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Tytuł:
Lysyl oxidase inhibits BMP9-induced osteoblastic differentiation through reducing Wnt/β-catenin via HIF-1a repression in 3T3-L1 cells.
Autorzy:
Zhang J; Department of Pharmacology, School of Pharmacy, Chongqing Medical University, No. 1 Yixueyuan Road, Yuzhong, Chongqing, 400016, People's Republic of China.; Key Laboratory of Biochemistry and Molecular Pharmacology of Chongqing, Chongqing Medical University, Chongqing, 400016, People's Republic of China.
Ye F; Department of Pharmacology, School of Pharmacy, Chongqing Medical University, No. 1 Yixueyuan Road, Yuzhong, Chongqing, 400016, People's Republic of China.; Key Laboratory of Biochemistry and Molecular Pharmacology of Chongqing, Chongqing Medical University, Chongqing, 400016, People's Republic of China.
Ye A; Department of Pharmacology, School of Pharmacy, Chongqing Medical University, No. 1 Yixueyuan Road, Yuzhong, Chongqing, 400016, People's Republic of China.; Key Laboratory of Biochemistry and Molecular Pharmacology of Chongqing, Chongqing Medical University, Chongqing, 400016, People's Republic of China.
He B; Department of Pharmacology, School of Pharmacy, Chongqing Medical University, No. 1 Yixueyuan Road, Yuzhong, Chongqing, 400016, People's Republic of China. .; Key Laboratory of Biochemistry and Molecular Pharmacology of Chongqing, Chongqing Medical University, Chongqing, 400016, People's Republic of China. .
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Źródło:
Journal of orthopaedic surgery and research [J Orthop Surg Res] 2023 Nov 29; Vol. 18 (1), pp. 911. Date of Electronic Publication: 2023 Nov 29.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/genetics
Growth Differentiation Factor 2*/genetics
Growth Differentiation Factor 2*/metabolism
Animals ; Mice ; 3T3-L1 Cells ; Cell Differentiation/genetics ; Osteogenesis/genetics ; Protein-Lysine 6-Oxidase/antagonists & inhibitors ; Protein-Lysine 6-Oxidase/genetics ; Protein-Lysine 6-Oxidase/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Cyclin-dependent kinase 5 negatively regulates antiviral immune response by disrupting myeloid differentiation primary response protein 88 self-association.
Autorzy:
Ren JP; State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.; University of Chinese Academy of Sciences, Beijing, China.; Beijing Institute for Stem Cell and Regenerative Medicine, Beijing, China.
Cong HL; Chinese Academy of Inspection and Quarantine, Beijing, China.
Gao LJ; State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.; University of Chinese Academy of Sciences, Beijing, China.; Beijing Institute for Stem Cell and Regenerative Medicine, Beijing, China.
Jiang DF; State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.; University of Chinese Academy of Sciences, Beijing, China.; Beijing Institute for Stem Cell and Regenerative Medicine, Beijing, China.
Li XT; State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.; University of Chinese Academy of Sciences, Beijing, China.; Beijing Institute for Stem Cell and Regenerative Medicine, Beijing, China.
Wang Y; State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.; University of Chinese Academy of Sciences, Beijing, China.; Beijing Institute for Stem Cell and Regenerative Medicine, Beijing, China.
Wang JQ; State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.; Peninsular Cancer Center, Binzhou Medical University, Yantai, China.
Tang TS; State Key Laboratory of Membrane Biology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.; University of Chinese Academy of Sciences, Beijing, China.; Beijing Institute for Stem Cell and Regenerative Medicine, Beijing, China.
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Źródło:
Virulence [Virulence] 2023 Dec; Vol. 14 (1), pp. 2223394.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Myeloid Differentiation Factor 88*/genetics
Virus Diseases*/immunology
Adaptor Proteins, Signal Transducing/metabolism ; Cell Differentiation ; Cyclin-Dependent Kinase 5/metabolism ; Immunity, Innate ; Toll-Like Receptors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
BMP-9 Improves the Osteogenic Differentiation Ability over BMP-2 through p53 Signaling In Vitro in Human Periosteum-Derived Cells.
Autorzy:
Park JH; Department of Nutritional Science, University of Michigan School of Public Health, Ann Arbor, MI 48109, USA.
