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Tytuł :
Chronic exposure of hydrogen peroxide alters redox state, apoptosis and endoplasmic reticulum stress in common carp (Cyprinus carpio).
Autorzy :
Jia R; Key Laboratory of Freshwater Fisheries and Germplasm Resources Utilization, Ministry of Agriculture and Rural Affairs, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China; International Joint Research Laboratory for Fish Immunopharmacology, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China. Electronic address: .
Du J; Key Laboratory of Freshwater Fisheries and Germplasm Resources Utilization, Ministry of Agriculture and Rural Affairs, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China; International Joint Research Laboratory for Fish Immunopharmacology, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China.
Cao L; Key Laboratory of Freshwater Fisheries and Germplasm Resources Utilization, Ministry of Agriculture and Rural Affairs, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China; International Joint Research Laboratory for Fish Immunopharmacology, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China.
Feng W; Key Laboratory of Freshwater Fisheries and Germplasm Resources Utilization, Ministry of Agriculture and Rural Affairs, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China; International Joint Research Laboratory for Fish Immunopharmacology, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China.
He Q; Key Laboratory of Freshwater Fisheries and Germplasm Resources Utilization, Ministry of Agriculture and Rural Affairs, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China.
Xu P; Key Laboratory of Freshwater Fisheries and Germplasm Resources Utilization, Ministry of Agriculture and Rural Affairs, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China; International Joint Research Laboratory for Fish Immunopharmacology, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China. Electronic address: .
Yin G; Key Laboratory of Freshwater Fisheries and Germplasm Resources Utilization, Ministry of Agriculture and Rural Affairs, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China; International Joint Research Laboratory for Fish Immunopharmacology, Freshwater Fisheries Research Center, Chinese Academy of Fishery Sciences, Wuxi 214081, China. Electronic address: .
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Źródło :
Aquatic toxicology (Amsterdam, Netherlands) [Aquat Toxicol] 2020 Dec; Vol. 229, pp. 105657. Date of Electronic Publication: 2020 Oct 13.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Carps/*metabolism
Endoplasmic Reticulum Stress/*drug effects
Hydrogen Peroxide/*toxicity
Animals ; Antioxidants/metabolism ; Apoptosis/genetics ; Carps/genetics ; Endoplasmic Reticulum Stress/genetics ; Gene Expression Regulation/drug effects ; Gills/drug effects ; Gills/metabolism ; Glutathione Peroxidase/metabolism ; Liver/drug effects ; Liver/metabolism ; Organ Specificity/drug effects ; Oxidation-Reduction/drug effects ; Oxidative Stress/drug effects ; Oxidative Stress/genetics ; Principal Component Analysis ; Water Pollutants, Chemical/toxicity
Czasopismo naukowe
Tytuł :
Trichinella spiralis muscle larvae excretory/secretory products trigger apoptosis and S-phase arrest of the non-small-cell lung cancer line A549.
Autorzy :
Wu H; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China.
Li M; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China.
Shao X; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China.
An Z; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China.
Du J; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China.
Yin H; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China.
Pan J; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China.
Li S; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China.
Zhang Y; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China.
Du L; Department of Pathogenic Biology, Chengde Medical University, Chengde, 067000, Hebei, China. Electronic address: .
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Źródło :
Experimental parasitology [Exp Parasitol] 2020 Nov; Vol. 218, pp. 107983. Date of Electronic Publication: 2020 Aug 28.
Typ publikacji :
Journal Article
MeSH Terms :
A549 Cells/*drug effects
Antigens, Helminth/*pharmacology
Apoptosis/*drug effects
Cell Cycle Checkpoints/*drug effects
Helminth Proteins/*pharmacology
S Phase/*drug effects
Trichinella spiralis/*chemistry
A549 Cells/cytology ; Analysis of Variance ; Animals ; Blotting, Western ; Carcinoma, Non-Small-Cell Lung/pathology ; Cell Proliferation/drug effects ; Culture Media, Serum-Free ; Female ; Humans ; Larva/chemistry ; Mice ; Trichinella spiralis/immunology
Czasopismo naukowe
Tytuł :
Toxoplasma gondii Chinese I genotype Wh6 strain infection induces tau phosphorylation via activating GSK3β and causes hippocampal neuron apoptosis.
Autorzy :
Tao Q; Anhui Provincial Laboratory of Microbiology and Parasitology, Anhui Medical University, Hefei, China.
Wang X; Department of Pediatrics, First Affiliated Hospital of Anhui Medical University, Heifei, China.
Liu L; Department of Parasitology, Medical College of Soochow University, Suzhou, China.
Ji Y; Anhui Provincial Laboratory of Microbiology and Parasitology, Anhui Medical University, Hefei, China.
