Informacja

Drogi użytkowniku, aplikacja do prawidłowego działania wymaga obsługi JavaScript. Proszę włącz obsługę JavaScript w Twojej przeglądarce.

Wyszukujesz frazę ""Chen S"" wg kryterium: Autor


Tytuł:
The Dual Roles of Activating Transcription Factor 3 (ATF3) in Inflammation, Apoptosis, Ferroptosis, and Pathogen Infection Responses.
Autorzy:
Liu S; State Key Laboratory for Animal Disease Control and Prevention, College of Veterinary Medicine, Lanzhou University, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730000, China.; Gansu Province Research Center for Basic Disciplines of Pathogen Biology, Lanzhou 730046, China.; Key Laboratory of Veterinary Etiological Biology, Key Laboratory of Ruminant Disease Prevention and Control (West), Ministry of Agricultural and Rural Affairs, Lanzhou 730046, China.
Li Z; State Key Laboratory for Animal Disease Control and Prevention, College of Veterinary Medicine, Lanzhou University, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730000, China.; Gansu Province Research Center for Basic Disciplines of Pathogen Biology, Lanzhou 730046, China.; Key Laboratory of Veterinary Etiological Biology, Key Laboratory of Ruminant Disease Prevention and Control (West), Ministry of Agricultural and Rural Affairs, Lanzhou 730046, China.
Lan S; State Key Laboratory for Animal Disease Control and Prevention, College of Veterinary Medicine, Lanzhou University, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730000, China.; Gansu Province Research Center for Basic Disciplines of Pathogen Biology, Lanzhou 730046, China.; Key Laboratory of Veterinary Etiological Biology, Key Laboratory of Ruminant Disease Prevention and Control (West), Ministry of Agricultural and Rural Affairs, Lanzhou 730046, China.
Hao H; State Key Laboratory for Animal Disease Control and Prevention, College of Veterinary Medicine, Lanzhou University, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730000, China.; Gansu Province Research Center for Basic Disciplines of Pathogen Biology, Lanzhou 730046, China.; Key Laboratory of Veterinary Etiological Biology, Key Laboratory of Ruminant Disease Prevention and Control (West), Ministry of Agricultural and Rural Affairs, Lanzhou 730046, China.
Baz AA; State Key Laboratory for Animal Disease Control and Prevention, College of Veterinary Medicine, Lanzhou University, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730000, China.; Gansu Province Research Center for Basic Disciplines of Pathogen Biology, Lanzhou 730046, China.; Key Laboratory of Veterinary Etiological Biology, Key Laboratory of Ruminant Disease Prevention and Control (West), Ministry of Agricultural and Rural Affairs, Lanzhou 730046, China.
Yan X; State Key Laboratory for Animal Disease Control and Prevention, College of Veterinary Medicine, Lanzhou University, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730000, China.; Gansu Province Research Center for Basic Disciplines of Pathogen Biology, Lanzhou 730046, China.; Key Laboratory of Veterinary Etiological Biology, Key Laboratory of Ruminant Disease Prevention and Control (West), Ministry of Agricultural and Rural Affairs, Lanzhou 730046, China.
Gao P; State Key Laboratory for Animal Disease Control and Prevention, College of Veterinary Medicine, Lanzhou University, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730000, China.; Gansu Province Research Center for Basic Disciplines of Pathogen Biology, Lanzhou 730046, China.; Key Laboratory of Veterinary Etiological Biology, Key Laboratory of Ruminant Disease Prevention and Control (West), Ministry of Agricultural and Rural Affairs, Lanzhou 730046, China.
Chen S; State Key Laboratory for Animal Disease Control and Prevention, College of Veterinary Medicine, Lanzhou University, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730000, China.; Gansu Province Research Center for Basic Disciplines of Pathogen Biology, Lanzhou 730046, China.; Key Laboratory of Veterinary Etiological Biology, Key Laboratory of Ruminant Disease Prevention and Control (West), Ministry of Agricultural and Rural Affairs, Lanzhou 730046, China.
Chu Y; State Key Laboratory for Animal Disease Control and Prevention, College of Veterinary Medicine, Lanzhou University, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730000, China.; Gansu Province Research Center for Basic Disciplines of Pathogen Biology, Lanzhou 730046, China.; Key Laboratory of Veterinary Etiological Biology, Key Laboratory of Ruminant Disease Prevention and Control (West), Ministry of Agricultural and Rural Affairs, Lanzhou 730046, China.
Pokaż więcej
Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Jan 09; Vol. 25 (2). Date of Electronic Publication: 2024 Jan 09.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Activating Transcription Factor 3*/genetics
Apoptosis*
Cyclic AMP Response Element-Binding Protein*
Ferroptosis*
Humans ; Inflammation
Czasopismo naukowe
Tytuł:
PANoptosis: Mechanism and Role in Pulmonary Diseases.
