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Tytuł:
CXCL9:SPP1 macrophage polarity identifies a network of cellular programs that control human cancers.
Autorzy:
Bill R; Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.; AGORA Cancer Research Center, Lausanne, Switzerland.; Swiss Cancer Center Leman, Lausanne, Switzerland.; Center for Systems Biology, Massachusetts General Hospital Research Institute and Harvard Medical School, Boston, MA, USA.; Department of Medical Oncology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.
Wirapati P; Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.; AGORA Cancer Research Center, Lausanne, Switzerland.; Swiss Cancer Center Leman, Lausanne, Switzerland.
Messemaker M; Center for Systems Biology, Massachusetts General Hospital Research Institute and Harvard Medical School, Boston, MA, USA.; Division of Molecular Oncology and Immunology, Netherlands Cancer Institute, Amsterdam, Netherlands.
Roh W; Broad Institute of Harvard and MIT, Cambridge, MA, USA.
Zitti B; Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.; AGORA Cancer Research Center, Lausanne, Switzerland.; Swiss Cancer Center Leman, Lausanne, Switzerland.
Duval F; Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.; AGORA Cancer Research Center, Lausanne, Switzerland.; Swiss Cancer Center Leman, Lausanne, Switzerland.
Kiss M; Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.; AGORA Cancer Research Center, Lausanne, Switzerland.; Swiss Cancer Center Leman, Lausanne, Switzerland.
Park JC; Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.; Massachusetts General Hospital Cancer Center, Charlestown, MA, USA.
Saal TM; Center for Systems Biology, Massachusetts General Hospital Research Institute and Harvard Medical School, Boston, MA, USA.
Hoelzl J; Center for Systems Biology, Massachusetts General Hospital Research Institute and Harvard Medical School, Boston, MA, USA.; Department of Medical Oncology, Heidelberg University Hospital, Heidelberg, Germany.
Tarussio D; Swiss Cancer Center Leman, Lausanne, Switzerland.; Department of Oncology, Center for Experimental Therapeutics, Lausanne University Hospital (CHUV), Lausanne, Switzerland.; Ludwig Institute for Cancer Research, Lausanne, Switzerland.
Benedetti F; Department of Oncology, Center for Experimental Therapeutics, Lausanne University Hospital (CHUV), Lausanne, Switzerland.; Ludwig Institute for Cancer Research, Lausanne, Switzerland.
Tissot S; Swiss Cancer Center Leman, Lausanne, Switzerland.; Department of Oncology, Center for Experimental Therapeutics, Lausanne University Hospital (CHUV), Lausanne, Switzerland.; Ludwig Institute for Cancer Research, Lausanne, Switzerland.
Kandalaft L; AGORA Cancer Research Center, Lausanne, Switzerland.; Swiss Cancer Center Leman, Lausanne, Switzerland.; Department of Oncology, Center for Experimental Therapeutics, Lausanne University Hospital (CHUV), Lausanne, Switzerland.; Ludwig Institute for Cancer Research, Lausanne, Switzerland.
Varrone M; Swiss Cancer Center Leman, Lausanne, Switzerland.; Department of Computational Biology, University of Lausanne (UNIL), Lausanne, Switzerland.; Swiss Institute of Bioinformatics, Lausanne, Switzerland.
Ciriello G; AGORA Cancer Research Center, Lausanne, Switzerland.; Swiss Cancer Center Leman, Lausanne, Switzerland.; Department of Computational Biology, University of Lausanne (UNIL), Lausanne, Switzerland.; Swiss Institute of Bioinformatics, Lausanne, Switzerland.
McKee TA; Division of Clinical Pathology, Diagnostic Department, Geneva University Hospitals (HUG), Geneva, Switzerland.
Monnier Y; Department of Otorhinolaryngology-Head and Neck Surgery, Geneva University Hospitals (HUG), Geneva, Switzerland.
Mermod M; Department of Otorhinolaryngology-Head and Neck Surgery, Geneva University Hospitals (HUG), Geneva, Switzerland.
Blaum EM; Broad Institute of Harvard and MIT, Cambridge, MA, USA.; Massachusetts General Hospital Cancer Center, Charlestown, MA, USA.
Gushterova I; Broad Institute of Harvard and MIT, Cambridge, MA, USA.; Massachusetts General Hospital Cancer Center, Charlestown, MA, USA.
Gonye ALK; Broad Institute of Harvard and MIT, Cambridge, MA, USA.; Massachusetts General Hospital Cancer Center, Charlestown, MA, USA.
Hacohen N; Broad Institute of Harvard and MIT, Cambridge, MA, USA.; Massachusetts General Hospital Cancer Center, Charlestown, MA, USA.
Getz G; Broad Institute of Harvard and MIT, Cambridge, MA, USA.; Massachusetts General Hospital Cancer Center, Charlestown, MA, USA.; Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.
Mempel TR; Center for Systems Biology, Massachusetts General Hospital Research Institute and Harvard Medical School, Boston, MA, USA.; Center for Immunology and Inflammatory Diseases, Massachusetts General Hospital, Boston, MA, USA.
Klein AM; Department of Systems Biology, Harvard Medical School, Boston, MA, USA.
Weissleder R; Center for Systems Biology, Massachusetts General Hospital Research Institute and Harvard Medical School, Boston, MA, USA.; Department of Systems Biology, Harvard Medical School, Boston, MA, USA.; Department of Radiology, Massachusetts General Hospital, Boston, MA, USA.
Faquin WC; Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.; Department of Otolaryngology Head and Neck Surgery, Massachusetts Eye and Ear Infirmary, Harvard Medical School, Boston, MA, USA.
Sadow PM; Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.; Department of Otolaryngology Head and Neck Surgery, Massachusetts Eye and Ear Infirmary, Harvard Medical School, Boston, MA, USA.
Lin D; Department of Otolaryngology Head and Neck Surgery, Massachusetts Eye and Ear Infirmary, Harvard Medical School, Boston, MA, USA.
Pai SI; Center for Systems Biology, Massachusetts General Hospital Research Institute and Harvard Medical School, Boston, MA, USA.; Broad Institute of Harvard and MIT, Cambridge, MA, USA.; Massachusetts General Hospital Cancer Center, Charlestown, MA, USA.; Department of Surgery, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.
