Informacja

Drogi użytkowniku, aplikacja do prawidłowego działania wymaga obsługi JavaScript. Proszę włącz obsługę JavaScript w Twojej przeglądarce.

Wyszukujesz frazę ""E2F Transcription Factors"" wg kryterium: Temat


Tytuł:
Reproductive period and epigenetic modifications of the oxidative phosphorylation pathway in the human prefrontal cortex.
Autorzy:
Bove RM; Weill Institute for the Neurosciences, Department of Neurology, University of California, San Francisco, San Francisco, CA, United States of America.; Harvard Medical School, Boston, MA, United States of America.
Patrick E; Harvard Medical School, Boston, MA, United States of America.; Program in Translational NeuroPsychiatric Genomics, Institute for the Neurosciences, Departments of Neurology and Psychiatry, Brigham and Women's Hospital, Boston, MA, United States of America.; Program in Medical and Population Genetics, Broad Institute, Cambridge, MA, United States of America.; School of Mathematics and Statistics, University of Sydney, Westmead, NSW, Australia.; The Westmead Institute for Medical Research, The University of Sydney, Westmead, NSW, Australia.
Aubin CM; Program in Medical and Population Genetics, Broad Institute, Cambridge, MA, United States of America.
Srivastava G; Program in Translational NeuroPsychiatric Genomics, Institute for the Neurosciences, Departments of Neurology and Psychiatry, Brigham and Women's Hospital, Boston, MA, United States of America.
Schneider JA; Rush Alzheimer's Disease Center, Rush University Medical Center, Chicago, IL, United States of America.
Bennett DA; Rush Alzheimer's Disease Center, Rush University Medical Center, Chicago, IL, United States of America.
De Jager PL; Harvard Medical School, Boston, MA, United States of America.; Program in Translational NeuroPsychiatric Genomics, Institute for the Neurosciences, Departments of Neurology and Psychiatry, Brigham and Women's Hospital, Boston, MA, United States of America.; Program in Medical and Population Genetics, Broad Institute, Cambridge, MA, United States of America.; Center for Translational & Computational Neuroimmunology, Department of Neurology, Columbia University Medical Center, New York City, NY, United States of America.
Chibnik LB; Harvard Medical School, Boston, MA, United States of America.; Program in Translational NeuroPsychiatric Genomics, Institute for the Neurosciences, Departments of Neurology and Psychiatry, Brigham and Women's Hospital, Boston, MA, United States of America.
Pokaż więcej
Źródło:
PloS one [PLoS One] 2018 Jul 27; Vol. 13 (7), pp. e0199073. Date of Electronic Publication: 2018 Jul 27 (Print Publication: 2018).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Epigenesis, Genetic*
Cognition/*physiology
E2F Transcription Factors/*genetics
Estradiol/*metabolism
Prefrontal Cortex/*metabolism
Stroke/*genetics
Aged, 80 and over ; Autopsy ; CpG Islands ; DNA Methylation ; DNA Repair ; Disease Susceptibility ; E2F Transcription Factors/metabolism ; Estradiol/pharmacology ; Fatty Acids/genetics ; Fatty Acids/metabolism ; Female ; Humans ; Lipid Metabolism/genetics ; Mechanistic Target of Rapamycin Complex 1/genetics ; Mechanistic Target of Rapamycin Complex 1/metabolism ; Oxidative Phosphorylation/drug effects ; Prefrontal Cortex/pathology ; Prospective Studies ; Proto-Oncogene Proteins c-myc/genetics ; Proto-Oncogene Proteins c-myc/metabolism ; Reproduction ; Stroke/diagnosis ; Stroke/pathology
Czasopismo naukowe
Tytuł:
Positive regulation of ataxia-telangiectasia-mutated protein (ATM) by E2F transcription Factor 1 (E2F-1) in cisplatin-resistant nasopharyngeal carcinoma cells.
Autorzy:
Zhou ZY; Department of Oncology, The First People's Hospital of Jingzhou, Jingzhou, 434000, China.
Yang JY; Department of Oncology, The First People's Hospital of Jingzhou, Jingzhou, 434000, China.
Shao CZ; Department of Oncology, The First People's Hospital of Jingzhou, Jingzhou, 434000, China.
Luo F; Department of Oncology, The First People's Hospital of Jingzhou, Jingzhou, 434000, China.
Du W; Department of Oncology, The First People's Hospital of Jingzhou, Jingzhou, 434000, China. duweiweidu_.
Pokaż więcej
Źródło:
World journal of surgical oncology [World J Surg Oncol] 2022 Mar 18; Vol. 20 (1), pp. 88. Date of Electronic Publication: 2022 Mar 18.
Typ publikacji:
Journal Article
MeSH Terms:
Ataxia Telangiectasia*
Nasopharyngeal Neoplasms*/drug therapy
Nasopharyngeal Neoplasms*/genetics
Nasopharyngeal Neoplasms*/metabolism
E2F1 Transcription Factor/*metabolism
Apoptosis ; Ataxia Telangiectasia Mutated Proteins/genetics ; Ataxia Telangiectasia Mutated Proteins/metabolism ; Ataxia Telangiectasia Mutated Proteins/pharmacology ; Cell Line, Tumor ; Cisplatin/pharmacology ; Drug Resistance, Neoplasm ; E2F Transcription Factors/metabolism ; Humans ; Nasopharyngeal Carcinoma/genetics
Czasopismo naukowe
Tytuł:
Actinidia chinensis Planch Root extract suppresses the growth and metastasis of hypopharyngeal carcinoma by inhibiting E2F Transcription Factor 1-mediated MNX1 antisense RNA 1.