Autorzy:
Chen S; Shanghai Frontiers Science Center of Drug Target Identification and Delivery, School of Pharmacy, Shanghai Jiao Tong University, Shanghai 200240, China.
Jiang J; Shanghai Frontiers Science Center of Drug Target Identification and Delivery, School of Pharmacy, Shanghai Jiao Tong University, Shanghai 200240, China.
Li T; Shanghai Frontiers Science Center of Drug Target Identification and Delivery, School of Pharmacy, Shanghai Jiao Tong University, Shanghai 200240, China.
Huang L; Shanghai Frontiers Science Center of Drug Target Identification and Delivery, School of Pharmacy, Shanghai Jiao Tong University, Shanghai 200240, China.
Pokaż więcej
Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Oct 19; Vol. 24 (20). Date of Electronic Publication: 2023 Oct 19.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Apoptosis*
Acute Lung Injury*
Humans ; Cell Death ; Homeostasis ; Inflammation ; Pyroptosis
Czasopismo naukowe
Tytuł:
Machine learning-based identification of cuproptosis-related markers and immune infiltration in severe community-acquired pneumonia.
Autorzy:
Chen S; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, Shanghai, China.
Zhou Z; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, Shanghai, China.
Wang Y; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, Shanghai, China.
Chen S; Department of Pulmonary and Critical Care Medicine, Zhongshan Hospital, Fudan University, Shanghai, China.
Jiang J; Shanghai Respiratory Research Institute, Zhongshan Hospital, Fudan University, Shanghai, China.
Pokaż więcej
Źródło:
The clinical respiratory journal [Clin Respir J] 2023 Jul; Vol. 17 (7), pp. 618-628. Date of Electronic Publication: 2023 Jun 06.
Typ publikacji:
Journal Article
MeSH Terms:
Community-Acquired Infections*
Pneumonia*
Respiratory Distress Syndrome*
Apoptosis*
Humans ; Algorithms ; Machine Learning ; Copper
Czasopismo naukowe
Tytuł:
In Silico Identification and Validation of Cuproptosis-Related LncRNA Signature as a Novel Prognostic Model and Immune Function Analysis in Colon Adenocarcinoma.
Autorzy:
Wang Y; State Key Laboratory of Southwestern Chinese Medicine Resources, School of Pharmacy, Chengdu University of Traditional Chinese Medicine, Chengdu 611137, China.
Huang X; State Key Laboratory of Southwestern Chinese Medicine Resources, School of Pharmacy, Chengdu University of Traditional Chinese Medicine, Chengdu 611137, China.
Chen S; State Key Laboratory of Southwestern Chinese Medicine Resources, School of Pharmacy, Chengdu University of Traditional Chinese Medicine, Chengdu 611137, China.
Jiang H; State Key Laboratory of Southwestern Chinese Medicine Resources, School of Pharmacy, Chengdu University of Traditional Chinese Medicine, Chengdu 611137, China.
Rao H; State Key Laboratory of Southwestern Chinese Medicine Resources, School of Pharmacy, Chengdu University of Traditional Chinese Medicine, Chengdu 611137, China.
Lu L; State Key Laboratory of Southwestern Chinese Medicine Resources, School of Pharmacy, Chengdu University of Traditional Chinese Medicine, Chengdu 611137, China.
Wen F; State Key Laboratory of Southwestern Chinese Medicine Resources, School of Pharmacy, Chengdu University of Traditional Chinese Medicine, Chengdu 611137, China.
Pei J; State Key Laboratory of Southwestern Chinese Medicine Resources, School of Pharmacy, Chengdu University of Traditional Chinese Medicine, Chengdu 611137, China.
Pokaż więcej
Źródło:
Current oncology (Toronto, Ont.) [Curr Oncol] 2022 Sep 15; Vol. 29 (9), pp. 6573-6593. Date of Electronic Publication: 2022 Sep 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adenocarcinoma*/pathology
Colonic Neoplasms*/genetics
Colonic Neoplasms*/pathology
MicroRNAs*/genetics
MicroRNAs*/metabolism
RNA, Long Noncoding*/genetics
RNA, Long Noncoding*/metabolism
Apoptosis*
Humans ; Gene Expression Regulation, Neoplastic ; Gene Regulatory Networks ; Immunity ; Prognosis ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Copper
Czasopismo naukowe
Tytuł:
Porcine TRIM21 Enhances Porcine Circovirus 2 Infection and Host Immune Responses, But Inhibits Apoptosis of PCV2-Infected Cells.
Autorzy:
Yang L; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Liu X; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Zhang L; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Li X; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Zhang X; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Niu G; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Ji W; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Chen S; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Ouyang H; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Ren L; College of Animal Sciences, Key Lab for Zoonoses Research, Ministry of Education, Jilin University, 5333 Xi'an Road, Changchun 130062, China.
