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Wyszukujesz frazę ""Dendritic Cells/metabolism"" wg kryterium: Temat


Tytuł :
Formation of the Intrathymic Dendritic Cell Pool Requires CCL21-Mediated Recruitment of CCR7+ Progenitors to the Thymus.
Autorzy :
Cosway, E.J.
Ohigashi, I.
Schauble, K.
Parnell, S.M.
Jenkinson, W.E.
Luther, S.
Takahama, Y.
Anderson, G.
Pokaż więcej
Temat :
Animals
Cell Movement/physiology
Chemokine CCL21/metabolism
Dendritic Cells/metabolism
Immune Tolerance/physiology
Mice
Mice, Inbred C57BL
Receptors, CCR7/metabolism
Thymocytes/metabolism
Thymus Gland/metabolism
Źródło :
Journal of immunology, vol. 201, no. 2, pp. 516-523
Opis pliku :
application/pdf
Tytuł :
The CLEC12A receptor marks human basophils::Potential implications for minimal residual disease detection in acute myeloid leukemia
Autorzy :
Toft-Petersen, Marie
Stidsholt Roug, Anne
Plesner, Trine
Ebbesen, Lene
Brown, Gordon D.
Nederby, Line
Pokaż więcej
Temat :
hMICL
CLEC12A
basophils
CD371
Neoplasm, Residual/diagnosis
Leukemia, Myeloid, Acute/diagnosis
Dendritic Cells/metabolism
immunophenotyping
Lectins, C-Type/metabolism
Basophils/metabolism
Receptors, Mitogen/metabolism
Humans
Flow Cytometry/methods
Biomarkers, Tumor/metabolism
Źródło :
Toft-Petersen, M, Stidsholt Roug, A, Plesner, T, Ebbesen, L, Brown, G D & Nederby, L 2018, ' The CLEC12A receptor marks human basophils : Potential implications for minimal residual disease detection in acute myeloid leukemia ', Cytometry. Part B: Clinical Cytometry, vol. 94, no. 3, pp. 520-526 . https://doi.org/10.1002/cyto.b.21540
Toft-Petersen, M, Stidsholt Roug, A, Plesner, T, Ebbesen, L, Brown, G D & Nederby, L 2018, ' The CLEC12A receptor marks human basophils: Potential implications for minimal residual disease detection in acute myeloid leukemia ', Cytometry. Part B: Clinical Cytometry, vol. 94, no. 3, pp. 520-526 . https://doi.org/10.1002/cyto.b.21540
Opis pliku :
application/pdf
Tytuł :
Sumoylation coordinates the repression of inflammatory and anti-viral gene-expression programs during innate sensing
Autorzy :
Decque, Adrien
Joffre, Olivier
Magalhaes, Joao G
Cossec, Jack-Christophe
Blecher-Gonen, Ronnie
Lapaquette, Pierre
Silvin, Aymeric
Manel, Nicolas
Joubert, Pierre-Emmanuel
Seeler, Sebastian
Albert, Matthew L
Amit, Ido
Amigorena, Sebastian
Dejean, Anne
Pokaż więcej
Temat :
MESH: Inflammation/virology
MESH: Shock, Septic/metabolism
MESH: Dendritic Cells/metabolism
MESH: Animals
MESH: Receptor, Interferon alpha-beta/metabolism
MESH: Gene Expression Regulation
MESH: Sumoylation*/genetics
MESH: Disease Models, Animal
[SDV.IMM]Life Sciences [q-bio]/Immunology
MESH: Disease Susceptibility
MESH: Immunity, Innate
MESH: Shock, Septic/immunology
MESH: Chromatin/metabolism
MESH: Disease Resistance
MESH: Lipopolysaccharides/immunology
MESH: Regulatory Elements, Transcriptional
MESH: Cytokines/metabolism
MESH: Inflammation Mediators/metabolism
Inflammation
MESH: Sumoylation*/immunology
MESH: Dendritic Cells/immunology
MESH: Gene Expression Profiling
MESH: Inflammation/immunology
MESH: Inflammation/genetics
MESH: Toll-Like Receptors/metabolism
MESH: Immunomodulation
MESH: Genetic Loci
MESH: Chromatin/genetics
MESH: Interferon-beta/metabolism
MESH: Inflammation/metabolism
Pattern recognition receptors
MESH: Protein Binding
MESH: Shock, Septic/genetics
MESH: Mice
Gene regulation in immune cells
MESH: Mice, Knockout
MESH: Signal Transduction
MESH: Enhancer Elements, Genetic
MESH: SUMO-1 Protein/metabolism
Źródło :
Nature Immunology, Nature Publishing Group, 2016, 17 (2), pp.140 - 149. ⟨10.1038/ni.3342⟩
Tytuł :
