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Wyszukujesz frazę ""Cyclin B1"" wg kryterium: Temat


Tytuł :
Toxoplasma gondii ROP16 kinase silences the cyclin B1 gene promoter by hijacking host cell UHRF1-dependent epigenetic pathways.
Autorzy :
Sabou M; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France.; Service de Parasitologie et Mycologie Médicale, Hôpitaux Universitaires de Strasbourg, Centre National de Référence de la Toxoplasmose, Pôle Sérologie, Strasbourg, France.
Doderer-Lang C; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France.
Leyer C; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France.
Konjic A; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France.
Kubina S; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France.
Lennon S; Laboratoire de Spectrométrie de Masse BioOrganique (LSMBO), Université de Strasbourg, IPHC, CNRS, UMR7178, Strasbourg, France.
Rohr O; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France.
Viville S; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France.
Cianférani S; Laboratoire de Spectrométrie de Masse BioOrganique (LSMBO), Université de Strasbourg, IPHC, CNRS, UMR7178, Strasbourg, France.
Candolfi E; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France.; Service de Parasitologie et Mycologie Médicale, Hôpitaux Universitaires de Strasbourg, Centre National de Référence de la Toxoplasmose, Pôle Sérologie, Strasbourg, France.
Pfaff AW; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France. .; Service de Parasitologie et Mycologie Médicale, Hôpitaux Universitaires de Strasbourg, Centre National de Référence de la Toxoplasmose, Pôle Sérologie, Strasbourg, France. .
Brunet J; Institut de Parasitologie et de Pathologie Tropicale de Strasbourg, « Dynamics of Host-Pathogen Interactions » EA 7292, Fédération de Médecine Translationelle Université de Strasbourg, Strasbourg, France.; Service de Parasitologie et Mycologie Médicale, Hôpitaux Universitaires de Strasbourg, Centre National de Référence de la Toxoplasmose, Pôle Sérologie, Strasbourg, France.
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Źródło :
Cellular and molecular life sciences : CMLS [Cell Mol Life Sci] 2020 Jun; Vol. 77 (11), pp. 2141-2156. Date of Electronic Publication: 2019 Sep 06.
Typ publikacji :
Journal Article
MeSH Terms :
CCAAT-Enhancer-Binding Proteins/*genetics
Cyclin B1/*genetics
Protein-Tyrosine Kinases/*metabolism
Protozoan Proteins/*metabolism
Toxoplasma/*physiology
Toxoplasmosis/*genetics
Ubiquitin-Protein Ligases/*genetics
CCAAT-Enhancer-Binding Proteins/metabolism ; Cell Cycle Checkpoints ; Cell Line ; Cyclin B1/metabolism ; Epigenesis, Genetic ; Host-Parasite Interactions ; Humans ; Phosphorylation ; Promoter Regions, Genetic ; Toxoplasmosis/metabolism ; Ubiquitin-Protein Ligases/metabolism
Czasopismo naukowe
Tytuł :
Orchestration of the spindle assembly checkpoint by CDK1-cyclin B1.
Autorzy :
Hayward D; Sir William Dunn School of Pathology, University of Oxford, UK.
Alfonso-Pérez T; Department of Biochemistry, University of Oxford, UK.
Gruneberg U; Sir William Dunn School of Pathology, University of Oxford, UK.
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Źródło :
FEBS letters [FEBS Lett] 2019 Oct; Vol. 593 (20), pp. 2889-2907. Date of Electronic Publication: 2019 Sep 13.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms :
M Phase Cell Cycle Checkpoints*
CDC2 Protein Kinase/*genetics
Cyclin B1/*genetics
Kinetochores/*metabolism
Microtubules/*metabolism
Spindle Apparatus/*metabolism
CDC2 Protein Kinase/metabolism ; Cell Cycle Proteins/genetics ; Cell Cycle Proteins/metabolism ; Chromosome Segregation ; Chromosomes, Human/chemistry ; Chromosomes, Human/metabolism ; Cyclin B1/metabolism ; Gene Expression Regulation ; HeLa Cells ; Humans ; Kinetochores/ultrastructure ; Microtubules/ultrastructure ; Protein Phosphatase 2/genetics ; Protein Phosphatase 2/metabolism ; Protein-Serine-Threonine Kinases/genetics ; Protein-Serine-Threonine Kinases/metabolism ; Protein-Tyrosine Kinases/genetics ; Protein-Tyrosine Kinases/metabolism ; Receptors, Neuropeptide Y/genetics ; Receptors, Neuropeptide Y/metabolism ; Signal Transduction ; Spindle Apparatus/ultrastructure
Czasopismo naukowe
Tytuł :
CyclinB1 deubiquitination by USP14 regulates cell cycle progression in breast cancer.
Autorzy :
Liu B; School of Basic Medical Sciences, Nanchang University, Nanchang, Jiangxi, PR China.
Liu Y; School of Basic Medical Sciences, Nanchang University, Nanchang, Jiangxi, PR China.
Wang Y; School of Basic Medical Sciences, Nanchang University, Nanchang, Jiangxi, PR China.
Xie C; School of Basic Medical Sciences, Nanchang University, Nanchang, Jiangxi, PR China.
Gan M; School of Basic Medical Sciences, Nanchang University, Nanchang, Jiangxi, PR China.
Han T; Jiangxi Institute of Respiratory Disease, the First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi, PR China. Electronic address: .
