medicine and Diagnostics, Department of Urology, Guangzhou First People's Hospital, School of Medicine, South China University of Technology, Guangzhou, Guangdong, China.
Han ZD; Guangdong Key Laboratory of Clinical Molecular Medicine and Diagnostics, Department of Urology, Guangzhou First People's Hospital, School of Medicine, South China University of Technology, Guangzhou, Guangdong, China.
He HC; Guangdong Key Laboratory of Urology, Department of Urology, Minimally Invasive Surgery Center, The First Affiliated Hospital, Guangzhou Institute of Urology, Guangzhou, Guangdong, China.
Mo RJ; Guangdong Key Laboratory of Clinical Molecular Medicine and Diagnostics, Department of Urology, Guangzhou First People's Hospital, School of Medicine, South China University of Technology, Guangzhou, Guangdong, China.
Lu JM; Guangdong Key Laboratory of Clinical Molecular Medicine and Diagnostics, Department of Urology, Guangzhou First People's Hospital, School of Medicine, South China University of Technology, Guangzhou, Guangdong, China.
Pan B; Department of Urology, The First Affiliated Hospital of Jinan University, Guangzhou, Guangdong, China.
Wu CL; Department of Pathology and Urology, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts.
Wang F; Institute of Biosciences and Technology, Texas A&M Health Science Center, Houston, Texas.
Zhong WD; Department of Urology, The First Affiliated Hospital of Jinan University, Guangzhou, Guangdong, China.; Guangdong Provincial Institute of Nephrology, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Key Laboratory of Clinical Molecular Medicine and Diagnostics, Department of Urology, Guangzhou First People's Hospital, School of Medicine, South China University of Technology, Guangzhou, Guangdong, China.
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Źródło :
Molecular carcinogenesis [Mol Carcinog] 2020 Aug; Vol. 59 (8), pp. 897-907. Date of Electronic Publication: 2020 Apr 22.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Gene Expression Regulation, Neoplastic*
Aryl Hydrocarbon Receptor Nuclear Translocator/*metabolism
Biomarkers, Tumor/*metabolism
Chemokines, CC/*metabolism
L-Lactate Dehydrogenase/*metabolism
Prostatic Neoplasms/*pathology
Receptors, CCR8/*metabolism
Apoptosis ; Aryl Hydrocarbon Receptor Nuclear Translocator/genetics ; Biomarkers, Tumor/genetics ; Cell Proliferation ; Chemokines, CC/genetics ; Humans ; Isoenzymes ; L-Lactate Dehydrogenase/genetics ; Male ; Prognosis ; Prostatic Neoplasms/genetics ; Prostatic Neoplasms/metabolism ; Receptors, CCR8/genetics ; Survival Rate ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł :
Upregulation and pathogenic roles of CCL18-CCR8 axis in IgG4-related disease.
Autorzy :
Tsuboi H; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Iizuka-Koga M; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.; Department of Microbiology and Immunology, Keio University School of Medicine, Tokyo, Japan.
Asashima H; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Takahashi H; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Kudo H; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Ono Y; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.; Section of Oral and Maxillofacial Oncology, Division of Maxillofacial Diagnostic and Surgical Sciences, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Honda F; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Iizuka A; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Segawa S; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Abe S; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Yagishita M; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Yokosawa M; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Kondo Y; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Moriyama M; Section of Oral and Maxillofacial Oncology, Division of Maxillofacial Diagnostic and Surgical Sciences, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Matsumoto I; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
Nakamura S; Section of Oral and Maxillofacial Oncology, Division of Maxillofacial Diagnostic and Surgical Sciences, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Sumida T; Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki, Japan.
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Źródło :
Modern rheumatology [Mod Rheumatol] 2020 Jul; Vol. 30 (4), pp. 729-737. Date of Electronic Publication: 2019 Aug 08.
Typ publikacji :
Journal Article
MeSH Terms :
Chemokines, CC/*blood
Immunoglobulin G4-Related Disease/*metabolism
Receptors, CCR8/*blood
Adult ; Chemokines, CC/metabolism ; Female ; Humans ; Immunoglobulin G4-Related Disease/blood ; Leukocytes/metabolism ; Male ; Middle Aged ; Receptors, CCR8/metabolism ; Salivary Glands, Minor/metabolism ; Up-Regulation
Czasopismo naukowe
Tytuł :
A CCL1/CCR8-dependent feed-forward mechanism drives ILC2 functions in type 2-mediated inflammation.
Autorzy :
Knipfer L; Department of Medicine 1, University Hospital Center, Friedrich-Alexander University, Erlangen-Nuremberg, Germany.
Schulz-Kuhnt A; Department of Medicine 1, University Hospital Center, Friedrich-Alexander University, Erlangen-Nuremberg, Germany.
Kindermann M; Department of Medicine 1, University Hospital Center, Friedrich-Alexander University, Erlangen-Nuremberg, Germany.
Greif V; Department of Medicine 1, University Hospital Center, Friedrich-Alexander University, Erlangen-Nuremberg, Germany.
Symowski C; Department of Infection Biology, University Hospital Center, Friedrich-Alexander University, Erlangen-Nuremberg, Germany.
