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Tytuł:
Genes and compounds that increase type VII collagen expression as potential treatments for dystrophic epidermolysis bullosa.
Autorzy:
Thompson EL; Department of Biochemistry, Molecular Biology, and Biophysics, University of Minnesota, Minneapolis, Minnesota, USA.
Pickett-Leonard M; Department of Pediatrics, University of Minnesota Medical School, Minneapolis, Minnesota, USA.
Riddle MJ; Department of Pediatrics, University of Minnesota Medical School, Minneapolis, Minnesota, USA.
Chen W; Department of Pediatrics, University of Minnesota Medical School, Minneapolis, Minnesota, USA.
Albert FW; Department of Genetics, Cell Biology and Development, University of Minnesota, Minneapolis, Minnesota, USA.
Tolar J; Department of Pediatrics, University of Minnesota Medical School, Minneapolis, Minnesota, USA.; Stem Cell Institute, University of Minnesota, Minnesota, USA.
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Źródło:
Experimental dermatology [Exp Dermatol] 2022 Jul; Vol. 31 (7), pp. 1065-1075. Date of Electronic Publication: 2022 Mar 09.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
MeSH Terms:
Collagen Type VII*
Epidermolysis Bullosa Dystrophica*/drug therapy
Epidermolysis Bullosa Dystrophica*/genetics
Cell Line ; Humans ; Keratinocytes/metabolism ; Mutation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Murine type VII collagen distorts outcome in human skin graft mouse model for dystrophic epidermolysis bullosa.
Autorzy:
Bremer J; Department of Dermatology, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Department of Genetics, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Kramer D; Department of Dermatology, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Eichhorn DS; Department of Dermatology, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Gostyński A; Department of Dermatology, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Diercks GFH; Department of Dermatology, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Department of Pathology, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Jonkman MF; Department of Dermatology, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
van den Akker PC; Department of Dermatology, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.; Department of Genetics, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Pasmooij AMG; Department of Dermatology, Center of Expertise of Blistering Diseases, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
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Źródło:
Experimental dermatology [Exp Dermatol] 2019 Oct; Vol. 28 (10), pp. 1153-1155. Date of Electronic Publication: 2018 Aug 20.
Typ publikacji:
Letter; Research Support, Non-U.S. Gov't
MeSH Terms:
Skin Transplantation*
Collagen Type VII/*biosynthesis
Collagen Type VII/*metabolism
Epidermolysis Bullosa Dystrophica/*pathology
Fibroblasts/*metabolism
Keratinocytes/*metabolism
Animals ; Antibodies, Heterophile/immunology ; Basement Membrane/metabolism ; Cells, Cultured ; Collagen Type VII/deficiency ; Collagen Type VII/genetics ; Collagen Type VII/immunology ; Dermis/pathology ; Epidermolysis Bullosa Dystrophica/immunology ; Female ; Fibroblasts/transplantation ; Gene Expression ; Heterografts ; Humans ; Keratinocytes/transplantation ; Male ; Mice ; Mice, SCID ; Models, Animal ; Skin Window Technique
Raport
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
T-cell activation and bacterial infection in skin wounds of recessive dystrophic epidermolysis bullosa patients.
Autorzy:
Alexeev V; Department of Dermatology and Cutaneous Biology, Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, Pennsylvania, USA.
Huitema L; Department of Dermatology and Cutaneous Biology, Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, Pennsylvania, USA.
Phillips T; Department of Dermatology and Cutaneous Biology, Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, Pennsylvania, USA.
Cepeda R; DEBRA MEXICO, Guadalupe, Mexico.; Julio Salas Dermatology, Guadalupe, Mexico.
de Los Cobos D; DEBRA MEXICO, Guadalupe, Mexico.
Perez RIM; DEBRA MEXICO, Guadalupe, Mexico.
Salas-Garza M; DEBRA MEXICO, Guadalupe, Mexico.
Fajardo-Ramirez OR; School of Medicine and Health Sciences, Thechnological University of Monterrey, Monterrey, Mexico.
Ringpfeil F; Ringpfeil Advanced Dermatology, Haverford, Pennsylvania, USA.
Uitto J; Department of Dermatology and Cutaneous Biology, Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, Pennsylvania, USA.
Salas-Alanis JC; DEBRA MEXICO, Guadalupe, Mexico.; Julio Salas Dermatology, Guadalupe, Mexico.
Igoucheva O; Department of Dermatology and Cutaneous Biology, Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, Pennsylvania, USA.
