Temat: "CONNEXIN 43" - OpacWWW

Skocz do pozycji: 1 1.

Tytuł :: Staphylococcus aureus impairs cutaneous wound healing by activating the expression of a gap junction protein, connexin-43 in keratinocytes.
Autorzy :: Xu W; Department of Life Sciences, College of Science and Engineering, Texas A&M University-Corpus Christi, Corpus Christi, TX, 78412, USA. .
Dielubanza E; Laboratory for Wound Repair and Regenerative Surgery, Department of Surgery, Northwestern University Feinberg School of Medicine, Chicago, IL, 60611, USA.
Maisel A; Laboratory for Wound Repair and Regenerative Surgery, Department of Surgery, Northwestern University Feinberg School of Medicine, Chicago, IL, 60611, USA.
Leung K; Division of Combat Wound Repair, US Army Institute of Surgical Research, JB Fort Sam Houston, San Antonio, TX, 78234, USA.
Mustoe T; Laboratory for Wound Repair and Regenerative Surgery, Department of Surgery, Northwestern University Feinberg School of Medicine, Chicago, IL, 60611, USA.
Hong S; Laboratory for Wound Repair and Regenerative Surgery, Department of Surgery, Northwestern University Feinberg School of Medicine, Chicago, IL, 60611, USA. .
Galiano R; Laboratory for Wound Repair and Regenerative Surgery, Department of Surgery, Northwestern University Feinberg School of Medicine, Chicago, IL, 60611, USA. .; Pokaż więcej
Źródło :: Cellular and molecular life sciences : CMLS [Cell Mol Life Sci] 2021 Feb; Vol. 78 (3), pp. 935-947. Date of Electronic Publication: 2020 May 14.
Typ publikacji :: Journal Article
MeSH Terms :: Connexin 43/*metabolism
Skin Diseases, Bacterial/*pathology
Staphylococcus aureus/*pathogenicity
Wound Healing/*physiology
Animals ; Cell Line ; Cell Movement/drug effects ; Connexin 43/antagonists & inhibitors ; Connexin 43/genetics ; Disease Models, Animal ; Humans ; Keratinocytes/cytology ; Keratinocytes/metabolism ; Mice ; Mitogen-Activated Protein Kinase 1/metabolism ; Mitogen-Activated Protein Kinase 3/metabolism ; Phosphatidylinositol 3-Kinases/chemistry ; Phosphatidylinositol 3-Kinases/metabolism ; Phosphoinositide-3 Kinase Inhibitors/pharmacology ; Phosphorylation ; Proto-Oncogene Proteins c-akt/antagonists & inhibitors ; Proto-Oncogene Proteins c-akt/metabolism ; RNA Interference ; RNA, Small Interfering/metabolism ; Skin Diseases, Bacterial/metabolism ; Skin Diseases, Bacterial/microbiology ; Staphylococcus aureus/isolation & purification ; Up-Regulation
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 2 2.

Tytuł :: Pulpal upregulation of connexin 43 during pulpitis.
Autorzy :: Lim WY; National Dental Research Institute Singapore, National Dental Centre Singapore, Singapore, Singapore. .; Endodontic Unit, Restorative Department, National Dental Centre Singapore, 5 Second Hospital Ave, Singapore, 168938, Singapore. .; Lee Kong Chian School of Medicine, Nanyang Technological University, Novena Campus, Headquarters & Clinical Sciences Building, 11 Mandalay Road, Singapore, 308232, Singapore. .; Department of Restorative Dentistry, Endodontic Unit, National Dental Centre of Singapore, 5 Second Hospital Ave, Singapore, 168938, Singapore. .
Madden LE; Lee Kong Chian School of Medicine, Nanyang Technological University, Novena Campus, Headquarters & Clinical Sciences Building, 11 Mandalay Road, Singapore, 308232, Singapore.; Skin Research Institute Singapore, Agency for Science, Technology and Research, Singapore, Singapore.
Becker DL; Lee Kong Chian School of Medicine, Nanyang Technological University, Novena Campus, Headquarters & Clinical Sciences Building, 11 Mandalay Road, Singapore, 308232, Singapore.; Skin Research Institute Singapore, Agency for Science, Technology and Research, Singapore, Singapore.; Pokaż więcej
Źródło :: Clinical oral investigations [Clin Oral Investig] 2021 Mar; Vol. 25 (3), pp. 1327-1335. Date of Electronic Publication: 2020 Jul 04.
Typ publikacji :: Journal Article
MeSH Terms :: Connexin 43*/metabolism
Pulpitis*
Animals ; Dental Pulp/metabolism ; Rats ; Rats, Sprague-Dawley ; Up-Regulation
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 3 3.

Tytuł :: Role of connexin 43 in odontoblastic differentiation and structural maintenance in pulp damage repair.
Autorzy :: Yin J; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases & West China Hospital of Stomatology, Sichuan University, Chengdu, China.; Department of Endodontics, School of Stomatology, Capital Medical University, Beijing, China.
Xu J; West China School of Public Health and West China Fourth Hospital, Sichuan University, Chengdu, China.
Cheng R; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases & West China Hospital of Stomatology, Sichuan University, Chengdu, China.
Shao M; West China School of Public Health and West China Fourth Hospital, Sichuan University, Chengdu, China.
Qin Y; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases & West China Hospital of Stomatology, Sichuan University, Chengdu, China.
Yang H; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases & West China Hospital of Stomatology, Sichuan University, Chengdu, China. .
Hu T; State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases & West China Hospital of Stomatology, Sichuan University, Chengdu, China. .; Pokaż więcej
Źródło :: International journal of oral science [Int J Oral Sci] 2021 Jan 08; Vol. 13 (1), pp. 1. Date of Electronic Publication: 2021 Jan 08.
Typ publikacji :: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :: Connexin 43*
Dental Pulp*
Animals ; Cell Differentiation ; Extracellular Matrix Proteins ; Odontoblasts ; Phosphoproteins ; Rats ; Rats, Sprague-Dawley
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 4 4.