Pokaż więcej
Źródło:
Viruses [Viruses] 2022 Jan 15; Vol. 14 (1). Date of Electronic Publication: 2022 Jan 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Host Microbial Interactions*/genetics
Host Microbial Interactions*/immunology
Immunity*
Apoptosis/*genetics
Circovirus/*immunology
Swine Diseases/*immunology
Tripartite Motif Proteins/*genetics
Animals ; Apoptosis/immunology ; Cell Line ; HEK293 Cells ; Humans ; Interferons/immunology ; Swine ; Swine Diseases/virology ; Tripartite Motif Proteins/classification ; Virus Replication
Czasopismo naukowe
Tytuł:
Enhanced H3K4 Trimethylation in TNF- α Promoter Gene Locus with Cell Apoptosis in the Ventral-Medial Striatum following Opioid Withdrawal of Neonatal Rat Offspring from Morphine-Addicted Mothers.
Autorzy:
Wu PL; School of Medicine, College of Medicine, I-Shou University, Kaohsiung, Taiwan.; Department of Pediatrics, E-Da Hospital, Kaohsiung, Taiwan.; Graduate Institute of Medicine, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan.
Suen JL; Graduate Institute of Medicine, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan.
Yang CH; Department of Pediatrics, E-Da Hospital, Kaohsiung, Taiwan.
Kuo KC; Department of Pediatrics, Kaohsiung Chang Gung Memorial Hospital and Chang Gung University College of Medicine, Kaohsiung, Taiwan.
Yang YSH; Joint Biobank, Office of Human Research, Taipei Medical University, Taipei, Taiwan.
Yang SN; School of Medicine, College of Medicine, I-Shou University, Kaohsiung, Taiwan.; Department of Pediatrics, E-Da Hospital, Kaohsiung, Taiwan.
Pokaż więcej
Źródło:
Mediators of inflammation [Mediators Inflamm] 2021 Jun 16; Vol. 2021, pp. 9828995. Date of Electronic Publication: 2021 Jun 16 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Morphine*
Pregnancy, Animal*
Promoter Regions, Genetic*
Histones/*metabolism
Morphine Dependence/*metabolism
Tumor Necrosis Factor-alpha/*genetics
Analgesics, Opioid ; Animals ; Animals, Newborn ; Brain/pathology ; Caspase 3/metabolism ; Corpus Striatum ; Epigenesis, Genetic ; Female ; Maternal Exposure ; Methylation ; Pregnancy ; Rats ; Rats, Sprague-Dawley ; Substance Withdrawal Syndrome/pathology
Czasopismo naukowe
Tytuł:
Agomelatine Softens Depressive-Like Behavior through the Regulation of Autophagy and Apoptosis.
Autorzy:
Chen F; The Second Clinical Medical College of Zhejiang Chinese Medical University, Hangzhou 310053, China.
Chen S; Institute of Neuroscience, Zhejiang University School of Medicine, Hangzhou 310058, China.
Liu J; The Second Clinical Medical College of Zhejiang Chinese Medical University, Hangzhou 310053, China.
Amin N; Institute of Neuroscience, Zhejiang University School of Medicine, Hangzhou 310058, China.; Department of Zoology, Faculty of Science, Aswan University, Egypt.
Jin W; The Second Clinical Medical College of Zhejiang Chinese Medical University, Hangzhou 310053, China.; Zhejiang Mental Health Center, Tongde Hospital of Zhejiang province, Hangzhou 310012, China.
Fang M; Institute of Neuroscience, Zhejiang University School of Medicine, Hangzhou 310058, China.
Pokaż więcej
Źródło:
BioMed research international [Biomed Res Int] 2021 Mar 17; Vol. 2021, pp. 6664591. Date of Electronic Publication: 2021 Mar 17 (Print Publication: 2021).
Typ publikacji:
Journal Article; Retracted Publication
MeSH Terms:
Depression*/drug therapy
Depression*/metabolism
Depression*/pathology
Stress, Psychological*/drug therapy
Stress, Psychological*/metabolism
Stress, Psychological*/pathology
Acetamides/*pharmacology
Apoptosis/*drug effects
Autophagy/*drug effects
Behavior, Animal/*drug effects
MAP Kinase Signaling System/*drug effects
Animals ; Brain-Derived Neurotrophic Factor/metabolism ; Disease Models, Animal ; Male ; Membrane Glycoproteins/metabolism ; Mice ; Protein-Tyrosine Kinases/metabolism ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł:
Oxidative stress-mediated mitochondrial pathway-dependent apoptosis is induced by silica nanoparticles in H9c2 cardiomyocytes.
Autorzy:
Cui G; Department of Respiratory Medicine, Qilu Children's Hospital of Shandong University, Ji'nan, People's Republic of China.
Zhang H; Department of Emergency, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Ji'nan, People's Republic of China.
Guo Q; Department of Toxicology, Shandong Academy of Occupational Health and Occupational Medicine, Shandong First Medical University & Shandong Academy of Medical Sciences, Ji'nan, People's Republic of China.
Shan S; School of Public Health Shandong University, Ji'nan, People's Republic of China.
