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Tytuł:
High-fat, high-sucrose, and combined high-fat/high-sucrose diets effects in oxidative stress and inflammation in male rats under presence or absence of obesity.
Autorzy:
Kobi JBBS; Postgraduate Program in Nutrition and Health, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
Matias AM; Postgraduate Program in Nutrition and Health, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
Gasparini PVF; Postgraduate Program in Nutrition and Health, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
Torezani-Sales S; Postgraduate Program in Physiological Science, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
Madureira AR; Postgraduate Program in Nutrition and Health, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
da Silva DS; Postgraduate Program in Physical Education, Center of Physical Education and Sports, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
Correa CR; The Medical School, São Paulo State University, Botucatu, São Paulo, Brazil.
Garcia JL; The Medical School, São Paulo State University, Botucatu, São Paulo, Brazil.
Haese D; University of Vila Velha, Vila Velha, Espírito Santo, Brazil.
Nogueira BV; Department of Morphology, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
de Assis ALEM; Department of Morphology, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
Lima-Leopoldo AP; Postgraduate Program in Nutrition and Health, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.; Center of Physical Education and Sports, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
Leopoldo AS; Postgraduate Program in Nutrition and Health, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.; Postgraduate Program in Physiological Science, Health Sciences Center, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.; Center of Physical Education and Sports, Federal University of Espírito Santo, Vitória, Espírito Santo, Brazil.
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Źródło:
Physiological reports [Physiol Rep] 2023 Apr; Vol. 11 (7), pp. e15635.
Typ publikacji:
Evaluation Study; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Diet, Carbohydrate Loading*/adverse effects
Diet, High-Fat*/adverse effects
Dietary Sucrose*/adverse effects
Inflammation*/etiology
Inflammation*/metabolism
Obesity*/etiology
Obesity*/metabolism
Oxidative Stress*/physiology
Animals ; Male ; Rats ; Disease Models, Animal ; Rats, Wistar
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Increased Weight Gain and Insulin Resistance in HF-Fed PLTP Deficient Mice Is Related to Altered Inflammatory Response and Plasma Transport of Gut-Derived LPS.
Autorzy:
Lebrun LJ; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.; Institut Agro Dijon, 1 Esplanade Erasme, 21000 Dijon, France.
Pallot G; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.
Nguyen M; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.; Department of Anesthesiology and Intensive Care, Dijon University Hospital, 21000 Dijon, France.
Tavernier A; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.; Institut Agro Dijon, 1 Esplanade Erasme, 21000 Dijon, France.
Dusuel A; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.
Pilot T; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.
Deckert V; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.
Dugail I; Faculté de Médecine Pitié-Salpêtrière, UMR1269, 75000 Paris, France.
Le Guern N; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.
Pais De Barros JP; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.; Lipidomic Analytic Plate-Forme, UBFC, Bâtiment B3, 21000 Dijon, France.
Benkhaled A; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.
Choubley H; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.; Lipidomic Analytic Plate-Forme, UBFC, Bâtiment B3, 21000 Dijon, France.
Lagrost L; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.
Masson D; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.; Laboratory of Clinical Chemistry, François Mitterrand University Hospital, 21000 Dijon, France.
Gautier T; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.
Grober J; INSERM, LNC UMR1231, Université Bourgogne Franche-Comté, 21000 Dijon, France.; FCS Bourgogne-Franche Comté, LipSTIC LabEx, 21000 Dijon, France.; Institut Agro Dijon, 1 Esplanade Erasme, 21000 Dijon, France.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Oct 30; Vol. 23 (21). Date of Electronic Publication: 2022 Oct 30.
Typ publikacji:
Journal Article
MeSH Terms:
Diet, High-Fat*/adverse effects
Endotoxemia*/chemically induced
Endotoxemia*/metabolism
Inflammation*/chemically induced
Inflammation*/metabolism
Insulin Resistance*/physiology
Weight Gain*/physiology
Animals ; Mice ; Lipopolysaccharides/adverse effects ; Lipoproteins/metabolism ; Obesity/etiology ; Phospholipid Transfer Proteins/genetics ; Phospholipid Transfer Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
High-Fat, Western-Style Diet, Systemic Inflammation, and Gut Microbiota: A Narrative Review.
Autorzy:
Malesza IJ; Department of Pediatric Gastroenterology and Metabolic Diseases, Poznan University of Medical Sciences, 61-701 Poznań, Poland.
Malesza M; Department of Physiology, Poznan University of Medical Sciences, 61-701 Poznań, Poland.
Walkowiak J; Department of Pediatric Gastroenterology and Metabolic Diseases, Poznan University of Medical Sciences, 61-701 Poznań, Poland.
Mussin N; Department of General Surgery, West Kazakhstan Marat Ospanov Medical University, Aktobe 030012, Kazakhstan.
Walkowiak D; Department of Organization and Management in Health Care, Poznan University of Medical Sciences, 61-701 Poznań, Poland.