Tytuł :: Long Noncoding RNA HOTAIR Functions as a Competitive Endogenous RNA to Regulate Connexin43 Remodeling in Atrial Fibrillation by Sponging MicroRNA-613.
Autorzy :: Dai W; Department of Cardiology, The First Affiliated Hospital of Guangxi Medical University, Guangxi Cardiovascular Institute, Nanning, Guangxi, China.
Chao X; Department of Cardiology, The First Affiliated Hospital of Guangxi Medical University, Guangxi Cardiovascular Institute, Nanning, Guangxi, China.
Li S; Department of Cardiology, The First Affiliated Hospital of Guangxi Medical University, Guangxi Cardiovascular Institute, Nanning, Guangxi, China.
Zhou S; Department of Cardiology, The First Affiliated Hospital of Guangxi Medical University, Guangxi Cardiovascular Institute, Nanning, Guangxi, China.
Zhong G; Department of Cardiology, The First Affiliated Hospital of Guangxi Medical University, Guangxi Cardiovascular Institute, Nanning, Guangxi, China.
Jiang Z; Hypertension Division, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi 530021, China.; Pokaż więcej
Źródło :: Cardiovascular therapeutics [Cardiovasc Ther] 2020 Nov 17; Vol. 2020, pp. 5925342. Date of Electronic Publication: 2020 Nov 17 (Print Publication: 2020).
Typ publikacji :: Journal Article
MeSH Terms :: Atrial Remodeling*
Atrial Appendage/*metabolism
Atrial Fibrillation/*metabolism
Connexin 43/*metabolism
MicroRNAs/*metabolism
Myocytes, Cardiac/*metabolism
RNA, Long Noncoding/*metabolism
Adult ; Aged ; Animals ; Atrial Appendage/physiopathology ; Atrial Fibrillation/genetics ; Atrial Fibrillation/physiopathology ; Cell Line ; Connexin 43/genetics ; Female ; Gene Expression Regulation ; Heart Rate ; Humans ; Male ; Mice ; MicroRNAs/genetics ; Middle Aged ; RNA, Long Noncoding/genetics ; Signal Transduction
: Czasopismo naukowe

Szczegóły Pełny tekst Pełny tekst

Skocz do pozycji: 5 5.

Tytuł :: [Butorphanol alleviates ischemic arrhythmia in SD rats by up-regulating connexin 43 (Cx43) pathway through miR-1-3p].
Autorzy :: Wang M; Department of Anesthesiology, General Hospital of Ningxia Medical University, Yinchuan 750004, China.
Ma H; Department of Anesthesiology, Cancer Hospital, General Hospital of Ningxia Medical University, Yinchuan 750004, China.
Gao Y; Department of Anesthesiology, General Hospital of Ningxia Medical University, Yinchuan 750004, China.
Zhao N; Department of Anesthesiology, General Hospital of Ningxia Medical University, Yinchuan 750004, China.
Zhang D; Department of Anesthesiology, General Hospital of Ningxia Medical University, Yinchuan 750004, China. *Corresponding author, E-mail: .; Pokaż więcej
Źródło :: Xi bao yu fen zi mian yi xue za zhi = Chinese journal of cellular and molecular immunology [Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi] 2020 Nov; Vol. 36 (11), pp. 990-995.
Typ publikacji :: Journal Article
MeSH Terms :: Arrhythmias, Cardiac*/drug therapy
Arrhythmias, Cardiac*/genetics
Butorphanol*/pharmacology
Connexin 43*/genetics
MicroRNAs*/genetics
Up-Regulation*/drug effects
Animals ; Narcotic Antagonists/pharmacology ; Rats ; Rats, Sprague-Dawley
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 6 6.

Tytuł :: TGF-β1-induced connexin43 promotes scar formation via the Erk/MMP-1/collagen III pathway.
Autorzy :: Li X; Laboratory of Tissue Regeneration and Immunology and Department of Periodontics, Beijing Key Laboratory of Tooth Regeneration and Function Reconstruction, School of Stomatology, Capital Medical University, Beijing, China.
Guo L; Department of Orthodontics, School of Stomatology, Capital Medical University, Beijing, China.
Yang X; Laboratory of Tissue Regeneration and Immunology and Department of Periodontics, Beijing Key Laboratory of Tooth Regeneration and Function Reconstruction, School of Stomatology, Capital Medical University, Beijing, China.
Wang J; School of Dental Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, USA.
Hou Y; Department of Orthodontics the Third Dental Center, Peking University School of Stomatology, Beijing, China.
Zhu S; Laboratory of Tissue Regeneration and Immunology and Department of Periodontics, Beijing Key Laboratory of Tooth Regeneration and Function Reconstruction, School of Stomatology, Capital Medical University, Beijing, China.
Du J; Laboratory of Tissue Regeneration and Immunology and Department of Periodontics, Beijing Key Laboratory of Tooth Regeneration and Function Reconstruction, School of Stomatology, Capital Medical University, Beijing, China.
Feng J; Department of General Dentistry, School of Stomatology, Capital Medical University, Beijing, China.
Xie Y; Department of General Dentistry, School of Stomatology, Capital Medical University, Beijing, China.
Zhuang L; Department of Orthodontics, School of Stomatology, Capital Medical University, Beijing, China.
He X; Department of Orthodontics, School of Stomatology, Capital Medical University, Beijing, China.
Liu Y; Laboratory of Tissue Regeneration and Immunology and Department of Periodontics, Beijing Key Laboratory of Tooth Regeneration and Function Reconstruction, School of Stomatology, Capital Medical University, Beijing, China.; Pokaż więcej
Źródło :: Journal of oral rehabilitation [J Oral Rehabil] 2020 Nov; Vol. 47 Suppl 1, pp. 99-106. Date of Electronic Publication: 2019 Oct 31.
Typ publikacji :: Journal Article
MeSH Terms :: Connexin 43*
Transforming Growth Factor beta1*
Animals ; Cicatrix ; Collagen ; Humans ; Matrix Metalloproteinase 1/genetics ; Mice ; Mice, Inbred C57BL
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 7 7.