Voehringer D; Department of Infection Biology, University Hospital Center, Friedrich-Alexander University, Erlangen-Nuremberg, Germany.
Neurath MF; Department of Medicine 1, University Hospital Center, Friedrich-Alexander University, Erlangen-Nuremberg, Germany.
Atreya I; Department of Medicine 1, University Hospital Center, Friedrich-Alexander University, Erlangen-Nuremberg, Germany.
Wirtz S; Department of Medicine 1, University Hospital Center, Friedrich-Alexander University, Erlangen-Nuremberg, Germany .
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Źródło :
The Journal of experimental medicine [J Exp Med] 2019 Dec 02; Vol. 216 (12), pp. 2763-2777. Date of Electronic Publication: 2019 Sep 19.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Immunity, Innate*
Chemokine CCL1/*metabolism
Inflammation/*etiology
Inflammation/*metabolism
Lymphocyte Subsets/*immunology
Lymphocyte Subsets/*metabolism
Receptors, CCR8/*metabolism
Animals ; Autocrine Communication ; Biomarkers ; Cell Movement/genetics ; Cell Movement/immunology ; Cytokines/metabolism ; Gene Expression ; Helminths/immunology ; Host-Parasite Interactions/genetics ; Host-Parasite Interactions/immunology ; Humans ; Immunophenotyping ; Inflammation Mediators/metabolism ; Mice ; Mice, Knockout ; Receptors, CCR8/genetics
Czasopismo naukowe
Tytuł :
CCR8 blockade primes anti-tumor immunity through intratumoral regulatory T cells destabilization in muscle-invasive bladder cancer.
Autorzy :
Wang T; Department of Urology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200080, China.
Zhou Q; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Fudan University, Shanghai, 200032, China.
Zeng H; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Fudan University, Shanghai, 200032, China.
Zhang H; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Fudan University, Shanghai, 200032, China.
Liu Z; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Fudan University, Shanghai, 200032, China.
Shao J; Department of Urology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200080, China.
Wang Z; Department of Urology, Zhongshan Hospital, Fudan University, Shanghai, China.
Xiong Y; Department of Urology, Zhongshan Hospital, Fudan University, Shanghai, China.
Wang J; Department of Urology, Zhongshan Hospital, Fudan University, Shanghai, China.
Bai Q; Department of Urology, Zhongshan Hospital, Fudan University, Shanghai, China.
Xia Y; Department of Urology, Zhongshan Hospital, Fudan University, Shanghai, China.
Wang Y; Department of Urology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Liu L; Department of Urology, Zhongshan Hospital, Fudan University, Shanghai, China.
Zhu Y; Department of Urology, Fudan University Shanghai Cancer Center, Shanghai, 200032, China.
Xu L; Department of Urology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Dai B; Department of Urology, Fudan University Shanghai Cancer Center, Shanghai, 200032, China.
Guo J; Department of Urology, Zhongshan Hospital, Fudan University, Shanghai, China.
Chang Y; Department of Urology, Fudan University Shanghai Cancer Center, Shanghai, 200032, China. .
Wang X; Department of Urology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200080, China. .
Xu J; Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Fudan University, Shanghai, 200032, China. .
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Źródło :
Cancer immunology, immunotherapy : CII [Cancer Immunol Immunother] 2020 Sep; Vol. 69 (9), pp. 1855-1867. Date of Electronic Publication: 2020 May 04.
Typ publikacji :
Journal Article
MeSH Terms :
Receptors, CCR8/*immunology
T-Lymphocytes, Regulatory/*immunology
Urinary Bladder Neoplasms/*immunology
Urinary Bladder Neoplasms/*therapy
Female ; Humans ; Immune Tolerance/immunology ; Immunotherapy/methods ; Male ; Prognosis ; Retrospective Studies ; Tumor Microenvironment/immunology
Czasopismo naukowe
Tytuł :
CCR8 leads to eosinophil migration and regulates neutrophil migration in murine allergic enteritis.
Autorzy :
Blanco-Pérez F; Vice President Research Group 'Molecular Allergology', Paul-Ehrlich-Institut, Langen, Germany.
Kato Y; Department of Pathology, Tokyo Women's Medical University, Tokyo, Japan.
Gonzalez-Menendez I; Institute of Pathology and Neuropathology, Eberhard Karls University of Tübingen and Comprehensive Cancer Center, University Hospital Tübingen, Tübingen, Germany.
Laiño J; Vice President Research Group 'Molecular Allergology', Paul-Ehrlich-Institut, Langen, Germany.
Ohbayashi M; Department of Nursing, Graduate School of Health Sciences, Toyohashi SOZO University, Toyohashi, Japan.
Burggraf M; Junior Research Group 1 Experimental Allergy Models', Paul-Ehrlich-Institut, Langen, Germany.
Krause M; Vice President Research Group 'Molecular Allergology', Paul-Ehrlich-Institut, Langen, Germany.
Kirberg J; Division of Immunology, Paul-Ehrlich-Institut, Langen, Germany.
Iwakura Y; Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science (TUS), Chiba, Japan.
Martella M; Institute of Pathology and Neuropathology, Eberhard Karls University of Tübingen and Comprehensive Cancer Center, University Hospital Tübingen, Tübingen, Germany.