Chen S; Department of Toxicology, Shandong Academy of Occupational Health and Occupational Medicine, Shandong First Medical University & Shandong Academy of Medical Sciences, Ji'nan, People's Republic of China.
Li C; Department of Toxicology, Shandong Academy of Occupational Health and Occupational Medicine, Shandong First Medical University & Shandong Academy of Medical Sciences, Ji'nan, People's Republic of China.
Yang X; Department of Toxicology, Shandong Academy of Occupational Health and Occupational Medicine, Shandong First Medical University & Shandong Academy of Medical Sciences, Ji'nan, People's Republic of China.
Li Z; Department of Toxicology, Shandong Academy of Occupational Health and Occupational Medicine, Shandong First Medical University & Shandong Academy of Medical Sciences, Ji'nan, People's Republic of China.
Mu Y; Department of Toxicology, Shandong Academy of Occupational Health and Occupational Medicine, Shandong First Medical University & Shandong Academy of Medical Sciences, Ji'nan, People's Republic of China.
Shao H; Department of Toxicology, Shandong Academy of Occupational Health and Occupational Medicine, Shandong First Medical University & Shandong Academy of Medical Sciences, Ji'nan, People's Republic of China.
Du Z; Department of Toxicology, Shandong Academy of Occupational Health and Occupational Medicine, Shandong First Medical University & Shandong Academy of Medical Sciences, Ji'nan, People's Republic of China.
Pokaż więcej
Źródło:
Toxicology mechanisms and methods [Toxicol Mech Methods] 2020 Nov; Vol. 30 (9), pp. 646-655. Date of Electronic Publication: 2020 Aug 20.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Heart Diseases/*chemically induced
Mitochondria, Heart/*drug effects
Myocytes, Cardiac/*drug effects
Nanoparticles/*toxicity
Oxidative Stress/*drug effects
Silicon Dioxide/*toxicity
Animals ; Cardiotoxicity ; Cell Line ; Dose-Response Relationship, Drug ; Heart Diseases/metabolism ; Heart Diseases/pathology ; Mitochondria, Heart/metabolism ; Mitochondria, Heart/pathology ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/pathology ; Rats ; Reactive Oxygen Species/metabolism ; Risk Assessment ; Signal Transduction ; Time Factors
Czasopismo naukowe
Tytuł:
JAK2/STAT3 involves oxidative stress-induced cell injury in N2a cells and a rat MCAO model.
Autorzy:
Sun Y; Department of Cell Biology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Cheng M; Department of Nutrition and Food Science, School of Public Health, Tianjin Medical University, Tianjin, China.
Liang X; Department of Nutrition and Food Science, School of Public Health, Tianjin Medical University, Tianjin, China.
Chen S; Department of Nutrition and Food Science, School of Public Health, Tianjin Medical University, Tianjin, China.
Wang M; Department of Nutrition and Food Science, School of Public Health, Tianjin Medical University, Tianjin, China.
Zhang X; Department of Nutrition and Food Science, School of Public Health, Tianjin Medical University, Tianjin, China.
Pokaż więcej
Źródło:
The International journal of neuroscience [Int J Neurosci] 2020 Nov; Vol. 130 (11), pp. 1142-1150. Date of Electronic Publication: 2020 Feb 24.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*/drug effects
Neuroblastoma*
Oxidative Stress*/drug effects
Signal Transduction*/drug effects
Enzyme Inhibitors/*pharmacology
Hydrogen Peroxide/*pharmacology
Infarction, Middle Cerebral Artery/*metabolism
Janus Kinase 2/*metabolism
Oxidants/*pharmacology
Reperfusion Injury/*metabolism
STAT3 Transcription Factor/*metabolism
8-Hydroxy-2'-Deoxyguanosine/metabolism ; Animals ; Cell Line, Tumor ; Cell Survival/drug effects ; Disease Models, Animal ; Janus Kinase 2/antagonists & inhibitors ; Janus Kinase 2/drug effects ; Mice ; Rats ; STAT3 Transcription Factor/drug effects ; Tyrphostins/pharmacology
Czasopismo naukowe
Tytuł:
Necrostatin-1 Synergizes the Pan Caspase Inhibitor to Attenuate Lung Injury Induced by Ischemia Reperfusion in Rats.
Autorzy:
Wang L; Department of Anesthesiology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou 325000, China.
Chen B; Department of Anesthesiology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou 325000, China.
Xiong X; Department of Anesthesiology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou 325000, China.
Chen S; Department of Anesthesiology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou 325000, China.
Jin L; Department of Anesthesiology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou 325000, China.
Zhu M; Department of Anesthesiology, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou 325000, China.