Luo Q; Anhui Provincial Laboratory of Microbiology and Parasitology, Anhui Medical University, Hefei, China.
Du J; Anhui Provincial Laboratory of Microbiology and Parasitology, Anhui Medical University, Hefei, China.
Yu L; Anhui Provincial Laboratory of Microbiology and Parasitology, Anhui Medical University, Hefei, China.
Shen J; Anhui Provincial Laboratory of Zoonoses of High Institutions, Anhui Medical University, Hefei, China.
Chu D; Anhui Provincial Laboratory of Microbiology and Parasitology, Anhui Medical University, Hefei, China. Electronic address: .
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Źródło :
Acta tropica [Acta Trop] 2020 Oct; Vol. 210, pp. 105560. Date of Electronic Publication: 2020 May 31.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Glycogen Synthase Kinase 3 beta/*physiology
Hippocampus/*pathology
Toxoplasma/*classification
Toxoplasmosis, Animal/*metabolism
tau Proteins/*metabolism
Animals ; Cells, Cultured ; Genotype ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; Neurons/pathology ; Phosphorylation ; Toxoplasma/genetics ; Toxoplasmosis, Animal/pathology
Czasopismo naukowe
Tytuł :
The first visualization of chemotherapy-induced tumor apoptosis via magnetic particle imaging in a mouse model.
Autorzy :
Liang X; Department of Radiology, First Clinical Medical College, Shanxi Medical University, Taiyuan, Shanxi Province, People's Republic of China. College of Medical Imaging, Shanxi Medical University, Taiyuan, Shanxi Province, People's Republic of China.
Wang K
Du J
Tian J
Zhang H
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Źródło :
Physics in medicine and biology [Phys Med Biol] 2020 Sep 24; Vol. 65 (19), pp. 195004. Date of Electronic Publication: 2020 Sep 24.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*
Antineoplastic Agents/*pharmacology
Magnetic Resonance Imaging/*methods
Magnetite Nanoparticles/*chemistry
Thymoma/*pathology
Thymus Neoplasms/*pathology
Animals ; Cell Proliferation ; Contrast Media ; Female ; Humans ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Thymoma/diagnostic imaging ; Thymoma/drug therapy ; Thymus Neoplasms/diagnostic imaging ; Thymus Neoplasms/drug therapy ; Tumor Cells, Cultured ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Knocking down LSD1 inhibits the stemness features of colorectal cancer stem cells.
Autorzy :
Chen J; Department of Gastrointestinal Surgery, Guizhou Provincial Bijie City Qixingguan District People's Hospital, Bijie, China.
Zhao J; Department of Gastrointestinal Surgery, Guizhou Provincial Staff Hospital, Guiyang, China.
Ding J; Department of Gastrointestinal Surgery, Guizhou Provincial People's Hospital, Guiyang, China.
Wang Z; Department of Gastrointestinal Surgery, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.
Du J; Department of Gastrointestinal Surgery, The First People's Hospital of Guiyang, Guiyang, China.
Wu C; Department of Gastrointestinal Surgery, Guizhou Provincial Bijie City Qixingguan District People's Hospital, Bijie, China.
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Źródło :
Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas [Braz J Med Biol Res] 2020 Jun 03; Vol. 53 (7), pp. e9230. Date of Electronic Publication: 2020 Jun 03 (Print Publication: 2020).
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Cell Movement/*drug effects
Cell Proliferation/*drug effects
Colorectal Neoplasms/*pathology
Histone Demethylases/*pharmacology
Neoplastic Stem Cells/*drug effects
Animals ; Blotting, Western ; Cell Line, Tumor ; Colony-Forming Units Assay ; Gene Expression Regulation, Neoplastic ; Humans ; Mice ; Neoplastic Stem Cells/metabolism
Czasopismo naukowe
Tytuł :
MAY, a novel tubulin inhibitor, induces cell apoptosis in A549 and A549/Taxol cells and inhibits epithelial-mesenchymal transition in A549/Taxol cells.
Autorzy :
Du J; Department of Pharmacology, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China.
Li J; Department of Pharmacology, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China.
Gao M; Department of Pharmacology, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China.
Guan Q; Key Laboratory of Structure-Based Drug Design and Discovery, Ministry of Education, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China.
Liu T; Department of Pharmacology, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China.
Wu Y; Key Laboratory of Structure-Based Drug Design and Discovery, Ministry of Education, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China.
Li Z; Department of Pharmacology, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China.
Zuo D; Department of Pharmacology, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China. Electronic address: .
Zhang W; Key Laboratory of Structure-Based Drug Design and Discovery, Ministry of Education, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China. Electronic address: .