Quintanilla-Martinez L; Institute of Pathology and Neuropathology, Eberhard Karls University of Tübingen and Comprehensive Cancer Center, University Hospital Tübingen, Tübingen, Germany.
Shibata N; Department of Pathology, Tokyo Women's Medical University, Tokyo, Japan.
Vieths S; Vice President Research Group 'Molecular Allergology', Paul-Ehrlich-Institut, Langen, Germany.
Scheurer S; Vice President Research Group 'Molecular Allergology', Paul-Ehrlich-Institut, Langen, Germany.
Toda M; Vice President Research Group 'Molecular Allergology', Paul-Ehrlich-Institut, Langen, Germany. .; Junior Research Group 1 Experimental Allergy Models', Paul-Ehrlich-Institut, Langen, Germany. .; Laboratory of Food and Biomolecular Science, Graduate School of Agricultural Science, Tohoku University, Sendai, Japan. .
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Źródło :
Scientific reports [Sci Rep] 2019 Jul 03; Vol. 9 (1), pp. 9608. Date of Electronic Publication: 2019 Jul 03.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Enteritis/*etiology
Eosinophils/*immunology
Eosinophils/*metabolism
Hypersensitivity/*complications
Neutrophils/*immunology
Neutrophils/*metabolism
Receptors, CCR8/*genetics
Animals ; Cytokines/blood ; Cytokines/metabolism ; Disease Models, Animal ; Disease Susceptibility ; Enteritis/metabolism ; Enteritis/pathology ; Female ; Immunoglobulin E/blood ; Immunoglobulin E/immunology ; Immunoglobulin G/blood ; Immunoglobulin G/immunology ; Intestinal Mucosa/immunology ; Intestinal Mucosa/metabolism ; Intestinal Mucosa/pathology ; Mice ; Mice, Knockout ; Receptors, CCR8/metabolism ; T-Lymphocyte Subsets/immunology ; T-Lymphocyte Subsets/metabolism
Czasopismo naukowe
Tytuł :
CCL18 enhances migration, invasion and EMT by binding CCR8 in bladder cancer cells.
Autorzy :
Liu X; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Xu X; Department of Urology, The Third Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Deng W; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Huang M; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Wu Y; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Zhou Z; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Zhu K; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Wang Y; Department of Emergency, The Second Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Cheng X; Department of Urology, The Second People's Hospital of Jingdezhen, Jingdezhen, Jiangxi 333000, P.R. China.
Zhou X; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Chen L; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Li Y; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Wang G; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Fu B; Department of Urology, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2019 Mar; Vol. 19 (3), pp. 1678-1686. Date of Electronic Publication: 2018 Dec 24.
Typ publikacji :
Journal Article
MeSH Terms :
Chemokines, CC/*genetics
Receptors, CCR8/*genetics
Urinary Bladder Neoplasms/*genetics
Vascular Endothelial Growth Factor C/*genetics
Cadherins/genetics ; Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; Chemokines, CC/antagonists & inhibitors ; Epithelial-Mesenchymal Transition/genetics ; Gene Expression Regulation, Neoplastic ; Humans ; Matrix Metalloproteinase 2/genetics ; Neoplasm Invasiveness/genetics ; Neoplasm Invasiveness/pathology ; Neoplasm Metastasis ; Protein Binding ; RNA, Small Interfering/pharmacology ; Receptors, CCR8/antagonists & inhibitors ; Signal Transduction/genetics ; Small Molecule Libraries/pharmacology ; Urinary Bladder Neoplasms/pathology
Czasopismo naukowe
Tytuł :
[Effect of acupuncture and moxibustion at "Feishu""Dazhui"and "Fengmen"on the expression of CC chemokine ligand 1 and CC chemokine receptor 8 in lung tissue of rats with asthma].
Autorzy :
Hu XJ; The Third Affiliated Hospital of Henan University of Traditional Chinese Medicine, Zhengzhou 450008, China.
Shao SJ; The Third Affiliated Hospital of Henan University of Traditional Chinese Medicine, Zhengzhou 450008, China.
Hua JS; The Third Affiliated Hospital of Henan University of Traditional Chinese Medicine, Zhengzhou 450008, China.
Wang PY; The Third Affiliated Hospital of Henan University of Traditional Chinese Medicine, Zhengzhou 450008, China.
Zhang J; The Third Affiliated Hospital of Henan University of Traditional Chinese Medicine, Zhengzhou 450008, China.
Ren Z; The Third Affiliated Hospital of Henan University of Traditional Chinese Medicine, Zhengzhou 450008, China.
Xu N; The Third Affiliated Hospital of Henan University of Traditional Chinese Medicine, Zhengzhou 450008, China.
Tian L; The Third Affiliated Hospital of Henan University of Traditional Chinese Medicine, Zhengzhou 450008, China.
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Źródło :
Zhen ci yan jiu = Acupuncture research [Zhen Ci Yan Jiu] 2020 May 25; Vol. 45 (5), pp. 363-7.
Typ publikacji :
Journal Article
MeSH Terms :
Acupuncture Therapy*
Asthma*
Moxibustion*
Animals ; Chemokine CCL1 ; Rats ; Rats, Sprague-Dawley ; Receptors, CCR8
Czasopismo naukowe
Tytuł :
The Chemokine Receptor CCR8 Promotes the Migration of Dendritic Cells into the Lymph Node Parenchyma to Initiate the Allergic Immune Response.