Sade-Feldman M; Broad Institute of Harvard and MIT, Cambridge, MA, USA.; Massachusetts General Hospital Cancer Center, Charlestown, MA, USA.
Pittet MJ; Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.; AGORA Cancer Research Center, Lausanne, Switzerland.; Swiss Cancer Center Leman, Lausanne, Switzerland.; Center for Systems Biology, Massachusetts General Hospital Research Institute and Harvard Medical School, Boston, MA, USA.; Ludwig Institute for Cancer Research, Lausanne, Switzerland.; Department of Oncology, Geneva University Hospitals (HUG), Geneva, Switzerland.
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Źródło:
Science (New York, N.Y.) [Science] 2023 Aug 04; Vol. 381 (6657), pp. 515-524. Date of Electronic Publication: 2023 Aug 03.
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CXCL9*/analysis
Chemokine CXCL9*/metabolism
Head and Neck Neoplasms*/immunology
Head and Neck Neoplasms*/pathology
Macrophages*/immunology
Osteopontin*/analysis
Osteopontin*/metabolism
Squamous Cell Carcinoma of Head and Neck*/immunology
Squamous Cell Carcinoma of Head and Neck*/pathology
Tumor Microenvironment*
Cell Polarity*/immunology
Humans ; Prognosis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
C-X-C Motif Chemokine Ligand 9 and Its CXCR3 Receptor Are the Salt and Pepper for T Cells Trafficking in a Mouse Model of Gaucher Disease.
Autorzy:
Magnusen AF; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.
Rani R; Division of Immunobiology, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.
McKay MA; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.
Hatton SL; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.
Nyamajenjere TC; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.
Magnusen DNA; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.
Köhl J; Institute for Systemic Inflammation Research, University of Lübeck, 23562 Lübeck, Germany.; Department of Pediatrics and Division of Immunobiology, Cincinnati Children's Hospital Medical Center, College of Medicine, University of Cincinnati, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.
Grabowski GA; Department of Molecular Genetics, Biochemistry and Microbiology, Division of Human Genetics, Cincinnati Children's Hospital Medical Center, College of Medicine, University of Cincinnati, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.; Department of Pediatrics, Division of Human Genetics, Cincinnati Children's Hospital Medical Center, College of Medicine, University of Cincinnati, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.
Pandey MK; Department of Pediatrics, Division of Human Genetics, Cincinnati Children's Hospital Medical Center, College of Medicine, University of Cincinnati, 3333 Burnet Avenue, Cincinnati, OH 45229, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Nov 24; Vol. 22 (23). Date of Electronic Publication: 2021 Nov 24.
Typ publikacji:
Journal Article
MeSH Terms:
Disease Models, Animal*
CD8-Positive T-Lymphocytes/*immunology
Chemokine CXCL9/*metabolism
Gaucher Disease/*immunology
Glucosylceramidase/*physiology
Inflammation/*immunology
Receptors, CXCR3/*metabolism
Animals ; CD8-Positive T-Lymphocytes/pathology ; Chemokine CXCL9/genetics ; Gaucher Disease/metabolism ; Gaucher Disease/pathology ; Inflammation/metabolism ; Inflammation/pathology ; Ligands ; Macrophages/immunology ; Macrophages/pathology ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Receptors, CXCR3/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CXCL9 as a key biomarker of vitiligo activity and prediction of the success of cultured melanocyte transplantation.
Autorzy:
Lin F; Department of Dermatology, Hangzhou Third People's Hospital, Affiliated Hangzhou Dermatology Hospital, Zhejiang University School of Medicine, Hangzhou, China.
Hu W; Department of Dermatology, Hangzhou Third People's Hospital, Affiliated Hangzhou Dermatology Hospital, Zhejiang University School of Medicine, Hangzhou, China.
Xu W; Department of Dermatology, Hangzhou Third People's Hospital, Affiliated Hangzhou Dermatology Hospital, Zhejiang University School of Medicine, Hangzhou, China.
Zhou M; Department of Dermatology, Hangzhou Third People's Hospital, Affiliated Hangzhou Dermatology Hospital, Zhejiang University School of Medicine, Hangzhou, China. .
Xu AE; Department of Dermatology, Hangzhou Third People's Hospital, Affiliated Hangzhou Dermatology Hospital, Zhejiang University School of Medicine, Hangzhou, China. .
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Źródło:
Scientific reports [Sci Rep] 2021 Sep 14; Vol. 11 (1), pp. 18298. Date of Electronic Publication: 2021 Sep 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Chemokine CXCL9/*blood
Melanocytes/*transplantation
Vitiligo/*therapy
Adult ; Biomarkers/blood ; Blister/metabolism ; Case-Control Studies ; Chemokine CXCL9/metabolism ; Female ; Humans ; Male ; Melanocytes/metabolism ; Skin Pigmentation ; Treatment Outcome ; Vitiligo/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The effects of T-DXd on the expression of HLA class I and chemokines CXCL9/10/11 in HER2-overexpressing gastric cancer cells.
Autorzy:
Nakajima S; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.; Department of Obesity and Inflammation Research, Fukushima Medical University School of Medicine, Fukushima, Japan.
Mimura K; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.; Department of Blood Transfusion and Transplantation Immunology, Fukushima Medical University School of Medicine, Fukushima, Japan.
Matsumoto T; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Thar Min AK; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Ito M; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Nakano H; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Neupane P; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Kanke Y; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Okayama H; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Saito M; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Momma T; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Watanabe Y; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Hanayama H; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Hayase S; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Saze Z; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan.
