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Wyświetlanie 61-80 z 174402
Tytuł:
Short cell cycle duration is a phenotype of human epidermal stem cells.
Autorzy:
Xiao T; Department of Dermatology, The Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA.; Department of Dermatology, VA Medical Center, San Francisco, CA, USA.
Eze UC; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA.
Charruyer-Reinwald A; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA.; Department of Dermatology, VA Medical Center, San Francisco, CA, USA.
Weisenberger T; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA.; Department of Dermatology, VA Medical Center, San Francisco, CA, USA.
Khalifa A; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA.; Department of Dermatology, VA Medical Center, San Francisco, CA, USA.; Faculty of Science, Zagazig University, Zagazig, Egypt.
Abegaze B; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA.
Schwab GK; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA.; Department of Dermatology, VA Medical Center, San Francisco, CA, USA.
Elsabagh RH; Immunology Department, Animal Health Research Institute (AHRI), Giza, Egypt.
Parenteau TR; Division of Geriatrics, UC San Francisco, San Francisco, CA, USA.
Kochanowski K; Department of Pharmaceutical Chemistry, UC San Francisco, San Francisco, CA, USA.
Piper M; Department of Plastic Surgery, UC San Francisco, San Francisco, CA, USA.
Xia Y; Department of Dermatology, The Second Affiliated Hospital of Xi'an Jiaotong University, Xi'an, Shaanxi, China.
Cheng JB; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA.; Department of Dermatology, VA Medical Center, San Francisco, CA, USA.
Cho RJ; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA.
Ghadially R; Department of Dermatology, San Francisco Co-Director Epithelial Section Eli and Edythe Broad Center of Regeneration Medicine and Stem Cell Research, University of California, 1700 Owens Street, San Francisco, CA, 94158, USA. .; Department of Dermatology, VA Medical Center, San Francisco, CA, USA. .
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Źródło:
Stem cell research & therapy [Stem Cell Res Ther] 2024 Mar 13; Vol. 15 (1), pp. 76. Date of Electronic Publication: 2024 Mar 13.
Typ publikacji:
Journal Article
MeSH Terms:
Pluripotent Stem Cells*
Adult ; Humans ; Cell Proliferation ; Cell Cycle ; Cell Division ; Cell Differentiation ; Phenotype ; Keratinocytes/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Large-scale phosphoproteomics reveals activation of the MAPK/GADD45β/P38 axis and cell cycle inhibition in response to BMP9 and BMP10 stimulation in endothelial cells.
Autorzy:
Al Tarrass M; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France.
Belmudes L; Grenoble Alpes University, CEA, INSERM, UA13 BGE, CNRS, CEA, FR2048, Grenoble, France.
Koça D; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France.
Azemard V; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France.
Liu H; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France.
Al Tabosh T; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France.
Ciais D; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France.; Present address: Université Côte d'Azur, CNRS, INSERM, iBV, Nice, France.
Desroches-Castan A; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France.
Battail C; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France.; Grenoble Alpes University, CEA, INSERM, UA13 BGE, CNRS, CEA, FR2048, Grenoble, France.
Couté Y; Grenoble Alpes University, CEA, INSERM, UA13 BGE, CNRS, CEA, FR2048, Grenoble, France.
Bouvard C; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France.
Bailly S; Biosanté Unit U1292, Grenoble Alpes University, CEA, Grenoble, 38000, France. .
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2024 Mar 04; Vol. 22 (1), pp. 158. Date of Electronic Publication: 2024 Mar 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Endothelial Cells*
Bone Morphogenetic Proteins*
Cell Cycle Checkpoints ; Phosphorylation ; G1 Phase Cell Cycle Checkpoints
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CDC20 assists its catalytic incorporation in the mitotic checkpoint complex.
Autorzy:
Piano V; Department of Mechanistic Cell Biology, Max Planck Institute of Molecular Physiology, 44227 Dortmund, Germany. .
Alex A; Department of Mechanistic Cell Biology, Max Planck Institute of Molecular Physiology, 44227 Dortmund, Germany.
Stege P; Department of Mechanistic Cell Biology, Max Planck Institute of Molecular Physiology, 44227 Dortmund, Germany.