Wu Y; Department of Pharmacology, Shenyang Pharmaceutical University, 103 Wenhua Road, Shenhe District, Shenyang, 110016, China. Electronic address: .
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Źródło :
Chemico-biological interactions [Chem Biol Interact] 2020 May 25; Vol. 323, pp. 109074. Date of Electronic Publication: 2020 Mar 23.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Epithelial-Mesenchymal Transition/*drug effects
Paclitaxel/*pharmacology
Triazoles/*pharmacology
Tubulin Modulators/*pharmacology
A549 Cells ; ATP Binding Cassette Transporter, Subfamily B, Member 1/metabolism ; Cell Cycle Checkpoints/drug effects ; Cell Survival/drug effects ; Humans ; Inhibitory Concentration 50 ; Microtubules/drug effects ; Microtubules/metabolism ; Mitochondria/drug effects ; Mitochondria/metabolism ; Nuclear Proteins/metabolism ; Paclitaxel/chemistry ; Polymerization ; Signal Transduction/drug effects ; Triazoles/chemistry ; Tubulin Modulators/chemistry ; Twist-Related Protein 1/metabolism
Czasopismo naukowe
Tytuł :
microRNA-137 downregulates MCL1 in ovarian cancer cells and mediates cisplatin-induced apoptosis.
Autorzy :
Chen W; Department of Gynecology, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, PR China.
Du J; Department of Biological Sciences & Technology, School of Life Sciences, Sun Yat-sen University, Guangzhou, PR China.
Li X; Department of Biological Sciences & Technology, School of Life Sciences, Sun Yat-sen University, Guangzhou, PR China.
Zhi Z; Department of Gynecology, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, PR China.
Jiang S; Department of Biological Sciences & Technology, School of Life Sciences, Sun Yat-sen University, Guangzhou, PR China.
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Źródło :
Pharmacogenomics [Pharmacogenomics] 2020 Feb; Vol. 21 (3), pp. 195-207.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Gene Expression Regulation, Neoplastic*
Apoptosis/*genetics
Carcinoma, Ovarian Epithelial/*pathology
MicroRNAs/*genetics
Myeloid Cell Leukemia Sequence 1 Protein/*genetics
Ovarian Neoplasms/*pathology
Carcinoma, Ovarian Epithelial/genetics ; Female ; HEK293 Cells ; Humans ; Ovarian Neoplasms/genetics ; Primary Cell Culture ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł :
K ATP Channel Blocker Glibenclamide Prevents Radiation-Induced Lung Injury and Inhibits Radiation-Induced Apoptosis of Vascular Endothelial Cells by Increased Ca Influx and Subsequent PKC Activation.
Autorzy :
Xia P; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Cao K; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Hu X; Department of Gastrointestinal Surgery, Changhai Hospital, Shanghai, P.R. China.
Liu L; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Yu D; Ophthalmology Department of Kunming General Hospital of Chengdu Military Area Command, Kunming, Yunnan, China.
Dong S; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Du J; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Xu Y; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Liu B; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Yang Y; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Gao F; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Sun X; Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
Liu H; Department of Radiation Medicine, Faculty of Naval Medicine, Second Military Medical University, Shanghai, China.
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Źródło :
Radiation research [Radiat Res] 2020 Feb; Vol. 193 (2), pp. 171-185. Date of Electronic Publication: 2019 Dec 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Calcium/*metabolism
Glyburide/*pharmacology
KATP Channels/*antagonists & inhibitors
Lung Injury/*prevention & control
Protein Kinase C/*metabolism
Radiation Injuries, Experimental/*prevention & control
Animals ; Apoptosis/radiation effects ; Biological Transport/drug effects ; Biological Transport/radiation effects ; Enzyme Activation/drug effects ; Enzyme Activation/radiation effects ; Gene Expression Regulation/drug effects ; Gene Expression Regulation/radiation effects ; Human Umbilical Vein Endothelial Cells/cytology ; Human Umbilical Vein Endothelial Cells/drug effects ; Human Umbilical Vein Endothelial Cells/radiation effects ; Humans ; Lung Injury/etiology ; Lung Injury/metabolism ; Lung Injury/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Phosphorylation/drug effects ; Phosphorylation/radiation effects ; Potassium Channel Blockers/pharmacology ; Radiation Injuries, Experimental/metabolism ; Radiation Injuries, Experimental/pathology ; Radiation Pneumonitis/prevention & control
Czasopismo naukowe
Tytuł :
L-Cystathionine Protects against Homocysteine-Induced Mitochondria-Dependent Apoptosis of Vascular Endothelial Cells.