Autorzy:
Zheng Y; Medical College, Soochow University, Suzhou, China.; Head and Neck & Otolaryngology Center, Plastic Surgery Center, Cancer Center, Department of Otolaryngology, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou, China.
Su L; Head and Neck & Otolaryngology Center, Plastic Surgery Center, Cancer Center, Department of Otolaryngology, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou, China.
Tan J; Head and Neck & Otolaryngology Center, Plastic Surgery Center, Cancer Center, Department of Otolaryngology, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou, China.
Dong F; Head and Neck & Otolaryngology Center, Plastic Surgery Center, Cancer Center, Department of Otolaryngology, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou, China.
Pokaż więcej
Źródło:
Bioengineered [Bioengineered] 2022 Mar; Vol. 13 (3), pp. 4911-4922.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Actinidia*/chemistry
Carcinoma, Hepatocellular*/genetics
Liver Neoplasms*/genetics
MicroRNAs*/genetics
Plant Extracts*/pharmacology
Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; E2F Transcription Factors/genetics ; E2F1 Transcription Factor ; Gene Expression Regulation, Neoplastic ; Homeodomain Proteins/metabolism ; Humans ; RNA, Antisense/genetics ; Transcription Factors/metabolism
Czasopismo naukowe
Tytuł:
Up-Regulated Interleukin-10 Induced by E2F Transcription Factor 2-MicroRNA-17-5p Circuitry in Extrafollicular Effector B Cells Contributes to Autoantibody Production in Systemic Lupus Erythematosus.
Autorzy:
Xu L; Medical University of South Carolina, Charleston, and The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China.
Wang L; The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China.
Shi Y; Medical University of South Carolina, Charleston, and The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China.
Deng Y; Medical University of South Carolina, Charleston.
Oates JC; Medical University of South Carolina, Charleston, and Ralph H. Johnson VA Medical Center, Medical Service, Charleston, South Carolina.
Kamen DL; Medical University of South Carolina, Charleston.
Gilkeson GS; Medical University of South Carolina, Charleston, and Ralph H. Johnson VA Medical Center, Medical Service, Charleston, South Carolina.
Wang F; The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China.
Zhang M; The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China.
Tan W; The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China.
Tsao BP; Medical University of South Carolina, Charleston.
Pokaż więcej
Źródło:
Arthritis & rheumatology (Hoboken, N.J.) [Arthritis Rheumatol] 2022 Mar; Vol. 74 (3), pp. 496-507. Date of Electronic Publication: 2022 Feb 09.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Autoantibodies*
Up-Regulation*
B-Lymphocyte Subsets/*metabolism
E2F Transcription Factors/*metabolism
Interleukin-10/*metabolism
Lupus Erythematosus, Systemic/*metabolism
Adolescent ; Adult ; Aged ; Female ; Humans ; Interleukin-10/genetics ; Leukocytes, Mononuclear/metabolism ; Lupus Erythematosus, Systemic/genetics ; Male ; MicroRNAs/genetics ; MicroRNAs/metabolism ; Middle Aged ; Young Adult
Czasopismo naukowe
Tytuł:
ATAD2 drives colorectal cancer progression by regulating TRIM25 expression via a positive feedback loop with E2F transcriptional factors.
Autorzy:
Tong Y; Department of Gastroenterology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, 450052, Henan, China. Electronic address: .
Li J; Department of Cardiology, The Second Affiliated Hospital of Zhengzhou University, Zhengzhou, 450014, Henan, China.
Peng M; Department of Clinical Laboratory, Henan Provincial Third People's Hospital, Zhengzhou, 450006, Henan, China.
Qian Q; Jiangsu Key Laboratory of Marine Pharmaceutical Compound Screening, College of Pharmacy, Jiangsu Ocean University, Lianyungang, 222005, China.
Shi W; Jiangsu Key Laboratory of Marine Pharmaceutical Compound Screening, College of Pharmacy, Jiangsu Ocean University, Lianyungang, 222005, China.
Chen Z; Jiangsu Key Laboratory of Marine Pharmaceutical Compound Screening, College of Pharmacy, Jiangsu Ocean University, Lianyungang, 222005, China.
Liu B; Jiangsu Key Laboratory of Marine Pharmaceutical Compound Screening, College of Pharmacy, Jiangsu Ocean University, Lianyungang, 222005, China. Electronic address: .
