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Wyświetlanie 21-40 z 26729
Tytuł:
Connexin 43 participates in atrial electrical remodelling through colocalization with calcium channels in atrial myocytes.
Autorzy:
Peng DW; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Lai YY; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Luo XS; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Li X; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Deng CY; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Guo HM; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.
Zhao JF; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.
Yang H; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Liu Y; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Wang ZY; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Xu YW; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Kuang SJ; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Wu SL; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Xue YM; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
Rao F; Guangdong Cardiovascular Institute, Guangdong Provincial People's Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China.; Guangdong Provincial Key Laboratory of Clinical Pharmacology, Research Center of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.
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Źródło:
Clinical and experimental pharmacology & physiology [Clin Exp Pharmacol Physiol] 2022 Jan; Vol. 49 (1), pp. 25-34. Date of Electronic Publication: 2021 Oct 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Atrial Remodeling*/physiology
Calcium Channels/*physiology
Connexin 43/*physiology
Heart Atria/*metabolism
Myocytes, Cardiac/*metabolism
Animals ; Atrial Fibrillation/metabolism ; Blotting, Western ; Calcium Channels/metabolism ; Calcium Channels, L-Type/metabolism ; Calcium Channels, L-Type/physiology ; Cell Line ; Cells, Cultured ; Connexin 43/metabolism ; Heart Atria/drug effects ; Heart Atria/physiopathology ; Humans ; Mibefradil/pharmacology ; Mice ; Mice, Inbred BALB C ; Microscopy, Confocal ; Myocytes, Cardiac/drug effects ; Myocytes, Cardiac/physiology ; Nifedipine/pharmacology ; Patch-Clamp Techniques
Czasopismo naukowe
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Tytuł:
Dilation of Pregnant Rat Uterine Arteries with Phenols from Extra Virgin Olive Oil Is Endothelium-Dependent and Involves Calcium and Potassium Channels.
Autorzy:
Esposito, Milena (AUTHOR)
Gatto, Mariacarmela (AUTHOR)
Cipolla, Marilyn J. (AUTHOR)
Bernstein, Ira M. (AUTHOR)
Mandalà, Maurizio (AUTHOR)
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Źródło:
Cells (2073-4409). Apr2024, Vol. 13 Issue 7, p619. 12p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Multivariate Approaches in Quantitative Structure–Property Relationships Study for the Photostability Assessment of 1,4-Dihydropyridine Derivatives.
Autorzy:
Chieffallo, Martina (AUTHOR)
De Luca, Michele (AUTHOR)
Grande, Fedora (AUTHOR)
Occhiuzzi, Maria Antonietta (AUTHOR)
Gündüz, Miyase Gözde (AUTHOR)
Garofalo, Antonio (AUTHOR)
Ioele, Giuseppina (AUTHOR)
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Źródło:
Pharmaceutics. Feb2024, Vol. 16 Issue 2, p206. 23p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A Cataract-Causing Mutation in the TRPM3 Cation Channel Disrupts Calcium Dynamics in the Lens.
Autorzy:
Zhou, Yuefang (AUTHOR)
Bennett, Thomas M. (AUTHOR)
Ruzycki, Philip A. (AUTHOR)
Guo, Zhaohua (AUTHOR)
Cao, Yu-Qing (AUTHOR)
Shahidullah, Mohammad (AUTHOR)
Delamere, Nicholas A. (AUTHOR)
Shiels, Alan (AUTHOR)
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Źródło:
Cells (2073-4409). Feb2024, Vol. 13 Issue 3, p257. 20p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Role of Orai3-Mediated Store-Operated Calcium Entry in Radiation-Induced Brain Microvascular Endothelial Cell Injury.
Autorzy:
Wu Q; Department of Radiotherapy, The First Affiliated Hospital of Anhui Medical University, Hefei 230032, China.
Fang Y; School of Basic Medical Sciences, Anhui Medical University, Hefei 230032, China.
Huang X; Department of Radiotherapy, The First Affiliated Hospital of Anhui Medical University, Hefei 230032, China.