Tytuł :: Hypoxic Melanoma Cells Deliver microRNAs to Dendritic Cells and Cytotoxic T Lymphocytes through Connexin-43 Channels.
Autorzy :: Tittarelli A; Programa Institucional de Fomento a la Investigación, Desarrollo e Innovación (PIDi), Universidad Tecnológica Metropolitana (UTEM), 8940577 Santiago, Chile.
Navarrete M; Disciplinary Program of Immunology, Institute of Biomedical Sciences, Faculty of Medicine, Universidad de Chile, 8380453 Santiago, Chile.; Millennium Institute on Immunology and Immunotherapy, Faculty of Medicine, Universidad de Chile, 8380453 Santiago, Chile.
Lizana M; Disciplinary Program of Immunology, Institute of Biomedical Sciences, Faculty of Medicine, Universidad de Chile, 8380453 Santiago, Chile.
Hofmann-Vega F; Disciplinary Program of Immunology, Institute of Biomedical Sciences, Faculty of Medicine, Universidad de Chile, 8380453 Santiago, Chile.
Salazar-Onfray F; Disciplinary Program of Immunology, Institute of Biomedical Sciences, Faculty of Medicine, Universidad de Chile, 8380453 Santiago, Chile.; Millennium Institute on Immunology and Immunotherapy, Faculty of Medicine, Universidad de Chile, 8380453 Santiago, Chile.; Pokaż więcej
Źródło :: International journal of molecular sciences [Int J Mol Sci] 2020 Oct 13; Vol. 21 (20). Date of Electronic Publication: 2020 Oct 13.
Typ publikacji :: Journal Article
MeSH Terms :: Tumor Hypoxia*
Connexin 43/*metabolism
Dendritic Cells/*metabolism
Melanoma/*metabolism
MicroRNAs/*metabolism
T-Lymphocytes, Cytotoxic/*metabolism
Animals ; Cell Line, Tumor ; Connexin 43/genetics ; Gap Junctions/metabolism ; Humans ; Mice ; Mice, Inbred C57BL ; MicroRNAs/genetics
: Czasopismo naukowe

Szczegóły Pełny tekst Pełny tekst

Skocz do pozycji: 8 8.

Tytuł :: Gap19, a Cx43 Hemichannel Inhibitor, Acts as a Gating Modifier That Decreases Main State Opening While Increasing Substate Gating.
Autorzy :: Lissoni A; Department of Basic and Applied Medical Sciences-Physiology Group, Ghent University, 9000 Ghent, Belgium.
Wang N; Department of Basic and Applied Medical Sciences-Physiology Group, Ghent University, 9000 Ghent, Belgium.
Nezlobinskii T; Department of Physics and Astronomy, Ghent University, 9000 Ghent, Belgium.
De Smet M; Department of Basic and Applied Medical Sciences-Physiology Group, Ghent University, 9000 Ghent, Belgium.
Panfilov AV; Department of Physics and Astronomy, Ghent University, 9000 Ghent, Belgium.; Laboratory of Computational Biology and Medicine, Ural Federal University, 620075 Ekaterinburg, Russia.
Vandersickel N; Department of Physics and Astronomy, Ghent University, 9000 Ghent, Belgium.
Leybaert L; Department of Basic and Applied Medical Sciences-Physiology Group, Ghent University, 9000 Ghent, Belgium.
Witschas K; Department of Basic and Applied Medical Sciences-Physiology Group, Ghent University, 9000 Ghent, Belgium.; Pokaż więcej
Źródło :: International journal of molecular sciences [Int J Mol Sci] 2020 Oct 05; Vol. 21 (19). Date of Electronic Publication: 2020 Oct 05.
Typ publikacji :: Journal Article
MeSH Terms :: Software*
Connexin 43/*chemistry
Green Fluorescent Proteins/*chemistry
Ion Channel Gating/*genetics
Connexin 43/antagonists & inhibitors ; Gap Junctions ; Green Fluorescent Proteins/genetics ; HeLa Cells ; Humans ; Kinetics ; Peptides/chemistry
: Czasopismo naukowe

Szczegóły Pełny tekst Pełny tekst

Skocz do pozycji: 9 9.