Koh EB; Department of Oral and Maxillofacial Surgery, Gyeongsang National University School of Medicine, Gyeongsang National University Hospital, Institute of Medical Sciences, Gyeongsang National University, Jinju 52727, Republic of Korea.; Department of Convergence Medical Science, Gyeongsang National University, Jinju 52828, Republic of Korea.
Seo YJ; Department of Oral and Maxillofacial Surgery, Gyeongsang National University School of Medicine, Gyeongsang National University Hospital, Institute of Medical Sciences, Gyeongsang National University, Jinju 52727, Republic of Korea.; Department of Convergence Medical Science, Gyeongsang National University, Jinju 52828, Republic of Korea.
Oh HS; Department of Oral and Maxillofacial Surgery, Gyeongsang National University School of Medicine, Gyeongsang National University Hospital, Institute of Medical Sciences, Gyeongsang National University, Jinju 52727, Republic of Korea.; Department of Convergence Medical Science, Gyeongsang National University, Jinju 52828, Republic of Korea.
Byun JH; Department of Oral and Maxillofacial Surgery, Gyeongsang National University School of Medicine, Gyeongsang National University Hospital, Institute of Medical Sciences, Gyeongsang National University, Jinju 52727, Republic of Korea.; Department of Convergence Medical Science, Gyeongsang National University, Jinju 52828, Republic of Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Oct 17; Vol. 24 (20). Date of Electronic Publication: 2023 Oct 17.
Typ publikacji:
Journal Article
MeSH Terms:
Growth Differentiation Factor 2*/metabolism
Osteogenesis*
Humans ; Bone Morphogenetic Protein 2/pharmacology ; Bone Morphogenetic Protein 2/metabolism ; Bone Morphogenetic Proteins/metabolism ; Cell Differentiation ; Osteoblasts/metabolism ; Periosteum/metabolism ; Signal Transduction ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
GDF15 Promotes the Osteogenic Cell Fate of Periodontal Ligament Fibroblasts, thus Affecting Their Mechanobiological Response.
Autorzy:
Lösch L; Department of Orthodontics, University Hospital Jena, Leutragraben 3, 07743 Jena, Germany.
Stemmler A; Department of Orthodontics, University Hospital Jena, Leutragraben 3, 07743 Jena, Germany.
Fischer A; Department of Orthodontics, University Hospital Jena, Leutragraben 3, 07743 Jena, Germany.
Steinmetz J; Department of Orthodontics, University Hospital Jena, Leutragraben 3, 07743 Jena, Germany.
Schuldt L; Department of Orthodontics, University Hospital Jena, Leutragraben 3, 07743 Jena, Germany.
Hennig CL; Department of Orthodontics, University Hospital Jena, Leutragraben 3, 07743 Jena, Germany.
Symmank J; Department of Orthodontics, University Hospital Jena, Leutragraben 3, 07743 Jena, Germany.
Jacobs C; Department of Orthodontics, University Hospital Jena, Leutragraben 3, 07743 Jena, Germany.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Jun 11; Vol. 24 (12). Date of Electronic Publication: 2023 Jun 11.
Typ publikacji:
Journal Article
MeSH Terms:
Periodontal Ligament*
Growth Differentiation Factor 15*/genetics
Growth Differentiation Factor 15*/metabolism
Humans ; Cells, Cultured ; Cell Differentiation ; Fibroblasts/metabolism ; Inflammation/metabolism ; Tooth Movement Techniques
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
SESN1 functions as a new tumor suppressor gene via Toll-like receptor signaling pathway in neuroblastoma.
Autorzy:
Hua Z; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.; Liaoning Key Laboratory of Research and Application of Animal Models for Environmental and Metabolic Diseases, Medical Research Center, Shengjing Hospital of China Medical University, Shenyang, China.
Chen B; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.; Liaoning Key Laboratory of Research and Application of Animal Models for Environmental and Metabolic Diseases, Medical Research Center, Shengjing Hospital of China Medical University, Shenyang, China.
Gong B; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.; Liaoning Key Laboratory of Research and Application of Animal Models for Environmental and Metabolic Diseases, Medical Research Center, Shengjing Hospital of China Medical University, Shenyang, China.
Lin M; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.; Liaoning Key Laboratory of Research and Application of Animal Models for Environmental and Metabolic Diseases, Medical Research Center, Shengjing Hospital of China Medical University, Shenyang, China.
Ma Y; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.; Liaoning Key Laboratory of Research and Application of Animal Models for Environmental and Metabolic Diseases, Medical Research Center, Shengjing Hospital of China Medical University, Shenyang, China.