Autorzy :
Sokol CL; Center for Immunology and Inflammatory Diseases, Division of Rheumatology, Allergy, and Immunology, Massachusetts General Hospital and Harvard Medical School, Boston, MA 02114, USA.
Camire RB; Center for Immunology and Inflammatory Diseases, Division of Rheumatology, Allergy, and Immunology, Massachusetts General Hospital and Harvard Medical School, Boston, MA 02114, USA.
Jones MC; Center for Immunology and Inflammatory Diseases, Division of Rheumatology, Allergy, and Immunology, Massachusetts General Hospital and Harvard Medical School, Boston, MA 02114, USA.
Luster AD; Center for Immunology and Inflammatory Diseases, Division of Rheumatology, Allergy, and Immunology, Massachusetts General Hospital and Harvard Medical School, Boston, MA 02114, USA. Electronic address: .
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Źródło :
Immunity [Immunity] 2018 Sep 18; Vol. 49 (3), pp. 449-463.e6. Date of Electronic Publication: 2018 Aug 28.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Dendritic Cells/*physiology
Hypersensitivity/*immunology
Lymph Nodes/*immunology
Receptors, CCR7/*metabolism
Receptors, CCR8/*metabolism
Animals ; Antigens, CD/metabolism ; Cell Movement ; Cells, Cultured ; Chemokine CCL8/metabolism ; Disease Models, Animal ; Female ; Integrin alpha Chains/metabolism ; Lymphocyte Activation ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Receptors, CCR8/genetics
Czasopismo naukowe
Tytuł :
Targeting CCR8 Induces Protective Antitumor Immunity and Enhances Vaccine-Induced Responses in Colon Cancer.
Autorzy :
Villarreal DO; Advaxis Immunotherapies, Princeton, New Jersey. .
L'Huillier A; Advaxis Immunotherapies, Princeton, New Jersey.
Armington S; Advaxis Immunotherapies, Princeton, New Jersey.
Mottershead C; Advaxis Immunotherapies, Princeton, New Jersey.
Filippova EV; Advaxis Immunotherapies, Princeton, New Jersey.
Coder BD; Advaxis Immunotherapies, Princeton, New Jersey.
Petit RG; Advaxis Immunotherapies, Princeton, New Jersey.
Princiotta MF; Advaxis Immunotherapies, Princeton, New Jersey.
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Źródło :
Cancer research [Cancer Res] 2018 Sep 15; Vol. 78 (18), pp. 5340-5348. Date of Electronic Publication: 2018 Jul 19.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cancer Vaccines/*immunology
Colonic Neoplasms/*immunology
Colonic Neoplasms/*therapy
Receptors, CCR8/*antagonists & inhibitors
Animals ; Antibodies, Monoclonal/immunology ; CD8-Positive T-Lymphocytes/immunology ; Cell Line, Tumor ; Humans ; Immune Tolerance ; Immunosuppression ; Immunotherapy ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Receptors, CCR8/immunology ; T-Lymphocytes, Regulatory/immunology ; Treatment Outcome ; Tumor Microenvironment/immunology ; Up-Regulation
Czasopismo naukowe
Tytuł :
CCR8 is expressed by post-positive selection CD4-lineage thymocytes but is dispensable for central tolerance induction.
Autorzy :
Thyagarajan HM; Department of Molecular Biosciences, Institute for Cellular and Molecular Biology, The University of Texas at Austin, Austin, Texas, United States of America.
Lancaster JN; Department of Molecular Biosciences, Institute for Cellular and Molecular Biology, The University of Texas at Austin, Austin, Texas, United States of America.
Lira SA; Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, New York, United States of America.
Ehrlich LIR; Department of Molecular Biosciences, Institute for Cellular and Molecular Biology, The University of Texas at Austin, Austin, Texas, United States of America.; Livestrong Cancer Institutes, Dell Medical School, The University of Texas at Austin, Austin, Texas, United States of America.
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Źródło :
PloS one [PLoS One] 2018 Jul 19; Vol. 13 (7), pp. e0200765. Date of Electronic Publication: 2018 Jul 19 (Print Publication: 2018).
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
CD4 Antigens/*metabolism
Receptors, CCR8/*metabolism
Thymocytes/*metabolism
Animals ; Antigen-Presenting Cells/metabolism ; Flow Cytometry ; Fluorescent Antibody Technique ; Mice ; Mice, Inbred C57BL ; Receptors, CCR7/genetics ; Receptors, CCR7/metabolism ; Receptors, CCR8/genetics
Czasopismo naukowe
Tytuł :
The Systemic Administration of the Chemokine CCL1 Evokes Thermal Analgesia in Mice Through the Activation of the Endocannabinoid System.
Autorzy :
García-Domínguez M; Laboratorio de Farmacología, Facultad de Medicina, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), Universidad de Oviedo, C/Julián Clavería 6, 33006, Oviedo, Asturias, Spain.
Aguirre A; Laboratorio de Farmacología, Facultad de Medicina, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), Universidad de Oviedo, C/Julián Clavería 6, 33006, Oviedo, Asturias, Spain.
