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Tytuł :
MicroRNA-92a serves as a risk factor in sepsis-induced ARDS and regulates apoptosis and cell migration in lipopolysaccharide-induced HPMEC and A549 cell injury.
Autorzy :
Xu F; Department of Critical Care Medicine, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400016, PR China. Electronic address: .
Yuan J; Department of Critical Care Medicine, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400016, PR China.
Tian S; Department of Critical Care Medicine, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400016, PR China.
Chen Y; Emergency Department, Chongqing Emergency Medical Center, Chongqing 400016, PR China.
Zhou F; Department of Critical Care Medicine, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400016, PR China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2020 Sep 01; Vol. 256, pp. 117957. Date of Electronic Publication: 2020 Jun 10.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*/drug effects
Apoptosis*/genetics
Cell Movement*/drug effects
Cell Movement*/genetics
Endothelial Cells/*pathology
Lung/*blood supply
MicroRNAs/*metabolism
Respiratory Distress Syndrome, Adult/*etiology
Respiratory Distress Syndrome, Adult/*genetics
Sepsis/*complications
A549 Cells ; Aged ; Endothelial Cells/drug effects ; Endothelial Cells/metabolism ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; Inflammation/pathology ; Intensive Care Units ; Lipopolysaccharides ; MicroRNAs/blood ; MicroRNAs/genetics ; Microvessels/pathology ; Middle Aged ; Proto-Oncogene Proteins c-akt/metabolism ; Respiratory Distress Syndrome, Adult/blood ; Respiratory Distress Syndrome, Adult/pathology ; Risk Factors ; Signal Transduction ; TOR Serine-Threonine Kinases/metabolism
Czasopismo naukowe
Tytuł :
Cyclophilin A inhibits trophoblast migration and invasion in vitro and vivo through p38/ERK/JNK pathways and causes features of preeclampsia in mice.
Autorzy :
Hu H; Department of Obstetrics and Gynecology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Jiang J; Department of Obstetrics and Gynecology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Chen Q; Department of Obstetrics and Gynecology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Wei S; Department of Neuropharmacology and Drug Discovery, School of Pharmaceutical Sciences, Southern Medical University, China.
Liu M; Department of Obstetrics and Gynecology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Chen X; Department of Obstetrics and Gynecology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Fan C; Institute of Neuroscience and Department of Neurology, the Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, China.
Ma J; Department of Obstetrics and Gynecology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Chen W; Department of Obstetrics and Gynecology, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Wang X; Department of Obstetrics and Gynecology, Nanfang Hospital, Southern Medical University, Guangzhou, China. Electronic address: .
Zhong M; Department of Obstetrics and Gynecology, Nanfang Hospital, Southern Medical University, Guangzhou, China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2020 Nov 15; Vol. 261, pp. 118351. Date of Electronic Publication: 2020 Aug 26.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Movement*
MAP Kinase Signaling System*
Cyclophilin A/*metabolism
Pre-Eclampsia/*enzymology
Pre-Eclampsia/*pathology
Trophoblasts/*enzymology
Trophoblasts/*pathology
Adult ; Animals ; Down-Regulation/genetics ; Female ; Gene Knockdown Techniques ; Gene Silencing ; Humans ; Kidney/pathology ; Matrix Metalloproteinase 2/metabolism ; Matrix Metalloproteinase 9/metabolism ; Mice ; Placenta/enzymology ; Placenta/pathology ; Pregnancy ; Pregnancy Outcome
Czasopismo naukowe
Tytuł :
Orai-1 and Orai-2 regulate oral cancer cell migration and colonisation by suppressing Akt/mTOR/NF-κB signalling.
Autorzy :
Singh AK; Cancer Biology Laboratory, Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati, Assam 781039, India.
Roy NK; Cancer Biology Laboratory, Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati, Assam 781039, India.
Bordoloi D; Cancer Biology Laboratory, Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati, Assam 781039, India.
Padmavathi G; Cancer Biology Laboratory, Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati, Assam 781039, India.
Banik K; Cancer Biology Laboratory, Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati, Assam 781039, India.
Khwairakpam AD; Cancer Biology Laboratory, Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati, Assam 781039, India.
Kunnumakkara AB; Cancer Biology Laboratory, Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati, Assam 781039, India. Electronic address: .