Zheng F; School of Basic Medical Sciences, Anhui Medical University, Hefei 230032, China.
Ma S; School of Basic Medical Sciences, Anhui Medical University, Hefei 230032, China.
Zhang X; Department of Radiotherapy, The First Affiliated Hospital of Anhui Medical University, Hefei 230032, China.
Han T; Department of Radiotherapy, The First Affiliated Hospital of Anhui Medical University, Hefei 230032, China.
Gao H; School of Basic Medical Sciences, Anhui Medical University, Hefei 230032, China.
Shen B; School of Basic Medical Sciences, Anhui Medical University, Hefei 230032, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Apr 06; Vol. 24 (7). Date of Electronic Publication: 2023 Apr 06.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Injuries*
Calcium Channels*/genetics
Calcium Channels*/metabolism
Endothelial Cells*/metabolism
Animals ; Rats ; Brain/metabolism ; Calcium/metabolism ; Calcium Signaling ; Rats, Sprague-Dawley
Czasopismo naukowe
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Tytuł:
Regulation of Cardiac Cav1.2 Channels by Calmodulin.
Autorzy:
Kameyama M; Department of Physiology, Graduate School of Medical & Dental Sciences, Kagoshima University, Sakura-ga-oka, Kagoshima 890-8544, Japan.
Minobe E; Department of Physiology, Graduate School of Medical & Dental Sciences, Kagoshima University, Sakura-ga-oka, Kagoshima 890-8544, Japan.
Shao D; Department of Pharmaceutical Toxicology, School of Pharmacy, China Medical University, Shenyang 110012, China.
Xu J; Department of Physiology, Graduate School of Medical & Dental Sciences, Kagoshima University, Sakura-ga-oka, Kagoshima 890-8544, Japan.
Gao Q; Department of Pharmaceutical Toxicology, School of Pharmacy, China Medical University, Shenyang 110012, China.
Hao L; Department of Pharmaceutical Toxicology, School of Pharmacy, China Medical University, Shenyang 110012, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Mar 29; Vol. 24 (7). Date of Electronic Publication: 2023 Mar 29.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Calmodulin*/metabolism
Calcium Channels, L-Type*/genetics
Calcium Channels, L-Type*/metabolism
Myocytes, Cardiac/metabolism ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/metabolism ; Calcium/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Age-related deficits in neuronal physiology and cognitive function are recapitulated in young mice overexpressing the L-type calcium channel, Ca V 1.3.
Autorzy:
Moore SJ; Michigan Neuroscience Institute, Ann Arbor, Michigan, USA.; Molecular & Integrative Physiology, University of Michigan, Ann Arbor, Michigan, USA.
Cazares VA; Molecular & Integrative Physiology, University of Michigan, Ann Arbor, Michigan, USA.; Department of Psychology, Williams College, Williamstown, Massachusetts, USA.
Temme SJ; Michigan Neuroscience Institute, Ann Arbor, Michigan, USA.
Murphy GG; Michigan Neuroscience Institute, Ann Arbor, Michigan, USA.; Molecular & Integrative Physiology, University of Michigan, Ann Arbor, Michigan, USA.
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Źródło:
Aging cell [Aging Cell] 2023 Mar; Vol. 22 (3), pp. e13781. Date of Electronic Publication: 2023 Jan 26.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
MeSH Terms:
Calcium Channels, L-Type*/genetics
Calcium Channels, L-Type*/metabolism
Calcium*/metabolism
Mice ; Animals ; Cognition/physiology ; Learning ; Mice, Transgenic ; Maze Learning ; Mice, Inbred C57BL
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Chlorogenic Acid Decreases Glutamate Release from Rat Cortical Nerve Terminals by P/Q-Type Ca Channel Suppression: A Possible Neuroprotective Mechanism.
Autorzy:
Hung YC; Chi Mei Medical Center, Department of Neurosurgery, Tainan 71004, Taiwan.
Kuo YH; Graduate Institute of Biomedical and Pharmaceutical Science, Fu Jen Catholic University, New Taipei City 24205, Taiwan.