Kono K; Department of Gastrointestinal Tract Surgery, Fukushima Medical University School of Medicine, 1 Hikariga-oka, Fukushima City, Fukushima, 960-1295, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2021 Aug 19; Vol. 11 (1), pp. 16891. Date of Electronic Publication: 2021 Aug 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Camptothecin/*analogs & derivatives
Chemokine CXCL10/*genetics
Chemokine CXCL11/*genetics
Chemokine CXCL9/*genetics
Histocompatibility Antigens Class I/*metabolism
Immunoconjugates/*therapeutic use
Receptor, ErbB-2/*metabolism
Stomach Neoplasms/*drug therapy
Stomach Neoplasms/*immunology
Trastuzumab/*therapeutic use
Camptothecin/pharmacology ; Camptothecin/therapeutic use ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Chemokine CXCL10/metabolism ; Chemokine CXCL11/metabolism ; Chemokine CXCL9/metabolism ; Chemotactic Factors/pharmacology ; DNA Damage ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; Immunoconjugates/pharmacology ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Stomach Neoplasms/genetics ; Trastuzumab/pharmacology ; Up-Regulation/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Pro-Inflammatory Chemokines CXCL9, CXCL10 and CXCL11 Are Upregulated Following SARS-CoV-2 Infection in an AKT-Dependent Manner.
Autorzy:
Callahan V; National Center for Biodefense and Infectious Diseases, School of Systems Biology, George Mason University, Manassas, VA 20110, USA.
Hawks S; Department of Biomedical Science and Pathobiology, Virginia-Maryland College of Veterinary Medicine, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.
Crawford MA; Division of Infectious Diseases and International Health, Department of Medicine, University of Virginia, Charlottesville, VA 22908, USA.
Lehman CW; Department of Biomedical Science and Pathobiology, Virginia-Maryland College of Veterinary Medicine, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.
Morrison HA; Department of Biomedical Science and Pathobiology, Virginia-Maryland College of Veterinary Medicine, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.
Ivester HM; Graduate Program in Translational Biology, Medicine and Health, Virginia Polytechnic Institute and State University, Roanoke, VA 24061, USA.
Akhrymuk I; Department of Biomedical Science and Pathobiology, Virginia-Maryland College of Veterinary Medicine, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.
Boghdeh N; National Center for Biodefense and Infectious Diseases, School of Systems Biology, George Mason University, Manassas, VA 20110, USA.
Flor R; National Center for Biodefense and Infectious Diseases, School of Systems Biology, George Mason University, Manassas, VA 20110, USA.
Finkielstein CV; Integrated Cellular Responses Laboratory, Department of Biological Sciences and Center for Drug Discovery, Fralin Biomedical Research Institute, Virginia Polytechnic Institute and State University, Roanoke, VA 24016, USA.
Allen IC; Department of Biomedical Science and Pathobiology, Virginia-Maryland College of Veterinary Medicine, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.; Virginia Tech Carilion School of Medicine, Virginia Polytechnic Institute and State University, Blacksburg, VA 24016, USA.; Center for Zoonotic and Arthropod-borne Pathogens, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.
Weger-Lucarelli J; Department of Biomedical Science and Pathobiology, Virginia-Maryland College of Veterinary Medicine, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.; Center for Zoonotic and Arthropod-borne Pathogens, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.
Duggal N; Department of Biomedical Science and Pathobiology, Virginia-Maryland College of Veterinary Medicine, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.; Center for Zoonotic and Arthropod-borne Pathogens, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.
Hughes MA; Division of Infectious Diseases and International Health, Department of Medicine, University of Virginia, Charlottesville, VA 22908, USA.
Kehn-Hall K; National Center for Biodefense and Infectious Diseases, School of Systems Biology, George Mason University, Manassas, VA 20110, USA.; Department of Biomedical Science and Pathobiology, Virginia-Maryland College of Veterinary Medicine, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.; Center for Zoonotic and Arthropod-borne Pathogens, Virginia Polytechnic Institute and State University, Blacksburg, VA 24060, USA.
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Źródło:
Viruses [Viruses] 2021 Jun 03; Vol. 13 (6). Date of Electronic Publication: 2021 Jun 03.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Up-Regulation*
COVID-19/*immunology
Chemokine CXCL10/*genetics
Chemokine CXCL11/*genetics
Chemokine CXCL9/*genetics
Proto-Oncogene Proteins c-akt/*metabolism
Angiotensin-Converting Enzyme 2/genetics ; Animals ; Cell Line ; Chemokine CXCL10/immunology ; Chemokine CXCL11/immunology ; Chemokine CXCL9/immunology ; Cytokine Release Syndrome/genetics ; Cytokine Release Syndrome/immunology ; Epithelial Cells/immunology ; Epithelial Cells/virology ; Female ; Humans ; Inflammation ; Lung/cytology ; Mice ; Mice, Transgenic ; Proto-Oncogene Proteins c-akt/genetics ; Signal Transduction/genetics ; Signal Transduction/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Characterization of Type I Interferon-Associated Chemokines and Cytokines in Lacrimal Glands of Nonobese Diabetic Mice.
Autorzy:
Allred MG; Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, IA 52242, USA.; Immunology Graduate Program, University of Iowa, Iowa City, IA 52242, USA.
Chimenti MS; Iowa Institute of Human Genetics, Carver College of Medicine, University of Iowa, Iowa City, IA 52242, USA.
Ciecko AE; Department of Pediatrics, Medical College of Wisconsin, Milwaukee, WI 53226, USA.; Department of Microbiology and Immunology, Medical College of Wisconsin, Milwaukee, WI 53226, USA.; Max McGee National Research Center for Juvenile Diabetes, Medical College of Wisconsin, Milwaukee, WI 53226, USA.
Chen YG; Department of Pediatrics, Medical College of Wisconsin, Milwaukee, WI 53226, USA.; Department of Microbiology and Immunology, Medical College of Wisconsin, Milwaukee, WI 53226, USA.; Max McGee National Research Center for Juvenile Diabetes, Medical College of Wisconsin, Milwaukee, WI 53226, USA.
Lieberman SM; Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, IA 52242, USA.; Immunology Graduate Program, University of Iowa, Iowa City, IA 52242, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Apr 05; Vol. 22 (7). Date of Electronic Publication: 2021 Apr 05.
