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Tytuł:
CX3CR1 knockout aggravates Coxsackievirus B3-induced myocarditis.
Autorzy:
Müller I; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Berlin, Germany.; DZHK (German Center for Cardiovascular Research), partner site Berlin, Germany.; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin-Brandenburg Center for Regenerative Therapies, Campus Virchow Klinikum, Berlin, Germany.
Pappritz K; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Berlin, Germany.; DZHK (German Center for Cardiovascular Research), partner site Berlin, Germany.; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin-Brandenburg Center for Regenerative Therapies, Campus Virchow Klinikum, Berlin, Germany.
Savvatis K; Inherited Cardiovascular Diseases Unit, Barts Health NHS Trust, Barts Heart Centre, London, United Kingdom.; William Harvey Research Institute, Queen Mary University London, London, United Kingdom.
Puhl K; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Berlin, Germany.; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin-Brandenburg Center for Regenerative Therapies, Campus Virchow Klinikum, Berlin, Germany.
Dong F; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Berlin, Germany.; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin-Brandenburg Center for Regenerative Therapies, Campus Virchow Klinikum, Berlin, Germany.
El-Shafeey M; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Berlin, Germany.; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin-Brandenburg Center for Regenerative Therapies, Campus Virchow Klinikum, Berlin, Germany.
Hamdani N; Department of Cardiovascular Physiology, Ruhr University Bochum, Bochum, Germany.
Hamann I; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Institute for Medical Immunology, Campus Virchow Klinikum, Berlin, Germany.
Noutsias M; Department of Internal Medicine III, Division of Cardiology, Angiology and Intensive Medical Care, University Hospital Halle, Halle (Saale), Germany.
Infante-Duarte C; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Institute for Medical Immunology, Campus Virchow Klinikum, Berlin, Germany.
Linke WA; Department of Cardiovascular Physiology, Ruhr University Bochum, Bochum, Germany.
Van Linthout S; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Berlin, Germany.; DZHK (German Center for Cardiovascular Research), partner site Berlin, Germany.; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin-Brandenburg Center for Regenerative Therapies, Campus Virchow Klinikum, Berlin, Germany.
Tschöpe C; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Berlin, Germany.; DZHK (German Center for Cardiovascular Research), partner site Berlin, Germany.; Charité -Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin-Brandenburg Center for Regenerative Therapies, Campus Virchow Klinikum, Berlin, Germany.
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Źródło:
PloS one [PLoS One] 2017 Aug 11; Vol. 12 (8), pp. e0182643. Date of Electronic Publication: 2017 Aug 11 (Print Publication: 2017).
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CX3CL1/*immunology
Coxsackievirus Infections/*genetics
Enterovirus B, Human/*pathogenicity
Host-Pathogen Interactions/*immunology
Myocarditis/*genetics
Receptors, Chemokine/*immunology
Animals ; Apoptosis ; CX3C Chemokine Receptor 1 ; Cell Adhesion Molecules/genetics ; Cell Adhesion Molecules/immunology ; Chemokine CX3CL1/genetics ; Coxsackievirus Infections/immunology ; Coxsackievirus Infections/pathology ; Coxsackievirus Infections/virology ; Disease Models, Animal ; Enterovirus B, Human/growth & development ; Gene Expression Regulation ; Heart Function Tests ; Humans ; Interleukins/genetics ; Interleukins/immunology ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Myocarditis/immunology ; Myocarditis/pathology ; Myocarditis/virology ; Myocytes, Cardiac/immunology ; Myocytes, Cardiac/pathology ; Phosphorylation ; Protein Kinases/genetics ; Protein Kinases/immunology ; Receptors, Chemokine/deficiency ; Receptors, Chemokine/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Role of the CX3C chemokine receptor CX3CR1 in the pathogenesis of atherosclerosis after aortic transplantation.
Autorzy:
Rowinska Z; Department of Vascular Surgery and Interdisciplinary Vein Center, St. Josef-Hospital, Ruhr- University Bochum, Bochum, Germany.; Institute of Molecular Cardiovascular Research, University Hospital, RWTH Aachen University, Aachen, Germany.
Koeppel TA; Division of Vascular Surgery, Hospital Asklepios St. Georg Hamburg, Hamburg, Germany.
Sanati M; Institute of Molecular Cardiovascular Research, University Hospital, RWTH Aachen University, Aachen, Germany.
Schelzig H; Department of Vascular and Endovascular Surgery, Düsseldorf University Hospital, Düsseldorf, Germany.
Jankowski J; Institute of Molecular Cardiovascular Research, University Hospital, RWTH Aachen University, Aachen, Germany.; School for Cardiovascular Diseases (CARIM), University of Maastricht, Maastricht, The Netherlands.
Weber C; Institut for Prevention and Epidemiology of Cardiovascular Disease, Ludwig-Maximilian-University of Munich, Munich, Germany.
Zernecke A; Institute of Experimental Biomedicine, University Hospital Würzburg, Würzburg, Germany.
Liehn EA; Institute of Molecular Cardiovascular Research, University Hospital, RWTH Aachen University, Aachen, Germany.; Human Genetic Laboratory, University for Medicine and Pharmacy, Craiova, Romania.
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Źródło:
PloS one [PLoS One] 2017 Feb 24; Vol. 12 (2), pp. e0170644. Date of Electronic Publication: 2017 Feb 24 (Print Publication: 2017).
Typ publikacji:
Journal Article
MeSH Terms:
Aorta/*transplantation
Apolipoproteins E/*genetics
Atherosclerosis/*therapy
Receptors, Chemokine/*genetics
Animals ; Aorta/physiopathology ; Atherosclerosis/genetics ; Atherosclerosis/physiopathology ; CX3C Chemokine Receptor 1 ; Chemokines, CX3C ; Disease Models, Animal ; Humans ; Macrophages ; Mice ; Mice, Knockout ; Monocytes ; Myocytes, Smooth Muscle/metabolism ; Myocytes, Smooth Muscle/pathology ; Plaque, Atherosclerotic/genetics ; Plaque, Atherosclerotic/physiopathology ; Plaque, Atherosclerotic/therapy ; Tunica Intima/metabolism ; Tunica Intima/physiopathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CX 3 C chemokine receptor 1 deficiency modulates microglia morphology but does not affect lesion size and short-term deficits after experimental stroke.