Tytuł :: Inhibition of MicroRNA-206 Ameliorates Ischemia-Reperfusion Arrhythmia in a Mouse Model by Targeting Connexin43.
Autorzy :: Jin Y; Department of Cardiology, The Affiliated Wuxi No.2 People's Hospital of Nanjing Medical University, No. 68 Zhongshan Road, Wuxi, 214002, Jiangsu, China.
Zhou T; Department of Cardiology, The Affiliated Wuxi No.2 People's Hospital of Nanjing Medical University, No. 68 Zhongshan Road, Wuxi, 214002, Jiangsu, China.
Feng Q; Department of Cardiology, The Affiliated Wuxi No.2 People's Hospital of Nanjing Medical University, No. 68 Zhongshan Road, Wuxi, 214002, Jiangsu, China.
Yang J; Department of Cardiology, The Affiliated Wuxi No.2 People's Hospital of Nanjing Medical University, No. 68 Zhongshan Road, Wuxi, 214002, Jiangsu, China.
Cao J; Department of Cardiology, The Affiliated Wuxi No.2 People's Hospital of Nanjing Medical University, No. 68 Zhongshan Road, Wuxi, 214002, Jiangsu, China.
Xu X; Department of Cardiology, The Affiliated Wuxi No.2 People's Hospital of Nanjing Medical University, No. 68 Zhongshan Road, Wuxi, 214002, Jiangsu, China.
Yang C; Department of Cardiology, The Affiliated Wuxi No.2 People's Hospital of Nanjing Medical University, No. 68 Zhongshan Road, Wuxi, 214002, Jiangsu, China. .; Pokaż więcej
Źródło :: Journal of cardiovascular translational research [J Cardiovasc Transl Res] 2020 Aug; Vol. 13 (4), pp. 584-592. Date of Electronic Publication: 2019 Dec 02.
Typ publikacji :: Journal Article
MeSH Terms :: Heart Rate*
Connexin 43/*metabolism
Gap Junctions/*metabolism
MicroRNAs/*metabolism
Myocardial Reperfusion Injury/*prevention & control
Myocytes, Cardiac/*metabolism
Oligonucleotides, Antisense/*administration & dosage
Tachycardia, Ventricular/*prevention & control
Ventricular Fibrillation/*prevention & control
Animals ; Connexin 43/genetics ; Disease Models, Animal ; Gap Junctions/genetics ; Male ; Mice, Inbred C57BL ; MicroRNAs/genetics ; Myocardial Reperfusion Injury/genetics ; Myocardial Reperfusion Injury/metabolism ; Myocardial Reperfusion Injury/physiopathology ; Signal Transduction ; Tachycardia, Ventricular/genetics ; Tachycardia, Ventricular/physiopathology ; Ventricular Fibrillation/genetics ; Ventricular Fibrillation/metabolism ; Ventricular Fibrillation/physiopathology
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 10 10.

Tytuł :: The atypical chemokine receptor 3 interacts with Connexin 43 inhibiting astrocytic gap junctional intercellular communication.
Autorzy :: Fumagalli A; Institut de Génomique Fonctionnelle, Université de Montpellier, CNRS, INSERM, Montpellier, France.
Heuninck J; Institut de Génomique Fonctionnelle, Université de Montpellier, CNRS, INSERM, Montpellier, France.
Pizzoccaro A; Institut de Génomique Fonctionnelle, Université de Montpellier, CNRS, INSERM, Montpellier, France.
Moutin E; Institut de Génomique Fonctionnelle, Université de Montpellier, CNRS, INSERM, Montpellier, France.
Koenen J; Université Paris-Saclay, Inserm, Inflammation, Microbiome and Immunosurveillance, 92140, Clamart, France.; Amsterdam Institute for Molecules Medicines and Systems, Division of Medicinal Chemistry, Faculty of Sciences, VU University Amsterdam, 1081, HV, Amsterdam, The Netherlands.
Séveno M; Biocampus Montpellier, Université de Montpellier, CNRS, INSERM, Montpellier, France.
Durroux T; Institut de Génomique Fonctionnelle, Université de Montpellier, CNRS, INSERM, Montpellier, France.
Junier MP; CNRS UMR8246, Inserm U1130, Neuroscience Paris Seine-IBPS, Sorbonne Universités, Paris, France.
Schlecht-Louf G; Université Paris-Saclay, Inserm, Inflammation, Microbiome and Immunosurveillance, 92140, Clamart, France.
Bachelerie F; Université Paris-Saclay, Inserm, Inflammation, Microbiome and Immunosurveillance, 92140, Clamart, France.
Schütz D; Institute of Pharmacology and Toxicology, Jena University Hospital, 07747, Jena, Germany.
Stumm R; Institute of Pharmacology and Toxicology, Jena University Hospital, 07747, Jena, Germany.
Smit MJ; Amsterdam Institute for Molecules Medicines and Systems, Division of Medicinal Chemistry, Faculty of Sciences, VU University Amsterdam, 1081, HV, Amsterdam, The Netherlands.
Guérineau NC; Institut de Génomique Fonctionnelle, Université de Montpellier, CNRS, INSERM, Montpellier, France.
Chaumont-Dubel S; Institut de Génomique Fonctionnelle, Université de Montpellier, CNRS, INSERM, Montpellier, France.
Marin P; Institut de Génomique Fonctionnelle, Université de Montpellier, CNRS, INSERM, Montpellier, France. .; Pokaż więcej
Źródło :: Nature communications [Nat Commun] 2020 Sep 25; Vol. 11 (1), pp. 4855. Date of Electronic Publication: 2020 Sep 25.
Typ publikacji :: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :: Astrocytes/*metabolism
Cell Communication/*physiology
Connexin 43/*metabolism
Gap Junctions/*pathology
Receptors, CXCR/*metabolism
Animals ; Cell Proliferation ; Connexin 43/drug effects ; Connexins/metabolism ; Gene Knock-In Techniques ; Glioblastoma/metabolism ; HEK293 Cells ; Humans ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Protein Interaction Domains and Motifs ; Receptors, CXCR/agonists ; Receptors, CXCR/genetics ; Signal Transduction/physiology
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 11 11.