Hsieh PW; Research Center for Chinese Herbal Medicine, College of Human Ecology, Chang Gung University of Science and Technology, Taoyuan 33303, Taiwan.; Graduate Institute of Natural Products, School of Traditional Chinese Medicine, Graduate Institute of Biomedical Sciences, College of Medicine, Chang Gung University, Taoyuan 33303, Taiwan.
Hsieh TY; Program in Nutrition & Food Science, Fu Jen Catholic University, New Taipei City 24205, Taiwan.
Kuo JR; Chi Mei Medical Center, Department of Neurosurgery, Tainan 71004, Taiwan.
Wang SJ; Research Center for Chinese Herbal Medicine, College of Human Ecology, Chang Gung University of Science and Technology, Taoyuan 33303, Taiwan.; School of Medicine, Fu Jen Catholic University, New Taipei City 24205, Taiwan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Oct 23; Vol. 22 (21). Date of Electronic Publication: 2021 Oct 23.
Typ publikacji:
Journal Article
MeSH Terms:
Calcium Channels/*metabolism
Chlorogenic Acid/*pharmacology
Glutamic Acid/*metabolism
Animals ; Calcium/metabolism ; Calcium Channels/drug effects ; Calcium Channels, P-Type/metabolism ; Calcium Channels, Q-Type/metabolism ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/metabolism ; Calmodulin/metabolism ; Cerebral Cortex/metabolism ; Cerebral Cortex/physiology ; Chlorogenic Acid/metabolism ; Excitatory Amino Acid Agents ; Glutamic Acid/drug effects ; Kainic Acid/metabolism ; Male ; Membrane Potentials/drug effects ; Molecular Docking Simulation ; Neurons/drug effects ; Neurons/metabolism ; Neuroprotection/drug effects ; Neuroprotective Agents/pharmacology ; Rats ; Rats, Sprague-Dawley ; Synapses/metabolism ; Synaptic Vesicles/metabolism ; Synaptosomes/metabolism
Czasopismo naukowe
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Tytuł:
Rab11-dependent recycling of calcium channels is mediated by auxiliary subunit α 2 δ-1 but not α 2 δ-3.
Autorzy:
Meyer JO; Department of Neuroscience, Physiology and Pharmacology, University College London, Gower Street, London, WC1E 6BT, UK.
Dolphin AC; Department of Neuroscience, Physiology and Pharmacology, University College London, Gower Street, London, WC1E 6BT, UK. .
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Źródło:
Scientific reports [Sci Rep] 2021 May 13; Vol. 11 (1), pp. 10256. Date of Electronic Publication: 2021 May 13.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Calcium Channels/*metabolism
Calcium Channels, L-Type/*metabolism
rab GTP-Binding Proteins/*metabolism
Animals ; Brain/metabolism ; Calcium/metabolism ; Calcium Channels/genetics ; Calcium Channels/physiology ; Calcium Channels, L-Type/genetics ; Calcium Channels, N-Type/genetics ; Calcium Channels, N-Type/metabolism ; Cell Line ; Cell Membrane/metabolism ; Cyclohexanecarboxylic Acids/metabolism ; Gabapentin/metabolism ; Hippocampus/metabolism ; Neurites/metabolism ; Neurons/metabolism ; Presynaptic Terminals/metabolism ; Primary Cell Culture ; Protein Transport ; Rats ; gamma-Aminobutyric Acid/metabolism ; rab GTP-Binding Proteins/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Functional implications of Ca v 2.3 R-type voltage-gated calcium channels in the murine auditory system - novel vistas from brainstem-evoked response audiometry.
Autorzy:
Lundt A; Experimental Neuropsychopharmacology, Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
Soós J; Experimental Neuropsychopharmacology, Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
Seidel R; Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
Henseler C; Experimental Neuropsychopharmacology, Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
Müller R; Cognitive Neurophysiology, Department of Psychiatry and Psychotherapy and University Hospital Cologne, Faculty of Medicine, University of Cologne, Cologne, Germany.