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Źródło:
Experimental dermatology [Exp Dermatol] 2022 Sep; Vol. 31 (9), pp. 1431-1442. Date of Electronic Publication: 2022 Jun 06.
Typ publikacji:
Journal Article
MeSH Terms:
Bacterial Infections*
Epidermolysis Bullosa Dystrophica*/pathology
T-Lymphocytes*/immunology
Antigens ; Collagen Type VII ; Humans ; Keratinocytes/pathology ; Lymphocyte Activation ; Programmed Cell Death 1 Receptor ; Staphylococcus aureus
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Transcriptomic profiling of recessive dystrophic epidermolysis bullosa wounded skin highlights drug repurposing opportunities to improve wound healing.
Autorzy:
Onoufriadis A; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK.
Proudfoot LE; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK.
Ainali C; Dignosis Ltd, London, UK.
Torre D; Department of Pharmacological Sciences, Mount Sinai Center for Bioinformatics, Icahn School of Medicine at Mount Sinai, New York City, New York, USA.
Papanikolaou M; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK.
Rayinda T; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK.; Department of Dermatology and Venereology, Faculty of Medicine, Public Health, and Nursing, Universitas Gadjah Mada/Dr. Sardjito Hospital, Yogyakarta, Indonesia.
Rashidghamat E; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK.
Danarti R; Department of Dermatology and Venereology, Faculty of Medicine, Public Health, and Nursing, Universitas Gadjah Mada/Dr. Sardjito Hospital, Yogyakarta, Indonesia.
Mellerio JE; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK.
Ma'ayan A; Department of Pharmacological Sciences, Mount Sinai Center for Bioinformatics, Icahn School of Medicine at Mount Sinai, New York City, New York, USA.
McGrath JA; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK.
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Źródło:
Experimental dermatology [Exp Dermatol] 2022 Mar; Vol. 31 (3), pp. 420-426. Date of Electronic Publication: 2021 Nov 02.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Drug Repositioning*
Epidermolysis Bullosa Dystrophica*/drug therapy
Epidermolysis Bullosa Dystrophica*/genetics
Collagen Type VII/genetics ; Collagen Type VII/metabolism ; Cytokines/genetics ; Genes, Recessive ; Humans ; Skin/metabolism ; Transcriptome ; Wound Healing
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Evidence for a role of extracellular heat shock protein 70 in epidermolysis bullosa acquisita.
Autorzy:
Tukaj S; Department of Molecular Biology, Faculty of Biology, University of Gdańsk, Gdańsk, Poland.
Mantej J; Department of Molecular Biology, Faculty of Biology, University of Gdańsk, Gdańsk, Poland.
Sitko K; Department of Molecular Biology, Faculty of Biology, University of Gdańsk, Gdańsk, Poland.
Bednarek M; Department of Molecular Biology, Faculty of Biology, University of Gdańsk, Gdańsk, Poland.
Zillikens D; Department of Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Ludwig RJ; Lübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Bieber K; Lübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Kasperkiewicz M; Department of Dermatology, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA.
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Źródło:
Experimental dermatology [Exp Dermatol] 2022 Apr; Vol. 31 (4), pp. 528-534. Date of Electronic Publication: 2021 Nov 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autoimmune Diseases*
Epidermolysis Bullosa Acquisita*
Animals ; Autoantibodies ; Collagen Type VII ; HSP70 Heat-Shock Proteins ; Leukocytes, Mononuclear/metabolism ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A non-viral gene therapy for treatment of recessive dystrophic epidermolysis bullosa.
Autorzy:
Cutlar L; Charles Institute of Dermatology, School of Medicine and Medical Science, University College Dublin, Dublin 4, Ireland.
Zhou D; Charles Institute of Dermatology, School of Medicine and Medical Science, University College Dublin, Dublin 4, Ireland.
Hu X; Centre for Medical Research, School of Medicine, Dalian University, Dalian Economical and Technological Development Zone, Liao Ning Province, China.
Duarte B; Cutaneous Diseases Modelling Unit, Division of Biomedicine, CIEMAT, Madrid, Spain.
Greiser U; Charles Institute of Dermatology, School of Medicine and Medical Science, University College Dublin, Dublin 4, Ireland.
Larcher F; Cutaneous Diseases Modelling Unit, Division of Biomedicine, CIEMAT, Madrid, Spain.
Wang W; Charles Institute of Dermatology, School of Medicine and Medical Science, University College Dublin, Dublin 4, Ireland. .