Sukumar P; Leeds Institute of Cardiovascular and Metabolic Medicine, School of Medicine, University of Leeds, Leeds LS2 9JT, UK. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2020 Nov 15; Vol. 261, pp. 118372. Date of Electronic Publication: 2020 Aug 31.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Movement*/drug effects
Signal Transduction*/drug effects
Mouth Neoplasms/*pathology
NF-kappa B/*metabolism
ORAI1 Protein/*metabolism
ORAI2 Protein/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
TOR Serine-Threonine Kinases/*metabolism
Calcium/metabolism ; Calcium Signaling ; Carcinogens/toxicity ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Gene Expression Regulation, Neoplastic/drug effects ; Gene Silencing/drug effects ; Humans ; Mouth Neoplasms/genetics ; Tobacco/chemistry ; Tumor Stem Cell Assay
Czasopismo naukowe
Tytuł :
Identification of stool miR-135b-5p as a non-invasive diaognostic biomarker in later tumor stage of colorectal cancer.
Autorzy :
Li L; Department of Gastroenterology, Yangpu Hospital, Tongji University School of Medicine, Shanghai 200090, China. Electronic address: .
Wang A; Institute of Gastrointestinal Surgery and Translational Medicine, Yangpu Hospital, Tongji University School of Medicine, Shanghai, China.
Cai M; Department of Gastroenterology, Yangpu Hospital, Tongji University School of Medicine, Shanghai 200090, China.
Tong M; Department of Gastroenterology, Yangpu Hospital, Tongji University School of Medicine, Shanghai 200090, China.
Chen F; Department of Gastroenterology, Yangpu Hospital, Tongji University School of Medicine, Shanghai 200090, China.
Huang L; Department of Gastroenterology, Yangpu Hospital, Tongji University School of Medicine, Shanghai 200090, China.
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Źródło :
Life sciences [Life Sci] 2020 Nov 01; Vol. 260, pp. 118417. Date of Electronic Publication: 2020 Sep 12.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Movement*
Gene Expression Regulation, Neoplastic*
Biomarkers, Tumor/*genetics
Colorectal Neoplasms/*diagnosis
Feces/*chemistry
MicroRNAs/*genetics
Apoptosis ; Case-Control Studies ; Cell Proliferation ; Colorectal Neoplasms/blood ; Colorectal Neoplasms/genetics ; Humans ; Neoplasm Staging ; ROC Curve ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł :
PADI4 modulates the invasion and migration of osteosarcoma cells by down-regulation of epithelial-mesenchymal transition.
Autorzy :
Zhai Q; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China.
Qin J; Department of Oncology, Gaoqing People's Hospital, Zibo 256300, China.
Jin X; Department of Geriatrics, Zibo Central Hospital, Zibo 255036, China.
Sun X; Department of Pathology, Zibo Central Hospital, Zibo 255036, China.
Wang L; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China.
Du W; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China.
Li T; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China. Electronic address: .
Xiang X; Center of Translational Medicine, Zibo Central Hospital, Zibo 255036, China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2020 Sep 01; Vol. 256, pp. 117968. Date of Electronic Publication: 2020 Jun 13.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Movement*
Down-Regulation*/genetics
Epithelial-Mesenchymal Transition*/genetics
Osteosarcoma/*enzymology
Osteosarcoma/*pathology
Protein-Arginine Deiminase Type 4/*metabolism
Antigens, CD/metabolism ; Cadherins/metabolism ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Humans ; Lung Neoplasms/secondary ; Neoplasm Invasiveness ; Osteosarcoma/genetics ; Protein-Arginine Deiminase Type 4/genetics
Czasopismo naukowe
Tytuł :
Sevoflurane inhibits migration and invasion of glioma cells via regulating miR-34a-5p/MMP-2 axis.
Autorzy :
Zhao H; Department of Anesthesiology and Perioperative Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, China.
Xing F; Department of Anesthesiology and Perioperative Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, China.
Yuan J; Department of Anesthesiology and Perioperative Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, China.
Li Z; Department of Anesthesiology and Perioperative Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, China.
Zhang W; Department of Anesthesiology and Perioperative Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2020 Sep 01; Vol. 256, pp. 117897. Date of Electronic Publication: 2020 Jun 02.