Pokaż więcej
Źródło:
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2022 Feb 26; Vol. 594, pp. 146-152. Date of Electronic Publication: 2022 Jan 13.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
ATPases Associated with Diverse Cellular Activities/*metabolism
Colorectal Neoplasms/*metabolism
DNA-Binding Proteins/*metabolism
E2F Transcription Factors/*metabolism
Transcription Factors/*metabolism
Tripartite Motif Proteins/*metabolism
Ubiquitin/*metabolism
Ubiquitin-Protein Ligases/*metabolism
Animals ; Cell Proliferation ; Disease Progression ; Gene Expression Profiling ; Gene Expression Regulation, Neoplastic ; HCT116 Cells ; Humans ; Male ; Mice ; Mice, Inbred BALB C ; Neoplasm Transplantation ; Oligonucleotide Array Sequence Analysis ; Proteasome Endopeptidase Complex/metabolism ; RNA Interference
Czasopismo naukowe
Tytuł:
Systematic study on expression and prognosis of E2Fs in human colorectal cancer.
Autorzy:
Ma Y; Nankai University School of Medicine, Nankai University, Tianjin, 300071, China.; Department of Clinical Laboratory, The First Medical Centre, The PLA General Hospital, Beijing, 100853, China.
Wang S; Nankai University School of Medicine, Nankai University, Tianjin, 300071, China.
Bao J; Department of Clinical Laboratory, The First Medical Centre, The PLA General Hospital, Beijing, 100853, China.
Wang C; Nankai University School of Medicine, Nankai University, Tianjin, 300071, China. .; Department of Clinical Laboratory, The First Medical Centre, The PLA General Hospital, Beijing, 100853, China. .
Pokaż więcej
Źródło:
International journal of clinical oncology [Int J Clin Oncol] 2022 Feb; Vol. 27 (2), pp. 362-372. Date of Electronic Publication: 2021 Oct 18.
Typ publikacji:
Journal Article
MeSH Terms:
Colorectal Neoplasms*/diagnosis
Colorectal Neoplasms*/genetics
Gene Expression Regulation, Neoplastic*
E2F Transcription Factors/*genetics
Humans ; Prognosis ; RNA, Messenger
Czasopismo naukowe
Tytuł:
Early 2 factor (E2F) transcription factors contribute to malignant progression and have clinical prognostic value in lower-grade glioma.
Autorzy:
Luo H; Department of Neurosurgery, The Second Affiliated Hospital of Nanchang University, Nanchang, Jiangxi Province, China.
Tao C; Department of Neurosurgery, The Second Affiliated Hospital of Nanchang University, Nanchang, Jiangxi Province, China.; Scientific Research Center, East China Institute of Digital Medical Engineering, Shangrao, Jiangxi Province, China.
Long X; Scientific Research Center, East China Institute of Digital Medical Engineering, Shangrao, Jiangxi Province, China.
Zhu X; Department of Neurosurgery, The Second Affiliated Hospital of Nanchang University, Nanchang, Jiangxi Province, China.; Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China.
Huang K; Department of Neurosurgery, The Second Affiliated Hospital of Nanchang University, Nanchang, Jiangxi Province, China.; Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China.
Pokaż więcej
Źródło:
Bioengineered [Bioengineered] 2021 Dec; Vol. 12 (1), pp. 7765-7779.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Brain Neoplasms*/diagnosis
Brain Neoplasms*/genetics
Brain Neoplasms*/mortality
Brain Neoplasms*/pathology
Glioma*/diagnosis
Glioma*/genetics
Glioma*/mortality
Glioma*/pathology
E2F Transcription Factors/*genetics
Transcriptome/*genetics
Brain/metabolism ; Brain/pathology ; Humans ; Prognosis ; ROC Curve
Czasopismo naukowe
Tytuł:
Simultaneous expression of MMB-FOXM1 complex components enables efficient bypass of senescence.
Autorzy:
Kumari R; MRC Prion Unit at UCL, UCL Institute of Prion Diseases, 33 Cleveland Street, London, W1W 7FF, UK.
Hummerich H; MRC Prion Unit at UCL, UCL Institute of Prion Diseases, 33 Cleveland Street, London, W1W 7FF, UK.
Shen X; The UCL Cancer Institute, Paul O'Gorman Building, 72 Huntley Street, London, WC1E 6DD, UK.
Fischer M; Computational Biology Group, Leibniz Institute on Aging - Fritz Lipmann Institute (FLI), Beutenbergstraße 11, 07745, Jena, Germany.
Litovchick L; Division of Hematology, Oncology and Palliative Care, Department of Internal Medicine, Massey Cancer Centre, Virginia Commonwealth University, Richmond, VA, 23298, USA.
Mittnacht S; The UCL Cancer Institute, Paul O'Gorman Building, 72 Huntley Street, London, WC1E 6DD, UK.
DeCaprio JA; Department of Medical Oncology, Dana-Farber Cancer Institute, 450 Brookline Avenue, Boston, MA, 02215, USA.; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, 450 Brookline Avenue, Boston, MA, 02115, USA.
Jat PS; MRC Prion Unit at UCL, UCL Institute of Prion Diseases, 33 Cleveland Street, London, W1W 7FF, UK. .