Autorzy:
van der Maten G; Laboratory for Experimental Brain Research, Division of Neurosurgery, Department of Clinical Sciences, Wallenberg Neuroscience Center, Lund University, BMC A13, 22184, Lund, Sweden.
Henck V; Laboratory for Experimental Brain Research, Division of Neurosurgery, Department of Clinical Sciences, Wallenberg Neuroscience Center, Lund University, BMC A13, 22184, Lund, Sweden.
Wieloch T; Laboratory for Experimental Brain Research, Division of Neurosurgery, Department of Clinical Sciences, Wallenberg Neuroscience Center, Lund University, BMC A13, 22184, Lund, Sweden.
Ruscher K; Laboratory for Experimental Brain Research, Division of Neurosurgery, Department of Clinical Sciences, Wallenberg Neuroscience Center, Lund University, BMC A13, 22184, Lund, Sweden. .
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Źródło:
BMC neuroscience [BMC Neurosci] 2017 Jan 06; Vol. 18 (1), pp. 11. Date of Electronic Publication: 2017 Jan 06.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Microglia/*metabolism
Microglia/*pathology
Receptors, Chemokine/*deficiency
Stroke/*metabolism
Stroke/*pathology
Animals ; Brain/metabolism ; Brain/pathology ; CX3C Chemokine Receptor 1 ; Calcium-Binding Proteins/metabolism ; Disease Models, Animal ; Green Fluorescent Proteins/genetics ; Green Fluorescent Proteins/metabolism ; Immunohistochemistry ; Male ; Mice, Inbred C57BL ; Mice, Transgenic ; Microfilament Proteins/metabolism ; Receptors, Chemokine/genetics ; Recovery of Function/physiology ; Time Factors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Activation of cannabinoid receptor 2 attenuates mechanical allodynia and neuroinflammatory responses in a chronic post-ischemic pain model of complex regional pain syndrome type I in rats.
Autorzy:
Xu J; Department of Pain Management, Cleveland Clinic, Cleveland, OH, USA.; Department of Immunology, Cleveland Clinic, Cleveland, OH, USA.
Tang Y; Department of Anesthesiology, West China Second Hospital, Sichuan University, Chengdu, Sichuan, China.; Department of General Anesthesiology, Cleveland Clinic, Cleveland, OH, USA.
Xie M; Department of Pain Management, Cleveland Clinic, Cleveland, OH, USA.
Bie B; Department of General Anesthesiology, Cleveland Clinic, Cleveland, OH, USA.
Wu J; Department of General Anesthesiology, Cleveland Clinic, Cleveland, OH, USA.
Yang H; Department of General Anesthesiology, Cleveland Clinic, Cleveland, OH, USA.
Foss JF; Department of General Anesthesiology, Cleveland Clinic, Cleveland, OH, USA.
Yang B; Department of Pathology, Cleveland Clinic, Cleveland, OH, USA.
Rosenquist RW; Department of Pain Management, Cleveland Clinic, Cleveland, OH, USA.
Naguib M; Department of General Anesthesiology, Cleveland Clinic, Cleveland, OH, USA.; Anesthesiology Institute, Cleveland Clinic, 9500 Euclid Ave. - NE6-306, Cleveland, OH, 44195, USA.
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Źródło:
The European journal of neuroscience [Eur J Neurosci] 2016 Dec; Vol. 44 (12), pp. 3046-3055. Date of Electronic Publication: 2016 Oct 13.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Encephalitis/*physiopathology
Hyperalgesia/*physiopathology
Microglia/*physiology
Receptor, Cannabinoid, CB2/*physiology
Receptors, Chemokine/*physiology
Reflex Sympathetic Dystrophy/*physiopathology
Animals ; Benzofurans/administration & dosage ; CX3C Chemokine Receptor 1 ; Disease Models, Animal ; Encephalitis/complications ; Encephalitis/prevention & control ; Epidermis/innervation ; Hyperalgesia/complications ; Hyperalgesia/prevention & control ; Ischemia/physiopathology ; Male ; Microglia/drug effects ; Pain/prevention & control ; Piperidines/administration & dosage ; Rats ; Rats, Sprague-Dawley ; Receptor, Cannabinoid, CB2/agonists ; Reflex Sympathetic Dystrophy/complications ; Spinal Cord Dorsal Horn/drug effects ; Spinal Cord Dorsal Horn/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Progesterone Attenuates Microglial-Driven Retinal Degeneration and Stimulates Protective Fractalkine-CX3CR1 Signaling.
Autorzy:
Roche SL; Cell Development and Disease Laboratory, Biochemistry Department, Biosciences Institute, University College Cork, Cork, Ireland.
Wyse-Jackson AC; Cell Development and Disease Laboratory, Biochemistry Department, Biosciences Institute, University College Cork, Cork, Ireland.
Gómez-Vicente V; Departamento de Óptica, Farmacología y Anatomía, Universidad de Alicante, Alicante, Spain.
Lax P; Departamento de Fisiología, Genética y Microbiología, Universidad de Alicante, Alicante, Spain.
Ruiz-Lopez AM; Cell Development and Disease Laboratory, Biochemistry Department, Biosciences Institute, University College Cork, Cork, Ireland.
Byrne AM; Cell Development and Disease Laboratory, Biochemistry Department, Biosciences Institute, University College Cork, Cork, Ireland.
Cuenca N; Departamento de Fisiología, Genética y Microbiología, Universidad de Alicante, Alicante, Spain.
Cotter TG; Cell Development and Disease Laboratory, Biochemistry Department, Biosciences Institute, University College Cork, Cork, Ireland.