Cao J; Department of Gastrointestinal Surgery, the Second Affiliated Hospital of Nanchang University, Nanchang, Jiangxi, PR China. Electronic address: .
Wang J; School of Basic Medical Sciences, Nanchang University, Nanchang, Jiangxi, PR China. Electronic address: .
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Źródło :
Pathology, research and practice [Pathol Res Pract] 2019 Oct; Vol. 215 (10), pp. 152592. Date of Electronic Publication: 2019 Aug 17.
Typ publikacji :
Journal Article
MeSH Terms :
Ubiquitination*
Breast Neoplasms/*metabolism
Cell Cycle/*physiology
Cyclin B1/*metabolism
Ubiquitin Thiolesterase/*metabolism
Breast Neoplasms/genetics ; Breast Neoplasms/pathology ; Cell Line, Tumor ; Cell Movement/physiology ; Cyclin B1/genetics ; Female ; Humans ; Ubiquitin Thiolesterase/genetics
Czasopismo naukowe
Tytuł :
Molecular mechanism of Forkhead box M1 inhibition by thiostrepton in breast cancer cells.
Autorzy :
Kongsema M; Department of Zoology, Faculty of Science, Kasetsart University, Bangkok 10900, Thailand.
Wongkhieo S; Department of Zoology, Faculty of Science, Kasetsart University, Bangkok 10900, Thailand.
Khongkow M; National Nanotechnology Centre (NANOTEC), National Science and Technology Development Agency, Pathum Thani 12120, Thailand.
Lam EW; Department of Surgery and Cancer, Imperial College London, Hammersmith Hospital Campus, London W12 0NN, UK.
Boonnoy P; Computational Biomodelling Laboratory for Agricultural Science and Technology (CBLAST), Faculty of Science, Kasetsart University, Bangkok 10900, Thailand.
Vongsangnak W; Department of Zoology, Faculty of Science, Kasetsart University, Bangkok 10900, Thailand.
Wong-Ekkabut J; Computational Biomodelling Laboratory for Agricultural Science and Technology (CBLAST), Faculty of Science, Kasetsart University, Bangkok 10900, Thailand.
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Źródło :
Oncology reports [Oncol Rep] 2019 Sep; Vol. 42 (3), pp. 953-962. Date of Electronic Publication: 2019 Jul 08.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Bacterial Agents/*pharmacology
Breast Neoplasms/*pathology
Cyclin B1/*antagonists & inhibitors
Forkhead Box Protein M1/*antagonists & inhibitors
Gene Expression Regulation, Neoplastic/*drug effects
Thiostrepton/*pharmacology
Apoptosis ; Biomarkers, Tumor/genetics ; Biomarkers, Tumor/metabolism ; Breast Neoplasms/drug therapy ; Breast Neoplasms/metabolism ; Cell Proliferation ; Cyclin B1/genetics ; Cyclin B1/metabolism ; DNA, Neoplasm/genetics ; DNA, Neoplasm/metabolism ; Female ; Forkhead Box Protein M1/genetics ; Forkhead Box Protein M1/metabolism ; Humans ; Molecular Docking Simulation ; Protein Conformation ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł :
HnRNPR-CCNB1/CENPF axis contributes to gastric cancer proliferation and metastasis.
Autorzy :
Chen EB; Department of Medical Oncology, Zhongshan Hospital, Fudan University, Shanghai, China.
Qin X; School of Chemical Biology and Biotechnology, Shenzhen Graduate School of Peking University, Shenzhen, China.
Peng K; Department of Medical Oncology, Zhongshan Hospital, Fudan University, Shanghai, China.
Li Q; Department of Medical Oncology, Zhongshan Hospital, Fudan University, Shanghai, China.
Tang C; Department of Medical Oncology, Zhongshan Hospital, Fudan University, Shanghai, China.
Wei YC; Department of Medical Oncology, Zhongshan Hospital, Fudan University, Shanghai, China.
Yu S; Department of Medical Oncology, Zhongshan Hospital, Fudan University, Shanghai, China.
Gan L; Department of Medical Oncology, Zhongshan Hospital, Fudan University, Shanghai, China.
Liu TS; Department of Medical Oncology, Zhongshan Hospital, Fudan University, Shanghai, China.; Center of Evidence-based Medicine, Fudan University, Shanghai, China.
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Źródło :
Aging [Aging (Albany NY)] 2019 Sep 16; Vol. 11 (18), pp. 7473-7491. Date of Electronic Publication: 2019 Sep 16.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Proliferation/*physiology
Chromosomal Proteins, Non-Histone/*metabolism
Cyclin B1/*metabolism
Heterogeneous-Nuclear Ribonucleoproteins/*metabolism
Microfilament Proteins/*metabolism
Neoplasm Metastasis/*physiopathology
Stomach Neoplasms/*metabolism
Animals ; Cell Line, Tumor ; Cell Survival ; Chromosomal Proteins, Non-Histone/genetics ; Cyclin B1/genetics ; Gastric Mucosa ; Gene Expression Regulation, Neoplastic ; Heterogeneous-Nuclear Ribonucleoproteins/genetics ; Male ; Mice, Nude ; Microfilament Proteins/genetics ; Neoplasms, Experimental ; RNA Interference ; RNA, Messenger
Czasopismo naukowe
Tytuł :
The circular RNA circ-Ccnb1 dissociates Ccnb1/Cdk1 complex suppressing cell invasion and tumorigenesis.
