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Tytuł:
Loss of the TRPM4 channel in humans causes immune dysregulation with defective monocyte migration.
Autorzy:
Yu F; Calcium Signaling Group, Research Department, Weill Cornell Medicine-Qatar, Doha, Qatar; Department of Physiology and Biophysics, Weill Cornell Medicine, New York, NY.
Hubrack S; Research Department, Sidra Medicine, Doha, Qatar.
Raynaud CM; Research Department, Sidra Medicine, Doha, Qatar.
Elmi A; Research Department, Sidra Medicine, Doha, Qatar.
Mackeh R; Research Department, Sidra Medicine, Doha, Qatar.
Agrebi N; Research Department, Sidra Medicine, Doha, Qatar.
Thareja G; Department of Physiology and Biophysics, Weill Cornell Medicine, New York, NY; Research Department, Weill Cornell Medicine-Qatar, Doha, Qatar.
Belkadi A; Department of Physiology and Biophysics, Weill Cornell Medicine, New York, NY; Research Department, Weill Cornell Medicine-Qatar, Doha, Qatar.
Al Saloos H; Division of Cardiology, Sidra Medicine, Doha, Qatar.
Ahmed AA; Genomics Core, Weill Cornell Medicine-Qatar, Doha, Qatar.
Purayil SC; Allergy & Immunology Division, Department of Medicine, Hamad Medical Corporation, Doha, Qatar.
Mohamoud YA; Genomics Core, Weill Cornell Medicine-Qatar, Doha, Qatar.
Suhre K; Department of Physiology and Biophysics, Weill Cornell Medicine, New York, NY; Research Department, Weill Cornell Medicine-Qatar, Doha, Qatar.
Abi Khalil C; Research Department, Weill Cornell Medicine-Qatar, Doha, Qatar; Heart Hospital, Hamad Medical Corporation, Doha, Qatar.
Schmidt F; Research Department, Sidra Medicine, Doha, Qatar; Department of Biochemistry, Weill Cornell Medicine, New York, NY.
Lo B; Research Department, Sidra Medicine, Doha, Qatar; College of Health and Life Sciences, Hamad Bin Khalifa University, Doha, Qatar. Electronic address: .
Hassan A; Pediatric Allergy and Immunology Department, Sidra Medicine, Doha, Qatar. Electronic address: .
Machaca K; Calcium Signaling Group, Research Department, Weill Cornell Medicine-Qatar, Doha, Qatar; Department of Physiology and Biophysics, Weill Cornell Medicine, New York, NY. Electronic address: .
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Źródło:
The Journal of allergy and clinical immunology [J Allergy Clin Immunol] 2024 Sep; Vol. 154 (3), pp. 792-806. Date of Electronic Publication: 2024 May 14.
Typ publikacji:
Journal Article; Case Reports
MeSH Terms:
TRPM Cation Channels*/genetics
TRPM Cation Channels*/immunology
Monocytes*/immunology
Cell Movement*/genetics
Cell Movement*/immunology
Humans ; T-Lymphocytes/immunology ; Male ; Female ; THP-1 Cells
Czasopismo naukowe
Tytuł:
Effects of 630 nm laser on apoptosis, metastasis, invasion and epithelial-to-mesenchymal transition of human lung squamous cell carcinoma H520 cells mediated by hematoporphyrin derivatives.
Autorzy:
Liu T; Department of Respiratory and Critical Care Medicine, the Affiliated Hospital of Qingdao University, Qingdao, China.
Zhang E; Department of Respiratory and Critical Care Medicine, Linyi Central Hospital, Linyi, China.
Cui S; Department of Respiratory and Critical Care Medicine, the Affiliated Hospital of Qingdao University, Qingdao, China.
Dai H; Department of Respiratory and Critical Care Medicine, the Affiliated Hospital of Qingdao University, Qingdao, China.
Yang X; Department of Respiratory and Critical Care Medicine, the Affiliated Hospital of Qingdao University, Qingdao, China.
Lin C; Department of Respiratory and Critical Care Medicine, the Affiliated Hospital of Qingdao University, Qingdao, China. .
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Źródło:
Lasers in medical science [Lasers Med Sci] 2024 Aug 30; Vol. 39 (1), pp. 228. Date of Electronic Publication: 2024 Aug 30.
Typ publikacji:
Journal Article
MeSH Terms:
Epithelial-Mesenchymal Transition*/drug effects
Epithelial-Mesenchymal Transition*/radiation effects
Apoptosis*/drug effects
Apoptosis*/radiation effects
Lung Neoplasms*/pathology
Lung Neoplasms*/radiotherapy
Lung Neoplasms*/drug therapy
Photochemotherapy*/methods
Carcinoma, Squamous Cell*/pathology
Carcinoma, Squamous Cell*/therapy
Carcinoma, Squamous Cell*/radiotherapy
Carcinoma, Squamous Cell*/drug therapy
Cell Movement*/drug effects
Cell Movement*/radiation effects
Cell Proliferation*/drug effects
Cell Proliferation*/radiation effects
Neoplasm Invasiveness*
Humans ; Cell Line, Tumor ; Photosensitizing Agents/pharmacology ; Photosensitizing Agents/therapeutic use ; Hematoporphyrin Derivative/pharmacology ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Proto-Oncogene Proteins c-bcl-2/genetics ; Cadherins/metabolism ; Vimentin/metabolism ; Caspase 9/metabolism ; Caspase 9/genetics
Czasopismo naukowe
Tytuł:
Role of miR-93-5p and Its Opposing Effect of Ionizing Radiation in Non-Small Cell Lung Cancer.
Autorzy:
Ni Q; Department of Oncology Jiangsu Taizhou People's Hospital, The Affiliated Taizhou People's Hospital of Nanjing Medical University Taizhou School of Clinical Medicine Nanjing Medical University, Taizhou 225300, China.
