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Wyszukujesz frazę ""Cell Cycle Checkpoints"" wg kryterium: Temat


Tytuł :
The quinazoline derivative, 04NB-03, induces cell cycle arrest and apoptosis in hepatocellular carcinoma cells in a reactive oxygen species-dependent manner.
Autorzy :
Wang R; Guangzhou Jinan Biomedicine Research and Development Center, Key Laboratory of Bioengineering Medicine of Guangdong Province, Institute of Biomedicine, College of Life Science and Technology, Jinan University, Guangzhou, 510632, China.
Zhu Y; Guangzhou Jinan Biomedicine Research and Development Center, Key Laboratory of Bioengineering Medicine of Guangdong Province, Institute of Biomedicine, College of Life Science and Technology, Jinan University, Guangzhou, 510632, China.
Chen J; Guangzhou Jinan Biomedicine Research and Development Center, Key Laboratory of Bioengineering Medicine of Guangdong Province, Institute of Biomedicine, College of Life Science and Technology, Jinan University, Guangzhou, 510632, China.
Wang Y; Guangzhou Jinan Biomedicine Research and Development Center, Key Laboratory of Bioengineering Medicine of Guangdong Province, Institute of Biomedicine, College of Life Science and Technology, Jinan University, Guangzhou, 510632, China.
Song X; Guangzhou Jinan Biomedicine Research and Development Center, Key Laboratory of Bioengineering Medicine of Guangdong Province, Institute of Biomedicine, College of Life Science and Technology, Jinan University, Guangzhou, 510632, China.
Wu Y; Guangzhou Jinan Biomedicine Research and Development Center, Key Laboratory of Bioengineering Medicine of Guangdong Province, Institute of Biomedicine, College of Life Science and Technology, Jinan University, Guangzhou, 510632, China.
Jin F; Guangzhou Jinan Biomedicine Research and Development Center, Key Laboratory of Bioengineering Medicine of Guangdong Province, Institute of Biomedicine, College of Life Science and Technology, Jinan University, Guangzhou, 510632, China; Integrated Chinese and Western Medicine Postdoctoral Research Station, Jinan University, Guangzhou, 510632, China. Electronic address: .
Wang Y; Guangzhou Jinan Biomedicine Research and Development Center, Key Laboratory of Bioengineering Medicine of Guangdong Province, Institute of Biomedicine, College of Life Science and Technology, Jinan University, Guangzhou, 510632, China; Key Laboratory of Virology of Guangzhou, Jinan University, Guangzhou, 510632, China. Electronic address: .
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Źródło :
Chemico-biological interactions [Chem Biol Interact] 2021 Apr 01; Vol. 338, pp. 109371. Date of Electronic Publication: 2021 Feb 12.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Carcinoma, Hepatocellular/*pathology
Cell Cycle Checkpoints/*drug effects
Liver Neoplasms/*pathology
Quinazolines/*pharmacology
Reactive Oxygen Species/*metabolism
Animals ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Female ; G2 Phase Cell Cycle Checkpoints/drug effects ; Humans ; Inhibitory Concentration 50 ; M Phase Cell Cycle Checkpoints/drug effects ; Mice, Inbred BALB C ; Mice, Nude ; Quinazolines/chemistry
Czasopismo naukowe
Tytuł :
The Insulin Receptor Adaptor IRS2 is an APC/C Substrate That Promotes Cell Cycle Protein Expression and a Robust Spindle Assembly Checkpoint.
Autorzy :
Manohar S; Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA.
Yu Q; Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA.
Gygi SP; Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA.
King RW; Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA. Electronic address: .
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Źródło :
Molecular & cellular proteomics : MCP [Mol Cell Proteomics] 2020 Sep; Vol. 19 (9), pp. 1450-1467. Date of Electronic Publication: 2020 Jun 18.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
M Phase Cell Cycle Checkpoints*/drug effects
Anaphase-Promoting Complex-Cyclosome/*metabolism
Cell Cycle Proteins/*metabolism
G1 Phase Cell Cycle Checkpoints/*genetics
Insulin Receptor Substrate Proteins/*metabolism
Amino Acid Motifs ; Anaphase-Promoting Complex-Cyclosome/drug effects ; Animals ; Antigens, CD/metabolism ; Cadherins/metabolism ; Cell Line ; Chromatography, Liquid ; G1 Phase Cell Cycle Checkpoints/drug effects ; Gene Expression Regulation/drug effects ; Gene Expression Regulation/genetics ; Gene Knockout Techniques ; Humans ; Insulin/metabolism ; Insulin Receptor Substrate Proteins/genetics ; Mice ; Microtubules/drug effects ; Microtubules/metabolism ; Phosphorylation ; Piperazines/pharmacology ; Protein Kinase Inhibitors/pharmacology ; Protein Stability ; Proteomics ; Pyridines/pharmacology ; Tandem Mass Spectrometry ; Time-Lapse Imaging ; Ubiquitination/drug effects ; Ubiquitination/genetics
Czasopismo naukowe
Tytuł :
Amentoflavone Induces Cell-cycle Arrest, Apoptosis, and Invasion Inhibition in Non-small Cell Lung Cancer Cells.
Autorzy :
Chen WT; Department of Psychiatry, Zuoying Branch of Kaohsiung Armed Forces General Hospital, Kaohsiung, Taiwan, R.O.C.