Autorzy :
Fang L; Sunnybrook Research Institute, Toronto, Canada; China-Japan Union Hospital of Jilin University, Jilin, China.
Du WW; Sunnybrook Research Institute, Toronto, Canada.
Awan FM; Sunnybrook Research Institute, Toronto, Canada; Atta-ur-Rahman School of Applied Biosciences (ASAB), National University of Sciences and Technology (NUST), H-12, Islamabad, Pakistan.
Dong J; Sunnybrook Research Institute, Toronto, Canada; The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, China. Electronic address: .
Yang BB; Sunnybrook Research Institute, Toronto, Canada; Department of Laboratory Medicine and Pathobiology, University of Toronto, Toronto, Canada. Electronic address: .
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Źródło :
Cancer letters [Cancer Lett] 2019 Sep 10; Vol. 459, pp. 216-226. Date of Electronic Publication: 2019 Jun 12.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Breast Neoplasms/*therapy
Cyclin B1/*genetics
DNA, Circular/*administration & dosage
Melanoma, Experimental/*therapy
Animals ; Breast Neoplasms/genetics ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; CDC2 Protein Kinase/metabolism ; Carcinogenesis ; Cell Movement/physiology ; Cyclin B1/metabolism ; DNA, Circular/genetics ; Female ; Humans ; Melanoma, Experimental/genetics ; Melanoma, Experimental/metabolism ; Melanoma, Experimental/pathology ; Mice ; Mice, Inbred C57BL ; Models, Molecular ; Neoplasm Invasiveness ; Signal Transduction ; Transfection
Czasopismo naukowe
Tytuł :
Lack of Cyclin B1 in zebrafish causes lengthening of G2 and M phases.
Autorzy :
Petrachkova T; Department of Biological Sciences, Western Michigan University, Kalamazoo, MI, 49008, USA. Electronic address: .
Wortinger LA; Department of Biological Sciences, Western Michigan University, Kalamazoo, MI, 49008, USA.
Bard AJ; Department of Biological Sciences, Western Michigan University, Kalamazoo, MI, 49008, USA.
Singh J; Department of Biological Sciences, Western Michigan University, Kalamazoo, MI, 49008, USA.
Warga RM; Department of Biological Sciences, Western Michigan University, Kalamazoo, MI, 49008, USA.
Kane DA; Department of Biological Sciences, Western Michigan University, Kalamazoo, MI, 49008, USA.
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Źródło :
Developmental biology [Dev Biol] 2019 Jul 15; Vol. 451 (2), pp. 167-179. Date of Electronic Publication: 2019 Mar 28.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
Cyclin B1/*metabolism
Zebrafish/*embryology
Zebrafish Proteins/*metabolism
Alternative Splicing ; Animals ; Apoptosis ; Chromosomal Instability ; Cyclin B1/genetics ; Embryo, Nonmammalian/cytology ; Embryo, Nonmammalian/metabolism ; G2 Phase ; Gene Editing ; Liver/cytology ; Liver/metabolism ; Mitosis ; Mutation ; Zebrafish/metabolism ; Zebrafish Proteins/genetics
Czasopismo naukowe
Tytuł :
Cyclins B1, T1, and H differ in their molecular mode of interaction with cytomegalovirus protein kinase pUL97.
Autorzy :
Steingruber M; From the Institute for Clinical and Molecular Virology, .
Keller L; From the Institute for Clinical and Molecular Virology.
Socher E; Division of Bioinformatics, Institute of Biochemistry Friedrich-Alexander University of Erlangen-Nürnberg, 91054 Erlangen, Germany.
Ferre S; the Université Grenoble Alpes, CEA, Inserm, BIG-BGE, 38000 Grenoble, France, and.
Hesse AM; the Université Grenoble Alpes, CEA, Inserm, BIG-BGE, 38000 Grenoble, France, and.
Couté Y; the Université Grenoble Alpes, CEA, Inserm, BIG-BGE, 38000 Grenoble, France, and.
Hahn F; From the Institute for Clinical and Molecular Virology.
Büscher N; the Institute for Virology, University Medical Center of the Johannes Gutenberg-University Mainz, 55131 Mainz, Germany.
Plachter B; the Institute for Virology, University Medical Center of the Johannes Gutenberg-University Mainz, 55131 Mainz, Germany.
Sticht H; Division of Bioinformatics, Institute of Biochemistry Friedrich-Alexander University of Erlangen-Nürnberg, 91054 Erlangen, Germany, .
Marschall M; From the Institute for Clinical and Molecular Virology, .
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Źródło :
The Journal of biological chemistry [J Biol Chem] 2019 Apr 12; Vol. 294 (15), pp. 6188-6203. Date of Electronic Publication: 2019 Feb 19.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cyclin B1/*metabolism
Cyclin H/*metabolism
Cyclin T/*metabolism
Cytomegalovirus/*physiology
Viral Proteins/*metabolism
Virus Replication/*physiology
Cyclin B1/genetics ; Cyclin H/genetics ; Cyclin T/genetics ; HEK293 Cells ; Humans ; Phosphorylation ; Protein Domains ; Protein Structure, Quaternary ; Viral Proteins/genetics
Czasopismo naukowe
Tytuł :
CDK1-CCNB1 creates a spindle checkpoint-permissive state by enabling MPS1 kinetochore localization.
Autorzy :
Hayward D; Sir William Dunn School of Pathology, University of Oxford, South Parks Road, Oxford, England, UK.