Autorzy :
Wang X; Department of Pediatrics, Peking University First Hospital, Beijing 100034, China.; Research Unit of Clinical Diagnosis and Treatment of Pediatric Syncope and Cardiovascular Diseases, Chinese Academy of Medical Sciences, Beijing, China.
Wang Y; Department of Pediatrics, Peking University First Hospital, Beijing 100034, China.
Zhang L; Department of Pediatrics, Peking University First Hospital, Beijing 100034, China.
Zhang D; Department of Pediatrics, Peking University First Hospital, Beijing 100034, China.
Bai L; Department of Pediatrics, Peking University First Hospital, Beijing 100034, China.
Kong W; Department of Physiology and Pathophysiology, Peking University Health Science Center, Beijing 100191, China.
Huang Y; Department of Pediatrics, Peking University First Hospital, Beijing 100034, China.
Tang C; Department of Physiology and Pathophysiology, Peking University Health Science Center, Beijing 100191, China.; Key Lab. of Ministry of Education of China, Beijing 100191, China.
Du J; Department of Pediatrics, Peking University First Hospital, Beijing 100034, China.
Jin H; Department of Pediatrics, Peking University First Hospital, Beijing 100034, China.
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Źródło :
Oxidative medicine and cellular longevity [Oxid Med Cell Longev] 2019 Nov 25; Vol. 2019, pp. 1253289. Date of Electronic Publication: 2019 Nov 25 (Print Publication: 2019).
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Cystathionine/*pharmacology
Homocysteine/*toxicity
Mitochondria/*metabolism
Protective Agents/*pharmacology
Caspase 3/metabolism ; Caspase 9/metabolism ; Human Umbilical Vein Endothelial Cells ; Humans ; Membrane Potential, Mitochondrial/drug effects ; Mitochondrial Membrane Transport Proteins/drug effects ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
ROS-dependent DNA damage contributes to crizotinib-induced hepatotoxicity via the apoptotic pathway.
Autorzy :
Yan H; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Du J; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Chen X; Department of Oncology, Hangzhou First People's Hospital, Zhejiang University, Hangzhou 310006, China.
Yang B; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
He Q; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Yang X; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.. Electronic address: .
Luo P; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.. Electronic address: .
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Źródło :
Toxicology and applied pharmacology [Toxicol Appl Pharmacol] 2019 Nov 15; Vol. 383, pp. 114768. Date of Electronic Publication: 2019 Oct 19.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Crizotinib/*toxicity
DNA Damage/*drug effects
Hepatocytes/*drug effects
Hepatocytes/*metabolism
Reactive Oxygen Species/*metabolism
Adult ; Apoptosis/physiology ; Cell Line ; Cell Survival/drug effects ; Cell Survival/physiology ; DNA Damage/physiology ; Dose-Response Relationship, Drug ; Hepatocytes/pathology ; Humans ; Male ; Protein Kinase Inhibitors/toxicity
Czasopismo naukowe
Tytuł :
The antitumor effect of hinesol, extract from Atractylodes lancea (Thunb.) DC. by proliferation, inhibition, and apoptosis induction via MEK/ERK and NF-κB pathway in non-small cell lung cancer cell lines A549 and NCI-H1299.
Autorzy :
Guo W; School of Chemistry, Biology and Material Engineering, Suzhou University of Science and Technology, Suzhou, China.
Liu S; Suzhou Key Laboratory for Medical Biotechnology, Suzhou Vocational Health College, Suzhou, China.
Ju X; School of Chemistry, Biology and Material Engineering, Suzhou University of Science and Technology, Suzhou, China.
Du J; Suzhou Key Laboratory for Medical Biotechnology, Suzhou Vocational Health College, Suzhou, China.
Xu B; School of Chemistry, Biology and Material Engineering, Suzhou University of Science and Technology, Suzhou, China.
Yuan H; School of Chemistry, Biology and Material Engineering, Suzhou University of Science and Technology, Suzhou, China.
Qin F; School of Chemistry, Biology and Material Engineering, Suzhou University of Science and Technology, Suzhou, China.
Li L; School of Chemistry, Biology and Material Engineering, Suzhou University of Science and Technology, Suzhou, China.
