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Tytuł:
TRIM3 Inhibits H 2 O 2 -Induced Apoptosis in Human Lens Epithelial Cells by Decreasing p53 via Ubiquitination.
Autorzy:
Deng Y; Department of Ophtalmology, Shanghai Eighth People Hospital, Shanghai, China.
Shi Y; Department of Ophtalmology, Shanghai Eighth People Hospital, Shanghai, China.
Wen C; Department of Ophtalmology, Shanghai Eighth People Hospital, Shanghai, China.
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Źródło:
Current eye research [Curr Eye Res] 2022 May; Vol. 47 (5), pp. 747-752. Date of Electronic Publication: 2022 Mar 23.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Carrier Proteins*/metabolism
Epithelial Cells*/cytology
Lens, Crystalline*/cytology
Tumor Suppressor Protein p53*/genetics
Cataract/metabolism ; Cells, Cultured ; Gene Knockdown Techniques ; Humans ; Hydrogen Peroxide/toxicity ; Oxidative Stress ; Ubiquitination
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Fyn gene silencing reduces oligodendrocytes apoptosis through inhibiting ERK1/2 phosphorylation in epilepsy.
Autorzy:
Luo X; Department of Neurology, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Li Z; Department of Neurology, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Zhao J; Department of Neurology, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Deng Y; Department of Neurology, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Zhong Y; Department of Neurology, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Zhang M; Department of Neurology, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
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Źródło:
Artificial cells, nanomedicine, and biotechnology [Artif Cells Nanomed Biotechnol] 2020 Dec; Vol. 48 (1), pp. 298-304.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Silencing*
Apoptosis/*genetics
Epilepsy/*genetics
Mitogen-Activated Protein Kinase 1/*metabolism
Mitogen-Activated Protein Kinase 3/*metabolism
Oligodendroglia/*pathology
Proto-Oncogene Proteins c-fyn/*genetics
Animals ; Epilepsy/pathology ; Gene Expression Regulation, Enzymologic/genetics ; Phosphorylation/genetics ; Proto-Oncogene Proteins c-fyn/deficiency ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Aflatoxin B1 Induces Neurotoxicity through Reactive Oxygen Species Generation, DNA Damage, Apoptosis, and S-Phase Cell Cycle Arrest.
Autorzy:
Huang B; Guangdong Provincial Key Laboratory of Protein Function and Regulation in Agricultural Organisms, College of Life Sciences, South China Agricultural University, Tianhe District, Guangzhou 510642, China.; Key Laboratory of Zoonosis of Ministry of Agriculture and Rural Affairs, South China Agricultural University, Guangzhou 510642, China.
Chen Q; Guangdong Provincial Key Laboratory of Protein Function and Regulation in Agricultural Organisms, College of Life Sciences, South China Agricultural University, Tianhe District, Guangzhou 510642, China.; Key Laboratory of Zoonosis of Ministry of Agriculture and Rural Affairs, South China Agricultural University, Guangzhou 510642, China.
Wang L; Guangdong Provincial Key Laboratory of Protein Function and Regulation in Agricultural Organisms, College of Life Sciences, South China Agricultural University, Tianhe District, Guangzhou 510642, China.; Key Laboratory of Zoonosis of Ministry of Agriculture and Rural Affairs, South China Agricultural University, Guangzhou 510642, China.
Gao X; Guangdong Provincial Key Laboratory of Protein Function and Regulation in Agricultural Organisms, College of Life Sciences, South China Agricultural University, Tianhe District, Guangzhou 510642, China.; Key Laboratory of Zoonosis of Ministry of Agriculture and Rural Affairs, South China Agricultural University, Guangzhou 510642, China.
Zhu W; Guangdong Provincial Key Laboratory of Protein Function and Regulation in Agricultural Organisms, College of Life Sciences, South China Agricultural University, Tianhe District, Guangzhou 510642, China.; Key Laboratory of Zoonosis of Ministry of Agriculture and Rural Affairs, South China Agricultural University, Guangzhou 510642, China.
Mu P; Guangdong Provincial Key Laboratory of Protein Function and Regulation in Agricultural Organisms, College of Life Sciences, South China Agricultural University, Tianhe District, Guangzhou 510642, China.; Key Laboratory of Zoonosis of Ministry of Agriculture and Rural Affairs, South China Agricultural University, Guangzhou 510642, China.