Pokaż więcej
Źródło:
Mediators of inflammation [Mediators Inflamm] 2020 Oct 24; Vol. 2020, pp. 7059304. Date of Electronic Publication: 2020 Oct 24 (Print Publication: 2020).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Caspase Inhibitors/*pharmacology
Imidazoles/*metabolism
Indoles/*metabolism
Lung Injury/*metabolism
Reperfusion Injury/*metabolism
Amino Acid Chloromethyl Ketones ; Animals ; Bronchoalveolar Lavage Fluid ; Caspase 8/metabolism ; Cell Death ; Flow Cytometry ; HMGB1 Protein/metabolism ; Inflammation ; Male ; Necrosis ; Protein Serine-Threonine Kinases/metabolism ; Pulmonary Edema ; Rats ; Rats, Sprague-Dawley ; Receptor-Interacting Protein Serine-Threonine Kinases/metabolism
Czasopismo naukowe
Tytuł:
Cytotoxic effects of vitamins K1, K2, and K3 against human T lymphoblastoid leukemia cells through apoptosis induction and cell cycle arrest.
Autorzy:
Xu W; Department of Pharmacy, Hubei Provincial Hospital of Traditional Chinese Medicine, Wuhan, China.; Institute of Traditional Chinese Medicine, Hubei Province Academy of Traditional Chinese Medicine, Wuhan, China.
Wu H; Department of Clinical Pharmacology, School of Pharmacy, Tokyo University of Pharmacy and Life Sciences, Tokyo, Japan.
Chen S; Department of Pharmacy, Hubei Provincial Hospital of Traditional Chinese Medicine, Wuhan, China.; Institute of Traditional Chinese Medicine, Hubei Province Academy of Traditional Chinese Medicine, Wuhan, China.
Wang X; Institute of Traditional Chinese Medicine, Hubei Province Academy of Traditional Chinese Medicine, Wuhan, China.; Department of Nephrology, Hubei Provincial Hospital of Traditional Chinese Medicine, Wuhan, China.
Tanaka S; Department of Clinical Pharmacology, School of Pharmacy, Tokyo University of Pharmacy and Life Sciences, Tokyo, Japan.
Sugiyama K; Department of Clinical Pharmacology, School of Pharmacy, Tokyo University of Pharmacy and Life Sciences, Tokyo, Japan.
Yamada H; Department of Clinical Pharmacology, School of Pharmacy, Tokyo University of Pharmacy and Life Sciences, Tokyo, Japan.
Hirano T; Department of Clinical Pharmacology, School of Pharmacy, Tokyo University of Pharmacy and Life Sciences, Tokyo, Japan.
Pokaż więcej
Źródło:
Chemical biology & drug design [Chem Biol Drug Des] 2020 Oct; Vol. 96 (4), pp. 1134-1147. Date of Electronic Publication: 2020 Aug 06.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Cell Cycle/*drug effects
Leukemia, T-Cell/*pathology
Vitamin K 1/*pharmacology
Vitamin K 2/*pharmacology
Vitamin K 3/*pharmacology
Cell Line, Tumor ; Humans
Czasopismo naukowe
Tytuł:
Gypenoside Inhibits Endothelial Cell Apoptosis in Atherosclerosis by Modulating Mitochondria through PI3K/Akt/Bad Pathway.
Autorzy:
Song N; Key Laboratory of Ministry of Education for TCM Viscera-State Theory and Applications, Liaoning University of Traditional Chinese Medicine, Shenyang 110847, China.; National Local Joint Engineering Laboratory for the Prevention and Treatment of Cardioencephalopathy with Integrated Traditional Chinese and Western Medicine, Shenyang 110847, China.
Jia L; Key Laboratory of Ministry of Education for TCM Viscera-State Theory and Applications, Liaoning University of Traditional Chinese Medicine, Shenyang 110847, China.; National Local Joint Engineering Laboratory for the Prevention and Treatment of Cardioencephalopathy with Integrated Traditional Chinese and Western Medicine, Shenyang 110847, China.
Cao H; Key Laboratory of Ministry of Education for TCM Viscera-State Theory and Applications, Liaoning University of Traditional Chinese Medicine, Shenyang 110847, China.; National Local Joint Engineering Laboratory for the Prevention and Treatment of Cardioencephalopathy with Integrated Traditional Chinese and Western Medicine, Shenyang 110847, China.
Ma Y; Key Laboratory of Ministry of Education for TCM Viscera-State Theory and Applications, Liaoning University of Traditional Chinese Medicine, Shenyang 110847, China.; National Local Joint Engineering Laboratory for the Prevention and Treatment of Cardioencephalopathy with Integrated Traditional Chinese and Western Medicine, Shenyang 110847, China.
Chen N; Key Laboratory of Ministry of Education for TCM Viscera-State Theory and Applications, Liaoning University of Traditional Chinese Medicine, Shenyang 110847, China.; National Local Joint Engineering Laboratory for the Prevention and Treatment of Cardioencephalopathy with Integrated Traditional Chinese and Western Medicine, Shenyang 110847, China.
Chen S; Key Laboratory of Ministry of Education for TCM Viscera-State Theory and Applications, Liaoning University of Traditional Chinese Medicine, Shenyang 110847, China.; National Local Joint Engineering Laboratory for the Prevention and Treatment of Cardioencephalopathy with Integrated Traditional Chinese and Western Medicine, Shenyang 110847, China.
