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Tytuł:
Hypertensive Pressure Mechanosensing Alone Triggers Lipid Droplet Accumulation and Transdifferentiation of Vascular Smooth Muscle Cells to Foam Cells.
Autorzy:
Swiatlowska P; School of Engineering and Materials Science, Queen Mary University of London, London, E1 4NS, UK.
Tipping W; Department of Pure and Applied Chemistry, University of Strathclyde, Glasgow, G1 1QA, UK.
Marhuenda E; School of Engineering and Materials Science, Queen Mary University of London, London, E1 4NS, UK.
Severi P; School of Engineering and Materials Science, Queen Mary University of London, London, E1 4NS, UK.; Department of Translational Medicine, Laboratory for Technologies of Advanced Therapies (LTTA), University of Ferrara, Ferrara, 44121, Italy.
Fomin V; Y.ai, New York, 10037, USA.
Yang Z; William Harvey Research Institute, Queen Mary University of London, London, EC1M 6BQ, UK.
Xiao Q; William Harvey Research Institute, Queen Mary University of London, London, EC1M 6BQ, UK.
Graham D; Department of Pure and Applied Chemistry, University of Strathclyde, Glasgow, G1 1QA, UK.
Shanahan C; School of Cardiovascular Medicine and Sciences, King's College London, London, SE5 9NU, UK.
Iskratsch T; School of Engineering and Materials Science, Queen Mary University of London, London, E1 4NS, UK.
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Źródło:
Advanced science (Weinheim, Baden-Wurttemberg, Germany) [Adv Sci (Weinh)] 2024 Mar; Vol. 11 (9), pp. e2308686. Date of Electronic Publication: 2023 Dec 25.
Typ publikacji:
Journal Article
MeSH Terms:
Foam Cells*/metabolism
Foam Cells*/pathology
Atherosclerosis*/genetics
Humans ; Muscle, Smooth, Vascular/metabolism ; Muscle, Smooth, Vascular/pathology ; Lipid Droplets/metabolism ; Lipid Droplets/pathology ; Cell Transdifferentiation ; Epigenesis, Genetic
Czasopismo naukowe
Tytuł:
The effects of glycosylation modifications on monocyte recruitment and foam cell formation in atherosclerosis.
Autorzy:
Teng D; Yantai Yuhuangding Hospital affiliated to Qingdao University, Yantai, Shandong, People's Republic of China; Qingdao University, Qingdao, Shandong, People's Republic of China.
Wang W; Yantai Yuhuangding Hospital affiliated to Qingdao University, Yantai, Shandong, People's Republic of China; Qingdao University, Qingdao, Shandong, People's Republic of China.
Jia W; Yantai Yuhuangding Hospital affiliated to Qingdao University, Yantai, Shandong, People's Republic of China; Qingdao University, Qingdao, Shandong, People's Republic of China.
Song J; Yantai Yuhuangding Hospital affiliated to Qingdao University, Yantai, Shandong, People's Republic of China; Qingdao University, Qingdao, Shandong, People's Republic of China.
Gong L; Yantai Yuhuangding Hospital affiliated to Qingdao University, Yantai, Shandong, People's Republic of China.
Zhong L; Yantai Yuhuangding Hospital affiliated to Qingdao University, Yantai, Shandong, People's Republic of China. Electronic address: .
Yang J; Yantai Yuhuangding Hospital affiliated to Qingdao University, Yantai, Shandong, People's Republic of China; Qingdao University, Qingdao, Shandong, People's Republic of China. Electronic address: .
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Źródło:
Biochimica et biophysica acta. Molecular basis of disease [Biochim Biophys Acta Mol Basis Dis] 2024 Mar; Vol. 1870 (3), pp. 167027. Date of Electronic Publication: 2024 Jan 17.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Foam Cells*
Atherosclerosis*/metabolism
Humans ; Monocytes/metabolism ; Glycosylation ; Macrophages/metabolism
Czasopismo naukowe
Tytuł:
HIV Infection Drives Foam Cell Formation via NLRP3 Inflammasome Activation.