Deng Y; Guangdong Provincial Key Laboratory of Protein Function and Regulation in Agricultural Organisms, College of Life Sciences, South China Agricultural University, Tianhe District, Guangzhou 510642, China.; Key Laboratory of Zoonosis of Ministry of Agriculture and Rural Affairs, South China Agricultural University, Guangzhou 510642, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Sep 06; Vol. 21 (18). Date of Electronic Publication: 2020 Sep 06.
Typ publikacji:
Journal Article
MeSH Terms:
DNA Damage*/physiology
Neurotoxicity Syndromes*/metabolism
Neurotoxicity Syndromes*/pathology
Aflatoxin B1/*toxicity
Apoptosis/*drug effects
Reactive Oxygen Species/*metabolism
S Phase/*drug effects
Aflatoxin B1/pharmacology ; Animals ; Apoptosis/physiology ; Cell Cycle/drug effects ; Cell Cycle/genetics ; Cell Cycle Checkpoints/drug effects ; Cell Cycle Checkpoints/genetics ; Cell Survival/drug effects ; Cell Survival/genetics ; Cells, Cultured ; Male ; Mice ; Oxidative Stress/drug effects ; Oxidative Stress/physiology ; S Phase/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Phospholipase D1 Ameliorates Apoptosis in Chronic Renal Toxicity Caused by Low-Dose Cadmium Exposure.
Autorzy:
Huang K; Department of Occupational Health and Occupational Medicine, School of Public Health, Southern Medical University, Guangzhou 510515, China.; The First People's Hospital of Zhaoqing City, Zhaoqing 526000, China.
Deng Y; Department of Occupational Health and Occupational Medicine, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Yuan W; Department of Occupational Health and Occupational Medicine, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Geng J; Department of Pathology, Southern Medical University, Guangzhou 510515, China.
Wang G; Department of Occupational Health and Occupational Medicine, School of Public Health, Southern Medical University, Guangzhou 510515, China.
Zou F; Department of Occupational Health and Occupational Medicine, School of Public Health, Southern Medical University, Guangzhou 510515, China.
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Źródło:
BioMed research international [Biomed Res Int] 2020 Mar 31; Vol. 2020, pp. 7091053. Date of Electronic Publication: 2020 Mar 31 (Print Publication: 2020).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Cadmium/*toxicity
Kidney/*drug effects
Phospholipase D/*genetics
Renal Insufficiency/*drug therapy
3' Untranslated Regions ; Animals ; Cell Line ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Gene Expression Regulation ; Kidney/cytology ; Kidney/metabolism ; Male ; MicroRNAs/genetics ; MicroRNAs/metabolism ; Phospholipase D/metabolism ; Rats ; Rats, Sprague-Dawley ; Renal Insufficiency/etiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Protein Kinase A Is Involved in Neuropathic Pain by Activating the p38MAPK Pathway to Mediate Spinal Cord Cell Apoptosis.
Autorzy:
Deng Y; Department of Orthopaedics, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; Key Laboratory of Orthopaedics Disease of Gansu Province, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; The International Cooperation Base of Gansu Province for The Pain Research in Spinal Disorders, Lanzhou, Gansu Province 730030, China.
Yang L; Department of Orthopaedics, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; Key Laboratory of Orthopaedics Disease of Gansu Province, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; The International Cooperation Base of Gansu Province for The Pain Research in Spinal Disorders, Lanzhou, Gansu Province 730030, China.
Xie Q; Department of Orthopaedics, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; Key Laboratory of Orthopaedics Disease of Gansu Province, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; The International Cooperation Base of Gansu Province for The Pain Research in Spinal Disorders, Lanzhou, Gansu Province 730030, China.
Yang F; Department of Neurosurgery, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; Institute of Neurology of Gansu Province, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.
Li G; Department of Neurosurgery, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; Institute of Neurology of Gansu Province, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.
Zhang G; Department of Orthopaedics, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; Key Laboratory of Orthopaedics Disease of Gansu Province, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; The International Cooperation Base of Gansu Province for The Pain Research in Spinal Disorders, Lanzhou, Gansu Province 730030, China.
Li S; Department of Orthopaedics, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; Key Laboratory of Orthopaedics Disease of Gansu Province, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.
Wu Z; Department of Orthopaedics, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; Key Laboratory of Orthopaedics Disease of Gansu Province, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.
Wang J; The Department of Osteoporosis, The People's Hospital of Baoan District, Shenzhen, Guangdong Province 518000, China.
Kang X; Department of Orthopaedics, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; Key Laboratory of Orthopaedics Disease of Gansu Province, Lanzhou University Second Hospital, Lanzhou, Gansu Province 730030, China.; The International Cooperation Base of Gansu Province for The Pain Research in Spinal Disorders, Lanzhou, Gansu Province 730030, China.
