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Tytuł:
The highly expressed calcium-insensitive synaptotagmin-11 and synaptotagmin-13 modulate insulin secretion.
Autorzy:
Ofori JK; Department of Clinical Sciences Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
Karagiannopoulos A; Department of Clinical Sciences Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
Barghouth M; Islet Pathophysiology, Department of Clinical Sciences Malmö, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
Nagao M; Department of Clinical Sciences Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.; Department of Endocrinology, Diabetes and Metabolism, Graduate School of Medicine, Nippon Medical School, Tokyo, Japan.
Andersson ME; Department of Clinical Sciences Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
Salunkhe VA; Department of Clinical Sciences Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.; Department of Physiology, Metabolism Research Unit, Institute of Neuroscience and Physiology, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
Zhang E; Islet Pathophysiology, Department of Clinical Sciences Malmö, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
Wendt A; Department of Clinical Sciences Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
Eliasson L; Department of Clinical Sciences Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
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Źródło:
Acta physiologica (Oxford, England) [Acta Physiol (Oxf)] 2022 Sep; Vol. 236 (1), pp. e13857. Date of Electronic Publication: 2022 Jul 02.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Calcium*/metabolism
Insulin-Secreting Cells*/metabolism
Glucose/metabolism ; Humans ; Insulin/metabolism ; Insulin Secretion ; Synaptotagmins/genetics ; Synaptotagmins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MAFA and MAFB regulate exocytosis-related genes in human β-cells.
Autorzy:
Cataldo LR; Endocrine Cell Differentiation and Function Group, Stem Cell Centre, Lund University, Lund, Sweden.; Lund University Diabetes Centre, Clinical Research Center, Malmo, Sweden.; The Novo Nordisk Foundation Centre for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark.
Singh T; Endocrine Cell Differentiation and Function Group, Stem Cell Centre, Lund University, Lund, Sweden.; Lund University Diabetes Centre, Clinical Research Center, Malmo, Sweden.
Achanta K; Endocrine Cell Differentiation and Function Group, Stem Cell Centre, Lund University, Lund, Sweden.; Lund University Diabetes Centre, Clinical Research Center, Malmo, Sweden.
Bsharat S; Endocrine Cell Differentiation and Function Group, Stem Cell Centre, Lund University, Lund, Sweden.; Lund University Diabetes Centre, Clinical Research Center, Malmo, Sweden.
Prasad RB; Lund University Diabetes Centre, Clinical Research Center, Malmo, Sweden.; Department of Clinical Sciences in Malmö, Malmo, Sweden.
Luan C; Lund University Diabetes Centre, Clinical Research Center, Malmo, Sweden.
Renström E; Lund University Diabetes Centre, Clinical Research Center, Malmo, Sweden.
Eliasson L; Lund University Diabetes Centre, Clinical Research Center, Malmo, Sweden.; Department of Clinical Sciences in Malmö, Malmo, Sweden.; Islet Cell Exocytosis, Lund University, Lund, Sweden.
Artner I; Endocrine Cell Differentiation and Function Group, Stem Cell Centre, Lund University, Lund, Sweden.; Lund University Diabetes Centre, Clinical Research Center, Malmo, Sweden.
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Źródło:
Acta physiologica (Oxford, England) [Acta Physiol (Oxf)] 2022 Feb; Vol. 234 (2), pp. e13761. Date of Electronic Publication: 2022 Jan 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Diabetes Mellitus, Type 2*/genetics
Diabetes Mellitus, Type 2*/metabolism
Insulin-Secreting Cells*/metabolism
Animals ; Exocytosis ; Humans ; Insulin/metabolism ; Insulin Secretion ; Maf Transcription Factors, Large/genetics ; Maf Transcription Factors, Large/metabolism ; MafB Transcription Factor/genetics ; MafB Transcription Factor/metabolism ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
One more piece in the pancreatic chloride puzzle.
Autorzy:
Eliasson L; Unit of Islet Cell Exocytosis, Lund University Diabetes Centre (LUDC), Department of Clinical Sciences Malmö, Lund University, Malmö, Sweden.; Clinical Research Centre, Skåne University Hospital (SUS), Malmö, Sweden.