Maffini S; Department of Mechanistic Cell Biology, Max Planck Institute of Molecular Physiology, 44227 Dortmund, Germany.
Stoppiello GA; Department of Mechanistic Cell Biology, Max Planck Institute of Molecular Physiology, 44227 Dortmund, Germany.
Huis In 't Veld PJ; Department of Mechanistic Cell Biology, Max Planck Institute of Molecular Physiology, 44227 Dortmund, Germany.
Vetter IR; Department of Mechanistic Cell Biology, Max Planck Institute of Molecular Physiology, 44227 Dortmund, Germany.
Musacchio A; Department of Mechanistic Cell Biology, Max Planck Institute of Molecular Physiology, 44227 Dortmund, Germany. .; Centre for Medical Biotechnology, Faculty of Biology, University Duisburg-Essen, 45141 Essen, Germany.
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Źródło:
Science (New York, N.Y.) [Science] 2021 Jan 01; Vol. 371 (6524), pp. 67-71.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
M Phase Cell Cycle Checkpoints*
Cdc20 Proteins/*metabolism
Kinetochores/*metabolism
Mad2 Proteins/*metabolism
Multiprotein Complexes/*metabolism
Biocatalysis ; Cell Cycle Proteins/metabolism ; HeLa Cells ; Humans ; Mad2 Proteins/genetics ; Mutation ; Poly-ADP-Ribose Binding Proteins/metabolism ; Protein Serine-Threonine Kinases ; Spindle Apparatus/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Chalkiness and premature controlled by energy homeostasis in OsNAC02 Ko-mutant during vegetative endosperm development.
Autorzy:
Yan, Mei (AUTHOR)
Jiao, Guiai (AUTHOR)
Shao, Gaoneng (AUTHOR)
Chen, Ying (AUTHOR)
Zhu, Maodi (AUTHOR)
Yang, Lingwei (AUTHOR)
Xie, Lihong (AUTHOR)
Hu, Peisong (AUTHOR)
Tang, Shaoqing (AUTHOR)
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Źródło:
BMC Plant Biology. 3/18/2024, Vol. 24 Issue 1, p1-24. 24p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The dynamic role of nucleoprotein SHCBP1 in the cancer cell cycle and its potential as a synergistic target for DNA-damaging agents in cancer therapy.
Autorzy:
Zhou, Mei (AUTHOR)
Duan, Limin (AUTHOR)
Chen, Jiangbin (AUTHOR)
Li, Yumei (AUTHOR)
Yin, Zhengrong (AUTHOR)
Song, Siwei (AUTHOR)
Cao, Yaqi (AUTHOR)
Luo, Ping (AUTHOR)
Hu, Fan (AUTHOR)
Yang, Guanghai (AUTHOR)
Xu, Juanjuan (AUTHOR)
Liao, Tingting (AUTHOR)
Jin, Yang (AUTHOR)
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Źródło:
Cell Communication & Signaling. 2/16/2024, Vol. 22 Issue 1, p1-29. 29p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Osmolar Modulation Drives Reversible Cell Cycle Exit and Human Pluripotent Cell Differentiation via NF‐κВ and WNT Signaling.
Autorzy:
Chui, Jonathan Sai‐Hong (AUTHOR)
Izuel‐Idoype, Teresa (AUTHOR)
Qualizza, Alessandra (AUTHOR)
de Almeida, Rita Pires (AUTHOR)
Piessens, Lindsey (AUTHOR)
van der Veer, Bernard K. (AUTHOR)
Vanmarcke, Gert (AUTHOR)
Malesa, Aneta (AUTHOR)
Athanasouli, Paraskevi (AUTHOR)
Boon, Ruben (AUTHOR)
Vriens, Joris (AUTHOR)
van Grunsven, Leo (AUTHOR)
Koh, Kian Peng (AUTHOR)
Verfaillie, Catherine M. (AUTHOR)
Lluis, Frederic (AUTHOR)
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Źródło:
Advanced Science. 2/16/2024, Vol. 11 Issue 7, p1-21. 21p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Cell Cycle Regulation in Macrophages and Susceptibility to HIV-1.