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CXCL10/*immunology
Chemokine CXCL9/*immunology
Interleukins/*immunology
Lacrimal Apparatus/*immunology
Signal Transduction/*immunology
T-Lymphocytes, Regulatory/*immunology
Animals ; Chemokine CXCL10/genetics ; Chemokine CXCL9/genetics ; Inflammation/genetics ; Inflammation/immunology ; Inflammation/pathology ; Interleukins/genetics ; Lacrimal Apparatus/pathology ; Mice ; Mice, Inbred NOD ; Mice, Knockout ; Receptors, CXCR3/genetics ; Receptors, CXCR3/immunology ; Signal Transduction/genetics ; T-Lymphocytes, Regulatory/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CXCL9 secreted by tumor-associated dendritic cells up-regulates PD-L1 expression in bladder cancer cells by activating the CXCR3 signaling.
Autorzy:
Xiu W; Department of Laboratory Medicine, West China Second University Hospital, Sichuan University, Chengdu, 610041, PR China.; Key Laboratory of Birth Defects and Related Diseases of Women and Children (Sichuan University), Ministry of Education, Chengdu, 610041, PR China.; Department of Thoracic Oncology and State Key Laboratory of Biotherapy, Cancer Center, West China Hospital, Sichuan University, Chengdu, 610041, PR China.
Luo J; Department of Laboratory Medicine, West China Second University Hospital, Sichuan University, Chengdu, 610041, PR China. .; Key Laboratory of Birth Defects and Related Diseases of Women and Children (Sichuan University), Ministry of Education, Chengdu, 610041, PR China. .
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Źródło:
BMC immunology [BMC Immunol] 2021 Jan 06; Vol. 22 (1), pp. 3. Date of Electronic Publication: 2021 Jan 06.
Typ publikacji:
Journal Article
MeSH Terms:
B7-H1 Antigen/*metabolism
Chemokine CXCL9/*immunology
Dendritic Cells/*immunology
Urinary Bladder Neoplasms/*metabolism
Cell Line, Tumor ; Chemokine CXCL9/metabolism ; Coculture Techniques ; Dendritic Cells/metabolism ; Humans ; Lymphocyte Activation ; Phosphorylation ; Proto-Oncogene Proteins c-akt/metabolism ; Receptors, CXCR3/metabolism ; STAT3 Transcription Factor/metabolism ; Signal Transduction ; T-Lymphocytes/immunology ; Urinary Bladder Neoplasms/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CXCL9, CXCL10, and CXCL11; biomarkers of pulmonary inflammation associated with autoimmunity in patients with collagen vascular diseases-associated interstitial lung disease and interstitial pneumonia with autoimmune features.
Autorzy:
Kameda M; Department of Respiratory Medicine and Allergology, Sapporo Medical University School of Medicine, Sapporo, Hokkaido, Japan.
Otsuka M; Department of Respiratory Medicine, Sapporo-Kosei General Hospital, Sapporo, Hokkaido, Japan.
Chiba H; Department of Respiratory Medicine and Allergology, Sapporo Medical University School of Medicine, Sapporo, Hokkaido, Japan.
Kuronuma K; Department of Respiratory Medicine and Allergology, Sapporo Medical University School of Medicine, Sapporo, Hokkaido, Japan.
Hasegawa T; Sysmex Corporation, Kobe, Japan.
Takahashi H; Department of Rheumatology, Sapporo Medical University School of Medicine, Sapporo, Hokkaido, Japan.
Takahashi H; Department of Respiratory Medicine and Allergology, Sapporo Medical University School of Medicine, Sapporo, Hokkaido, Japan.
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Źródło:
PloS one [PLoS One] 2020 Nov 02; Vol. 15 (11), pp. e0241719. Date of Electronic Publication: 2020 Nov 02 (Print Publication: 2020).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Biomarkers/*blood
Chemokine CXCL10/*blood
Chemokine CXCL11/*blood
Chemokine CXCL9/*blood
Lung Diseases, Interstitial/*diagnosis
Vascular Diseases/*complications
Aged ; Autoimmunity ; Biomarkers/analysis ; Bronchoalveolar Lavage Fluid/chemistry ; Bronchoalveolar Lavage Fluid/cytology ; Bronchoalveolar Lavage Fluid/immunology ; C-Reactive Protein/analysis ; Chemokine CXCL10/analysis ; Chemokine CXCL11/analysis ; Chemokine CXCL9/analysis ; Collagen/metabolism ; Female ; Humans ; Idiopathic Pulmonary Fibrosis/diagnosis ; Lung Diseases, Interstitial/etiology ; Lymphocytes/cytology ; Lymphocytes/metabolism ; Macrophages/cytology ; Macrophages/metabolism ; Male ; Middle Aged ; Retrospective Studies ; Vascular Diseases/pathology ; Vital Capacity
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Peyer's patches-derived CD11b B cells recruit regulatory T cells through CXCL9 in dextran sulphate sodium-induced colitis.
Autorzy:
Wang Z; Department of Immunology, School of Basic Medical Sciences, Institute of Biomedical Sciences, Fudan University, Shanghai, China.
Zhang H; Department of Immunology, School of Basic Medical Sciences, Institute of Biomedical Sciences, Fudan University, Shanghai, China.
Liu R; Department of Immunology, School of Basic Medical Sciences, Institute of Biomedical Sciences, Fudan University, Shanghai, China.
Qian T; Department of Immunology, School of Basic Medical Sciences, Institute of Biomedical Sciences, Fudan University, Shanghai, China.
Liu J; Department of Immunology, School of Basic Medical Sciences, Institute of Biomedical Sciences, Fudan University, Shanghai, China.
Huang E; Department of Pathology, The University of Hong Kong, Hong Kong, China.
Lu Z; Department of Immunology, School of Basic Medical Sciences, Institute of Biomedical Sciences, Fudan University, Shanghai, China.
Zhao C; Northfield Mount Hermon School, Mount Hermon, MA, USA.
Wang L; Department of Immunology, School of Basic Medical Sciences, Institute of Biomedical Sciences, Fudan University, Shanghai, China.; Biotherapy Research Centre, Fudan University, Shanghai, China.
Chu Y; Department of Immunology, School of Basic Medical Sciences, Institute of Biomedical Sciences, Fudan University, Shanghai, China.; Biotherapy Research Centre, Fudan University, Shanghai, China.