Lv X; Key Laboratory of Ministry of Education for TCM Viscera-State Theory and Applications, Liaoning University of Traditional Chinese Medicine, Shenyang 110847, China.; National Local Joint Engineering Laboratory for the Prevention and Treatment of Cardioencephalopathy with Integrated Traditional Chinese and Western Medicine, Shenyang 110847, China.
Yang G; Key Laboratory of Ministry of Education for TCM Viscera-State Theory and Applications, Liaoning University of Traditional Chinese Medicine, Shenyang 110847, China.; National Local Joint Engineering Laboratory for the Prevention and Treatment of Cardioencephalopathy with Integrated Traditional Chinese and Western Medicine, Shenyang 110847, China.
Pokaż więcej
Źródło:
BioMed research international [Biomed Res Int] 2020 Jun 20; Vol. 2020, pp. 2819658. Date of Electronic Publication: 2020 Jun 20 (Print Publication: 2020).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Atherosclerosis/*pathology
Endothelial Cells/*pathology
Mitochondria/*metabolism
Phosphatidylinositol 3-Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
bcl-Associated Death Protein/*metabolism
Adenosine Triphosphate/metabolism ; Animals ; Aorta/drug effects ; Aorta/metabolism ; Aorta/pathology ; Atherosclerosis/blood ; Caspase 3/metabolism ; Electron Transport/drug effects ; Endothelial Cells/drug effects ; Energy Metabolism/genetics ; Gene Expression Regulation/drug effects ; Ginsenosides/pharmacology ; Gynostemma ; Lipids/blood ; Lipoproteins, LDL ; Male ; Mice, Inbred C57BL ; Mitochondria/drug effects ; Mitochondrial Dynamics/drug effects ; Mitochondrial Proteins/metabolism ; Plant Extracts/pharmacology ; Plaque, Atherosclerotic/pathology ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł:
Low shear stress induces endothelial cell apoptosis and monocyte adhesion by upregulating PECAM‑1 expression.
Autorzy:
Xie X; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Wang F; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Zhu L; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Yang H; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Pan D; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Liu Y; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Qu X; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Gu Y; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Li X; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Chen S; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, Jiangsu 210006, P.R. China.
Pokaż więcej
Źródło:
Molecular medicine reports [Mol Med Rep] 2020 Jun; Vol. 21 (6), pp. 2580-2588. Date of Electronic Publication: 2020 Apr 08.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Shear Strength*
Cell Adhesion/*physiology
Platelet Endothelial Cell Adhesion Molecule-1/*metabolism
Cell Line ; Coculture Techniques ; Endothelial Cells/cytology ; Endothelial Cells/metabolism ; Forkhead Box Protein O1/metabolism ; Humans ; Monocytes/cytology ; Monocytes/metabolism ; Phosphorylation ; Platelet Endothelial Cell Adhesion Molecule-1/antagonists & inhibitors ; Platelet Endothelial Cell Adhesion Molecule-1/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; RNA Interference ; RNA, Small Interfering/metabolism
Czasopismo naukowe
Tytuł:
Downregulated long non-coding RNA LINC01093 in liver fibrosis promotes hepatocyte apoptosis via increasing ubiquitination of SIRT1.
Autorzy:
Tang Y; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital, Wenzhou Medical University, 205 Wenrui Avenue, Wenzhou 325000, People's Republic of China.
Ma N; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital, Wenzhou Medical University, 205 Wenrui Avenue, Wenzhou 325000, People's Republic of China.
Luo H; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital, Wenzhou Medical University, 205 Wenrui Avenue, Wenzhou 325000, People's Republic of China.
Chen S; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital, Wenzhou Medical University, 205 Wenrui Avenue, Wenzhou 325000, People's Republic of China.
Yu F; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital, Wenzhou Medical University, 205 Wenrui Avenue, Wenzhou 325000, People's Republic of China.
Pokaż więcej
Źródło:
Journal of biochemistry [J Biochem] 2020 May 01; Vol. 167 (5), pp. 525-534.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Down-Regulation*
Ubiquitination*
Hepatocytes/*metabolism
Liver Cirrhosis/*metabolism
RNA, Long Noncoding/*metabolism
Sirtuin 1/*metabolism
Animals ; Carbon Tetrachloride/administration & dosage ; Cells, Cultured ; Disease Models, Animal ; Injections, Intraperitoneal ; Liver Cirrhosis/chemically induced ; Liver Cirrhosis/pathology ; Male ; Mice ; Mice, Inbred BALB C ; RNA, Long Noncoding/genetics
Czasopismo naukowe
Tytuł:
SPAG5-AS1 inhibited autophagy and aggravated apoptosis of podocytes via SPAG5/AKT/mTOR pathway.