Alfonso-Pérez T; Department of Biochemistry, University of Oxford, South Parks Road, Oxford, England, UK.
Cundell MJ; Department of Biochemistry, University of Oxford, South Parks Road, Oxford, England, UK.
Hopkins M; Department of Biochemistry, University of Oxford, South Parks Road, Oxford, England, UK.
Holder J; Department of Biochemistry, University of Oxford, South Parks Road, Oxford, England, UK.
Bancroft J; Sir William Dunn School of Pathology, University of Oxford, South Parks Road, Oxford, England, UK.
Hutter LH; Department of Biochemistry, University of Oxford, South Parks Road, Oxford, England, UK.
Novak B; Department of Biochemistry, University of Oxford, South Parks Road, Oxford, England, UK.
Barr FA; Department of Biochemistry, University of Oxford, South Parks Road, Oxford, England, UK .
Gruneberg U; Sir William Dunn School of Pathology, University of Oxford, South Parks Road, Oxford, England, UK .
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Źródło :
The Journal of cell biology [J Cell Biol] 2019 Apr 01; Vol. 218 (4), pp. 1182-1199. Date of Electronic Publication: 2019 Jan 23.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Video-Audio Media
MeSH Terms :
M Phase Cell Cycle Checkpoints*
CDC2 Protein Kinase/*metabolism
Cell Cycle Proteins/*metabolism
Cell Nucleus/*enzymology
Cyclin B1/*metabolism
Kinetochores/*enzymology
Protein-Serine-Threonine Kinases/*metabolism
Protein-Tyrosine Kinases/*metabolism
Aurora Kinase B/genetics ; Aurora Kinase B/metabolism ; CDC2 Protein Kinase/genetics ; Cell Cycle Proteins/genetics ; Cell Nucleus/genetics ; Cyclin B1/genetics ; HEK293 Cells ; HeLa Cells ; Humans ; Phosphorylation ; Protein Phosphatase 2/genetics ; Protein Phosphatase 2/metabolism ; Protein-Serine-Threonine Kinases/genetics ; Protein-Tyrosine Kinases/genetics ; Retinal Pigment Epithelium/enzymology ; Signal Transduction ; Time Factors
Czasopismo naukowe
Tytuł :
MAD1-dependent recruitment of CDK1-CCNB1 to kinetochores promotes spindle checkpoint signaling.
Autorzy :
Alfonso-Pérez T; Department of Biochemistry, University of Oxford, Oxford, UK.
Hayward D; Sir William Dunn School of Pathology, University of Oxford, Oxford, UK.
Holder J; Department of Biochemistry, University of Oxford, Oxford, UK.
Gruneberg U; Sir William Dunn School of Pathology, University of Oxford, Oxford, UK .
Barr FA; Department of Biochemistry, University of Oxford, Oxford, UK .
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Źródło :
The Journal of cell biology [J Cell Biol] 2019 Apr 01; Vol. 218 (4), pp. 1108-1117. Date of Electronic Publication: 2019 Jan 23.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Video-Audio Media
MeSH Terms :
M Phase Cell Cycle Checkpoints*
Signal Transduction*
CDC2 Protein Kinase/*metabolism
Cell Cycle Proteins/*metabolism
Cyclin B1/*metabolism
Kinetochores/*enzymology
Spindle Apparatus/*enzymology
CDC2 Protein Kinase/genetics ; Cell Cycle Proteins/genetics ; Cyclin B1/genetics ; HEK293 Cells ; HeLa Cells ; Humans ; Protein Binding ; Protein-Serine-Threonine Kinases/genetics ; Protein-Serine-Threonine Kinases/metabolism ; Protein-Tyrosine Kinases/genetics ; Protein-Tyrosine Kinases/metabolism ; Spindle Apparatus/genetics ; Time Factors
Czasopismo naukowe
Tytuł :
The subcellular location of cyclin B1 and CDC25 associated with the formation of polyploid giant cancer cells and their clinicopathological significance.
Autorzy :
Fei F; Nankai University School of Medicine, Nankai University, Tianjin, 300071, China.; Departments of Pathology, Tianjin Union Medical Center, Tianjin, 300121, China.
Qu J; Nankai University School of Medicine, Nankai University, Tianjin, 300071, China.; Departments of Pathology, Tianjin Union Medical Center, Tianjin, 300121, China.
Liu K; Tianjin Medical University, Tianjin, 300070, China.
Li C; Nankai University School of Medicine, Nankai University, Tianjin, 300071, China.; Departments of Pathology, Tianjin Union Medical Center, Tianjin, 300121, China.
Wang X; Departments of Pathology, Tianjin Union Medical Center, Tianjin, 300121, China.; Graduate School, Tianjin University of Traditional Chinese Medicine, Tianjin, China.
Li Y; Departments of Colorectal Surgery, Tianjin Union Medical Center, Tianjin, 300121, China.
Zhang S; Departments of Pathology, Tianjin Union Medical Center, Tianjin, 300121, China. .