Lastra A; Laboratorio de Farmacología, Facultad de Medicina, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), Universidad de Oviedo, C/Julián Clavería 6, 33006, Oviedo, Asturias, Spain.
Hidalgo A; Laboratorio de Farmacología, Facultad de Medicina, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), Universidad de Oviedo, C/Julián Clavería 6, 33006, Oviedo, Asturias, Spain.
Baamonde A; Laboratorio de Farmacología, Facultad de Medicina, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), Universidad de Oviedo, C/Julián Clavería 6, 33006, Oviedo, Asturias, Spain.
Menéndez L; Laboratorio de Farmacología, Facultad de Medicina, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), Universidad de Oviedo, C/Julián Clavería 6, 33006, Oviedo, Asturias, Spain. .
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Źródło :
Cellular and molecular neurobiology [Cell Mol Neurobiol] 2019 Nov; Vol. 39 (8), pp. 1115-1124. Date of Electronic Publication: 2019 Jun 15.
Typ publikacji :
Journal Article
MeSH Terms :
Analgesia*
Temperature*
Chemokine CCL1/*administration & dosage
Endocannabinoids/*metabolism
Analgesics/pharmacology ; Animals ; Arachidonic Acids/metabolism ; Cyclophosphamide ; Glycerides/metabolism ; Leukocytes/drug effects ; Leukocytes/metabolism ; Lumbar Vertebrae/drug effects ; Lumbar Vertebrae/metabolism ; Male ; Mice ; Models, Biological ; Neurons/drug effects ; Neurons/metabolism ; Proto-Oncogene Proteins c-fos/metabolism ; Receptor, Cannabinoid, CB1/metabolism ; Receptor, Cannabinoid, CB2/metabolism ; Receptors, CCR8/metabolism
Czasopismo naukowe
Tytuł :
Disruption of the CCL1-CCR8 axis inhibits vascular Treg recruitment and function and promotes atherosclerosis in mice.
Autorzy :
Vila-Caballer M; Instituto de Biomedicina de Valencia (IBV-CSIC), Valencia, Spain; Universidad Cardenal Herrera-CEU (CEU Universities), Valencia, Spain.
González-Granado JM; Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain; CIBER de Enfermedades Cardiovasculares (CIBER-CV), Spain; LamImSys Laboratory, Instituto de Investigación Sanitaria Hospital 12 de Octubre (imas12), Madrid, Spain; Departamento de Fisiología, Facultad de Medicina, Universidad Autónoma de Madrid (UAM), Madrid, Spain.
Zorita V; Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.
Abu Nabah YN; Instituto de Biomedicina de Valencia (IBV-CSIC), Valencia, Spain.
Silvestre-Roig C; Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain; Institute for Cardiovascular Prevention (IPEK), Ludwig Maximilian University, Munich, Germany.
Del Monte-Monge A; Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain; CIBER de Enfermedades Cardiovasculares (CIBER-CV), Spain.
Molina-Sánchez P; Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.
Ait-Oufella H; Institut National de la Santé et de la Recherche Médicale (INSERM), Paris Cardiovascular Research Center, Paris, France.
Andrés-Manzano MJ; Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain; CIBER de Enfermedades Cardiovasculares (CIBER-CV), Spain.
Sanz MJ; Departamento de Farmacología, Universidad de Valencia and Instituto de Investigación Sanitaria-INCLIVA, Valencia, Spain.
Weber C; Institute for Cardiovascular Prevention (IPEK), Ludwig Maximilian University, Munich, Germany.
Kremer L; Departamento de Inmunología y Oncología, Centro Nacional de Biotecnología-CSIC, Madrid, Spain.
Gutiérrez J; Departamento de Inmunología y Oncología, Centro Nacional de Biotecnología-CSIC, Madrid, Spain.
Mallat Z; Institut National de la Santé et de la Recherche Médicale (INSERM), Paris Cardiovascular Research Center, Paris, France; Division of Cardiovascular Medicine, University of Cambridge, Cambridge, UK.
Andrés V; Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain; CIBER de Enfermedades Cardiovasculares (CIBER-CV), Spain. Electronic address: .
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Źródło :
Journal of molecular and cellular cardiology [J Mol Cell Cardiol] 2019 Jul; Vol. 132, pp. 154-163. Date of Electronic Publication: 2019 May 21.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Atherosclerosis/*immunology
Chemokine CCL1/*immunology
Receptors, CCR8/*immunology
T-Lymphocytes, Regulatory/*immunology
Animals ; Apolipoproteins E/immunology ; Cytokines/immunology ; Inflammation/immunology ; Interleukin-10/immunology ; Leukocytes/immunology ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Th1 Cells/immunology ; Th2 Cells/immunology
Czasopismo naukowe
Tytuł :
Role of Conserved Disulfide Bridges and Aromatic Residues in Extracellular Loop 2 of Chemokine Receptor CCR8 for Chemokine and Small Molecule Binding.
Autorzy :
Barington L; From the Department of Neuroscience and Pharmacology.
Rummel PC; From the Department of Neuroscience and Pharmacology.