Raj Ginde V; Experimental Neuropsychopharmacology, Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
Imran Arshaad M; Experimental Neuropsychopharmacology, Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
Ehninger D; Molecular and Cellular Cognition, German Center for Neurodegenerative Diseases, (Deutsches Zentrum für Neurodegenerative Erkrankungen, DZNE), Bonn, Germany.
Hescheler J; Institute of Neurophysiology, Faculty of Medicine, University of Cologne, Cologne, Germany.
Sachinidis A; Institute of Neurophysiology, Faculty of Medicine, University of Cologne, Cologne, Germany.
Broich K; Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
Wormuth C; Experimental Neuropsychopharmacology, Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
Papazoglou A; Experimental Neuropsychopharmacology, Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
Weiergräber M; Experimental Neuropsychopharmacology, Federal Institute for Drugs and Medical Devices (Bundesinstitut für Arzneimittel und Medizinprodukte, BfArM), Bonn, Germany.
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Źródło:
The European journal of neuroscience [Eur J Neurosci] 2020 Apr; Vol. 51 (7), pp. 1583-1604. Date of Electronic Publication: 2019 Nov 03.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Audiometry, Evoked Response*
Auditory Threshold*
Calcium Channels, N-Type*
Evoked Potentials, Auditory, Brain Stem*
Acoustic Stimulation ; Animals ; Brain Stem ; Calcium Channels ; Female ; Male ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Whole transcriptome sequencing analyses of islets reveal ncRNA regulatory networks underlying impaired insulin secretion and increased β-cell mass in high fat diet-induced diabetes mellitus.
Autorzy:
Ma J; Department of Endocrinology and Metabolism, West China Hospital, Sichuan University, Chengdu, China.; Center for Diabetes and Metabolism Research, West China Hospital, Sichuan University, Chengdu, China.
Gao R; Oxford Centre for Diabetes, Endocrinology and Metabolism, Radcliffe Department of Medicine, University of Oxford, Oxford, United Kingdom.
Xie Q; Department of Endocrinology and Metabolism, West China Hospital, Sichuan University, Chengdu, China.; Center for Diabetes and Metabolism Research, West China Hospital, Sichuan University, Chengdu, China.
Pan X; Department of Endocrinology and Metabolism, West China Hospital, Sichuan University, Chengdu, China.; Center for Diabetes and Metabolism Research, West China Hospital, Sichuan University, Chengdu, China.
Tong N; Department of Endocrinology and Metabolism, West China Hospital, Sichuan University, Chengdu, China.; Center for Diabetes and Metabolism Research, West China Hospital, Sichuan University, Chengdu, China.
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Źródło:
PloS one [PLoS One] 2024 Apr 01; Vol. 19 (4), pp. e0300965. Date of Electronic Publication: 2024 Apr 01 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
RNA, Long Noncoding*/genetics
RNA, Long Noncoding*/metabolism
MicroRNAs*/genetics
MicroRNAs*/metabolism
Diabetes Mellitus*
Mice ; Animals ; Diet, High-Fat/adverse effects ; RNA, Circular/metabolism ; Insulin Secretion ; Exome Sequencing ; Mice, Inbred C57BL ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Obesity/genetics ; Gene Regulatory Networks ; Calcium Channels, N-Type/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Polycystin-2 Mediated Calcium Signalling in the Dictyostelium Model for Autosomal Dominant Polycystic Kidney Disease.
Autorzy:
Allan CY; Department of Microbiology, Anatomy, Physiology and Pharmacology, La Trobe University, Bundoora, Melbourne, VIC 3086, Australia.
Sanislav O; Department of Microbiology, Anatomy, Physiology and Pharmacology, La Trobe University, Bundoora, Melbourne, VIC 3086, Australia.
Fisher PR; Department of Microbiology, Anatomy, Physiology and Pharmacology, La Trobe University, Bundoora, Melbourne, VIC 3086, Australia.
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Źródło:
Cells [Cells] 2024 Mar 31; Vol. 13 (7). Date of Electronic Publication: 2024 Mar 31.