Pokaż więcej
Źródło:
Scientific reports [Sci Rep] 2021 Nov 02; Vol. 11 (1), pp. 21506. Date of Electronic Publication: 2021 Nov 02.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Cellular Senescence*
Gene Expression Regulation*
Breast/*metabolism
Cell Cycle Proteins/*metabolism
Fibroblasts/*metabolism
Forkhead Box Protein M1/*metabolism
Multiprotein Complexes/*metabolism
Trans-Activators/*metabolism
Breast/cytology ; Cell Cycle Proteins/genetics ; Cyclin-Dependent Kinase Inhibitor p21/genetics ; Cyclin-Dependent Kinase Inhibitor p21/metabolism ; E2F Transcription Factors/genetics ; E2F Transcription Factors/metabolism ; Female ; Fibroblasts/cytology ; Forkhead Box Protein M1/genetics ; Humans ; Kv Channel-Interacting Proteins/genetics ; Kv Channel-Interacting Proteins/metabolism ; Multiprotein Complexes/genetics ; Phosphorylation ; Repressor Proteins/genetics ; Repressor Proteins/metabolism ; Retinoblastoma Binding Proteins/genetics ; Retinoblastoma Binding Proteins/metabolism ; Trans-Activators/genetics ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/metabolism ; Ubiquitin-Protein Ligases/genetics ; Ubiquitin-Protein Ligases/metabolism ; YAP-Signaling Proteins/genetics ; YAP-Signaling Proteins/metabolism
Czasopismo naukowe
Tytuł:
DREAM represses distinct targets by cooperating with different THAP domain proteins.
Autorzy:
Gal C; Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Genetics, University of Cambridge, Cambridge, UK.
Carelli FN; Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Genetics, University of Cambridge, Cambridge, UK.
Appert A; Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Genetics, University of Cambridge, Cambridge, UK.
Cerrato C; Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Genetics, University of Cambridge, Cambridge, UK.
Huang N; Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Genetics, University of Cambridge, Cambridge, UK.
Dong Y; Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Genetics, University of Cambridge, Cambridge, UK.
Murphy J; Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Genetics, University of Cambridge, Cambridge, UK.
Frapporti A; Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Genetics, University of Cambridge, Cambridge, UK.
Ahringer J; Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Genetics, University of Cambridge, Cambridge, UK. Electronic address: .
Pokaż więcej
Źródło:
Cell reports [Cell Rep] 2021 Oct 19; Vol. 37 (3), pp. 109835.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Caenorhabditis elegans/*metabolism
Caenorhabditis elegans Proteins/*metabolism
Retinoblastoma Protein/*metabolism
Transcription Factors/*metabolism
Animals ; Animals, Genetically Modified ; Caenorhabditis elegans/genetics ; Caenorhabditis elegans Proteins/genetics ; Cell Cycle Proteins/genetics ; Cell Cycle Proteins/metabolism ; DNA Methylation ; E2F Transcription Factors/genetics ; E2F Transcription Factors/metabolism ; Gene Expression Regulation ; Histones/genetics ; Histones/metabolism ; Promoter Regions, Genetic ; Protein Binding ; Protein Interaction Domains and Motifs ; Retinoblastoma Protein/genetics ; Transcription Factors/genetics
Czasopismo naukowe
Tytuł:
Asxl1 deficiency in embryonic fibroblasts leads to cellular senescence via impairment of the AKT-E2F pathway and Ezh2 inactivation.
Autorzy:
Youn HS; Department of Bioscience and Biotechnology, BK21 Graduate Program, Sejong University, 209 Neungdong-ro, Gwangjin-gu, Seoul, 143-747, Korea.
Kim TY; Department of Bioscience and Biotechnology, BK21 Graduate Program, Sejong University, 209 Neungdong-ro, Gwangjin-gu, Seoul, 143-747, Korea.
Park UH; Department of Bioscience and Biotechnology, BK21 Graduate Program, Sejong University, 209 Neungdong-ro, Gwangjin-gu, Seoul, 143-747, Korea.
Moon ST; Department of Bioscience and Biotechnology, BK21 Graduate Program, Sejong University, 209 Neungdong-ro, Gwangjin-gu, Seoul, 143-747, Korea.
An SJ; Department of Bioscience and Biotechnology, BK21 Graduate Program, Sejong University, 209 Neungdong-ro, Gwangjin-gu, Seoul, 143-747, Korea.
Lee YK; Department of Bioscience and Biotechnology, BK21 Graduate Program, Sejong University, 209 Neungdong-ro, Gwangjin-gu, Seoul, 143-747, Korea.
Hwang JT; Korea Food Research Institute, Bundang-gu, Seongnam-si, 463-746, Korea.
Kim EJ; Department of Molecular Biology, Dankook University, Gyeonggi-do, 448-701, Korea.
Um SJ; Department of Bioscience and Biotechnology, BK21 Graduate Program, Sejong University, 209 Neungdong-ro, Gwangjin-gu, Seoul, 143-747, Korea. .
Pokaż więcej
Źródło:
Scientific reports [Sci Rep] 2017 Jul 12; Vol. 7 (1), pp. 5198. Date of Electronic Publication: 2017 Jul 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cellular Senescence*
E2F Transcription Factors/*antagonists & inhibitors
Embryo, Mammalian/*pathology
Enhancer of Zeste Homolog 2 Protein/*antagonists & inhibitors
Fibroblasts/*pathology
Proto-Oncogene Proteins c-akt/*antagonists & inhibitors
Repressor Proteins/*physiology
Animals ; Cell Proliferation ; Cells, Cultured ; E2F Transcription Factors/genetics ; E2F Transcription Factors/metabolism ; Embryo, Mammalian/metabolism ; Enhancer of Zeste Homolog 2 Protein/genetics ; Enhancer of Zeste Homolog 2 Protein/metabolism ; Fibroblasts/metabolism ; Mice ; Mice, Knockout ; Phosphorylation ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; Reactive Oxygen Species/metabolism ; Signal Transduction
Czasopismo naukowe
Tytuł:
BRD4 regulates cellular senescence in gastric cancer cells via E2F/miR-106b/p21 axis.