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Źródło:
Mediators of inflammation [Mediators Inflamm] 2020 Mar 23; Vol. 2020, pp. 6420425. Date of Electronic Publication: 2020 Mar 23 (Print Publication: 2020).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*physiology
Cyclic AMP-Dependent Protein Kinases/*metabolism
Neuralgia/*metabolism
Neuralgia/*pathology
p38 Mitogen-Activated Protein Kinases/*metabolism
p38 Mitogen-Activated Protein Kinases/*physiology
Animals ; Apoptosis/genetics ; Blotting, Western ; Cyclic AMP-Dependent Protein Kinases/genetics ; Enzyme-Linked Immunosorbent Assay ; Imidazoles/pharmacology ; Immunohistochemistry ; In Situ Nick-End Labeling ; Male ; Pyridines/pharmacology ; Rats ; Software ; p38 Mitogen-Activated Protein Kinases/antagonists & inhibitors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
SPAG5-AS1 inhibited autophagy and aggravated apoptosis of podocytes via SPAG5/AKT/mTOR pathway.
Autorzy:
Xu J; Department of Geriatrics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
Deng Y; Department of Endocrinology, The Affiliated Hospital of Qingdao University, Qingdao, China.
Wang Y; Department of Nephrology, Yueyang Hospital of Integrated Traditional Chinese and Western Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Sun X; Department of Endocrinology, The Affiliated Hospital of Qingdao University, Qingdao, China.
Chen S; Department of Endocrinology and Metabolism, Ningbo First Hospital, Ningbo, China.
Fu G; Department of Geriatrics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
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Źródło:
Cell proliferation [Cell Prolif] 2020 Feb; Vol. 53 (2), pp. e12738. Date of Electronic Publication: 2020 Jan 19.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*physiology
Autophagy/*physiology
Cell Cycle Proteins/*metabolism
Podocytes/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Signal Transduction/*physiology
TOR Serine-Threonine Kinases/*metabolism
Cells, Cultured ; Diabetic Nephropathies/metabolism ; Diabetic Nephropathies/pathology ; Glucose/metabolism ; Humans ; Podocytes/physiology ; RNA, Long Noncoding/metabolism ; Transcription, Genetic/physiology ; Ubiquitin Thiolesterase/metabolism ; Ubiquitination/physiology ; Up-Regulation/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Potential Role of MicroRNA-124 in Cerebral Ischemia Injury.
Autorzy:
Liu X; Third-Grade Pharmacological Laboratory on Chinese Medicine Approved by State Administration of Traditional Chinese Medicine, Medical College of China Three Gorges University, Yichang 443002, China.
Feng Z; Third-Grade Pharmacological Laboratory on Chinese Medicine Approved by State Administration of Traditional Chinese Medicine, Medical College of China Three Gorges University, Yichang 443002, China.
Du L; Third-Grade Pharmacological Laboratory on Chinese Medicine Approved by State Administration of Traditional Chinese Medicine, Medical College of China Three Gorges University, Yichang 443002, China.
Huang Y; Third-Grade Pharmacological Laboratory on Chinese Medicine Approved by State Administration of Traditional Chinese Medicine, Medical College of China Three Gorges University, Yichang 443002, China.
Ge J; The Key Laboratory of Hunan Province for Integrated Traditional Chinese and Western Medicine on Prevention and Treatment of Cardio-Cerebral Diseases, Hunan University of Chinese Medicine, Changsha 410208, China.
Deng Y; The Key Laboratory of Hunan Province for Integrated Traditional Chinese and Western Medicine on Prevention and Treatment of Cardio-Cerebral Diseases, Hunan University of Chinese Medicine, Changsha 410208, China.
Mei Z; Third-Grade Pharmacological Laboratory on Chinese Medicine Approved by State Administration of Traditional Chinese Medicine, Medical College of China Three Gorges University, Yichang 443002, China.; The Key Laboratory of Hunan Province for Integrated Traditional Chinese and Western Medicine on Prevention and Treatment of Cardio-Cerebral Diseases, Hunan University of Chinese Medicine, Changsha 410208, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Dec 23; Vol. 21 (1). Date of Electronic Publication: 2019 Dec 23.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Apoptosis*
Autophagic Cell Death*
Oxidative Stress*
Brain Injuries/*metabolism
Brain Ischemia/*metabolism
MicroRNAs/*metabolism
Alzheimer Disease/metabolism ; Alzheimer Disease/pathology ; Animals ; Brain Injuries/pathology ; Brain Ischemia/pathology ; Humans ; Stroke/metabolism ; Stroke/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Methyl-mercury induces apoptosis through ROS-mediated endoplasmic reticulum stress and mitochondrial apoptosis pathways activation in rat cortical neurons.