Tytuł :: Auxiliary trafficking subunit GJA1-20k protects connexin-43 from degradation and limits ventricular arrhythmias.
Autorzy :: Xiao S; Nora Eccles Harrison Cardiovascular Research and Training Institute, University of Utah, Salt Lake City, Utah, USA.; Department of Neurology, UCLA, Los Angeles, California, USA.
Shimura D; Nora Eccles Harrison Cardiovascular Research and Training Institute, University of Utah, Salt Lake City, Utah, USA.
Baum R; Nora Eccles Harrison Cardiovascular Research and Training Institute, University of Utah, Salt Lake City, Utah, USA.; Department of Biomedical Sciences, Cedars-Sinai Medical Center, Los Angeles, California, USA.
Hernandez DM; Department of Biomedical Sciences, Cedars-Sinai Medical Center, Los Angeles, California, USA.
Agvanian S; Department of Biomedical Sciences, Cedars-Sinai Medical Center, Los Angeles, California, USA.
Nagaoka Y; Department of Biomedical Sciences, Cedars-Sinai Medical Center, Los Angeles, California, USA.; Department of Pathology, University of Alabama at Birmingham, Birmingham, Alabama, USA.
Katsumata M; Department of Biomedical Sciences, Cedars-Sinai Medical Center, Los Angeles, California, USA.
Lampe PD; Translational Research Program, Public Health Sciences and Human Biology Divisions, Fred Hutchinson Cancer Research Center, Seattle, Washington, USA.
Kleber AG; Department of Pathology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA.
Hong T; Nora Eccles Harrison Cardiovascular Research and Training Institute, University of Utah, Salt Lake City, Utah, USA.; Department of Biomedical Sciences, Cedars-Sinai Medical Center, Los Angeles, California, USA.
Shaw RM; Nora Eccles Harrison Cardiovascular Research and Training Institute, University of Utah, Salt Lake City, Utah, USA.; Pokaż więcej
Źródło :: The Journal of clinical investigation [J Clin Invest] 2020 Sep 01; Vol. 130 (9), pp. 4858-4870.
Typ publikacji :: Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :: Proteolysis*
Arrhythmias, Cardiac/*metabolism
Connexin 43/*metabolism
Gap Junctions/*metabolism
Heart Ventricles/*metabolism
Animals ; Arrhythmias, Cardiac/genetics ; Arrhythmias, Cardiac/pathology ; CRISPR-Cas Systems ; Connexin 43/genetics ; Gap Junctions/genetics ; Gap Junctions/pathology ; Heart Ventricles/pathology ; Mice ; Mice, Mutant Strains ; Protein Transport
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 12 12.

Tytuł :: Myofibers deficient in connexins 43 and 45 expression protect mice from skeletal muscle and systemic dysfunction promoted by a dysferlin mutation.
Autorzy :: Fernández G; Instituto de Ciencias Biomédicas, Facultad de Ciencias de la Salud, Universidad Autónoma de Chile, Santiago, Chile.
Arias-Bravo G; Instituto de Ciencias Biomédicas, Facultad de Ciencias de la Salud, Universidad Autónoma de Chile, Santiago, Chile; Instituto de Ciencias de la Salud, Universidad de O'Higgins, Rancagua, Chile.
Bevilacqua JA; Departamento de Neurología y Neurocirugía, Hospital Clínico Univeridad de Chile y Departamento de Anatomía y Medicina legal, Facultad de Medicina, Universidad de Chile, Santiago, Chile; Departamento de Neurología y Neurocirugía, Clínica Davila, y Corporacion de Investigaciones Neurológicas de Santiago (CINSAN), Santiago, Chile.
Castillo-Ruiz M; Escuela de Química y Farmacia, Facultad de Medicina, Universidad Andres Bello, Santiago, Chile; Departamento de Ciencias Químicas y Biológicas, Facultad de Salud, Universidad Bernardo O Higgins, Santiago, Chile.
Caviedes P; Programa de Farmacología, Instituto de Ciencias Biomédicas, Facultad de Medicina, Universidad de Chile, Santiago, Chile; Centro de Biotecnología y Bioingeniería (CeBiB), Depto de Ingeniería Química, Biotecnología y Materiales, Fac. de Ciencias Físicas y Matemáticas, Universidad de Chile, Santiago, Chile.
Sáez JC; Departamento de Fisiología, Fac. de Ciencias Biológicas, Pontificia Universidad Católica de Chile, Santiago, Chile; Instituto de Neurociencias, Centro Interdisciplinario de Neurociencias, Universidad de Valparaíso, Valparaíso, Chile. Electronic address: .
Cea LA; Instituto de Ciencias Biomédicas, Facultad de Ciencias de la Salud, Universidad Autónoma de Chile, Santiago, Chile. Electronic address: .; Pokaż więcej
Źródło :: Biochimica et biophysica acta. Molecular basis of disease [Biochim Biophys Acta Mol Basis Dis] 2020 Aug 01; Vol. 1866 (8), pp. 165800. Date of Electronic Publication: 2020 Apr 16.
Typ publikacji :: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :: Connexin 43/*genetics
Connexins/*genetics
Dysferlin/*genetics
Muscular Dystrophies, Limb-Girdle/*genetics
Muscular Dystrophies, Limb-Girdle/*prevention & control
Myofibrils/*genetics
Animals ; Calcium/metabolism ; Connexin 43/deficiency ; Connexins/deficiency ; Creatine Kinase/blood ; Creatine Kinase/genetics ; Disease Models, Animal ; Dysferlin/deficiency ; Gene Expression ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Muscular Dystrophies, Limb-Girdle/metabolism ; Muscular Dystrophies, Limb-Girdle/pathology ; Mutation ; Myofibrils/metabolism ; Myofibrils/pathology ; Permeability ; Physical Conditioning, Animal ; Rotarod Performance Test ; Sarcolemma/metabolism
SCR Disease Name :: Dysferlinopathy
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 13 13.