Sang K; Department of General Surgery Jiangsu Taizhou People's Hospital, The Affiliated Taizhou People's Hospital of Nanjing Medical University Taizhou School of Clinical Medicine Nanjing Medical University, Taizhou 225300, China.
Zhou J; Department of General Surgery Jiangsu Taizhou People's Hospital, The Affiliated Taizhou People's Hospital of Nanjing Medical University Taizhou School of Clinical Medicine Nanjing Medical University, Taizhou 225300, China.
Pan C; Department of General Surgery Jiangsu Taizhou People's Hospital, The Affiliated Taizhou People's Hospital of Nanjing Medical University Taizhou School of Clinical Medicine Nanjing Medical University, Taizhou 225300, China.
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Źródło:
Analytical cellular pathology (Amsterdam) [Anal Cell Pathol (Amst)] 2024 Aug 10; Vol. 2024, pp. 4218464. Date of Electronic Publication: 2024 Aug 10 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
MicroRNAs*/genetics
MicroRNAs*/metabolism
Lung Neoplasms*/radiotherapy
Lung Neoplasms*/genetics
Lung Neoplasms*/pathology
Cell Movement*/radiation effects
Cell Movement*/genetics
Carcinoma, Non-Small-Cell Lung*/radiotherapy
Carcinoma, Non-Small-Cell Lung*/genetics
Carcinoma, Non-Small-Cell Lung*/pathology
Carcinoma, Non-Small-Cell Lung*/metabolism
Radiation, Ionizing*
Cell Proliferation*/radiation effects
Cell Proliferation*/genetics
Apoptosis*/radiation effects
Apoptosis*/genetics
Gene Expression Regulation, Neoplastic*/radiation effects
Humans ; A549 Cells
Czasopismo naukowe
Tytuł:
Plasma membrane SK2 channel activity regulates migration and chemosensitivity of high-grade serous ovarian cancer cells.
Autorzy:
Romito O; Inserm UMR 1069, Nutrition Croissance Cancer, Faculté de Médecine, Université de Tours, Tours, France.; Réseau Molécules Marines, Métabolisme et Cancer and Réseau CasTHOR Cancéropôle Grand Ouest, Tours, France.
Lemettre A; Inserm UMR 1069, Nutrition Croissance Cancer, Faculté de Médecine, Université de Tours, Tours, France.; Réseau Molécules Marines, Métabolisme et Cancer and Réseau CasTHOR Cancéropôle Grand Ouest, Tours, France.
Chantôme A; Inserm UMR 1069, Nutrition Croissance Cancer, Faculté de Médecine, Université de Tours, Tours, France.; Réseau Molécules Marines, Métabolisme et Cancer and Réseau CasTHOR Cancéropôle Grand Ouest, Tours, France.
Champion O; Inserm UMR 1069, Nutrition Croissance Cancer, Faculté de Médecine, Université de Tours, Tours, France.; Réseau Molécules Marines, Métabolisme et Cancer and Réseau CasTHOR Cancéropôle Grand Ouest, Tours, France.
Couty N; Inserm UMR 1069, Nutrition Croissance Cancer, Faculté de Médecine, Université de Tours, Tours, France.; Réseau Molécules Marines, Métabolisme et Cancer and Réseau CasTHOR Cancéropôle Grand Ouest, Tours, France.
Ouldamer L; Inserm UMR 1069, Nutrition Croissance Cancer, Faculté de Médecine, Université de Tours, Tours, France.; Réseau Molécules Marines, Métabolisme et Cancer and Réseau CasTHOR Cancéropôle Grand Ouest, Tours, France.; CHRU de Tours, Service de Gynécologie et d'Obstétrique, Tours, France.
Hempel N; UPMC Hillman Cancer Center, Division of Hematology & Oncology, Department of Medicine, University of Pittsburgh, PA, USA.
Trebak M; Department of Pharmacology and Chemical Biology, Vascular Medicine Institute, University of Pittsburgh, PA, USA.
Goupille C; Inserm UMR 1069, Nutrition Croissance Cancer, Faculté de Médecine, Université de Tours, Tours, France.; Réseau Molécules Marines, Métabolisme et Cancer and Réseau CasTHOR Cancéropôle Grand Ouest, Tours, France.; CHRU de Tours, Service de Gynécologie et d'Obstétrique, Tours, France.
Potier-Cartereau M; Inserm UMR 1069, Nutrition Croissance Cancer, Faculté de Médecine, Université de Tours, Tours, France.; Réseau Molécules Marines, Métabolisme et Cancer and Réseau CasTHOR Cancéropôle Grand Ouest, Tours, France.
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Źródło:
Molecular oncology [Mol Oncol] 2024 Aug; Vol. 18 (8), pp. 1853-1865. Date of Electronic Publication: 2024 Mar 13.
Typ publikacji:
Journal Article
MeSH Terms:
Ovarian Neoplasms*/pathology
Ovarian Neoplasms*/metabolism
Ovarian Neoplasms*/genetics
Ovarian Neoplasms*/drug therapy
Cell Movement*/drug effects
Cell Movement*/genetics
Small-Conductance Calcium-Activated Potassium Channels*/metabolism
Small-Conductance Calcium-Activated Potassium Channels*/genetics
Cell Membrane*/metabolism
Humans ; Female ; Cell Line, Tumor ; Drug Resistance, Neoplasm/genetics ; Lysophospholipids/metabolism ; Calcium/metabolism
Czasopismo naukowe
Tytuł:
Silencing MFN2 Drives WNT/β-catenin Nucleation to Reduce Sorafenib Sensitivity in Hepatocellular Carcinoma Cells.
Autorzy:
Zeng CM; Department of Geriatric Medicine, Shengli Clinical Medical College of Fujian Medical University, Fujian Key Laboratory of Geriatrics Diseases, Fujian Provincial Center for Geriatrics, Fujian Provincial Hospital, Fuzhou, 350001, China.