Autorzy:
Ferreira IATM; Cambridge Institute of Therapeutic Immunology & Infectious Disease (CITIID), Cambridge CB20AW, UK.; Department of Medicine, University of Cambridge, Cambridge CB20QQ, UK.
Porterfield JZ; Department of Microbiology, University of Kentucky, Lexington, KY 40536, USA.; Africa Health Research Institute, Durban 4001, South Africa.
Gupta RK; Cambridge Institute of Therapeutic Immunology & Infectious Disease (CITIID), Cambridge CB20AW, UK.; Department of Medicine, University of Cambridge, Cambridge CB20QQ, UK.; Africa Health Research Institute, Durban 4001, South Africa.
Mlcochova P; Cambridge Institute of Therapeutic Immunology & Infectious Disease (CITIID), Cambridge CB20AW, UK.; Department of Medicine, University of Cambridge, Cambridge CB20QQ, UK.
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Źródło:
Viruses [Viruses] 2020 Jul 31; Vol. 12 (8). Date of Electronic Publication: 2020 Jul 31.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Cell Cycle*
HIV-1/*physiology
Macrophages/*physiology
Macrophages/*virology
Animals ; Cell Cycle Checkpoints ; Cells, Cultured ; DNA Damage ; G1 Phase ; Gram-Negative Bacteria/immunology ; Histone Deacetylase Inhibitors/pharmacology ; Humans ; Macrophages/immunology ; Phosphorylation ; Resting Phase, Cell Cycle ; SAM Domain and HD Domain-Containing Protein 1/metabolism ; vif Gene Products, Human Immunodeficiency Virus/metabolism ; vpr Gene Products, Human Immunodeficiency Virus/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Biomedical association analysis between G2/M checkpoint genes and susceptibility to HIV-1 infection and AIDS progression from a northern chinese MSM population.
Autorzy:
Wu J; College of Basic Medicine, Harbin Medical University-Daqing Campus, Daqing, Heilongjiang Province, 163319, China.
Xu L; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China.
Liu B; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.
Sun W; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China.
Hu Y; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.
Yang Y; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.
Guo K; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.
Jia X; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China.
Sun H; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China.
Wu J; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China.
Huang Y; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China.
Ji W; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China.
Fu S; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China.; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China.
Qiao Y; College of Basic Medicine, Harbin Medical University-Daqing Campus, Daqing, Heilongjiang Province, 163319, China. .; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China. .; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China. .
Zhang X; Laboratory of Medical Genetics, Harbin Medical University, Harbin, Heilongjiang Province, 150081, China. .; Key Laboratory of Preservation of Human Genetic Resources and Disease Control in China, Harbin Medical University, Ministry of Education, Harbin, Heilongjiang Province, 150081, China. .
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Źródło:
AIDS research and therapy [AIDS Res Ther] 2023 Jul 19; Vol. 20 (1), pp. 51. Date of Electronic Publication: 2023 Jul 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Acquired Immunodeficiency Syndrome*/epidemiology
Acquired Immunodeficiency Syndrome*/genetics
HIV Infections*/epidemiology
HIV Infections*/genetics
Sexual and Gender Minorities*
Cell Cycle Checkpoints*/genetics
Humans ; Male ; East Asian People ; HIV-1 ; Homosexuality, Male
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Novel variants, muscle imaging, and myopathological changes in Chinese patients with VCP-related multisystem proteinopathy.
Autorzy:
Wan Y; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Wang Q; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Zheng Y; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Yu M; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Xie Z; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Ling C; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Meng L; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Yu J; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Zheng Y; Department of Neurology, the First Affiliated Hospital of Nanchang University, 330006, Jiangxi, Nanchang, 17 Yongwaizheng Street, China.
Wang Y; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Zhang W; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Liu C; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Zhao Y; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Yuan Y; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Deng J; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.
Gang Q; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.; Department of Neurology, Peking University First Hospital, Peking University, 100034, Beijing, China.
Wang Z; Department of Neurology, Peking University First Hospital, 100034, Beijing, China.; Department of Neurology, Peking University First Hospital, Peking University, 100034, Beijing, China.