Tytuł :: Cx43 in Neural Progenitors Promotes Glioma Invasion in a 3D Culture System.
Autorzy :: Khosla K; Department of Cellular and Physiological Sciences, Life Sciences Institute, The University of British Columbia, Vancouver, BC V6T 1Z3, Canada.
Naus CC; Department of Cellular and Physiological Sciences, Life Sciences Institute, The University of British Columbia, Vancouver, BC V6T 1Z3, Canada.
Sin WC; Department of Cellular and Physiological Sciences, Life Sciences Institute, The University of British Columbia, Vancouver, BC V6T 1Z3, Canada.; Pokaż więcej
Źródło :: International journal of molecular sciences [Int J Mol Sci] 2020 Jul 23; Vol. 21 (15). Date of Electronic Publication: 2020 Jul 23.
Typ publikacji :: Journal Article
MeSH Terms :: Models, Biological*
Connexin 43/*metabolism
Glioma/*metabolism
Neoplasm Proteins/*metabolism
Neural Stem Cells/*metabolism
Animals ; Coculture Techniques ; Connexin 43/genetics ; Glioma/genetics ; Glioma/pathology ; Mice ; Mice, Knockout ; Neoplasm Invasiveness ; Neoplasm Proteins/genetics ; Neural Stem Cells/pathology ; Tumor Cells, Cultured
: Czasopismo naukowe

Szczegóły Pełny tekst Pełny tekst

Skocz do pozycji: 14 14.

Tytuł :: Connexin 43 prevents the progression of diabetic renal tubulointerstitial fibrosis by regulating the SIRT1-HIF-1α signaling pathway.
Autorzy :: Sun X; Laboratory of Pharmacology and Toxicology, School of Pharmaceutical Sciences, Sun Yat-sen University, Guangzhou 510006, China.
Huang K; Phase I Clinical Trial Center, Guangzhou Eighth People's Hospital, Guangzhou Medical University, Guangzhou 510060, China.
Haiming X; Laboratory of Pharmacology and Toxicology, School of Pharmaceutical Sciences, Sun Yat-sen University, Guangzhou 510006, China.
Lin Z; Laboratory of Pharmacology and Toxicology, School of Pharmaceutical Sciences, Sun Yat-sen University, Guangzhou 510006, China.
Yang Y; Laboratory of Pharmacology and Toxicology, School of Pharmaceutical Sciences, Sun Yat-sen University, Guangzhou 510006, China.
Zhang M; Laboratory of Pharmacology and Toxicology, School of Pharmaceutical Sciences, Sun Yat-sen University, Guangzhou 510006, China.
Liu P; Laboratory of Pharmacology and Toxicology, School of Pharmaceutical Sciences, Sun Yat-sen University, Guangzhou 510006, China.
Huang H; Laboratory of Pharmacology and Toxicology, School of Pharmaceutical Sciences, Sun Yat-sen University, Guangzhou 510006, China.; Guangdong Provincial Key Laboratory of New Drug Design and Evaluation, Sun Yat-sen University, Guangzhou 510006, China.; Pokaż więcej
Źródło :: Clinical science (London, England : 1979) [Clin Sci (Lond)] 2020 Jul 17; Vol. 134 (13), pp. 1573-1592.
Typ publikacji :: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :: Connexin 43/*metabolism
Diabetic Nephropathies/*metabolism
Hypoxia-Inducible Factor 1, alpha Subunit/*metabolism
Kidney Tubules/*metabolism
Sirtuin 1/*metabolism
Animals ; Connexin 43/genetics ; Diabetic Nephropathies/genetics ; Diabetic Nephropathies/pathology ; Disease Progression ; Fibrosis/genetics ; Fibrosis/metabolism ; Fibrosis/pathology ; Humans ; Hypoxia-Inducible Factor 1, alpha Subunit/genetics ; Kidney Tubules/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Signal Transduction ; Sirtuin 1/genetics
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 15 15.

Tytuł :: Adenovirus targets transcriptional and posttranslational mechanisms to limit gap junction function.
Autorzy :: Calhoun PJ; Fralin Biomedical Research Institute at VTC, Roanoke, VA, USA.; Department of Biological Sciences, Virginia Tech, Blacksburg, VA, USA.
Phan AV; Virginia Tech Carilion School of Medicine, Roanoke, VA, USA.
Taylor JD; Virginia Tech Carilion School of Medicine, Roanoke, VA, USA.
James CC; Fralin Biomedical Research Institute at VTC, Roanoke, VA, USA.; Graduate Program in Translational Biology, Medicine, and Health, Virginia Tech, Blacksburg, VA, USA.
Padget RL; Fralin Biomedical Research Institute at VTC, Roanoke, VA, USA.; Graduate Program in Translational Biology, Medicine, and Health, Virginia Tech, Blacksburg, VA, USA.
Zeitz MJ; Fralin Biomedical Research Institute at VTC, Roanoke, VA, USA.
Smyth JW; Fralin Biomedical Research Institute at VTC, Roanoke, VA, USA.; Department of Biological Sciences, Virginia Tech, Blacksburg, VA, USA.; Virginia Tech Carilion School of Medicine, Roanoke, VA, USA.; Pokaż więcej
Źródło :: FASEB journal : official publication of the Federation of American Societies for Experimental Biology [FASEB J] 2020 Jul; Vol. 34 (7), pp. 9694-9712. Date of Electronic Publication: 2020 Jun 02.
Typ publikacji :: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :: Cell Communication*
Transcription, Genetic*
Adenoviridae/*physiology
Adenoviridae Infections/*pathology
Connexin 43/*metabolism
Gap Junctions/*pathology
Myocytes, Cardiac/*pathology
Adenoviridae Infections/metabolism ; Adenoviridae Infections/virology ; Cells, Cultured ; Connexin 43/genetics ; Gap Junctions/virology ; Gene Expression Regulation ; HEK293 Cells ; Humans ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/virology ; Phosphorylation
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 16 16.