Shao B; Department of Rehabilitation, Shengli Clinical Medical College of Fujian Medical University; Fujian Provincial Hospital, Fuzhou, 350001, China.
Chen YP; Department of Gynecology, Shengli Clinical Medical College of Fujian Medical University; Fujian Provincial Hospital, Fuzhou, 350001, China.
Ding GS; Department of Ultrasonography, Shengli Clinical Medical College of Fujian Medical University; Fujian Provincial Hospital, Fuzhou, 350001, China. .
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Źródło:
Current medical science [Curr Med Sci] 2024 Aug; Vol. 44 (4), pp. 789-798. Date of Electronic Publication: 2024 Jun 27.
Typ publikacji:
Journal Article
MeSH Terms:
Sorafenib*/pharmacology
Carcinoma, Hepatocellular*/genetics
Carcinoma, Hepatocellular*/drug therapy
Carcinoma, Hepatocellular*/metabolism
Carcinoma, Hepatocellular*/pathology
Liver Neoplasms*/genetics
Liver Neoplasms*/drug therapy
Liver Neoplasms*/metabolism
Liver Neoplasms*/pathology
GTP Phosphohydrolases*/genetics
GTP Phosphohydrolases*/metabolism
beta Catenin*/metabolism
beta Catenin*/genetics
Wnt Signaling Pathway*/drug effects
Wnt Signaling Pathway*/genetics
Cell Proliferation*/drug effects
Cell Movement*/drug effects
Cell Movement*/genetics
Drug Resistance, Neoplasm*/genetics
Drug Resistance, Neoplasm*/drug effects
Humans ; Cell Line, Tumor ; Gene Silencing ; Antineoplastic Agents/pharmacology ; Mitochondrial Proteins/genetics ; Mitochondrial Proteins/metabolism ; Epithelial-Mesenchymal Transition/drug effects ; Epithelial-Mesenchymal Transition/genetics ; Apoptosis/drug effects ; Apoptosis/genetics ; Gene Expression Regulation, Neoplastic/drug effects
Czasopismo naukowe
Tytuł:
SLC4A4 as a novel biomarker involved in immune system response and lung adenocarcinoma progression.
Autorzy:
Quan S; Department of Thoracic Surgery, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan 250021, China.
Li N; Nephrology Department, Jinan Zhangqiu District People's Hospital, Jinan 250200, China.
Lian S; Out-patient Department, Zaozhuang Municipal Hospital, Zaozhuang 277102, China.
Wang Y; The Department of Hospital Infection, Jinan Fifth People's Hospital, Jinan 250022, China.
Liu Y; Thoracic Surgery, PLA 80th Group Army Hospital, Weifang 261011, China.
Liu J; Department of Thoracic Surgery, The Fourth People's Hospital of Heze, Heze 274100, China.
Zhang Z; Department of Thoracic Surgery, Gaoqing County People's Hospital, Gaoqing 256399, China.
Gao D; Department of Thoracic Surgery, The Second People's Hospital of Liaocheng, Liaocheng 252600, China.
Li Y; Department of Thoracic Surgery, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan 250021, China. Electronic address: .
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Źródło:
International immunopharmacology [Int Immunopharmacol] 2024 Oct 25; Vol. 140, pp. 112756. Date of Electronic Publication: 2024 Jul 30.
Typ publikacji:
Journal Article
MeSH Terms:
Lung Neoplasms*/immunology
Lung Neoplasms*/genetics
Lung Neoplasms*/pathology
Lung Neoplasms*/mortality
Biomarkers, Tumor*/genetics
Biomarkers, Tumor*/metabolism
Adenocarcinoma of Lung*/immunology
Adenocarcinoma of Lung*/genetics
Adenocarcinoma of Lung*/pathology
Adenocarcinoma of Lung*/mortality
Tumor Microenvironment*/immunology
Cell Movement*
Humans ; Animals ; Cell Line, Tumor ; Mice ; Prognosis ; Gene Expression Regulation, Neoplastic ; Epithelial-Mesenchymal Transition ; Female ; Mice, Nude ; Male ; Disease Progression ; Apoptosis ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł:
Fucoxanthin Attenuates Angiogenesis by Blocking the VEGFR2-Mediated Signaling Pathway through Binding the Vascular Endothelial Growth Factor.
Autorzy:
Guo GX; College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.
Qiu YH; College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.; Nansha-South China Agricultural University Fishery Research Institute, Guangzhou 511464, China.
Liu Y; College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.
Yu LL; College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.
Zhang X; College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.; Joint Laboratory of Guangdong Province and Hong Kong Region on Marine Bioresource Conservation and Exploitation, College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.
Tsim KW; Nansha-South China Agricultural University Fishery Research Institute, Guangzhou 511464, China.; Joint Laboratory of Guangdong Province and Hong Kong Region on Marine Bioresource Conservation and Exploitation, College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.; Division of Life Science and Center for Chinese Medicine, The Hong Kong University of Science and Technology, Hong Kong 999077, China.
Qin QW; College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.; Nansha-South China Agricultural University Fishery Research Institute, Guangzhou 511464, China.; Joint Laboratory of Guangdong Province and Hong Kong Region on Marine Bioresource Conservation and Exploitation, College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.
Hu WH; College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.; Nansha-South China Agricultural University Fishery Research Institute, Guangzhou 511464, China.; Joint Laboratory of Guangdong Province and Hong Kong Region on Marine Bioresource Conservation and Exploitation, College of Marine Sciences, South China Agricultural University, Guangzhou 510642, China.
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Źródło:
Journal of agricultural and food chemistry [J Agric Food Chem] 2024 Oct 02; Vol. 72 (39), pp. 21610-21623. Date of Electronic Publication: 2024 Sep 18.