Sumoylation coordinates the repression of inflammatory and anti-viral gene-expression programs during innate sensing
Autorzy :
Decque, Adrien
Joffre, Olivier
Magalhaes, Joao G
Cossec, Jack-Christophe
Blecher-Gonen, Ronnie
Lapaquette, Pierre
Silvin, Aymeric
Manel, Nicolas
Joubert, Pierre-Emmanuel
Seeler, Sebastian
Albert, Matthew L
Amit, Ido
Amigorena, Sebastian
Dejean, Anne
Pokaż więcej
Temat :
Gene regulation in immune cells
Inflammation
Pattern recognition receptors
MESH: Animals
MESH: Chromatin/genetics
MESH: Gene Expression Profiling
MESH: Gene Expression Regulation
MESH: Genetic Loci
MESH: Immunity, Innate
MESH: Immunomodulation
MESH: Inflammation/genetics
MESH: Inflammation/immunology
MESH: Inflammation/metabolism
MESH: Inflammation/virology
MESH: Inflammation Mediators/metabolism
MESH: Chromatin/metabolism
MESH: Interferon-beta/metabolism
MESH: Lipopolysaccharides/immunology
MESH: Mice
MESH: Mice, Knockout
MESH: Protein Binding
MESH: Receptor, Interferon alpha-beta/metabolism
MESH: Regulatory Elements, Transcriptional
MESH: SUMO-1 Protein/metabolism
MESH: Shock, Septic/genetics
MESH: Shock, Septic/immunology
MESH: Cytokines/metabolism
MESH: Shock, Septic/metabolism
MESH: Signal Transduction
MESH: Sumoylation*/genetics
MESH: Sumoylation*/immunology
MESH: Toll-Like Receptors/metabolism
MESH: Dendritic Cells/immunology
MESH: Dendritic Cells/metabolism
MESH: Disease Models, Animal
MESH: Disease Resistance
MESH: Disease Susceptibility
MESH: Enhancer Elements, Genetic
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Nature Immunology, Nature Publishing Group, 2016, 17 (2), pp.140 - 149. ⟨10.1038/ni.3342⟩
Tytuł :
Targeted Delivery of α-Galactosylceramide to CD8α + Dendritic Cells Optimizes Type I NKT Cell–Based Antitumor Responses
Autorzy :
Macho-Fernandez, Elodie
Cruz, Luis Javier
Ghinnagow, Reem
Fontaine, Josette
Bialecki, Emilie
Frisch, Benoît
Trottein, François
Faveeuw, Christelle
Pokaż więcej
Temat :
MESH: Animals
MESH: Antibodies/chemistry
MESH: Dendritic Cells/immunology
MESH: Dendritic Cells/metabolism
MESH: Drug Delivery Systems/methods
MESH: Galactosylceramides/administration & dosage
MESH: Galactosylceramides/chemistry
MESH: Galactosylceramides/immunology
MESH: Lectins, C-Type/immunology
MESH: Lymphocyte Activation/immunology
MESH: Male
MESH: Mice
MESH: Antibodies/immunology
MESH: Mice, Inbred C57BL
MESH: Mice, Knockout
MESH: Mice, Transgenic
MESH: Minor Histocompatibility Antigens
MESH: Nanoparticles/administration & dosage
MESH: Nanoparticles/chemistry
MESH: Natural Killer T-Cells/immunology
MESH: Natural Killer T-Cells/metabolism
MESH: Neoplasms, Experimental/immunology
MESH: Neoplasms, Experimental/pathology
MESH: Antigen Presentation/immunology
MESH: Neoplasms, Experimental/therapy
MESH: Receptors, Antigen, T-Cell, gamma-delta/immunology
MESH: Receptors, Antigen, T-Cell, gamma-delta/metabolism
MESH: Receptors, Cell Surface/immunology
MESH: T-Lymphocytes/immunology
MESH: T-Lymphocytes/metabolism
MESH: T-Lymphocytes, Cytotoxic/immunology
MESH: T-Lymphocytes, Cytotoxic/metabolism
MESH: Tumor Burden/immunology
MESH: Antigens, CD/immunology
MESH: CD8 Antigens/immunology
MESH: CD8 Antigens/metabolism
MESH: CD8-Positive T-Lymphocytes/immunology
MESH: CD8-Positive T-Lymphocytes/metabolism
MESH: Cell Line, Tumor
[SDV.IMM.II]Life Sciences [q-bio]/Immunology/Innate immunity
Źródło :
Journal of Immunology, Publisher : Baltimore : Williams & Wilkins, c1950-. Latest Publisher : Bethesda, MD : American Association of Immunologists, 2014, 193 (2), pp.961-969. ⟨10.4049/jimmunol.1303029⟩
Tytuł :
Pivotal role of M-DC8+ monocytes from viremic HIV-infected patients in TNF overproduction in response to microbial products.