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Źródło:
Experimental dermatology [Exp Dermatol] 2016 Oct; Vol. 25 (10), pp. 818-20. Date of Electronic Publication: 2016 Jun 30.
Typ publikacji:
Evaluation Study; Letter; Research Support, Non-U.S. Gov't
MeSH Terms:
Collagen Type VII/*genetics
Epidermolysis Bullosa Dystrophica/*therapy
Genetic Therapy/*methods
Animals ; Collagen Type VII/metabolism ; Humans ; Mice ; Skin/metabolism
Raport
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Translational perspectives to treat Epidermolysis bullosa-Where do we stand?
Autorzy:
Prodinger C; Department of Dermatology and Allergology, University Hospital of the Paracelsus Medical University Salzburg, Salzburg, Austria.
Bauer JW; Department of Dermatology and Allergology, University Hospital of the Paracelsus Medical University Salzburg, Salzburg, Austria.
Laimer M; Department of Dermatology and Allergology, University Hospital of the Paracelsus Medical University Salzburg, Salzburg, Austria.
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Źródło:
Experimental dermatology [Exp Dermatol] 2020 Nov; Vol. 29 (11), pp. 1112-1122.
Typ publikacji:
Journal Article
MeSH Terms:
Genetic Therapy*
Carcinoma, Squamous Cell/*drug therapy
Epidermolysis Bullosa/*therapy
Skin Neoplasms/*drug therapy
Antineoplastic Agents/therapeutic use ; Carcinoma, Squamous Cell/etiology ; Cell- and Tissue-Based Therapy ; Clinical Trials as Topic ; Codon, Nonsense ; Collagen Type VII/therapeutic use ; Epidermolysis Bullosa/complications ; Epidermolysis Bullosa/genetics ; Humans ; Immunotherapy ; Molecular Targeted Therapy ; Pain/drug therapy ; Pain/etiology ; Pruritus/drug therapy ; Pruritus/etiology ; Recombinant Proteins/therapeutic use ; Skin Neoplasms/etiology ; Translational Research, Biomedical
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Mutation analysis and characterization of COL7A1 mutations in dystrophic epidermolysis bullosa.
Autorzy:
Dang N; Department of Dermatology, St George Hospital, Sydney, Australia.
Murrell DF
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Źródło:
Experimental dermatology [Exp Dermatol] 2008 Jul; Vol. 17 (7), pp. 553-68.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Mutation*
Collagen Type VII/*genetics
Epidermolysis Bullosa Dystrophica/*genetics
Amino Acid Sequence ; Amino Acid Substitution ; Base Sequence ; Collagen Type VII/chemistry ; Collagen Type VII/metabolism ; DNA Mutational Analysis ; Epidermolysis Bullosa Dystrophica/metabolism ; Epidermolysis Bullosa Dystrophica/pathology ; Epidermolysis Bullosa Dystrophica/therapy ; Exons ; Genetic Therapy ; Glycine/genetics ; Humans ; Inheritance Patterns/genetics ; Molecular Sequence Data ; Phenotype ; Skin/metabolism ; Skin/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Quantitative analysis of the synthesis and secretion of type VII collagen in cultured human dermal fibroblasts with a sensitive sandwich enzyme-linked immunoassay.
Autorzy:
Amano S; Shiseido Life Science Research Center, Yokohama, Japan. />Ogura Y
Akutsu N
Nishiyama T
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Źródło:
Experimental dermatology [Exp Dermatol] 2007 Feb; Vol. 16 (2), pp. 151-5.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Collagen Type VII/*biosynthesis
Enzyme-Linked Immunosorbent Assay/*methods
Fibroblasts/*metabolism
Skin/*metabolism
Adolescent ; Adult ; Age Factors ; Aged ; Aged, 80 and over ; Ascorbic Acid/physiology ; Cells, Cultured ; Child ; Child, Preschool ; Collagen Type VII/analysis ; Collagen Type VII/metabolism ; Cytokines/physiology ; Humans ; Infant ; Infant, Newborn ; Middle Aged ; Transforming Growth Factor beta1/physiology ; Tumor Necrosis Factor-alpha/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Unique mouse monoclonal antibodies reactive with maturation-related epitopes on type VII collagen.
Autorzy:
Hayakawa T; Department of Dermatology, Kurume University School of Medicine, Kurume University Institute of Cutaneous Cell Biology, Kurume, Japan.; Department of Oral and Maxillofacial Surgery, School of Dentistry, Aichi-Gakuin University, Nagoya, Japan.
Hirako Y; Division of Biological Science, Graduate School of Science, Nagoya University, Nagoya, Japan.