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Źródło:
Immunology [Immunology] 2018 Nov; Vol. 155 (3), pp. 356-366. Date of Electronic Publication: 2018 Jul 26.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
B-Lymphocytes/*immunology
CD11b Antigen/*immunology
Chemokine CXCL9/*immunology
Colitis/*immunology
Peyer's Patches/*immunology
T-Lymphocytes, Regulatory/*immunology
Animals ; B-Lymphocytes/pathology ; CD11b Antigen/genetics ; Chemokine CXCL9/genetics ; Colitis/chemically induced ; Colitis/genetics ; Colitis/pathology ; Dextran Sulfate/toxicity ; Disease Models, Animal ; Mice ; Mice, Knockout ; Peyer's Patches/pathology ; T-Lymphocytes, Regulatory/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Interleukin-1β induces CXCR3-mediated chemotaxis to promote umbilical cord mesenchymal stem cell transendothelial migration.
Autorzy:
Guo YC; Institute of Anatomy and Cell Biology, School of Medicine, National Yang Ming University, Peitou, Taipei, 112, Taiwan, Republic of China.
Chiu YH; Institute of Anatomy and Cell Biology, School of Medicine, National Yang Ming University, Peitou, Taipei, 112, Taiwan, Republic of China.
Chen CP; Division of High Risk Pregnancy, Mackay Memorial Hospital, Taipei, Taiwan, Republic of China.
Wang HS; Institute of Anatomy and Cell Biology, School of Medicine, National Yang Ming University, Peitou, Taipei, 112, Taiwan, Republic of China. .
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Źródło:
Stem cell research & therapy [Stem Cell Res Ther] 2018 Oct 25; Vol. 9 (1), pp. 281. Date of Electronic Publication: 2018 Oct 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Chemokine CXCL9/*genetics
Human Umbilical Vein Endothelial Cells/*drug effects
Interleukin-1beta/*pharmacology
Mesenchymal Stem Cells/*drug effects
RNA, Messenger/*genetics
Receptors, CXCR3/*genetics
Acetamides/pharmacology ; Chemokine CXCL9/metabolism ; Chemotaxis/drug effects ; Coculture Techniques ; Cycloheximide/pharmacology ; Diffusion Chambers, Culture ; Gene Expression Regulation ; Human Umbilical Vein Endothelial Cells/cytology ; Human Umbilical Vein Endothelial Cells/metabolism ; Humans ; Imidazoles/pharmacology ; Mesenchymal Stem Cells/cytology ; Mesenchymal Stem Cells/metabolism ; Pyridines/pharmacology ; Pyrimidinones/pharmacology ; RNA, Messenger/metabolism ; Receptors, CXCR3/antagonists & inhibitors ; Receptors, CXCR3/metabolism ; Umbilical Cord/cytology ; p38 Mitogen-Activated Protein Kinases/antagonists & inhibitors ; p38 Mitogen-Activated Protein Kinases/genetics ; p38 Mitogen-Activated Protein Kinases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Induction of Th1 chemokine secretion in dermal fibroblasts by vanadium pentoxide.
Autorzy:
Fallahi P; Department of Clinical and Experimental Medicine, University of Pisa, I‑56126 Pisa, Italy.
Foddis R; Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, I‑56126 Pisa, Italy.
Elia G; Department of Clinical and Experimental Medicine, University of Pisa, I‑56126 Pisa, Italy.
Ragusa F; Department of Clinical and Experimental Medicine, University of Pisa, I‑56126 Pisa, Italy.
Patrizio A; Department of Clinical and Experimental Medicine, University of Pisa, I‑56126 Pisa, Italy.
Guglielmi G; U.O. Medicina Preventiva del Lavoro, Azienda Ospedaliero‑Universitaria Pisana, I‑56124 Pisa, Italy.
Frenzilli G; Department of Clinical and Experimental Medicine, University of Pisa, I‑56126 Pisa, Italy.
Benvenga S; Department of Clinical and Experimental Medicine, University of Messina, I‑98125 Messina, Italy.
Cristaudo A; Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, I‑56126 Pisa, Italy.
Antonelli A; Department of Clinical and Experimental Medicine, University of Pisa, I‑56126 Pisa, Italy.
Ferrari SM; Department of Clinical and Experimental Medicine, University of Pisa, I‑56126 Pisa, Italy.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2018 May; Vol. 17 (5), pp. 6914-6918. Date of Electronic Publication: 2018 Mar 09.
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CXCL10/*immunology
Chemokine CXCL9/*immunology
Fibroblasts/*drug effects
Vanadium Compounds/*immunology
Cell Proliferation/drug effects ; Cell Survival/drug effects ; Cells, Cultured ; Chemokine CXCL10/analysis ; Chemokine CXCL9/analysis ; Fibroblasts/cytology ; Fibroblasts/immunology ; Humans ; Skin/cytology ; Skin/drug effects ; Skin/immunology ; Vanadium Compounds/adverse effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Spinal CXCL9 and CXCL11 are not involved in neuropathic pain despite an upregulation in the spinal cord following spinal nerve injury.
Autorzy:
Wu XB; 1 Pain Research Laboratory, Institute of Nautical Medicine, Jiangsu Key Laboratory of Neuroregeneration, Nantong University, Nantong, Jiangsu, China.
He LN; 1 Pain Research Laboratory, Institute of Nautical Medicine, Jiangsu Key Laboratory of Neuroregeneration, Nantong University, Nantong, Jiangsu, China.
Jiang BC; 1 Pain Research Laboratory, Institute of Nautical Medicine, Jiangsu Key Laboratory of Neuroregeneration, Nantong University, Nantong, Jiangsu, China.
Shi H; 1 Pain Research Laboratory, Institute of Nautical Medicine, Jiangsu Key Laboratory of Neuroregeneration, Nantong University, Nantong, Jiangsu, China.
Bai XQ; 1 Pain Research Laboratory, Institute of Nautical Medicine, Jiangsu Key Laboratory of Neuroregeneration, Nantong University, Nantong, Jiangsu, China.
Zhang WW; 1 Pain Research Laboratory, Institute of Nautical Medicine, Jiangsu Key Laboratory of Neuroregeneration, Nantong University, Nantong, Jiangsu, China.