Aringazina R; Department of Internal Diseases No. 1, West Kazakhstan Marat Ospanov Medical University, Aktobe 030012, Kazakhstan.
Bartkowiak-Wieczorek J; Department of Physiology, Poznan University of Medical Sciences, 61-701 Poznań, Poland.
Mądry E; Department of Physiology, Poznan University of Medical Sciences, 61-701 Poznań, Poland.
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Źródło:
Cells [Cells] 2021 Nov 14; Vol. 10 (11). Date of Electronic Publication: 2021 Nov 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Diet, High-Fat*
Diet, Western*
Gastrointestinal Microbiome*
Inflammation/*pathology
Animals ; Dysbiosis/microbiology ; Endoplasmic Reticulum Stress ; Humans
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Storage conditions of high-fat diets affect pulmonary inflammation.
Autorzy:
Kokoszynska M; Department of Medicine, Pulmonary Disease and Critical Care Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.; Vermont Lung Center, Burlington, Vermont, USA.
Ubags ND; Faculty of Biology and Medicine, University of Lausanne, Service de Pneumologie, CHUV, Lausanne, Switzerland.
Bivona JJ 3rd; Department of Medicine, Pulmonary Disease and Critical Care Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.; Vermont Lung Center, Burlington, Vermont, USA.; Cellular, Molecular, and Biomedical Sciences Doctoral Program, University of Vermont, Burlington, Vermont, USA.
Ventrone S; Department of Medicine, Pulmonary Disease and Critical Care Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.; Vermont Lung Center, Burlington, Vermont, USA.
Reed LF; Department of Medicine, Pulmonary Disease and Critical Care Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.; Vermont Lung Center, Burlington, Vermont, USA.
Dixon AE; Department of Medicine, Pulmonary Disease and Critical Care Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.; Vermont Lung Center, Burlington, Vermont, USA.
Wargo MJ; Department of Medicine, Pulmonary Disease and Critical Care Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.; Vermont Lung Center, Burlington, Vermont, USA.
Poynter ME; Department of Medicine, Pulmonary Disease and Critical Care Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.; Vermont Lung Center, Burlington, Vermont, USA.
Suratt BT; Department of Medicine, Pulmonary Disease and Critical Care Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.; Vermont Lung Center, Burlington, Vermont, USA.
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Źródło:
Physiological reports [Physiol Rep] 2021 Nov; Vol. 9 (22), pp. e15116.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Animal Feed*
Diet, High-Fat*
Food Storage*
Temperature*
Acute Lung Injury/*metabolism
Inflammation/*metabolism
Malondialdehyde/*metabolism
Obesity/*metabolism
Pneumonia/*metabolism
Acute Lung Injury/chemically induced ; Acute Lung Injury/microbiology ; Animals ; Diet, Fat-Restricted ; Disease Models, Animal ; Gastrointestinal Microbiome ; Inflammation/microbiology ; Lipid Metabolism ; Lipopolysaccharides/toxicity ; Mice ; Mice, Inbred C57BL ; Obesity/microbiology ; Pneumonia/chemically induced ; Pneumonia/microbiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Delphinidin-3- O -glucoside, an active compound of Hibiscus sabdariffa calyces, inhibits oxidative stress and inflammation in rabbits with atherosclerosis.
Autorzy:
Sun B; Department of Vascular Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences, Beijing, China.; Department of Vascular Surgery, Weifang People's Hospital, Weifang, China.
Li F; Department of Vascular Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences, Beijing, China.
Zhang X; Department of Vascular Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences, Beijing, China.
Wang W; Department of Vascular Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences, Beijing, China.
Shao J; Department of Vascular Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences, Beijing, China.
Zheng Y; Department of Vascular Surgery, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences, Beijing, China.
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Źródło:
Pharmaceutical biology [Pharm Biol] 2022 Dec; Vol. 60 (1), pp. 247-254.
Typ publikacji:
Journal Article
MeSH Terms:
Anthocyanins/*pharmacology
Atherosclerosis/*drug therapy
Glucosides/*pharmacology
Hibiscus/*chemistry
Inflammation/*drug therapy
Animals ; Anthocyanins/isolation & purification ; Atherosclerosis/pathology ; Diet, High-Fat ; Disease Models, Animal ; Glucosides/isolation & purification ; Inflammation/pathology ; Lipid Metabolism/drug effects ; Male ; Oxidative Stress/drug effects ; Rabbits
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Adipose Tissue-Derived CCL5 Enhances Local Pro-Inflammatory Monocytic MDSCs Accumulation and Inflammation via CCR5 Receptor in High-Fat Diet-Fed Mice.
Autorzy:
Chan PC; Department of Physiology & Biophysics, National Defense Medical Center (NDMC), Taipei 114, Taiwan.
Lu CH; Division of Endocrinology and Metabolism, Department of Internal Medicine, Tri-Service General Hospital, NDMC, Taipei 114, Taiwan.