Chen CH; Surgical department of Show Chwan Memorial Hospital, Changhua, Taiwan, R.O.C.
Su HT; Division of Anesthesiology, Taichung Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation, Taichung, Taiwan, R.O.C.; School of Medicine, Tzu Chi University, Hualien, Taiwan, R.O.C.
Yueh PF; Institute of Traditional Medicine, School of Medicine, National Yang Ming Chiao Tung University, Taipei, Taiwan, R.O.C.; Department of Biological Science and Technology, China Medical University, Taichung, Taiwan, R.O.C.
Hsu FT; Department of Biological Science and Technology, China Medical University, Taichung, Taiwan, R.O.C.
Chiang IT; Department of Radiation Oncology, Show Chwan Memorial Hospital, Changhua, Taiwan, R.O.C.; .; Department of Radiation Oncology, Chang Bing Show Chwan Memorial Hospital, Changhua, Taiwan, R.O.C.; Department of Medical Imaging and Radiological Sciences, Central Taiwan University of Science and Technology, Taichung, Taiwan, R.O.C.
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Źródło :
Anticancer research [Anticancer Res] 2021 Mar; Vol. 41 (3), pp. 1357-1364.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Biflavonoids/*pharmacology
Carcinoma, Non-Small-Cell Lung/*pathology
Cell Cycle Checkpoints/*drug effects
Lung Neoplasms/*pathology
Carcinoma, Non-Small-Cell Lung/metabolism ; Cell Line, Tumor ; Cell Movement/drug effects ; Cell Survival/drug effects ; Cytochrome P-450 CYP2C9 Inhibitors/pharmacology ; G1 Phase Cell Cycle Checkpoints ; Humans ; Lung Neoplasms/metabolism ; Matrix Metalloproteinases/metabolism ; NF-kappa B/metabolism ; Neoplasm Invasiveness ; Signal Transduction/drug effects ; Vascular Endothelial Growth Factor A/metabolism
Czasopismo naukowe
Tytuł :
Transmembrane and Immunoglobulin Domain Containing 1, a Putative Tumor Suppressor, Induces G2/M Cell Cycle Checkpoint Arrest in Colon Cancer Cells.
Autorzy :
De La Cena KOC; Department of Pathology, School of Medicine, Boston University Medical Campus, Boston, Massachusetts.
Ho RX; Department of Pathology, School of Medicine, Boston University Medical Campus, Boston, Massachusetts.
Amraei R; Department of Pathology, School of Medicine, Boston University Medical Campus, Boston, Massachusetts.
Woolf N; Department of Pathology, School of Medicine, Boston University Medical Campus, Boston, Massachusetts.
Tashjian JY; Department of Pathology, School of Medicine, Boston University Medical Campus, Boston, Massachusetts.
Zhao Q; Department of Pathology, School of Medicine, Boston University Medical Campus, Boston, Massachusetts.
Richards S; Renal Section, Department of Medicine, Boston Medical Center, Boston, Massachusetts.
Walker J; Renal Section, Department of Medicine, Boston Medical Center, Boston, Massachusetts.
Huang J; Department of Pathology, School of Medicine, Boston University Medical Campus, Boston, Massachusetts.
Chitalia VC; Renal Section, Department of Medicine, Boston Medical Center, Boston, Massachusetts; Boston Veterans Affairs Healthcare System, Boston, Massachusetts.
Rahimi N; Department of Pathology, School of Medicine, Boston University Medical Campus, Boston, Massachusetts. Electronic address: .
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Źródło :
The American journal of pathology [Am J Pathol] 2021 Jan; Vol. 191 (1), pp. 157-167. Date of Electronic Publication: 2020 Oct 29.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Cell Cycle Checkpoints/*physiology
Colonic Neoplasms/*metabolism
Membrane Glycoproteins/*metabolism
Adenoma/genetics ; Adenoma/metabolism ; Adenoma/pathology ; Animals ; Cell Movement/genetics ; Cell Proliferation/genetics ; Cell Transformation, Neoplastic/metabolism ; Colonic Neoplasms/genetics ; Colonic Neoplasms/pathology ; G2 Phase Cell Cycle Checkpoints/physiology ; Genes, Tumor Suppressor/physiology ; Humans ; Membrane Glycoproteins/genetics ; Mice ; Mice, Knockout ; Tumor Suppressor Proteins/genetics ; Tumor Suppressor Proteins/metabolism
Czasopismo naukowe
Tytuł :
Cyclin Y binds and activates CDK4 to promote the G1/S phase transition in hepatocellular carcinoma cells via Rb signaling.
Autorzy :
Chen L; Department of Pathology, School of Basic Medical Sciences, Fudan University, Shanghai, China.
Wang X; The State Key Laboratory of Genetic Engineering, Fudan University, Shanghai, China.
Cheng H; Department of Pathology, School of Basic Medical Sciences, Fudan University, Shanghai, China.
Zhang W; Department of Pathology, School of Basic Medical Sciences, Fudan University, Shanghai, China.
Liu Y; Department of Pathology, School of Basic Medical Sciences, Fudan University, Shanghai, China.
Zeng W; Department of Pathology, School of Basic Medical Sciences, Fudan University, Shanghai, China.