Tytuł :: Connexin43 Expression and Associated Chronic Inflammation Presages the Development of Cerebral Radiation Necrosis.
Autorzy :: Feldman LA; Department of Pharmacology and Clinical Pharmacology, School of Medical Sciences, Faculty of Medical and Health Sciences, University of Auckland, Auckland, New Zealand.; Centre for Brain Research, University of Auckland, Auckland, New Zealand.
Haldankar S; Department of Pharmacology and Clinical Pharmacology, School of Medical Sciences, Faculty of Medical and Health Sciences, University of Auckland, Auckland, New Zealand.; Centre for Brain Research, University of Auckland, Auckland, New Zealand.
O'Carroll SJ; Centre for Brain Research, University of Auckland, Auckland, New Zealand.; Department of Anatomy and Medical Imaging, School of Medical Sciences, Faculty of Medical and Health Sciences, University of Auckland, Auckland, New Zealand.
Liu K; Department of Pharmacology and Clinical Pharmacology, School of Medical Sciences, Faculty of Medical and Health Sciences, University of Auckland, Auckland, New Zealand.; Centre for Brain Research, University of Auckland, Auckland, New Zealand.
Fackelmeier B; Centre for Brain Research, University of Auckland, Auckland, New Zealand.; Department of Anatomy and Medical Imaging, School of Medical Sciences, Faculty of Medical and Health Sciences, University of Auckland, Auckland, New Zealand.
Broaddus WC; Department of Neurosurgery, Virginia Commonwealth University, Richmond, Virginia.
Anene-Maidoh T; Department of Neurosurgery, Virginia Commonwealth University, Richmond, Virginia.
Green CR; Department of Ophthalmology, School of Medicine, Faculty of Medical and Health Sciences, University of Auckland, Auckland, New Zealand.
Garbow JR; Biomedical MR Laboratory, Mallinckrodt Institute of Radiology, Washington University, St. Louis, Missouri.
Guan J; Department of Pharmacology and Clinical Pharmacology, School of Medical Sciences, Faculty of Medical and Health Sciences, University of Auckland, Auckland, New Zealand.; Centre for Brain Research, University of Auckland, Auckland, New Zealand.; Pokaż więcej
Źródło :: Journal of neuropathology and experimental neurology [J Neuropathol Exp Neurol] 2020 Jul 01; Vol. 79 (7), pp. 791-799.
Typ publikacji :: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :: Brain/*metabolism
Brain/*radiation effects
Brain Injuries/*metabolism
Connexin 43/*biosynthesis
Radiation Injuries/*metabolism
Animals ; Brain/pathology ; Brain Injuries/genetics ; Brain Injuries/pathology ; Connexin 43/genetics ; Female ; Gene Expression ; Inflammation/metabolism ; Inflammation/pathology ; Mice ; Mice, Inbred BALB C ; Necrosis/metabolism ; Necrosis/pathology ; Radiation Injuries/genetics ; Radiation Injuries/pathology
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 17 17.

Tytuł :: Increased expression of connexin 43 in a mouse model of spinal motoneuronal loss.
Autorzy :: Spitale FM; Department of Biomedical and Biotechnological Sciences, Section of Physiology, University of Catania, Catania 95123, Italy.
Vicario N; Department of Biomedical and Biotechnological Sciences, Section of Physiology, University of Catania, Catania 95123, Italy.
Rosa MD; Department of Biomedical and Biotechnological Sciences, Section of Anatomy, Histology and Movement Sciences, University of Catania, Catania 95123, Italy.
Tibullo D; Department of Biomedical and Biotechnological Sciences, Section of Biochemistry, University of Catania, Catania 95123, Italy.
Vecchio M; Department of Biomedical and Biotechnological Sciences, Section of Pharmacology, University of Catania, Catania 95123, Italy.; Rehabilitation Unit, 'AOU Policlinico Vittorio Emanuele', University of Catania, Catania 95123, Italy.
Gulino R; Department of Biomedical and Biotechnological Sciences, Section of Physiology, University of Catania, Catania 95123, Italy.
Parenti R; Department of Biomedical and Biotechnological Sciences, Section of Physiology, University of Catania, Catania 95123, Italy.; Pokaż więcej
Źródło :: Aging [Aging (Albany NY)] 2020 Jun 24; Vol. 12 (13), pp. 12598-12608. Date of Electronic Publication: 2020 Jun 24.
Typ publikacji :: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :: Amyotrophic Lateral Sclerosis*/metabolism
Amyotrophic Lateral Sclerosis*/physiopathology
Spinal Cord*/chemistry
Spinal Cord*/cytology
Spinal Cord*/metabolism
Spinal Cord*/physiopathology
Connexin 43/*metabolism
Motor Neurons/*metabolism
Adult ; Animals ; Astrocytes/metabolism ; Connexin 43/genetics ; Disease Models, Animal ; Female ; Gap Junctions/metabolism ; Glial Fibrillary Acidic Protein/metabolism ; Humans ; Male ; Mice ; Middle Aged ; Motor Neurons/cytology
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 18 18.