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Źródło:
Acta physiologica (Oxford, England) [Acta Physiol (Oxf)] 2022 Jan; Vol. 234 (1), pp. e13737. Date of Electronic Publication: 2021 Nov 01.
Typ publikacji:
Editorial; Comment
MeSH Terms:
Chlorides*
Pancreas*
Opinia redakcyjna
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Islet Function in the Pathogenesis of Cystic Fibrosis-Related Diabetes Mellitus.
Autorzy:
Westholm E; Department of Clinical Sciences in Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
Wendt A; Department of Clinical Sciences in Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
Eliasson L; Department of Clinical Sciences in Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden.
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Źródło:
Clinical medicine insights. Endocrinology and diabetes [Clin Med Insights Endocrinol Diabetes] 2021 Jul 13; Vol. 14, pp. 11795514211031204. Date of Electronic Publication: 2021 Jul 13 (Print Publication: 2021).
Typ publikacji:
Journal Article; Review
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The calcium channel subunit gamma-4 is regulated by MafA and necessary for pancreatic beta-cell specification.
Autorzy:
Luan C; 1Lund University Diabetes Center, Department of Clinical Sciences Malmö, Lund University, 202 13 Malmö, Sweden.
Ye Y; 1Lund University Diabetes Center, Department of Clinical Sciences Malmö, Lund University, 202 13 Malmö, Sweden.
Singh T; 2Stem Cell Center, Department of Laboratory Medicine, Lund University, 221 85 Lund, Sweden.
Barghouth M; 1Lund University Diabetes Center, Department of Clinical Sciences Malmö, Lund University, 202 13 Malmö, Sweden.
Eliasson L; 1Lund University Diabetes Center, Department of Clinical Sciences Malmö, Lund University, 202 13 Malmö, Sweden.
Artner I; 1Lund University Diabetes Center, Department of Clinical Sciences Malmö, Lund University, 202 13 Malmö, Sweden.; 2Stem Cell Center, Department of Laboratory Medicine, Lund University, 221 85 Lund, Sweden.
Zhang E; 1Lund University Diabetes Center, Department of Clinical Sciences Malmö, Lund University, 202 13 Malmö, Sweden.
Renström E; 1Lund University Diabetes Center, Department of Clinical Sciences Malmö, Lund University, 202 13 Malmö, Sweden.
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Źródło:
Communications biology [Commun Biol] 2019 Mar 15; Vol. 2, pp. 106. Date of Electronic Publication: 2019 Mar 15 (Print Publication: 2019).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation*
Calcium Channels, N-Type/*genetics
Calcium Channels, N-Type/*metabolism
Insulin-Secreting Cells/*metabolism
Maf Transcription Factors, Large/*metabolism
Animals ; Biomarkers ; Calcium/metabolism ; Calcium Signaling ; Gene Expression ; Glucose/metabolism ; Humans ; Insulin Secretion ; Mice ; Mice, Knockout ; Models, Biological ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
miR-483-5p associates with obesity and insulin resistance and independently associates with new onset diabetes mellitus and cardiovascular disease.
Autorzy:
Gallo W; Hypertension and Cardiovascular Disease, Department of Clinical Sciences-Malmö, Lund University, Malmö, Sweden.; Lund University Diabetes Centre, Skåne University Hospital, Malmö, Sweden.
Esguerra JLS; Lund University Diabetes Centre, Skåne University Hospital, Malmö, Sweden.; Islet Cell Exocytosis, Department of Clinical Sciences-Malmö, Lund University, Malmö, Sweden.
Eliasson L; Lund University Diabetes Centre, Skåne University Hospital, Malmö, Sweden.; Islet Cell Exocytosis, Department of Clinical Sciences-Malmö, Lund University, Malmö, Sweden.
Melander O; Hypertension and Cardiovascular Disease, Department of Clinical Sciences-Malmö, Lund University, Malmö, Sweden.; Lund University Diabetes Centre, Skåne University Hospital, Malmö, Sweden.