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Źródło:
Molecular genetics & genomic medicine [Mol Genet Genomic Med] 2023 Jul; Vol. 11 (7), pp. e2176. Date of Electronic Publication: 2023 Mar 31.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Cycle Proteins*/genetics
Muscular Diseases*/genetics
Adult ; Humans ; Valosin Containing Protein/genetics ; East Asian People ; Muscle, Skeletal/diagnostic imaging ; Muscle, Skeletal/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
SMCHD1 and LRIF1 converge at the FSHD-associated D4Z4 repeat and LRIF1 promoter yet display different modes of action.
Autorzy:
Šikrová D; Department of Human Genetics, Leiden University Medical Center, 2333ZC, Leiden, The Netherlands.
Testa AM; Department of Human Genetics, Leiden University Medical Center, 2333ZC, Leiden, The Netherlands.; Department of Biomedical Sciences, University of Padua, 35100, Padua, Italy.
Willemsen I; Department of Human Genetics, Leiden University Medical Center, 2333ZC, Leiden, The Netherlands.
van den Heuvel A; Department of Human Genetics, Leiden University Medical Center, 2333ZC, Leiden, The Netherlands.
Tapscott SJ; Human Biology Division, Fred Hutchinson Cancer Research Center, Seattle, WA, 98109, USA.
Daxinger L; Department of Human Genetics, Leiden University Medical Center, 2333ZC, Leiden, The Netherlands.
Balog J; Department of Human Genetics, Leiden University Medical Center, 2333ZC, Leiden, The Netherlands.
van der Maarel SM; Department of Human Genetics, Leiden University Medical Center, 2333ZC, Leiden, The Netherlands. S.M.van_der_.
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Źródło:
Communications biology [Commun Biol] 2023 Jun 28; Vol. 6 (1), pp. 677. Date of Electronic Publication: 2023 Jun 28.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Chromosomal Proteins, Non-Histone*/genetics
Muscular Dystrophy, Facioscapulohumeral*/genetics
Cell Cycle Proteins*/genetics
Humans ; Chromatin/genetics ; Epigenomics ; Genes, Homeobox ; Muscle, Skeletal
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
α-catenin interaction with YAP/FoxM1/TEAD-induced CEP55 supports liver cancer cell migration.
Autorzy:
Tang Y; Institute of Pathology, University Hospital Heidelberg, Heidelberg, Germany.
Thiess L; Institute of Pathology, University Hospital Heidelberg, Heidelberg, Germany.
Weiler SME; Institute of Pathology, University Hospital Heidelberg, Heidelberg, Germany.
Tóth M; Institute of Pathology, University Hospital Heidelberg, Heidelberg, Germany.
Rose F; Institute of Pathology, University Hospital Heidelberg, Heidelberg, Germany.
Merker S; CFMP, Core Facility for Mass Spectrometry & Proteomics at the Center for Molecular Biology (ZMBH), Heidelberg University, Heidelberg, Germany.
Ruppert T; CFMP, Core Facility for Mass Spectrometry & Proteomics at the Center for Molecular Biology (ZMBH), Heidelberg University, Heidelberg, Germany.
Schirmacher P; Institute of Pathology, University Hospital Heidelberg, Heidelberg, Germany.
Breuhahn K; Institute of Pathology, University Hospital Heidelberg, Heidelberg, Germany. .
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2023 Jun 28; Vol. 21 (1), pp. 162. Date of Electronic Publication: 2023 Jun 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adenocarcinoma*
Carcinoma, Hepatocellular*
Colonic Neoplasms*
Liver Neoplasms*
Cell Cycle Proteins*
Animals ; Humans ; Mice ; alpha Catenin ; Cell Line ; Cell Movement ; Neoplasm Recurrence, Local ; Proto-Oncogene Proteins c-akt
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Optineurin deficiency impairs autophagy to cause interferon beta overproduction and increased survival of mice following viral infection.
Autorzy:
Fukushi M; Department of Virology, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima, Japan.
Ohsawa R; Department of Epidemiology, Research Institute of Radiation Biology and Medicine, Hiroshima University, Hiroshima, Japan.
Okinaka Y; Graduate School of Integrated Sciences for Life, Hiroshima University, Higashi-Hiroshima City, Hiroshima, Japan.
Oikawa D; Department of Medical Biochemistry, Graduate School of Medicine, Osaka Metropolitan University, Osaka, Japan.
