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    Wyświetlanie 1-18 z 18
    Tytuł:
    Targeting autophagy overcomes Enzalutamide resistance in castration-resistant prostate cancer cells and improves therapeutic response in a xenograft model.
    Autorzy:
    Nguyen HG; Department of Urology, UC Davis School of Medicine, Sacramento, CA, USA.
    Yang JC; Department of Urology, UC Davis School of Medicine, Sacramento, CA, USA.
    Kung HJ; 1] Department of Biochemistry and Molecular Medicine, UC Davis School of Medicine, Sacramento, CA, USA [2] UC Davis Comprehensive Cancer Center, UC Davis School of Medicine, Sacramento, CA, USA.
    Shi XB; Department of Urology, UC Davis School of Medicine, Sacramento, CA, USA.
    Tilki D; Department of Urology, UC Davis School of Medicine, Sacramento, CA, USA.
    Lara PN Jr; UC Davis Comprehensive Cancer Center, UC Davis School of Medicine, Sacramento, CA, USA.
    DeVere White RW; 1] Department of Urology, UC Davis School of Medicine, Sacramento, CA, USA [2] UC Davis Comprehensive Cancer Center, UC Davis School of Medicine, Sacramento, CA, USA.
    Gao AC; 1] Department of Urology, UC Davis School of Medicine, Sacramento, CA, USA [2] UC Davis Comprehensive Cancer Center, UC Davis School of Medicine, Sacramento, CA, USA.
    Evans CP; 1] Department of Urology, UC Davis School of Medicine, Sacramento, CA, USA [2] UC Davis Comprehensive Cancer Center, UC Davis School of Medicine, Sacramento, CA, USA.
    Pokaż więcej
    Źródło:
    Oncogene [Oncogene] 2014 Sep 04; Vol. 33 (36), pp. 4521-30. Date of Electronic Publication: 2014 Mar 24.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Androgen Receptor Antagonists/*therapeutic use
    Autophagy/*drug effects
    Chloroquine/*pharmacology
    Clomipramine/*pharmacology
    Metformin/*pharmacology
    Phenylthiohydantoin/*analogs & derivatives
    Animals ; Benzamides ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; Male ; Mice ; Mice, SCID ; Nitriles ; Phenylthiohydantoin/therapeutic use ; Prostatic Neoplasms, Castration-Resistant/drug therapy ; Prostatic Neoplasms, Castration-Resistant/pathology ; Signal Transduction/drug effects ; Xenograft Model Antitumor Assays
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    miR-30 as a tumor suppressor connects EGF/Src signal to ERG and EMT.
    Autorzy:
    Kao CJ; Department of Biochemistry and Molecular Medicine, University of California-Davis, Davis, CA, USA.
    Martiniez A; Department of Biochemistry and Molecular Medicine, University of California-Davis, Davis, CA, USA.
    Shi XB; Department of Urology, University of California-Davis, Sacramento, CA, USA.
    Yang J; Department of Urology, University of California-Davis, Sacramento, CA, USA.
    Evans CP; Department of Urology, University of California-Davis, Sacramento, CA, USA.
    Dobi A; Center for Prostate Disease Research, Department of Surgery, Uniformed Services University of the Health Sciences, Rockville, MD, USA.
    deVere White RW; Department of Urology, University of California-Davis, Sacramento, CA, USA.
    Kung HJ; 1] Department of Biochemistry and Molecular Medicine, University of California-Davis, Davis, CA, USA [2] Institute of Molecular and Genomic Medicine, National Health Research Institutes, Maioli, Taiwan [3] Translational Medicine and Integrated Lab, Taipei Medical University, Taipei, Taiwan.
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    Źródło:
    Oncogene [Oncogene] 2014 May 08; Vol. 33 (19), pp. 2495-503. Date of Electronic Publication: 2013 Jun 03.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Epithelial-Mesenchymal Transition/*genetics
    MicroRNAs/*genetics
    Prostatic Neoplasms/*genetics
    Signal Transduction/*genetics
    Animals ; Blotting, Western ; Cell Line, Tumor ; Epidermal Growth Factor/genetics ; Epidermal Growth Factor/metabolism ; Fluorescent Antibody Technique ; Genes, Tumor Suppressor ; Heterografts ; Humans ; Male ; Oligonucleotide Array Sequence Analysis ; Oncogene Proteins, Fusion/genetics ; Oncogene Proteins, Fusion/metabolism ; Prostatic Neoplasms/metabolism ; Protein Kinase Inhibitors/pharmacology ; Real-Time Polymerase Chain Reaction ; Signal Transduction/drug effects ; Trans-Activators/genetics ; Trans-Activators/metabolism ; Transcriptional Regulator ERG ; src-Family Kinases/genetics ; src-Family Kinases/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Tumor suppressive miR-124 targets androgen receptor and inhibits proliferation of prostate cancer cells.
