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Tytuł:
Curcumin Alleviates Oxygen-Glucose-Deprivation/Reperfusion-Induced Oxidative Damage by Regulating miR-1287-5p/LONP2 Axis in SH-SY5Y Cells.
Autorzy:
Zhang T; Department of Traditional Chinese Medicine Encephalopathy, Hubei Provincial Hospital Traditional Chinese Medicine, Wuhan 430074, China.; Department of Traditional Chinese Medicine Encephalopathy, Hubei Province Traditional Chinese Medicine Research Institute, Wuhan 430074, China.
Chen X; Department of Traditional Chinese Medicine Encephalopathy, Hubei Provincial Hospital Traditional Chinese Medicine, Wuhan 430074, China.; Department of Traditional Chinese Medicine Encephalopathy, Hubei Province Traditional Chinese Medicine Research Institute, Wuhan 430074, China.
Qu Y; Department of Traditional Chinese Medicine Encephalopathy, Hubei Provincial Hospital Traditional Chinese Medicine, Wuhan 430074, China.; Department of Traditional Chinese Medicine Encephalopathy, Hubei Province Traditional Chinese Medicine Research Institute, Wuhan 430074, China.
Ding Y; Department of Traditional Chinese Medicine Encephalopathy, Hubei Provincial Hospital Traditional Chinese Medicine, Wuhan 430074, China.; Department of Traditional Chinese Medicine Encephalopathy, Hubei Province Traditional Chinese Medicine Research Institute, Wuhan 430074, China.
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Źródło:
Analytical cellular pathology (Amsterdam) [Anal Cell Pathol (Amst)] 2021 Sep 18; Vol. 2021, pp. 5548706. Date of Electronic Publication: 2021 Sep 18 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Antioxidants*/pharmacology
Brain*/drug effects
Brain*/enzymology
Brain*/pathology
Curcumin*/pharmacology
Ischemic Stroke*/drug therapy
Ischemic Stroke*/enzymology
Ischemic Stroke*/genetics
Ischemic Stroke*/pathology
Neurons*/drug effects
Neurons*/enzymology
Neurons*/pathology
Neuroprotective Agents*/pharmacology
Oxidative Stress*/drug effects
Reperfusion Injury*/enzymology
Reperfusion Injury*/genetics
Reperfusion Injury*/pathology
Reperfusion Injury*/prevention & control
Humans ; Apoptosis/drug effects ; Cell Hypoxia ; Cell Line, Tumor ; Glucose/deficiency ; MicroRNAs/genetics ; MicroRNAs/metabolism ; Signal Transduction
Czasopismo naukowe
Tytuł:
Ginkgolide B‑induced AMPK pathway activation protects astrocytes by regulating endoplasmic reticulum stress, oxidative stress and energy metabolism induced by Aβ1‑42.
Autorzy:
Wang J; School of Food Science and Technology, Dalian Polytechnic University, Dalian, Liaoning 116034, P.R. China.
Ding Y; School of Food Science and Technology, Dalian Polytechnic University, Dalian, Liaoning 116034, P.R. China.
Zhuang L; School of Food Science and Technology, Dalian Polytechnic University, Dalian, Liaoning 116034, P.R. China.
Wang Z; Jiangsu Kanion Pharmaceutical Co. Ltd., Lianyungang, Jiangsu 222000, P.R. China.
Xiao W; Jiangsu Kanion Pharmaceutical Co. Ltd., Lianyungang, Jiangsu 222000, P.R. China.
Zhu J; School of Food Science and Technology, Dalian Polytechnic University, Dalian, Liaoning 116034, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Jun; Vol. 23 (6). Date of Electronic Publication: 2021 Apr 21.
Typ publikacji:
Journal Article
MeSH Terms:
AMP-Activated Protein Kinases/*drug effects
Amyloid beta-Peptides/*metabolism
Astrocytes/*metabolism
Endoplasmic Reticulum Stress/*drug effects
Energy Metabolism/*drug effects
Ginkgolides/*pharmacology
Lactones/*pharmacology
Oxidative Stress/*drug effects
Peptide Fragments/*metabolism
AMP-Activated Protein Kinases/metabolism ; Animals ; Ginkgolides/metabolism ; Glutathione Peroxidase/metabolism ; Heme Oxygenase-1/metabolism ; Lactones/metabolism ; Male ; Malondialdehyde ; Metabolic Diseases ; Rats ; Rats, Sprague-Dawley ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects ; Superoxide Dismutase
Czasopismo naukowe
Tytuł:
Preparation of Antioxidant Protein Hydrolysates from Pleurotus geesteranus and Their Protective Effects on H 2 O 2 Oxidative Damaged PC12 Cells.