Chien HC; Department of Physiology & Biophysics, National Defense Medical Center (NDMC), Taipei 114, Taiwan.
Tian YF; Department of Surgery, Chi Mei Medical Center, Tainan 717, Taiwan.
Hsieh PS; Department of Physiology & Biophysics, National Defense Medical Center (NDMC), Taipei 114, Taiwan.; Graduate Institute of Medical Sciences, NDMC, Taipei 114, Taiwan.; Department of Medical Research, Tri-Service General Hospital, Taipei 114, Taiwan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Nov 17; Vol. 23 (22). Date of Electronic Publication: 2022 Nov 17.
Typ publikacji:
Journal Article
MeSH Terms:
Adipose Tissue*/chemistry
Adipose Tissue*/metabolism
Inflammation*/metabolism
Myeloid-Derived Suppressor Cells*/metabolism
Receptors, CCR5*/genetics
Receptors, CCR5*/metabolism
Chemokine CCL5*/metabolism
Chemokine CCL5*/pharmacology
Animals ; Mice ; Diet, High-Fat/adverse effects ; Insulin Resistance/genetics ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Obese ; Obesity/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Papain Ameliorates Lipid Accumulation and Inflammation in High-Fat Diet-Induced Obesity Mice and 3T3-L1 Adipocytes via AMPK Activation.
Autorzy:
Kang YM; Department of Pharmacology, College of Korean Medicine, Sangji University, Wonju 26339, Gangwon-do, Korea.
Kang HA; Department of Pharmacology, College of Korean Medicine, Sangji University, Wonju 26339, Gangwon-do, Korea.
Cominguez DC; Department of Pharmacology, College of Korean Medicine, Sangji University, Wonju 26339, Gangwon-do, Korea.
Kim SH; Department of Obstetrics and Gynecology, College of Korean Medicine, Sangji University, Wonju 26339, Gangwon-do, Korea.
An HJ; Department of Pharmacology, College of Korean Medicine, Sangji University, Wonju 26339, Gangwon-do, Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Sep 14; Vol. 22 (18). Date of Electronic Publication: 2021 Sep 14.
Typ publikacji:
Journal Article
MeSH Terms:
Diet, High-Fat*
AMP-Activated Protein Kinases/*metabolism
Adipocytes/*pathology
Inflammation/*pathology
Lipid Metabolism/*drug effects
Obesity/*metabolism
Obesity/*pathology
Papain/*pharmacology
3T3-L1 Cells ; Adipocytes/drug effects ; Adipocytes/metabolism ; Adipogenesis/drug effects ; Adipokines/metabolism ; Adiposity/drug effects ; Animals ; Biomarkers/metabolism ; Body Weight/drug effects ; Enzyme Activation/drug effects ; Fatty Liver/pathology ; Forkhead Box Protein O1/metabolism ; Hypertrophy ; Lipids/blood ; Liver/drug effects ; Liver/pathology ; Macrophages/drug effects ; Macrophages/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Obese ; Organ Size/drug effects ; Sirtuin 1/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Maternal exposure to high-fat diet during pregnancy and lactation predisposes normal weight offspring mice to develop hepatic inflammation and insulin resistance.
Autorzy:
Saengnipanthkul S; Division of Nutrition, Department of Pediatrics, Faculty of Medicine, Khon Kaen University, Khon Kaen, Thailand.; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Noh HL; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Friedline RH; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Suk S; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Choi S; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Acosta NK; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Tran DA; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Hu X; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Inashima K; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Kim AM; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.
Lee KW; Department of Agricultural Biotechnology, College of Agricultural and Life Sciences, Seoul National University, Seoul, South Korea.
Kim JK; Program in Molecular Medicine, University of Massachusetts Medical School, Worcester, MA, USA.; Division of Endocrinology, Metabolism, and Diabetes, Department of Medicine, University of Massachusetts Medical School, Worcester, MA, USA.; Department of Agricultural Biotechnology, College of Agricultural and Life Sciences, Seoul National University, Seoul, South Korea.
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Źródło:
Physiological reports [Physiol Rep] 2021 Mar; Vol. 9 (6), pp. e14811.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Diet, High-Fat*
Insulin Resistance*
Maternal Nutritional Physiological Phenomena*
Inflammation/*metabolism
Liver Diseases/*metabolism
Prenatal Exposure Delayed Effects/*metabolism
Animals ; Female ; Gene Expression ; Inflammation/complications ; Lactation ; Liver Diseases/complications ; Male ; Mice, Inbred C57BL ; Pregnancy ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Thymoquinone Protects Neurons in the Cerebellum of Rats through Mitigating Oxidative Stress and Inflammation Following High-Fat Diet Supplementation.
Autorzy:
Alrafiah A; Department of Medical Laboratory Technology, Faculty of Applied Medical Sciences, King Abdulaziz University, Jeddah 21589, Saudi Arabia.