Autorzy:
Dong X; Department of Biochemistry, University of Illinois at Urbana-Champaign, Urbana, IL, 61801, USA.
Hu X; Department of Biochemistry, University of Illinois at Urbana-Champaign, Urbana, IL, 61801, USA.
Chen J; Department of Biochemistry, University of Illinois at Urbana-Champaign, Urbana, IL, 61801, USA.
Hu D; Department of Pathology, Fujian Provincial Cancer Hospital, The Affiliated Hospital of Fujian Medical University, Fujian, China, 350108.
Chen LF; Department of Biochemistry, University of Illinois at Urbana-Champaign, Urbana, IL, 61801, USA. .; Institute for Translational Medicine, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, Fujian, China, 350108. .; Department of Medical Biochemistry, College of Medicine, University of Illinois at Urbana-Champaign, Urbana, IL, 61801, USA. .
Pokaż więcej
Źródło:
Cell death & disease [Cell Death Dis] 2018 Feb 12; Vol. 9 (2), pp. 203. Date of Electronic Publication: 2018 Feb 12.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Cellular Senescence*
Cyclin-Dependent Kinase Inhibitor p21/*metabolism
E2F Transcription Factors/*metabolism
MicroRNAs/*metabolism
Neoplasm Proteins/*metabolism
Nuclear Proteins/*metabolism
Stomach Neoplasms/*metabolism
Transcription Factors/*metabolism
Cell Cycle Proteins ; Cell Line, Tumor ; Cyclin-Dependent Kinase Inhibitor p21/genetics ; E2F Transcription Factors/genetics ; Female ; Humans ; Male ; MicroRNAs/genetics ; Neoplasm Proteins/genetics ; Nuclear Proteins/genetics ; Stomach Neoplasms/genetics ; Transcription Factors/genetics
Czasopismo naukowe
Tytuł:
Functional interrelationship between TFII-I and E2F transcription factors at specific cell cycle gene loci.
Autorzy:
Shen Y; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Nar R; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Fan AX; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Aryan M; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Hossain MA; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Gurumurthy A; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Wassel PC; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Tang M; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Lu J; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Strouboulis J; Institute of Molecular Biology and Biotechnology, Foundation of Research and Technology-Hellas, Heraklion, Crete, Greece.
Bungert J; Department of Biochemistry and Molecular Biology, Center for Epigenetics, Genetics Institute, Health Cancer Center, Powell-Gene Therapy Center, University of Florida, Gainesville, Florida.
Pokaż więcej
Źródło:
Journal of cellular biochemistry [J Cell Biochem] 2018 Jan; Vol. 119 (1), pp. 712-722. Date of Electronic Publication: 2017 Jul 31.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Cycle Proteins/*genetics
E2F Transcription Factors/*metabolism
Transcription Factors, TFII/*metabolism
Cell Cycle ; Cell Proliferation ; Chromatin Immunoprecipitation ; Computational Biology/methods ; E2F Transcription Factors/genetics ; Humans ; K562 Cells ; Promoter Regions, Genetic ; Protein Binding ; Transcription Factors, TFII/genetics
Czasopismo naukowe
Tytuł:
RB depletion is required for the continuous growth of tumors initiated by loss of RB.
Autorzy:
Doan A; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
Arand J; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.; Center of Anatomy and Cell Biology, Department of Cell and Developmental Biology, Medical University of Vienna, Vienna, Austria.
Gong D; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.; Department of Bioengineering, Stanford University, Stanford, California, United States of America.
Drainas AP; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
Shue YT; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
Lee MC; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
Zhang S; Department of Biology, Stanford University, Stanford, California, United States of America.
Walter DM; Department of Cancer Biology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.; Abramson Family Cancer Research Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Chaikovsky AC; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
Feldser DM; Department of Cancer Biology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.; Abramson Family Cancer Research Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
Vogel H; Department of Pathology, Stanford University, Stanford, California, United States of America.
Dow LE; Sandra and Edward Meyer Cancer Center, Weill Cornell Medicine, New York, New York, United States of America.; Department of Biochemistry, Weill Cornell Medicine, New York, New York, United States of America.; Department of Medicine, Weill Cornell Medicine, New York, New York, United States of America.
Skotheim JM; Department of Biology, Stanford University, Stanford, California, United States of America.
Sage J; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
Pokaż więcej
Źródło:
PLoS genetics [PLoS Genet] 2021 Dec 08; Vol. 17 (12), pp. e1009941. Date of Electronic Publication: 2021 Dec 08 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Pituitary Neoplasms/*genetics
Retinoblastoma Binding Proteins/*genetics
Thyroid Neoplasms/*genetics
Animals ; Cell Cycle Checkpoints/genetics ; Cell Line, Tumor ; Disease Models, Animal ; E2F Transcription Factors/metabolism ; Female ; Gene Expression Regulation, Neoplastic ; Gene Knockdown Techniques ; Humans ; Male ; Mice ; Mice, Transgenic ; NIH 3T3 Cells ; Pituitary Neoplasms/pathology ; RNA, Small Interfering/metabolism ; Thyroid Neoplasms/pathology
Czasopismo naukowe
Tytuł:
Cancer cells with defective RB and CDKN2A are resistant to the apoptotic effects of rapamycin.