Gao YJ; 1 Pain Research Laboratory, Institute of Nautical Medicine, Jiangsu Key Laboratory of Neuroregeneration, Nantong University, Nantong, Jiangsu, China.; 2 Co-innovation Center of Neuroregeneration, Nantong University, Nantong, Jiangsu, China.
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Źródło:
Molecular pain [Mol Pain] 2018 Jan-Dec; Vol. 14, pp. 1744806918777401. Date of Electronic Publication: 2018 Apr 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Chemokine CXCL11/*genetics
Chemokine CXCL9/*genetics
Neuralgia/*genetics
Spinal Cord/*metabolism
Spinal Nerves/*injuries
Spinal Nerves/*metabolism
Up-Regulation/*genetics
Animals ; Astrocytes/metabolism ; Astrocytes/pathology ; Chemokine CXCL11/metabolism ; Chemokine CXCL9/metabolism ; Enzyme Activation ; Excitatory Postsynaptic Potentials ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Ligation ; Male ; Mice, Inbred ICR ; Neural Inhibition ; Neuralgia/enzymology ; Neuralgia/pathology ; Neurons/metabolism ; Neurons/pathology ; Spinal Cord/pathology ; Spinal Cord/physiopathology ; Spinal Nerves/pathology ; Spinal Nerves/physiopathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Hepatectomy leads to loss of TRAIL-expressing liver NK cells via downregulation of the CXCL9-CXCR3 axis in mice.
Autorzy:
Yano T; Department of Gastroenterological and Transplant Surgery, Applied Life Sciences, Institute of Biomedical & Health Sciences, Hiroshima University, Hiroshima, Japan.
Ohira M; Department of Gastroenterological and Transplant Surgery, Applied Life Sciences, Institute of Biomedical & Health Sciences, Hiroshima University, Hiroshima, Japan.
Nakano R; Department of Gastroenterological and Transplant Surgery, Applied Life Sciences, Institute of Biomedical & Health Sciences, Hiroshima University, Hiroshima, Japan.
Tanaka Y; Department of Gastroenterological and Transplant Surgery, Applied Life Sciences, Institute of Biomedical & Health Sciences, Hiroshima University, Hiroshima, Japan.
Ohdan H; Department of Gastroenterological and Transplant Surgery, Applied Life Sciences, Institute of Biomedical & Health Sciences, Hiroshima University, Hiroshima, Japan.
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Źródło:
PloS one [PLoS One] 2017 Oct 31; Vol. 12 (10), pp. e0186997. Date of Electronic Publication: 2017 Oct 31 (Print Publication: 2017).
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CXCL9/*genetics
Hepatectomy/*methods
Killer Cells, Natural/*metabolism
Liver/*metabolism
Receptors, CXCR3/*genetics
TNF-Related Apoptosis-Inducing Ligand/*genetics
Animals ; Chemokine CXCL9/metabolism ; Down-Regulation ; Female ; Flow Cytometry ; Gene Expression ; Male ; Mice, Inbred C57BL ; Mice, Knockout ; Perioperative Period ; Receptors, CXCR3/metabolism ; Reverse Transcriptase Polymerase Chain Reaction ; TNF-Related Apoptosis-Inducing Ligand/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Thioredoxin-1, chemokine (C-X-C motif) ligand-9 and interferon-γ expression in the neoplastic cells and macrophages of Hodgkin lymphoma: clinicopathologic correlations and potential prognostic implications.
Autorzy:
P Vassilakopoulos T; a Department of Haematology, Laikon General Hospital, National and Kapodistrian University of Athens, Athens, Greece.
Levidou G; b Department of Pathology , Laikon General Hospital, National and Kapodistrian University of Athens , Athens , Greece.
Milionis V; b Department of Pathology , Laikon General Hospital, National and Kapodistrian University of Athens , Athens , Greece.
Hartmann S; c Dr Senckenberg Institute of Pathology, Goethe University Hospital Frankfurt , Frankfurt , Germany.
Lakiotaki E; b Department of Pathology , Laikon General Hospital, National and Kapodistrian University of Athens , Athens , Greece.
Sepsa A; b Department of Pathology , Laikon General Hospital, National and Kapodistrian University of Athens , Athens , Greece.
Thymara I; b Department of Pathology , Laikon General Hospital, National and Kapodistrian University of Athens , Athens , Greece.
Ntailiani P; b Department of Pathology , Laikon General Hospital, National and Kapodistrian University of Athens , Athens , Greece.
Spanou K; b Department of Pathology , Laikon General Hospital, National and Kapodistrian University of Athens , Athens , Greece.
K Angelopoulou M; a Department of Haematology, Laikon General Hospital, National and Kapodistrian University of Athens, Athens, Greece.
P Siakantaris M; a Department of Haematology, Laikon General Hospital, National and Kapodistrian University of Athens, Athens, Greece.
Moschogiannis M; d Department of Haematology , Athens Medical Center , Psychikon , Athens , Greece.
A Pangalis G; d Department of Haematology , Athens Medical Center , Psychikon , Athens , Greece.
Panayiotidis P; c Dr Senckenberg Institute of Pathology, Goethe University Hospital Frankfurt , Frankfurt , Germany.
Konstantopoulos K; c Dr Senckenberg Institute of Pathology, Goethe University Hospital Frankfurt , Frankfurt , Germany.
Patsouris E; b Department of Pathology , Laikon General Hospital, National and Kapodistrian University of Athens , Athens , Greece.
Hansmann ML; c Dr Senckenberg Institute of Pathology, Goethe University Hospital Frankfurt , Frankfurt , Germany.
Korkolopoulou P; b Department of Pathology , Laikon General Hospital, National and Kapodistrian University of Athens , Athens , Greece.
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Źródło:
Leukemia & lymphoma [Leuk Lymphoma] 2017 Sep; Vol. 58 (9), pp. 1-13. Date of Electronic Publication: 2017 Jun 02.