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Źródło:
PloS one [PLoS One] 2018 Nov 08; Vol. 13 (11), pp. e0206974. Date of Electronic Publication: 2018 Nov 08 (Print Publication: 2018).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cardiovascular Diseases/*diagnosis
Diabetes Mellitus, Type 2/*diagnosis
Insulin Resistance/*genetics
MicroRNAs/*blood
Obesity/*pathology
Aged ; Biomarkers/blood ; Body Mass Index ; Cardiovascular Diseases/epidemiology ; Cardiovascular Diseases/genetics ; Cross-Sectional Studies ; Diabetes Mellitus, Type 2/epidemiology ; Diabetes Mellitus, Type 2/genetics ; Female ; Humans ; Incidence ; Insulin/blood ; Lipoproteins, HDL/blood ; Logistic Models ; Male ; Middle Aged ; Obesity/genetics ; Odds Ratio ; Risk Factors ; Triglycerides/blood
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MiR-335 overexpression impairs insulin secretion through defective priming of insulin vesicles.
Autorzy:
Salunkhe VA; Department of Clinical Sciences Malmö, Islet Cell Exocytosis Lund University Diabetes Centre Lund University, Malmö, Sweden.
Ofori JK; Department of Clinical Sciences Malmö, Islet Cell Exocytosis Lund University Diabetes Centre Lund University, Malmö, Sweden.
Gandasi NR; Department of Medical Cell Biology, Uppsala University, Uppsala, Sweden.
Salö SA; Department of Clinical Sciences Malmö, Islet Cell Exocytosis Lund University Diabetes Centre Lund University, Malmö, Sweden.
Hansson S; Department of Clinical Sciences Malmö, Islet Cell Exocytosis Lund University Diabetes Centre Lund University, Malmö, Sweden.
Andersson ME; Department of Clinical Sciences Malmö, Islet Cell Exocytosis Lund University Diabetes Centre Lund University, Malmö, Sweden.
Wendt A; Department of Clinical Sciences Malmö, Islet Cell Exocytosis Lund University Diabetes Centre Lund University, Malmö, Sweden.
Barg S; Department of Medical Cell Biology, Uppsala University, Uppsala, Sweden.
Esguerra JLS; Department of Clinical Sciences Malmö, Islet Cell Exocytosis Lund University Diabetes Centre Lund University, Malmö, Sweden lena.eliasson@med.lu.se .
Eliasson L; Department of Clinical Sciences Malmö, Islet Cell Exocytosis Lund University Diabetes Centre Lund University, Malmö, Sweden lena.eliasson@med.lu.se .
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Źródło:
Physiological reports [Physiol Rep] 2017 Nov; Vol. 5 (21).
Typ publikacji:
Journal Article
MeSH Terms:
Diabetes Mellitus, Type 2/*metabolism
Insulin/*metabolism
MicroRNAs/*metabolism
Animals ; Cell Line ; Disease Models, Animal ; Exocytosis ; Humans ; Insulin Secretion ; Insulin-Secreting Cells/metabolism ; Islets of Langerhans/metabolism ; Munc18 Proteins/metabolism ; Rats ; Synaptosomal-Associated Protein 25/metabolism ; Synaptotagmins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Elevated miR-130a/miR130b/miR-152 expression reduces intracellular ATP levels in the pancreatic beta cell.
Autorzy:
Ofori JK; Islet Cell Exocytosis, Department of Clinical Sciences-Malmö, Lund University, Malmö, 205 02, Sweden.; Lund University Diabetes Centre, Skåne University Hospital, Lund and Malmö, Sweden.
Salunkhe VA; Islet Cell Exocytosis, Department of Clinical Sciences-Malmö, Lund University, Malmö, 205 02, Sweden.; Lund University Diabetes Centre, Skåne University Hospital, Lund and Malmö, Sweden.
Bagge A; Lund University Diabetes Centre, Skåne University Hospital, Lund and Malmö, Sweden.; Molecular Metabolism, Department of Clinical Sciences-Malmö, Lund University, Malmö, 20502, Sweden.