Autorzy:
Xu J; Department of Geriatrics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Deng Y; Department of Endocrinology, The Affiliated Hospital of Qingdao University, Qingdao, China.
Wang Y; Department of Nephrology, Yueyang Hospital of Integrated Traditional Chinese and Western Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Sun X; Department of Endocrinology, The Affiliated Hospital of Qingdao University, Qingdao, China.
Chen S; Department of Endocrinology and Metabolism, Ningbo First Hospital, Ningbo, China.
Fu G; Department of Geriatrics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Pokaż więcej
Źródło:
Cell proliferation [Cell Prolif] 2020 Feb; Vol. 53 (2), pp. e12738. Date of Electronic Publication: 2020 Jan 19.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*physiology
Autophagy/*physiology
Cell Cycle Proteins/*metabolism
Podocytes/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Signal Transduction/*physiology
TOR Serine-Threonine Kinases/*metabolism
Cells, Cultured ; Diabetic Nephropathies/metabolism ; Diabetic Nephropathies/pathology ; Glucose/metabolism ; Humans ; Podocytes/physiology ; RNA, Long Noncoding/metabolism ; Transcription, Genetic/physiology ; Ubiquitin Thiolesterase/metabolism ; Ubiquitination/physiology ; Up-Regulation/physiology
Czasopismo naukowe
Tytuł:
Autophagy Promotes Porcine Parvovirus Replication and Induces Non-Apoptotic Cell Death in Porcine Placental Trophoblasts.
Autorzy:
Zhang X
Xiong Y
Zhang J; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Shao T; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Chen S; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Miao B; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Wang Z; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Du Q; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Huang Y; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Tong D; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Pokaż więcej
Źródło:
Viruses [Viruses] 2019 Dec 20; Vol. 12 (1). Date of Electronic Publication: 2019 Dec 20.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Autophagy*
Virus Replication*
Parvoviridae Infections/*veterinary
Parvovirus, Porcine/*physiology
Swine Diseases/*virology
Trophoblasts/*virology
Animals ; Autophagosomes/metabolism ; Biomarkers ; Cell Line ; Female ; Lysosomes/metabolism ; Parvovirus, Porcine/ultrastructure ; Placenta/metabolism ; Placenta/virology ; Pregnancy ; Swine ; Swine Diseases/metabolism ; Trophoblasts/metabolism
Czasopismo naukowe
Tytuł:
Acupuncture Reduces Apoptosis of Granulosa Cells in Rats with Premature Ovarian Failure Via Restoring the PI3K/Akt Signaling Pathway.
Autorzy:
Wang S; Department of Acupuncture, School of Traditional Chinese Medicine, Jinan University, Guangzhou 510632, China.
Lin S; Department of Acupuncture, School of Traditional Chinese Medicine, Jinan University, Guangzhou 510632, China.
Zhu M; Department of Acupuncture, School of Traditional Chinese Medicine, Jinan University, Guangzhou 510632, China.
Li C; Department of Acupuncture, School of Traditional Chinese Medicine, Jinan University, Guangzhou 510632, China.
Chen S; Department of Acupuncture, School of Traditional Chinese Medicine, Jinan University, Guangzhou 510632, China.
Pu L; Department of Acupuncture, School of Traditional Chinese Medicine, Jinan University, Guangzhou 510632, China.
Lin J; Department of Acupuncture, School of Traditional Chinese Medicine, Jinan University, Guangzhou 510632, China.
Cao L; Formula-pattern Research Center, School of Traditional Chinese Medicine, Jinan University, Guangzhou 510632, China.
Zhang Y; Department of Acupuncture, School of Traditional Chinese Medicine, Jinan University, Guangzhou 510632, China.
Pokaż więcej
Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Dec 13; Vol. 20 (24). Date of Electronic Publication: 2019 Dec 13.
Typ publikacji:
Journal Article
MeSH Terms:
Acupuncture Therapy*
Apoptosis*
Primary Ovarian Insufficiency*/metabolism
Primary Ovarian Insufficiency*/pathology
Primary Ovarian Insufficiency*/therapy
Signal Transduction*
Granulosa Cells/*metabolism
Phosphatidylinositol 3-Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Animals ; Female ; Granulosa Cells/pathology ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł:
A mitochondrial-targeted peptide ameliorated podocyte apoptosis through a HOCl-alb-enhanced and mitochondria-dependent signalling pathway in diabetic rats and in vitro.
Autorzy:
Wang X; a Division of Nephrology, Zhujiang Hospital , Southern Medical University , Guangzhou , China.
Tang D; b Division of Nephrology, Huadu District people's Hospital , Southern Medical University , Guangzhou , China.
Zou Y; a Division of Nephrology, Zhujiang Hospital , Southern Medical University , Guangzhou , China.
Wu X; a Division of Nephrology, Zhujiang Hospital , Southern Medical University , Guangzhou , China.
Chen Y; a Division of Nephrology, Zhujiang Hospital , Southern Medical University , Guangzhou , China.