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Źródło:
PloS one [PLoS One] 2016 Nov 04; Vol. 11 (11), pp. e0165197. Date of Electronic Publication: 2016 Nov 04 (Print Publication: 2016).
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CX3CL1/*metabolism
Microglia/*metabolism
Neuroprotective Agents/*metabolism
Progesterone/*metabolism
Receptors, Chemokine/*metabolism
Retinal Degeneration/*metabolism
Signal Transduction/*physiology
Animals ; CX3C Chemokine Receptor 1 ; Cell Line ; Central Nervous System Stimulants/metabolism ; Disease Models, Animal ; Female ; Male ; Mice ; Mice, Inbred C57BL ; Norgestrel/metabolism ; Photoreceptor Cells, Vertebrate/metabolism ; Retina/metabolism ; Retinitis Pigmentosa/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CX3CR1 Disruption Differentially Influences Dopaminergic Neuron Degeneration in Parkinsonian Mice Depending on the Neurotoxin and Route of Administration.
Autorzy:
Tristão FS; Sorbonne Université UPMC UM75 INSERM U1127, CNRS UMR 7225, Institut du Cerveau et de la Moelle Epinière, Thérapeutique Expérimentale de la neurodégénérescence, Hôpital de la Salpêtrière - Bâtiment ICM, 47 boulevard de l'Hôpital, 75651, Paris, France.; Department of Morphology, Physiology and Basic Pathology (MFPb-Fisiologia), School of Odontology, University of Sao Paulo (USP), Av Café s/n 14040-220, Ribeirão Preto, SP, Brazil.; Center for Interdisciplinary Research on Applied Neurosciences (NAPNA), USP, Ribeirão Preto, Brazil.
Lazzarini M; Department of Molecular Biology of Neuronal Signals, Max Planck Institute of Experimental Medicine, Göttingen, Germany.
Martin S; Department of Molecular Biology of Neuronal Signals, Max Planck Institute of Experimental Medicine, Göttingen, Germany.; Center Nanoscale Microscopy and Molecular Physiology of the Brain (CNMPB), Göttingen, Germany.
Amar M; Sorbonne Université UPMC UM75 INSERM U1127, CNRS UMR 7225, Institut du Cerveau et de la Moelle Epinière, Thérapeutique Expérimentale de la neurodégénérescence, Hôpital de la Salpêtrière - Bâtiment ICM, 47 boulevard de l'Hôpital, 75651, Paris, France.
Stühmer W; Department of Molecular Biology of Neuronal Signals, Max Planck Institute of Experimental Medicine, Göttingen, Germany.; Center Nanoscale Microscopy and Molecular Physiology of the Brain (CNMPB), Göttingen, Germany.
Kirchhoff F; Department of Molecular Physiology, University of Saarland, Homburg, Germany.
Gomes LA; Department of Molecular Biology of Neuronal Signals, Max Planck Institute of Experimental Medicine, Göttingen, Germany.
Lanfumey L; INSERM UMR S894, Université Pierre et Marie Curie, UPMC, Paris, France.
Prediger RD; Departamento de Farmacologia, Centro de Ciências Biológicas, Universidade Federal de Santa Catarina, UFSC, Campus Trindade, Florianópolis, SC, Brazil.
Sepulveda JE; Sorbonne Université UPMC UM75 INSERM U1127, CNRS UMR 7225, Institut du Cerveau et de la Moelle Epinière, Thérapeutique Expérimentale de la neurodégénérescence, Hôpital de la Salpêtrière - Bâtiment ICM, 47 boulevard de l'Hôpital, 75651, Paris, France.
Del-Bel EA; Department of Morphology, Physiology and Basic Pathology (MFPb-Fisiologia), School of Odontology, University of Sao Paulo (USP), Av Café s/n 14040-220, Ribeirão Preto, SP, Brazil. .; Center for Interdisciplinary Research on Applied Neurosciences (NAPNA), USP, Ribeirão Preto, Brazil. .
Raisman-Vozari R; Sorbonne Université UPMC UM75 INSERM U1127, CNRS UMR 7225, Institut du Cerveau et de la Moelle Epinière, Thérapeutique Expérimentale de la neurodégénérescence, Hôpital de la Salpêtrière - Bâtiment ICM, 47 boulevard de l'Hôpital, 75651, Paris, France. .
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Źródło:
Neurotoxicity research [Neurotox Res] 2016 Apr; Vol. 29 (3), pp. 364-80. Date of Electronic Publication: 2015 Sep 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Corpus Striatum/*drug effects
Dopaminergic Neurons/*drug effects
Dopaminergic Neurons/*pathology
Oxidopamine/*toxicity
Parkinsonian Disorders/*metabolism
Parkinsonian Disorders/*pathology
Receptors, Chemokine/*metabolism
Substantia Nigra/*drug effects
Administration, Intranasal ; Animals ; Astrocytes/drug effects ; Astrocytes/metabolism ; CX3C Chemokine Receptor 1 ; Corpus Striatum/metabolism ; Corpus Striatum/pathology ; Dopamine/metabolism ; Dopaminergic Neurons/metabolism ; Encephalitis/chemically induced ; Encephalitis/metabolism ; Gliosis/chemically induced ; Gliosis/metabolism ; Injections, Intraperitoneal ; Mice ; Mice, Transgenic ; Microglia/drug effects ; Microglia/metabolism ; Parkinsonian Disorders/chemically induced ; Receptors, Chemokine/genetics ; Substantia Nigra/metabolism ; Substantia Nigra/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Protectin D1 reduces concanavalin A-induced liver injury by inhibiting NF-κB-mediated CX3CL1/CX3CR1 axis and NLR family, pyrin domain containing 3 inflammasome activation.
Autorzy:
Ren J; Department of Digestive System, Yantaishan Hospital, Yantai, Shandong 264000, P.R. China.
Meng S; Department of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin 130041, P.R. China.