Kiyono T; Project for Prevention of HPV-related Cancer, Exploratory Oncology Research and Clinical Trial Center, National Cancer Center, Kashiwa, Chiba, Japan.
Moriwaki M; Department of Epidemiology, Research Institute of Radiation Biology and Medicine, Hiroshima University, Hiroshima, Japan.
Irie T; Department of Virology, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima, Japan.
Oda K; Department of Virology, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima, Japan.
Kamei Y; Spectrography and Bioimaging Facility, National Institute for Basic Biology, Okazaki, Aichi, Japan.
Tokunaga F; Department of Medical Biochemistry, Graduate School of Medicine, Osaka Metropolitan University, Osaka, Japan.
Sotomaru Y; Natural Science Center for Basic Research and Development, Hiroshima University, Hiroshima, Japan.
Maruyama H; Department of Clinical Neuroscience & Therapeutics, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima, Japan.
Kawakami H; Department of Epidemiology, Research Institute of Radiation Biology and Medicine, Hiroshima University, Hiroshima, Japan.
Sakaguchi T; Department of Virology, Graduate School of Biomedical and Health Sciences, Hiroshima University, Hiroshima, Japan.
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Źródło:
PloS one [PLoS One] 2023 Jun 23; Vol. 18 (6), pp. e0287545. Date of Electronic Publication: 2023 Jun 23 (Print Publication: 2023).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Amyotrophic Lateral Sclerosis*/genetics
Cell Cycle Proteins*/genetics
Virus Diseases*
Membrane Transport Proteins*/genetics
Animals ; Humans ; Mice ; Autophagy/genetics ; Immunity, Innate ; Interferon-beta/genetics ; Transcription Factor TFIIIA/genetics ; Transcription Factor TFIIIA/metabolism ; Mice, Knockout
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The p38/MK2 Pathway Functions as Chk1-Backup Downstream of ATM/ATR in G 2 -Checkpoint Activation in Cells Exposed to Ionizing Radiation.
Autorzy:
Luo D; Institute of Medical Radiation Biology, University Hospital Essen, University of Duisburg-Essen, 45147 Essen, Germany.; Division of Experimental Radiation Biology, Department of Radiation Therapy, University Hospital Essen, University of Duisburg-Essen, 45147 Essen, Germany.
Mladenov E; Institute of Medical Radiation Biology, University Hospital Essen, University of Duisburg-Essen, 45147 Essen, Germany.; Division of Experimental Radiation Biology, Department of Radiation Therapy, University Hospital Essen, University of Duisburg-Essen, 45147 Essen, Germany.
Soni A; Institute of Medical Radiation Biology, University Hospital Essen, University of Duisburg-Essen, 45147 Essen, Germany.; Division of Experimental Radiation Biology, Department of Radiation Therapy, University Hospital Essen, University of Duisburg-Essen, 45147 Essen, Germany.
Stuschke M; Division of Experimental Radiation Biology, Department of Radiation Therapy, University Hospital Essen, University of Duisburg-Essen, 45147 Essen, Germany.; German Cancer Consortium (DKTK), Partner Site University Hospital Essen, 45147 Essen, Germany.; German Cancer Research Center (DKFZ), 69120 Heidelberg, Germany.
Iliakis G; Institute of Medical Radiation Biology, University Hospital Essen, University of Duisburg-Essen, 45147 Essen, Germany.; Division of Experimental Radiation Biology, Department of Radiation Therapy, University Hospital Essen, University of Duisburg-Essen, 45147 Essen, Germany.
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Źródło:
Cells [Cells] 2023 May 14; Vol. 12 (10). Date of Electronic Publication: 2023 May 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Cycle Proteins*/metabolism
Radiation, Ionizing*
Phosphorylation ; Ataxia Telangiectasia Mutated Proteins/metabolism ; DNA/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
GINS2 regulates the proliferation and apoptosis of colon cancer cells through PTP4A1.
Autorzy:
Hu H; Department of Endoscopy, Guizhou Provincial People's Hospital, Guiyang, Guizhou 550002, P.R. China.
