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    Wyświetlanie 1-6 z 6
    Tytuł:
    Protective Effects of PEP-1-GSTA2 Protein in Hippocampal Neuronal Cell Damage Induced by Oxidative Stress.
    Autorzy:
    Choi YJ; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Shin MJ; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Youn GS; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Park JH; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Yeo HJ; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Yeo EJ; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Kwon HJ; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Lee LR; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Kim NY; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Kwon SY; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Jung HY; Department of Veterinary Medicine, Institute of Veterinary Science, Chungnam National University, Daejeon 34134, Republic of Korea.
    Cho YJ; Department of Neurosurgery, Hallym University Medical Center, Chuncheon 24253, Republic of Korea.
    Kim DW; Department of Biochemistry and Molecular Biology, Research Institute of Oral Sciences, College of Dentistry, Gangneung-Wonju National University, Gangneung 25457, Republic of Korea.
    Park J; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Han KH; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Lee KW; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Park JK; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Lee CH; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Eum WS; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
    Choi SY; Department of Biomedical Science, Research Institute of Bioscience & Biotechnology, Hallym University, Chuncheon 24252, Republic of Korea.
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    Źródło:
    International journal of molecular sciences [Int J Mol Sci] 2023 Feb 01; Vol. 24 (3). Date of Electronic Publication: 2023 Feb 01.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Hippocampus*/metabolism
    Oxidative Stress*
    Animals ; Apoptosis ; Ischemia/metabolism ; Neurons/metabolism ; Reactive Oxygen Species/metabolism ; Glutathione Transferase/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Tat-Biliverdin Reductase A Exerts a Protective Role in Oxidative Stress-Induced Hippocampal Neuronal Cell Damage by Regulating the Apoptosis and MAPK Signaling.
    Autorzy:
    Kim SJ; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Shin MJ; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Kim DW; Department of Biochemistry and Molecular Biology, Research Institute of Oral Sciences, College of Dentistry, Gangneung-Wonju National University, Gangneung 25457, Korea.
    Yeo HJ; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Yeo EJ; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Choi YJ; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Sohn EJ; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Han KH; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Park J; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Lee KW; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Park JK; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Cho YJ; Department of Neurosurgery, Hallym University Medical Center, Chuncheon 24253, Korea.
    Kim DS; Department of Anatomy, College of Medicine, Soonchunhyang University, Cheonan-Si 31538, Korea.
    Eum WS; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
    Choi SY; Department of Biomedical Science and Research, Institute of Bioscience and Biotechnology, Hallym University, Chuncheon 24252, Korea.
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    Źródło:
    International journal of molecular sciences [Int J Mol Sci] 2020 Apr 11; Vol. 21 (8). Date of Electronic Publication: 2020 Apr 11.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Gene Products, tat*/genetics
    Oxidoreductases Acting on CH-CH Group Donors*/genetics
    Apoptosis/*drug effects
    MAP Kinase Signaling System/*drug effects
    Oxidative Stress/*drug effects
    Pyramidal Cells/*drug effects
    Pyramidal Cells/*metabolism
    Recombinant Fusion Proteins/*pharmacology
    Animals ; Brain Ischemia/drug therapy ; Brain Ischemia/etiology ; Brain Ischemia/metabolism ; Brain Ischemia/pathology ; Cell Line ; Disease Models, Animal ; Gerbillinae ; Hydrogen Peroxide/metabolism ; Male ; Neuroprotective Agents/pharmacology ; Reactive Oxygen Species/metabolism ; Recombinant Fusion Proteins/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    PEP‑1‑glutaredoxin 1 protects against hippocampal neuronal cell damage from oxidative stress via regulation of MAPK and apoptotic signaling pathways.
    Autorzy:
    Ryu EJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Kim DW; Department of Biochemistry and Molecular Biology, Research Institute of Oral Sciences, College of Dentistry, Gangneung‑Wonju National University, Gangneung, Gangwon 25457, Republic of South Korea.
    Shin MJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Jo HS; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Park JH; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Cho SB; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Lee CH; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Yeo HJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Yeo EJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Choi YJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Kim DS; Department of Anatomy, College of Medicine, Soonchunhyang University, Cheonan‑Si, South Chungcheong 31538, Republic of South Korea.
    Cho SW; Department of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, Seoul 05505, Republic of South Korea.
    Cho YJ; Department of Neurosurgery, Hallym University Medical Center, Chuncheon, Gangwon 24253, Republic of South Korea.
    Sohn EJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Son O; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Lee KW; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Han KH; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Park J; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Eum WS; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
    Choi SY; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Gangwon 24252, Republic of South Korea.
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    Źródło:
    Molecular medicine reports [Mol Med Rep] 2018 Aug; Vol. 18 (2), pp. 2216-2228. Date of Electronic Publication: 2018 Jun 15.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Cysteamine/*analogs & derivatives
    Glutaredoxins/*pharmacology
    Hippocampus/*metabolism
    MAP Kinase Signaling System/*drug effects
    Neurons/*metabolism
    Oxidative Stress/*drug effects
    Peptides/*pharmacology
    Animals ; Brain Ischemia/drug therapy ; Brain Ischemia/metabolism ; Brain Ischemia/pathology ; Cell Line ; Cysteamine/pharmacology ; Hippocampus/pathology ; Mice ; Neurons/pathology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    N-Adamantyl-4-Methylthiazol-2-Amine Attenuates Glutamate-Induced Oxidative Stress and Inflammation in the Brain.