Teye K; Department of Dermatology, Kurume University School of Medicine, Kurume University Institute of Cutaneous Cell Biology, Kurume, Japan.
Tsuchisaka A; Department of Dermatology, Kurume University School of Medicine, Kurume University Institute of Cutaneous Cell Biology, Kurume, Japan.
Koga H; Department of Dermatology, Kurume University School of Medicine, Kurume University Institute of Cutaneous Cell Biology, Kurume, Japan.
Ishii N; Department of Dermatology, Kurume University School of Medicine, Kurume University Institute of Cutaneous Cell Biology, Kurume, Japan.
Karashima T; Department of Dermatology, Kurume University School of Medicine, Kurume University Institute of Cutaneous Cell Biology, Kurume, Japan.
Kaneda M; Division of Biological Science, Graduate School of Science, Nagoya University, Nagoya, Japan.
Oyu Y; Division of Biological Science, Graduate School of Science, Nagoya University, Nagoya, Japan.
Tateishi C; Department of Dermatology, Osaka City University Graduate School of Medicine, Osaka, Japan.
Sugawara K; Department of Dermatology, Osaka City University Graduate School of Medicine, Osaka, Japan.
Yonamine A; Department of Dermatology, Osaka City University Graduate School of Medicine, Osaka, Japan.
Shinkuma S; Department of Dermatology, Hokkaido University Graduate School of Medicine, Sapporo, Japan.
Shimizu H; Department of Dermatology, Hokkaido University Graduate School of Medicine, Sapporo, Japan.
Fukano H; Department of Oral and Maxillofacial Surgery, School of Dentistry, Aichi-Gakuin University, Nagoya, Japan.
Shimozato K; Department of Oral and Maxillofacial Surgery, School of Dentistry, Aichi-Gakuin University, Nagoya, Japan.
Nguyen NT; Department of Dermatology, Stanford University School of Medicine, Center for Clinical Sciences Research, Stanford, CA, USA.
Marinkovich MP; Department of Dermatology, Stanford University School of Medicine, Center for Clinical Sciences Research, Stanford, CA, USA.; Department of Dermatology, Veterans Administration Hospital, Palo Alto Medical Center, Palo Alto, CA, USA.
Tsuruta D; Department of Dermatology, Osaka City University Graduate School of Medicine, Osaka, Japan.
Hashimoto T; Department of Dermatology, Kurume University School of Medicine, Kurume University Institute of Cutaneous Cell Biology, Kurume, Japan.
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Źródło:
Experimental dermatology [Exp Dermatol] 2017 Sep; Vol. 26 (9), pp. 811-819. Date of Electronic Publication: 2017 Apr 20.
Typ publikacji:
Evaluation Study; Journal Article
MeSH Terms:
Basement Membrane/*metabolism
Collagen Type VII/*immunology
Collagen Type VII/*metabolism
Animals ; Antibodies, Monoclonal/biosynthesis ; Cattle ; HEK293 Cells ; Humans ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Immunomapping of EBA sera to multiple epitopes on collagen VII: further evidence that anchoring fibrils originate and terminate in the lamina densa.
Autorzy:
McMillan JR; Department of Dermatology, Hokkaido University Graduate School of Medicine, Sapporo, Japan. />Matsumura T
Hashimoto T
Schumann H
Bruckner-Tuderman L
Shimizu H
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Źródło:
Experimental dermatology [Exp Dermatol] 2003 Jun; Vol. 12 (3), pp. 261-7.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epitope Mapping*
Collagen Type VII/*immunology
Epidermis/*immunology
Epidermolysis Bullosa Acquisita/*immunology
Adult ; Aged ; Autoantibodies/pharmacology ; Blotting, Western ; Collagen Type VII/chemistry ; Epidermis/pathology ; Epidermis/ultrastructure ; Epidermolysis Bullosa Acquisita/pathology ; Female ; Humans ; Male ; Microscopy, Immunoelectron ; Middle Aged ; Protein Structure, Tertiary
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Evidence for a contributory role of a xenogeneic immune response in experimental epidermolysis bullosa acquisita.
Autorzy:
Niebuhr M; Institute of Anatomy, University of Lübeck, Lübeck, Germany.
Kasperkiewicz M; Department of Dermatology, University of Lübeck, Lübeck, Germany.
Maass S; Institute of Anatomy, University of Lübeck, Lübeck, Germany.
Hauenschild E; Institute of Anatomy, University of Lübeck, Lübeck, Germany.