Autorzy:
Liao X; Department of Food Science and Technology, Institute of Food Safety and Nutrition, College of Science & Engineering, Jinan University, Guangzhou 510632, China.; State Key Laboratory of Applied Microbiology Southern China, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Provincial Key Laboratory of Microbial Safety and Health, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Open Laboratory of Applied Microbiology, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.
Zhu Z; Department of Food Science and Technology, Institute of Food Safety and Nutrition, College of Science & Engineering, Jinan University, Guangzhou 510632, China.
Wu S; Department of Food Science and Technology, Institute of Food Safety and Nutrition, College of Science & Engineering, Jinan University, Guangzhou 510632, China.; State Key Laboratory of Applied Microbiology Southern China, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Provincial Key Laboratory of Microbial Safety and Health, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Open Laboratory of Applied Microbiology, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; College of Life Science and Technology, Jinan University, Guangzhou 510632, China.
Chen M; Department of Food Science and Technology, Institute of Food Safety and Nutrition, College of Science & Engineering, Jinan University, Guangzhou 510632, China.; State Key Laboratory of Applied Microbiology Southern China, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Provincial Key Laboratory of Microbial Safety and Health, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Open Laboratory of Applied Microbiology, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.
Huang R; Department of Food Science and Technology, Institute of Food Safety and Nutrition, College of Science & Engineering, Jinan University, Guangzhou 510632, China.; State Key Laboratory of Applied Microbiology Southern China, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Provincial Key Laboratory of Microbial Safety and Health, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Open Laboratory of Applied Microbiology, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.
Wang J; College of Food Science, South China Agricultural University, Guangzhou 510642, China.
Wu Q; State Key Laboratory of Applied Microbiology Southern China, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Provincial Key Laboratory of Microbial Safety and Health, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Open Laboratory of Applied Microbiology, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.
Ding Y; Department of Food Science and Technology, Institute of Food Safety and Nutrition, College of Science & Engineering, Jinan University, Guangzhou 510632, China.; State Key Laboratory of Applied Microbiology Southern China, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Provincial Key Laboratory of Microbial Safety and Health, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.; Guangdong Open Laboratory of Applied Microbiology, Guangdong Institute of Microbiology, Guangdong Academy of Sciences, Guangzhou 510070, China.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2020 Nov 19; Vol. 25 (22). Date of Electronic Publication: 2020 Nov 19.
Typ publikacji:
Journal Article
MeSH Terms:
Antioxidants/*pharmacology
Hydrogen Peroxide/*toxicity
Oxidative Stress/*drug effects
Pleurotus/*chemistry
Protective Agents/*pharmacology
Protein Hydrolysates/*pharmacology
Amino Acids/analysis ; Animals ; Hydrolysis ; PC12 Cells ; Pleurotus/enzymology ; Rats ; Subtilisins/metabolism
Czasopismo naukowe
Tytuł:
Dihydromyricetin Improves Endothelial Dysfunction in Diabetic Mice via Oxidative Stress Inhibition in a SIRT3-Dependent Manner.
Autorzy:
Hua YY; Department of Pharmacology, School of Pharmacy, Key Laboratory of Inflammation and Molecular Drug Target of Jiangsu Province, Nantong University, Nantong 226001, China.
Zhang Y; Department of Pharmacology, School of Pharmacy, Key Laboratory of Inflammation and Molecular Drug Target of Jiangsu Province, Nantong University, Nantong 226001, China.
Gong WW; Department of Pharmacology, School of Pharmacy, Key Laboratory of Inflammation and Molecular Drug Target of Jiangsu Province, Nantong University, Nantong 226001, China.
Ding Y; Department of Pharmacology, School of Pharmacy, Key Laboratory of Inflammation and Molecular Drug Target of Jiangsu Province, Nantong University, Nantong 226001, China.