    Autorzy:
    Shi XB; Department of Urology, University of California, Davis, School of Medicine, Sacramento, CA 95817, USA.
    Xue L
    Ma AH
    Tepper CG
    Gandour-Edwards R
    Kung HJ
    deVere White RW
    Pokaż więcej
    Źródło:
    Oncogene [Oncogene] 2013 Aug 29; Vol. 32 (35), pp. 4130-8. Date of Electronic Publication: 2012 Oct 15.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Cell Proliferation*
    Genes, Tumor Suppressor/*physiology
    MicroRNAs/*physiology
    Prostatic Neoplasms/*genetics
    Receptors, Androgen/*genetics
    Animals ; Apoptosis ; Cell Line, Tumor ; DNA Methylation ; Down-Regulation ; Humans ; Male ; Mice ; Prostatic Neoplasms/pathology ; Prostatic Neoplasms/prevention & control ; Xenograft Model Antitumor Assays
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Oncomir miR-125b suppresses p14(ARF) to modulate p53-dependent and p53-independent apoptosis in prostate cancer.
    Autorzy:
    Amir S; Department of Urology, University of California Davis, Sacramento, California, United States of America.
    Ma AH
    Shi XB
    Xue L
    Kung HJ
    Devere White RW
    Pokaż więcej
    Źródło:
    PloS one [PLoS One] 2013 Apr 09; Vol. 8 (4), pp. e61064. Date of Electronic Publication: 2013 Apr 09 (Print Publication: 2013).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Gene Expression Regulation, Neoplastic*
    Apoptosis/*genetics
    MicroRNAs/*genetics
    Prostatic Neoplasms/*genetics
    Proto-Oncogene Proteins c-mdm2/*genetics
    Tumor Suppressor Protein p14ARF/*genetics
    Tumor Suppressor Protein p53/*genetics
    Apoptosis Regulatory Proteins/genetics ; Apoptosis Regulatory Proteins/metabolism ; Cell Line, Tumor ; Cell Proliferation ; Cyclin-Dependent Kinase Inhibitor p21/genetics ; Cyclin-Dependent Kinase Inhibitor p21/metabolism ; Humans ; Male ; MicroRNAs/metabolism ; Prostatic Neoplasms/metabolism ; Prostatic Neoplasms/pathology ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins/metabolism ; Proto-Oncogene Proteins c-mdm2/metabolism ; Signal Transduction ; Tumor Suppressor Protein p14ARF/antagonists & inhibitors ; Tumor Suppressor Protein p14ARF/metabolism ; Tumor Suppressor Protein p53/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    MicroRNA let-7c is downregulated in prostate cancer and suppresses prostate cancer growth.
    Autorzy:
    Nadiminty N; Department of Urology, University of California Davis, Sacramento, California, United States of America. />Tummala R
    Lou W
    Zhu Y
    Shi XB
    Zou JX
    Chen H
    Zhang J
    Chen X
    Luo J
    deVere White RW
    Kung HJ
    Evans CP
    Gao AC
    Pokaż więcej
    Źródło:
    PloS one [PLoS One] 2012; Vol. 7 (3), pp. e32832. Date of Electronic Publication: 2012 Mar 30.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Cell Proliferation*
    Down-Regulation*
    MicroRNAs/*genetics
    Prostatic Neoplasms/*genetics
    Androgens/metabolism ; Animals ; Blotting, Northern ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Genes, Tumor Suppressor ; Green Fluorescent Proteins/genetics ; Green Fluorescent Proteins/metabolism ; Humans ; In Situ Hybridization ; Lentivirus/genetics ; Lentivirus/metabolism ; Male ; Mice ; Mice, Nude ; Neoplasms, Experimental/genetics ; Neoplasms, Experimental/metabolism ; Neoplasms, Experimental/pathology ; Orchiectomy ; Prostatic Neoplasms/metabolism ; Prostatic Neoplasms/pathology ; Reverse Transcriptase Polymerase Chain Reaction ; Transplantation, Heterologous ; Tumor Burden/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Nucleic Acid, Antibody, and Virus Culture Methods to Detect Xenotropic MLV-Related Virus in Human Blood Samples.
    Autorzy:
    Kearney MF; HIV Drug Resistance Program, National Cancer Institute at Frederick, Frederick, MD 21702-1201, USA.