Yan B; Department of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin 130041, P.R. China.
Yu J; Department of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin 130041, P.R. China.
Liu J; Department of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin 130041, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2016 Apr; Vol. 13 (4), pp. 3627-38. Date of Electronic Publication: 2016 Mar 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Chemokine CX3CL1/*metabolism
Docosahexaenoic Acids/*pharmacology
NF-kappa B/*metabolism
NLR Family, Pyrin Domain-Containing 3 Protein/*metabolism
Receptors, Chemokine/*metabolism
Signal Transduction/*drug effects
Animals ; CX3C Chemokine Receptor 1 ; Chemical and Drug Induced Liver Injury/etiology ; Chemical and Drug Induced Liver Injury/pathology ; Chemical and Drug Induced Liver Injury/prevention & control ; Chemokine CX3CL1/antagonists & inhibitors ; Concanavalin A/toxicity ; Cytokines/analysis ; Docosahexaenoic Acids/therapeutic use ; Inflammasomes/metabolism ; Liver/cytology ; Liver/metabolism ; Liver/pathology ; Male ; Mice ; Mice, Inbred C57BL ; NLR Family, Pyrin Domain-Containing 3 Protein/genetics ; Receptors, Chemokine/antagonists & inhibitors ; T-Lymphocytes/cytology ; T-Lymphocytes/drug effects ; T-Lymphocytes/metabolism ; Toll-Like Receptor 4/genetics ; Toll-Like Receptor 4/metabolism ; Transaminases/blood
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Respiratory Syncytial Virus Uses CX3CR1 as a Receptor on Primary Human Airway Epithelial Cultures.
Autorzy:
Johnson SM; Center for Vaccines and Immunity, The Research Institute at Nationwide Children's Hospital, Columbus, Ohio, United States of America.; Department of Pediatrics, The Ohio State University College of Medicine, Columbus, Ohio, United States of America.
McNally BA; Center for Vaccines and Immunity, The Research Institute at Nationwide Children's Hospital, Columbus, Ohio, United States of America.; Department of Pediatrics, The Ohio State University College of Medicine, Columbus, Ohio, United States of America.
Ioannidis I; Center for Vaccines and Immunity, The Research Institute at Nationwide Children's Hospital, Columbus, Ohio, United States of America.; Department of Pediatrics, The Ohio State University College of Medicine, Columbus, Ohio, United States of America.
Flano E; Center for Vaccines and Immunity, The Research Institute at Nationwide Children's Hospital, Columbus, Ohio, United States of America.; Department of Pediatrics, The Ohio State University College of Medicine, Columbus, Ohio, United States of America.
Teng MN; Division of Allergy and Immunology, Department of Internal Medicine, Morsani College of Medicine, University of South Florida, Tampa, Florida, United States of America.
Oomens AG; Department of Veterinary Pathobiology, Oklahoma State University, Stillwater, Oklahoma, United States of America.
Walsh EE; School of Medicine and Dentistry, University of Rochester Medical Center, Rochester, New York, United States of America.
Peeples ME; Center for Vaccines and Immunity, The Research Institute at Nationwide Children's Hospital, Columbus, Ohio, United States of America.; Department of Pediatrics, The Ohio State University College of Medicine, Columbus, Ohio, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2015 Dec 11; Vol. 11 (12), pp. e1005318. Date of Electronic Publication: 2015 Dec 11 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Host-Parasite Interactions/*physiology
Receptors, Chemokine/*metabolism
Respiratory Mucosa/*virology
Respiratory Syncytial Virus Infections/*metabolism
Respiratory Syncytial Virus, Human/*metabolism
Animals ; CX3C Chemokine Receptor 1 ; Cell Line ; Cells, Cultured ; GTP-Binding Proteins/metabolism ; Humans ; Immunohistochemistry ; Mice ; Mice, Inbred C57BL ; Transfection ; Viral Proteins/metabolism ; Virus Internalization
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Examination of the Fractalkine and Fractalkine Receptor Expression in Fallopian Adenocarcinoma Reveals Differences When Compared to Ovarian Carcinoma.
Autorzy:
Gurler H; Department of Biopharmaceutical Sciences, University of Illinois at Chicago, 833 South Wood Street, Chicago, IL 60612, USA. .
Macias V; Department of Pathology, University of Illinois at Chicago, 840 South Wood Street, Chicago, IL 60612, USA. .
Kajdacsy-Balla AA; Department of Pathology, University of Illinois at Chicago, 840 South Wood Street, Chicago, IL 60612, USA. .
Barbolina MV; Department of Biopharmaceutical Sciences, University of Illinois at Chicago, 833 South Wood Street, Chicago, IL 60612, USA. .
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Źródło:
Biomolecules [Biomolecules] 2015 Dec 03; Vol. 5 (4), pp. 3438-47. Date of Electronic Publication: 2015 Dec 03.
Typ publikacji:
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adenocarcinoma/*metabolism
Chemokine CX3CL1/*metabolism
Fallopian Tube Neoplasms/*metabolism
Ovarian Neoplasms/*metabolism
Receptors, Chemokine/*metabolism
Adenocarcinoma/genetics ; CX3C Chemokine Receptor 1 ; Chemokine CX3CL1/genetics ; Epithelium/metabolism ; Fallopian Tube Neoplasms/genetics ; Fallopian Tubes/metabolism ; Female ; Gene Expression Regulation, Neoplastic ; Humans ; Ovarian Neoplasms/genetics ; Receptors, Chemokine/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Effect of schistosomiasis on CX3CR1-expressing mononuclear phagocytes in the ileum and mesenteric lymph nodes of the mouse.
Autorzy:
Alpaerts K; Laboratory of Cell Biology and Histology, Department of Veterinary Sciences, University of Antwerp, Antwerp, Belgium.
Buckinx R; Laboratory of Cell Biology and Histology, Department of Veterinary Sciences, University of Antwerp, Antwerp, Belgium.