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Źródło:
Biomolecules [Biomolecules] 2021 Jan 27; Vol. 11 (2). Date of Electronic Publication: 2021 Jan 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Diet, High-Fat*
Benzoquinones/*pharmacology
Cerebellum/*drug effects
Inflammation/*metabolism
Neurons/*drug effects
Animals ; Antioxidants/pharmacology ; Body Weight ; Brain ; Cerebellum/metabolism ; Cytokines/metabolism ; Dietary Supplements ; Glutathione/metabolism ; Malondialdehyde/metabolism ; Neurons/metabolism ; Obesity ; Overweight ; Oxidative Stress ; Rats ; Rats, Wistar ; Superoxide Dismutase/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Antiobesity efficacy of asiatic acid: down-regulation of adipogenic and inflammatory processes in high fat diet induced obese rats.
Autorzy:
Uddandrao VVS; Centre for Biological Sciences, Department of Biochemistry, K.S. Rangasamy College of Arts and Science (Autonomous), Tiruchengode, India.
Rameshreddy P; Centre for Biological Sciences, Department of Biochemistry, K.S. Rangasamy College of Arts and Science (Autonomous), Tiruchengode, India.
Brahmanaidu P; Animal Physiology and Biochemistry Laboratory, ICMR-National Animal Resource Facility for Biomedical Research, Hyderabad, India.
Ponnusamy P; Department of Botany, Bharathiar University, Coimbatore, India.
Balakrishnan S; College of Applied Medical Sciences, Majmaah University, Al Majmaah, The Kingdom of Saudi Arabia.; Department of Bio-Medical Engineering, Velalar College of Engineering and Technology, Thindal, Tamil Nadu, India.
Ramavat RN; Animal Physiology and Biochemistry Laboratory, ICMR-National Animal Resource Facility for Biomedical Research, Hyderabad, India.
Swapna K; Centre for Biological Sciences, Department of Biochemistry, K.S. Rangasamy College of Arts and Science (Autonomous), Tiruchengode, India.
Pothani S; Animal Physiology and Biochemistry Laboratory, ICMR-National Animal Resource Facility for Biomedical Research, Hyderabad, India.
Nemani H; Animal Physiology and Biochemistry Laboratory, ICMR-National Animal Resource Facility for Biomedical Research, Hyderabad, India.
Meriga B; Department of Biochemistry, Animal Physiology and Biochemistry Laboratory, Sri Venkateswara University, Tirupati, India.
Vadivukkarasi S; Centre for Biological Sciences, Department of Biochemistry, K.S. Rangasamy College of Arts and Science (Autonomous), Tiruchengode, India.
P R N; Centre for Biological Sciences, Department of Biochemistry, K.S. Rangasamy College of Arts and Science (Autonomous), Tiruchengode, India.
Ganapathy S; Centre for Biological Sciences, Department of Biochemistry, K.S. Rangasamy College of Arts and Science (Autonomous), Tiruchengode, India.
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Źródło:
Archives of physiology and biochemistry [Arch Physiol Biochem] 2020 Dec; Vol. 126 (5), pp. 453-462. Date of Electronic Publication: 2019 Feb 10.
Typ publikacji:
Journal Article
MeSH Terms:
Adipogenesis*
Diet, High-Fat*
Biomarkers/*metabolism
Inflammation/*drug therapy
Obesity/*complications
Pentacyclic Triterpenes/*pharmacology
Animals ; Inflammation/etiology ; Inflammation/metabolism ; Inflammation/pathology ; Lipid Metabolism ; Male ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Caloric restriction-induced weight loss with a high-fat diet does not fully recover visceral adipose tissue inflammation in previously obese C57BL/6 mice.
Autorzy:
Rodrigues MOM; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
Evangelista-Silva PH; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
Neves NN; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
Moreno LG; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
Santos CS; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
Rocha KLS; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
Ottone VO; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
Batista-da-Silva B; Instituto de Biotecnologia, Universidade Federal de Uberlândia, Uberlândia, MG 38408-100, Brazil.
Dias-Peixoto MF; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
Magalhães FC; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
Esteves EA; Programa Multicêntrico de Pós-graduação em Ciências Fisiológicas, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, MG 39100-000, Brazil.
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Źródło:
Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme [Appl Physiol Nutr Metab] 2020 Dec; Vol. 45 (12), pp. 1353-1359. Date of Electronic Publication: 2020 Jun 23.
Typ publikacji:
Journal Article
MeSH Terms:
Caloric Restriction*
Diet, High-Fat*
Weight Loss*
Inflammation/*pathology
Intra-Abdominal Fat/*pathology
Adipokines ; Adiposity ; Animals ; Diet, Fat-Restricted ; Male ; Mice, Inbred C57BL ; Obesity
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
P2X 4 deficiency reduces atherosclerosis and plaque inflammation in mice.