    Autorzy:
    Yang SJ; Department of Biomedical Laboratory Science, Konyang University, Daejeon, 35365, South Korea.
    Kim EA; Department of Biomedical Laboratory Science, Konyang University, Daejeon, 35365, South Korea.
    Chang MJ; Department of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, 88, Olympic-ro 43-Gil, Songpa-gu, Seoul, 05505, South Korea.; Sema High School, 559-17, Sumokwon-Ro, Osan-si, Kyeonggi-do, 18107, South Korea.
    Kim J; Department of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, 88, Olympic-ro 43-Gil, Songpa-gu, Seoul, 05505, South Korea.
    Na JM; Department of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, 88, Olympic-ro 43-Gil, Songpa-gu, Seoul, 05505, South Korea.
    Choi SY; Department of Biomedical Science and Research Institute for Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, South Korea.
    Cho SW; Department of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, 88, Olympic-ro 43-Gil, Songpa-gu, Seoul, 05505, South Korea. .
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    Źródło:
    Neurotoxicity research [Neurotox Res] 2017 Jul; Vol. 32 (1), pp. 107-120. Date of Electronic Publication: 2017 Mar 11.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Encephalitis*/chemically induced
    Encephalitis*/drug therapy
    Encephalitis*/physiopathology
    Anti-Inflammatory Agents/*therapeutic use
    Azetidines/*therapeutic use
    Excitatory Amino Acid Agonists/*toxicity
    Glutamic Acid/*toxicity
    Naphthalenes/*therapeutic use
    Oxidative Stress/*drug effects
    Adenosine Triphosphate/metabolism ; Animals ; Cytokines/metabolism ; Disease Models, Animal ; Electron Transport Complex IV/metabolism ; Lipid Peroxidation/drug effects ; Male ; Mitochondria/drug effects ; Nitric Oxide/metabolism ; Rats ; Rats, Sprague-Dawley ; Reactive Oxygen Species/metabolism ; Sodium-Potassium-Exchanging ATPase/metabolism ; Statistics, Nonparametric
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Tat-HSP22 inhibits oxidative stress-induced hippocampal neuronal cell death by regulation of the mitochondrial pathway.
    Autorzy:
    Jo HS; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Kim DW; Department of Biochemistry and Molecular Biology, Research Institute of Oral Sciences, College of Dentistry, Gangneung-Wonju National University, Gangneung, 25457, Korea.
    Shin MJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Cho SB; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Park JH; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Lee CH; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Yeo EJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Choi YJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Yeo HJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Sohn EJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Son O; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Cho SW; Department of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, Seoul, 05505, Korea.
    Kim DS; Department of Anatomy, College of Medicine, Soonchunhyang University, Cheonan-Si, 31538, Korea.
    Yu YH; Department of Anatomy, College of Medicine, Soonchunhyang University, Cheonan-Si, 31538, Korea.
    Lee KW; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Park J; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea.
    Eum WS; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea. .
    Choi SY; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, 24252, Korea. .
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    Źródło:
    Molecular brain [Mol Brain] 2017 Jan 04; Vol. 10 (1), pp. 1. Date of Electronic Publication: 2017 Jan 04.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Gene Products, tat/*pharmacology
    Heat-Shock Proteins/*pharmacology
    Hippocampus/*pathology
    Mitochondria/*metabolism
    Neurons/*pathology
    Oxidative Stress/*drug effects
    Protein Serine-Threonine Kinases/*pharmacology
    Recombinant Fusion Proteins/*pharmacology
    Signal Transduction/*drug effects
    Animals ; Cell Death/drug effects ; Cell Membrane Permeability/drug effects ; Gerbillinae ; Hydrogen Peroxide/pharmacology ; Male ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/drug effects ; Molecular Chaperones ; Neurons/drug effects ; Neurons/metabolism ; Neuroprotective Agents/pharmacology ; Recombinant Fusion Proteins/isolation & purification ; Transduction, Genetic
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Transduced Tat-DJ-1 protein protects against oxidative stress-induced SH-SY5Y cell death and Parkinson disease in a mouse model.
    Autorzy:
    Jeong HJ; Department of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chunchon 200-702, Korea.
    Kim DW
    Woo SJ
    Kim HR
    Kim SM
    Jo HS
    Park M
    Kim DS
    Kwon OS
    Hwang IK
    Han KH
    Park J
    Eum WS
    Choi SY
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    Źródło:
    Molecules and cells [Mol Cells] 2012 May; Vol. 33 (5), pp. 471-8. Date of Electronic Publication: 2012 Apr 17.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Gene Products, tat/*genetics
    Gene Products, tat/*metabolism
    Oncogene Proteins/*genetics
    Oxidative Stress/*physiology
    Parkinson Disease/*genetics
    Animals ; Antioxidants/metabolism ; Cell Death/genetics ; Cell Death/physiology ; Dopaminergic Neurons/metabolism ; Dopaminergic Neurons/physiology ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; Neuroprotective Agents/metabolism ; Oncogene Proteins/metabolism ; Parkinson Disease/metabolism ; Parkinson Disease/pathology ; Peroxiredoxins ; Protein Deglycase DJ-1 ; Reactive Oxygen Species/metabolism ; Substantia Nigra/metabolism ; Substantia Nigra/physiology ; Transduction, Genetic/methods
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-6 z 6

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