Autorzy:
Liu W; a Department of Environmental Health, School of Public Health , China Medical University , Shenyang , People's Republic of China.
Yang T; a Department of Environmental Health, School of Public Health , China Medical University , Shenyang , People's Republic of China.
Xu Z; a Department of Environmental Health, School of Public Health , China Medical University , Shenyang , People's Republic of China.
Xu B; a Department of Environmental Health, School of Public Health , China Medical University , Shenyang , People's Republic of China.
Deng Y; a Department of Environmental Health, School of Public Health , China Medical University , Shenyang , People's Republic of China.
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Źródło:
Free radical research [Free Radic Res] 2019 Jan; Vol. 53 (1), pp. 26-44. Date of Electronic Publication: 2018 Dec 04.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Cerebral Cortex/*cytology
Endoplasmic Reticulum Stress/*drug effects
Methylmercury Compounds/*pharmacology
Mitochondria/*drug effects
Neurons/*cytology
Neurons/*drug effects
Reactive Oxygen Species/*metabolism
Animals ; Cell Survival/drug effects ; Dose-Response Relationship, Drug ; Female ; Male ; Mitochondria/metabolism ; Neurons/metabolism ; Rats ; Rats, Wistar ; Structure-Activity Relationship
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Evaluation of a combination "lymphocyte apoptosis model" to predict survival of sepsis patients in an intensive care unit.
Autorzy:
Jiang W; The Second School of Clinical Medicine, Southern Medical University, 1063 Shatai Nan road, Guangzhou, 510515, China.; Department of Emergency and Critical Care Medicine, Guangdong General Hospital, Guangdong Academy of Medical Sciences, 106 Zhongshan Er Road, Guangzhou, 510080, Guangdong, China.
Zhong W; The Second School of Clinical Medicine, Southern Medical University, 1063 Shatai Nan road, Guangzhou, 510515, China.; Department of Emergency and Critical Care Medicine, Guangdong General Hospital, Guangdong Academy of Medical Sciences, 106 Zhongshan Er Road, Guangzhou, 510080, Guangdong, China.
Deng Y; Department of Emergency and Critical Care Medicine, Guangdong General Hospital, Guangdong Academy of Medical Sciences, 106 Zhongshan Er Road, Guangzhou, 510080, Guangdong, China.
Chen C; Department of Emergency and Critical Care Medicine, Guangdong General Hospital, Guangdong Academy of Medical Sciences, 106 Zhongshan Er Road, Guangzhou, 510080, Guangdong, China.
Wang Q; The Second School of Clinical Medicine, Southern Medical University, 1063 Shatai Nan road, Guangzhou, 510515, China.; Department of Emergency and Critical Care Medicine, Guangdong General Hospital, Guangdong Academy of Medical Sciences, 106 Zhongshan Er Road, Guangzhou, 510080, Guangdong, China.
Zhou M; Division of Laboratory, Guangdong General Hospital, Guangdong Academy of Medical Sciences, 106 Zhongshan Er Road, Guangzhou, 510080, Guangdong, China.
Li X; The Second School of Clinical Medicine, Southern Medical University, 1063 Shatai Nan road, Guangzhou, 510515, China.; Department of Emergency and Critical Care Medicine, Guangdong General Hospital, Guangdong Academy of Medical Sciences, 106 Zhongshan Er Road, Guangzhou, 510080, Guangdong, China.
Sun C; Department of Emergency and Critical Care Medicine, Guangdong General Hospital, Guangdong Academy of Medical Sciences, 106 Zhongshan Er Road, Guangzhou, 510080, Guangdong, China.
Zeng H; The Second School of Clinical Medicine, Southern Medical University, 1063 Shatai Nan road, Guangzhou, 510515, China. .; Department of Emergency and Critical Care Medicine, Guangdong General Hospital, Guangdong Academy of Medical Sciences, 106 Zhongshan Er Road, Guangzhou, 510080, Guangdong, China. .