Autorzy:
Peikert A; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
König S; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Suchanek D; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Rofa K; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Schäfer I; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Dimanski D; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Karnbrock L; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Bulatova K; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Engelmann J; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Hoppe N; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Wadle C; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Heidt T; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Albrecht P; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
von Garlen S; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Härdtner C; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Hilgendorf I; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Wolf D; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
von Zur Mühlen C; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Bode C; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Zirlik A; Department of Cardiology, Medical University of Graz, Graz, Austria.
Duerschmied D; First Department of Medicine (Cardiology), Medical Faculty Mannheim, University Medical Centre Mannheim, Mannheim, Germany.
Merz J; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany.
Stachon P; Department of Cardiology and Angiology I, University Heart Center Freiburg, Medical Faculty, University of Freiburg, Hugstetter Straße 55, 79106, Freiburg, Germany. .; First Department of Medicine (Cardiology), Medical Faculty Mannheim, University Medical Centre Mannheim, Mannheim, Germany. .
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Źródło:
Scientific reports [Sci Rep] 2022 Feb 18; Vol. 12 (1), pp. 2801. Date of Electronic Publication: 2022 Feb 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Atherosclerosis/*genetics
Inflammation/*genetics
Receptors, LDL/*genetics
Receptors, Purinergic P2X4/*genetics
Adenosine Triphosphate/metabolism ; Animals ; Atherosclerosis/pathology ; Blood Vessels/drug effects ; Blood Vessels/pathology ; Chemokine CCL2/genetics ; Chemokine CXCL1/genetics ; Cholesterol/pharmacology ; Diet, High-Fat/adverse effects ; Endarterectomy, Carotid ; Humans ; Inflammation/pathology ; Interleukin-6/genetics ; Macrophages/metabolism ; Macrophages/pathology ; Mice ; Mice, Knockout ; NLR Family, Pyrin Domain-Containing 3 Protein/genetics ; Plaque, Atherosclerotic/genetics ; Plaque, Atherosclerotic/pathology ; Signal Transduction/genetics ; Tumor Necrosis Factor-alpha/genetics ; Vascular Cell Adhesion Molecule-1/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Microglial and peripheral immune priming is partially sexually dimorphic in adolescent mouse offspring exposed to maternal high-fat diet.
Autorzy:
Bordeleau M; Integrated Program in Neuroscience, McGill University, Montreal, QC, Canada.; Axe neurosciences, Centre de recherche du CHU de Québec-Université Laval, Québec, QC, Canada.; Cerebral Imaging Center, Douglas Mental Health University Institute, McGill University, Montréal, QC, Canada.
Lacabanne C; Integrated Program in Neuroscience, McGill University, Montreal, QC, Canada.
Fernández de Cossío L; Department of Neurosciences, University of California, La Jolla, San Diego, CA, USA.
Vernoux N; Axe neurosciences, Centre de recherche du CHU de Québec-Université Laval, Québec, QC, Canada.
Savage JC; Axe neurosciences, Centre de recherche du CHU de Québec-Université Laval, Québec, QC, Canada.; Département de médecine moléculaire, Université Laval, Québec, QC, Canada.
González-Ibáñez F; Axe neurosciences, Centre de recherche du CHU de Québec-Université Laval, Québec, QC, Canada.; Département de médecine moléculaire, Université Laval, Québec, QC, Canada.
Tremblay MÈ; Axe neurosciences, Centre de recherche du CHU de Québec-Université Laval, Québec, QC, Canada. .; Département de médecine moléculaire, Université Laval, Québec, QC, Canada. .; Department of Neurology and Neurosurgery, McGill University, Montréal, QC, Canada. .; Division of Medical Sciences, University of Victoria, Victoria, BC, Canada. .; Department of Biochemistry and Molecular Biology, University of British Columbia, Vancouver, BC, Canada. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2020 Sep 05; Vol. 17 (1), pp. 264. Date of Electronic Publication: 2020 Sep 05.
Typ publikacji:
Journal Article
MeSH Terms:
Diet, High-Fat*
Hippocampus/*pathology
Inflammation/*pathology
Maternal Nutritional Physiological Phenomena/*physiology
Microglia/*pathology
Prenatal Exposure Delayed Effects/*metabolism
Adolescent ; Animals ; CX3C Chemokine Receptor 1/metabolism ; Cell Shape/physiology ; Female ; Hippocampus/drug effects ; Hippocampus/metabolism ; Humans ; Inflammation/metabolism ; Interleukin-6/blood ; Lipopolysaccharides/pharmacology ; Male ; Membrane Glycoproteins/metabolism ; Membrane Proteins/metabolism ; Mice ; Microglia/drug effects ; Microglia/metabolism ; Neurons/drug effects ; Neurons/metabolism ; Neurons/pathology ; Pregnancy ; Receptors, Immunologic/metabolism ; Sex Factors ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Effect of a freeze-dried coffee solution in a high-fat diet-induced obesity model in rats: Impact on inflammatory response, lipid profile, and gut microbiota.