Vishnu N; Lund University Diabetes Centre, Skåne University Hospital, Lund and Malmö, Sweden.; Molecular Metabolism, Department of Clinical Sciences-Malmö, Lund University, Malmö, 20502, Sweden.
Nagao M; Islet Cell Exocytosis, Department of Clinical Sciences-Malmö, Lund University, Malmö, 205 02, Sweden.; Lund University Diabetes Centre, Skåne University Hospital, Lund and Malmö, Sweden.
Mulder H; Lund University Diabetes Centre, Skåne University Hospital, Lund and Malmö, Sweden.; Molecular Metabolism, Department of Clinical Sciences-Malmö, Lund University, Malmö, 20502, Sweden.
Wollheim CB; Lund University Diabetes Centre, Skåne University Hospital, Lund and Malmö, Sweden.; Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, 1211, Switzerland.
Eliasson L; Islet Cell Exocytosis, Department of Clinical Sciences-Malmö, Lund University, Malmö, 205 02, Sweden.; Lund University Diabetes Centre, Skåne University Hospital, Lund and Malmö, Sweden.
Esguerra JL; Islet Cell Exocytosis, Department of Clinical Sciences-Malmö, Lund University, Malmö, 205 02, Sweden.; Lund University Diabetes Centre, Skåne University Hospital, Lund and Malmö, Sweden.
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Źródło:
Scientific reports [Sci Rep] 2017 Mar 23; Vol. 7, pp. 44986. Date of Electronic Publication: 2017 Mar 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation*
RNA Interference*
Adenosine Triphosphate/*metabolism
Insulin-Secreting Cells/*metabolism
MicroRNAs/*genetics
Adenosine Diphosphate ; Animals ; Cell Line ; Cells, Cultured ; Cytoplasm/metabolism ; Diabetes Mellitus, Type 2/metabolism ; Gene Silencing ; Glucose/metabolism ; Glucose Intolerance ; Humans ; Insulin/metabolism ; Islets of Langerhans/metabolism ; RNA, Messenger/genetics ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Osteopontin Affects Insulin Vesicle Localization and Ca2+ Homeostasis in Pancreatic Beta Cells from Female Mice.
Autorzy:
Wendt A; Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Department of Clinical Sciences in Malmö, Lund University, Clinical Research Centre, Malmö, Sweden.
Mollet IG; Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Department of Clinical Sciences in Malmö, Lund University, Clinical Research Centre, Malmö, Sweden.
Knutsson A; Vessel Wall Biology, Department of Experimental Medical Science, Lund University, Biomedical Center, Lund, Sweden.
Bolmgren VS; Vessel Wall Biology, Department of Experimental Medical Science, Lund University, Biomedical Center, Lund, Sweden.
Hultgårdh-Nilsson A; Vessel Wall Biology, Department of Experimental Medical Science, Lund University, Biomedical Center, Lund, Sweden.
Gomez MF; Unit of Vascular excitation-transcription (ET) coupling, Lund University Diabetes Centre, Department of Clinical Sciences in Malmö, Lund University, Clinical Research Centre, Malmö, Sweden.
Eliasson L; Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Department of Clinical Sciences in Malmö, Lund University, Clinical Research Centre, Malmö, Sweden.
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Źródło:
PloS one [PLoS One] 2017 Jan 20; Vol. 12 (1), pp. e0170498. Date of Electronic Publication: 2017 Jan 20 (Print Publication: 2017).
Typ publikacji:
Journal Article
MeSH Terms:
Calcium/*metabolism
Insulin-Secreting Cells/*physiology
Inulin/*physiology
Osteopontin/*physiology
Animals ; Calcium/physiology ; Female ; Homeostasis/physiology ; Insulin-Secreting Cells/ultrastructure ; Inulin/metabolism ; Mice ; Mice, Knockout ; Microscopy, Electron, Transmission ; Real-Time Polymerase Chain Reaction ; Secretory Vesicles/physiology ; Secretory Vesicles/ultrastructure
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Inhibition of phosphodiesterase 3, 4, and 5 induces endolymphatic hydrops in mouse inner ear, as evaluated with repeated 9.4T MRI.