Typ publikacji:
Journal Article
MeSH Terms:
Polycystic Kidney, Autosomal Dominant*/genetics
Dictyostelium*/metabolism
Humans ; TRPP Cation Channels/genetics ; Calcium/metabolism ; Calcium Signaling/physiology ; Calcium Channels/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MCU-independent Ca overload and necrotic cell death in a mouse model of Duchenne muscular dystrophy.
Autorzy:
Bround MJ; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, 240 Albert Sabin Way, MLC 7020, Cincinnati, OH, 45229-3039, USA.
Abay E; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, 240 Albert Sabin Way, MLC 7020, Cincinnati, OH, 45229-3039, USA.
Huo J; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, 240 Albert Sabin Way, MLC 7020, Cincinnati, OH, 45229-3039, USA.
Havens JR; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, 240 Albert Sabin Way, MLC 7020, Cincinnati, OH, 45229-3039, USA.
York AJ; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, 240 Albert Sabin Way, MLC 7020, Cincinnati, OH, 45229-3039, USA.
Bers DM; Department of Pharmacology, University of California, Davis, CA, 95616, USA.
Molkentin JD; Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati, 240 Albert Sabin Way, MLC 7020, Cincinnati, OH, 45229-3039, USA. .
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Źródło:
Scientific reports [Sci Rep] 2024 Mar 21; Vol. 14 (1), pp. 6751. Date of Electronic Publication: 2024 Mar 21.
Typ publikacji:
Journal Article
MeSH Terms:
Muscular Dystrophy, Duchenne*/pathology
Animals ; Humans ; Mice ; Calcium/metabolism ; Calcium Channels/metabolism ; Cell Death ; Mitochondria/metabolism ; Mitochondrial Membrane Transport Proteins/metabolism ; Necrosis/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Aland Island Eye Disease with Retinoschisis in the Clinical Spectrum of CACNA1F -Associated Retinopathy-A Case Report.
Autorzy:
Wyględowska-Promieńska D; Department of Ophthalmology, Faculty of Medical Sciences in Katowice, Medical University of Silesia, 40-514 Katowice, Poland.; Kornel Gibiński University Clinical Center, 40-514 Katowice, Poland.
Świerczyńska M; Department of Ophthalmology, Faculty of Medical Sciences in Katowice, Medical University of Silesia, 40-514 Katowice, Poland.; Kornel Gibiński University Clinical Center, 40-514 Katowice, Poland.
Śpiewak D; Kornel Gibiński University Clinical Center, 40-514 Katowice, Poland.
Pojda-Wilczek D; Department of Ophthalmology, Faculty of Medical Sciences in Katowice, Medical University of Silesia, 40-514 Katowice, Poland.; Kornel Gibiński University Clinical Center, 40-514 Katowice, Poland.
Tronina A; Kornel Gibiński University Clinical Center, 40-514 Katowice, Poland.; Department of Pediatric Ophthalmology, Faculty of Medical Sciences in Katowice, Medical University of Silesia, 40-514 Katowice, Poland.
Dorecka M; Department of Ophthalmology, Faculty of Medical Sciences in Katowice, Medical University of Silesia, 40-514 Katowice, Poland.; Kornel Gibiński University Clinical Center, 40-514 Katowice, Poland.
Smędowski A; Department of Ophthalmology, Faculty of Medical Sciences in Katowice, Medical University of Silesia, 40-514 Katowice, Poland.; Kornel Gibiński University Clinical Center, 40-514 Katowice, Poland.; GlaucoTech Co., 40-282 Katowice, Poland.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Mar 02; Vol. 25 (5). Date of Electronic Publication: 2024 Mar 02.
Typ publikacji:
Case Reports
MeSH Terms:
Retinoschisis*
Genetic Diseases, X-Linked*/genetics
Retinal Diseases*
Night Blindness*
Myopia*
Albinism, Ocular*
Retinitis Pigmentosa*
Eye Diseases, Hereditary*
Male ; Humans ; Child, Preschool ; Infant ; Middle Aged ; Calcium Channels, L-Type/metabolism ; Retina/metabolism ; Mutation
SCR Disease Name:
Night blindness, congenital stationary; Cone-Rod Dystrophy, X-Linked, 3; Aland Island Eye Disease; Cone-Rod Dystrophy 3
Raport
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The difference in endothelium-dependent relaxation components in proximal and distal thoracic aorta regions of male rats.