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Źródło :
Laboratory investigation; a journal of technical methods and pathology [Lab Invest] 2019 Apr; Vol. 99 (4), pp. 483-498. Date of Electronic Publication: 2018 Nov 28.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Breast Neoplasms*/chemistry
Breast Neoplasms*/metabolism
Breast Neoplasms*/pathology
Breast Neoplasms*/physiopathology
Ovarian Neoplasms*/chemistry
Ovarian Neoplasms*/metabolism
Ovarian Neoplasms*/pathology
Ovarian Neoplasms*/physiopathology
Cell Cycle Proteins/*metabolism
Cyclin B1/*metabolism
cdc25 Phosphatases/*metabolism
Cell Cycle Proteins/analysis ; Cell Line, Tumor ; Cyclin B1/analysis ; Female ; Humans ; Intracellular Space/metabolism ; Neoplastic Processes ; Polyploidy ; cdc25 Phosphatases/analysis
Czasopismo naukowe
Tytuł :
PKMYT1 is associated with prostate cancer malignancy and may serve as a therapeutic target.
Autorzy :
Wang J; Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300211, China.
Wang L; Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300211, China.
Chen S; Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300211, China.
Peng H; Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300211, China.
Xiao L; Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300211, China.
E Du; Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300211, China.
Liu Y; Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300211, China.
Lin D; Vancouver Prostate Centre, Department of Urologic Sciences, Faculty of Medicine, University of British Columbia, Vancouver, BC V6H 3Z6, Canada; Department of Experimental Therapeutics, BC Cancer Agency, Vancouver, BC V5Z 1L3, Canada.
Wang Y; Vancouver Prostate Centre, Department of Urologic Sciences, Faculty of Medicine, University of British Columbia, Vancouver, BC V6H 3Z6, Canada; Department of Experimental Therapeutics, BC Cancer Agency, Vancouver, BC V5Z 1L3, Canada.
Xu Y; Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300211, China. Electronic address: .
Yang K; Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300211, China. Electronic address: .
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Źródło :
Gene [Gene] 2020 Jun 20; Vol. 744, pp. 144608. Date of Electronic Publication: 2020 Mar 29.
Typ publikacji :
Journal Article
MeSH Terms :
Prostatic Neoplasms/*enzymology
Cell Cycle ; Cell Line, Tumor ; Cell Proliferation ; Cyclin B1/genetics ; Cyclin B1/metabolism ; Cyclin E/genetics ; Cyclin E/metabolism ; Humans ; Male ; Membrane Proteins/antagonists & inhibitors ; Membrane Proteins/metabolism ; Oncogene Proteins/genetics ; Oncogene Proteins/metabolism ; Oxazines/therapeutic use ; Prognosis ; Prostatic Neoplasms/drug therapy ; Prostatic Neoplasms/metabolism ; Prostatic Neoplasms/pathology ; Protein Kinase Inhibitors/therapeutic use ; Protein-Serine-Threonine Kinases/antagonists & inhibitors ; Protein-Serine-Threonine Kinases/metabolism ; Protein-Tyrosine Kinases/antagonists & inhibitors ; Protein-Tyrosine Kinases/metabolism ; Pyridines/therapeutic use
Czasopismo naukowe
Tytuł :
Acetylation of MORC2 by NAT10 regulates cell-cycle checkpoint control and resistance to DNA-damaging chemotherapy and radiotherapy in breast cancer.
Autorzy :
Liu HY; Fudan University Shanghai Cancer Center and Shanghai Key Laboratory of Medical Epigenetics, Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China.
Liu YY; Fudan University Shanghai Cancer Center and Shanghai Key Laboratory of Medical Epigenetics, Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China.; Cancer Institute, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Breast Surgery, Shanghai Medical College, Fudan University, Shanghai 200032, China.
Yang F; Fudan University Shanghai Cancer Center and Shanghai Key Laboratory of Medical Epigenetics, Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China.; Cancer Institute, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Breast Surgery, Shanghai Medical College, Fudan University, Shanghai 200032, China.
Zhang L; Fudan University Shanghai Cancer Center and Shanghai Key Laboratory of Medical Epigenetics, Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China.; Cancer Institute, Shanghai Medical College, Fudan University, Shanghai 200032, China.
Zhang FL; Fudan University Shanghai Cancer Center and Shanghai Key Laboratory of Medical Epigenetics, Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China.; Cancer Institute, Shanghai Medical College, Fudan University, Shanghai 200032, China.
Hu X; Fudan University Shanghai Cancer Center and Shanghai Key Laboratory of Medical Epigenetics, Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China.; Cancer Institute, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Breast Surgery, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Shanghai Key Laboratory of Breast Cancer, Shanghai Medical College, Fudan University, Shanghai 200032, China.
Shao ZM; Fudan University Shanghai Cancer Center and Shanghai Key Laboratory of Medical Epigenetics, Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China.; Cancer Institute, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Breast Surgery, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Shanghai Key Laboratory of Breast Cancer, Shanghai Medical College, Fudan University, Shanghai 200032, China.
Li DQ; Fudan University Shanghai Cancer Center and Shanghai Key Laboratory of Medical Epigenetics, Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China.; Cancer Institute, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Department of Breast Surgery, Shanghai Medical College, Fudan University, Shanghai 200032, China.; Shanghai Key Laboratory of Breast Cancer, Shanghai Medical College, Fudan University, Shanghai 200032, China.; International Co-laboratory of Medical Epigenetics and Metabolism, Ministry of Science and Technology, Shanghai 200032, China.