Lückmann M; From the Department of Neuroscience and Pharmacology, the Novo Nordisk Foundation Center for Basic Metabolic Research, and.
Pihl H; From the Department of Neuroscience and Pharmacology.
Larsen O; From the Department of Neuroscience and Pharmacology.
Daugvilaite V; From the Department of Neuroscience and Pharmacology.
Johnsen AH; the Department of Clinical Biochemistry, Rigshospitalet, DK-2100 Copenhagen, Denmark.
Frimurer TM; the Novo Nordisk Foundation Center for Basic Metabolic Research, and the Novo Nordisk Foundation Center for Protein Research, University of Copenhagen, DK-2200 Copenhagen, Denmark and.
Karlshøj S; From the Department of Neuroscience and Pharmacology.
Rosenkilde MM; From the Department of Neuroscience and Pharmacology, .
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Źródło :
The Journal of biological chemistry [J Biol Chem] 2016 Jul 29; Vol. 291 (31), pp. 16208-20. Date of Electronic Publication: 2016 May 19.
Typ publikacji :
Journal Article
MeSH Terms :
Chemokine CCL1/*chemistry
Chemokines, CC/*chemistry
Disulfides/*chemistry
Inositol 1,4,5-Trisphosphate/*chemistry
Receptors, CCR8/*chemistry
Viral Proteins/*chemistry
Animals ; COS Cells ; Chemokine CCL1/metabolism ; Chemokines, CC/metabolism ; Chlorocebus aethiops ; Disulfides/metabolism ; Humans ; Inositol 1,4,5-Trisphosphate/metabolism ; Protein Binding ; Protein Domains ; Protein Structure, Secondary ; Receptors, CCR8/genetics ; Receptors, CCR8/metabolism ; Viral Proteins/metabolism
Czasopismo naukowe
Tytuł :
CCR8 T reg cells as master drivers of immune regulation.
Autorzy :
Barsheshet Y; Department of Immunology, Technion, Haifa 31096, Israel.
Wildbaum G; Department of Immunology, Technion, Haifa 31096, Israel.
Levy E; Department of Immunology, Technion, Haifa 31096, Israel.
Vitenshtein A; Department of Immunology, Technion, Haifa 31096, Israel.
Akinseye C; GlaxoSmithKline Medicines Research Centre, Stevenage, Hertfordshire, SG1 2NY, United Kingdom.
Griggs J; GlaxoSmithKline Medicines Research Centre, Stevenage, Hertfordshire, SG1 2NY, United Kingdom.
Lira SA; Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY 10029.
Karin N; Department of Immunology, Technion, Haifa 31096, Israel; .; Rappaport Family Institute for Research in the Medical Sciences and Bruce Rappaport Faculty of Medicine, Technion, Haifa 31096, Israel.
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Źródło :
Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2017 Jun 06; Vol. 114 (23), pp. 6086-6091. Date of Electronic Publication: 2017 May 22.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Forkhead Transcription Factors/*immunology
Receptors, CCR8/*immunology
T-Lymphocytes, Regulatory/*immunology
Animals ; Chemokine CCL1/immunology ; Chemokine CCL1/metabolism ; Female ; Forkhead Transcription Factors/metabolism ; Humans ; Mice ; Mice, Inbred C57BL ; Receptors, CCR8/metabolism ; T-Lymphocytes, Regulatory/metabolism ; Up-Regulation
Czasopismo naukowe
Tytuł :
CC chemokine CCL1 receptor CCR8 mediates conversion of mesenchymal stem cells to embryoid bodies expressing FOXP3+CCR8+ regulatory T cells.
Autorzy :
Haque NS; Department of Pathology, New York Medical College, Valhalla, NY, United States of America.; Department of Biological Sciences, New York City College of Technology, City University of New York, Brooklyn, NY, United States of America.
Tuteja A; Department of Pathology, New York Medical College, Valhalla, NY, United States of America.
Haque N; Department of Biological Sciences, New York City College of Technology, City University of New York, Brooklyn, NY, United States of America.
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Źródło :
PloS one [PLoS One] 2019 Jul 17; Vol. 14 (7), pp. e0218944. Date of Electronic Publication: 2019 Jul 17 (Print Publication: 2019).
Typ publikacji :
Journal Article
MeSH Terms :
Chemokine CCL1/*genetics
Embryoid Bodies/*metabolism
Forkhead Transcription Factors/*genetics
Receptors, CCR8/*genetics
Animals ; Chemotaxis/genetics ; Forkhead Transcription Factors/metabolism ; Gene Expression Regulation, Developmental ; Humans ; Mesenchymal Stem Cells/metabolism ; Mice ; Rats ; T-Lymphocytes, Regulatory/metabolism ; Th2 Cells
Czasopismo naukowe
Tytuł :
CCR8 regulates contact hypersensitivity by restricting cutaneous dendritic cell migration to the draining lymph nodes.
Autorzy :
Yabe R; Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba 278-0022, Japan Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan Medical Mycology Research Center, Chiba University, Inohana Chuo-ku, Chiba 260-8673, Japan.
Shimizu K; Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba 278-0022, Japan Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan.
Shimizu S; Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan.
Azechi S; Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan.
Choi BI; Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan.
Sudo K; Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan.