Li Z; Department of Pediatrics, Shengjing Hospital of China Medical University, Shenyang, China.; Liaoning Key Laboratory of Research and Application of Animal Models for Environmental and Metabolic Diseases, Medical Research Center, Shengjing Hospital of China Medical University, Shenyang, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14664.
Typ publikacji:
Journal Article
MeSH Terms:
Myeloid Differentiation Factor 88*/genetics
Myeloid Differentiation Factor 88*/metabolism
Neuroblastoma*/pathology
Child ; Humans ; Animals ; Mice ; N-Myc Proto-Oncogene Protein/genetics ; N-Myc Proto-Oncogene Protein/metabolism ; Transcription Factors/genetics ; Signal Transduction/genetics ; Genes, Tumor Suppressor ; Toll-Like Receptors/genetics ; Toll-Like Receptors/metabolism ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Tumor Microenvironment ; Sestrins/genetics ; Sestrins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Targeted Disruption of Lats1 and Lats2 in Mice Impairs Testis Development and Alters Somatic Cell Fate.
Autorzy:
Abou Nader N; Centre de Recherche en Reproduction et Fertilité, Faculté de Médecine Vétérinaire, Université de Montréal, Saint-Hyacinthe, QC J2S 7C6, Canada.
Ménard A; Centre de Recherche en Reproduction et Fertilité, Faculté de Médecine Vétérinaire, Université de Montréal, Saint-Hyacinthe, QC J2S 7C6, Canada.
Levasseur A; Centre de Recherche en Reproduction et Fertilité, Faculté de Médecine Vétérinaire, Université de Montréal, Saint-Hyacinthe, QC J2S 7C6, Canada.
St-Jean G; Centre de Recherche en Reproduction et Fertilité, Faculté de Médecine Vétérinaire, Université de Montréal, Saint-Hyacinthe, QC J2S 7C6, Canada.
Boerboom D; Centre de Recherche en Reproduction et Fertilité, Faculté de Médecine Vétérinaire, Université de Montréal, Saint-Hyacinthe, QC J2S 7C6, Canada.
Zamberlam G; Centre de Recherche en Reproduction et Fertilité, Faculté de Médecine Vétérinaire, Université de Montréal, Saint-Hyacinthe, QC J2S 7C6, Canada.
Boyer A; Centre de Recherche en Reproduction et Fertilité, Faculté de Médecine Vétérinaire, Université de Montréal, Saint-Hyacinthe, QC J2S 7C6, Canada.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Nov 05; Vol. 23 (21). Date of Electronic Publication: 2022 Nov 05.
Typ publikacji:
Journal Article
MeSH Terms:
Adaptor Proteins, Signal Transducing*/metabolism
Sex Differentiation*
Animals ; Male ; Mice ; Cell Cycle Proteins/metabolism ; Cell Differentiation/genetics ; Phosphoproteins/metabolism ; Protein Serine-Threonine Kinases/genetics ; Testis/metabolism ; YAP-Signaling Proteins
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
HDAC6 Enhances Endoglin Expression through Deacetylation of Transcription Factor SP1, Potentiating BMP9-Induced Angiogenesis.
Autorzy:
Sun C; Shanghai Key Laboratory of Bioactive Small Molecules, Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University Shanghai Medical College, Shanghai 200032, China.; State Key Laboratory of Quality Research in Chinese Medicine and School of Pharmacy, Macau University of Science and Technology, Avenida WaiLong, Taipa, Macau 999078, China.
Xie K; Shanghai Key Laboratory of Bioactive Small Molecules, Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University Shanghai Medical College, Shanghai 200032, China.
Yang L; Shanghai Key Laboratory of Bioactive Small Molecules, Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University Shanghai Medical College, Shanghai 200032, China.
Cai S; Shanghai Key Laboratory of Bioactive Small Molecules, Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University Shanghai Medical College, Shanghai 200032, China.
Wang M; Shanghai Key Laboratory of Bioactive Small Molecules, Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University Shanghai Medical College, Shanghai 200032, China.
Zhu Y; State Key Laboratory of Quality Research in Chinese Medicine and School of Pharmacy, Macau University of Science and Technology, Avenida WaiLong, Taipa, Macau 999078, China.; Department of Pharmacology, School of Pharmacy, Fudan University, Shanghai 200433, China.