Bieber K; Lübeck Institute of Experimental Dermatology, University of Lübeck, Lübeck, Germany.
Ludwig RJ; Department of Dermatology, University of Lübeck, Lübeck, Germany.; Lübeck Institute of Experimental Dermatology, University of Lübeck, Lübeck, Germany.
Westermann J; Institute of Anatomy, University of Lübeck, Lübeck, Germany.
Kalies K; Institute of Anatomy, University of Lübeck, Lübeck, Germany.
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Źródło:
Experimental dermatology [Exp Dermatol] 2017 Dec; Vol. 26 (12), pp. 1207-1213.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antibodies, Heterophile/*metabolism
Collagen Type VII/*immunology
Epidermolysis Bullosa Acquisita/*immunology
Animals ; Disease Models, Animal ; Fingolimod Hydrochloride ; Immunoglobulin G/blood ; Interferon-gamma/metabolism ; Lymph Nodes/immunology ; Mice, Inbred C57BL ; Rabbits ; T-Lymphocytes/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Structural proteins of the dermal-epidermal junction targeted by autoantibodies in pemphigoid diseases.
Autorzy:
Goletz S; Lübeck Institute of Experimental Dermatology (LIED), University of Lübeck, Lübeck, Germany.
Zillikens D; Department of Dermatology, University of Lübeck, Lübeck, Germany.
Schmidt E; Lübeck Institute of Experimental Dermatology (LIED), University of Lübeck, Lübeck, Germany.; Department of Dermatology, University of Lübeck, Lübeck, Germany.
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Źródło:
Experimental dermatology [Exp Dermatol] 2017 Dec; Vol. 26 (12), pp. 1154-1162.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Autoantibodies/*immunology
Skin Diseases, Vesiculobullous/*immunology
Animals ; Autoantigens/immunology ; Collagen Type VII/immunology ; Dystonin/immunology ; Humans ; Integrin alpha6beta4/immunology ; Laminin/immunology ; Non-Fibrillar Collagens/immunology ; Plectin/immunology ; Collagen Type XVII
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
In vivo enzymatic modulation of IgG antibodies prevents immune complex-dependent skin injury.
Autorzy:
Mihai S; Department of Biology, Institute of Genetics, University of Erlangen-Nuremberg, Erlangen, Germany.; Department of Clinical Chemistry, University Hospital Erlangen, Erlangen, Germany.
Albert H; Department of Biology, Institute of Genetics, University of Erlangen-Nuremberg, Erlangen, Germany.
Ludwig RJ; Department of Dermatology, University of Lübeck, Lübeck, Germany.
Iwata H; Department of Dermatology, University of Lübeck, Lübeck, Germany.
Björck L; Division of Infection Medicine, Department of Clinical Sciences, Lund University, Lund, Sweden.
Collin M; Division of Infection Medicine, Department of Clinical Sciences, Lund University, Lund, Sweden.
Nimmerjahn F; Department of Biology, Institute of Genetics, University of Erlangen-Nuremberg, Erlangen, Germany.
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Źródło:
Experimental dermatology [Exp Dermatol] 2017 Aug; Vol. 26 (8), pp. 691-696. Date of Electronic Publication: 2016 Dec 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Arthus Reaction/*prevention & control
Bacterial Proteins/*therapeutic use
Epidermolysis Bullosa Acquisita/*prevention & control
Glycoside Hydrolases/*therapeutic use
Animals ; Bacterial Proteins/metabolism ; Bacterial Proteins/pharmacology ; Collagen Type VII/immunology ; Drug Evaluation, Preclinical ; Female ; Glycoside Hydrolases/metabolism ; Glycoside Hydrolases/pharmacology ; Immunoglobulin G/metabolism ; Mice, Inbred C57BL
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Heat shock protein 90 is required for ex vivo neutrophil-driven autoantibody-induced tissue damage in experimental epidermolysis bullosa acquisita.
Autorzy:
Tukaj S; Department of Dermatology, University of Lübeck, Lübeck, Germany.
Hellberg L; Institute of Medical Microbiology and Hygiene, University of Lübeck, Lübeck, Germany.
Ueck C; Institute of Medical Microbiology and Hygiene, University of Lübeck, Lübeck, Germany.
Hänsel M; Department of Dermatology, University of Lübeck, Lübeck, Germany.
Samavedam U; Department of Dermatology, University of Lübeck, Lübeck, Germany.
Zillikens D; Department of Dermatology, University of Lübeck, Lübeck, Germany.