Autorzy:
Chakraborty S; Department of Biological Sciences, Hunter College of the City University of New York, New York, NY, USA; Biochemistry Program, Graduate Center of the City University of New York, NY, New York, USA.
Utter MB; Department of Biological Sciences, Hunter College of the City University of New York, New York, NY, USA; Biochemistry Program, Graduate Center of the City University of New York, NY, New York, USA.
Frias MA; Department of Biological Sciences, Hunter College of the City University of New York, New York, NY, USA.
Foster DA; Department of Biological Sciences, Hunter College of the City University of New York, New York, NY, USA; Biochemistry Program, Graduate Center of the City University of New York, NY, New York, USA; Department of Pharmacology, Weill Cornell Medicine, New York, NY, USA. Electronic address: .
Pokaż więcej
Źródło:
Cancer letters [Cancer Lett] 2021 Dec 01; Vol. 522, pp. 164-170. Date of Electronic Publication: 2021 Sep 23.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Cyclin-Dependent Kinase Inhibitor p16/*genetics
Lung Neoplasms/*drug therapy
Prostatic Neoplasms/*drug therapy
Retinoblastoma Protein/*genetics
Transforming Growth Factor beta/*genetics
Apoptosis/drug effects ; Cell Cycle/drug effects ; Cell Line, Tumor ; Drug Resistance, Neoplasm/drug effects ; E2F Transcription Factors/genetics ; Humans ; Lung Neoplasms/genetics ; Lung Neoplasms/pathology ; Male ; Mutation/genetics ; Phosphorylation/drug effects ; Prostatic Neoplasms/genetics ; Prostatic Neoplasms/pathology ; Signal Transduction/drug effects ; Sirolimus/adverse effects ; Sirolimus/pharmacology
Czasopismo naukowe
Tytuł:
CircularRNA_0119872 regulates the microRNA-582-3p/E2F transcription factor 3 pathway to promote the progression of malignant melanoma.
Autorzy:
Qu J; Department of Dermatology, Linyi Central Hospital, Linyi 276400, Shandong, China.
Yuan C; Department of Dermatology, Dongying People's Hospital, Dongying 257091, Shandong, China.
Jia Q; Department of Dermatology, Haiyang People's Hospital, Haiyang 265100, Shandong, China.
Sun M; Department of Dermatology, Haiyang People's Hospital, Haiyang 265100, Shandong, China.
Jiang M; Department of Dermatology, Haiyang People's Hospital, Haiyang 265100, Shandong, China.
Zuo F; Department of Dermatology, Haiyang People's Hospital, Haiyang 265100, Shandong, China.
Pokaż więcej
Źródło:
Clinics (Sao Paulo, Brazil) [Clinics (Sao Paulo)] 2021 Oct 11; Vol. 76, pp. e3036. Date of Electronic Publication: 2021 Oct 11 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Melanoma*/genetics
MicroRNAs*/genetics
Cell Line, Tumor ; E2F Transcription Factors ; Gene Expression Regulation, Neoplastic ; Humans
Czasopismo naukowe
Tytuł:
Cell cycle transcription control: DREAM/MuvB and RB-E2F complexes.
Autorzy:
Fischer M; a Molecular Oncology, Medical School, University of Leipzig , Leipzig , Germany.; b Department of Medical Oncology , Dana-Farber Cancer Institute , Boston , MA , USA.; c Department of Medicine, Brigham and Women's Hospital , Harvard Medical School , Boston , MA , USA.
Müller GA; a Molecular Oncology, Medical School, University of Leipzig , Leipzig , Germany.
Pokaż więcej
Źródło:
Critical reviews in biochemistry and molecular biology [Crit Rev Biochem Mol Biol] 2017 Dec; Vol. 52 (6), pp. 638-662. Date of Electronic Publication: 2017 Aug 11.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Cycle*
Transcriptional Activation*
E2F Transcription Factors/*metabolism
Kv Channel-Interacting Proteins/*metabolism
Nuclear Proteins/*metabolism
Repressor Proteins/*metabolism
Retinoblastoma Protein/*metabolism
Trans-Activators/*metabolism
Animals ; Cell Differentiation ; E2F Transcription Factors/genetics ; Gene Expression Regulation, Neoplastic ; Humans ; Kv Channel-Interacting Proteins/genetics ; Neoplasms/genetics ; Neoplasms/metabolism ; Nuclear Proteins/genetics ; Protein Interaction Maps ; Repressor Proteins/genetics ; Retinoblastoma Protein/genetics ; Trans-Activators/genetics ; Transcription, Genetic
Czasopismo naukowe
Tytuł:
Effect of HPV E6/E7 siRNA with Chemotherapeutic Agents on the Regulation of TP53/E2F Dynamic Behavior for Cell Fate Decisions.