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Źródło :
Nucleic acids research [Nucleic Acids Res] 2020 Apr 17; Vol. 48 (7), pp. 3638-3656.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
DNA Damage*
G2 Phase Cell Cycle Checkpoints*
Breast Neoplasms/*enzymology
N-Terminal Acetyltransferases/*metabolism
Transcription Factors/*metabolism
Acetylation ; Antineoplastic Agents ; Breast Neoplasms/genetics ; Breast Neoplasms/metabolism ; CDC2 Protein Kinase/genetics ; CDC2 Protein Kinase/metabolism ; Cell Line, Tumor ; Cyclin B1/genetics ; Cyclin B1/metabolism ; Drug Resistance, Neoplasm ; Gene Expression Regulation, Neoplastic ; Histones/metabolism ; Humans ; Lysine/metabolism ; Radiation, Ionizing ; Sirtuin 2/metabolism ; Transcription Factors/chemistry
Czasopismo naukowe
Tytuł :
Mitotic CDK Promotes Replisome Disassembly, Fork Breakage, and Complex DNA Rearrangements.
Autorzy :
Deng L; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA 02215, USA; Department of Cell Biology, Harvard Medical School, Blavatnik Institute, Boston, MA 02115, USA; Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Blavatnik Institute, Boston, MA 02115, USA.
Wu RA; Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Blavatnik Institute, Boston, MA 02115, USA.
Sonneville R; MRC Protein Phosphorylation and Ubiquitylation Unit, Sir James Black Centre, School of Life Sciences, University of Dundee, Dow Street, Dundee DD1 5EH, UK.
Kochenova OV; Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Blavatnik Institute, Boston, MA 02115, USA.
Labib K; MRC Protein Phosphorylation and Ubiquitylation Unit, Sir James Black Centre, School of Life Sciences, University of Dundee, Dow Street, Dundee DD1 5EH, UK.
Pellman D; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA 02215, USA; Department of Cell Biology, Harvard Medical School, Blavatnik Institute, Boston, MA 02115, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA; Howard Hughes Medical Institute, Boston, MA 02115, USA. Electronic address: .
Walter JC; Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Blavatnik Institute, Boston, MA 02115, USA; Howard Hughes Medical Institute, Boston, MA 02115, USA. Electronic address: .
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Źródło :
Molecular cell [Mol Cell] 2019 Mar 07; Vol. 73 (5), pp. 915-929.e6.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
DNA Damage*
DNA Replication*
Gene Rearrangement*
Mitosis*
Cell Cycle Proteins/*metabolism
Cyclin B1/*metabolism
DNA/*biosynthesis
Protein Kinases/*metabolism
Xenopus Proteins/*metabolism
Adenosine Triphosphatases/genetics ; Adenosine Triphosphatases/metabolism ; Animals ; Caenorhabditis elegans/genetics ; Caenorhabditis elegans/metabolism ; Caenorhabditis elegans Proteins/genetics ; Caenorhabditis elegans Proteins/metabolism ; Cell Cycle Proteins/genetics ; Cyclin B1/genetics ; DNA/genetics ; DNA Repair ; DNA-Directed DNA Polymerase/genetics ; DNA-Directed DNA Polymerase/metabolism ; Minichromosome Maintenance Proteins/genetics ; Minichromosome Maintenance Proteins/metabolism ; Nuclear Proteins/genetics ; Nuclear Proteins/metabolism ; Protein Kinases/genetics ; Protein-Serine-Threonine Kinases/genetics ; Protein-Serine-Threonine Kinases/metabolism ; Ubiquitin-Protein Ligases/genetics ; Ubiquitin-Protein Ligases/metabolism ; Ubiquitination ; Xenopus Proteins/genetics ; Xenopus laevis/genetics ; Xenopus laevis/metabolism
Czasopismo naukowe
Tytuł :
Identification of putative drugs for gastric adenocarcinoma utilizing differentially expressed genes and connectivity map.
Autorzy :
Chen ZX; Department of Medical Oncology, The Second Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Zou XP; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Yan HQ; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Zhang R; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Pang JS; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Qin XG; Department of Gastrointestinal Surgery, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
He RQ; Department of Medical Oncology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Ma J; Department of Medical Oncology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Feng ZB; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Chen G; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Gan TQ; Department of Medical Oncology, The Second Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2019 Feb; Vol. 19 (2), pp. 1004-1015. Date of Electronic Publication: 2018 Dec 13.
Typ publikacji :
Journal Article
MeSH Terms :
Gene Expression Regulation, Neoplastic*
Adenocarcinoma/*genetics
Antineoplastic Agents/*therapeutic use
Cell Cycle Proteins/*genetics
Cyclin B1/*genetics
Nuclear Proteins/*genetics
Stomach Neoplasms/*genetics
Tumor Suppressor Protein p53/*genetics
Adenocarcinoma/drug therapy ; Adenocarcinoma/metabolism ; Adenocarcinoma/pathology ; Antineoplastic Agents/classification ; Cell Cycle Proteins/metabolism ; Cell Line, Tumor ; Computational Biology/methods ; Cyclin B1/metabolism ; Databases, Genetic ; Gene Expression Profiling ; Gene Ontology ; Gene Regulatory Networks ; Humans ; Molecular Sequence Annotation ; Nuclear Proteins/metabolism ; Pharmacogenetics/methods ; Protein Interaction Mapping ; Signal Transduction ; Stomach Neoplasms/drug therapy ; Stomach Neoplasms/metabolism ; Stomach Neoplasms/pathology ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
Tytuł :
Identification of genes associated with cancer progression and prognosis in lung adenocarcinoma: Analyses based on microarray from Oncomine and The Cancer Genome Atlas databases.
Autorzy :
Liu W; Department of Gastroenterology in the First Affiliated Hospital, Zhengzhou University, Zhengzhou, China.