Shen JR; Department of Pharmacology, School of Pharmacy, Key Laboratory of Inflammation and Molecular Drug Target of Jiangsu Province, Nantong University, Nantong 226001, China.
Li H; Department of Pharmacology, School of Pharmacy, Key Laboratory of Inflammation and Molecular Drug Target of Jiangsu Province, Nantong University, Nantong 226001, China.
Chen Y; Department of Pharmacology, School of Pharmacy, Key Laboratory of Inflammation and Molecular Drug Target of Jiangsu Province, Nantong University, Nantong 226001, China.
Meng GL; Department of Pharmacology, School of Pharmacy, Key Laboratory of Inflammation and Molecular Drug Target of Jiangsu Province, Nantong University, Nantong 226001, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Sep 13; Vol. 21 (18). Date of Electronic Publication: 2020 Sep 13.
Typ publikacji:
Journal Article
MeSH Terms:
Diabetes Mellitus, Experimental/*metabolism
Endothelial Cells/*drug effects
Endothelium, Vascular/*drug effects
Flavonols/*pharmacology
Oxidative Stress/*drug effects
Sirtuin 3/*metabolism
Vascular Diseases/*drug therapy
Animals ; Aorta, Thoracic/drug effects ; Aorta, Thoracic/metabolism ; Blood Glucose/drug effects ; DNA/metabolism ; DNA Copy Number Variations/drug effects ; DNA, Mitochondrial/drug effects ; DNA, Mitochondrial/metabolism ; Endothelial Cells/metabolism ; Endothelium, Vascular/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Reactive Oxygen Species/metabolism ; Superoxide Dismutase/metabolism ; Vascular Diseases/metabolism
Czasopismo naukowe
Tytuł:
Protective Effects of Novel Antioxidant Peptide Purified from Alcalase Hydrolysate of Velvet Antler Against Oxidative Stress in Chang Liver Cells in Vitro and in a Zebrafish Model In Vivo.
Autorzy:
Ding Y; Department of Pharmaceutical Engineering, Soonchunhyang University, Asan 31538, Korea. .
Ko SC; National Marine Bio-Resources and Information Center, National Marine Biodiversity Institute of Korea, Seochun 33662, Korea. .
Moon SH; Division of Food Bioscience, Konkuk University, Chungju 27478, Korea. .
Lee SH; Department of Pharmaceutical Engineering, Soonchunhyang University, Asan 31538, Korea. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Oct 19; Vol. 20 (20). Date of Electronic Publication: 2019 Oct 19.
Typ publikacji:
Journal Article
MeSH Terms:
Antioxidants/*pharmacology
Hepatocytes/*drug effects
Hepatocytes/*metabolism
Oxidative Stress/*drug effects
Peptide Fragments/*pharmacology
Subtilisins/*chemistry
Animals ; Antioxidants/chemistry ; Antioxidants/isolation & purification ; Antlers/chemistry ; Free Radical Scavengers/chemistry ; Free Radical Scavengers/pharmacology ; Humans ; Hydrolysis ; Peptide Fragments/chemistry ; Peptide Fragments/isolation & purification ; Reactive Oxygen Species/metabolism ; Zebrafish
Czasopismo naukowe
Tytuł:
Fluoride-induced iron overload contributes to hepatic oxidative damage in mouse and the protective role of Grape seed proanthocyanidin extract.
Autorzy:
Niu Q; Department of Public Health, Medical College, Shihezi University, China.
He P; Occupational disease prevention and control of hospital of the Xinjiang Uygur Autonomous Region, China.
Xu S; Department of Public Health, Medical College, Shihezi University, China.
Ma R; Department of Public Health, Medical College, Shihezi University, China.
Ding Y; Department of Public Health, Medical College, Shihezi University, China.
Mu L; Department of Public Health, Medical College, Shihezi University, China.
Li S; Department of Public Health, Medical College, Shihezi University, China.
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Źródło:
The Journal of toxicological sciences [J Toxicol Sci] 2018; Vol. 43 (5), pp. 311-319.