Yu L; The State Key Laboratory of Genetic Engineering, Fudan University, Shanghai, China.
Huang C; Department of Liver Surgery & Transplantation, Liver Cancer Institute, Zhongshan Hospital, Fudan University, Shanghai, China. Electronic address: .
Liu G; Department of Pathology, School of Basic Medical Sciences, Fudan University, Shanghai, China. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 Dec 17; Vol. 533 (4), pp. 1162-1169. Date of Electronic Publication: 2020 Oct 07.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Carcinoma, Hepatocellular/*metabolism
Cyclin-Dependent Kinase 4/*metabolism
Cyclins/*metabolism
G1 Phase Cell Cycle Checkpoints/*genetics
Liver Neoplasms/*metabolism
Retinoblastoma Protein/*metabolism
S Phase Cell Cycle Checkpoints/*genetics
Carcinoma, Hepatocellular/genetics ; Carcinoma, Hepatocellular/mortality ; Cell Cycle Checkpoints/genetics ; Cyclin A/genetics ; Cyclin A/metabolism ; Cyclin-Dependent Kinase 2/genetics ; Cyclin-Dependent Kinase 2/metabolism ; Humans ; Immunohistochemistry ; Liver Neoplasms/genetics ; Liver Neoplasms/mortality ; Phosphorylation ; Prognosis ; Retinoblastoma Protein/antagonists & inhibitors ; Signal Transduction ; Tissue Array Analysis
Czasopismo naukowe
Tytuł :
Pyoluteorin induces cell cycle arrest and apoptosis in human triple-negative breast cancer cells MDA-MB-231.
Autorzy :
Ding T; State Key Laboratory of New Drug and Pharmaceutical Process, Shanghai Institute of Pharmaceutical Industry, China State Institute of Pharmaceutical Industry, Shanghai, China.
Yang LJ; Institute of Interdisciplinary Integrative Medicine Research, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Zhang WD; State Key Laboratory of New Drug and Pharmaceutical Process, Shanghai Institute of Pharmaceutical Industry, China State Institute of Pharmaceutical Industry, Shanghai, China.; School of Pharmacy, Second Military Medical University, Shanghai, China.
Shen YH; School of Pharmacy, Second Military Medical University, Shanghai, China.
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Źródło :
The Journal of pharmacy and pharmacology [J Pharm Pharmacol] 2020 Jul; Vol. 72 (7), pp. 969-978. Date of Electronic Publication: 2020 Apr 04.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Cell Proliferation/*drug effects
G2 Phase Cell Cycle Checkpoints/*drug effects
M Phase Cell Cycle Checkpoints/*drug effects
Phenols/*pharmacology
Pyrroles/*pharmacology
Triple Negative Breast Neoplasms/*drug therapy
Antineoplastic Agents/pharmacology ; Caspase 3/metabolism ; Cell Cycle Checkpoints/drug effects ; Cell Line, Tumor ; Cell Survival/drug effects ; Drug Discovery ; Humans ; Membrane Potential, Mitochondrial/drug effects
Czasopismo naukowe
Tytuł :
Glaucocalyxin A induces cell cycle arrest and apoptosis via inhibiting NF-κB/p65 signaling pathway in melanoma cells.
Autorzy :
Chen J; Department of Burns and Plastic Surgery, The Second Affiliated Hospital of Shantou University Medical College, Shantou, Guangdong, PR China.
Zhang W; Department of Burns and Plastic Surgery, The Second Affiliated Hospital of Shantou University Medical College, Shantou, Guangdong, PR China.
Pan C; Department of Chemistry, Shantou University Medical College, Shantou, Guangdong, PR China.
Fan J; Department of Plastic and Reconstructive Surgery, Beijing Chao-Yang Hospital, Capital Medical University, Beijing, PR China. Electronic address: .
Zhong X; Department of Burns and Plastic Surgery, The Second Affiliated Hospital of Shantou University Medical College, Shantou, Guangdong, PR China. Electronic address: .
Tang S; Department of Burns and Plastic Surgery, The Second Affiliated Hospital of Shantou University Medical College, Shantou, Guangdong, PR China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2021 Apr 15; Vol. 271, pp. 119185. Date of Electronic Publication: 2021 Feb 09.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Cell Cycle Checkpoints/*drug effects
Diterpenes, Kaurane/*pharmacology
Melanoma/*metabolism
Transcription Factor RelA/*metabolism
Animals ; Antineoplastic Agents/therapeutic use ; Apoptosis/physiology ; Cell Cycle Checkpoints/physiology ; Cell Line, Tumor ; Cell Survival/drug effects ; Cell Survival/physiology ; Diterpenes, Kaurane/therapeutic use ; Dose-Response Relationship, Drug ; Humans ; Melanoma/drug therapy ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Signal Transduction/drug effects ; Signal Transduction/physiology ; Transcription Factor RelA/antagonists & inhibitors ; Xenograft Model Antitumor Assays/methods
Czasopismo naukowe
Tytuł :
HIV-1 Vif Triggers Cell Cycle Arrest by Degrading Cellular PPP2R5 Phospho-regulators.
Autorzy :
Salamango DJ; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA. Electronic address: .