Ouyang S; Department of Respiratory and Sleep Medicine in the First Affiliated Hospital, Zhengzhou University, Zhengzhou, China.
Zhou Z; Department of Radiology in the First Affiliated Hospital, Zhengzhou University, Zhengzhou, China.
Wang M; Department of Radiology in the First Affiliated Hospital, Zhengzhou University, Zhengzhou, China.
Wang T; Department of Medical Examination in the First Affiliated Hospital, Zhengzhou University, Zhengzhou, China.
Qi Y; Department of Thoracic Surgery in the First Affiliated Hospital, Zhengzhou University, Zhengzhou, China.
Zhao C; Department of Respiratory and Sleep Medicine in the First Affiliated Hospital, Zhengzhou University, Zhengzhou, China.
Chen K; Department of Pathology in the First Affiliated Hospital, Zhengzhou University, Zhengzhou, China.
Dai L; Department of Respiratory and Sleep Medicine in the First Affiliated Hospital, Zhengzhou University, Zhengzhou, China.; Department of Tumor Research in the Institute of Medical and Pharmaceutical Sciences, Zhengzhou University, Zhengzhou, China.
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Źródło :
Molecular genetics & genomic medicine [Mol Genet Genomic Med] 2019 Feb; Vol. 7 (2), pp. e00528. Date of Electronic Publication: 2018 Dec 16.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Gene Expression Regulation, Neoplastic*
Adenocarcinoma/*genetics
Biomarkers, Tumor/*genetics
Cyclin B1/*genetics
Lung Neoplasms/*genetics
Receptor for Advanced Glycation End Products/*genetics
Adenocarcinoma/pathology ; Biomarkers, Tumor/metabolism ; Cyclin B1/metabolism ; Databases, Genetic ; Humans ; Lung/metabolism ; Lung Neoplasms/pathology ; Receptor for Advanced Glycation End Products/metabolism
Czasopismo naukowe
Tytuł :
CYCLIN-B1/2 and -D1 act in opposition to coordinate cortical progenitor self-renewal and lineage commitment.
Autorzy :
Hagey DW; Department of Cell and Molecular Biology, Karolinska Institutet, Solnavägen 9, SE-171 65, Stockholm, Sweden. .
Topcic D; Department of Cell and Molecular Biology, Karolinska Institutet, Solnavägen 9, SE-171 65, Stockholm, Sweden.
Kee N; Department of Cell and Molecular Biology, Karolinska Institutet, Solnavägen 9, SE-171 65, Stockholm, Sweden.
Reynaud F; Department of Cell and Molecular Biology, Karolinska Institutet, Solnavägen 9, SE-171 65, Stockholm, Sweden.
Bergsland M; Department of Cell and Molecular Biology, Karolinska Institutet, Solnavägen 9, SE-171 65, Stockholm, Sweden.
Perlmann T; Department of Cell and Molecular Biology, Karolinska Institutet, Solnavägen 9, SE-171 65, Stockholm, Sweden.
Muhr J; Department of Cell and Molecular Biology, Karolinska Institutet, Solnavägen 9, SE-171 65, Stockholm, Sweden. .
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Źródło :
Nature communications [Nat Commun] 2020 Jun 09; Vol. 11 (1), pp. 2898. Date of Electronic Publication: 2020 Jun 09.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Gene Expression Regulation, Developmental*
Cyclin B1/*physiology
Cyclin B2/*physiology
Cyclin D1/*physiology
Animals ; CDC2 Protein Kinase/physiology ; Cell Cycle ; Cell Differentiation ; Cell Lineage ; Cell Separation ; Cerebral Cortex/embryology ; Female ; Flow Cytometry ; Mice ; Mice, Inbred C57BL ; Neuroglia/metabolism ; Sequence Analysis, RNA ; Signal Transduction ; Stem Cells/cytology
Czasopismo naukowe
Tytuł :
Bioinformatical Analysis of Gene Expression Omnibus Database Associates TAF7/CCNB1, TAF7/CCNA2, and GTF2E2/CDC20 Pathways with Glioblastoma Development and Prognosis.
Autorzy :
Yang L; Department of Neurosurgery, The First Affiliated Hospital of Hainan Medical University, Haikou, China.
Zeng W; Department of General Medicine, The First Affiliated Hospital of Hainan Medical University, Haikou, China.
Sun H; Department of Medical Oncology, The First Affiliated Hospital of Hainan Medical University, Haikou, China.
Huang F; Department of Medical Oncology, The First Affiliated Hospital of Hainan Medical University, Haikou, China.
Yang C; Department of Medical Oncology, The First Affiliated Hospital of Hainan Medical University, Haikou, China.
Cai X; Department of Medical Oncology, The First Affiliated Hospital of Hainan Medical University, Haikou, China.
Lu Y; Department of Medical Oncology, The First Affiliated Hospital of Hainan Medical University, Haikou, China.
Zeng J; Department of Medical Oncology, The First Affiliated Hospital of Hainan Medical University, Haikou, China.
Yang K; Department of Neurosurgery, The First Affiliated Hospital of Hainan Medical University, Haikou, China. Electronic address: .
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Źródło :
World neurosurgery [World Neurosurg] 2020 Jun; Vol. 138, pp. e492-e514. Date of Electronic Publication: 2020 Mar 05.