Cools N; Laboratory of Experimental Hematology, Vaccine and Infectious Disease Institute (Vaxinfectio), Faculty of Medicine and Health Sciences, University of Antwerp, Antwerp University Hospital (UZA), Edegem, Belgium.
Heylen M; Laboratory of Experimental Medicine and Pediatrics, Faculty of Medicine and Health Sciences, University of Antwerp, Antwerp, Belgium.
Nullens S; Laboratory of Experimental Medicine and Pediatrics, Faculty of Medicine and Health Sciences, University of Antwerp, Antwerp, Belgium.
Berneman Z; Laboratory of Experimental Hematology, Vaccine and Infectious Disease Institute (Vaxinfectio), Faculty of Medicine and Health Sciences, University of Antwerp, Antwerp University Hospital (UZA), Edegem, Belgium.; Center for Cell Therapy and Regenerative Medicine, Antwerp University Hospital (UZA), Edegem, Belgium.
De Winter B; Laboratory of Experimental Medicine and Pediatrics, Faculty of Medicine and Health Sciences, University of Antwerp, Antwerp, Belgium.
Adriaensen D; Laboratory of Cell Biology and Histology, Department of Veterinary Sciences, University of Antwerp, Antwerp, Belgium.
Van Nassauw L; Laboratory of Human Anatomy and Embryology, Faculty of Medicine and Health Sciences, University of Antwerp, Antwerp, Belgium.
Timmermans JP; Laboratory of Cell Biology and Histology, Department of Veterinary Sciences, University of Antwerp, Antwerp, Belgium.
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Źródło:
Neurogastroenterology and motility [Neurogastroenterol Motil] 2015 Nov; Vol. 27 (11), pp. 1587-99. Date of Electronic Publication: 2015 Aug 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Dendritic Cells/*immunology
Ileum/*immunology
Macrophages/*immunology
Receptors, Chemokine/*immunology
Schistosomiasis mansoni/*immunology
Animals ; CX3C Chemokine Receptor 1 ; Disease Models, Animal ; Flow Cytometry ; Immunohistochemistry ; Intestinal Mucosa/immunology ; Lymph Nodes/immunology ; Male ; Mesentery/immunology ; Mice ; Mice, Inbred C57BL ; Microscopy, Confocal
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Fractalkine Signaling Regulates the Inflammatory Response in an α-Synuclein Model of Parkinson Disease.
Autorzy:
Thome AD; Center for Neurodegeneration and Experimental Therapeutics, Department of Neurology, The University of Alabama at Birmingham, Birmingham, Alabama, United States of America.
Standaert DG; Center for Neurodegeneration and Experimental Therapeutics, Department of Neurology, The University of Alabama at Birmingham, Birmingham, Alabama, United States of America.
Harms AS; Center for Neurodegeneration and Experimental Therapeutics, Department of Neurology, The University of Alabama at Birmingham, Birmingham, Alabama, United States of America.
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Źródło:
PloS one [PLoS One] 2015 Oct 15; Vol. 10 (10), pp. e0140566. Date of Electronic Publication: 2015 Oct 15 (Print Publication: 2015).
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CX3CL1/*metabolism
Parkinson Disease/*immunology
Receptors, Chemokine/*genetics
Receptors, Chemokine/*metabolism
alpha-Synuclein/*metabolism
Animals ; CX3C Chemokine Receptor 1 ; Dependovirus/genetics ; Disease Models, Animal ; Dopaminergic Neurons/metabolism ; Gene Knockout Techniques ; Genetic Vectors ; Humans ; Mice ; Microglia/drug effects ; Parkinson Disease/genetics ; Phagocytosis ; alpha-Synuclein/genetics ; alpha-Synuclein/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Expression pattern of Ccr2 and Cx3cr1 in inherited retinal degeneration.
Autorzy:
Kohno H; Department of Ophthalmology, The Jikei University School of Medicine, 105-8461, Tokyo, Japan. .; Tokyu Hospital, 145-0062, Tokyo, Japan. .
Koso H; Division of Molecular and Developmental Biology, The Institute of Medical Science, The University of Tokyo, 108-8639, Tokyo, Japan.
Okano K; Department of Ophthalmology, The Jikei University School of Medicine, 105-8461, Tokyo, Japan.
Sundermeier TR; Department of Pharmacology, School of Medicine, Case Western Reserve University, Cleveland, OH, USA.
Saito S; Department of Molecular Immunology, The Jikei University School of Medicine, 105-8461, Tokyo, Japan.
Watanabe S; Division of Molecular and Developmental Biology, The Institute of Medical Science, The University of Tokyo, 108-8639, Tokyo, Japan.
Tsuneoka H; Department of Ophthalmology, The Jikei University School of Medicine, 105-8461, Tokyo, Japan.
Sakai T; Department of Ophthalmology, The Jikei University School of Medicine, 105-8461, Tokyo, Japan. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2015 Oct 12; Vol. 12, pp. 188. Date of Electronic Publication: 2015 Oct 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation/*genetics
Receptors, CCR2/*metabolism
Receptors, Chemokine/*metabolism
Retinal Degeneration/*genetics
Retinal Degeneration/*metabolism
Animals ; CX3C Chemokine Receptor 1 ; Cell Movement/genetics ; Disease Models, Animal ; Female ; Leukocytes/metabolism ; Leukocytes/pathology ; Luminescent Proteins/genetics ; Luminescent Proteins/metabolism ; Macrophages/metabolism ; Macrophages/pathology ; Male ; Mice ; Mice, Transgenic ; Microglia/metabolism ; Microglia/pathology ; Mutation/genetics ; Neurons/metabolism ; Neurons/pathology ; Proto-Oncogene Proteins/deficiency ; Proto-Oncogene Proteins/genetics ; Receptor Protein-Tyrosine Kinases/deficiency ; Receptor Protein-Tyrosine Kinases/genetics ; Receptors, CCR2/genetics ; Receptors, Chemokine/genetics ; Retina/metabolism ; Retina/pathology ; Retinal Pigment Epithelium/metabolism ; Retinal Pigment Epithelium/pathology ; Time Factors ; c-Mer Tyrosine Kinase
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Metabolic Effects of CX3CR1 Deficiency in Diet-Induced Obese Mice.