Typ publikacji:
Journal Article
MeSH Terms:
Liver*/enzymology
Liver*/pathology
Phytotherapy*
Grape Seed Extract/*pharmacology
Iron Overload/*chemically induced
Liver Diseases/*etiology
Liver Diseases/*prevention & control
Oxidative Stress/*drug effects
Proanthocyanidins/*pharmacology
Sodium Fluoride/*adverse effects
Alanine Transaminase/metabolism ; Animals ; Aspartate Aminotransferases/metabolism ; Cation Transport Proteins/metabolism ; Hepcidins/metabolism ; Iron/metabolism ; Iron Overload/complications ; Iron Overload/metabolism ; Male ; Mice
Czasopismo naukowe
Tytuł:
Acetyl-11-Keto-β-Boswellic Acid Attenuates Prooxidant and Profibrotic Mechanisms Involving Transforming Growth Factor-β1, and Improves Vascular Remodeling in Spontaneously Hypertensive Rats.
Autorzy:
Shang P; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Liu W; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Liu T; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Zhang Y; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Mu F; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Zhu Z; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Liang L; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Zhai X; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Ding Y; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Li Y; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Wen A; Department of Pharmacy, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
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Źródło:
Scientific reports [Sci Rep] 2016 Dec 23; Vol. 6, pp. 39809. Date of Electronic Publication: 2016 Dec 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation/*drug effects
Hypertension/*metabolism
Oxidative Stress/*drug effects
Transforming Growth Factor beta1/*biosynthesis
Triterpenes/*pharmacology
Vascular Remodeling/*drug effects
Animals ; Fibrosis ; Hypertension/pathology ; Rats ; Rats, Inbred SHR ; Rats, Inbred WKY ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł:
AMPK activation protects cells from oxidative stress-induced senescence via autophagic flux restoration and intracellular NAD(+) elevation.
Autorzy:
Han X; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Tai H; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Wang X; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Wang Z; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Zhou J; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Wei X; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Ding Y; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Gong H; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Mo C; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Zhang J; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Qin J; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Ma Y; Center of Infectious Diseases, West China Hospital, Sichuan University, Chengdu, China.
Huang N; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
Xiang R; Department of Clinical Medicine, Medical School of Nankai University, Tianjin, China.
Xiao H; Lab for Aging Research, State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University and Collaborative Innovation Center for Biotherapy, Chengdu, China.
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Źródło:
Aging cell [Aging Cell] 2016 Jun; Vol. 15 (3), pp. 416-27. Date of Electronic Publication: 2016 Feb 18.
Typ publikacji:
Journal Article
MeSH Terms:
Autophagy*/drug effects
Cellular Senescence*/drug effects
Cytoprotection*/drug effects
Oxidative Stress*/drug effects
AMP-Activated Protein Kinases/*metabolism
Intracellular Space/*metabolism
NAD/*metabolism
Animals ; Basic Helix-Loop-Helix Leucine Zipper Transcription Factors/metabolism ; Cell Nucleus/drug effects ; Cell Nucleus/metabolism ; Enzyme Activation/drug effects ; Human Umbilical Vein Endothelial Cells/metabolism ; Humans ; Hydrogen Peroxide/toxicity ; Lysosomes/drug effects ; Lysosomes/metabolism ; Mice ; NIH 3T3 Cells ; Protein Transport/drug effects ; TOR Serine-Threonine Kinases/metabolism
Czasopismo naukowe
Tytuł:
Attenuating Oxidative Stress by Paeonol Protected against Acetaminophen-Induced Hepatotoxicity in Mice.
Autorzy:
Ding Y; Department of Geriatrics, Third Affiliated Hospital of Soochow University, Changzhou, China.
Li Q; Department of Pathology, Third Affiliated Hospital of Soochow University, Changzhou, China.
Xu Y; Modern Medical Research Center, Third Affiliated Hospital of Soochow University, Changzhou, China.
Chen Y; Department of Geriatrics, Third Affiliated Hospital of Soochow University, Changzhou, China.
Deng Y; Department of Geriatrics, Third Affiliated Hospital of Soochow University, Changzhou, China.
Zhi F; Modern Medical Research Center, Third Affiliated Hospital of Soochow University, Changzhou, China.
Qian K; Department of Geriatrics, Third Affiliated Hospital of Soochow University, Changzhou, China.