Typ publikacji:
Journal Article
MeSH Terms:
Xanthophylls*/pharmacology
Xanthophylls*/chemistry
Signal Transduction*/drug effects
Human Umbilical Vein Endothelial Cells*/drug effects
Human Umbilical Vein Endothelial Cells*/metabolism
Vascular Endothelial Growth Factor A*/metabolism
Vascular Endothelial Growth Factor A*/genetics
Vascular Endothelial Growth Factor Receptor-2*/metabolism
Vascular Endothelial Growth Factor Receptor-2*/genetics
Cell Proliferation*/drug effects
Cell Movement*/drug effects
Zebrafish*
Humans ; Animals ; Angiogenesis Inhibitors/pharmacology ; Angiogenesis Inhibitors/chemistry ; Phaeophyceae/chemistry ; Neovascularization, Pathologic/drug therapy ; Neovascularization, Pathologic/metabolism ; Angiogenesis
Czasopismo naukowe
Tytuł:
The activation of asparagine synthetase by the transcription factor FOXM1 plays a pivotal role in the initiation and progression of ESCC.
Autorzy:
Lian JJ; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Li ZX; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Lin HL; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Sun MC; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Wu H; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Feng AQ; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Fang K; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Wang XY
Xu AP; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Chu Y; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Zhang L; Department of Pathology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Chen T; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
Xu MD; Endoscopy Center, Department of Gastroenterology, Shanghai East Hospital, School of Medicine, Tongji University, Shanghai, China.
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Źródło:
The journal of gene medicine [J Gene Med] 2024 Oct; Vol. 26 (10), pp. e3741.
Typ publikacji:
Journal Article
MeSH Terms:
Forkhead Box Protein M1*/metabolism
Forkhead Box Protein M1*/genetics
Aspartate-Ammonia Ligase*/genetics
Aspartate-Ammonia Ligase*/metabolism
Gene Expression Regulation, Neoplastic*
Cell Proliferation*/genetics
Cell Movement*/genetics
Disease Progression*
Esophageal Neoplasms*/genetics
Esophageal Neoplasms*/metabolism
Esophageal Neoplasms*/pathology
Esophageal Squamous Cell Carcinoma*/genetics
Esophageal Squamous Cell Carcinoma*/pathology
Esophageal Squamous Cell Carcinoma*/metabolism
Humans ; Cell Line, Tumor ; Prognosis ; Animals ; Mice ; Male ; Drug Resistance, Neoplasm/genetics ; Female ; Neoplastic Stem Cells/metabolism ; Neoplastic Stem Cells/pathology ; Carbon-Nitrogen Ligases with Glutamine as Amide-N-Donor
Czasopismo naukowe
Tytuł:
Decellularized extracellular matrix derived from dental pulp stem cells promotes gingival fibroblast adhesion and migration.
Autorzy:
Nowwarote N; Centre de Recherche des Cordeliers, Molecular Oral Pathophysiology, INSERM UMRS 1138, Université Paris Cité, Sorbonne Université, Paris, 75006, France. .; Department of Oral Biology, Dental Faculty Garancière, Université Paris Cité, Paris, 75006, France. .
Chahlaoui Z; Centre de Recherche des Cordeliers, Molecular Oral Pathophysiology, INSERM UMRS 1138, Université Paris Cité, Sorbonne Université, Paris, 75006, France.
Petit S; Centre de Recherche des Cordeliers, Molecular Oral Pathophysiology, INSERM UMRS 1138, Université Paris Cité, Sorbonne Université, Paris, 75006, France.
Duong LT; Centre de Recherche des Cordeliers, Molecular Oral Pathophysiology, INSERM UMRS 1138, Université Paris Cité, Sorbonne Université, Paris, 75006, France.
Dingli F; Centre de Recherche, CurieCoreTech Spectrométrie de Masse Protéomique, Institut Curie, PSL Research University, Paris, France.
Loew D; Centre de Recherche, CurieCoreTech Spectrométrie de Masse Protéomique, Institut Curie, PSL Research University, Paris, France.
Chansaenroj A; Center of Excellence for Dental Stem Cell Biology, Faculty of Dentistry, Chulalongkorn University, Bangkok, 10330, Thailand.
Kornsuthisopon C; Center of Excellence for Dental Stem Cell Biology, Faculty of Dentistry, Chulalongkorn University, Bangkok, 10330, Thailand. .; Department of Anatomy, Faculty of Dentistry, Chulalongkorn University, Bangkok, 10330, Thailand. .
Osathanon T; Center of Excellence for Dental Stem Cell Biology, Faculty of Dentistry, Chulalongkorn University, Bangkok, 10330, Thailand.; Department of Anatomy, Faculty of Dentistry, Chulalongkorn University, Bangkok, 10330, Thailand.
Ferre FC; Centre de Recherche des Cordeliers, Molecular Oral Pathophysiology, INSERM UMRS 1138, Université Paris Cité, Sorbonne Université, Paris, 75006, France.; Department of Oral Biology, Dental Faculty Garancière, Université Paris Cité, Paris, 75006, France.
Fournier BPJ; Centre de Recherche des Cordeliers, Molecular Oral Pathophysiology, INSERM UMRS 1138, Université Paris Cité, Sorbonne Université, Paris, 75006, France.; Department of Oral Biology, Dental Faculty Garancière, Université Paris Cité, Paris, 75006, France.
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Źródło:
BMC oral health [BMC Oral Health] 2024 Oct 01; Vol. 24 (1), pp. 1166. Date of Electronic Publication: 2024 Oct 01.
Typ publikacji:
Journal Article
MeSH Terms:
Dental Pulp*/cytology
Gingiva*/cytology
Cell Movement*
Extracellular Matrix*/metabolism
Cell Adhesion*
Fibroblasts*
Stem Cells*
Humans ; Cell Proliferation ; Cells, Cultured ; Fibronectins/metabolism
Czasopismo naukowe
Tytuł:
Ladinin-1 in actin arcs of oral squamous cell carcinoma is involved in cell migration and epithelial phenotype.