Autorzy:
Shah R; Division of Endocrinology and Diabetes, The Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, United States of America.
O'Neill SM; Cardiovascular Institute, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Hinkle C; Cardiovascular Institute, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Caughey J; Cardiovascular Institute, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Stephan S; Cardiovascular Institute, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Lynch E; Cardiovascular Institute, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Bermingham K; Cardiovascular Institute, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Lynch G; Cardiovascular Institute, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Ahima RS; Division of Endocrinology, Diabetes and Metabolism, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Reilly MP; Cardiovascular Institute, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
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Źródło:
PloS one [PLoS One] 2015 Sep 22; Vol. 10 (9), pp. e0138317. Date of Electronic Publication: 2015 Sep 22 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Diet, High-Fat*
Receptors, Chemokine/*genetics
Adipokines/metabolism ; Adipose Tissue/cytology ; Animals ; CX3C Chemokine Receptor 1 ; Chemokine CCL2/blood ; Chemokine CX3CL1/blood ; Diabetes Mellitus, Type 2/etiology ; Energy Metabolism ; Glucose Intolerance ; Glycogen Synthase Kinase 3/genetics ; Glycogen Synthase Kinase 3/metabolism ; Glycogen Synthase Kinase 3 beta ; Insulin/metabolism ; Insulin Resistance ; Interleukin-6/blood ; Liver/metabolism ; Macrophages/cytology ; Macrophages/immunology ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; Receptors, Chemokine/deficiency
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Time-dependent effects of CX3CR1 in a mouse model of mild traumatic brain injury.
Autorzy:
Febinger HY; Department of Anesthesiology and Pain Medicine, University of Washington, BOX # 359724, Seattle, WA, 98001, USA.; Present address: Interdepartmental Program in Neuroscience, University of Utah School of Medicine, Salt Lake City, Utah, USA.
Thomasy HE; Department of Anesthesiology and Pain Medicine, University of Washington, BOX # 359724, Seattle, WA, 98001, USA.; Neuroscience Graduate Program, University of Washington, Seattle, WA, 98104, USA.
Pavlova MN; Department of Anesthesiology and Pain Medicine, University of Washington, BOX # 359724, Seattle, WA, 98001, USA.
Ringgold KM; Department of Anesthesiology and Pain Medicine, University of Washington, BOX # 359724, Seattle, WA, 98001, USA.
Barf PR; Department of Anesthesiology and Pain Medicine, University of Washington, BOX # 359724, Seattle, WA, 98001, USA.
George AM; Department of Anesthesiology and Pain Medicine, University of Washington, BOX # 359724, Seattle, WA, 98001, USA.
Grillo JN; Department of Anesthesiology and Pain Medicine, University of Washington, BOX # 359724, Seattle, WA, 98001, USA.; Department of Biology, University of Washington, Seattle, WA, 98104, USA.
Bachstetter AD; Sanders-Brown Center on Aging, University of Kentucky, Lexington, KY, 40536, USA.
Garcia JA; Department of Biology and South Texas Center for Emerging Infectious Diseases, University of Texas at San Antonio, San Antonio, TX, 78249, USA.
Cardona AE; Department of Biology and South Texas Center for Emerging Infectious Diseases, University of Texas at San Antonio, San Antonio, TX, 78249, USA.
Opp MR; Department of Anesthesiology and Pain Medicine, University of Washington, BOX # 359724, Seattle, WA, 98001, USA.
Gemma C; Department of Anesthesiology and Pain Medicine, University of Washington, BOX # 359724, Seattle, WA, 98001, USA. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2015 Sep 02; Vol. 12, pp. 154. Date of Electronic Publication: 2015 Sep 02.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Brain Injuries*/complications
Brain Injuries*/metabolism
Brain Injuries*/pathology
Brain/*pathology
Receptors, Chemokine/*metabolism
Analysis of Variance ; Animals ; CX3C Chemokine Receptor 1 ; Disease Models, Animal ; Exploratory Behavior/physiology ; Flow Cytometry ; Fluoresceins/metabolism ; Green Fluorescent Proteins/genetics ; Green Fluorescent Proteins/metabolism ; Leukocytes, Mononuclear/pathology ; Macrophage Activation/genetics ; Macrophage Activation/physiology ; Male ; Maze Learning/physiology ; Mice ; Mice, Transgenic ; Neurons/pathology ; Psychomotor Disorders/etiology ; Receptors, Chemokine/genetics ; Rotarod Performance Test ; Time Factors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Fractalkine in the nervous system: neuroprotective or neurotoxic molecule?
Autorzy:
Lauro C; Department of Physiology and Pharmacology, Istituto Pasteur Fondazione Cenci Bolognetti, Sapienza University of Rome, Rome, Italy.
Catalano M; Department of Physiology and Pharmacology, Istituto Pasteur Fondazione Cenci Bolognetti, Sapienza University of Rome, Rome, Italy.; IRCCS NeuroMed, Pozzilli, Italy.
Trettel F; Department of Physiology and Pharmacology, Istituto Pasteur Fondazione Cenci Bolognetti, Sapienza University of Rome, Rome, Italy.
Limatola C; Department of Physiology and Pharmacology, Istituto Pasteur Fondazione Cenci Bolognetti, Sapienza University of Rome, Rome, Italy.; IRCCS NeuroMed, Pozzilli, Italy.