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Źródło:
BMC anesthesiology [BMC Anesthesiol] 2018 Jul 18; Vol. 18 (1), pp. 89. Date of Electronic Publication: 2018 Jul 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Lymphocytes*
Models, Biological*
Predictive Value of Tests*
Biomarkers/*blood
Sepsis/*diagnosis
Aged ; Female ; Humans ; Intensive Care Units/statistics & numerical data ; Male ; Middle Aged ; Prognosis ; Sepsis/blood ; Sepsis/mortality ; Survival Analysis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Vibrio alginolyticus T3SS effectors, Val1686 and Val1680, induce cell rounding, apoptosis and lysis of fish epithelial cells.
Autorzy:
Zhao Z; a Institute of Marine Biology, College of Oceanography, Hohai University , Nanjing , Jiangsu, PR China.; b Key Laboratory of Marine Bio-resources Sustainable Utilization, Key Laboratory of Applied Marine Biology of Guangdong Province, South China Sea Institute of Oceanology, Chinese Academy of Sciences , Guangzhou, Guangdong , PR China.
Liu J; a Institute of Marine Biology, College of Oceanography, Hohai University , Nanjing , Jiangsu, PR China.; c Paul G. Allen School for Global Animal Health, Washington State University , Pullman , WA , U.S.
Deng Y; b Key Laboratory of Marine Bio-resources Sustainable Utilization, Key Laboratory of Applied Marine Biology of Guangdong Province, South China Sea Institute of Oceanology, Chinese Academy of Sciences , Guangzhou, Guangdong , PR China.; d Key Laboratory of South China Sea Fishery Resources Exploitation & Utilization, Ministry of Agriculture, South China Sea Fisheries Research Institute, Chinese Academy of Fishery Sciences , Guangzhou, Guangdong , PR China.
Huang W; b Key Laboratory of Marine Bio-resources Sustainable Utilization, Key Laboratory of Applied Marine Biology of Guangdong Province, South China Sea Institute of Oceanology, Chinese Academy of Sciences , Guangzhou, Guangdong , PR China.
Ren C; b Key Laboratory of Marine Bio-resources Sustainable Utilization, Key Laboratory of Applied Marine Biology of Guangdong Province, South China Sea Institute of Oceanology, Chinese Academy of Sciences , Guangzhou, Guangdong , PR China.
Call DR; c Paul G. Allen School for Global Animal Health, Washington State University , Pullman , WA , U.S.
Hu C; b Key Laboratory of Marine Bio-resources Sustainable Utilization, Key Laboratory of Applied Marine Biology of Guangdong Province, South China Sea Institute of Oceanology, Chinese Academy of Sciences , Guangzhou, Guangdong , PR China.
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Źródło:
Virulence [Virulence] 2018 Jan 01; Vol. 9 (1), pp. 318-330.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Cell Shape/*drug effects
Cytotoxins/*metabolism
Epithelial Cells/*drug effects
Epithelial Cells/*physiology
Vibrio alginolyticus/*metabolism
Virulence Factors/*metabolism
Animals ; Cells, Cultured ; Cytotoxins/genetics ; Fishes ; Gene Deletion ; Type III Secretion Systems/metabolism ; Virulence Factors/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
mTOR signaling pathway is inhibited downstream of the cyclophilin D-mediated mitochondrial permeability transition in honokiol-triggered regulated necrosis.
Autorzy:
Tian W; Department of Surgical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Xiong J; Department of Surgical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Zhu S; Department of Surgical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Xu D; Department of Surgical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Shen H; Department of Medical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Deng Y; Department of Surgical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2016 Apr; Vol. 13 (4), pp. 3227-35. Date of Electronic Publication: 2016 Feb 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Biphenyl Compounds/*toxicity
Cyclophilins/*pharmacology
Lignans/*toxicity
Mitochondria/*metabolism
Signal Transduction/*drug effects
TOR Serine-Threonine Kinases/*metabolism
Amino Acid Chloromethyl Ketones/pharmacology ; Blotting, Western ; Caspase 3/metabolism ; Caspase 8/metabolism ; Peptidyl-Prolyl Isomerase F ; Cyclosporine/pharmacology ; HEK293 Cells ; Humans ; Imidazoles/pharmacology ; Indoles/pharmacology ; Nuclear Pore Complex Proteins/metabolism ; Permeability/drug effects ; RNA-Binding Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
MicroRNA-101 sensitizes hepatocellular carcinoma cells to doxorubicin-induced apoptosis via targeting Mcl-1.