Typ publikacji :
Journal Article
MeSH Terms :
Gene Expression Regulation, Neoplastic*
Brain Neoplasms/*genetics
Glioblastoma/*genetics
Brain Neoplasms/mortality ; Brain Neoplasms/pathology ; Cdc20 Proteins/genetics ; Computational Biology ; Cyclin A2/genetics ; Cyclin B1/genetics ; Databases, Genetic ; Gene Expression Profiling ; Glioblastoma/mortality ; Glioblastoma/pathology ; Humans ; Prognosis ; Survival Rate ; TATA-Binding Protein Associated Factors/genetics ; Transcription Factor TFIID/genetics
Czasopismo naukowe
Tytuł :
Paradoxical mitotic exit induced by a small molecule inhibitor of APC/C .
Autorzy :
Richeson KV; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Bodrug T; Department of Biochemistry and Biophysics, University of North Carolina School of Medicine, Chapel Hill, NC, USA.
Sackton KL; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Yamaguchi M; Department of Structural Biology, St. Jude Children's Research Hospital, Memphis, TN, USA.
Paulo JA; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Gygi SP; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Schulman BA; Department of Structural Biology, St. Jude Children's Research Hospital, Memphis, TN, USA.; Department of Molecular Machines and Signaling, Max Planck Institute of Biochemistry, Martinsried, Germany.
Brown NG; Department of Pharmacology and Lineberger Comprehensive Cancer Center, University of North Carolina School of Medicine, Chapel Hill, NC, USA.
King RW; Department of Cell Biology, Harvard Medical School, Boston, MA, USA. .
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Źródło :
Nature chemical biology [Nat Chem Biol] 2020 May; Vol. 16 (5), pp. 546-555. Date of Electronic Publication: 2020 Mar 09.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
Anaphase-Promoting Complex-Cyclosome/*antagonists & inhibitors
Carbamates/*pharmacology
Cdc20 Proteins/*antagonists & inhibitors
Diamines/*pharmacology
Mitosis/*drug effects
Adaptor Proteins, Signal Transducing/metabolism ; Anaphase-Promoting Complex-Cyclosome/metabolism ; Binding Sites ; Cell Cycle Proteins/metabolism ; Cyclin B1/metabolism ; HCT116 Cells ; HeLa Cells ; Humans ; Nocodazole/pharmacology ; Nuclear Proteins/metabolism ; Spindle Apparatus/drug effects ; Spindle Apparatus/metabolism ; Telomerase/genetics ; Telomerase/metabolism ; Time-Lapse Imaging ; Ubiquitination
Czasopismo naukowe
Tytuł :
Ribosome binding protein GCN1 regulates the cell cycle and cell proliferation and is essential for the embryonic development of mice.
Autorzy :
Yamazaki H; Department of Stress Response Science, Center for Advanced Medical Research, Hirosaki University, Hirosaki, Japan.
Kasai S; Department of Stress Response Science, Center for Advanced Medical Research, Hirosaki University, Hirosaki, Japan.
Mimura J; Department of Stress Response Science, Center for Advanced Medical Research, Hirosaki University, Hirosaki, Japan.
Ye P; Department of Stress Response Science, Center for Advanced Medical Research, Hirosaki University, Hirosaki, Japan.
Inose-Maruyama A; Department of Stress Response Science, Center for Advanced Medical Research, Hirosaki University, Hirosaki, Japan.
Tanji K; Department of Neuropathology, Institute of Brain Science Graduate School of Medicine, Hirosaki University, Hirosaki, Japan.
Wakabayashi K; Department of Neuropathology, Institute of Brain Science Graduate School of Medicine, Hirosaki University, Hirosaki, Japan.
Mizuno S; Transborder Medical Research Center and Laboratory Animal Resource Center, University of Tsukuba, Tsukuba, Japan.
Sugiyama F; Transborder Medical Research Center and Laboratory Animal Resource Center, University of Tsukuba, Tsukuba, Japan.
Takahashi S; Transborder Medical Research Center and Laboratory Animal Resource Center, University of Tsukuba, Tsukuba, Japan.
Sato T; Department of Stress Response Science, Center for Advanced Medical Research, Hirosaki University, Hirosaki, Japan.; Department of Chemistry and Biological Sciences, Faculty of Science and Engineering, Iwate University, Morioka, Japan.
Ozaki T; Department of Chemistry and Biological Sciences, Faculty of Science and Engineering, Iwate University, Morioka, Japan.
Cavener DR; Department of Biology, Center for Cellular Dynamics and the Huck Institute of the Life Sciences, Pennsylvania State University, University Park, Pennsylvania, United States of America.
Yamamoto M; Department of Medical Biochemistry, Tohoku University Graduate School of Medicine, Sendai, Japan.
Itoh K; Department of Stress Response Science, Center for Advanced Medical Research, Hirosaki University, Hirosaki, Japan.
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Źródło :
PLoS genetics [PLoS Genet] 2020 Apr 23; Vol. 16 (4), pp. e1008693. Date of Electronic Publication: 2020 Apr 23 (Print Publication: 2020).
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Cycle*
Cell Proliferation*
Fetal Development*
Stress, Physiological*
RNA-Binding Proteins/*metabolism
Trans-Activators/*metabolism
Animals ; CDC2 Protein Kinase/metabolism ; Cells, Cultured ; Cyclin B1/metabolism ; Fibroblasts/metabolism ; HeLa Cells ; Humans ; Mice ; Mice, Inbred C57BL ; Protein-Serine-Threonine Kinases/metabolism ; RNA-Binding Proteins/genetics ; Trans-Activators/genetics
Czasopismo naukowe

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