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CXCL9/*genetics
Hodgkin Disease/*genetics
Interferon-gamma/*genetics
Macrophages/*metabolism
Thioredoxins/*genetics
Adolescent ; Adult ; Aged ; Chemokine CXCL9/metabolism ; Female ; Gene Expression ; Histiocytes/metabolism ; Histiocytes/pathology ; Hodgkin Disease/metabolism ; Hodgkin Disease/mortality ; Hodgkin Disease/pathology ; Humans ; Immunohistochemistry ; Interferon-gamma/metabolism ; Male ; Middle Aged ; Neoplasm Staging ; Prognosis ; Proportional Hazards Models ; Thioredoxins/metabolism ; Tumor Microenvironment ; Young Adult
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CXCL9, a promising biomarker in the diagnosis of chronic Q fever.
Autorzy:
Jansen AFM; Department of Internal Medicine 463, Radboud center for Infectious Diseases (RCI), Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands. .; Radboud Expert Center for Q fever, Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands. .
Schoffelen T; Department of Internal Medicine 463, Radboud center for Infectious Diseases (RCI), Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.; Radboud Expert Center for Q fever, Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.
Textoris J; Université Claude Bernard Lyon 1, Hospices Civils de Lyon, bioMérieux; EA7426 'Pathophysiology of injury induced immunosuppression (PI3)', Hôpital E. Herriot, Lyon, France.
Mege JL; URMITE, Aix-Marseille University, Marseille, France.
Nabuurs-Franssen M; Department of Medical Microbiology and Infectious Diseases, Canisius Wilhelmina Hospital, Nijmegen, The Netherlands.
Raijmakers RPH; Department of Internal Medicine 463, Radboud center for Infectious Diseases (RCI), Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.; Radboud Expert Center for Q fever, Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.
Netea MG; Department of Internal Medicine 463, Radboud center for Infectious Diseases (RCI), Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.; Radboud Expert Center for Q fever, Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.
Joosten LAB; Department of Internal Medicine 463, Radboud center for Infectious Diseases (RCI), Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.; Radboud Expert Center for Q fever, Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.
Bleeker-Rovers CP; Department of Internal Medicine 463, Radboud center for Infectious Diseases (RCI), Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.; Radboud Expert Center for Q fever, Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands.
van Deuren M; Department of Internal Medicine 463, Radboud center for Infectious Diseases (RCI), Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands. .; Radboud Expert Center for Q fever, Radboud University Medical Center, P.O. Box 9101, 6500 HB, Nijmegen, The Netherlands. .
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Źródło:
BMC infectious diseases [BMC Infect Dis] 2017 Aug 09; Vol. 17 (1), pp. 556. Date of Electronic Publication: 2017 Aug 09.
Typ publikacji:
Journal Article
MeSH Terms:
Biomarkers/*blood
Chemokine CXCL9/*blood
Q Fever/*diagnosis
Case-Control Studies ; Chemokine CCL8/blood ; Chemokine CCL8/genetics ; Chemokine CXCL10/blood ; Chemokine CXCL10/genetics ; Chemokine CXCL11/blood ; Chemokine CXCL11/genetics ; Chemokine CXCL9/genetics ; Coxiella burnetii/pathogenicity ; Gene Expression Profiling ; Gene Expression Regulation ; Humans ; Leukocytes, Mononuclear/microbiology ; Q Fever/blood ; Q Fever/genetics ; Q Fever/therapy
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
IL-1α Modulates IFN-γ-Induced Production of CXCL9/MIG during Herpes Simplex Virus Type-1 Corneal Infection.
Autorzy:
Armstrong S; Department of Biology, University of West Georgia , Carrollton, GA, USA.
Arroyo M; Department of Biology, University of West Georgia , Carrollton, GA, USA.
Decker-Pulice K; Department of Biology, University of West Georgia , Carrollton, GA, USA.
Lane M; Department of Biology, University of West Georgia , Carrollton, GA, USA.
Mckinney M; Department of Biology, University of West Georgia , Carrollton, GA, USA.
Molesworth-Kenyon SJ; Department of Biology, University of West Georgia , Carrollton, GA, USA.
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Źródło:
Current eye research [Curr Eye Res] 2021 Mar; Vol. 46 (3), pp. 309-317. Date of Electronic Publication: 2020 Sep 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Chemokine CXCL9/*metabolism
Eye Infections, Viral/*metabolism
Herpesvirus 1, Human/*genetics
Interferon-gamma/*pharmacology
Interleukin-1alpha/*metabolism
Keratitis, Herpetic/*metabolism
Cornea/metabolism ; Enzyme-Linked Immunosorbent Assay ; Eye Infections, Viral/virology ; Herpes Simplex/genetics ; Humans ; Keratitis, Herpetic/virology ; Neutrophils/drug effects ; RNA, Viral/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Inhibition of the CXCL9-CXCR3 axis suppresses the progression of experimental apical periodontitis by blocking macrophage migration and activation.
Autorzy:
Hasegawa T; Division of Operative Dentistry, Department of Ecological Dentistry, Graduate School of Dentistry, Tohoku University, 4-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Venkata Suresh V; Division of Operative Dentistry, Department of Ecological Dentistry, Graduate School of Dentistry, Tohoku University, 4-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Yahata Y; Division of Operative Dentistry, Department of Ecological Dentistry, Graduate School of Dentistry, Tohoku University, 4-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Nakano M; Division of Operative Dentistry, Department of Ecological Dentistry, Graduate School of Dentistry, Tohoku University, 4-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Suzuki S; Division of Operative Dentistry, Department of Ecological Dentistry, Graduate School of Dentistry, Tohoku University, 4-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.
Suzuki S; Division of Periodontology and Endodontology, Department of Ecological Dentistry, Graduate School of Dentistry, Tohoku University, Sendai, Miyagi, Japan.
Yamada S; Division of Periodontology and Endodontology, Department of Ecological Dentistry, Graduate School of Dentistry, Tohoku University, Sendai, Miyagi, Japan.
Kitaura H; Division of Orthodontics and Dentofacial Orthopedics, Graduate School of Dentistry, Department of Community Social Dentistry, Tohoku University, Sendai, Miyagi, Japan.
Mizoguchi I; Division of Orthodontics and Dentofacial Orthopedics, Graduate School of Dentistry, Department of Community Social Dentistry, Tohoku University, Sendai, Miyagi, Japan.
Noiri Y; Division of Cariology, Operative Dentistry and Endodontics, Department of Oral Health Science, Graduate School of Medical and Dental Sciences, Niigata University, Niigata, Japan.