Autorzy:
Degerman E; a Department of Experimental Medical Sciences, Section for Diabetes , Metabolism and Endocrinology Lund University Diabetes Centre , Lund , Sweden.
In 't Zandt R; b Lund University Bioimaging Center, Lund University , Lund , Sweden.
Pålbrink A; a Department of Experimental Medical Sciences, Section for Diabetes , Metabolism and Endocrinology Lund University Diabetes Centre , Lund , Sweden.
Eliasson L; c Department of Clinical Sciences in Malmö, Islet Cell Exocytosis , Lund University Diabetes Centre, Lund University , Lund , Sweden.; d Clinical Research Centre, Skåne University Hospital , Malmö , Sweden.
Cayé-Thomasen P; e Department of Otorhinolaryngology, Head and Neck Surgery , Copenhagen University Hospital Rigshospitalet , Copenhagen , Denmark.
Magnusson M; f Department of Clinical Sciences, Section for Otorhinolaryngology , Lund University & Skåne University Hospital , Lund , Sweden.
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Źródło:
Acta oto-laryngologica [Acta Otolaryngol] 2017 Jan; Vol. 137 (1), pp. 8-15. Date of Electronic Publication: 2016 Aug 15.
Typ publikacji:
Journal Article
MeSH Terms:
Phosphodiesterase Inhibitors*
Endolymphatic Hydrops/*enzymology
Phosphoric Diester Hydrolases/*metabolism
Saccule and Utricle/*enzymology
Animals ; Endolymphatic Hydrops/diagnostic imaging ; Female ; Humans ; Magnetic Resonance Imaging ; Mice, Inbred CBA ; Quinolones ; Rolipram ; Sildenafil Citrate
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Rosuvastatin Treatment Affects Both Basal and Glucose-Induced Insulin Secretion in INS-1 832/13 Cells.
Autorzy:
Salunkhe VA; Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Dept. Clinical Sciences in Malmö, Lund University, Clinical Research Centre, SUS Malmö, Malmö, Sweden.
Elvstam O; Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Dept. Clinical Sciences in Malmö, Lund University, Clinical Research Centre, SUS Malmö, Malmö, Sweden.
Eliasson L; Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Dept. Clinical Sciences in Malmö, Lund University, Clinical Research Centre, SUS Malmö, Malmö, Sweden.
Wendt A; Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Dept. Clinical Sciences in Malmö, Lund University, Clinical Research Centre, SUS Malmö, Malmö, Sweden.
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Źródło:
PloS one [PLoS One] 2016 Mar 17; Vol. 11 (3), pp. e0151592. Date of Electronic Publication: 2016 Mar 17 (Print Publication: 2016).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Glucose/*pharmacology
Insulin/*metabolism
Insulin-Secreting Cells/*drug effects
Rosuvastatin Calcium/*pharmacology
Animals ; Calcium/metabolism ; Calcium Channels/metabolism ; Cell Line ; Diazoxide/pharmacology ; Dose-Response Relationship, Drug ; Hydroxymethylglutaryl-CoA Reductase Inhibitors/pharmacology ; Insulin Secretion ; Insulin-Secreting Cells/metabolism ; Ion Channel Gating/drug effects ; KATP Channels/metabolism ; Membrane Potentials/drug effects ; Mevalonic Acid/pharmacology ; Patch-Clamp Techniques ; Rats ; Vasodilator Agents/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Calcium current inactivation rather than pool depletion explains reduced exocytotic rate with prolonged stimulation in insulin-secreting INS-1 832/13 cells.
Autorzy:
Pedersen MG; Islet Cell Exocytosis, Lund University Diabetes Centre, Department of Clinical Sciences Malmö, Lund University, Malmö, Sweden.
Salunkhe VA; Islet Cell Exocytosis, Lund University Diabetes Centre, Department of Clinical Sciences Malmö, Lund University, Malmö, Sweden.
Svedin E; Center for Infectious Medicine, Department of Medicine, The Karolinska Institute, Huddinge University Hospital, Stockholm, Sweden.
Edlund A; Islet Cell Exocytosis, Lund University Diabetes Centre, Department of Clinical Sciences Malmö, Lund University, Malmö, Sweden.