Autorzy:
Cavalcanti MH; Postgraduate Program in Human Nutrition, Faculty of Health Sciences, Campus Universitário Darcy Ribeiro, Universidade de Brasília, Brasília, Distrito Federal, Brazil.
Roseira JPS; Department of Animal Science, Universidade Federal de Viçosa, Viçosa, Minas Gerais, Brazil.
Leandro EDS; Postgraduate Program in Human Nutrition, Faculty of Health Sciences, Campus Universitário Darcy Ribeiro, Universidade de Brasília, Brasília, Distrito Federal, Brazil.; Department of Nutrition, Faculty of Health Sciences, Campus Universitário Darcy Ribeiro, Universidade de Brasília, Brasília, Distrito Federal, Brazil.
Arruda SF; Postgraduate Program in Human Nutrition, Faculty of Health Sciences, Campus Universitário Darcy Ribeiro, Universidade de Brasília, Brasília, Distrito Federal, Brazil.; Department of Nutrition, Faculty of Health Sciences, Campus Universitário Darcy Ribeiro, Universidade de Brasília, Brasília, Distrito Federal, Brazil.
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Źródło:
PloS one [PLoS One] 2022 Jan 26; Vol. 17 (1), pp. e0262270. Date of Electronic Publication: 2022 Jan 26 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Coffee*
Gastrointestinal Microbiome/*drug effects
Inflammation/*metabolism
Lipid Metabolism/*drug effects
Lipids/*blood
Obesity/*metabolism
Animals ; Body Weight/drug effects ; Diet, High-Fat/adverse effects ; Eating/drug effects ; Male ; Obesity/etiology ; Rats ; Rats, Wistar
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Sitagliptin Is More Effective Than Gliclazide in Preventing  Pro-Fibrotic and Pro-Inflammatory Changes in a Rodent Model of Diet-Induced Non-Alcoholic Fatty Liver Disease.
Autorzy:
Ren J; Greg Brown Diabetes and Endocrinology Research Laboratories, Sydney Medical School (Central), Faculty of Medicine and Health, The University of Sydney, Camperdown, NSW 2006, Australia.
Wang X; Greg Brown Diabetes and Endocrinology Research Laboratories, Sydney Medical School (Central), Faculty of Medicine and Health, The University of Sydney, Camperdown, NSW 2006, Australia.
Yee C; Greg Brown Diabetes and Endocrinology Research Laboratories, Sydney Medical School (Central), Faculty of Medicine and Health, The University of Sydney, Camperdown, NSW 2006, Australia.
Gorrell MD; Liver Enzymes in Metabolism and Inflammation Program, Centenary Institute, The University of Sydney, Newtown, NSW 2042, Australia.; A.W. Morrow Gastroenterology and Liver Centre, Royal Prince Alfred Hospital, Camperdown, NSW 2050, Australia.
McLennan SV; Greg Brown Diabetes and Endocrinology Research Laboratories, Sydney Medical School (Central), Faculty of Medicine and Health, The University of Sydney, Camperdown, NSW 2006, Australia.; Department of Endocrinology, Royal Prince Alfred Hospital, Camperdown, NSW 2050, Australia.; New South Wales Health Pathology (Eastern), Camperdown, NSW 2050, Australia.
Twigg SM; Greg Brown Diabetes and Endocrinology Research Laboratories, Sydney Medical School (Central), Faculty of Medicine and Health, The University of Sydney, Camperdown, NSW 2006, Australia.; Department of Endocrinology, Royal Prince Alfred Hospital, Camperdown, NSW 2050, Australia.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2022 Jan 22; Vol. 27 (3). Date of Electronic Publication: 2022 Jan 22.
Typ publikacji:
Comparative Study; Journal Article
MeSH Terms:
Diet, High-Fat/*adverse effects
Gliclazide/*pharmacology
Inflammation/*prevention & control
Liver Cirrhosis/*prevention & control
Non-alcoholic Fatty Liver Disease/*drug therapy
Sitagliptin Phosphate/*pharmacology
Animals ; Hypoglycemic Agents/pharmacology ; Inflammation/etiology ; Inflammation/pathology ; Liver Cirrhosis/etiology ; Liver Cirrhosis/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Non-alcoholic Fatty Liver Disease/etiology ; Non-alcoholic Fatty Liver Disease/metabolism ; Non-alcoholic Fatty Liver Disease/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Apolipoprotein CIII Reduction Protects White Adipose Tissues against Obesity-Induced Inflammation and Insulin Resistance in Mice.
Autorzy:
Recio-López P; The Rolf Luft Research Center for Diabetes and Endocrinology, Karolinska Institutet, Karolinska University Hospital, SE-171 76 Stockholm, Sweden.
Valladolid-Acebes I; The Rolf Luft Research Center for Diabetes and Endocrinology, Karolinska Institutet, Karolinska University Hospital, SE-171 76 Stockholm, Sweden.