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Źródło :
Journal of cellular biochemistry [J Cell Biochem] 2019 Nov; Vol. 120 (11), pp. 18600-18607. Date of Electronic Publication: 2019 Jul 24.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Antineoplastic Agents, Phytogenic/*pharmacology
Apoptosis/*drug effects
Atractylodes/*chemistry
Cell Proliferation/*drug effects
MAP Kinase Signaling System/*drug effects
NF-kappa B/*metabolism
Sesquiterpenes/*pharmacology
Spiro Compounds/*pharmacology
A549 Cells ; Apoptosis Regulatory Proteins/metabolism ; Carcinoma, Non-Small-Cell Lung/metabolism ; Carcinoma, Non-Small-Cell Lung/pathology ; Cell Line, Tumor ; Dose-Response Relationship, Drug ; G1 Phase Cell Cycle Checkpoints/drug effects ; Humans ; Lung Neoplasms/metabolism ; Lung Neoplasms/pathology ; Molecular Structure ; Plant Extracts/pharmacology ; Resting Phase, Cell Cycle/drug effects ; Sesquiterpenes/chemistry ; Spiro Compounds/chemistry ; Time Factors
Czasopismo naukowe
Tytuł :
S100a8/a9 Signaling Causes Mitochondrial Dysfunction and Cardiomyocyte Death in Response to Ischemic/Reperfusion Injury.
Autorzy :
Li Y; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
Chen B; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
Yang X; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
Zhang C; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
Jiao Y; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
Li P; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
Liu Y; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
Li Z; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
Qiao B; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
Bond Lau W; Department of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA (W.B.L., X.-l.M.).
Ma XL; Department of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA (W.B.L., X.-l.M.).
Du J; Beijing Anzhen Hospital of Capital Medical University and Beijing Institute of Heart Lung and Blood Vessel Diseases, China (Y. Li, B.C., Z.Y., C.Z., Y.J., P.L., Y. Liu, Z.L., B.Q., J.D.).
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Źródło :
Circulation [Circulation] 2019 Aug 27; Vol. 140 (9), pp. 751-764. Date of Electronic Publication: 2019 Jun 21.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*
Calgranulin B/*metabolism
Mitochondria/*metabolism
Myocardial Reperfusion Injury/*pathology
Animals ; Antibodies, Neutralizing/administration & dosage ; Calgranulin A/blood ; Calgranulin B/genetics ; Calgranulin B/immunology ; Disease Models, Animal ; Electron Transport Complex I/antagonists & inhibitors ; Electron Transport Complex I/metabolism ; Heart Failure/etiology ; Humans ; Membrane Potential, Mitochondrial ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Transgenic ; Myocardial Infarction/diagnosis ; Myocardial Infarction/surgery ; Myocardial Reperfusion Injury/drug therapy ; Myocytes, Cardiac/cytology ; Myocytes, Cardiac/metabolism ; Percutaneous Coronary Intervention ; Signal Transduction
Czasopismo naukowe
Tytuł :
GKN2 promotes oxidative stress-induced gastric cancer cell apoptosis via the Hsc70 pathway.
Autorzy :
Zhang Z; Departments of General Surgery, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China.
Xue H; Departments of General Surgery, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China.
Dong Y; Departments of General Surgery, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China.
Zhang J; Departments of Digestive Diseases, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China.
Pan Y; Departments of Digestive Diseases, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China.
Shi L; Departments of General Surgery, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China.
Xiong P; Departments of Digestive Diseases, Dongfang Hospital, Tongji University, Shanghai, China.
Zhu J; Departments of Digestive Diseases, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China.
Li W; Departments of Digestive Diseases, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China.
Zheng W; Departments of Digestive Diseases, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China.
Liu J; Departments of Digestive Diseases, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China. .
Du J; Departments of General Surgery, Huashan Hospital, Fudan University, 12 Middle Urumqi Road, Shanghai, 200040, People's Republic of China. .
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Źródło :
Journal of experimental & clinical cancer research : CR [J Exp Clin Cancer Res] 2019 Aug 05; Vol. 38 (1), pp. 338. Date of Electronic Publication: 2019 Aug 05.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Oxidative Stress*
Signal Transduction*
Carrier Proteins/*metabolism
HSC70 Heat-Shock Proteins/*metabolism
Stomach Neoplasms/*metabolism
Adult ; Aged ; Animals ; Antineoplastic Agents/therapeutic use ; Carrier Proteins/genetics ; Caspases ; Cell Line, Tumor ; Dioxolanes/pharmacology ; Disease Models, Animal ; Female ; Gene Expression ; Gene Knockdown Techniques ; Genes, Reporter ; Humans ; JNK Mitogen-Activated Protein Kinases/metabolism ; Male ; Mice ; Middle Aged ; NF-kappa B/metabolism ; Oxidation-Reduction ; Protein Binding ; Stomach Neoplasms/diagnosis ; Stomach Neoplasms/drug therapy ; Stomach Neoplasms/genetics ; Treatment Outcome ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Epigallocatechin-3-gallate stimulates autophagy and reduces apoptosis levels in retinal Müller cells under high-glucose conditions.