Autorzy:
Rajasekaran N; Research Institute of Pharmaceutical Science, College of Pharmacy, Seoul National University, Seoul 08826, Republic of Korea.
Jung HS; Research Institute of Pharmaceutical Science, College of Pharmacy, Seoul National University, Seoul 08826, Republic of Korea; ABION Inc. R&D Center, 9th Floor, HanWha Biz Metro Building, 242 Digital-ro, Guro-gu, Seoul 08394, Republic of Korea.
Bae SH; PIPET (Pharmacometrics Institute for Practical Education and Training), College of Medicine, The Catholic University of Korea, Seoul 06591, Republic of Korea.
Chelakkot C; Division of Integrative Biosciences and Biotechnology, Pohang University of Science and Technology (POSTECH), Pohang, Kyungpook 37673, Republic of Korea.
Hong S; Research Institute of Pharmaceutical Science, College of Pharmacy, Seoul National University, Seoul 08826, Republic of Korea.
Choi JS; The center for Anti-cancer Companion Diagnostics, School of Biological Science, Institute of Entrepreneurial BioConvergence, Seoul National University, Seoul 08826, Republic of Korea.
Yim DS; PIPET (Pharmacometrics Institute for Practical Education and Training), College of Medicine, The Catholic University of Korea, Seoul 06591, Republic of Korea.
Oh YK; Research Institute of Pharmaceutical Science, College of Pharmacy, Seoul National University, Seoul 08826, Republic of Korea.
Choi YL; Department of Pathology, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul 06351, Republic of Korea.
Shin YK; Research Institute of Pharmaceutical Science, College of Pharmacy, Seoul National University, Seoul 08826, Republic of Korea; The Center for Anti-cancer CDx, N-Bio, Seoul National University, Seoul 08826, Republic of Korea; Tumor Microenvironment Global Core Research Center, Seoul National University, Seoul 08826, Republic of Korea; Molecular Medicine and Biopharmaceutical Sciences, Graduate School of Convergence Science and Technology, Seoul National University, Seoul 08826, Republic of Korea. Electronic address: .
Pokaż więcej
Źródło:
Neoplasia (New York, N.Y.) [Neoplasia] 2017 Oct; Vol. 19 (10), pp. 735-749. Date of Electronic Publication: 2017 Aug 24.
Typ publikacji:
Journal Article
MeSH Terms:
RNA Interference*
RNA, Small Interfering*
Antineoplastic Agents/*pharmacology
E2F Transcription Factors/*genetics
Oncogene Proteins, Viral/*genetics
Papillomaviridae/*genetics
Tumor Suppressor Protein p53/*genetics
Animals ; Apoptosis/drug effects ; Apoptosis/genetics ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Proliferation/genetics ; Cell Tracking ; Disease Models, Animal ; E2F Transcription Factors/metabolism ; Gene Expression ; Gene Expression Regulation/drug effects ; Genes, Reporter ; Humans ; Mice ; Microscopy, Confocal ; Signal Transduction/drug effects ; Tumor Suppressor Protein p53/metabolism ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł:
MAZ induces MYB expression during the exit from quiescence via the E2F site in the MYB promoter.
Autorzy:
Álvaro-Blanco J; Department of Cancer Biology, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC-UAM, Madrid 28029, Spain.
Urso K; Gene regulation in cardiovascular remodeling and inflammation group, Centro Nacional de Investigaciones Cardiovasculares, Madrid 28029, Spain.
Chiodo Y; Department of Cancer Biology, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC-UAM, Madrid 28029, Spain.
Martín-Cortázar C; Department of Cancer Biology, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC-UAM, Madrid 28029, Spain.
Kourani O; Department of Cancer Biology, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC-UAM, Madrid 28029, Spain.
Arco PG; Gene regulation in cardiovascular remodeling and inflammation group, Centro Nacional de Investigaciones Cardiovasculares, Madrid 28029, Spain.; Department of Molecular Biology, Universidad Autónoma de Madrid, Centro de Biología Molecular, Cantoblanco, Madrid 28049, Spain.; CIBERCV, Spain.
Rodríguez-Martínez M; Department of Cancer Biology, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC-UAM, Madrid 28029, Spain.
Calonge E; Unidad de Inmunopatología del SIDA, Centro Nacional de Microbiología, Majadahonda 28220, Spain.
Alcamí J; Unidad de Inmunopatología del SIDA, Centro Nacional de Microbiología, Majadahonda 28220, Spain.
Redondo JM; Gene regulation in cardiovascular remodeling and inflammation group, Centro Nacional de Investigaciones Cardiovasculares, Madrid 28029, Spain.; CIBERCV, Spain.
Iglesias T; Department of Endocrine and Nervous Systems Pathophysiology, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC-UAM, Madrid 28029, Spain.; CIBERNED, Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas, Spain.
Campanero MR; Department of Cancer Biology, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC-UAM, Madrid 28029, Spain.; CIBERCV, Spain.