Autorzy:
He H; Department of Surgical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Tian W; Department of Surgical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Chen H; Department of Surgical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Deng Y; Department of Surgical Oncology, The Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2016 Feb; Vol. 13 (2), pp. 1923-9. Date of Electronic Publication: 2015 Dec 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Carcinoma, Hepatocellular/*genetics
Carcinoma, Hepatocellular/*pathology
Doxorubicin/*pharmacology
Liver Neoplasms/*genetics
Liver Neoplasms/*pathology
MicroRNAs/*genetics
Base Sequence ; Cell Line, Tumor ; Down-Regulation/drug effects ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; MicroRNAs/metabolism ; Molecular Sequence Data ; Myeloid Cell Leukemia Sequence 1 Protein/genetics ; Myeloid Cell Leukemia Sequence 1 Protein/metabolism ; Up-Regulation/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Identification of low Ca(2+) stress-induced embryo apoptosis response genes in Arachis hypogaea by SSH-associated library lift (SSHaLL).
Autorzy:
Chen H; Fujian Provincial Key Laboratory of Crop Molecular and Cell Biology, Fujian Agriculture and Forestry University, Fuzhou, China.
Zhang C; Fujian Provincial Key Laboratory of Crop Molecular and Cell Biology, Fujian Agriculture and Forestry University, Fuzhou, China.
Cai TC; Fujian Provincial Key Laboratory of Crop Molecular and Cell Biology, Fujian Agriculture and Forestry University, Fuzhou, China.
Deng Y; Fujian Provincial Key Laboratory of Crop Molecular and Cell Biology, Fujian Agriculture and Forestry University, Fuzhou, China.
Zhou S; Fujian Provincial Key Laboratory of Crop Molecular and Cell Biology, Fujian Agriculture and Forestry University, Fuzhou, China.
Zheng Y; Fujian Provincial Key Laboratory of Crop Molecular and Cell Biology, Fujian Agriculture and Forestry University, Fuzhou, China.
Ma S; Fujian Provincial Key Laboratory of Crop Molecular and Cell Biology, Fujian Agriculture and Forestry University, Fuzhou, China.
Tang R; Cash Crops Research Institute, Guangxi Academy of Agricultural Sciences, Nanning, China.
Varshney RK; International Crops Research Institute for the Semi-Arid Tropics (ICRISAT), Patancheru, India.
Zhuang W; Fujian Provincial Key Laboratory of Crop Molecular and Cell Biology, Fujian Agriculture and Forestry University, Fuzhou, China.
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Źródło:
Plant biotechnology journal [Plant Biotechnol J] 2016 Feb; Vol. 14 (2), pp. 682-98. Date of Electronic Publication: 2015 Jun 16.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Library*
Genes, Plant*
Apoptosis/*drug effects
Arachis/*embryology
Calcium/*pharmacology
Nucleic Acid Hybridization/*methods
Seeds/*cytology
Stress, Physiological/*drug effects
Abscisic Acid/metabolism ; Apoptosis/genetics ; Arachis/cytology ; Arachis/drug effects ; Arachis/genetics ; Cytochrome P-450 Enzyme System/metabolism ; Down-Regulation/drug effects ; Down-Regulation/genetics ; Gene Expression Profiling ; Gene Expression Regulation, Plant/drug effects ; Molecular Sequence Annotation ; Phenotype ; Plants, Genetically Modified ; Real-Time Polymerase Chain Reaction ; Seeds/drug effects ; Stress, Physiological/genetics ; Nicotiana/genetics ; Up-Regulation/drug effects ; Up-Regulation/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
EBP50 induces apoptosis in macrophages by upregulating nitric oxide production to eliminate intracellular Mycobacterium tuberculosis.
Autorzy:
Guo Y; Department of Clinical Laboratory, The First Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Deng Y; Department of Clinical Laboratory, The First Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Huang Z; Department of Clinical Laboratory, The First Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Luo Q; Department of Clinical Laboratory, The First Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Peng Y; Department of Tuberculosis, Jiangxi Chest Hospital, Nanchang, 330006, China.