Autorzy :
Wang L; The Second Affiliated Hospital of Soochow University. Suzhou, 215004, China; Department of Ophthalmology, Lixiang Eye Hospital of Soochow University, Suzhou, 215021, China.
Sun X; Shanghai Key Laboratory of Fundus Disease, Shanghai, 200080, China; Department of Ophthalmology, Shanghai First People's Hospital, School of Medicine, Shanghai JiaoTong University, 100 Haining Road, Shanghai, 200080, China.
Zhu M; Department of Ophthalmology, Lixiang Eye Hospital of Soochow University, Suzhou, 215021, China.
Du J; Department of Ophthalmology, Lixiang Eye Hospital of Soochow University, Suzhou, 215021, China.
Xu J; Department of Ophthalmology, Lixiang Eye Hospital of Soochow University, Suzhou, 215021, China.
Qin X; Department of Ophthalmology, Lixiang Eye Hospital of Soochow University, Suzhou, 215021, China.
Xu X; Shanghai Key Laboratory of Fundus Disease, Shanghai, 200080, China; Department of Ophthalmology, Shanghai First People's Hospital, School of Medicine, Shanghai JiaoTong University, 100 Haining Road, Shanghai, 200080, China. Electronic address: .
Song E; Department of Ophthalmology, Lixiang Eye Hospital of Soochow University, Suzhou, 215021, China. Electronic address: .
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Źródło :
Experimental cell research [Exp Cell Res] 2019 Jul 15; Vol. 380 (2), pp. 149-158. Date of Electronic Publication: 2019 Apr 15.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Autophagy/*drug effects
Catechin/*analogs & derivatives
Glucose/*metabolism
Glucose/*pharmacology
Retina/*drug effects
Animals ; Catechin/pharmacology ; Cell Count ; Cell Proliferation/drug effects ; Cells, Cultured ; Dose-Response Relationship, Drug ; Rats ; Retina/cytology ; Structure-Activity Relationship
Czasopismo naukowe
Tytuł :
Echinacoside Alleviates Hypoxic-Ischemic Brain Injury in Neonatal Rat by Enhancing Antioxidant Capacity and Inhibiting Apoptosis.
Autorzy :
Wei W; Department of Pharmacology, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.
Lan XB; Department of Pharmacology, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.
Liu N; Department of Pharmacology, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.
Yang JM; Department of Pharmacology, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.
Du J; Department of Pharmacology, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.; Ningxia Key Laboratory of Craniocerebral Diseases of Ningxia Hui Autonomous Region, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.
Ma L; Ningxia Key Laboratory of Craniocerebral Diseases of Ningxia Hui Autonomous Region, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.
Zhang WJ; Department of Pharmacology, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.
Niu JG; Ningxia Key Laboratory of Craniocerebral Diseases of Ningxia Hui Autonomous Region, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.
Sun T; Ningxia Key Laboratory of Craniocerebral Diseases of Ningxia Hui Autonomous Region, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China.
Yu JQ; Department of Pharmacology, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China. .; Ningxia Hui Medicine Modern Engineering Research Center and Collaborative Innovation Center, Ningxia Medical University, Yinchuan, Ningxia Hui Autonomous Region, 750004, People's Republic of China. .
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Źródło :
Neurochemical research [Neurochem Res] 2019 Jul; Vol. 44 (7), pp. 1582-1592. Date of Electronic Publication: 2019 Mar 25.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Glycosides/*therapeutic use
Hypoxia-Ischemia, Brain/*drug therapy
Neuroprotective Agents/*therapeutic use
Oxidative Stress/*drug effects
Animals ; Animals, Newborn ; Brain/pathology ; Caspase 3/metabolism ; Catalase/metabolism ; Dose-Response Relationship, Drug ; Female ; Glutathione Peroxidase/metabolism ; Glycosides/administration & dosage ; Hypoxia-Ischemia, Brain/pathology ; Male ; Malondialdehyde/metabolism ; Neuroprotective Agents/administration & dosage ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Rats, Sprague-Dawley ; Superoxide Dismutase/metabolism ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
MiR-23 enhances cardiac fibroblast proliferation and suppresses fibroblast apoptosis via targeting TGF-β1 in atrial fibrillation.
Autorzy :
Yu RB; Department of Cardiovascular Surgery, The Affiliated Hospital of Qingdao University, Qingdao, China. .