Kubo S; Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba 278-0022, Japan Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan.
Nakae S; Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan.
Ishigame H; Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan.
Kakuta S; Department of Biomedical Science, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo 113-8657, Japan.
Iwakura Y; Center for Animal Disease Models, Research Institute for Biomedical Sciences (RIBS), Tokyo University of Science, Noda, Chiba 278-0022, Japan Center for Experimental Medicine and Systems Biology, The Institute of Medical Science, University of Tokyo (IMSUT), Minato-ku, Tokyo 108-8639, Japan Medical Mycology Research Center, Chiba University, Inohana Chuo-ku, Chiba 260-8673, Japan Core Research for Evolutional and Technology (CREST), Japan Science and Technology Agency, Kawaguchi, Saitama 332-0012, Japan .
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Źródło :
International immunology [Int Immunol] 2015 Apr; Vol. 27 (4), pp. 169-81. Date of Electronic Publication: 2014 Oct 25.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement/*immunology
Dermatitis, Contact/*immunology
Langerhans Cells/*immunology
Lymph Nodes/*cytology
Receptors, CCR8/*immunology
Adoptive Transfer ; Animals ; Cell Proliferation ; Dermatitis, Contact/genetics ; Dinitrofluorobenzene ; Inflammation/immunology ; Lymph Nodes/immunology ; Mice ; Mice, Inbred BALB C ; Mice, Knockout ; Mice, SCID ; Receptors, CCR8/biosynthesis ; Receptors, CCR8/genetics ; T-Lymphocytes/immunology ; T-Lymphocytes/transplantation ; Th1 Cells/immunology ; Th17 Cells/immunology
Czasopismo naukowe
Tytuł :
In silico characterization of binding mode of CCR8 inhibitor: homology modeling, docking and membrane based MD simulation study.
Autorzy :
Gadhe CG; a Department of Life Sciences, College of BioNano Technology , Gachon University , 1342 Seongnamdaero, Sujeong-gu, Seongnam-si , Gyeonggi-do 461-701 , Republic of Korea.
Balupuri A; b Department of Bio-New Drug Development, College of Medicine , Chosun University , Gwangju 501-759 , Republic of Korea.
Cho SJ; b Department of Bio-New Drug Development, College of Medicine , Chosun University , Gwangju 501-759 , Republic of Korea.; c Department of Cellular Molecular Medicine, College of Medicine , Chosun University , Gwangju 501-759 , Republic of Korea.
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Źródło :
Journal of biomolecular structure & dynamics [J Biomol Struct Dyn] 2015; Vol. 33 (11), pp. 2491-510. Date of Electronic Publication: 2015 Jan 23.
Typ publikacji :
Journal Article
MeSH Terms :
Drug Discovery*
Ligands*
Models, Molecular*
Receptors, CCR8/*chemistry
Amino Acid Sequence ; Binding Sites ; Catalytic Domain ; Humans ; Hydrophobic and Hydrophilic Interactions ; Molecular Docking Simulation ; Molecular Dynamics Simulation ; Molecular Sequence Data ; Protein Binding ; Protein Conformation ; Protein Structure, Secondary ; Quantitative Structure-Activity Relationship ; Receptors, CCR8/antagonists & inhibitors ; Reproducibility of Results ; Sequence Alignment
Czasopismo naukowe
Tytuł :
Changes in Chemokines and Chemokine Receptors Expression in a Mouse Model of Alzheimer's Disease.
Autorzy :
Jorda A; Department of Physiology, School of Medicine, University of Valencia. Spain.
Cauli O; Faculty of Surgery and Chiropody, University of Valencia. Spain.
Santonja JM; Clinic Hospital of Valencia. Spain.
Aldasoro M; Department of Physiology, School of Medicine, University of Valencia. Spain.
Aldasoro C; Department of Physiology, School of Medicine, University of Valencia. Spain.
Obrador E; Department of Physiology, School of Medicine, University of Valencia. Spain.
Vila JM; Department of Physiology, School of Medicine, University of Valencia. Spain.
Mauricio MD; Department of Physiology, School of Medicine, University of Valencia. Spain.
Iradi A; Department of Physiology, School of Medicine, University of Valencia. Spain.
Guerra-Ojeda S; Department of Physiology, School of Medicine, University of Valencia. Spain.
Marchio P; Department of Physiology, School of Medicine, University of Valencia. Spain.
Valles SL; Department of Physiology, School of Medicine, University of Valencia. Spain.
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Źródło :
International journal of biological sciences [Int J Biol Sci] 2019 Jan 01; Vol. 15 (2), pp. 453-463. Date of Electronic Publication: 2019 Jan 01 (Print Publication: 2019).
Typ publikacji :
Journal Article
MeSH Terms :
Alzheimer Disease/*metabolism
Chemokines/*metabolism
Receptors, Chemokine/*metabolism
Animals ; Chemokine CCL3/metabolism ; Chemokine CCL4/metabolism ; Chemotaxis/physiology ; Glial Fibrillary Acidic Protein/genetics ; Glial Fibrillary Acidic Protein/metabolism ; Hand Strength/physiology ; Inflammation/metabolism ; Mice ; Receptors, CCR8/metabolism
Czasopismo naukowe
Tytuł :
Neuropathic pain inhibitor, RAP-103, is a potent inhibitor of microglial CCL1/CCR8.