Ikeda T; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA; Howard Hughes Medical Institute, University of Minnesota, Minneapolis, MN 55455, USA.
Moghadasi SA; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA.
Wang J; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA.
McCann JL; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA.
Serebrenik AA; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA.
Ebrahimi D; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA.
Jarvis MC; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA.
Brown WL; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA.
Harris RS; Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN 55455, USA; Masonic Cancer Center, University of Minnesota, Minneapolis, MN 55455, USA; Institute for Molecular Virology, University of Minnesota, Minneapolis, MN 55455, USA; Howard Hughes Medical Institute, University of Minnesota, Minneapolis, MN 55455, USA. Electronic address: .
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Źródło :
Cell reports [Cell Rep] 2019 Oct 29; Vol. 29 (5), pp. 1057-1065.e4.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Cycle Checkpoints*
Proteolysis*
Protein Phosphatase 2/*metabolism
vif Gene Products, Human Immunodeficiency Virus/*metabolism
Cell Line ; G2 Phase Cell Cycle Checkpoints ; High-Throughput Nucleotide Sequencing ; Humans ; Phosphorylation ; Protein Binding ; Reproducibility of Results ; Static Electricity ; Substrate Specificity
Czasopismo naukowe
Tytuł :
Apigetrin induces extrinsic apoptosis, autophagy and G2/M phase cell cycle arrest through PI3K/AKT/mTOR pathway in AGS human gastric cancer cell.
Autorzy :
Kim SM; Research Institute of Life science and College of Veterinary Medicine, Gyeongsang National University, Gazwa, Jinju, 52828, Republic, Republic of Korea. Electronic address: .
Vetrivel P; Research Institute of Life science and College of Veterinary Medicine, Gyeongsang National University, Gazwa, Jinju, 52828, Republic, Republic of Korea. Electronic address: .
Ha SE; Research Institute of Life science and College of Veterinary Medicine, Gyeongsang National University, Gazwa, Jinju, 52828, Republic, Republic of Korea. Electronic address: .
Kim HH; Research Institute of Life science and College of Veterinary Medicine, Gyeongsang National University, Gazwa, Jinju, 52828, Republic, Republic of Korea. Electronic address: .
Kim JA; Department of Physical Therapy, International University of Korea, Jinju, 52833, Republic of Korea. Electronic address: .
Kim GS; Research Institute of Life science and College of Veterinary Medicine, Gyeongsang National University, Gazwa, Jinju, 52828, Republic, Republic of Korea. Electronic address: .
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Źródło :
The Journal of nutritional biochemistry [J Nutr Biochem] 2020 Sep; Vol. 83, pp. 108427. Date of Electronic Publication: 2020 May 21.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apigenin/*pharmacology
Apoptosis/*drug effects
Autophagy/*drug effects
M Phase Cell Cycle Checkpoints/*drug effects
Phosphatidylinositol 3-Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Stomach Neoplasms/*physiopathology
TOR Serine-Threonine Kinases/*metabolism
Cell Line, Tumor ; Cell Proliferation/drug effects ; G2 Phase Cell Cycle Checkpoints/drug effects ; Humans ; Phosphatidylinositol 3-Kinases/genetics ; Proto-Oncogene Proteins c-akt/genetics ; Stomach Neoplasms/drug therapy ; Stomach Neoplasms/genetics ; Stomach Neoplasms/metabolism ; TOR Serine-Threonine Kinases/genetics
Czasopismo naukowe
Tytuł :
Induction of different cellular arrest and molecular responses in low EGFR expressing A549 and high EGFR expressing A431 tumor cells treated with various doses of Lu-Nimotuzumab.
Autorzy :
Suman S; Radiopharmaceuticals Division, Bhabha Atomic Research Centre, Mumbai, India.
Priya R; Radiation Biology and Health Sciences Division, Bhabha Atomic Research Centre, Mumbai, India.
Kameswaran M; Radiopharmaceuticals Division, Bhabha Atomic Research Centre, Mumbai, India.
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Źródło :
International journal of radiation biology [Int J Radiat Biol] 2020 Sep; Vol. 96 (9), pp. 1144-1156. Date of Electronic Publication: 2020 Jul 23.
Typ publikacji :
Journal Article
MeSH Terms :
Antibodies, Monoclonal, Humanized/*therapeutic use
Cell Cycle Checkpoints/*radiation effects
Gene Expression Regulation, Neoplastic/*radiation effects
Lutetium/*therapeutic use
Radioisotopes/*therapeutic use
A549 Cells ; Apoptosis/radiation effects ; Cell Proliferation/radiation effects ; Dose-Response Relationship, Radiation ; ErbB Receptors/metabolism ; G2 Phase Cell Cycle Checkpoints/radiation effects ; Humans
Czasopismo naukowe
Tytuł :
Xanthatin mediates G 2 /M cell cycle arrest, autophagy and apoptosis via ROS/XIAP signaling in human colon cancer cells.
Autorzy :
Geng YD; Department of Pharmacy, Anhui Provincial Hospital, Anhui Medical University, Hefei, Anhui, China.
Zhang L; Department of Pharmacy, Anhui Provincial Hospital, Anhui Medical University, Hefei, Anhui, China.
Wang GY; Department of Pharmacy, Anhui Provincial Hospital, Anhui Medical University, Hefei, Anhui, China.