Tytuł :: Soluble silica stimulates osteogenic differentiation and gap junction communication in human dental follicle cells.
Autorzy :: Uribe P; Department of Orthodontics, Institute of Odontology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden. .
Johansson A; Unit of Molecular Periodontology, Department of Odontology, University of Umeå, Umeå, Sweden.
Jugdaohsingh R; Biomineral Research Group, Department of Veterinary Medicine, University of Cambridge, Cambridge, UK.
Powell JJ; Biomineral Research Group, Department of Veterinary Medicine, University of Cambridge, Cambridge, UK.
Magnusson C; Department of Orthodontics, Institute of Odontology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
Davila M; The Bioinformatics Core Facility at the University of Gothenburg, Gothenburg, Sweden.
Westerlund A; Department of Orthodontics, Institute of Odontology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
Ransjö M; Department of Orthodontics, Institute of Odontology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.; Pokaż więcej
Źródło :: Scientific reports [Sci Rep] 2020 Jun 18; Vol. 10 (1), pp. 9923. Date of Electronic Publication: 2020 Jun 18.
Typ publikacji :: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :: Cell Differentiation*
Osteogenesis*
Connexin 43/*metabolism
Dental Sac/*cytology
Gap Junctions/*physiology
Osteoblasts/*cytology
Silicon Dioxide/*pharmacology
Adolescent ; Cell Proliferation ; Cells, Cultured ; Child ; Connexin 43/genetics ; Dental Sac/drug effects ; Dental Sac/metabolism ; Humans ; Osteoblasts/drug effects ; Osteoblasts/metabolism
: Czasopismo naukowe

Szczegóły Pełny tekst Pełny tekst

Skocz do pozycji: 19 19.

Tytuł :: The Connexin 43 Regulator Rotigaptide Reduces Cytokine-Induced Cell Death in Human Islets.
Autorzy :: Ghiasi SM; Section of Cell Biology and Functional Genomics, Division of Diabetes, Endocrinology and Metabolism, Department of Metabolism, Digestion and Reproduction, Faculty of Medicine, Imperial College London, London W12 0NN, UK.; Department of Biomedical Sciences, University of Copenhagen, 2200 Copenhagen N, Denmark.
Hansen JB; Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, 2200 Copenhagen N, Denmark.
Christensen DP; Department of Biology, University of Copenhagen, 2200 Copenhagen N, Denmark.
Tyrberg B; Department of Physiology, Sahlgrenska Academy, Gothenburg University, 40530 Gothenburg, Sweden.; Cardiovascular and Metabolic Diseases, Institute de recherche Servier, 92150 Suresnes, France.
Mandrup-Poulsen T; Department of Biomedical Sciences, University of Copenhagen, 2200 Copenhagen N, Denmark.; Pokaż więcej
Źródło :: International journal of molecular sciences [Int J Mol Sci] 2020 Jun 17; Vol. 21 (12). Date of Electronic Publication: 2020 Jun 17.
Typ publikacji :: Journal Article
MeSH Terms :: Connexin 43/*metabolism
Cytokines/*metabolism
Islets of Langerhans/*drug effects
Islets of Langerhans/*metabolism
Oligopeptides/*pharmacology
Apoptosis/drug effects ; Biomarkers ; Cell Death/drug effects ; Cell Line ; Connexin 43/genetics ; Cytokines/genetics ; Gap Junctions/metabolism ; Gene Expression Regulation ; Humans ; Insulin/metabolism ; Insulin Secretion/drug effects ; Insulin-Secreting Cells/drug effects ; Insulin-Secreting Cells/metabolism ; Islets of Langerhans/cytology ; Mitochondria/drug effects ; Mitochondria/genetics ; Mitochondria/metabolism ; NF-kappa B/metabolism ; Reactive Oxygen Species/metabolism ; Stress, Physiological
: Czasopismo naukowe

Szczegóły Pełny tekst

Skocz do pozycji: 20 20.

Tytuł :: Cx30.2 deletion causes imbalances in testicular Cx43, Cx46, and Cx50 and insulin receptors. Reciprocally, diabetes/obesity alters Cx30.2 in mouse testis.
Autorzy :: Pelletier RM; Department of Pathology and Cell Biology, Université de Montréal, Québec, Canada.
Layeghkhavidaki H; Department of Pathology and Cell Biology, Université de Montréal, Québec, Canada.
Kumar NM; Department of Ophthalmology and Visual Sciences, University of Illinois, Chicago, Illinois.
Vitale ML; Department of Pathology and Cell Biology, Université de Montréal, Québec, Canada.; Pokaż więcej
Źródło :: American journal of physiology. Regulatory, integrative and comparative physiology [Am J Physiol Regul Integr Comp Physiol] 2020 Jun 01; Vol. 318 (6), pp. R1078-R1090. Date of Electronic Publication: 2020 Apr 29.
Typ publikacji :: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :: Connexin 43/*genetics
Connexins/*genetics
Diabetes Mellitus, Experimental/*genetics
Obesity/*genetics
Receptor, Insulin/*genetics
Testis/*metabolism
Animals ; Connexin 43/metabolism ; Connexins/metabolism ; Diabetes Mellitus, Experimental/metabolism ; Gene Expression Regulation ; Male ; Mice ; Mice, Knockout ; Obesity/metabolism ; Receptor, Insulin/metabolism
: Czasopismo naukowe

Szczegóły

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