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Źródło:
Annals of the New York Academy of Sciences [Ann N Y Acad Sci] 2015 Sep; Vol. 1351, pp. 141-8. Date of Electronic Publication: 2015 Jun 17.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Brain/*immunology
Brain Diseases/*physiopathology
Chemokine CX3CL1/*metabolism
Neuroprotective Agents/*metabolism
Receptors, Chemokine/*metabolism
Animals ; Anti-Inflammatory Agents/metabolism ; Brain Diseases/immunology ; CX3C Chemokine Receptor 1 ; Chemokine CX3CL1/biosynthesis ; Disease Models, Animal ; Humans ; Inflammation/immunology ; Microglia/metabolism ; Receptors, Chemokine/biosynthesis ; Signal Transduction/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CX3CR1 Is Expressed in Differentiated Human Ciliated Airway Cells and Co-Localizes with Respiratory Syncytial Virus on Cilia in a G Protein-Dependent Manner.
Autorzy:
Jeong KI; Sanofi Pasteur, Research North America, 38 Sidney St., Cambridge, MA 02139, United States of America.
Piepenhagen PA; Genzyme, Department of Pathology, 5 The Mountain Rd., Framingham, MA 01701, United States of America.
Kishko M; Sanofi Pasteur, Research North America, 38 Sidney St., Cambridge, MA 02139, United States of America.
DiNapoli JM; Sanofi Pasteur, Research North America, 38 Sidney St., Cambridge, MA 02139, United States of America.
Groppo RP; Sanofi Pasteur, Research North America, 38 Sidney St., Cambridge, MA 02139, United States of America.
Zhang L; Sanofi Pasteur, Research North America, 38 Sidney St., Cambridge, MA 02139, United States of America.
Almond J; Sanofi Pasteur, Research North America, 38 Sidney St., Cambridge, MA 02139, United States of America.
Kleanthous H; Sanofi Pasteur, Research North America, 38 Sidney St., Cambridge, MA 02139, United States of America.
Delagrave S; Sanofi Pasteur, Research North America, 38 Sidney St., Cambridge, MA 02139, United States of America.
Parrington M; Sanofi Pasteur, Research North America, 38 Sidney St., Cambridge, MA 02139, United States of America.
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Źródło:
PloS one [PLoS One] 2015 Jun 24; Vol. 10 (6), pp. e0130517. Date of Electronic Publication: 2015 Jun 24 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epithelial Cells/*metabolism
Receptors, Chemokine/*genetics
Respiratory Mucosa/*metabolism
Respiratory Syncytial Virus, Human/*genetics
Viral Envelope Proteins/*genetics
Viral Fusion Proteins/*genetics
Antibodies/pharmacology ; Base Sequence ; Binding Sites ; CX3C Chemokine Receptor 1 ; Cell Differentiation ; Child ; Cilia/metabolism ; Cilia/pathology ; Cilia/virology ; Epithelial Cells/pathology ; Epithelial Cells/virology ; Epitopes/chemistry ; Epitopes/immunology ; Gene Expression ; Humans ; Molecular Sequence Data ; Primary Cell Culture ; Protein Binding ; Receptors, Chemokine/antagonists & inhibitors ; Receptors, Chemokine/chemistry ; Receptors, Chemokine/metabolism ; Respiratory Mucosa/pathology ; Respiratory Mucosa/virology ; Respiratory Syncytial Virus, Human/metabolism ; Sequence Deletion ; Viral Envelope Proteins/antagonists & inhibitors ; Viral Envelope Proteins/chemistry ; Viral Envelope Proteins/metabolism ; Viral Fusion Proteins/chemistry ; Viral Fusion Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Neuronal Cx3cr1 Deficiency Protects against Amyloid β-Induced Neurotoxicity.
Autorzy:
Dworzak J; Laboratory of Molecular Immunology, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America; Neuronal Oscillations Group, Department of Physiology, Development, and Neuroscience, University of Cambridge, Cambridge, United Kingdom; Cell Biology Section, Neurogenetics Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, United States of America.
Renvoisé B; Cell Biology Section, Neurogenetics Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, United States of America.
Habchi J; Department of Chemistry, University of Cambridge, Cambridge, United Kingdom.
Yates EV; Department of Chemistry, University of Cambridge, Cambridge, United Kingdom.
Combadière C; Centre d'Immunologie et des Maladies Infectieuses-Paris, Institut National de la Santé et de la Recherche Médicale, Paris, France.
Knowles TP; Department of Chemistry, University of Cambridge, Cambridge, United Kingdom.
Dobson CM; Department of Chemistry, University of Cambridge, Cambridge, United Kingdom.
Blackstone C; Cell Biology Section, Neurogenetics Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, United States of America.
Paulsen O; Neuronal Oscillations Group, Department of Physiology, Development, and Neuroscience, University of Cambridge, Cambridge, United Kingdom.
Murphy PM; Laboratory of Molecular Immunology, National Institute of Allergy and Infectious Disease, National Institutes of Health, Bethesda, Maryland, United States of America.
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Źródło:
PloS one [PLoS One] 2015 Jun 03; Vol. 10 (6), pp. e0127730. Date of Electronic Publication: 2015 Jun 03 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Synaptic Transmission*
Alzheimer Disease/*metabolism
Amyloid beta-Peptides/*metabolism
Neurons/*metabolism
Protein Aggregation, Pathological/*metabolism
Receptors, Chemokine/*deficiency
Alzheimer Disease/genetics ; Alzheimer Disease/pathology ; Amyloid beta-Peptides/genetics ; Animals ; CX3C Chemokine Receptor 1 ; Disease Models, Animal ; Mice ; Mice, Knockout ; Neurons/pathology ; Protein Aggregation, Pathological/genetics ; Protein Aggregation, Pathological/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Transcriptome analysis in patients with chronic kidney disease on hemodialysis disclosing a key role for CD16+CX3CR1+ monocytes.
Autorzy:
Schepers E; Department of Internal Medicine, Nephrology Division, Ghent University Hospital, Ghent, Belgium.
Houthuys E; Unit for Medical Biotechnology, Inflammation Research Center (IRC), VIB and Laboratory for Protein Biochemistry and Biomolecular Engineering, Department of Biochemistry and Microbiology, Ghent University, Ghent, Belgium.