    Lee K
    Bagni RK
    Wiegand A
    Spindler J
    Maldarelli F
    Pinto PA
    Linehan WM
    Vocke CD
    Delviks-Frankenberry KA
    Devere White RW
    Del Prete GQ
    Mellors JW
    Lifson JD
    Kewalramani VN
    Pathak VK
    Coffin JM
    Le Grice SF
    Pokaż więcej
    Źródło:
    Advances in virology [Adv Virol] 2011; Vol. 2011, pp. 272193. Date of Electronic Publication: 2011 Nov 17.
    Typ publikacji:
    Journal Article
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Effects of a high dose, aglycone-rich soy extract on prostate-specific antigen and serum isoflavone concentrations in men with localized prostate cancer.
    Autorzy:
    deVere White RW; University of California, Davis, Sacramento, California 95616, USA.
    Tsodikov A
    Stapp EC
    Soares SE
    Fujii H
    Hackman RM
    Pokaż więcej
    Źródło:
    Nutrition and cancer [Nutr Cancer] 2010; Vol. 62 (8), pp. 1036-43.
    Typ publikacji:
    Journal Article; Randomized Controlled Trial; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Dietary Supplements*/adverse effects
    Dietary Supplements*/analysis
    Antineoplastic Agents, Phytogenic/*therapeutic use
    Genistein/*therapeutic use
    Isoflavones/*blood
    Isoflavones/*therapeutic use
    Polysaccharides/*therapeutic use
    Prostate-Specific Antigen/*blood
    Prostatic Neoplasms/*diet therapy
    Aged ; Aged, 80 and over ; Antineoplastic Agents, Phytogenic/adverse effects ; Antineoplastic Agents, Phytogenic/blood ; Antineoplastic Agents, Phytogenic/chemistry ; Biomarkers, Tumor/blood ; Double-Blind Method ; Equol ; Genistein/adverse effects ; Genistein/blood ; Genistein/chemistry ; Humans ; Isoflavones/adverse effects ; Male ; Middle Aged ; Phytotherapy/adverse effects ; Plant Extracts/adverse effects ; Plant Extracts/chemistry ; Plant Extracts/therapeutic use ; Polysaccharides/adverse effects ; Polysaccharides/chemistry ; Prostatic Neoplasms/blood ; Severity of Illness Index ; Glycine max/chemistry ; Time Factors
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Regulation of androgen receptor transcriptional activity by rapamycin in prostate cancer cell proliferation and survival.
    Autorzy:
    Wang Y; School of Medicine, University of California Davis, Sacramento, CA, USA.
    Mikhailova M
    Bose S
    Pan CX
    deVere White RW
    Ghosh PM
    Pokaż więcej
    Źródło:
    Oncogene [Oncogene] 2008 Nov 27; Vol. 27 (56), pp. 7106-17. Date of Electronic Publication: 2008 Sep 08.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Gene Expression Regulation, Neoplastic*
    Transcription, Genetic*
    Prostatic Neoplasms/*metabolism
    Receptors, Androgen/*metabolism
    Sirolimus/*pharmacology
    Adaptor Proteins, Signal Transducing ; Apoptosis ; Carrier Proteins/metabolism ; Cell Line, Tumor ; Cell Proliferation ; Cell Survival ; Humans ; Male ; Mechanistic Target of Rapamycin Complex 1 ; Multiprotein Complexes ; Proteins/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Rapamycin-Insensitive Companion of mTOR Protein ; Regulatory-Associated Protein of mTOR ; TOR Serine-Threonine Kinases ; Transcription Factors/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Cystectomy in the elderly: does the survival benefit in younger patients translate to the octogenarians?
    Autorzy:
    Chamie K; Department of Urology, University of California, Davis, Sacramento, CA, USA. />Hu B
    Devere White RW
    Ellison LM
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    Źródło:
    BJU international [BJU Int] 2008 Aug; Vol. 102 (3), pp. 284-90. Date of Electronic Publication: 2008 Apr 11.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Cystectomy/*mortality
    Urinary Bladder Neoplasms/*mortality
    Adult ; Age Factors ; Aged ; Aged, 80 and over ; Cohort Studies ; Cystectomy/methods ; Female ; Humans ; Lymph Node Excision/methods ; Male ; Middle Aged ; Neoplasm Invasiveness ; Prognosis ; Prospective Studies ; Sex Factors ; Survival Analysis ; Treatment Outcome ; Urinary Bladder Neoplasms/pathology ; Urinary Bladder Neoplasms/radiotherapy ; Urinary Bladder Neoplasms/surgery
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Inappropriate activation of androgen receptor by relaxin via beta-catenin pathway.
    Autorzy:
    Liu S; Department of Biochemistry and Molecular Medicine, School of Medicine and Cancer Center, University of California, Sacramento, CA, USA.