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Źródło:
PloS one [PLoS One] 2016 May 04; Vol. 11 (5), pp. e0154375. Date of Electronic Publication: 2016 May 04 (Print Publication: 2016).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Acetaminophen/*toxicity
Acetophenones/*pharmacology
Chemical and Drug Induced Liver Injury/*prevention & control
Oxidative Stress/*drug effects
Acetaminophen/administration & dosage ; Acetophenones/administration & dosage ; Analgesics, Non-Narcotic/administration & dosage ; Analgesics, Non-Narcotic/toxicity ; Animals ; Chemical and Drug Induced Liver Injury/etiology ; Chemical and Drug Induced Liver Injury/metabolism ; Disease Models, Animal ; Glutathione/metabolism ; Glutathione Peroxidase/metabolism ; Liver/drug effects ; Liver/metabolism ; Liver/pathology ; MAP Kinase Signaling System/drug effects ; Male ; Malondialdehyde/metabolism ; Mice ; Mice, Inbred C57BL ; NF-kappa B/metabolism ; Superoxide Dismutase/metabolism
Czasopismo naukowe
Tytuł:
Induction of ROS Overload by Alantolactone Prompts Oxidative DNA Damage and Apoptosis in Colorectal Cancer Cells.
Autorzy:
Ding Y; Department of Radiotherapy, the Second Hospital of Jilin University, 218 Ziqiang Street, Changchun 130041, China. .
Wang H; School of Life Sciences, Jilin University, 2699 Qianjin Street, Changchun 130012, China. .
Niu J; School of Life Sciences, Jilin University, 2699 Qianjin Street, Changchun 130012, China. .
Luo M; Department of Nephrology, the Second Hospital of Jilin University, 218 Ziqiang Street, Changchun 130041, China. .
Gou Y; School of Life Sciences, Jilin University, 2699 Qianjin Street, Changchun 130012, China. .
Miao L; Department of Nephrology, the Second Hospital of Jilin University, 218 Ziqiang Street, Changchun 130041, China. .
Zou Z; School of Life Sciences, Jilin University, 2699 Qianjin Street, Changchun 130012, China. .
Cheng Y; Department of Radiotherapy, the Second Hospital of Jilin University, 218 Ziqiang Street, Changchun 130041, China. .; Department of Thoracic Oncology, Jilin Provincial Cancer Hospital, 1018 Huguang Street, Changchun 130012, China. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2016 Apr 14; Vol. 17 (4), pp. 558. Date of Electronic Publication: 2016 Apr 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents, Phytogenic/*pharmacology
Apoptosis/*drug effects
Colorectal Neoplasms/*drug therapy
DNA Damage/*drug effects
Lactones/*pharmacology
Oxidative Stress/*drug effects
Reactive Oxygen Species/*metabolism
Sesquiterpenes, Eudesmane/*pharmacology
Antineoplastic Agents, Phytogenic/chemistry ; Cell Cycle Checkpoints ; Cell Line, Tumor ; Colon/drug effects ; Colon/metabolism ; Colon/pathology ; Colorectal Neoplasms/metabolism ; Colorectal Neoplasms/pathology ; Guanine/analogs & derivatives ; Guanine/metabolism ; Humans ; Inula/chemistry ; Lactones/chemistry ; Rectum/drug effects ; Rectum/metabolism ; Rectum/pathology ; Sesquiterpenes, Eudesmane/chemistry
Czasopismo naukowe
Tytuł:
Efficacy of Procyanidins against In Vivo Cellular Oxidative Damage: A Systematic Review and Meta-Analysis.
Autorzy:
Li S; Department of Public Health and Key Laboratory of Xinjiang Endemic and Ethnic Diseases (Ministry of Education), Shihezi University School of Medicine, Xinjiang, China.
Xu M; Department of Public Health and Key Laboratory of Xinjiang Endemic and Ethnic Diseases (Ministry of Education), Shihezi University School of Medicine, Xinjiang, China.
Niu Q; Department of Public Health and Key Laboratory of Xinjiang Endemic and Ethnic Diseases (Ministry of Education), Shihezi University School of Medicine, Xinjiang, China.
Xu S; Department of Public Health and Key Laboratory of Xinjiang Endemic and Ethnic Diseases (Ministry of Education), Shihezi University School of Medicine, Xinjiang, China.