Ye L; Department of Endoscopy, Guizhou Provincial People's Hospital, Guiyang, Guizhou 550002, P.R. China.
Liu Z; Department of Gastroenterology, Affiliated Cancer Hospital of Guizhou Medical University, Guiyang, Guizhou 550001, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2022 Apr; Vol. 25 (4). Date of Electronic Publication: 2022 Feb 09.
Typ publikacji:
Journal Article
MeSH Terms:
Cell Cycle Proteins/*metabolism
Chromosomal Proteins, Non-Histone/*metabolism
Colonic Neoplasms/*metabolism
Membrane Proteins/*metabolism
Protein Tyrosine Phosphatases/*metabolism
Apoptosis/genetics ; Cell Cycle Checkpoints/genetics ; Cell Cycle Proteins/genetics ; Cell Line ; Cell Proliferation/genetics ; Chromosomal Proteins, Non-Histone/genetics ; Colonic Neoplasms/genetics ; Gene Expression Regulation, Neoplastic ; Humans ; Membrane Proteins/genetics ; Protein Tyrosine Phosphatases/genetics ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
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Tytuł:
Exceptional origin activation revealed by comparative analysis in two laboratory yeast strains.
Autorzy:
Joshi I; Department of Biochemistry and Molecular Biology, SUNY Upstate Medical University, Syracuse, New York, United States of America.
Peng J; Department of Biochemistry and Molecular Biology, SUNY Upstate Medical University, Syracuse, New York, United States of America.
Alvino G; Department of Genome Sciences, University of Washington, Seattle, Washington, United States of America.
Kwan E; Department of Genome Sciences, University of Washington, Seattle, Washington, United States of America.
Feng W; Department of Biochemistry and Molecular Biology, SUNY Upstate Medical University, Syracuse, New York, United States of America.
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Źródło:
PloS one [PLoS One] 2022 Feb 14; Vol. 17 (2), pp. e0263569. Date of Electronic Publication: 2022 Feb 14 (Print Publication: 2022).
Typ publikacji:
Comparative Study; Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Replication Origin*
Cell Cycle Proteins/*genetics
Checkpoint Kinase 2/*genetics
DNA, Fungal/*genetics
Saccharomyces cerevisiae/*growth & development
Saccharomyces cerevisiae Proteins/*genetics
Cell Cycle Proteins/metabolism ; Checkpoint Kinase 2/metabolism ; Laboratories ; Mutation ; Phosphorylation ; Polymorphism, Single Nucleotide ; S Phase Cell Cycle Checkpoints ; Saccharomyces cerevisiae/genetics ; Saccharomyces cerevisiae Proteins/metabolism ; Transcription, Genetic ; Whole Genome Sequencing
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Divergence of the PIERCE1 expression between mice and humans as a p53 target gene.
Autorzy:
Kim HJ; Department of Biochemistry, College of Life Science and Biotechnology, Yonsei University, Seoul, Republic of Korea.
Lee SE; Department of Biochemistry, College of Life Science and Biotechnology, Yonsei University, Seoul, Republic of Korea.
Na H; Department of Biochemistry, College of Life Science and Biotechnology, Yonsei University, Seoul, Republic of Korea.
Roe JS; Department of Biochemistry, College of Life Science and Biotechnology, Yonsei University, Seoul, Republic of Korea.
Roh JI; Department of Biochemistry, College of Life Science and Biotechnology, Yonsei University, Seoul, Republic of Korea.
Lee HW; Department of Biochemistry, College of Life Science and Biotechnology, Yonsei University, Seoul, Republic of Korea.
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Źródło:
PloS one [PLoS One] 2020 Aug 03; Vol. 15 (8), pp. e0236881. Date of Electronic Publication: 2020 Aug 03 (Print Publication: 2020).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Cycle Proteins*/genetics
Cell Cycle Proteins*/metabolism
Response Elements/*genetics
Tumor Suppressor Protein p53/*metabolism
Animals ; Cell Cycle/genetics ; Cell Line, Tumor ; DNA Damage/genetics ; Humans ; Mice ; Promoter Regions, Genetic ; Transcription, Genetic/physiology
Czasopismo naukowe
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Tytuł:
Role of Polo-like Kinases Plk1 and Plk4 in the Initiation of Centriole Duplication-Impact on Cancer.