Typ publikacji :
Journal Article
MeSH Terms :
Brain Neoplasms/*pathology
Cell Movement/*drug effects
Glioma/*pathology
Matrix Metalloproteinase 2/*metabolism
MicroRNAs/*metabolism
Sevoflurane/*pharmacology
Base Sequence ; Brain Neoplasms/enzymology ; Brain Neoplasms/genetics ; Cell Line, Tumor ; Cell Movement/genetics ; Gene Expression Regulation, Neoplastic/drug effects ; Gene Silencing/drug effects ; Glioma/enzymology ; Glioma/genetics ; Humans ; MicroRNAs/genetics ; Neoplasm Invasiveness ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Up-Regulation/drug effects
Czasopismo naukowe
Tytuł :
Chlorpyrifos subthreshold exposure induces epithelial-mesenchymal transition in breast cancer cells.
Autorzy :
Lasagna M; Instituto de Química y Fisicoquímica Biológicas 'Prof. Alejandro C. Paladini' (IQUIFIB) UBA-CONICET, Buenos Aires, Argentina; Laboratorio de Radioisótopos, Cátedra de Física, Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Buenos Aires, Argentina.
Hielpos MS; Laboratorio de Radioisótopos, Cátedra de Física, Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Buenos Aires, Argentina.
Ventura C; Laboratorio de Radioisótopos, Cátedra de Física, Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Buenos Aires, Argentina; Instituto de Estudios Inmunológicos y Fisiopatológicos (IIFP) CONICET-UNLP, La Plata, Argentina.
Mardirosian MN; Instituto de Química y Fisicoquímica Biológicas 'Prof. Alejandro C. Paladini' (IQUIFIB) UBA-CONICET, Buenos Aires, Argentina.
Martín G; Laboratorio de Radioisótopos, Cátedra de Física, Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Buenos Aires, Argentina.
Miret N; Laboratorio de Efectos Biológicos de Contaminantes Ambientales, Departamento de Bioquímica Humana, Facultad de Medicina, Universidad de Buenos Aires, Buenos Aires, Argentina.
Randi A; Laboratorio de Efectos Biológicos de Contaminantes Ambientales, Departamento de Bioquímica Humana, Facultad de Medicina, Universidad de Buenos Aires, Buenos Aires, Argentina.
Núñez M; Laboratorio de Radioisótopos, Cátedra de Física, Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Buenos Aires, Argentina.
Cocca C; Instituto de Química y Fisicoquímica Biológicas 'Prof. Alejandro C. Paladini' (IQUIFIB) UBA-CONICET, Buenos Aires, Argentina; Laboratorio de Radioisótopos, Cátedra de Física, Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Buenos Aires, Argentina. Electronic address: .
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Źródło :
Ecotoxicology and environmental safety [Ecotoxicol Environ Saf] 2020 Dec 01; Vol. 205, pp. 111312. Date of Electronic Publication: 2020 Sep 18.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Movement/*drug effects
Chlorpyrifos/*toxicity
Endocrine Disruptors/*toxicity
Epithelial-Mesenchymal Transition/*drug effects
Pesticides/*toxicity
Animals ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; CSK Tyrosine-Protein Kinase/genetics ; Cell Line, Tumor ; Cell Movement/genetics ; Dose-Response Relationship, Drug ; Epithelial-Mesenchymal Transition/genetics ; Estrogen Receptor alpha/genetics ; Female ; Humans ; MCF-7 Cells ; Matrix Metalloproteinase 2/genetics ; Signal Transduction
Czasopismo naukowe
Tytuł :
Ezrin T567 phosphorylation regulates migration and invasion of ectopic endometrial stromal cells by changing actin cytoskeleton.
Autorzy :
Chen C; Department of Gynecology, Women's Hospital, School of Medicine, Zhejiang University, Zhejiang 310006, China.
Ye C; Department of Gynecology, Women's Hospital, School of Medicine, Zhejiang University, Zhejiang 310006, China.
Xia J; Department of Obstetrics and Gynecology, Hangzhou First People's Hospital, Zhejiang 310006, China.
Zhou Y; Department of Gynecology, Women's Hospital, School of Medicine, Zhejiang University, Zhejiang 310006, China.