Li H; c Special Medical Service Center, Zhujiang Hospital , Southern Medical University , Guangzhou , China.
Chen S; a Division of Nephrology, Zhujiang Hospital , Southern Medical University , Guangzhou , China.
Shi Y; c Special Medical Service Center, Zhujiang Hospital , Southern Medical University , Guangzhou , China.
Niu H; c Special Medical Service Center, Zhujiang Hospital , Southern Medical University , Guangzhou , China.
Pokaż więcej
Źródło:
Journal of enzyme inhibition and medicinal chemistry [J Enzyme Inhib Med Chem] 2019 Dec; Vol. 34 (1), pp. 394-404.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Diabetes Mellitus, Experimental/*drug therapy
Hypochlorous Acid/*pharmacology
Mitochondria/*drug effects
Podocytes/*drug effects
Serum Albumin/*metabolism
Animals ; Diabetes Mellitus, Experimental/metabolism ; Dose-Response Relationship, Drug ; Male ; Molecular Structure ; Rats ; Rats, Sprague-Dawley ; Signal Transduction/drug effects ; Structure-Activity Relationship
Czasopismo naukowe
Tytuł:
P38 Signal Transduction Pathway Has More Cofactors on Apoptosis of SGC-7901 Gastric Cancer Cells Induced by Combination of Rutin and Oxaliplatin.
Autorzy:
Li Q; Department of Pathology, The Third Affiliated Hospital of Jinzhou Medical University, Jinzhou, Liaoning, China.
Ren L; Department of Experimental Pharmacology and Toxicology, School of Pharmaceutical Sciences, Jilin University, Changchun, Jilin, China.
Zhang Y; Department of Experimental Pharmacology and Toxicology, School of Pharmaceutical Sciences, Jilin University, Changchun, Jilin, China.
Gu Z; Department of Pathology and Pathophysiology, Jinzhou Medical University, Jinzhou, Liaoning, China.
Tan Q; Department of Pathology and Pathophysiology, Jinzhou Medical University, Jinzhou, Liaoning, China.
Zhang T; Department of Experimental Pharmacology and Toxicology, School of Pharmaceutical Sciences, Jilin University, Changchun, Jilin, China.
Qin M; Department of Experimental Pharmacology and Toxicology, School of Pharmaceutical Sciences, Jilin University, Changchun, Jilin, China.
Chen S; Department of Pathology, The Third Affiliated Hospital of Jinzhou Medical University, Jinzhou, Liaoning, China.
Pokaż więcej
Źródło:
BioMed research international [Biomed Res Int] 2019 Nov 06; Vol. 2019, pp. 6407210. Date of Electronic Publication: 2019 Nov 06 (Print Publication: 2019).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Oxaliplatin/*pharmacology
Rutin/*pharmacology
Signal Transduction/*drug effects
Stomach Neoplasms/*drug therapy
Antineoplastic Agents/pharmacology ; Caspases/metabolism ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Drug Therapy, Combination ; G1 Phase/drug effects ; Humans ; MAP Kinase Signaling System/drug effects ; Resting Phase, Cell Cycle/drug effects ; Stomach Neoplasms/metabolism
Czasopismo naukowe
Tytuł:
Jujube honey induces apoptosis in human hepatocellular carcinoma HepG2 cell via DNA damage, p53 expression, and caspase activation.
Autorzy:
Cheng N; College of Food Science and Technology, Northwest University, Xi'an, China.; Bee Product Research Center of Shaanxi Province, Xi'an, China.
Zhao H; School of Chemical Engineering, Northwest University, Xi'an, China.; Bee Product Research Center of Shaanxi Province, Xi'an, China.
Chen S; College of Food Science and Technology, Northwest University, Xi'an, China.
He Q; College of Food Science and Technology, Northwest University, Xi'an, China.
Cao W; College of Food Science and Technology, Northwest University, Xi'an, China.; Bee Product Research Center of Shaanxi Province, Xi'an, China.
Pokaż więcej
Źródło:
Journal of food biochemistry [J Food Biochem] 2019 Nov; Vol. 43 (11), pp. e12998. Date of Electronic Publication: 2019 Aug 01.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Carcinoma, Hepatocellular/*drug therapy
Honey/*analysis
Liver Neoplasms/*drug therapy
Ziziphus/*chemistry
Cell Cycle/drug effects ; DNA Damage/drug effects ; Flavonoids/analysis ; Hep G2 Cells ; Humans ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/drug effects ; Phenols/analysis ; Up-Regulation
Czasopismo naukowe

Ta witryna wykorzystuje pliki cookies do przechowywania informacji na Twoim komputerze. Pliki cookies stosujemy w celu świadczenia usług na najwyższym poziomie, w tym w sposób dostosowany do indywidualnych potrzeb. Korzystanie z witryny bez zmiany ustawień dotyczących cookies oznacza, że będą one zamieszczane w Twoim komputerze. W każdym momencie możesz dokonać zmiany ustawień dotyczących cookies