Tao B; Shanghai Key Laboratory of Bioactive Small Molecules, Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University Shanghai Medical College, Shanghai 200032, China.
Zhu Y; Shanghai Key Laboratory of Bioactive Small Molecules, Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University Shanghai Medical College, Shanghai 200032, China.
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Źródło:
Cells [Cells] 2024 Mar 11; Vol. 13 (6). Date of Electronic Publication: 2024 Mar 11.
Typ publikacji:
Journal Article
MeSH Terms:
Growth Differentiation Factor 2*/metabolism
Endothelial Cells*/metabolism
Humans ; Histone Deacetylase 6/metabolism ; Endoglin/metabolism ; Phosphorylation ; Angiogenesis ; Proteomics ; Transcription Factors/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
STAP-2 facilitates insulin signaling through binding to CAP/c-Cbl and regulates adipocyte differentiation.
Autorzy:
Sekine Y; Department of Cell Biology, Kyoto Pharmaceutical University, Kyoto, 607-8412, Japan. .
Kikkawa K; Department of Cell Biology, Kyoto Pharmaceutical University, Kyoto, 607-8412, Japan.
Honda S; Department of Cell Biology, Kyoto Pharmaceutical University, Kyoto, 607-8412, Japan.
Sasaki Y; Department of Immunology, Graduate School of Pharmaceutical Sciences, Hokkaido University, Sapporo, 060-0812, Japan.
Kawahara S; Department of Immunology, Graduate School of Pharmaceutical Sciences, Hokkaido University, Sapporo, 060-0812, Japan.
Mizushima A; Department of Immunology, Graduate School of Pharmaceutical Sciences, Hokkaido University, Sapporo, 060-0812, Japan.
Togi S; Division of Genomic Medicine, Department of Advanced Medicine, Medical Research Institute, Kanazawa Medical University, Kahoku, Ishikawa, 920-0293, Japan.
Fujimuro M; Department of Cell Biology, Kyoto Pharmaceutical University, Kyoto, 607-8412, Japan.
Oritani K; Department of Hematology, International University of Health and Welfare, Narita, Chiba, 286-8686, Japan.
Matsuda T; Department of Immunology, Graduate School of Pharmaceutical Sciences, Hokkaido University, Sapporo, 060-0812, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2024 Mar 09; Vol. 14 (1), pp. 5799. Date of Electronic Publication: 2024 Mar 09.
Typ publikacji:
Journal Article
MeSH Terms:
Adaptor Proteins, Signal Transducing*/genetics
Adaptor Proteins, Signal Transducing*/metabolism
Insulin*/metabolism
Cell Differentiation*/genetics
Animals ; Humans ; Mice ; Adipocytes/metabolism ; Fibroblasts/metabolism ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CD163-Mediated Small-Vessel Injury in Alzheimer's Disease: An Exploration from Neuroimaging to Transcriptomics.
Autorzy:
Chen Y; Health Management Center, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.; Department of Neurology, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.; Renji-UNSW CHeBA (Centre for Healthy Brain Ageing of University of New South Wales) Neurocognitive Center, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.
Lu P; Health Management Center, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.; Renji-UNSW CHeBA (Centre for Healthy Brain Ageing of University of New South Wales) Neurocognitive Center, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.
Wu S; School of Biomedical Engineering, Shanghai Jiao Tong University, Shanghai 200030, China.
Yang J; Health Management Center, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.; Department of Neurology, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.; Renji-UNSW CHeBA (Centre for Healthy Brain Ageing of University of New South Wales) Neurocognitive Center, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.
Liu W; Health Management Center, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.
Zhang Z; School of Biomedical Engineering, Shanghai Jiao Tong University, Shanghai 200030, China.
Xu Q; Health Management Center, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.; Department of Neurology, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.; Renji-UNSW CHeBA (Centre for Healthy Brain Ageing of University of New South Wales) Neurocognitive Center, Renji Hospital of Medical School, Shanghai Jiao Tong University, Shanghai 200127, China.
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Corporate Authors:
Alzheimer’s Disease Neuroimaging Initiative
Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Feb 14; Vol. 25 (4). Date of Electronic Publication: 2024 Feb 14.