Li K
Wang G
Gao GM
Du JX
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Źródło :
European review for medical and pharmacological sciences [Eur Rev Med Pharmacol Sci] 2019 May; Vol. 23 (10), pp. 4419-4424.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*genetics
Atrial Fibrillation/*genetics
Atrial Fibrillation/*pathology
Fibroblasts/*pathology
MicroRNAs/*genetics
Transforming Growth Factor beta1/*genetics
Atrial Appendage/metabolism ; Cell Count ; Cell Line ; Cell Proliferation ; Disease Progression ; Gene Targeting ; Humans
Czasopismo naukowe
Tytuł :
lncRNA AWPPH promotes proliferation and inhibits apoptosis of non‑small cell lung cancer cells by activating the Wnt/β‑catenin signaling pathway.
Autorzy :
Song Z; Department of Respiratory Medicine, Cangzhou Central Hospital, Cangzhou, Hebei 061001, P.R. China.
Du J; Department of Respiratory Medicine, Cangzhou Central Hospital, Cangzhou, Hebei 061001, P.R. China.
Zhou L; Department of Respiratory Medicine, Cangzhou Central Hospital, Cangzhou, Hebei 061001, P.R. China.
Sun B; Department of Respiratory Medicine, Cangzhou Central Hospital, Cangzhou, Hebei 061001, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2019 May; Vol. 19 (5), pp. 4425-4432. Date of Electronic Publication: 2019 Mar 27.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Cell Proliferation*
Wnt Signaling Pathway*
Carcinoma, Non-Small-Cell Lung/*diagnosis
Lung Neoplasms/*diagnosis
RNA, Long Noncoding/*metabolism
Adult ; Aged ; Carcinoma, Non-Small-Cell Lung/genetics ; Carcinoma, Non-Small-Cell Lung/mortality ; Cell Line, Tumor ; Female ; Gene Expression Regulation, Neoplastic ; Humans ; Lung Neoplasms/genetics ; Lung Neoplasms/mortality ; Male ; Middle Aged ; Prognosis ; RNA, Long Noncoding/genetics ; Survival Rate ; Young Adult
Czasopismo naukowe
Tytuł :
Vitamin D receptor activation protects against lipopolysaccharide-induced acute kidney injury through suppression of tubular cell apoptosis.
Autorzy :
Du J; Department of Nephrology, Third Xiangya Hospital, Central South University , Changsha, Hunan , China.; Division of Biological Sciences, Department of Medicine, University of Chicago , Chicago, Illinois.
Jiang S; Department of Nephrology, Third Xiangya Hospital, Central South University , Changsha, Hunan , China.
Hu Z; Department of Nephrology, Third Xiangya Hospital, Central South University , Changsha, Hunan , China.
Tang S; Department of Nephrology, Third Xiangya Hospital, Central South University , Changsha, Hunan , China.
Sun Y; Institute of Health Sciences, China Medical University , Shenyang, Liaoning , China.
He J; Department of Nephrology, Third Xiangya Hospital, Central South University , Changsha, Hunan , China.
Li Z; Department of Nephrology, Third Xiangya Hospital, Central South University , Changsha, Hunan , China.
Yi B; Department of Nephrology, Third Xiangya Hospital, Central South University , Changsha, Hunan , China.
Wang J; Department of Nephrology, Third Xiangya Hospital, Central South University , Changsha, Hunan , China.
Zhang H; Department of Nephrology, Third Xiangya Hospital, Central South University , Changsha, Hunan , China.
Li YC; Division of Biological Sciences, Department of Medicine, University of Chicago , Chicago, Illinois.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2019 May 01; Vol. 316 (5), pp. F1068-F1077. Date of Electronic Publication: 2019 Mar 13.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Lipopolysaccharides*
Acute Kidney Injury/*prevention & control
Apoptosis/*drug effects
Ergocalciferols/*pharmacology
Kidney Tubules, Proximal/*drug effects
Receptors, Calcitriol/*agonists
Acute Kidney Injury/chemically induced ; Acute Kidney Injury/metabolism ; Acute Kidney Injury/pathology ; Animals ; Apoptosis Regulatory Proteins/metabolism ; Disease Models, Animal ; Humans ; Kidney Tubules, Proximal/metabolism ; Kidney Tubules, Proximal/pathology ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; NF-kappa B/metabolism ; Proto-Oncogene Proteins/metabolism ; RAW 264.7 Cells ; Receptors, Calcitriol/genetics ; Receptors, Calcitriol/metabolism ; Signal Transduction ; Tumor Suppressor Proteins/metabolism
Czasopismo naukowe
Tytuł :
Inhibition of miR-23a attenuates doxorubicin-induced mitochondria-dependent cardiomyocyte apoptosis by targeting the PGC-1α/Drp1 pathway.
Autorzy :
Du J; Institute of Clinical Pharmacology, the Second Affiliated Hospital of Harbin Medical University (The University Key Laboratory of Drug Research, Heilongjiang Province), Harbin 150086, PR China; Department of Clinical Pharmacology, College of Pharmacy, Harbin Medical University, Harbin 150081, PR China.