Autorzy:
Mezhenskyi OR; Department of Nervous and Muscular Physiology, Bogomoletz Institute of Physiology, Kiyv, Ukraine.
Philyppov IB; Department of Nervous and Muscular Physiology, Bogomoletz Institute of Physiology, Kiyv, Ukraine.
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Źródło:
Physiological reports [Physiol Rep] 2024 Mar; Vol. 12 (6), pp. e15992.
Typ publikacji:
Journal Article
MeSH Terms:
Aorta, Thoracic*
Acetylcholine*/pharmacology
Rats ; Male ; Animals ; Rats, Wistar ; Endothelium, Vascular ; Vasodilation ; Aorta, Abdominal ; Potassium Channels ; Calcium Channels
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Do calcium channel blockers applied to cardiomyocytes cause increased channel expression resulting in reduced efficacy?
Autorzy:
Jæger KH; Simula Research Laboratory, Oslo, Norway. .
Charwat V; Organos Inc., Berkeley, CA, USA.
Wall S; Simula Research Laboratory, Oslo, Norway.; Organos Inc., Berkeley, CA, USA.
Healy KE; Department of Bioengineering, University of California, Berkeley, CA, USA.; Department of Material Science and Engineering, University of California, Berkeley, CA, USA.
Tveito A; Simula Research Laboratory, Oslo, Norway.
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Źródło:
NPJ systems biology and applications [NPJ Syst Biol Appl] 2024 Mar 01; Vol. 10 (1), pp. 22. Date of Electronic Publication: 2024 Mar 01.
Typ publikacji:
Journal Article
MeSH Terms:
Myocytes, Cardiac*
Induced Pluripotent Stem Cells*
Humans ; Calcium Channel Blockers/pharmacology ; Calcium ; Calcium Channels
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Acute Downregulation but Not Genetic Ablation of Murine MCU Impairs Suppressive Capacity of Regulatory CD4 T Cells.
Autorzy:
Jost P; Molecular Biophysics, Saarland University, 66421 Homburg, Germany.
Klein F; Molecular Biophysics, Saarland University, 66421 Homburg, Germany.
Brand B; Würzburg Institute of Systems Immunology, Max Planck Research Group at Julius-Maximilians University of Würzburg, 97078 Würzburg, Germany.
Wahl V; Experimental Pharmacology, Center for Molecular Signaling (PZMS), School of Medicine, Saarland University, 66421 Homburg, Germany.
Wyatt A; Experimental Pharmacology, Center for Molecular Signaling (PZMS), School of Medicine, Saarland University, 66421 Homburg, Germany.
Yildiz D; Experimental Pharmacology, Center for Molecular Signaling (PZMS), School of Medicine, Saarland University, 66421 Homburg, Germany.
Boehm U; Experimental Pharmacology, Center for Molecular Signaling (PZMS), School of Medicine, Saarland University, 66421 Homburg, Germany.
Niemeyer BA; Molecular Biophysics, Saarland University, 66421 Homburg, Germany.
Vaeth M; Würzburg Institute of Systems Immunology, Max Planck Research Group at Julius-Maximilians University of Würzburg, 97078 Würzburg, Germany.
Alansary D; Molecular Biophysics, Saarland University, 66421 Homburg, Germany.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Apr 24; Vol. 24 (9). Date of Electronic Publication: 2023 Apr 24.
Typ publikacji:
Journal Article
MeSH Terms:
Calcium Channels*/metabolism
T-Lymphocytes, Regulatory*/metabolism
Animals ; Mice ; Calcium/metabolism ; CD4-Positive T-Lymphocytes/metabolism ; Down-Regulation ; Mitochondria/genetics ; Mitochondria/metabolism
Czasopismo naukowe
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