Typ publikacji:
Journal Article
MeSH Terms:
Alzheimer Disease*/diagnostic imaging
Alzheimer Disease*/genetics
Alzheimer Disease*/psychology
Cognitive Dysfunction*/diagnostic imaging
Cognitive Dysfunction*/genetics
Cognitive Dysfunction*/psychology
Folate Receptor 2*
White Matter*/diagnostic imaging
Antigens, Differentiation, Myelomonocytic*/metabolism
Animals ; Humans ; Mice ; Aldehyde Dehydrogenase, Mitochondrial ; Gene Expression Profiling ; Magnetic Resonance Imaging/methods ; Neuroimaging ; Transcription Factors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Involvement of M1-Activated Macrophages and Perforin/Granulysin Expressing Lymphocytes in IgA Vasculitis Nephritis.
Autorzy:
Laskarin G; Department of Physiology, Immunology and Pathophysiology, Faculty of Medicine, University of Rijeka, 51000 Rijeka, Croatia.; Hospital for Medical Rehabilitation of Hearth and Lung Diseases and Rheumatism 'Thalassotherapia-Opatija', 51410 Opatija, Croatia.
Babarovic E; Department of Pathology, Faculty of Medicine, University of Rijeka, 51000 Rijeka, Croatia.
Kifer N; Division of Rheumatology and Immunology, Department of Paediatrics, School of Medicine, University Hospital Centre Zagreb, University of Zagreb, 10000 Zagreb, Croatia.
Bulimbasic S; Department of Pathology and Cytology, School of Medicine, University Hospital Centre Zagreb, University of Zagreb, 10000 Zagreb, Croatia.
Sestan M; Division of Rheumatology and Immunology, Department of Paediatrics, School of Medicine, University Hospital Centre Zagreb, University of Zagreb, 10000 Zagreb, Croatia.
Held M; Division of Rheumatology and Immunology, Department of Paediatrics, School of Medicine, University Hospital Centre Zagreb, University of Zagreb, 10000 Zagreb, Croatia.
Frkovic M; Division of Rheumatology and Immunology, Department of Paediatrics, School of Medicine, University Hospital Centre Zagreb, University of Zagreb, 10000 Zagreb, Croatia.
Gagro A; Children's Hospital Zagreb, Faculty of Medicine, University of Osijek, 31000 Osijek, Croatia.
Coric M; Department of Pathology and Cytology, School of Medicine, University Hospital Centre Zagreb, University of Zagreb, 10000 Zagreb, Croatia.
Jelusic M; Division of Rheumatology and Immunology, Department of Paediatrics, School of Medicine, University Hospital Centre Zagreb, University of Zagreb, 10000 Zagreb, Croatia.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Feb 13; Vol. 25 (4). Date of Electronic Publication: 2024 Feb 13.
Typ publikacji:
Journal Article
MeSH Terms:
IgA Vasculitis*/complications
Killer Cells, Natural*/immunology
Macrophages*/immunology
Nephritis*/immunology
Perforin*/metabolism
Antigens, Differentiation, T-Lymphocyte*/metabolism
Macrophage Activation*
Child ; Humans ; Arginase/metabolism ; Adolescent ; Male ; Female
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
TGF-β Isoforms and GDF-15 in the Development and Progression of Atherosclerosis.
Autorzy:
Liuizė Abramavičiūtė A; Department of Biochemistry, Medical Academy, Lithuanian University of Health Sciences, LT-50161 Kaunas, Lithuania.
Mongirdienė A; Department of Biochemistry, Medical Academy, Lithuanian University of Health Sciences, LT-50161 Kaunas, Lithuania.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Feb 09; Vol. 25 (4). Date of Electronic Publication: 2024 Feb 09.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Atherosclerosis*/metabolism
Growth Differentiation Factor 15*/metabolism
Transforming Growth Factor beta*/metabolism
Humans ; Inflammation/metabolism ; Macrophages/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A single-centre, real-world study of BTK inhibitors for the initial treatment of MYD88 diffuse large B-cell lymphoma.
Autorzy:
Deng T; Department of Hematology, Chongqing Fifth People's Hospital, Chongqing, PR China.
Zhang S; Department of Hematology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, PR China.
Xiao M; Department of Hematology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, PR China.
Gu J; Department of Hematology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, PR China.
Huang L; Department of Hematology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, PR China.
Zhou X; Department of Hematology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, PR China.
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Źródło:
Cancer medicine [Cancer Med] 2024 Feb; Vol. 13 (4), pp. e7005.