Wu R; Department of Gynecology, Women's Hospital, School of Medicine, Zhejiang University, Zhejiang 310006, China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2020 Aug 01; Vol. 254, pp. 117681. Date of Electronic Publication: 2020 May 05.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Movement*
Actin Cytoskeleton/*metabolism
Cytoskeletal Proteins/*metabolism
Endometrium/*cytology
Stromal Cells/*cytology
Female ; Humans ; Phosphorylation
Czasopismo naukowe
Tytuł :
Long non-coding RNA MANCR is a target of BET bromodomain protein BRD4 and plays a critical role in cellular migration and invasion abilities of prostate cancer.
Autorzy :
Nagasawa M; Department of Biochemistry and Molecular Biology, Shiga University of Medical Science, Shiga, Japan; Department of Urology, Shiga University of Medical Science, Shiga, Japan.
Tomimatsu K; Department of Biochemistry and Molecular Biology, Shiga University of Medical Science, Shiga, Japan.
Terada K; Department of Biochemistry and Molecular Biology, Shiga University of Medical Science, Shiga, Japan.
Kondo K; Department of Biochemistry and Molecular Biology, Shiga University of Medical Science, Shiga, Japan.
Miyazaki K; Laboratory of Immunology, Institute for Frontier Life and Medical Sciences, Kyoto University, Kyoto, Japan.
Miyazaki M; Laboratory of Immunology, Institute for Frontier Life and Medical Sciences, Kyoto University, Kyoto, Japan.
Motooka D; Genome Information Research Center, Research Institute for Microbial Diseases, Osaka University, Osaka, Japan.
Okuzaki D; Genome Information Research Center, Research Institute for Microbial Diseases, Osaka University, Osaka, Japan.
Yoshida T; Department of Urology, Shiga University of Medical Science, Shiga, Japan.
Kageyama S; Department of Urology, Shiga University of Medical Science, Shiga, Japan.
Kawamoto H; Laboratory of Immunology, Institute for Frontier Life and Medical Sciences, Kyoto University, Kyoto, Japan.
Kawauchi A; Department of Urology, Shiga University of Medical Science, Shiga, Japan.
Agata Y; Department of Biochemistry and Molecular Biology, Shiga University of Medical Science, Shiga, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 May 21; Vol. 526 (1), pp. 128-134. Date of Electronic Publication: 2020 Mar 18.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*/drug effects
Cell Movement*/genetics
Cell Cycle Proteins/*metabolism
Prostatic Neoplasms/*metabolism
Prostatic Neoplasms/*pathology
RNA, Untranslated/*metabolism
Transcription Factors/*metabolism
Azepines/pharmacology ; Cell Line, Tumor ; Down-Regulation/drug effects ; Down-Regulation/genetics ; Enhancer Elements, Genetic/genetics ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; Male ; Neoplasm Invasiveness ; Prostatic Neoplasms/genetics ; RNA, Untranslated/genetics ; Triazoles/pharmacology
Czasopismo naukowe
Tytuł :
FOXM1 facilitates breast cancer cell stemness and migration in YAP1-dependent manner.
Autorzy :
Sun HL; College of Life Science & Bioengineering, Beijing University of Technology, Pingleyuan 100(#), District of Chaoyang, Beijing, 100124, China.
Men JR; College of Life Science & Bioengineering, Beijing University of Technology, Pingleyuan 100(#), District of Chaoyang, Beijing, 100124, China.
Liu HY; College of Life Science & Bioengineering, Beijing University of Technology, Pingleyuan 100(#), District of Chaoyang, Beijing, 100124, China.
Liu MY; College of Life Science & Bioengineering, Beijing University of Technology, Pingleyuan 100(#), District of Chaoyang, Beijing, 100124, China.
Zhang HS; College of Life Science & Bioengineering, Beijing University of Technology, Pingleyuan 100(#), District of Chaoyang, Beijing, 100124, China. Electronic address: .
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Źródło :
Archives of biochemistry and biophysics [Arch Biochem Biophys] 2020 May 30; Vol. 685, pp. 108349. Date of Electronic Publication: 2020 Mar 21.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Adaptor Proteins, Signal Transducing/*metabolism
Breast Neoplasms/*metabolism
Cell Movement/*physiology
Forkhead Box Protein M1/*metabolism
Neoplastic Stem Cells/*metabolism
Transcription Factors/*metabolism
Adaptor Proteins, Signal Transducing/chemistry ; Biomarkers/metabolism ; Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; Cell Proliferation/physiology ; Forkhead Box Protein M1/genetics ; Gene Knockdown Techniques ; Humans ; Nanog Homeobox Protein/metabolism ; Octamer Transcription Factor-3/metabolism ; Phosphorylation/genetics ; Transcription Factors/chemistry ; Up-Regulation
Czasopismo naukowe
Tytuł :
Downregulation of glypican-4 facilitates breast cancer progression by inducing cell migration and proliferation.