Feng XJ; Department of Pharmacy, Anhui Provincial Hospital, Anhui Medical University, Hefei, Anhui, China.
Chen ZL; Department of Pharmacy, Anhui Provincial Hospital, Anhui Medical University, Hefei, Anhui, China.
Jiang L; Department of Pharmacy, Anhui Provincial Hospital, Anhui Medical University, Hefei, Anhui, China.
Shen AZ; Department of Pharmacy, Anhui Provincial Hospital, Anhui Medical University, Hefei, Anhui, China.
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Źródło :
Natural product research [Nat Prod Res] 2020 Sep; Vol. 34 (18), pp. 2616-2620. Date of Electronic Publication: 2018 Dec 27.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Autophagy/*drug effects
Cell Cycle Checkpoints/*drug effects
Colonic Neoplasms/*drug therapy
Furans/*therapeutic use
Signal Transduction/*drug effects
Antineoplastic Agents, Phytogenic/isolation & purification ; Antineoplastic Agents, Phytogenic/pharmacology ; Asteraceae/chemistry ; Cell Line, Tumor ; Colonic Neoplasms/metabolism ; Colonic Neoplasms/pathology ; Furans/isolation & purification ; Furans/pharmacology ; G2 Phase Cell Cycle Checkpoints ; Humans ; Reactive Oxygen Species/metabolism ; X-Linked Inhibitor of Apoptosis Protein/metabolism ; Xanthium/chemistry
Czasopismo naukowe
Tytuł :
CDC6 regulates both G2/M transition and metaphase-to-anaphase transition during the first meiosis of mouse oocytes.
Autorzy :
Yi ZY; The Reproductive Medicine Center, Peking University Shenzhen Hospital, Shenzhen Peking University-The Hong Kong University of Science and Technology Medical Center, Shenzhen, China.
Meng TG; State Key Laboratory of Stem Cell and Reproductive Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.
Ma XS; State Key Laboratory of Stem Cell and Reproductive Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.
Li J; The Reproductive Medicine Center, Peking University Shenzhen Hospital, Shenzhen Peking University-The Hong Kong University of Science and Technology Medical Center, Shenzhen, China.
Zhang CH; The Reproductive Medicine Center, Peking University Shenzhen Hospital, Shenzhen Peking University-The Hong Kong University of Science and Technology Medical Center, Shenzhen, China.
Ouyang YC; State Key Laboratory of Stem Cell and Reproductive Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.
Schatten H; Department of Veterinary Pathobiology, University of Missouri, Columbia, Missouri.
Qiao J; Reproductive Medical Center, Peking University Third Hospital, Beijing, China.
Sun QY; State Key Laboratory of Stem Cell and Reproductive Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.
Qian WP; The Reproductive Medicine Center, Peking University Shenzhen Hospital, Shenzhen Peking University-The Hong Kong University of Science and Technology Medical Center, Shenzhen, China.
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Źródło :
Journal of cellular physiology [J Cell Physiol] 2020 Jul; Vol. 235 (7-8), pp. 5541-5554. Date of Electronic Publication: 2020 Jan 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Cycle Proteins/*genetics
G2 Phase Cell Cycle Checkpoints/*genetics
Meiosis/*genetics
Nuclear Proteins/*genetics
Oocytes/*growth & development
Anaphase/genetics ; Animals ; Centrosome ; Female ; M Phase Cell Cycle Checkpoints/genetics ; Metaphase/genetics ; Mice ; Oocytes/metabolism ; Spindle Apparatus/genetics
Czasopismo naukowe
Tytuł :
Resveratrol targets TyrRS acetylation to protect against radiation-induced damage.
Autorzy :
Gao P; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Li N; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Ji K; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Wang Y; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Xu C; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Liu Y; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Wang Q; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Wang J; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
He N; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Sun Z; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Du L; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
Liu Q; Tianjin Key Laboratory of Radiation Medicine and Molecular Nuclear Medicine, Institute of Radiation Medicine, Chinese Academy of Medical Sciences-Peking Union Medical College, Tianjin, China.
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Źródło :
FASEB journal : official publication of the Federation of American Societies for Experimental Biology [FASEB J] 2019 Jul; Vol. 33 (7), pp. 8083-8093. Date of Electronic Publication: 2019 Apr 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*/drug effects
Apoptosis*/genetics
Apoptosis*/radiation effects
G2 Phase Cell Cycle Checkpoints*/drug effects
G2 Phase Cell Cycle Checkpoints*/genetics
G2 Phase Cell Cycle Checkpoints*/radiation effects
Radiation Injuries, Experimental*/genetics
Radiation Injuries, Experimental*/metabolism
Radiation Injuries, Experimental*/pathology
Radiation Injuries, Experimental*/prevention & control
Signal Transduction*/drug effects
Signal Transduction*/genetics
Signal Transduction*/radiation effects
Tyrosine-tRNA Ligase*/genetics
Tyrosine-tRNA Ligase*/metabolism
Resveratrol/*pharmacology
Animals ; DNA Damage ; DNA Repair/drug effects ; DNA Repair/genetics ; DNA Repair/radiation effects ; HEK293 Cells ; Humans ; M Phase Cell Cycle Checkpoints/drug effects ; M Phase Cell Cycle Checkpoints/genetics ; M Phase Cell Cycle Checkpoints/radiation effects ; Sirtuin 1/genetics ; Sirtuin 1/metabolism
Czasopismo naukowe
Tytuł :
Norwogonin flavone suppresses the growth of human colon cancer cells via mitochondrial mediated apoptosis, autophagy induction and triggering G2/M phase cell cycle arrest.