Autorzy:
Abé T; Division of Oral Pathology, Faculty of Dentistry & Graduate School of Medical and Dental Sciences, Niigata University, 2-5274 Gakkocho-dori, Chuo-ku, Niigata, 951-8514, Japan. .
Yamazaki M; Division of Oral Pathology, Faculty of Dentistry & Graduate School of Medical and Dental Sciences, Niigata University, 2-5274 Gakkocho-dori, Chuo-ku, Niigata, 951-8514, Japan.
Nozumi M; Department of Neurochemistry and Molecular Cell Biology, Graduate School of Medicine, Niigata University, Niigata, Japan.
Maruyama S; Oral Pathology Section, Department of Surgical Pathology, Niigata University Hospital, Niigata, Japan.
Takamura K; Division of Molecular and Diagnostic Pathology, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan.
Ohashi R; Division of Molecular and Diagnostic Pathology, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan.
Ajioka Y; Division of Molecular and Diagnostic Pathology, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan.
Tanuma JI; Division of Oral Pathology, Faculty of Dentistry & Graduate School of Medical and Dental Sciences, Niigata University, 2-5274 Gakkocho-dori, Chuo-ku, Niigata, 951-8514, Japan.
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Źródło:
Scientific reports [Sci Rep] 2024 Oct 01; Vol. 14 (1), pp. 22778. Date of Electronic Publication: 2024 Oct 01.
Typ publikacji:
Journal Article
MeSH Terms:
Cell Movement*
Mouth Neoplasms*/pathology
Mouth Neoplasms*/metabolism
Mouth Neoplasms*/genetics
Carcinoma, Squamous Cell*/metabolism
Carcinoma, Squamous Cell*/pathology
Carcinoma, Squamous Cell*/genetics
Actins*/metabolism
Humans ; Cell Line, Tumor ; p21-Activated Kinases/metabolism ; p21-Activated Kinases/genetics ; Vimentin/metabolism ; Vimentin/genetics ; Caveolin 1/metabolism ; Caveolin 1/genetics ; Gene Expression Regulation, Neoplastic ; Phenotype ; Epithelial Cells/metabolism ; Epithelial Cells/pathology
Czasopismo naukowe
Tytuł:
Unraveling the role of EPHA2 in regulating migration and immunomodulation processes in cervical cancer: exploring the synergic effect of 17β-estradiol on cancer progression.
Autorzy:
Mubthasima PP; Cancer and Exosome Biology Laboratory, Department of Biochemistry, CSIR- Central Food Technological Research Institute, Mysuru, 570020, India.; Academy of Scientific and Innovative Research (AcSIR), Ghaziabad, 201002, India.
Kannan A; Cancer and Exosome Biology Laboratory, Department of Biochemistry, CSIR- Central Food Technological Research Institute, Mysuru, 570020, India. .; Academy of Scientific and Innovative Research (AcSIR), Ghaziabad, 201002, India. .
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Źródło:
Medical oncology (Northwood, London, England) [Med Oncol] 2024 Oct 01; Vol. 41 (11), pp. 255. Date of Electronic Publication: 2024 Oct 01.
Typ publikacji:
Journal Article
MeSH Terms:
Receptor, EphA2*/metabolism
Estradiol*/pharmacology
Uterine Cervical Neoplasms*/pathology
Uterine Cervical Neoplasms*/metabolism
Uterine Cervical Neoplasms*/immunology
Cell Movement*/drug effects
Disease Progression*
Immunomodulation*
Humans ; Female ; Cell Proliferation/drug effects ; Cell Line, Tumor ; Exosomes/metabolism ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł:
IGFBP7 regulates cell proliferation and migration through JAK/STAT pathway in gastric cancer and is regulated by DNA and RNA methylation.
Autorzy:
Mo W; Department of General Surgery, Changzhou No.7 People's Hospital, Changzhou, China.; Department of General Surgery, Changzhou Geriatric Hospital affiliated to Soochow University, Changzhou, China.
Deng L; Department of Oncology, Changzhou No.7 People's Hospital, Changzhou, China.; Department of Oncology, Changzhou Geriatric Hospital affiliated to Soochow University, Changzhou, China.
Cheng Y; Department of General Surgery, Changzhou No.7 People's Hospital, Changzhou, China.; Department of General Surgery, Changzhou Geriatric Hospital affiliated to Soochow University, Changzhou, China.
Ge S; Department of General Surgery, Changzhou No.7 People's Hospital, Changzhou, China.; Department of General Surgery, Changzhou Geriatric Hospital affiliated to Soochow University, Changzhou, China.
Wang J; School of Public Health, Suzhou Medical College of Soochow University, Suzhou, China.
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Źródło:
Journal of cellular and molecular medicine [J Cell Mol Med] 2024 Oct; Vol. 28 (19), pp. e70080.
Typ publikacji:
Journal Article
MeSH Terms:
Stomach Neoplasms*/pathology
Stomach Neoplasms*/genetics
Stomach Neoplasms*/metabolism
Cell Proliferation*
Cell Movement*/genetics
DNA Methylation*
Insulin-Like Growth Factor Binding Proteins*/metabolism
Insulin-Like Growth Factor Binding Proteins*/genetics
Gene Expression Regulation, Neoplastic*
Signal Transduction*
STAT Transcription Factors*/metabolism
Humans ; Animals ; Mice ; Cell Line, Tumor ; Mice, Nude ; Janus Kinases/metabolism ; Female ; Male ; RNA Methylation
Czasopismo naukowe
Tytuł:
Inhibition of HTR2B-mediated serotonin signaling in colorectal cancer suppresses tumor growth through ERK signaling.