Autorzy :
Munir J; Soonchunhyang Institute of Medi-bioscience (SIMS), Soonchunhyang University, Cheonan, South Korea; Department of Integrated Biomedicine, Soonchunhyang University, Cheonan, South Korea. Electronic address: .
Van Ngu T; Soonchunhyang Institute of Medi-bioscience (SIMS), Soonchunhyang University, Cheonan, South Korea; Department of Integrated Biomedicine, Soonchunhyang University, Cheonan, South Korea. Electronic address: .
Na Ayudthaya PD; Soonchunhyang Institute of Medi-bioscience (SIMS), Soonchunhyang University, Cheonan, South Korea; Laboratory of Biochemistry, Chulabhorn Research Institute, Bangkok, Thailand. Electronic address: .
Ryu S; Soonchunhyang Institute of Medi-bioscience (SIMS), Soonchunhyang University, Cheonan, South Korea; Department of Integrated Biomedicine, Soonchunhyang University, Cheonan, South Korea. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 May 21; Vol. 526 (1), pp. 91-97. Date of Electronic Publication: 2020 Mar 18.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*
Disease Progression*
Down-Regulation*
Breast Neoplasms/*metabolism
Breast Neoplasms/*pathology
Glypicans/*metabolism
Cell Line, Tumor ; Cell Proliferation ; Female ; Humans ; Neoplasm Invasiveness ; Neoplasm Metastasis
Czasopismo naukowe
Tytuł :
Cyclophilin a knokdown inhibits cell migration and invasion through the suppression of epithelial-mesenchymal transition in colorectal cancer cells.
Autorzy :
Yamamoto T; Pathological and Biomolecule Analyses Laboratory, Faculty of Pharmacy, Kindai University, Osaka, Japan.
Takakura H; Department of Molecular-Targeting Prevention, Kyoto Prefectural University of Medicine, Kyoto, Japan; Department of Drug Discovery Medicine, Kyoto Prefectural University of Medicine, Kyoto, Japan.
Mitamura K; Pathological and Biomolecule Analyses Laboratory, Faculty of Pharmacy, Kindai University, Osaka, Japan.
Taga A; Pathological and Biomolecule Analyses Laboratory, Faculty of Pharmacy, Kindai University, Osaka, Japan; Antiaging Center, Kindai University, Osaka, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 May 21; Vol. 526 (1), pp. 55-61. Date of Electronic Publication: 2020 Mar 16.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*
Epithelial-Mesenchymal Transition*/genetics
Gene Knockdown Techniques*
Colorectal Neoplasms/*pathology
Cyclophilin A/*metabolism
Cell Line, Tumor ; Cell Proliferation/genetics ; Colorectal Neoplasms/genetics ; Cyclophilin A/genetics ; Gene Expression Regulation, Neoplastic ; Humans ; Neoplasm Invasiveness ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Signal Transduction/genetics
Czasopismo naukowe
Tytuł :
Exosomal encapsulation of miR-125a-5p inhibited trophoblast cell migration and proliferation by regulating the expression of VEGFA in preeclampsia.
Autorzy :
Xueya Z; The International Peace Maternity and Child Health Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Shanghai Key Laboratory of Embryo Original Diseases, Shanghai, China.
Yamei L; The International Peace Maternity and Child Health Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Shanghai Municipal Key Clinical Specialty, Shanghai, China.
Sha C; The International Peace Maternity and Child Health Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Shanghai Municipal Key Clinical Specialty, Shanghai, China.
Dan C; The International Peace Maternity and Child Health Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Shanghai Key Laboratory of Embryo Original Diseases, Shanghai, China.
Hong S; The International Peace Maternity and Child Health Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Shanghai Municipal Key Clinical Specialty, Shanghai, China.
Xingyu Y; The International Peace Maternity and Child Health Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Shanghai Key Laboratory of Embryo Original Diseases, Shanghai, China. Electronic address: .