Ding Y; Department of Public Health and Key Laboratory of Xinjiang Endemic and Ethnic Diseases (Ministry of Education), Shihezi University School of Medicine, Xinjiang, China.
Yan Y; Department of Public Health and Key Laboratory of Xinjiang Endemic and Ethnic Diseases (Ministry of Education), Shihezi University School of Medicine, Xinjiang, China.
Guo S; Department of Public Health and Key Laboratory of Xinjiang Endemic and Ethnic Diseases (Ministry of Education), Shihezi University School of Medicine, Xinjiang, China.
Li F; Department of Pathology and Key Laboratory of Xinjiang Endemic and Ethnic Diseases (Ministry of Education), Shihezi University School of Medicine, Shihezi, Xinjiang, 832002, China.
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Źródło:
PloS one [PLoS One] 2015 Oct 01; Vol. 10 (10), pp. e0139455. Date of Electronic Publication: 2015 Oct 01 (Print Publication: 2015).
Typ publikacji:
Journal Article; Meta-Analysis; Research Support, Non-U.S. Gov't; Review; Systematic Review
MeSH Terms:
Antioxidants/*pharmacology
Biflavonoids/*pharmacology
Catechin/*pharmacology
Oxidative Stress/*drug effects
Proanthocyanidins/*pharmacology
Animals ; Humans
Czasopismo naukowe
Tytuł:
Urotensin II Protects Cardiomyocytes from Apoptosis Induced by Oxidative Stress through the CSE/H2S Pathway.
Autorzy:
Gong H; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China. .
Chen Z; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China. .
Zhang X; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China. .; Department of Geriatrics, Zhongshan Hospital, Fudan University, Shanghai 200032, China. .
Li Y; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China. liyang9_.
Zhang J; Department of Radiology, Shanghai First People's Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai 200080, China. .
Chen Y; Department of Physiology and Pathophysiology, Shanghai Medical College, Fudan University, Shanghai 200032, China. .
Ding Y; Department of Physiology and Pathophysiology, Shanghai Medical College, Fudan University, Shanghai 200032, China. .
Zhang G; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China. .
Yang C; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, China. .
Zhu Y; Department of Physiology and Pathophysiology, Shanghai Medical College, Fudan University, Shanghai 200032, China. .
Zou Y; Department of Radiology, Shanghai First People's Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai 200080, China. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2015 Jun 03; Vol. 16 (6), pp. 12482-98. Date of Electronic Publication: 2015 Jun 03.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cystathionine gamma-Lyase/*metabolism
Hydrogen Sulfide/*metabolism
Myocytes, Cardiac/*drug effects
Oxidative Stress/*drug effects
Urotensins/*pharmacology
Animals ; Apoptosis/drug effects ; Cells, Cultured ; Gene Expression Regulation/drug effects ; Hydrogen Peroxide/pharmacology ; Male ; Myocytes, Cardiac/metabolism ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Rats ; Rats, Sprague-Dawley ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł:
Genome sequencing of the perciform fish Larimichthys crocea provides insights into molecular and genetic mechanisms of stress adaptation.
Autorzy:
Ao J; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
Mu Y; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
Xiang LX; College of Life Sciences, ZheJiang University, Key Laboratory for Cell and Gene Engineering of Zhejiang Province, Hangzhou, ZheJiang, P. R. China.
Fan D; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Feng M; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Zhang S; Ocean University of China, Qingdao, Shandong, P. R. China.
Shi Q; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Zhu LY; College of Life Sciences, ZheJiang University, Key Laboratory for Cell and Gene Engineering of Zhejiang Province, Hangzhou, ZheJiang, P. R. China.
Li T; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
Ding Y; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
Nie L; College of Life Sciences, ZheJiang University, Key Laboratory for Cell and Gene Engineering of Zhejiang Province, Hangzhou, ZheJiang, P. R. China.
Li Q; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
Dong WR; College of Life Sciences, ZheJiang University, Key Laboratory for Cell and Gene Engineering of Zhejiang Province, Hangzhou, ZheJiang, P. R. China.
Jiang L; College of Life Sciences, Shenzhen University, Shenzhen, Guangdong, P. R. China.