Autorzy :
Wang Z; Department of Gastrointestinal Surgery, the Central Hospital of Enshi Tujia and Miao Autonomous Prefecture, Enshi, Hubei 445000, China.
Zhang Q
Zhou L
Liu G
Wu Q
Chen C
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Źródło :
Journal of B.U.ON. : official journal of the Balkan Union of Oncology [J BUON] 2020 May-Jun; Vol. 25 (3), pp. 1449-1454.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Autophagy/*drug effects
Cell Cycle Checkpoints/*drug effects
Cell Proliferation/*drug effects
Colonic Neoplasms/*drug therapy
Flavones/*pharmacology
Mitochondria/*drug effects
Cell Line, Tumor ; Colonic Neoplasms/metabolism ; G2 Phase Cell Cycle Checkpoints/drug effects ; Humans ; M Phase Cell Cycle Checkpoints/drug effects ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Signal Transduction/drug effects ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
CAMK2N1 suppresses hepatoma growth through inhibiting E2F1-mediated cell-cycle signaling.
Autorzy :
Peng JM; Department of Gastroenterology and Hepatology, Chang Gung Memorial Hospital, Taoyuan, 333, Taiwan; Institute for Translational Research in Biomedicine, Kaohsiung Chang Gung Memorial Hospital, Kaohsiung, Taiwan.
Tseng RH; Department of Gastroenterology and Hepatology, Chang Gung Memorial Hospital, Taoyuan, 333, Taiwan.
Shih TC; Department of Gastroenterology and Hepatology, Chang Gung Memorial Hospital, Taoyuan, 333, Taiwan.
Hsieh SY; Department of Gastroenterology and Hepatology, Chang Gung Memorial Hospital, Taoyuan, 333, Taiwan; Chang Gung University, Institute of Biomedical Sciences, College of Medicine, Taoyuan, 333, Taiwan. Electronic address: .
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Źródło :
Cancer letters [Cancer Lett] 2021 Jan 28; Vol. 497, pp. 66-76. Date of Electronic Publication: 2020 Oct 15.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Cycle Checkpoints*
Gene Expression Regulation, Neoplastic*
Biomarkers, Tumor/*metabolism
Carcinoma, Hepatocellular/*prevention & control
E2F1 Transcription Factor/*antagonists & inhibitors
Liver Neoplasms/*prevention & control
Proteins/*antagonists & inhibitors
Animals ; Apoptosis ; Biomarkers, Tumor/genetics ; Carcinoma, Hepatocellular/genetics ; Carcinoma, Hepatocellular/metabolism ; Carcinoma, Hepatocellular/pathology ; Cell Proliferation ; E2F1 Transcription Factor/genetics ; E2F1 Transcription Factor/metabolism ; Humans ; Liver Neoplasms/genetics ; Liver Neoplasms/metabolism ; Liver Neoplasms/pathology ; Mice ; Mice, Nude ; Prognosis ; Proteins/genetics ; Proteins/metabolism ; Survival Rate ; Tumor Cells, Cultured ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Hyperthermia induced disruption of mechanical balance leads to G1 arrest and senescence in cells.
Autorzy :
Mundhara N; Department of Biosciences and Bioengineering, Indian Institute of Technology Bombay, Mumbai 400076, India.
Majumder A; Department of Chemical Engineering, Indian Institute of Technology Bombay, Mumbai 400076, India.
Panda D; Department of Biosciences and Bioengineering, Indian Institute of Technology Bombay, Mumbai 400076, India.
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Źródło :
The Biochemical journal [Biochem J] 2021 Jan 15; Vol. 478 (1), pp. 179-196.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
G1 Phase Cell Cycle Checkpoints*/genetics
G1 Phase Cell Cycle Checkpoints*/physiology
Cellular Senescence/*physiology
Cytoskeleton/*metabolism
Histone Deacetylase 6/*metabolism
Hyperthermia/*metabolism
Stress Fibers/*metabolism
Acetylation ; Actins/metabolism ; Apoptosis/physiology ; Cell Adhesion/physiology ; Cell Division/physiology ; Cell Line, Tumor ; Cell Proliferation/physiology ; Cell Shape ; Cell Size ; HSP70 Heat-Shock Proteins/metabolism ; Histone Deacetylase 6/genetics ; Humans ; Hyperthermia/genetics ; Membrane Potential, Mitochondrial/physiology ; Microtubules/metabolism ; Reactive Oxygen Species/metabolism ; Up-Regulation
Czasopismo naukowe
Tytuł :
Discriminative SKP2 Interactions with CDK-Cyclin Complexes Support a Cyclin A-Specific Role in p27KIP1 Degradation.
Autorzy :
Salamina M; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Montefiore BC; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Liu M; Department of Biochemistry and Molecular Pharmacology, Perlmutter NYU Cancer Center, New York University Grossman School of Medicine, and Howard Hughes Medical Institute, The Alexandria Center of Life Science, East Tower, 450 E, 29th Street, New York, NY 10016, USA.