Weiwei C; The International Peace Maternity and Child Health Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, China; Shanghai Municipal Key Clinical Specialty, Shanghai, China. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 May 07; Vol. 525 (3), pp. 646-653. Date of Electronic Publication: 2020 Feb 28.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*/genetics
Gene Expression Regulation*
Exosomes/*metabolism
MicroRNAs/*metabolism
Pre-Eclampsia/*genetics
Trophoblasts/*cytology
Vascular Endothelial Growth Factor A/*genetics
Adult ; Cell Cycle Checkpoints/genetics ; Cell Line ; Cell Proliferation/genetics ; Female ; Fetal Blood/metabolism ; Gene Ontology ; Human Umbilical Vein Endothelial Cells/metabolism ; Humans ; MicroRNAs/genetics ; Neovascularization, Physiologic/drug effects ; Pre-Eclampsia/blood ; Pregnancy ; Vascular Endothelial Growth Factor A/metabolism
Czasopismo naukowe
Tytuł :
Lymphatic endothelial cells promote T lymphocyte migration into lymph nodes under hyperlipidemic conditions.
Autorzy :
Park M; Department of Microbiology, College of Medicine, Ewha Womans University, Seoul, 07804, South Korea.
Cho KA; Department of Microbiology, College of Medicine, Ewha Womans University, Seoul, 07804, South Korea.
Kim YH; Department of Microbiology, College of Medicine, Ewha Womans University, Seoul, 07804, South Korea.
Lee KH; Department of Dermatology, Bucheon St. Mary's Hospital, The Catholic University of Korea, 14647, South Korea. Electronic address: .
Woo SY; Department of Microbiology, College of Medicine, Ewha Womans University, Seoul, 07804, South Korea. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 May 07; Vol. 525 (3), pp. 786-792. Date of Electronic Publication: 2020 Mar 05.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*/drug effects
Endothelial Cells/*pathology
Hyperlipidemias/*immunology
Hyperlipidemias/*pathology
Lymph Nodes/*pathology
T-Lymphocytes/*pathology
Animals ; Cell Line ; Chemokines/metabolism ; Diet ; Endothelial Cells/drug effects ; Endothelial Cells/metabolism ; Heart Ventricles/drug effects ; Heart Ventricles/pathology ; Heart Ventricles/physiopathology ; Hyperlipidemias/physiopathology ; Male ; Mice, Inbred C57BL ; Neovascularization, Physiologic/drug effects ; Palmitic Acid/toxicity ; T-Lymphocytes/drug effects ; Ventricular Remodeling/drug effects
Czasopismo naukowe
Tytuł :
Talin B regulates collective cell migration via PI3K signaling in Dictyostelium discoideum mounds.
Autorzy :
Yamazaki SI; Laboratory for Cell Signaling Dynamics, BDR (Biosystems and Dynamics Research Center), RIKEN, Suita, Osaka, 565-0874, Japan.
Hashimura H; Laboratory for Cell Signaling Dynamics, BDR (Biosystems and Dynamics Research Center), RIKEN, Suita, Osaka, 565-0874, Japan; Graduate School of Arts and Sciences, University of Tokyo, Komaba, 153-8902, Tokyo, Japan.
Morimoto YV; Laboratory for Cell Signaling Dynamics, BDR (Biosystems and Dynamics Research Center), RIKEN, Suita, Osaka, 565-0874, Japan; Department of Physics and Information Technology, Faculty of Computer Science and Systems Engineering, Kyushu Institute of Technology, Iizuka, Fukuoka, 820-8502, Japan.
Miyanaga Y; Laboratory of Single Molecular Biology, Graduate School of Frontier Biosciences, Osaka University, 1-3 Yamadaoka, Suita, Osaka, 565-0871, Japan.
Matsuoka S; Laboratory of Single Molecular Biology, Graduate School of Frontier Biosciences, Osaka University, 1-3 Yamadaoka, Suita, Osaka, 565-0871, Japan.
Kamimura Y; Laboratory for Cell Signaling Dynamics, BDR (Biosystems and Dynamics Research Center), RIKEN, Suita, Osaka, 565-0874, Japan. Electronic address: .