Eliasson L; Islet Cell Exocytosis, Lund University Diabetes Centre, Department of Clinical Sciences Malmö, Lund University, Malmö, Sweden.
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Źródło:
PloS one [PLoS One] 2014 Aug 08; Vol. 9 (8), pp. e103874. Date of Electronic Publication: 2014 Aug 08 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Models, Biological*
Calcium/*metabolism
Exocytosis/*physiology
Insulin/*metabolism
Insulin-Secreting Cells/*physiology
Animals ; Cell Line, Tumor ; Cell Membrane/physiology ; Electric Capacitance ; Patch-Clamp Techniques ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Genome-wide DNA methylation analysis of human pancreatic islets from type 2 diabetic and non-diabetic donors identifies candidate genes that influence insulin secretion.
Autorzy:
Dayeh T; Epigenetics and Diabetes, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
Volkov P; Epigenetics and Diabetes, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
Salö S; Epigenetics and Diabetes, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
Hall E; Epigenetics and Diabetes, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
Nilsson E; Epigenetics and Diabetes, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
Olsson AH; Epigenetics and Diabetes, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
Kirkpatrick CL; Department of Cell Physiology and Metabolism, University Medical Center, Geneva, Switzerland.
Wollheim CB; Department of Cell Physiology and Metabolism, University Medical Center, Geneva, Switzerland.
Eliasson L; Islet Cell Exocytosis, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
Rönn T; Epigenetics and Diabetes, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
Bacos K; Epigenetics and Diabetes, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
Ling C; Epigenetics and Diabetes, Department of Clinical Sciences, Lund University Diabetes Centre, Lund University, CRC, Malmö, Sweden.
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Źródło:
PLoS genetics [PLoS Genet] 2014 Mar 06; Vol. 10 (3), pp. e1004160. Date of Electronic Publication: 2014 Mar 06 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epigenesis, Genetic*
DNA Methylation/*genetics
Diabetes Mellitus, Type 2/*genetics
Insulin/*genetics
CpG Islands/genetics ; Diabetes Mellitus, Type 2/metabolism ; Diabetes Mellitus, Type 2/pathology ; Disease Susceptibility ; Exocytosis/genetics ; Genome, Human ; Humans ; Insulin/metabolism ; Insulin Secretion ; Insulin-Secreting Cells/metabolism ; Islets of Langerhans/metabolism ; Islets of Langerhans/pathology ; Promoter Regions, Genetic
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
New drug for treatment of insulin-resistant complication?
Autorzy:
Eliasson L
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Źródło:
Acta physiologica (Oxford, England) [Acta Physiol (Oxf)] 2013 Mar; Vol. 207 (3), pp. 432-3.
Typ publikacji:
Editorial; Comment
MeSH Terms:
Insulin Resistance*
Adrenomedullin/*pharmacology
Aorta/*drug effects
Diabetes Mellitus, Experimental/*drug therapy
Diabetic Angiopathies/*prevention & control
Vascular Calcification/*prevention & control
Animals ; Male
Opinia redakcyjna
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Differential glucose-regulation of microRNAs in pancreatic islets of non-obese type 2 diabetes model Goto-Kakizaki rat.
Autorzy:
Esguerra JL; Islet Cell Exocytosis, Department of Clinical Sciences-Malmö, Lund University, Malmö, Sweden. />Bolmeson C
Cilio CM
Eliasson L
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Źródło:
PloS one [PLoS One] 2011 Apr 07; Vol. 6 (4), pp. e18613. Date of Electronic Publication: 2011 Apr 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Diabetes Mellitus, Type 2/*metabolism
Glucose/*pharmacology
Islets of Langerhans/*drug effects
Islets of Langerhans/*metabolism
MicroRNAs/*genetics
Animals ; Gene Expression Profiling ; In Vitro Techniques ; Polymerase Chain Reaction ; Rats ; Rats, Wistar
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Beta-cell specific deletion of Dicer1 leads to defective insulin secretion and diabetes mellitus.