Dhondt A; Department of Internal Medicine, Nephrology Division, Ghent University Hospital, Ghent, Belgium.
De Meyer G; Department of Internal Medicine, Cardiology Division, Ghent University Hospital, Ghent, Belgium.
Neirynck N; Department of Internal Medicine, Cardiology Division, Ghent University Hospital, Ghent, Belgium.
Bernaert P; Renal Division, AZ Maria Middelares, Ghent, Belgium.
Van den Bergh R; Department of Molecular and Cellular Interactions, VIB-Vrije Universiteit Brussel, Pleinlaan 2, B-1050 Brussels, Belgium; Laboratory of Cellular and Molecular Immunology, Vrije Universiteit Brussel, Pleinlaan 2, B-1050 Brussels, Belgium.
Brouckaert P; Department of Biomedical and Molecular Biology, Ghent University, Zwijnaarde, Belgium.
Vanholder R; Department of Internal Medicine, Nephrology Division, Ghent University Hospital, Ghent, Belgium.
Glorieux G; Department of Internal Medicine, Nephrology Division, Ghent University Hospital, Ghent, Belgium.
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Źródło:
PloS one [PLoS One] 2015 Apr 01; Vol. 10 (4), pp. e0121750. Date of Electronic Publication: 2015 Apr 01 (Print Publication: 2015).
Typ publikacji:
Journal Article
MeSH Terms:
Transcriptome*
Monocytes/*metabolism
Receptors, Chemokine/*metabolism
Receptors, IgG/*metabolism
Renal Insufficiency, Chronic/*metabolism
Aged ; CX3C Chemokine Receptor 1 ; Cardiovascular Diseases/genetics ; Cardiovascular Diseases/metabolism ; Case-Control Studies ; Cells, Cultured ; Female ; GPI-Linked Proteins/genetics ; GPI-Linked Proteins/metabolism ; Gene Expression Profiling ; Humans ; Male ; Middle Aged ; Receptors, Chemokine/genetics ; Receptors, IgG/genetics ; Renal Dialysis ; Renal Insufficiency, Chronic/genetics ; Renal Insufficiency, Chronic/therapy
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Fibrillar amyloid plaque formation precedes microglial activation.
Autorzy:
Jung CK; Department for Translational Brain Research, German Center for Neurodegenerative Diseases-site Munich (DZNE-M) and Center for Neuropathology and Prion Research (ZNP), Ludwig-Maximilians-University Munich, Munich, Germany.
Keppler K; Department for Translational Brain Research, German Center for Neurodegenerative Diseases-site Munich (DZNE-M) and Center for Neuropathology and Prion Research (ZNP), Ludwig-Maximilians-University Munich, Munich, Germany.
Steinbach S; Department for Translational Brain Research, German Center for Neurodegenerative Diseases-site Munich (DZNE-M) and Center for Neuropathology and Prion Research (ZNP), Ludwig-Maximilians-University Munich, Munich, Germany.
Blazquez-Llorca L; Department for Translational Brain Research, German Center for Neurodegenerative Diseases-site Munich (DZNE-M) and Center for Neuropathology and Prion Research (ZNP), Ludwig-Maximilians-University Munich, Munich, Germany.
Herms J; Department for Translational Brain Research, German Center for Neurodegenerative Diseases-site Munich (DZNE-M) and Center for Neuropathology and Prion Research (ZNP), Ludwig-Maximilians-University Munich, Munich, Germany; Munich Cluster of Systems Neurology (SyNergy), Munich, Germany.
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Źródło:
PloS one [PLoS One] 2015 Mar 23; Vol. 10 (3), pp. e0119768. Date of Electronic Publication: 2015 Mar 23 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Disease Models, Animal*
Alzheimer Disease/*complications
Amyloid beta-Protein Precursor/*physiology
Microglia/*pathology
Plaque, Amyloid/*pathology
Presenilin-1/*physiology
Receptors, Chemokine/*physiology
Animals ; Brain/metabolism ; Brain/pathology ; CX3C Chemokine Receptor 1 ; Cells, Cultured ; Female ; Humans ; Immunoenzyme Techniques ; Male ; Mice ; Mice, Transgenic ; Microglia/metabolism ; Plaque, Amyloid/etiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Involvement of CX3CL1/CX3CR1 signaling in spinal long term potentiation.
Autorzy:
Bian C; Institute of Neurobiology, Institutes of Brain Science and State Key Laboratory of Medical Neurobiology, Collaborative Innovation Center for Brain Science, Fudan University, Shanghai, 200032, China.
Zhao ZQ; Institute of Neurobiology, Institutes of Brain Science and State Key Laboratory of Medical Neurobiology, Collaborative Innovation Center for Brain Science, Fudan University, Shanghai, 200032, China.
Zhang YQ; Institute of Neurobiology, Institutes of Brain Science and State Key Laboratory of Medical Neurobiology, Collaborative Innovation Center for Brain Science, Fudan University, Shanghai, 200032, China.
Lü N; Institute of Neurobiology, Institutes of Brain Science and State Key Laboratory of Medical Neurobiology, Collaborative Innovation Center for Brain Science, Fudan University, Shanghai, 200032, China.
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Źródło:
PloS one [PLoS One] 2015 Mar 13; Vol. 10 (3), pp. e0118842. Date of Electronic Publication: 2015 Mar 13 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Long-Term Potentiation*
Signal Transduction*
Chemokine CX3CL1/*metabolism
Receptors, Chemokine/*metabolism
Spinal Cord/*cytology
Spinal Cord/*physiology
Animals ; CX3C Chemokine Receptor 1 ; Gene Expression Regulation ; Gene Knockout Techniques ; Interleukin-18/metabolism ; Interleukin-23/metabolism ; Male ; Mice ; Rats ; Receptors, Chemokine/deficiency ; Receptors, Chemokine/genetics ; Spinal Cord Dorsal Horn/cytology ; Spinal Cord Dorsal Horn/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
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