Ueda M; Laboratory for Cell Signaling Dynamics, BDR (Biosystems and Dynamics Research Center), RIKEN, Suita, Osaka, 565-0874, Japan; Laboratory of Single Molecular Biology, Graduate School of Frontier Biosciences, Osaka University, 1-3 Yamadaoka, Suita, Osaka, 565-0871, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 Apr 30; Vol. 525 (2), pp. 372-377. Date of Electronic Publication: 2020 Feb 22.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*
Dictyostelium/*cytology
Phosphatidylinositol 3-Kinases/*metabolism
Talin/*physiology
Cell Aggregation ; Cyclic AMP/metabolism ; Dictyostelium/metabolism ; Protozoan Proteins
Czasopismo naukowe
Tytuł :
TGF-β promotes fetal gene expression and cell migration velocity in a wound repair model of untransformed intestinal epithelial cells.
Autorzy :
Anzai S; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Kawamoto A; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Nagata S; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Takahashi J; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Kawai M; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Kuno R; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Kobayashi S; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Watanabe S; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Suzuki K; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Shimizu H; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Hiraguri Y; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Takeoka S; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Sugihara HY; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Yui S; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan; Center for Stem Cell and Regenerative Medicine, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Oshima S; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Watanabe M; Advanced Research Institute, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan.
Okamoto R; Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan; Center for Stem Cell and Regenerative Medicine, Tokyo Medical and Dental University (TMDU), Tokyo, 113-8519, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 Apr 09; Vol. 524 (3), pp. 533-541. Date of Electronic Publication: 2020 Feb 01.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Models, Biological*
Cell Movement/*genetics
Epithelial Cells/*pathology
Fetus/*metabolism
Gene Expression Regulation, Developmental/*drug effects
Intestines/*pathology
Transforming Growth Factor beta/*pharmacology
Wound Healing/*genetics
Animals ; Cell Movement/drug effects ; Epithelial Cells/drug effects ; Epithelial Cells/metabolism ; Fetus/drug effects ; Mice, Inbred C57BL ; Organoids/drug effects ; Organoids/metabolism ; Up-Regulation/drug effects ; Up-Regulation/genetics ; Wound Healing/drug effects
Czasopismo naukowe
Tytuł :
Cell motion-coordinated fibrillar assembly of soluble collagen I to promote MDCK cell branching formation.
Autorzy :
Wang J; Institute of Biomedical Engineering and Health Sciences, College of Pharmaceutical Engineering and Life Science, Changzhou University, 1 Gehu Road, Wujin District, Changzhou City, Jiangsu Province, 213164, China.
Guo J; Institute of Biomedical Engineering and Health Sciences, College of Pharmaceutical Engineering and Life Science, Changzhou University, 1 Gehu Road, Wujin District, Changzhou City, Jiangsu Province, 213164, China.
Che B; Institute of Biomedical Engineering and Health Sciences, College of Pharmaceutical Engineering and Life Science, Changzhou University, 1 Gehu Road, Wujin District, Changzhou City, Jiangsu Province, 213164, China.
Ouyang M; Institute of Biomedical Engineering and Health Sciences, College of Pharmaceutical Engineering and Life Science, Changzhou University, 1 Gehu Road, Wujin District, Changzhou City, Jiangsu Province, 213164, China. Electronic address: .
Deng L; Institute of Biomedical Engineering and Health Sciences, College of Pharmaceutical Engineering and Life Science, Changzhou University, 1 Gehu Road, Wujin District, Changzhou City, Jiangsu Province, 213164, China. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 Apr 02; Vol. 524 (2), pp. 317-324. Date of Electronic Publication: 2020 Jan 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*
Collagen Type I/*metabolism
Epithelial Cells/*metabolism
Extracellular Matrix/*metabolism
Actin Cytoskeleton/metabolism ; Animals ; Cytoskeleton/metabolism ; Dogs ; Epithelial Cells/cytology ; Madin Darby Canine Kidney Cells ; Myosin Type II/metabolism
Czasopismo naukowe
Tytuł :
Phosphatidic acid regulates subcellular distribution of RA-GEFs critical for chemokine-dependent migration.
Autorzy :
Momoi Y; Department of Biosciences, School of Science, Kitasato University, 1-15-1 Kitasato, Minamiku, Sagamihara, Kanagawa, 252-0337, Japan.