Autorzy:
Kalis M; Cellular Autoimmunity Unit, Lund University Diabetes Center, Department of Clinical Sciences, Lund University, Malmö University Hospital, Malmö, Sweden.
Bolmeson C
Esguerra JL
Gupta S
Edlund A
Tormo-Badia N
Speidel D
Holmberg D
Mayans S
Khoo NK
Wendt A
Eliasson L
Cilio CM
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Źródło:
PloS one [PLoS One] 2011; Vol. 6 (12), pp. e29166. Date of Electronic Publication: 2011 Dec 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Deletion*
DEAD-box RNA Helicases/*genetics
Diabetes Mellitus, Experimental/*physiopathology
Insulin/*metabolism
Islets of Langerhans/*metabolism
Ribonuclease III/*genetics
Animals ; Apoptosis ; Blood Glucose/metabolism ; Cell Proliferation ; Diabetes Mellitus, Experimental/genetics ; Flow Cytometry ; Gene Knockdown Techniques ; Homeostasis ; Immunohistochemistry ; Insulin Secretion ; Islets of Langerhans/pathology ; Islets of Langerhans/ultrastructure ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Why treatment fails in type 2 diabetes.
Autorzy:
Rosengren A; Lund University Diabetes Centre, Malmo, Sweden.
Jing X
Eliasson L
Renström E
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Źródło:
PLoS medicine [PLoS Med] 2008 Oct 28; Vol. 5 (10), pp. e215.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Diabetes Mellitus, Type 2/*drug therapy
Hypoglycemic Agents/*therapeutic use
Sulfonylurea Compounds/*therapeutic use
Animals ; Humans ; Mice ; Treatment Outcome
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
A K ATP channel-dependent pathway within alpha cells regulates glucagon release from both rodent and human islets of Langerhans.
Autorzy:
MacDonald PE; Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Oxford, United Kingdom. />De Marinis YZ
Ramracheya R
Salehi A
Ma X
Johnson PR
Cox R
Eliasson L
Rorsman P
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Źródło:
PLoS biology [PLoS Biol] 2007 Jun; Vol. 5 (6), pp. e143.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Glucagon/*metabolism
Glucagon-Secreting Cells/*metabolism
Glucose/*metabolism
Potassium Channels/*metabolism
Animals ; Calcium/metabolism ; Calcium Channels, N-Type/metabolism ; Exocytosis/physiology ; Female ; Humans ; Mice ; Mice, Inbred BALB C ; Mice, Knockout ; Sodium Channels/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
R-type Ca(2+)-channel-evoked CICR regulates glucose-induced somatostatin secretion.
Autorzy:
Zhang Q; Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford OX3 7LJ, UK.
Bengtsson M
Partridge C
Salehi A
Braun M
Cox R
Eliasson L
Johnson PR
Renström E
Schneider T
Berggren PO
Göpel S
Ashcroft FM
Rorsman P
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Źródło:
Nature cell biology [Nat Cell Biol] 2007 Apr; Vol. 9 (4), pp. 453-60. Date of Electronic Publication: 2007 Mar 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Calcium/*metabolism
Calcium Channels, R-Type/*physiology
Glucose/*pharmacology
Somatostatin/*metabolism
Animals ; Calcium/pharmacology ; Calcium Channels, R-Type/genetics ; Cytophotometry ; Diazoxide/pharmacology ; Dose-Response Relationship, Drug ; Electrophysiology ; Immunohistochemistry ; In Vitro Techniques ; Islets of Langerhans/cytology ; Islets of Langerhans/drug effects ; Islets of Langerhans/metabolism ; Isradipine/pharmacology ; Macrocyclic Compounds/pharmacology ; Mannoheptulose/pharmacology ; Membrane Potentials/drug effects ; Mice ; Mice, Inbred C57BL ; Mice, Inbred Strains ; Mice, Knockout ; Microscopy, Confocal ; Oxazoles/pharmacology ; Potassium/pharmacology ; Potassium Channel Blockers/pharmacology ; Potassium Channels/physiology ; Ryanodine/pharmacology ; Somatostatin-Secreting Cells/drug effects ; Somatostatin-Secreting Cells/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
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