Chen J; Department of Clinical Laboratory, The First Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Jiang H; Department of Clinical Laboratory, The First Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Ye J; Department of Clinical Laboratory, The First Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Li J; Department of Clinical Laboratory, The First Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
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Źródło:
Scientific reports [Sci Rep] 2016 Jan 05; Vol. 6, pp. 18961. Date of Electronic Publication: 2016 Jan 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/genetics
Apoptosis*/immunology
Macrophages/*microbiology
Macrophages/*physiology
Mycobacterium tuberculosis/*immunology
Nitric Oxide/*biosynthesis
Phosphoproteins/*metabolism
Sodium-Hydrogen Exchangers/*metabolism
Animals ; Caspase 3/metabolism ; Cell Line ; Cytotoxicity, Immunologic ; Flow Cytometry ; Gene Expression Regulation ; Gene Knockdown Techniques ; Humans ; Mice ; Nitric Oxide Synthase Type II/metabolism ; Phagocytosis/immunology ; Phagosomes/metabolism ; Phosphoproteins/genetics ; Sodium-Hydrogen Exchangers/genetics ; Tuberculosis/genetics ; Tuberculosis/immunology ; Tuberculosis/metabolism ; Tuberculosis/microbiology ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
A critical role of mir-199a in the cell biological behaviors of colorectal cancer.
Autorzy:
Ye H; Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical College, Zhanjiang, Guangdong, 524023, China. yehua_.
Pang L; Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical College, Zhanjiang, Guangdong, 524023, China. pangliping.
Wu Q; Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical College, Zhanjiang, Guangdong, 524023, China. wu_qiong_.
Zhu Y; Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical College, Zhanjiang, Guangdong, 524023, China. drzheng_.
Guo C; Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical College, Zhanjiang, Guangdong, 524023, China. .
Deng Y; Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical College, Zhanjiang, Guangdong, 524023, China. .
Zheng X; Guangdong Key Laboratory for Research and Development of Natural Drugs, Guangdong Medical College, Zhanjiang, Guangdong, 524023, China. zheng_.
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Źródło:
Diagnostic pathology [Diagn Pathol] 2015 Jun 12; Vol. 10, pp. 65. Date of Electronic Publication: 2015 Jun 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*genetics
Cell Proliferation/*genetics
Colorectal Neoplasms/*genetics
MicroRNAs/*biosynthesis
Neoplasm Invasiveness/*genetics
Blotting, Western ; Cell Movement/genetics ; Colorectal Neoplasms/pathology ; Down-Regulation ; Female ; Flow Cytometry ; Humans ; Male ; Middle Aged ; Real-Time Polymerase Chain Reaction ; Transfection
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Melatonin antagonizes Mn-induced oxidative injury through the activation of keap1-Nrf2-ARE signaling pathway in the striatum of mice.
Autorzy:
Deng Y; Department of Environmental Health, School of Public Health, China Medical University, Shenyang, 110001, Liaoning, People's Republic of China, .
Zhu J
Mi C
Xu B
Jiao C
Li Y
Xu D
Liu W
Xu Z
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Źródło:
Neurotoxicity research [Neurotox Res] 2015 Feb; Vol. 27 (2), pp. 156-71. Date of Electronic Publication: 2014 Oct 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antioxidants/*pharmacology
Apoptosis/*drug effects
Chlorides/*toxicity
Corpus Striatum/*drug effects
Corpus Striatum/*metabolism
Melatonin/*pharmacology
Signal Transduction/*drug effects
Adaptor Proteins, Signal Transducing/metabolism ; Animals ; Antioxidant Response Elements ; Body Weight/drug effects ; Cytoskeletal Proteins/metabolism ; Female ; Kelch-Like ECH-Associated Protein 1 ; Locomotion/drug effects ; Male ; Manganese Compounds ; Mice ; NF-E2-Related Factor 2/metabolism ; Neurons/drug effects ; Neurons/metabolism ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
HIV-1 Tat-mediated apoptosis in human blood-retinal barrier-associated cells.
Autorzy:
Che X; Department of Ophthalmology, Ninth People's Hospital, Shanghai Jiao Tong University, Shanghai, China.
He F; Department of Ophthalmology, Ninth People's Hospital, Shanghai Jiao Tong University, Shanghai, China.
Deng Y; Department of Ophthalmology, Ninth People's Hospital, Shanghai Jiao Tong University, Shanghai, China.
Xu S; Department of Ophthalmology, Ninth People's Hospital, Shanghai Jiao Tong University, Shanghai, China.
Fan X; Department of Ophthalmology, Ninth People's Hospital, Shanghai Jiao Tong University, Shanghai, China.
Gu P; Department of Ophthalmology, Ninth People's Hospital, Shanghai Jiao Tong University, Shanghai, China.
Wang Z; Department of Ophthalmology, Ninth People's Hospital, Shanghai Jiao Tong University, Shanghai, China.