Berggren PO; The Rolf Luft Research Center for Diabetes and Endocrinology, Karolinska Institutet, Karolinska University Hospital, SE-171 76 Stockholm, Sweden.
Juntti-Berggren L; The Rolf Luft Research Center for Diabetes and Endocrinology, Karolinska Institutet, Karolinska University Hospital, SE-171 76 Stockholm, Sweden.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Dec 22; Vol. 23 (1). Date of Electronic Publication: 2021 Dec 22.
Typ publikacji:
Journal Article
MeSH Terms:
Adipose Tissue, White/*metabolism
Apolipoprotein C-III/*metabolism
Inflammation/*metabolism
Insulin Resistance/*physiology
Obesity/*metabolism
Adipocytes/metabolism ; Adipose Tissue, Brown/metabolism ; Animals ; Diet, High-Fat ; Insulin/metabolism ; Lipolysis/physiology ; Liver/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Thermogenesis/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Adipose-Specific PPARα Knockout Mice Have Increased Lipogenesis by PASK-SREBP1 Signaling and a Polarity Shift to Inflammatory Macrophages in White Adipose Tissue.
Autorzy:
Hinds TD Jr; Department of Pharmacology and Nutritional Sciences, University of Kentucky, Lexington, KY 40508, USA.; Barnstable Brown Diabetes Center, University of Kentucky, Lexington, KY 40508, USA.; Markey Cancer Center, University of Kentucky, Lexington, KY 40508, USA.
Kipp ZA; Department of Pharmacology and Nutritional Sciences, University of Kentucky, Lexington, KY 40508, USA.
Xu M; Department of Pharmacology and Nutritional Sciences, University of Kentucky, Lexington, KY 40508, USA.
Yiannikouris FB; Department of Pharmacology and Nutritional Sciences, University of Kentucky, Lexington, KY 40508, USA.; Barnstable Brown Diabetes Center, University of Kentucky, Lexington, KY 40508, USA.
Morris AJ; Division of Cardiovascular Medicine, College of Medicine, University of Kentucky, Lexington, KY 40508, USA.; Lexington Veterans Affairs Medical Center, Lexington, KY 40508, USA.
Stec DF; Small Molecule NMR Facility Core, Vanderbilt Institute of Chemical Biology, Vanderbilt University, Nashville, TN 37235, USA.
Wahli W; Lee Kong Chian School of Medicine, Nanyang Technological University Singapore, Clinical Sciences Building, Singapore 308232, Singapore.; Toxalim Research Center in Food Toxicology (UMR 1331), INRAE, ENVT, INP-PURPAN, UPS, Université de Toulouse, F-31300 Toulouse, France.; Center for Integrative Genomics, Université de Lausanne, Le Génopode, CH-1015 Lausanne, Switzerland.
Stec DE; Department of Physiology & Biophysics, Cardiorenal and Metabolic Diseases Research Center, University of Mississippi Medical Center, Jackson, MS 39216, USA.
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Źródło:
Cells [Cells] 2021 Dec 21; Vol. 11 (1). Date of Electronic Publication: 2021 Dec 21.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Cell Polarity*
Lipogenesis*
Adipose Tissue, White/*metabolism
Inflammation/*pathology
Macrophages/*pathology
PPAR alpha/*deficiency
Protein Serine-Threonine Kinases/*metabolism
Sterol Regulatory Element Binding Protein 1/*metabolism
Adipocytes/metabolism ; Adiposity ; Amino Acids/blood ; Animals ; Biomarkers/metabolism ; Body Weight ; Cholesterol/blood ; Diet, High-Fat ; Female ; Inflammation/blood ; Lipidomics ; Macrophages/metabolism ; Male ; Metabolome ; Mice, Inbred C57BL ; Mice, Knockout ; Models, Biological ; Organ Size ; Organ Specificity ; PPAR alpha/metabolism ; Signal Transduction ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
PEX-168 improves insulin resistance, inflammatory response and adipokines in simple obese mice: a mechanistic exploration.
Autorzy:
Guo Z; College of Nursing, Yangzhou University, Yangzhou, China.
Wu Y; College of Nursing, Yangzhou University, Yangzhou, China.
Zhu L; Northern Jiangsu People's Hospital, Yangzhou, China.
Wang Y; General Surgery Institute of Yangzhou, Yangzhou University, Yangzhou, China.
Wang D; Northern Jiangsu People's Hospital, Yangzhou, China.; General Surgery Institute of Yangzhou, Yangzhou University, Yangzhou, China.; Clinical Medical College of Yangzhou University, Yangzhou, China.
Sun X; College of Nursing, Yangzhou University, Yangzhou, China. .; Northern Jiangsu People's Hospital, Yangzhou, China. .
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Źródło:
BMC endocrine disorders [BMC Endocr Disord] 2021 Dec 20; Vol. 21 (1), pp. 245. Date of Electronic Publication: 2021 Dec 20.