Autorzy :
Dutertre, Charles-Antoine
Amraoui, Sonia
DeRosa, Annalisa
Jourdain, Jean-Pierre
Vimeux, Lene
Goguet, Matthieu
Degrelle, Severine
Feuillet, Vincent
Liovat, Anne-Sophie
Müller-Trutwin, Michaela
Decroix, Nipa
Deveau, Christiane
Meyer, Laurence
Goujard, Cécile
Loulergue, Pierre
Launay, Odile
Richard, Yolande
Hosmalin, Anne
Pokaż więcej
Temat :
MESH: Dendritic Cells/metabolism
MESH: Macrophage Activation/drug effects
MESH: Male
MESH: Monocytes/metabolism
[SDV.IMM]Life Sciences [q-bio]/Immunology
MESH: Monocytes/immunology
MESH: Glycoproteins
MESH: Viremia/metabolism
[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseases
MESH: Lipopolysaccharides/toxicity
MESH: Macrophages/drug effects
MESH: Monocytes/drug effects
MESH: Viremia/pathology
MESH: Young Adult
MESH: Dendritic Cells/immunology
MESH: Tumor Necrosis Factor-alpha/metabolism
MESH: Dendritic Cells/drug effects
[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/Virology
MESH: Anti-HIV Agents/therapeutic use
MESH: Adult
MESH: Up-Regulation*/drug effects
MESH: Macrophages/immunology
MESH: Viremia/drug therapy
[SDV.IMM.IMM]Life Sciences [q-bio]/Immunology/Immunotherapy
MESH: HIV Infections/metabolism
MESH: Antigens, Surface/metabolism
MESH: Cells, Cultured
MESH: Humans
MESH: Macrophages/pathology
MESH: Monocytes/pathology
MESH: Flow Cytometry
MESH: HIV Infections/pathology
MESH: Antibodies, Monoclonal/metabolism
[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunology
MESH: Viremia/immunology
MESH: Macrophages/metabolism
MESH: Dendritic Cells/pathology Female
MESH: Middle Aged
MESH: HIV Infections/drug therapy
MESH: HIV Infections/immunology
MESH: Membrane Glycoproteins/antagonists & inhibitors
[SDV]Life Sciences [q-bio]
[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
MESH: Antigens, CD1
Źródło :
Blood, American Society of Hematology, 2012, 120 (11), pp.2259-2268. ⟨10.1182/blood-2012-03-418681⟩
Tytuł :
Pivotal role of M-DC8+ monocytes from viremic HIV-infected patients in TNF overproduction in response to microbial products.
Autorzy :
Dutertre, Charles-Antoine
Amraoui, Sonia
DeRosa, Annalisa
Jourdain, Jean-Pierre
Vimeux, Lene
Goguet, Matthieu
Degrelle, Severine
Feuillet, Vincent
Liovat, Anne-Sophie
Müller-Trutwin, Michaela
Decroix, Nipa
Deveau, Christiane
Meyer, Laurence
Goujard, Cécile
Loulergue, Pierre
Launay, Odile
Richard, Yolande
Hosmalin, Anne
Pokaż więcej
Temat :
MESH: Adult
MESH: Anti-HIV Agents/therapeutic use
MESH: Flow Cytometry
MESH: Glycoproteins
MESH: HIV Infections/drug therapy
MESH: HIV Infections/immunology
MESH: HIV Infections/metabolism
MESH: HIV Infections/pathology
MESH: Humans
MESH: Lipopolysaccharides/toxicity
MESH: Macrophage Activation/drug effects
MESH: Macrophages/drug effects
MESH: Antibodies, Monoclonal/metabolism
MESH: Macrophages/immunology
MESH: Macrophages/metabolism
MESH: Macrophages/pathology
MESH: Male
MESH: Membrane Glycoproteins/antagonists & inhibitors
MESH: Middle Aged
MESH: Monocytes/drug effects
MESH: Monocytes/immunology
MESH: Monocytes/metabolism
MESH: Monocytes/pathology
MESH: Antigens, CD1
MESH: Tumor Necrosis Factor-alpha/metabolism
MESH: Up-Regulation*/drug effects
MESH: Viremia/drug therapy
MESH: Viremia/immunology
MESH: Viremia/metabolism
MESH: Viremia/pathology
MESH: Young Adult
MESH: Antigens, Surface/metabolism
MESH: Cells, Cultured
MESH: Dendritic Cells/drug effects
MESH: Dendritic Cells/immunology
MESH: Dendritic Cells/metabolism
MESH: Dendritic Cells/pathology Female
[SDV.IMM.IMM]Life Sciences [q-bio]/Immunology/Immunotherapy
[SDV.IMM]Life Sciences [q-bio]/Immunology
Źródło :
Blood, American Society of Hematology, 2012, 120 (11), pp.2259-2268. ⟨10.1182/blood-2012-03-418681⟩

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