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Wyszukujesz frazę ""Che, Kepeng"" wg kryterium: Autor


Wyświetlanie 1-7 z 7
Tytuł:
Aurintricarboxylic acid is a canonical disruptor of the TAZ-TEAD transcriptional complex.
Autorzy:
Che K; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.
Pobbati AV; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.
Seavey CN; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.; Department of General Surgery, Digestive Disease and Surgery Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.
Fedorov Y; Small Molecule Drug Development Core, Case Western Reserve University School of Medicine, Cleveland, Ohio, United States of America.
Komar AA; Department of Biological, Geological and Environmental Sciences, Center for Gene Regulation in Health and Disease, Cleveland State University, Cleveland, Ohio, United States of America.; Genomic Medicine Institute, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.
Burtscher A; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.
Ma S; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.
Rubin BP; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.; Robert J. Tomsich Pathology and Laboratory Medicine Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.
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Źródło:
PloS one [PLoS One] 2022 Apr 13; Vol. 17 (4), pp. e0266143. Date of Electronic Publication: 2022 Apr 13 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Aurintricarboxylic Acid*
Transcription Factors*/genetics
Transcription Factors*/metabolism
Oncogene Proteins, Fusion
Czasopismo naukowe
Tytuł:
Inhibition of PI3K and MAPK pathways along with KIT inhibitors as a strategy to overcome drug resistance in gastrointestinal stromal tumors.
Autorzy:
Gupta A; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, Ohio, United States of America.
Ma S; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, Ohio, United States of America.
Che K; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, Ohio, United States of America.
Pobbati AV; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, Ohio, United States of America.
Rubin BP; Department of Cancer Biology, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, Ohio, United States of America.; Robert J. Tomsich Pathology and Laboratory Medicine Institute, Cleveland Clinic Foundation, Cleveland, Ohio, United States of America.
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Źródło:
PloS one [PLoS One] 2021 Jul 29; Vol. 16 (7), pp. e0252689. Date of Electronic Publication: 2021 Jul 29 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents/*pharmacology
Drug Resistance, Neoplasm/*drug effects
Gastrointestinal Neoplasms/*pathology
Gastrointestinal Stromal Tumors/*pathology
Phosphatidylinositol 3-Kinases/*metabolism
Protein Kinase Inhibitors/*pharmacology
Proto-Oncogene Proteins c-kit/*antagonists & inhibitors
Apoptosis/drug effects ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Humans ; Imatinib Mesylate/pharmacology ; MAP Kinase Signaling System/drug effects ; Xenograft Model Antitumor Assays
Czasopismo naukowe
    Wyświetlanie 1-7 z 7

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