Ludwig RJ; Department of Dermatology, University of Lübeck, Lübeck, Germany.
Laskay T; Institute of Medical Microbiology and Hygiene, University of Lübeck, Lübeck, Germany.
Kasperkiewicz M; Department of Dermatology, University of Lübeck, Lübeck, Germany.
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Źródło:
Experimental dermatology [Exp Dermatol] 2015 Jun; Vol. 24 (6), pp. 471-3. Date of Electronic Publication: 2015 Mar 25.
Typ publikacji:
Letter; Research Support, Non-U.S. Gov't
MeSH Terms:
Autoantibodies/*physiology
Epidermolysis Bullosa Acquisita/*pathology
Epidermolysis Bullosa Acquisita/*physiopathology
HSP90 Heat-Shock Proteins/*physiology
Neutrophils/*pathology
Skin/*pathology
Autoantibodies/immunology ; Benzoquinones/pharmacology ; Case-Control Studies ; Cells, Cultured ; Collagen Type VII/immunology ; Dose-Response Relationship, Drug ; Epidermolysis Bullosa Acquisita/immunology ; HSP90 Heat-Shock Proteins/antagonists & inhibitors ; HSP90 Heat-Shock Proteins/drug effects ; Humans ; Immunoprecipitation ; Lactams, Macrocyclic/pharmacology ; Matrix Metalloproteinase 12/physiology ; Matrix Metalloproteinase 2/physiology ; Neutrophils/drug effects ; Neutrophils/metabolism ; Reactive Oxygen Species/metabolism ; Skin/drug effects ; Skin/physiopathology
Raport
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Gene therapy: pursuing restoration of dermal adhesion in recessive dystrophic epidermolysis bullosa.
Autorzy:
Cutlar L; Network of Excellence for Functional Biomaterials, National University of Ireland, Galway, Ireland.
Greiser U
Wang W
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Źródło:
Experimental dermatology [Exp Dermatol] 2014 Jan; Vol. 23 (1), pp. 1-6.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Epidermolysis Bullosa Dystrophica/*genetics
Epidermolysis Bullosa Dystrophica/*therapy
Genetic Therapy/*methods
Adhesiveness ; Animals ; Autografts ; Collagen Type VII/deficiency ; Collagen Type VII/genetics ; Collagen Type VII/physiology ; Epidermolysis Bullosa Dystrophica/physiopathology ; Genes, Recessive ; Genetic Vectors ; Humans ; Induced Pluripotent Stem Cells/transplantation ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Keratinocyte cell lines derived from severe generalized recessive epidermolysis bullosa patients carrying a highly recurrent COL7A1 homozygous mutation: models to assess cell and gene therapies in vitro and in vivo.
Autorzy:
Chamorro C; Epithelial Biomedicine Division, Cutaneous Disease Modelling Unit, CIEMAT, Madrid, Spain.
Almarza D
Duarte B
Llames SG
Murillas R
García M
Cigudosa JC
Espinosa-Hevia L
Escámez MJ
Mencía A
Meana A
García-Escudero R
Moro R
Conti CJ
Del Río M
Larcher F
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Źródło:
Experimental dermatology [Exp Dermatol] 2013 Sep; Vol. 22 (9), pp. 601-3.
Typ publikacji:
Letter; Research Support, Non-U.S. Gov't
MeSH Terms:
Mutation*
Collagen Type VII/*genetics
Epidermolysis Bullosa Dystrophica/*genetics
Epidermolysis Bullosa Dystrophica/*therapy
Keratinocytes/*metabolism
Animals ; Cell Line ; Cell- and Tissue-Based Therapy ; Epidermolysis Bullosa Dystrophica/pathology ; Genetic Therapy ; Heterografts ; Homozygote ; Humans ; Keratinocytes/transplantation ; Mice ; Models, Genetic ; Regeneration
Raport
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Tytuł:
Wool-derived keratin stimulates human keratinocyte migration and types IV and VII collagen expression.
Autorzy:
Tang L
Sierra JO
Kelly R
Kirsner RS
Li J
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Źródło:
Experimental dermatology [Exp Dermatol] 2012 Jun; Vol. 21 (6), pp. 458-60.
Typ publikacji:
Letter; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Movement/*drug effects
Keratinocytes/*drug effects
Keratins/*pharmacology
Wound Healing/*drug effects
Animals ; Cells, Cultured ; Collagen Type IV/metabolism ; Collagen Type VII/metabolism ; Humans ; Keratinocytes/metabolism ; Wool/chemistry
Raport
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
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