Autorzy:
Lee JY; Department of Biomedical Science and Engineering, Gwangju Institute of Science and Technology, Gwangju, South Korea.
Park S; Department of Biomedical Science and Engineering, Gwangju Institute of Science and Technology, Gwangju, South Korea.
Park EJ; Division of Colon and Rectal Surgery, Department of Surgery, Asan Medical Center, University of Ulsan College of Medicine, Seoul, South Korea.
Pagire HS; Department of Chemistry, Gwangju Institute of Science and Technology, Gwangju, South Korea.
Pagire SH; Department of Chemistry, Gwangju Institute of Science and Technology, Gwangju, South Korea.
Choi BW; Department of Chemistry, Gwangju Institute of Science and Technology, Gwangju, South Korea.
Park M; Department of Biomedical Science and Engineering, Gwangju Institute of Science and Technology, Gwangju, South Korea.
Fang S; Department of Biomedical Sciences, Gangnam Severance Hospital, Yonsei University College of Medicine, Seoul, South Korea.
Ahn JH; Department of Chemistry, Gwangju Institute of Science and Technology, Gwangju, South Korea; JD Bioscience Inc., Gwangju, South Korea. Electronic address: .
Oh CM; Department of Biomedical Science and Engineering, Gwangju Institute of Science and Technology, Gwangju, South Korea. Electronic address: .
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Źródło:
Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie [Biomed Pharmacother] 2024 Oct; Vol. 179, pp. 117428. Date of Electronic Publication: 2024 Sep 09.
Typ publikacji:
Journal Article
MeSH Terms:
Colorectal Neoplasms*/pathology
Colorectal Neoplasms*/drug therapy
Colorectal Neoplasms*/metabolism
Cell Proliferation*/drug effects
Receptor, Serotonin, 5-HT2B*/metabolism
Serotonin*/metabolism
Serotonin*/pharmacology
Cell Movement*/drug effects
MAP Kinase Signaling System*/drug effects
Animals ; Humans ; Cell Line, Tumor ; Mice ; Xenograft Model Antitumor Assays ; Mice, Nude ; Mice, Inbred BALB C ; Serotonin 5-HT2 Receptor Antagonists/pharmacology ; Signal Transduction/drug effects ; Gene Expression Regulation, Neoplastic/drug effects
Czasopismo naukowe
Tytuł:
Polystyrene nanoplastics accelerate atherosclerosis: Unraveling the impact on smooth muscle cells through KIF15-mediated migration.
Autorzy:
Zhong Y; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China; Department of Cardiovascular Surgery, Zhujiang Hospital, Southern Medical University, Guangzhou 510280, China.
Feng Y; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Huang Y; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China; Department of Cardiovascular Surgery, Zhujiang Hospital, Southern Medical University, Guangzhou 510280, China.
Wang B; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Shi W; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Liang B; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Li Z; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Zhang B; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Du J; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Xiu J; State Key Laboratory of Organ Failure Research Department of Cardiology, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Yang X; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Huang Z; National Medical Products Administration (NMPA) Key Laboratory for Safety Evaluation of Cosmetics, Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Toxicology, School of Public Health, Southern Medical University, Guangzhou 510515, China; Department of Cardiovascular Surgery, Zhujiang Hospital, Southern Medical University, Guangzhou 510280, China. Electronic address: .
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Źródło:
Ecotoxicology and environmental safety [Ecotoxicol Environ Saf] 2024 Oct 01; Vol. 284, pp. 116983. Date of Electronic Publication: 2024 Sep 03.
Typ publikacji:
Journal Article
MeSH Terms:
Atherosclerosis*/chemically induced
Atherosclerosis*/pathology
Kinesins*/metabolism
Cell Movement*/drug effects
Polystyrenes*/toxicity
Myocytes, Smooth Muscle*/drug effects
Myocytes, Smooth Muscle*/pathology
Animals ; Mice ; Male ; Microplastics/toxicity ; Nanoparticles/toxicity ; Apolipoproteins E/genetics ; Mice, Knockout ; Mice, Knockout, ApoE ; Mice, Inbred C57BL
Czasopismo naukowe
Tytuł:
Silencing ANLN hinders the proliferation, migration, invasion, and angiogenesis of oral squamous cell carcinoma.
Autorzy:
Wu S; Guangxi Key Laboratory of Oral and Maxillofacial Rehabilitation and Reconstruction; Guangxi Health Commission Key laboratory of prevention and treatment for oral infectious diseases; Guangxi Clinical Research Center for Craniofacial Deformity, Nanning 530021, PR China; Department of Pathology, College & Hospital of Stomatology, Guangxi Medical University, Nanning, Guangxi 530021, PR China.
Li D; Guangxi Key Laboratory of Oral and Maxillofacial Rehabilitation and Reconstruction; Guangxi Health Commission Key laboratory of prevention and treatment for oral infectious diseases; Guangxi Clinical Research Center for Craniofacial Deformity, Nanning 530021, PR China; Department of Pathology, College & Hospital of Stomatology, Guangxi Medical University, Nanning, Guangxi 530021, PR China.
Li L; Guangxi Key Laboratory of Oral and Maxillofacial Rehabilitation and Reconstruction; Guangxi Health Commission Key laboratory of prevention and treatment for oral infectious diseases; Guangxi Clinical Research Center for Craniofacial Deformity, Nanning 530021, PR China; Department of Pathology, College & Hospital of Stomatology, Guangxi Medical University, Nanning, Guangxi 530021, PR China.
Zhao J; Guangxi Key Laboratory of Oral and Maxillofacial Rehabilitation and Reconstruction; Guangxi Health Commission Key laboratory of prevention and treatment for oral infectious diseases; Guangxi Clinical Research Center for Craniofacial Deformity, Nanning 530021, PR China; Department of Pathology, College & Hospital of Stomatology, Guangxi Medical University, Nanning, Guangxi 530021, PR China.