Autorzy:
Hoffmann I; F045, Cell Cycle Control and Carcinogenesis, Im Neuenheimer Feld 242, 69115 Heidelberg, Germany.
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Źródło:
Cells [Cells] 2022 Feb 24; Vol. 11 (5). Date of Electronic Publication: 2022 Feb 24.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Centrioles*/metabolism
Neoplasms*/genetics
Neoplasms*/metabolism
Cell Cycle Proteins/*metabolism
Protein Serine-Threonine Kinases/*metabolism
Proto-Oncogene Proteins/*metabolism
Cell Cycle/physiology ; Centrosome/metabolism ; Humans ; Mitosis ; Polo-Like Kinase 1
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interphase-arrested Drosophila embryos activate zygotic gene expression and initiate mid-blastula transition events at a low nuclear-cytoplasmic ratio.
Autorzy:
Strong IJT; Department of Biochemistry and Biophysics, University of California San Francisco, San Francisco, California, United States of America.
Lei X; Hunan Key Laboratory of Molecular Precision Medicine, Department of Oncology, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Chen F; Hunan Key Laboratory of Molecular Precision Medicine, Department of Oncology, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Yuan K; Hunan Key Laboratory of Molecular Precision Medicine, Department of Oncology, Xiangya Hospital, Central South University, Changsha, Hunan, China.; Hunan Key Laboratory of Medical Genetics, School of Life Sciences, Central South University, Changsha, Hunan, China.
O'Farrell PH; Department of Biochemistry and Biophysics, University of California San Francisco, San Francisco, California, United States of America.
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Źródło:
PLoS biology [PLoS Biol] 2020 Oct 22; Vol. 18 (10), pp. e3000891. Date of Electronic Publication: 2020 Oct 22 (Print Publication: 2020).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:
Cell Cycle Checkpoints*/genetics
Gene Expression Regulation, Developmental*
Interphase*/genetics
Blastula/*cytology
Cell Nucleus/*metabolism
Drosophila melanogaster/*embryology
Embryo, Nonmammalian/*cytology
Zygote/*metabolism
Animals ; Cell Cycle Proteins/metabolism ; Drosophila melanogaster/cytology ; Drosophila melanogaster/genetics ; Gastrulation ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Transcription, Genetic ; Transcriptome/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CHK1 monitors spindle assembly checkpoint and DNA damage repair during the first cleavage of mouse early embryos.
Autorzy:
Ju JQ; College of Animal Science and Technology, Nanjing Agricultural University, Nanjing, China.
Li XH; College of Animal Science and Technology, Nanjing Agricultural University, Nanjing, China.
Pan MH; College of Animal Science and Technology, Nanjing Agricultural University, Nanjing, China.
Xu Y; College of Animal Science and Technology, Nanjing Agricultural University, Nanjing, China.
Sun MH; College of Animal Science and Technology, Nanjing Agricultural University, Nanjing, China.
Xu Y; College of Animal Science and Technology, Nanjing Agricultural University, Nanjing, China.
Sun SC; College of Animal Science and Technology, Nanjing Agricultural University, Nanjing, China.
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Źródło:
Cell proliferation [Cell Prolif] 2020 Oct; Vol. 53 (10), pp. e12895. Date of Electronic Publication: 2020 Sep 10.
Typ publikacji:
Journal Article
MeSH Terms:
DNA Repair*
M Phase Cell Cycle Checkpoints*/drug effects
Checkpoint Kinase 1/*metabolism
Animals ; Apoptosis/drug effects ; Aurora Kinase B/metabolism ; Cell Cycle Proteins/metabolism ; Checkpoint Kinase 1/antagonists & inhibitors ; Chromosome Segregation/drug effects ; DNA Damage/drug effects ; Embryo, Mammalian/cytology ; Embryo, Mammalian/metabolism ; Embryonic Development/drug effects ; Kinetochores/metabolism ; Mice ; Microtubules/metabolism ; Oxidative Stress/drug effects ; Phenylurea Compounds/pharmacology ; Protein Serine-Threonine Kinases/metabolism ; Pyrazines/pharmacology ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 61-80 z 174402

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