Typ publikacji:
Journal Article
MeSH Terms:
Adipokines/*metabolism
Hyperinsulinism/*drug therapy
Hypoglycemic Agents/*pharmacology
Inflammation/*drug therapy
Obesity/*drug therapy
Peptides/*pharmacology
Polyethylene Glycols/*pharmacology
Animals ; Diet, High-Fat ; Disease Models, Animal ; Hypoglycemic Agents/administration & dosage ; Insulin Resistance/physiology ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Obese ; Peptides/administration & dosage ; Polyethylene Glycols/administration & dosage
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MEK6 Overexpression Exacerbates Fat Accumulation and Inflammatory Cytokines in High-Fat Diet-Induced Obesity.
Autorzy:
Lee S; Department of Food & Nutrition, Sungshin Women's University, Seoul 01133, Korea.
Lee M; Department of Food & Nutrition, Sungshin Women's University, Seoul 01133, Korea.; Research Institute of Obesity Sciences, Sungshin Women's University, Seoul 01133, Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Dec 17; Vol. 22 (24). Date of Electronic Publication: 2021 Dec 17.
Typ publikacji:
Journal Article
MeSH Terms:
Cytokines/*analysis
Fats/*analysis
Inflammation/*genetics
MAP Kinase Kinase 6/*genetics
Obesity/*genetics
Adipose Tissue/pathology ; Animals ; Diet, High-Fat/adverse effects ; Inflammation/etiology ; Inflammation/pathology ; Male ; Mice, Inbred C57BL ; Obesity/etiology ; Obesity/pathology ; Up-Regulation ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
IL-19 Contributes to the Development of Nonalcoholic Steatohepatitis by Altering Lipid Metabolism.
Autorzy:
Azuma YT; Laboratory of Veterinary Pharmacology, Division of Veterinary Science, Osaka Prefecture University Graduate School of Life and Environmental Sciences, Osaka 598-8531, Japan.
Fujita T; Molecular Toxicology Laboratory, Department of Pharmaceutical Sciences, Ritsumeikan University, Kusatsu 525-8577, Japan.
Izawa T; Laboratory of Veterinary Pathology, Division of Veterinary Science, Osaka Prefecture University Graduate School of Life and Environmental Sciences, Osaka 598-8531, Japan.
Hirota K; Laboratory of Veterinary Pharmacology, Division of Veterinary Science, Osaka Prefecture University Graduate School of Life and Environmental Sciences, Osaka 598-8531, Japan.
Nishiyama K; Laboratory of Veterinary Pharmacology, Division of Veterinary Science, Osaka Prefecture University Graduate School of Life and Environmental Sciences, Osaka 598-8531, Japan.
Ikegami A; Molecular Toxicology Laboratory, Department of Pharmaceutical Sciences, Ritsumeikan University, Kusatsu 525-8577, Japan.
Aoyama T; Molecular Toxicology Laboratory, Department of Pharmaceutical Sciences, Ritsumeikan University, Kusatsu 525-8577, Japan.
Ike M; Molecular Toxicology Laboratory, Department of Pharmaceutical Sciences, Ritsumeikan University, Kusatsu 525-8577, Japan.
Ushikai Y; Laboratory of Veterinary Pharmacology, Division of Veterinary Science, Osaka Prefecture University Graduate School of Life and Environmental Sciences, Osaka 598-8531, Japan.
Kuwamura M; Laboratory of Veterinary Pathology, Division of Veterinary Science, Osaka Prefecture University Graduate School of Life and Environmental Sciences, Osaka 598-8531, Japan.
Fujii H; Department of Premier Preventive Medicine, Graduate School of Medicine, Osaka City University, Osaka 545-8585, Japan.
Tsuneyama K; Department of Pathology and Laboratory Medicine, Institute of Biomedical Sciences, Tokushima University Graduate School, Tokushima 770-8503, Japan.
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Źródło:
Cells [Cells] 2021 Dec 13; Vol. 10 (12). Date of Electronic Publication: 2021 Dec 13.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Inflammation/*genetics
Interleukins/*genetics
Lipid Metabolism/*genetics
Non-alcoholic Fatty Liver Disease/*genetics
Adenosine Triphosphate/metabolism ; Animals ; Cholesterol/metabolism ; Diet, High-Fat/adverse effects ; Disease Models, Animal ; Fatty Acids/biosynthesis ; Fatty Acids/genetics ; Hep G2 Cells ; Humans ; Inflammation/etiology ; Inflammation/metabolism ; Inflammation/pathology ; Interleukin-6/genetics ; Liver/injuries ; Liver/metabolism ; Liver/pathology ; Mice ; Mice, Knockout ; Non-alcoholic Fatty Liver Disease/etiology ; Non-alcoholic Fatty Liver Disease/metabolism ; Non-alcoholic Fatty Liver Disease/pathology ; Palmitates/pharmacology ; Transforming Growth Factor beta/genetics ; Triglycerides/metabolism ; Tumor Necrosis Factor-alpha/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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