Typ publikacji:
Journal Article
MeSH Terms:
Myeloid Differentiation Factor 88*/genetics
Lymphoma, Large B-Cell, Diffuse*/drug therapy
Lymphoma, Large B-Cell, Diffuse*/genetics
Lymphoma, Large B-Cell, Diffuse*/pathology
Piperidines*
Pyridines*
Humans ; Aged ; Rituximab/therapeutic use ; Disease-Free Survival ; Retrospective Studies ; Vincristine/adverse effects ; Cyclophosphamide/adverse effects ; Doxorubicin/adverse effects ; Antineoplastic Combined Chemotherapy Protocols/adverse effects ; Prednisone/adverse effects ; CD79 Antigens/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
New insights into developmental fate decisions by autoreactive CD4 thymocytes.
Autorzy:
Tai X; Experimental Immunology Branch, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA.
Singer A; Experimental Immunology Branch, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA.
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Źródło:
Clinical and translational medicine [Clin Transl Med] 2024 Feb; Vol. 14 (2), pp. e1590.
Typ publikacji:
Editorial
MeSH Terms:
Thymocytes*/cytology
Thymocytes*/immunology
CD4-Positive T-Lymphocytes*/cytology
CD4-Positive T-Lymphocytes*/immunology
Cell Differentiation*
Animals ; Mice ; Clonal Deletion
Opinia redakcyjna
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
GDF15 is a dynamic biomarker of the integrated stress response in the central nervous system.
Autorzy:
Asundi J; Calico Life Sciences LLC, South San Francisco, California, USA.
Zhang C; Calico Life Sciences LLC, South San Francisco, California, USA.
Donnelly-Roberts D; AbbVie, North Chicago, Illinois, USA.
Solorio JZ; Calico Life Sciences LLC, South San Francisco, California, USA.
Challagundla M; AbbVie Deutschland GmbH & Co. KG, Ludwigshafen, Germany.
Connelly C; Calico Life Sciences LLC, South San Francisco, California, USA.
Boch C; AbbVie Deutschland GmbH & Co. KG, Ludwigshafen, Germany.
Chen J; AbbVie, North Chicago, Illinois, USA.
Richter M; AbbVie Deutschland GmbH & Co. KG, Ludwigshafen, Germany.
Maneshi MM; AbbVie, North Chicago, Illinois, USA.
Swensen AM; AbbVie, North Chicago, Illinois, USA.
Lebon L; Calico Life Sciences LLC, South San Francisco, California, USA.
Schiffmann R; Texas Christian University, Fort Worth, Texas, USA.
Sanyal S; Calico Life Sciences LLC, South San Francisco, California, USA.
Sidrauski C; Calico Life Sciences LLC, South San Francisco, California, USA.
Kolumam G; Calico Life Sciences LLC, South San Francisco, California, USA.
Baruch A; Calico Life Sciences LLC, South San Francisco, California, USA.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14600.
Typ publikacji:
Journal Article
MeSH Terms:
Growth Differentiation Factor 15*/genetics
Leukoencephalopathies*/genetics
Humans ; Mice ; Animals ; Central Nervous System/metabolism ; Eukaryotic Initiation Factor-2B/genetics ; Eukaryotic Initiation Factor-2B/metabolism ; Biomarkers
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
HIF1 α Promotes BMP9-Mediated Osteoblastic Differentiation and Vascularization by Interacting with CBFA1.
Autorzy:
Li Y; Department of Sports Medicine, Peking University Third Hospital, Institute of Sports Medicine of Peking University, Beijing Key Laboratory of Sports Injuries, Beijing 100191, China.
Liu Z; Department of Sports Medicine, Peking University Third Hospital, Institute of Sports Medicine of Peking University, Beijing Key Laboratory of Sports Injuries, Beijing 100191, China.
Wang HD; Department of Sports Medicine, Peking University Third Hospital, Institute of Sports Medicine of Peking University, Beijing Key Laboratory of Sports Injuries, Beijing 100191, China.
Zhang J; Department of Orthopaedics, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400016, China.
Lin M; Department of Orthopaedics, The First Affiliated Hospital of Zunyi Medical University, Zunyi, 563000 Guizhou, China.
Yang J; Department of Orthopaedics, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400016, China.
Huang J; Department of Orthopaedics, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400016, China.
Yan W; Department of Sports Medicine, Peking University Third Hospital, Institute of Sports Medicine of Peking University, Beijing Key Laboratory of Sports Injuries, Beijing 100191, China.