Sun B; Ocean University of China, Qingdao, Shandong, P. R. China.
Zhang X; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Li M; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
Zhang HQ; College of Life Sciences, ZheJiang University, Key Laboratory for Cell and Gene Engineering of Zhejiang Province, Hangzhou, ZheJiang, P. R. China.
Xie S; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Zhu Y; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Jiang X; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Wang X; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
Mu P; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
Chen W; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
Yue Z; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Wang Z; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Wang J; BGI-Tech, BGI-Shenzhen, Shenzhen, Guangdong, P. R. China.
Shao JZ; College of Life Sciences, ZheJiang University, Key Laboratory for Cell and Gene Engineering of Zhejiang Province, Hangzhou, ZheJiang, P. R. China.
Chen X; Key Laboratory of Marine Biogenetics and Resources, Third Institute of Oceanography, State Oceanic Administration, Fujian Collaborative Innovation Center for Exploitation and Utilization of Marine Biological Resources, Key Laboratory of Marine Genetic Resources of Fujian Province, Xiamen, P. R. China.
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Źródło:
PLoS genetics [PLoS Genet] 2015 Apr 02; Vol. 11 (4), pp. e1005118. Date of Electronic Publication: 2015 Apr 02 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adaptation, Physiological*
Genome*
Osmotic Pressure*
Oxidative Stress*
Fish Proteins/*genetics
Perciformes/*genetics
Animals ; Fish Proteins/metabolism ; Perciformes/metabolism ; Transcriptome
Czasopismo naukowe
Tytuł:
Alterations of the gut microbiota in high-fat diet mice is strongly linked to oxidative stress.
Autorzy:
Qiao Y; State Key Laboratory of Food Science and Technology, Jiangnan University, Wuxi, China.
Sun J
Ding Y
Le G
Shi Y
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Źródło:
Applied microbiology and biotechnology [Appl Microbiol Biotechnol] 2013 Feb; Vol. 97 (4), pp. 1689-97. Date of Electronic Publication: 2012 Sep 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Metagenome*
Oxidative Stress*
Diet, High-Fat/*adverse effects
Dietary Fats/*adverse effects
Intestines/*microbiology
Metabolic Syndrome/*metabolism
Animals ; Antioxidants/metabolism ; Bacteria/classification ; Bacteria/genetics ; Bacteria/isolation & purification ; Bacteria/metabolism ; Dietary Fats/metabolism ; Humans ; Intestinal Mucosa/metabolism ; Male ; Malondialdehyde/metabolism ; Metabolic Syndrome/microbiology ; Mice ; Molecular Sequence Data ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł:
A Fur-like protein PerR regulates two oxidative stress response related operons dpr and metQIN in Streptococcus suis.
Autorzy:
Zhang T; Division of Animal Infectious Diseases in the State Key Laboratory of Agricultural Microbiology, College of Veterinary Medicine, Huazhong Agricultural University, Shizishan Street, Wuhan 430070, China.
Ding Y
Li T
Wan Y
Li W
Chen H
Zhou R
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Źródło:
BMC microbiology [BMC Microbiol] 2012 May 30; Vol. 12, pp. 85. Date of Electronic Publication: 2012 May 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation, Bacterial*
Operon*
Oxidative Stress*
Bacterial Proteins/*metabolism
DNA-Binding Proteins/*metabolism
Membrane Transport Proteins/*metabolism
Repressor Proteins/*metabolism
Streptococcus suis/*genetics
Agouti Signaling Protein ; Animals ; Artificial Gene Fusion ; Bacterial Proteins/genetics ; DNA, Bacterial/metabolism ; Disease Models, Animal ; Electrophoretic Mobility Shift Assay ; Gene Deletion ; Gene Expression Profiling ; Genes, Reporter ; Green Fluorescent Proteins/analysis ; Green Fluorescent Proteins/genetics ; Hydrogen Peroxide/toxicity ; Mice ; Promoter Regions, Genetic ; Protein Binding ; Regulon ; Repressor Proteins/genetics ; Streptococcal Infections/microbiology ; Streptococcal Infections/pathology ; Streptococcus suis/drug effects ; Streptococcus suis/physiology ; Virulence Factors/genetics ; Virulence Factors/metabolism
Czasopismo naukowe

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