Autorzy:
Caocci M; Department of Microbiology, Immunology, and Inflammation, Center for Neurovirology and Gene Editing, 3500 N Broad St. MERB 760, Lewis Katz School of Medicine at Temple University, Philadelphia, PA 19140, USA.
Niu M; Department of Neurological Sciences, University of Nebraska Medical Center, Omaha, NE 68198, USA.
Fox HS; Department of Neurological Sciences, University of Nebraska Medical Center, Omaha, NE 68198, USA.
Burdo TH; Department of Microbiology, Immunology, and Inflammation, Center for Neurovirology and Gene Editing, 3500 N Broad St. MERB 760, Lewis Katz School of Medicine at Temple University, Philadelphia, PA 19140, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Feb 17; Vol. 25 (4). Date of Electronic Publication: 2024 Feb 17.
Typ publikacji:
Journal Article
MeSH Terms:
Atherosclerosis*/immunology
Foam Cells*/immunology
HIV Infections*/complications
HIV Infections*/immunology
Humans ; Cytokines ; Inflammasomes/metabolism ; Interleukin-1beta/metabolism ; Leukocytes, Mononuclear/metabolism ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism
Czasopismo naukowe
Tytuł:
Bioactive TNIIIA2 Sequence in Tenascin-C Is Responsible for Macrophage Foam Cell Transformation; Potential of FNIII14 Peptide Derived from Fibronectin in Suppression of Atherosclerotic Plaque Formation.
Autorzy:
Iyoda T; Department of Pharmacy, Faculty of Pharmaceutical Sciences, Sanyo-Onoda City University, Sanyo-Onoda 756-0884, Yamaguchi, Japan.; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, Noda 278-8510, Chiba, Japan.
Ohishi A; Department of Endocrinology and Metabolism, Faculty of Medicine, University of Tsukuba, Tsukuba 305-8575, Ibaraki, Japan.
Wang Y; Department of Endocrinology and Metabolism, Faculty of Medicine, University of Tsukuba, Tsukuba 305-8575, Ibaraki, Japan.
Yokoyama MS; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, Noda 278-8510, Chiba, Japan.
Kazama M; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, Noda 278-8510, Chiba, Japan.
Okita N; Department of Pharmacy, Faculty of Pharmaceutical Sciences, Sanyo-Onoda City University, Sanyo-Onoda 756-0884, Yamaguchi, Japan.
Inouye S; Department of Pharmacy, Faculty of Pharmaceutical Sciences, Sanyo-Onoda City University, Sanyo-Onoda 756-0884, Yamaguchi, Japan.
Nakagawa Y; Department of Endocrinology and Metabolism, Faculty of Medicine, University of Tsukuba, Tsukuba 305-8575, Ibaraki, Japan.; Department of Complex Biosystem Research, Institute of Natural Medicine, University of Toyama, Toyama 930-0194, Toyama, Japan.
Shimano H; Department of Endocrinology and Metabolism, Faculty of Medicine, University of Tsukuba, Tsukuba 305-8575, Ibaraki, Japan.
Fukai F; Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science, Noda 278-8510, Chiba, Japan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2024 Feb 02; Vol. 25 (3). Date of Electronic Publication: 2024 Feb 02.
Typ publikacji:
Journal Article
MeSH Terms:
Atherosclerosis*
Foam Cells*/metabolism
Plaque, Atherosclerotic*
Animals ; Mice ; Extracellular Matrix Proteins ; Fibronectins/metabolism ; Lipids ; Peptides/chemistry ; Tenascin/metabolism
Czasopismo naukowe
Tytuł:
Trimethylamine N-oxide promotes oxidative stress and lipid accumulation in macrophage foam cells via the Nrf2/ABCA1 pathway.
Autorzy:
Luo Z; Department of Laboratory Diagnostics, the First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, NanGang, Harbin, Heilongjiang, 150001, People's Republic of China.
Yu X; Department of Laboratory Diagnostics, the First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, NanGang, Harbin, Heilongjiang, 150001, People's Republic of China.