Wood DJ; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Heath R; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Ault JR; Astbury Centre for Structural Molecular Biology, School of Molecular and Cellular Biology, University of Leeds, Leeds LS2 9JT, UK.
Wang LZ; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Korolchuk S; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Baslé A; Biosciences Institute, Newcastle University, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Pastok MW; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Reeks J; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Tatum NJ; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Sobott F; Astbury Centre for Structural Molecular Biology, School of Molecular and Cellular Biology, University of Leeds, Leeds LS2 9JT, UK.
Arold ST; Division of Biological and Environmental Sciences and Engineering (BESE), Computational Bioscience Research Center (CBRC), King Abdullah University of Science and Technology (KAUST), Thuwal 23955-6900, Saudi Arabia; Centre de Biochimie Structurale, CNRS, INSERM, Université de Montpellier, 34090 Montpellier, France.
Pagano M; Department of Biochemistry and Molecular Pharmacology, Perlmutter NYU Cancer Center, New York University Grossman School of Medicine, and Howard Hughes Medical Institute, The Alexandria Center of Life Science, East Tower, 450 E, 29th Street, New York, NY 10016, USA.
Noble MEM; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK.
Endicott JA; Newcastle University Centre for Cancer, Translational and Clinical Research Institute, Newcastle University, Paul O'Gorman Building, Framlington Place, Newcastle upon Tyne NE2 4HH, UK. Electronic address: .
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Źródło :
Journal of molecular biology [J Mol Biol] 2021 Mar 05; Vol. 433 (5), pp. 166795. Date of Electronic Publication: 2021 Jan 07.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
G1 Phase Cell Cycle Checkpoints*
Cyclin A/*chemistry
Cyclin-Dependent Kinase 2/*chemistry
Cyclin-Dependent Kinase Inhibitor p27/*chemistry
S-Phase Kinase-Associated Proteins/*chemistry
Binding Sites ; CDC2-CDC28 Kinases/chemistry ; CDC2-CDC28 Kinases/genetics ; CDC2-CDC28 Kinases/metabolism ; Cyclin A/genetics ; Cyclin A/metabolism ; Cyclin E/chemistry ; Cyclin E/genetics ; Cyclin E/metabolism ; Cyclin-Dependent Kinase 2/genetics ; Cyclin-Dependent Kinase 2/metabolism ; Cyclin-Dependent Kinase Inhibitor p27/genetics ; Cyclin-Dependent Kinase Inhibitor p27/metabolism ; Gene Expression Regulation ; HEK293 Cells ; Humans ; Protein Binding ; Protein Conformation, alpha-Helical ; Protein Conformation, beta-Strand ; Protein Interaction Domains and Motifs ; Proteolysis ; Recombinant Proteins/chemistry ; Recombinant Proteins/genetics ; Recombinant Proteins/metabolism ; S-Phase Kinase-Associated Proteins/genetics ; S-Phase Kinase-Associated Proteins/metabolism ; Saccharomyces cerevisiae/genetics ; Saccharomyces cerevisiae/metabolism ; Signal Transduction
Czasopismo naukowe
Tytuł :
Betulinic Acid Restricts Human Bladder Cancer Cell Proliferation In Vitro by Inducing Caspase-Dependent Cell Death and Cell Cycle Arrest, and Decreasing Metastatic Potential.
Autorzy :
Kim SY; Anti-Aging Research Center, Dong-eui University, Busan 47340, Korea.; Department of Molecular Biology, Pusan National University, Busan 46241, Korea.; Department of Biochemistry, College of Korean Medicine, Dong-eui University, Busan 47227, Korea.
Hwangbo H; Anti-Aging Research Center, Dong-eui University, Busan 47340, Korea.; Department of Molecular Biology, Pusan National University, Busan 46241, Korea.; Department of Biochemistry, College of Korean Medicine, Dong-eui University, Busan 47227, Korea.
Kim MY; Anti-Aging Research Center, Dong-eui University, Busan 47340, Korea.; Department of Biochemistry, College of Korean Medicine, Dong-eui University, Busan 47227, Korea.
Ji SY; Anti-Aging Research Center, Dong-eui University, Busan 47340, Korea.; Department of Biochemistry, College of Korean Medicine, Dong-eui University, Busan 47227, Korea.
Kim DH; Anti-Aging Research Center, Dong-eui University, Busan 47340, Korea.; Department of Biochemistry, College of Korean Medicine, Dong-eui University, Busan 47227, Korea.
Lee H; Anti-Aging Research Center, Dong-eui University, Busan 47340, Korea.; Department of Biochemistry, College of Korean Medicine, Dong-eui University, Busan 47227, Korea.
Kim GY; Department of Marine Life Sciences, School of Marine Biomedical Sciences, Jeju National University, Jeju 63243, Korea.
Moon SK; Department of Food and Nutrition, Chung-Ang University, Anseong 17546, Korea.
Leem SH; Department of Biomedical Science, Dong-A University, Busan 49315, Korea.; Department of Health Sciences, Dong-A University, Busan 49315, Korea.
Yun SJ; Department of Urology, College of Medicine, Chungbuk National University, Cheongju 28644, Korea.