Handa K; Division of Operative Dentistry, Department of Ecological Dentistry, Graduate School of Dentistry, Tohoku University, 4-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan.; Division of Oral Biochemistry, Department of Oral Science, Graduate School of Dentistry, Kanagawa Dental University, Yokosuka, Kanagawa, Japan.
Saito M; Division of Operative Dentistry, Department of Ecological Dentistry, Graduate School of Dentistry, Tohoku University, 4-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, 980-8575, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2021 Jan 28; Vol. 11 (1), pp. 2613. Date of Electronic Publication: 2021 Jan 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Macrophage Activation*
Chemokine CXCL9/*metabolism
Macrophages/*metabolism
Periapical Periodontitis/*immunology
Receptors, CXCR3/*metabolism
Animals ; Cell Line, Tumor ; Cell Migration Assays, Macrophage ; Disease Models, Animal ; Host-Pathogen Interactions/immunology ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; Periapical Periodontitis/metabolism ; Periapical Periodontitis/pathology ; Receptors, CXCR3/antagonists & inhibitors ; Tooth Root/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CXCL9: evidence and contradictions for its role in tumor progression.
Autorzy:
Ding Q; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
Lu P; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
Xia Y; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
Ding S; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
Fan Y; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
Li X; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
Han P; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
Liu J; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
Tian D; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
Liu M; Department of Gastroenterology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei Province, 430030, China.
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Źródło:
Cancer medicine [Cancer Med] 2016 Nov; Vol. 5 (11), pp. 3246-3259. Date of Electronic Publication: 2016 Oct 10.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Chemokine CXCL9/*metabolism
Neoplasms/*metabolism
Neoplasms/*pathology
Animals ; Biomarkers ; Cell Transformation, Neoplastic/genetics ; Cell Transformation, Neoplastic/metabolism ; Chemokine CXCL9/genetics ; Disease Progression ; Humans ; Models, Biological ; Neoplasms/genetics ; Oncogene Proteins/genetics ; Oncogene Proteins/metabolism ; Promoter Regions, Genetic ; Receptors, CXCR3/metabolism ; T-Lymphocytes/metabolism ; Tumor Suppressor Proteins/genetics ; Tumor Suppressor Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Monokine induced by interferon gamma (MIG/CXCL9) is an independent prognostic factor in newly diagnosed myeloma.
Autorzy:
Bolomsky A; a Wilhelminen Cancer Research Institute, Department of Medicine I, Center for Oncology and Hematology , Wilhelminen Hospital , Vienna , Austria ;
Schreder M; a Wilhelminen Cancer Research Institute, Department of Medicine I, Center for Oncology and Hematology , Wilhelminen Hospital , Vienna , Austria ;
Hübl W; b Department of Laboratory Medicine , Wilhelminen Hospital , Vienna , Austria.
Zojer N; a Wilhelminen Cancer Research Institute, Department of Medicine I, Center for Oncology and Hematology , Wilhelminen Hospital , Vienna , Austria ;
Hilbe W; a Wilhelminen Cancer Research Institute, Department of Medicine I, Center for Oncology and Hematology , Wilhelminen Hospital , Vienna , Austria ;
Ludwig H; a Wilhelminen Cancer Research Institute, Department of Medicine I, Center for Oncology and Hematology , Wilhelminen Hospital , Vienna , Austria ;
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Źródło:
Leukemia & lymphoma [Leuk Lymphoma] 2016 Nov; Vol. 57 (11), pp. 2516-25. Date of Electronic Publication: 2016 Mar 21.
Typ publikacji:
Journal Article
MeSH Terms:
Chemokine CXCL9/*metabolism
Multiple Myeloma/*metabolism
Multiple Myeloma/*mortality
Adult ; Age Factors ; Aged ; Aged, 80 and over ; Biomarkers ; Chemokine CXCL9/blood ; Chemokines/metabolism ; Combined Modality Therapy ; Female ; Gene Expression ; Humans ; Male ; Middle Aged ; Multiple Myeloma/diagnosis ; Multiple Myeloma/therapy ; Neoplasm Staging ; Prognosis ; Proportional Hazards Models ; Protein Binding ; Receptors, CXCR3/metabolism ; Survival Analysis ; Treatment Outcome
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin-6, MCP-1, IP-10, and MIG are sequentially expressed in cerebrospinal fluid after subarachnoid hemorrhage.
Autorzy:
Niwa A; Department of Neurological Surgery, Aichi Medical University, 1-1 Yazakokarimata, Nagakute, Aichi, 480-1195, Japan.
Osuka K; Department of Neurological Surgery, Aichi Medical University, 1-1 Yazakokarimata, Nagakute, Aichi, 480-1195, Japan. .
Nakura T; Department of Neurological Surgery, Aichi Medical University, 1-1 Yazakokarimata, Nagakute, Aichi, 480-1195, Japan.
Matsuo N; Department of Neurological Surgery, Aichi Medical University, 1-1 Yazakokarimata, Nagakute, Aichi, 480-1195, Japan.
Watabe T; Department of Neurological Surgery, Aichi Medical University, 1-1 Yazakokarimata, Nagakute, Aichi, 480-1195, Japan.
Takayasu M; Department of Neurological Surgery, Aichi Medical University, 1-1 Yazakokarimata, Nagakute, Aichi, 480-1195, Japan.
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2016 Aug 30; Vol. 13 (1), pp. 217. Date of Electronic Publication: 2016 Aug 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Chemokine CCL2/*cerebrospinal fluid
Chemokine CXCL10/*cerebrospinal fluid
Chemokine CXCL9/*cerebrospinal fluid
Interleukin-6/*cerebrospinal fluid
Subarachnoid Hemorrhage/*cerebrospinal fluid
Subarachnoid Hemorrhage/*diagnosis
Adult ; Aged ; Biomarkers/cerebrospinal fluid ; Chemokine CCL2/genetics ; Chemokine CXCL10/genetics ; Chemokine CXCL9/genetics ; Female ; Gene Expression ; Humans ; Interleukin-6/genetics ; Male ; Middle Aged ; Subarachnoid Hemorrhage/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Wyświetlanie 1-20 z 996

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