Zhang H; Guangxi Key Laboratory of Oral and Maxillofacial Rehabilitation and Reconstruction; Guangxi Health Commission Key laboratory of prevention and treatment for oral infectious diseases; Guangxi Clinical Research Center for Craniofacial Deformity, Nanning 530021, PR China; Department of Pathology, College & Hospital of Stomatology, Guangxi Medical University, Nanning, Guangxi 530021, PR China.
Zhou X; Guangxi Key Laboratory of Oral and Maxillofacial Rehabilitation and Reconstruction; Guangxi Health Commission Key laboratory of prevention and treatment for oral infectious diseases; Guangxi Clinical Research Center for Craniofacial Deformity, Nanning 530021, PR China; Department of Pathology, College & Hospital of Stomatology, Guangxi Medical University, Nanning, Guangxi 530021, PR China.
Wang S; Department of Research, Guangxi Medical University Cancer Hospital, Nanning, Guangxi 530021, PR China.
Mo Y; Department of Research, Guangxi Medical University Cancer Hospital, Nanning, Guangxi 530021, PR China. Electronic address: .
Li P; Guangxi Key Laboratory of Oral and Maxillofacial Rehabilitation and Reconstruction; Guangxi Health Commission Key laboratory of prevention and treatment for oral infectious diseases; Guangxi Clinical Research Center for Craniofacial Deformity, Nanning 530021, PR China; Department of Pathology, College & Hospital of Stomatology, Guangxi Medical University, Nanning, Guangxi 530021, PR China. Electronic address: .
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Źródło:
Pathology, research and practice [Pathol Res Pract] 2024 Oct; Vol. 262, pp. 155563. Date of Electronic Publication: 2024 Aug 26.
Typ publikacji:
Journal Article
MeSH Terms:
Mouth Neoplasms*/pathology
Mouth Neoplasms*/genetics
Mouth Neoplasms*/metabolism
Cell Proliferation*/genetics
Neovascularization, Pathologic*/genetics
Neovascularization, Pathologic*/pathology
Neovascularization, Pathologic*/metabolism
Cell Movement*/genetics
Neoplasm Invasiveness*/genetics
Microfilament Proteins*/metabolism
Microfilament Proteins*/genetics
Humans ; Carcinoma, Squamous Cell/pathology ; Carcinoma, Squamous Cell/genetics ; Carcinoma, Squamous Cell/metabolism ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Animals ; Squamous Cell Carcinoma of Head and Neck/pathology ; Squamous Cell Carcinoma of Head and Neck/genetics ; Squamous Cell Carcinoma of Head and Neck/metabolism ; Gene Silencing ; Mice ; Epithelial-Mesenchymal Transition/genetics ; Female ; Male ; Angiogenesis
Czasopismo naukowe
Tytuł:
Mutant p53 achieves function by regulating EGR1 to induce epithelial mesenchymal transition.
Autorzy:
Meng W; Department of Toxicology, School of Public Health, Jilin University, Changchun, Jilin 130021, China.
Yu S; Interventional Center, Jilin Cancer Hospital, No. 1018 Huguang Rd, Chaoyang, Changchun 130012, China.
Li Y; Department of Toxicology, School of Public Health, Jilin University, Changchun, Jilin 130021, China.
Wang H; NHC Key Laboratory of Radiobiology, School of Public Health, Jilin University, Changchun 130021, China.
Feng Y; NHC Key Laboratory of Radiobiology, School of Public Health, Jilin University, Changchun 130021, China.
Sun W; Department of Toxicology, School of Public Health, Jilin University, Changchun, Jilin 130021, China.
Liu Y; Department of Toxicology, School of Public Health, Jilin University, Changchun, Jilin 130021, China.
Sun S; NHC Key Laboratory of Radiobiology, School of Public Health, Jilin University, Changchun 130021, China. Electronic address: .
Liu H; Interventional Center, Jilin Cancer Hospital, No. 1018 Huguang Rd, Chaoyang, Changchun 130012, China. Electronic address: .
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Źródło:
Tissue & cell [Tissue Cell] 2024 Oct; Vol. 90, pp. 102510. Date of Electronic Publication: 2024 Aug 02.
Typ publikacji:
Journal Article
MeSH Terms:
Epithelial-Mesenchymal Transition*/genetics
Early Growth Response Protein 1*/metabolism
Early Growth Response Protein 1*/genetics
Tumor Suppressor Protein p53*/metabolism
Tumor Suppressor Protein p53*/genetics
Gene Expression Regulation, Neoplastic*
Lung Neoplasms*/genetics
Lung Neoplasms*/pathology
Lung Neoplasms*/metabolism
Cell Movement*/genetics
Cell Proliferation*/genetics
Humans ; Cell Line, Tumor ; Carcinoma, Non-Small-Cell Lung/genetics ; Carcinoma, Non-Small-Cell Lung/pathology ; Carcinoma, Non-Small-Cell Lung/metabolism ; Mutation ; Neoplasm Invasiveness/genetics
Czasopismo naukowe
Tytuł:
Stearoyl-CoA desaturase 1 is targeted by EBV-encoded miR-BART20-5p and regulates cell autophagy, proliferation, and migration in EBV-associated gastric cancer.
Autorzy:
Gong Z; Department of Pathogenic Biology, School of Basic Medicine, Qingdao University, Qingdao, 266071, China.
Shi D; Department of Pathogenic Biology, School of Basic Medicine, Qingdao University, Qingdao, 266071, China.
Yan Z; Department of Pathogenic Biology, School of Basic Medicine, Qingdao University, Qingdao, 266071, China.
Sun L; Department of Pathology of the Affiliated Hospital of Qingdao University, 16 Jiangsu Road, Qingdao, 266003, China.