Hang P; Institute of Clinical Pharmacology, the Second Affiliated Hospital of Harbin Medical University (The University Key Laboratory of Drug Research, Heilongjiang Province), Harbin 150086, PR China; Department of Clinical Pharmacology, College of Pharmacy, Harbin Medical University, Harbin 150081, PR China.
Pan Y; Institute of Clinical Pharmacology, the Second Affiliated Hospital of Harbin Medical University (The University Key Laboratory of Drug Research, Heilongjiang Province), Harbin 150086, PR China; Department of Clinical Pharmacology, College of Pharmacy, Harbin Medical University, Harbin 150081, PR China.
Feng B; Institute of Clinical Pharmacology, the Second Affiliated Hospital of Harbin Medical University (The University Key Laboratory of Drug Research, Heilongjiang Province), Harbin 150086, PR China; Department of Clinical Pharmacology, College of Pharmacy, Harbin Medical University, Harbin 150081, PR China.
Zheng Y; Institute of Clinical Pharmacology, the Second Affiliated Hospital of Harbin Medical University (The University Key Laboratory of Drug Research, Heilongjiang Province), Harbin 150086, PR China; Department of Clinical Pharmacology, College of Pharmacy, Harbin Medical University, Harbin 150081, PR China.
Chen T; Institute of Clinical Pharmacology, the Second Affiliated Hospital of Harbin Medical University (The University Key Laboratory of Drug Research, Heilongjiang Province), Harbin 150086, PR China; Department of Clinical Pharmacology, College of Pharmacy, Harbin Medical University, Harbin 150081, PR China.
Zhao L; Institute of Clinical Pharmacology, the Second Affiliated Hospital of Harbin Medical University (The University Key Laboratory of Drug Research, Heilongjiang Province), Harbin 150086, PR China; Department of Clinical Pharmacology, College of Pharmacy, Harbin Medical University, Harbin 150081, PR China.
Du Z; Institute of Clinical Pharmacology, the Second Affiliated Hospital of Harbin Medical University (The University Key Laboratory of Drug Research, Heilongjiang Province), Harbin 150086, PR China; State Key Laboratory of Quality Research in Chinese Medicines, Macau University of Science and Technology, Macau, PR China; Department of Clinical Pharmacology, College of Pharmacy, Harbin Medical University, Harbin 150081, PR China. Electronic address: .
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Źródło :
Toxicology and applied pharmacology [Toxicol Appl Pharmacol] 2019 Apr 15; Vol. 369, pp. 73-81. Date of Electronic Publication: 2019 Mar 01.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Doxorubicin/*toxicity
Dynamins/*metabolism
MicroRNAs/*metabolism
Mitochondria, Heart/*drug effects
Myocytes, Cardiac/*drug effects
Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha/*metabolism
Animals ; Animals, Newborn ; Cardiotoxicity ; Caspase 3/metabolism ; Cells, Cultured ; Cytochromes c/metabolism ; Membrane Potential, Mitochondrial/drug effects ; MicroRNAs/genetics ; Mitochondria, Heart/genetics ; Mitochondria, Heart/metabolism ; Mitochondria, Heart/pathology ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/pathology ; Oxidative Stress/drug effects ; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha/genetics ; RNA Interference ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Rats, Sprague-Dawley ; Reactive Oxygen Species/metabolism ; Signal Transduction
Czasopismo naukowe
Tytuł :
Bisdemethoxycurcumin protects against renal fibrosis via activation of fibroblast apoptosis.
Autorzy :
Jin F; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Jin Y; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Du J; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Jiang L; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Zhang Y; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Zhao Z; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Yang B; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China.
Luo P; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China. Electronic address: .
He Q; Institute of Pharmacology & Toxicology, College of Pharmaceutical Sciences, Zhejiang University, Hangzhou 310058, China. Electronic address: .
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Źródło :
European journal of pharmacology [Eur J Pharmacol] 2019 Mar 15; Vol. 847, pp. 26-31. Date of Electronic Publication: 2019 Jan 17.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Curcumin/*analogs & derivatives
Fibroblasts/*drug effects
Fibrosis/*drug therapy
Kidney/*drug effects
Protective Agents/*pharmacology
Renal Insufficiency, Chronic/*drug therapy
Animals ; Biological Products/pharmacology ; Cell Line ; Curcumin/pharmacology ; Diarylheptanoids ; Disease Models, Animal ; Female ; Humans ; Male ; Mice ; Ureteral Obstruction/drug therapy ; Urinary Tract/drug effects
Czasopismo naukowe

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