Kim WJ; Department of Urology, College of Medicine, Chungbuk National University, Cheongju 28644, Korea.
Cheong J; Department of Molecular Biology, Pusan National University, Busan 46241, Korea.
Park C; Division of Basic Sciences, College of Liberal Studies, Dong-Eui University, Busan 47340, Korea.
Choi YH; Anti-Aging Research Center, Dong-eui University, Busan 47340, Korea.; Department of Biochemistry, College of Korean Medicine, Dong-eui University, Busan 47227, Korea.
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Źródło :
Molecules (Basel, Switzerland) [Molecules] 2021 Mar 04; Vol. 26 (5). Date of Electronic Publication: 2021 Mar 04.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Cycle Checkpoints*
Cell Movement*
Antineoplastic Agents, Phytogenic/*pharmacology
Caspase 3/*metabolism
Pentacyclic Triterpenes/*pharmacology
Urinary Bladder Neoplasms/*drug therapy
Apoptosis ; Caspase 3/genetics ; Cell Proliferation ; Humans ; In Vitro Techniques ; Neoplasm Metastasis ; Reactive Oxygen Species ; Tumor Cells, Cultured ; Urinary Bladder Neoplasms/metabolism ; Urinary Bladder Neoplasms/pathology
Czasopismo naukowe
Tytuł :
Disruption of p97/VCP induces autophagosome accumulation, cell cycle arrest and apoptosis in human choriocarcinoma cells.
Autorzy :
Desdicioglu R; Department of Obstetrics and Gynecology, Ankara Yıldırım Beyazıt University, Ankara, Turkey.
Sahin C; Department of Histology and Embryology, Ankara Yıldırım Beyazıt University, Ankara, Turkey.
Yavuz F; Department of Obstetrics and Gynecology, Ankara Yıldırım Beyazıt University, Ankara, Turkey.
Cayli S; Department of Histology and Embryology, Ankara Yıldırım Beyazıt University, Ankara, Turkey. .
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Źródło :
Molecular biology reports [Mol Biol Rep] 2021 Mar; Vol. 48 (3), pp. 2163-2171. Date of Electronic Publication: 2021 Feb 23.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*/drug effects
Cell Cycle Checkpoints*/drug effects
Autophagosomes/*metabolism
Choriocarcinoma/*metabolism
Choriocarcinoma/*pathology
Valosin Containing Protein/*metabolism
Autophagosomes/drug effects ; Autophagy/drug effects ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; G1 Phase/drug effects ; Humans ; Microtubule-Associated Proteins/metabolism ; Polyubiquitin/metabolism ; Quinazolines/pharmacology ; RNA, Small Interfering/metabolism ; Resting Phase, Cell Cycle/drug effects ; Sequestosome-1 Protein/metabolism ; Ubiquitination/drug effects
Czasopismo naukowe
Tytuł :
The regulatory roles of p53 in cardiovascular health and disease.
Autorzy :
Men H; Department of Cardiovascular Disease, First Hospital of Jilin University, Jilin University, Changchun, 130021, China.; Department of Pediatrics, Pediatric Research Institute, University of Louisville, Louisville, KY, 40202, USA.
Cai H; Department of Cardiovascular Disease, First Hospital of Jilin University, Jilin University, Changchun, 130021, China.
Cheng Q; Department of Cardiovascular Disease, First Hospital of Jilin University, Jilin University, Changchun, 130021, China.
Zhou W; Department of Cardiovascular Disease, First Hospital of Jilin University, Jilin University, Changchun, 130021, China.; Department of Pediatrics, Pediatric Research Institute, University of Louisville, Louisville, KY, 40202, USA.
Wang X; Department of Cardiovascular Disease, First Hospital of Jilin University, Jilin University, Changchun, 130021, China.; Department of Pediatrics, Pediatric Research Institute, University of Louisville, Louisville, KY, 40202, USA.
Huang S; Department of Cardiovascular Disease, First Hospital of Jilin University, Jilin University, Changchun, 130021, China.; Department of Pediatrics, Pediatric Research Institute, University of Louisville, Louisville, KY, 40202, USA.
Zheng Y; Department of Cardiovascular Disease, First Hospital of Jilin University, Jilin University, Changchun, 130021, China. .
Cai L; Department of Pediatrics, Pediatric Research Institute, University of Louisville, Louisville, KY, 40202, USA. .; Department of Pharmacology and Toxicology, University of Louisville, Louisville, KY, 40202, USA. .
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Źródło :
Cellular and molecular life sciences : CMLS [Cell Mol Life Sci] 2021 Mar; Vol. 78 (5), pp. 2001-2018. Date of Electronic Publication: 2020 Nov 11.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Cell Cycle Checkpoints/*physiology
Diabetic Cardiomyopathies/*metabolism
Myocytes, Cardiac/*metabolism
Signal Transduction/*physiology
Tumor Suppressor Protein p53/*metabolism
Animals ; Cell Cycle Checkpoints/genetics ; Cell Proliferation/genetics ; Cellular Senescence/genetics ; Diabetic Cardiomyopathies/genetics ; Gene Expression Regulation ; Humans ; Myocytes, Cardiac/cytology ; Signal Transduction/genetics ; Tumor Suppressor Protein p53/genetics
Czasopismo naukowe

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