Pokaż więcej
Źródło:
Nucleic acids research [Nucleic Acids Res] 2017 Sep 29; Vol. 45 (17), pp. 9960-9975.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression Regulation*
Promoter Regions, Genetic*
DNA-Binding Proteins/*genetics
E2F Transcription Factors/*genetics
G1 Phase/*genetics
Oncogene Proteins v-myb/*genetics
Transcription Factors/*genetics
Binding Sites ; Cell Line, Tumor ; Crk-Associated Substrate Protein/genetics ; Crk-Associated Substrate Protein/metabolism ; DNA-Binding Proteins/antagonists & inhibitors ; DNA-Binding Proteins/metabolism ; E2F Transcription Factors/metabolism ; Genes, Reporter ; HEK293 Cells ; Humans ; Jurkat Cells ; Luciferases/genetics ; Luciferases/metabolism ; Lymphocytes/cytology ; Lymphocytes/metabolism ; Oncogene Proteins v-myb/metabolism ; Osteoblasts/cytology ; Osteoblasts/metabolism ; Protein Binding ; Protein Isoforms/genetics ; Protein Isoforms/metabolism ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Retinoblastoma Protein/genetics ; Retinoblastoma Protein/metabolism ; Transcription Factors/antagonists & inhibitors ; Transcription Factors/metabolism ; Transcription, Genetic
Czasopismo naukowe
Tytuł:
Controlling Depth of Cellular Quiescence by an Rb-E2F Network Switch.
Autorzy:
Kwon JS; Department of Molecular & Cellular Biology, University of Arizona, Tucson, AZ 85721, USA.
Everetts NJ; Department of Molecular & Cellular Biology, University of Arizona, Tucson, AZ 85721, USA.
Wang X; Department of Molecular & Cellular Biology, University of Arizona, Tucson, AZ 85721, USA.
Wang W; Department of Computational and Systems Biology, University of Pittsburgh School of Medicine, Pittsburgh, PA 15260, USA.
Della Croce K; Department of Molecular & Cellular Biology, University of Arizona, Tucson, AZ 85721, USA.
Xing J; Department of Computational and Systems Biology, University of Pittsburgh School of Medicine, Pittsburgh, PA 15260, USA; UPMC-Hillman Cancer Center, University of Pittsburgh, Pittsburgh, PA 15232, USA.
Yao G; Department of Molecular & Cellular Biology, University of Arizona, Tucson, AZ 85721, USA; Arizona Cancer Center, University of Arizona, Tucson, AZ 85719, USA. Electronic address: .
Pokaż więcej
Źródło:
Cell reports [Cell Rep] 2017 Sep 26; Vol. 20 (13), pp. 3223-3235.
Typ publikacji:
Journal Article
MeSH Terms:
Models, Biological*
Cellular Senescence/*genetics
E2F Transcription Factors/*genetics
Retinoblastoma Protein/*genetics
Animals ; Cell Division ; Cell Proliferation/genetics ; E2F Transcription Factors/metabolism ; Fibroblasts ; Gene Regulatory Networks ; Humans ; Rats ; Retinoblastoma Protein/metabolism
Czasopismo naukowe
Tytuł:
SOG1 transcription factor promotes the onset of endoreduplication under salinity stress in Arabidopsis.
Autorzy:
Mahapatra K; Department of Botany, UGC Center for Advanced Studies, The University of Burdwan, Golapbag Campus, Burdwan, West Bengal, 713 104, India.
Roy S; Department of Botany, UGC Center for Advanced Studies, The University of Burdwan, Golapbag Campus, Burdwan, West Bengal, 713 104, India. .
Pokaż więcej
Źródło:
Scientific reports [Sci Rep] 2021 Jun 02; Vol. 11 (1), pp. 11659. Date of Electronic Publication: 2021 Jun 02.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Retracted Publication
MeSH Terms:
Endoreduplication*
Arabidopsis/*genetics
Arabidopsis Proteins/*genetics
Ataxia Telangiectasia Mutated Proteins/*genetics
DNA, Plant/*genetics
Salt Tolerance/*genetics
Transcription Factors/*genetics
Arabidopsis/drug effects ; Arabidopsis/metabolism ; Arabidopsis Proteins/metabolism ; Ataxia Telangiectasia Mutated Proteins/metabolism ; Cell Cycle Proteins/genetics ; Cell Cycle Proteins/metabolism ; Cell Size ; Cyclin B/genetics ; Cyclin B/metabolism ; Cyclin-Dependent Kinases/genetics ; Cyclin-Dependent Kinases/metabolism ; DNA, Plant/metabolism ; E2F Transcription Factors/genetics ; E2F Transcription Factors/metabolism ; G2 Phase Cell Cycle Checkpoints/genetics ; Gene Expression Regulation, Plant ; Plant Cells/drug effects ; Plant Cells/metabolism ; Polyploidy ; Protein Serine-Threonine Kinases/genetics ; Protein Serine-Threonine Kinases/metabolism ; Salt Stress/genetics ; Signal Transduction ; Sodium Chloride/pharmacology ; Transcription Factors/metabolism
Czasopismo naukowe

Ta witryna wykorzystuje pliki cookies do przechowywania informacji na Twoim komputerze. Pliki cookies stosujemy w celu świadczenia usług na najwyższym poziomie, w tym w sposób dostosowany do indywidualnych potrzeb. Korzystanie z witryny bez zmiany ustawień dotyczących cookies oznacza, że będą one zamieszczane w Twoim komputerze. W każdym momencie możesz dokonać zmiany ustawień dotyczących cookies