Nishikimi A; Department of Biosciences, School of Science, Kitasato University, 1-15-1 Kitasato, Minamiku, Sagamihara, Kanagawa, 252-0337, Japan.
Du G; Department of Integrative Biology & Pharmacology, University of Texas Health Science at Houston 6431 Fannin St, Houston, TX, 77030, USA.
Kataoka T; Division of Molecular Biology, Department of Biochemistry and Molecular Biology, Kobe University Graduate School of Medicine, 7-5-1 Kusunoki-cho, Chuo-ku, Kobe, 650-0017, Japan.
Katagiri K; Department of Biosciences, School of Science, Kitasato University, 1-15-1 Kitasato, Minamiku, Sagamihara, Kanagawa, 252-0337, Japan. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 Apr 02; Vol. 524 (2), pp. 325-331. Date of Electronic Publication: 2020 Jan 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*
Chemokines/*metabolism
Guanine Nucleotide Exchange Factors/*metabolism
Phosphatidic Acids/*metabolism
Animals ; Cell Line ; HEK293 Cells ; Humans ; Lymphocytes/cytology ; Lymphocytes/metabolism ; Mice
Czasopismo naukowe
Tytuł :
Ceramide synthase 2 knockdown suppresses trophozoite growth, migration, in vitro encystment and excystment of Entamoeba invadens.
Autorzy :
Jáuregui-Wade JM; Department of Biochemistry, CINVESTAV-IPN, P.O. Box 14-740, 07360, Ciudad de México, Mexico.
Cerbón-Solórzano J; Department of Biochemistry, CINVESTAV-IPN, P.O. Box 14-740, 07360, Ciudad de México, Mexico.
Ávila-García R; Department of Biochemistry, CINVESTAV-IPN, P.O. Box 14-740, 07360, Ciudad de México, Mexico.
Ayala-Sumuano JT; Idix S.A. de C.V, Querétaro, Mexico.
Valdés J; Department of Biochemistry, CINVESTAV-IPN, P.O. Box 14-740, 07360, Ciudad de México, Mexico. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 Mar 26; Vol. 524 (1), pp. 135-141. Date of Electronic Publication: 2020 Jan 21.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*
Gene Knockdown Techniques*
Entamoeba/*cytology
Entamoeba/*enzymology
Oxidoreductases/*metabolism
Trophozoites/*enzymology
Trophozoites/*growth & development
Cell Proliferation ; Cell Survival ; Down-Regulation/genetics ; Entamoeba/genetics ; Gene Amplification ; Life Cycle Stages ; Oxidoreductases/genetics ; Protein Transport ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Sphingomyelins/metabolism
Czasopismo naukowe
Tytuł :
Interaction between IDH1 WT and calmodulin and its implications for glioblastoma cell growth and migration.
Autorzy :
Kang S; Natural Product Research Institute, College of Pharmacy, Seoul National University, 1 Gwanak-ro, Gwanak-gu, Seoul, 08826, South Korea. Electronic address: .
Kwon HN; Natural Product Research Institute, College of Pharmacy, Seoul National University, 1 Gwanak-ro, Gwanak-gu, Seoul, 08826, South Korea; Stem Cells and Metabolism Research Program, Faculty of Medicine / Helsinki Institute of Life Science, University of Helsinki, Finland.
Kang S; Natural Product Research Institute, College of Pharmacy, Seoul National University, 1 Gwanak-ro, Gwanak-gu, Seoul, 08826, South Korea; Department of Biochemistry and Molecular Genetics, College of Medicine, University of Illinois at Chicago, Chicago, IL, 60607, USA.
Park S; Natural Product Research Institute, College of Pharmacy, Seoul National University, 1 Gwanak-ro, Gwanak-gu, Seoul, 08826, South Korea. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 Mar 26; Vol. 524 (1), pp. 224-230. Date of Electronic Publication: 2020 Jan 23.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*/drug effects
Brain Neoplasms/*metabolism
Brain Neoplasms/*pathology
Calmodulin/*metabolism
Glioblastoma/*metabolism
Glioblastoma/*pathology
Isocitrate Dehydrogenase/*metabolism
Amino Acid Sequence ; Binding Sites ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Humans ; Isocitrate Dehydrogenase/chemistry ; Models, Molecular ; Protein Binding/drug effects ; Trifluoperazine/pharmacology
Czasopismo naukowe

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