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Źródło:
PloS one [PLoS One] 2014 Apr 16; Vol. 9 (4), pp. e95420. Date of Electronic Publication: 2014 Apr 16 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Endothelial Cells/*drug effects
HIV-1/*chemistry
Retinal Pigment Epithelium/*drug effects
tat Gene Products, Human Immunodeficiency Virus/*pharmacology
Antibodies, Neutralizing/pharmacology ; Blood-Retinal Barrier/cytology ; Blood-Retinal Barrier/drug effects ; Blood-Retinal Barrier/metabolism ; Cell Line ; Cytochromes c/genetics ; Cytochromes c/metabolism ; Dose-Response Relationship, Drug ; Endothelial Cells/cytology ; Endothelial Cells/metabolism ; Gene Expression Regulation ; Humans ; Mitochondria/drug effects ; Mitochondria/metabolism ; Models, Biological ; Organ Specificity ; Receptors, N-Methyl-D-Aspartate/genetics ; Receptors, N-Methyl-D-Aspartate/metabolism ; Retinal Pigment Epithelium/cytology ; Retinal Pigment Epithelium/metabolism ; Signal Transduction ; bcl-2 Homologous Antagonist-Killer Protein/genetics ; bcl-2 Homologous Antagonist-Killer Protein/metabolism ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism ; tat Gene Products, Human Immunodeficiency Virus/antagonists & inhibitors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
BMI1 polycomb group protein acts as a master switch for growth and death of tumor cells: regulates TCF4-transcriptional factor-induced BCL2 signaling.
Autorzy:
Siddique HR; Department of Molecular Chemoprevention and Therapeutics, The Hormel Institute, University of Minnesota, Austin, Minnesota, United States of America.
Parray A
Tarapore RS
Wang L
Mukhtar H
Karnes RJ
Deng Y
Konety BR
Saleem M
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Źródło:
PloS one [PLoS One] 2013 May 06; Vol. 8 (5), pp. e60664. Date of Electronic Publication: 2013 May 06 (Print Publication: 2013).
Typ publikacji:
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:
Apoptosis*
Basic Helix-Loop-Helix Leucine Zipper Transcription Factors/*metabolism
Polycomb Repressive Complex 1/*physiology
Prostatic Neoplasms/*metabolism
Proto-Oncogene Proteins c-bcl-2/*metabolism
Transcription Factors/*metabolism
Animals ; Antineoplastic Agents/pharmacology ; Cell Proliferation/drug effects ; Cellular Senescence ; Cisplatin/pharmacology ; Docetaxel ; Drug Resistance, Neoplasm ; Gene Expression Regulation, Neoplastic ; HT29 Cells ; Humans ; Male ; Metribolone/pharmacology ; Mice ; Mice, Nude ; Neoplasm Transplantation ; Promoter Regions, Genetic ; Prostate/metabolism ; Prostatic Neoplasms/pathology ; Protein Binding ; Proto-Oncogene Proteins c-bcl-2/genetics ; Signal Transduction ; Taxoids/pharmacology ; Testosterone Congeners/pharmacology ; Transcription Factor 4
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Protective effects of grape seed procyanidin extract against nickel sulfate-induced apoptosis and oxidative stress in rat testes.
Autorzy:
Su L; School of Life Sciences, Lanzhou University, Lanzhou, P.R. China.
Deng Y
Zhang Y
Li C
Zhang R
Sun Y
Zhang K
Li J
Yao S
Pokaż więcej
Źródło:
Toxicology mechanisms and methods [Toxicol Mech Methods] 2011 Jul; Vol. 21 (6), pp. 487-94. Date of Electronic Publication: 2011 Mar 21.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antioxidants/*therapeutic use
Apoptosis/*drug effects
Grape Seed Extract/*therapeutic use
Nickel/*toxicity
Oxidative Stress/*drug effects
Proanthocyanidins/*therapeutic use
Testis/*drug effects
Animals ; Antioxidants/administration & dosage ; Dose-Response Relationship, Drug ; Grape Seed Extract/administration & dosage ; Heavy Metal Poisoning ; Male ; Metals, Heavy/metabolism ; Nickel/administration & dosage ; Oxidants/administration & dosage ; Oxidants/toxicity ; Oxidoreductases/metabolism ; Phytotherapy ; Poisoning/metabolism ; Poisoning/prevention & control ; Proanthocyanidins/administration & dosage ; Proto-Oncogene Proteins c-kit/metabolism ; Random Allocation ; Rats ; Rats, Wistar ; Reactive Oxygen Species/metabolism ; S Phase/drug effects ; Sperm Motility/drug effects ; Spermatozoa/drug effects ; Testis/metabolism ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 1-20 z 206

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