Ao Y; Department of Sports Medicine, Peking University Third Hospital, Institute of Sports Medicine of Peking University, Beijing Key Laboratory of Sports Injuries, Beijing 100191, China.
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Źródło:
BioMed research international [Biomed Res Int] 2022 Oct 01; Vol. 2022, pp. 2475169. Date of Electronic Publication: 2022 Oct 01 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Core Binding Factor Alpha 1 Subunit*/metabolism
Growth Differentiation Factor 2*/metabolism
Hypoxia-Inducible Factor 1, alpha Subunit*/metabolism
Mesenchymal Stem Cells*/metabolism
Animals ; Cell Differentiation ; Fibroblasts/metabolism ; Mice ; Neovascularization, Pathologic/metabolism ; Osteogenesis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Local anesthetic lidocaine-inducible gene, growth differentiation factor-15 suppresses the growth of cancer cell lines.
Autorzy:
Haraguchi-Suzuki K; Intensive Care Unit, Gunma University Hospital, 3-39-15, Showa-machi, Maebashi, Gunma, 371-8511, Japan. .
Kawabata-Iwakawa R; Division of Integrated Oncology Research, Initiative for Advanced Research, Gunma University, 3-39-15, Showa-machi, Maebashi, Gunma, 371-8511, Japan.
Suzuki T; Laboratory for Immunogenetics, RIKEN Center for Integrative Medical Sciences, 1-7-22, Suehiro-cho, Tsurumi-ku, Yokohama, Kanagawa, 230-0045, Japan.
Suto T; Department of Anesthesiology, Gunma University Hospital, 3-39-15, Showa-machi, Maebashi, Gunma, 371-8511, Japan.
Takazawa T; Intensive Care Unit, Gunma University Hospital, 3-39-15, Showa-machi, Maebashi, Gunma, 371-8511, Japan.
Saito S; Department of Anesthesiology, Gunma University Hospital, 3-39-15, Showa-machi, Maebashi, Gunma, 371-8511, Japan.
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Źródło:
Scientific reports [Sci Rep] 2022 Aug 25; Vol. 12 (1), pp. 14520. Date of Electronic Publication: 2022 Aug 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Growth Differentiation Factor 15*/genetics
Neoplasms*
Anesthetics, Local/pharmacology ; Growth Differentiation Factor 9 ; HeLa Cells ; Humans ; Lidocaine/pharmacology ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Dietary ingredient change induces a transient MyD88-dependent mucosal enteric glial cell response and promotes obesity.
Autorzy:
Liu Z; Laboratory of Infection and Immunity, Jiangsu Key Laboratory of Immunity and Metabolism, Department of Pathogenic Biology and Immunology, Xuzhou Medical University, Xuzhou, People's Republic of China.
Sun H; Laboratory of Infection and Immunity, Jiangsu Key Laboratory of Immunity and Metabolism, Department of Pathogenic Biology and Immunology, Xuzhou Medical University, Xuzhou, People's Republic of China.
Xu S; Laboratory of Infection and Immunity, Jiangsu Key Laboratory of Immunity and Metabolism, Department of Pathogenic Biology and Immunology, Xuzhou Medical University, Xuzhou, People's Republic of China.
Wang H; Laboratory of Infection and Immunity, Jiangsu Key Laboratory of Immunity and Metabolism, Department of Pathogenic Biology and Immunology, Xuzhou Medical University, Xuzhou, People's Republic of China.
Zhang Z; Laboratory of Infection and Immunity, Jiangsu Key Laboratory of Immunity and Metabolism, Department of Pathogenic Biology and Immunology, Xuzhou Medical University, Xuzhou, People's Republic of China.
Wei Y; Laboratory of Infection and Immunity, Jiangsu Key Laboratory of Immunity and Metabolism, Department of Pathogenic Biology and Immunology, Xuzhou Medical University, Xuzhou, People's Republic of China.
Kou Y; Laboratory of Infection and Immunity, Jiangsu Key Laboratory of Immunity and Metabolism, Department of Pathogenic Biology and Immunology, Xuzhou Medical University, Xuzhou, People's Republic of China.
Wang Y; Laboratory of Infection and Immunity, Jiangsu Key Laboratory of Immunity and Metabolism, Department of Pathogenic Biology and Immunology, Xuzhou Medical University, Xuzhou, People's Republic of China.