    Vinall RL
    Tepper C
    Shi XB
    Xue LR
    Ma AH
    Wang LY
    Fitzgerald LD
    Wu Z
    Gandour-Edwards R
    deVere White RW
    Kung HJ
    Pokaż więcej
    Źródło:
    Oncogene [Oncogene] 2008 Jan 17; Vol. 27 (4), pp. 499-505. Date of Electronic Publication: 2007 Jul 23.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Receptors, Androgen/*metabolism
    Relaxin/*physiology
    beta Catenin/*physiology
    Cell Nucleus/drug effects ; Chromones/pharmacology ; Enzyme Activation ; Enzyme Inhibitors/pharmacology ; Gene Expression Regulation, Neoplastic ; Glycogen Synthase Kinase 3/metabolism ; Glycogen Synthase Kinase 3 beta ; Humans ; Male ; Models, Biological ; Morpholines/pharmacology ; Phosphorylation ; Promoter Regions, Genetic ; Prostate-Specific Antigen/genetics ; Prostatic Neoplasms/genetics ; Prostatic Neoplasms/pathology ; Protein Binding ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction/physiology ; Tumor Cells, Cultured ; beta Catenin/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    A 90 kDa fragment of filamin A promotes Casodex-induced growth inhibition in Casodex-resistant androgen receptor positive C4-2 prostate cancer cells.
    Autorzy:
    Wang Y; Department of Urology, School of Medicine, University of California Davis, Sacramento, CA, USA.
    Kreisberg JI
    Bedolla RG
    Mikhailova M
    deVere White RW
    Ghosh PM
    Pokaż więcej
    Źródło:
    Oncogene [Oncogene] 2007 Sep 06; Vol. 26 (41), pp. 6061-70. Date of Electronic Publication: 2007 Apr 09.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Androgen Antagonists/*pharmacology
    Anilides/*pharmacology
    Antineoplastic Agents/*pharmacology
    Cell Division/*drug effects
    Contractile Proteins/*pharmacology
    Microfilament Proteins/*pharmacology
    Nitriles/*pharmacology
    Prostatic Neoplasms/*pathology
    Tosyl Compounds/*pharmacology
    Androgens/pharmacology ; Cell Line, Tumor ; Cell Nucleus/metabolism ; Drug Resistance, Neoplasm ; Filamins ; Humans ; Kinetics ; Male ; Oncogene Protein v-akt/metabolism ; Peptide Fragments/pharmacokinetics ; Peptide Fragments/pharmacology ; Phosphorylation ; Receptors, Androgen/drug effects ; Receptors, Androgen/physiology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Inhibition of Akt pathways in the treatment of prostate cancer.
    Autorzy:
    Nelson EC; Department of Urology, University of California at Davis, Sacramento, CA, USA.
    Evans CP
    Mack PC
    Devere-White RW
    Lara PN Jr
    Pokaż więcej
    Źródło:
    Prostate cancer and prostatic diseases [Prostate Cancer Prostatic Dis] 2007; Vol. 10 (4), pp. 331-9. Date of Electronic Publication: 2007 May 01.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Review
    MeSH Terms:
    Antineoplastic Agents/*therapeutic use
    Prostatic Neoplasms/*drug therapy
    Proto-Oncogene Proteins c-akt/*antagonists & inhibitors
    Signal Transduction/*drug effects
    Humans ; Male
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Abnormalities of apoptotic and cell cycle regulatory proteins in distinct histopathologic components of benign prostatic hyperplasia.
    Autorzy:
    Gandour-Edwards R; Department of Pathology, University of California Davis Health System, 4400V Street, Sacramento, CA 95817, USA. />Mack PC
    Devere-White RW
    Gumerlock PH
    Pokaż więcej
    Źródło:
    Prostate cancer and prostatic diseases [Prostate Cancer Prostatic Dis] 2004; Vol. 7 (4), pp. 321-6.
    Typ publikacji:
    Comparative Study; Journal Article; Research Support, U.S. Gov't, P.H.S.
    MeSH Terms:
    Apoptosis*
    Cell Cycle*
    Cell Cycle Proteins/*metabolism
    Prostatic Hyperplasia/*metabolism
    Prostatic Hyperplasia/*pathology
    Proto-Oncogene Proteins c-bcl-2/*metabolism
    Tumor Suppressor Proteins/*metabolism
    Aged ; Aged, 80 and over ; Cyclin-Dependent Kinase Inhibitor p27 ; Down-Regulation ; Enzyme Inhibitors/metabolism ; Epithelial Cells/metabolism ; Genes, Tumor Suppressor ; Humans ; Immunoenzyme Techniques ; Male ; Middle Aged ; Stromal Cells/metabolism ; bcl-2-Associated X Protein
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-18 z 18

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