Autorzy :
Noda M; Laboratory of Pathophysiology, Graduate School of Pharmaceutical Sciences, Kyushu University, 3-1-1 Maidashi, Higashi-ku, Fukuoka, 812-8582, Japan. Electronic address: .
Tomonaga D; Laboratory of Pathophysiology, Graduate School of Pharmaceutical Sciences, Kyushu University, 3-1-1 Maidashi, Higashi-ku, Fukuoka, 812-8582, Japan.
Kitazono K; Laboratory of Pathophysiology, Graduate School of Pharmaceutical Sciences, Kyushu University, 3-1-1 Maidashi, Higashi-ku, Fukuoka, 812-8582, Japan.
Yoshioka Y; Laboratory of Pathophysiology, Graduate School of Pharmaceutical Sciences, Kyushu University, 3-1-1 Maidashi, Higashi-ku, Fukuoka, 812-8582, Japan.
Liu J; Laboratory of Pathophysiology, Graduate School of Pharmaceutical Sciences, Kyushu University, 3-1-1 Maidashi, Higashi-ku, Fukuoka, 812-8582, Japan.
Rousseau JP; Department of Pediatrics, Université Laval, Centre de Recherche de l'Institut Universitaire de Cardiologie et Pneumologie de Québec, 2725 Chemin Ste-Foy, Québec, QC, G1V 4G5, Canada.
Kinkead R; Department of Pediatrics, Université Laval, Centre de Recherche de l'Institut Universitaire de Cardiologie et Pneumologie de Québec, 2725 Chemin Ste-Foy, Québec, QC, G1V 4G5, Canada.
Ruff MR; Creative Bio-Peptides, USA, Rockville, MD, 20854, USA.
Pert CB; Creative Bio-Peptides, USA, Rockville, MD, 20854, USA.
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Źródło :
Neurochemistry international [Neurochem Int] 2018 Oct; Vol. 119, pp. 184-189. Date of Electronic Publication: 2017 Dec 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Chemokine CCL1/*drug effects
Microglia/*drug effects
Neuralgia/*drug therapy
Peptides/*pharmacology
Receptors, CCR8/*drug effects
Animals ; Chemotaxis/drug effects ; Hyperalgesia/drug therapy ; Mice ; Sciatic Nerve/drug effects ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł :
Chemokine receptor CCR8 is required for lipopolysaccharide-triggered cytokine production in mouse peritoneal macrophages.
Autorzy :
Oshio T; Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Ichikawa, Chiba, Japan; Department of Dermatology, Jichi Medical University, Shimotsuke, Tochigi, Japan.
Kawashima R; Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Ichikawa, Chiba, Japan; Kitasato University Graduate School of Medical Sciences, Sagamihara, Kanagawa, Japan.
Kawamura YI; Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Ichikawa, Chiba, Japan.
Hagiwara T; Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Ichikawa, Chiba, Japan.
Mizutani N; Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Ichikawa, Chiba, Japan.
Okada T; Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Ichikawa, Chiba, Japan.
Otsubo T; Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Ichikawa, Chiba, Japan.
Inagaki-Ohara K; Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Ichikawa, Chiba, Japan.
Matsukawa A; Department of Pathology & Experimental Medicine, Okayama University, Okayama, Okayama, Japan.
Haga T; Department of Life Science, Gakushuin University, Mejiro-ku, Tokyo, Japan.
Kakuta S; Department of Biomedical Science, Graduate School of Agricultural and Life Sciences, The University of Tokyo, Bunkyo-ku, Tokyo, Japan.
Iwakura Y; Division of Experimental Animal Immunology, Research Institute for Biomedical Sciences, Tokyo University of Science, Noda, Chiba, Japan.
Hosokawa S; Department of Applied Chemistry, Faculty of Advanced Science and Engineering, Waseda University, Shinjuku-ku, Tokyo, Japan.
Dohi T; Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Ichikawa, Chiba, Japan.
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Źródło :
PloS one [PLoS One] 2014 Apr 08; Vol. 9 (4), pp. e94445. Date of Electronic Publication: 2014 Apr 08 (Print Publication: 2014).
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cytokines/*biosynthesis
Macrophages, Peritoneal/*metabolism
Receptors, CCR8/*metabolism
Animals ; Chemokine CCL1/antagonists & inhibitors ; Chemotaxis/drug effects ; Chemotaxis/immunology ; Colitis/genetics ; Colitis/immunology ; Colitis/metabolism ; Disease Models, Animal ; Inflammation/genetics ; Inflammation/immunology ; Inflammation/metabolism ; Intracellular Space/metabolism ; Lipopolysaccharides/immunology ; Macrophages/drug effects ; Macrophages/immunology ; Macrophages/metabolism ; Macrophages, Peritoneal/drug effects ; Macrophages, Peritoneal/immunology ; Male ; Mice ; Mice, Knockout ; Protein Transport ; Receptors, CCR8/antagonists & inhibitors ; Receptors, CCR8/deficiency ; Receptors, CCR8/genetics ; Signal Transduction/drug effects ; Toll-Like Receptors/metabolism
Czasopismo naukowe

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