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Źródło:
Nutritional neuroscience [Nutr Neurosci] 2023 Dec; Vol. 26 (12), pp. 1183-1193. Date of Electronic Publication: 2022 Nov 07.
Typ publikacji:
Journal Article
MeSH Terms:
Ecosystem*
Myeloid Differentiation Factor 88*/genetics
Myeloid Differentiation Factor 88*/metabolism
Animals ; Mice ; Body Weight ; Diet, High-Fat/adverse effects ; Mice, Inbred C57BL ; Mucous Membrane/metabolism ; Neuroglia/metabolism ; Obesity/metabolism ; Weight Gain
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Two duplicated gsdf homeologs cooperatively regulate male differentiation by inhibiting cyp19a1a transcription in a hexaploid fish.
Autorzy:
Wang MT; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Li Z; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Ding M; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Yao TZ; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Yang S; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Zhang XJ; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Miao C; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Du WX; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Shi Q; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Li S; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Mei J; College of Fisheries, Huazhong Agricultural University, Wuhan, China.
Wang Y; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Wang ZW; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Zhou L; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Li XY; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
Gui JF; State Key Laboratory of Freshwater Ecology and Biotechnology, Hubei Hongshan Laboratory, The Innovative Academy of Seed Design, Institute of Hydrobiology, Chinese Academy of Sciences, Wuhan, China.; University of Chinese Academy of Sciences, Beijing, China.
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Źródło:
PLoS genetics [PLoS Genet] 2022 Jun 29; Vol. 18 (6), pp. e1010288. Date of Electronic Publication: 2022 Jun 29 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gonads*/metabolism
Sex Differentiation*/genetics
Animals ; Cell Differentiation/genetics ; Female ; Fish Proteins/genetics ; Fishes/genetics ; Gene Expression Regulation, Developmental ; Male ; Polyploidy ; Transcription Factors/genetics ; Transcription Factors/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Deletion of Wt1 during early gonadogenesis leads to differences of sex development in male and female adult mice.
Autorzy:
Torres-Cano A; Celltec-UB, Department of Cell Biology, Physiology and Immunology, Faculty of Biology, University of Barcelona, Barcelona, Spain.; Institute of Biomedicine (IBUB), University of Barcelona, Barcelona, Spain.
Portella-Fortuny R; Celltec-UB, Department of Cell Biology, Physiology and Immunology, Faculty of Biology, University of Barcelona, Barcelona, Spain.; Institute of Biomedicine (IBUB), University of Barcelona, Barcelona, Spain.
Müller-Sánchez C; Celltec-UB, Department of Cell Biology, Physiology and Immunology, Faculty of Biology, University of Barcelona, Barcelona, Spain.
Porras-Marfil S; Celltec-UB, Department of Cell Biology, Physiology and Immunology, Faculty of Biology, University of Barcelona, Barcelona, Spain.; Institute of Biomedicine (IBUB), University of Barcelona, Barcelona, Spain.
Ramiro-Pareta M; Celltec-UB, Department of Cell Biology, Physiology and Immunology, Faculty of Biology, University of Barcelona, Barcelona, Spain.; Institute of Biomedicine (IBUB), University of Barcelona, Barcelona, Spain.
Chau YY; Centre for Cardiovascular Science, Queen's Medical Research Institute, University of Edinburgh, United Kingdom.
Reina M; Celltec-UB, Department of Cell Biology, Physiology and Immunology, Faculty of Biology, University of Barcelona, Barcelona, Spain.
Soriano FX; Celltec-UB, Department of Cell Biology, Physiology and Immunology, Faculty of Biology, University of Barcelona, Barcelona, Spain.; Institut de Neurociències, Universitat de Barcelona, Barcelona, Spain.
Martínez-Estrada OM; Celltec-UB, Department of Cell Biology, Physiology and Immunology, Faculty of Biology, University of Barcelona, Barcelona, Spain.; Institute of Biomedicine (IBUB), University of Barcelona, Barcelona, Spain.
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Źródło:
PLoS genetics [PLoS Genet] 2022 Jun 15; Vol. 18 (6), pp. e1010240. Date of Electronic Publication: 2022 Jun 15 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Gonads*/metabolism
Sex Differentiation*/genetics
Animals ; Cell Differentiation/genetics ; Female ; Male ; Mice ; Ovary/metabolism ; Sexual Development ; Testis/metabolism ; WT1 Proteins/genetics ; WT1 Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 1-20 z 455982

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