Liu W; Department of Pathogenic Biology, School of Basic Medicine, Qingdao University, Qingdao, 266071, China. .
Luo B; Department of Pathogenic Biology, School of Basic Medicine, Qingdao University, Qingdao, 266071, China. .
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Źródło:
Virus genes [Virus Genes] 2024 Oct; Vol. 60 (5), pp. 464-474. Date of Electronic Publication: 2024 Aug 03.
Typ publikacji:
Journal Article
MeSH Terms:
Stomach Neoplasms*/virology
Stomach Neoplasms*/genetics
Stomach Neoplasms*/pathology
MicroRNAs*/genetics
Stearoyl-CoA Desaturase*/genetics
Autophagy*/genetics
Cell Movement*/genetics
Herpesvirus 4, Human*/genetics
Cell Proliferation*/genetics
Humans ; Cell Line, Tumor ; Epstein-Barr Virus Infections/virology ; Epstein-Barr Virus Infections/genetics ; Gene Expression Regulation, Neoplastic ; 3' Untranslated Regions/genetics ; RNA, Viral/genetics
Czasopismo naukowe
Tytuł:
Hsp90aa1/JUN/Ccl2 regulatory axis mediates migration and differentiation of NSPCs, promoting the onset and progression of early post-ischemic stroke epilepsy.
Autorzy:
Hu S; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China.
Tang Y; Department of Anesthesiology, The Third Xiangya Hospital, Central South University, Changsha, China.
Li X; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China.
Li W; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China.
Zeng Y; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China.
Jiang M; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China; Department of Neurology, University of Michigan, Ann Arbor, MI, USA.
Chen R; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China.
Zheng P; Department of Neurosurgery, Shanghai Pudong New Area People's Hospital, Shanghai, China.
Yang L; Department of Neurosurgery, The Third Xiangya Hospital, Central South University, Changsha, China.
Song Z; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China.
Xie D; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China. Electronic address: .
Chen Y; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China. Electronic address: .
Yuan Y; Department of Neurology, The Third Xiangya Hospital, Central South University, Changsha, China. Electronic address: .
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Źródło:
Neurobiology of disease [Neurobiol Dis] 2024 Oct 01; Vol. 200, pp. 106635. Date of Electronic Publication: 2024 Aug 10.
Typ publikacji:
Journal Article
MeSH Terms:
Cell Differentiation*/physiology
Cell Movement*/physiology
Epilepsy*/metabolism
Epilepsy*/genetics
HSP90 Heat-Shock Proteins*/metabolism
HSP90 Heat-Shock Proteins*/genetics
Ischemic Stroke*/metabolism
Ischemic Stroke*/pathology
Neural Stem Cells*/metabolism
Animals ; Male ; Mice ; Disease Progression ; Mice, Inbred C57BL ; Proto-Oncogene Proteins c-jun
Czasopismo naukowe
Tytuł:
N-acetylgalactosaminyltransferase GALNT6 is a potential therapeutic target of clear cell renal cell carcinoma progression.
Autorzy:
Sun L; Department of Urology, The First Affiliated Hospital of China Medical University, Shenyang, China.
Li Z; Department of General Surgery, Shengjing Hospital of China Medical University, Shenyang, China.
Shu P; Department of Thoracic Surgery, The First Affiliated Hospital of China Medical University, Shenyang, China.
Lu M; Department of Colorectal Surgery, The First Affiliated Hospital of China Medical University, Shenyang, China.
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Źródło:
Cancer science [Cancer Sci] 2024 Oct; Vol. 115 (10), pp. 3320-3332. Date of Electronic Publication: 2024 Aug 06.
Typ publikacji:
Journal Article
MeSH Terms:
N-Acetylgalactosaminyltransferases*/metabolism
N-Acetylgalactosaminyltransferases*/genetics
Carcinoma, Renal Cell*/pathology
Carcinoma, Renal Cell*/metabolism
Carcinoma, Renal Cell*/genetics
Cell Proliferation*
Kidney Neoplasms*/pathology
Kidney Neoplasms*/genetics
Kidney Neoplasms*/metabolism
Cell Movement*
Disease Progression*
Humans ; Animals ; Cell Line, Tumor ; Mice ; Glycosylation ; Female ; Male ; Antigens, Tumor-Associated, Carbohydrate/metabolism ; Mice, Nude ; Gene Expression Regulation, Neoplastic
Czasopismo naukowe
Tytuł:
The long intergenic non-coding RNA LINC01140 modulates gastric cancer phenotypes and cancer cell lines aggressiveness.
Autorzy:
Singh J; Department of Gastroenterology, Institute of Medical Sciences, Banaras Hindu University, Varanasi, 221005, India.
Narayan G; Department of Molecular and Human Genetics, Banaras Hindu University, Varanasi, 221005, India.
Dixit VK; Department of Gastroenterology, Institute of Medical Sciences, Banaras Hindu University, Varanasi, 221005, India. Electronic address: .
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Źródło:
Digestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver [Dig Liver Dis] 2024 Oct; Vol. 56 (10), pp. 1776-1783. Date of Electronic Publication: 2024 Mar 31.
Typ publikacji:
Journal Article
MeSH Terms:
Stomach Neoplasms*/genetics
Stomach Neoplasms*/pathology
Stomach Neoplasms*/metabolism
RNA, Long Noncoding*/genetics
RNA, Long Noncoding*/metabolism
Cell Movement*/genetics
Fibroblast Growth Factor 9*/genetics
Fibroblast Growth Factor 9*/metabolism
Gene Expression Regulation, Neoplastic*
Humans ; Cell Line, Tumor ; Male ; Female ; Phenotype ; Middle Aged ; MicroRNAs/genetics ; MicroRNAs/metabolism ; Neoplasm Invasiveness/genetics ; Up-Regulation ; Cell Proliferation/genetics
Czasopismo naukowe

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