Wang C; Department of Laboratory Diagnostics, the First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, NanGang, Harbin, Heilongjiang, 150001, People's Republic of China.
Zhao H; Department of Laboratory Diagnostics, the First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, NanGang, Harbin, Heilongjiang, 150001, People's Republic of China.
Wang X; Department of Laboratory Diagnostics, the First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, NanGang, Harbin, Heilongjiang, 150001, People's Republic of China.
Guan X; Department of Laboratory Diagnostics, the First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, NanGang, Harbin, Heilongjiang, 150001, People's Republic of China. .
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Źródło:
Journal of physiology and biochemistry [J Physiol Biochem] 2024 Feb; Vol. 80 (1), pp. 67-79. Date of Electronic Publication: 2023 Nov 07.
Typ publikacji:
Meta-Analysis; Journal Article
MeSH Terms:
Atherosclerosis*/metabolism
Foam Cells*
Methylamines*/metabolism
Plaque, Atherosclerotic*
Humans ; ATP Binding Cassette Transporter 1/genetics ; Lipids/pharmacology ; Macrophages/metabolism ; NF-E2-Related Factor 2/metabolism ; Oxidative Stress
Czasopismo naukowe
Tytuł:
Macrophage KLF15 prevents foam cell formation and atherosclerosis via transcriptional suppression of OLR-1.
Autorzy:
Song ZK; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Zhao L; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Liu DS; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Zhao LN; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Peng QB; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Li ZY; Department of Thoracic Surgery, The First Affiliated Hospital of Xinjiang Medical University, Urumqi 830000, China.
Wu JY; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Chen SK; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Huang FZ; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Chen X; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Lin TX; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Guan L; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Meng WP; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Guo JW; Department of Pharmacology, School of Medicine, Yangtze University, Jingzhou 434023, China.
Su YN; Department of Rehabilitation, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
He XX; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Liang SJ; Department of Pharmacology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou 510080, China.
Zhu P; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.
Zheng SY; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China. Electronic address: .
Du SL; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China. Electronic address: .
Liu X; Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China. Electronic address: .
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Źródło:
Journal of molecular and cellular cardiology [J Mol Cell Cardiol] 2024 Jan; Vol. 186, pp. 57-70. Date of Electronic Publication: 2023 Nov 18.
Typ publikacji:
Journal Article
MeSH Terms:
Foam Cells*/metabolism
Atherosclerosis*/pathology
Humans ; Mice ; Animals ; Proprotein Convertase 9/metabolism ; Macrophages/metabolism ; Lipoproteins, LDL/metabolism ; Kruppel-Like Transcription Factors/genetics ; Kruppel-Like Transcription Factors/metabolism
Czasopismo naukowe
Tytuł:
SENP3 attenuates foam cell formation by deSUMOylating NLRP3 in macrophages stimulated with ox-LDL.
Autorzy:
Chen J; Department of Pathogen Biology, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan, China.
Sun X; Department of Pathogen Biology, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan, China.
Liu Y; Department of Pathogen Biology, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan, China.
Zhang Y; Department of Pathogen Biology, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan, China.
Zhao M; Department of Biochemistry, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan, China. Electronic address: .
Shao L; Department of Pathogen Biology, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan, China. Electronic address: .
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Źródło:
Cellular signalling [Cell Signal] 2024 May; Vol. 117, pp. 111092. Date of Electronic Publication: 2024 Feb 06.
Typ publikacji:
Journal Article
MeSH Terms:
NLR Family, Pyrin Domain-Containing 3 Protein*/metabolism
Foam Cells*/metabolism
Inflammasomes/metabolism ; Peptide Hydrolases/metabolism ; Macrophages/metabolism ; Lipoproteins, LDL/pharmacology ; Lipoproteins, LDL/metabolism ; Endopeptidases/metabolism
Czasopismo naukowe
Tytuł:
Bioengineered Vascular Model of Foam Cell Formation.
Autorzy:
Zhou Y; Baker Heart and Diabetes Institute, Melbourne, Victoria 3004, Australia.; School of Health and Biomedical Sciences, RMIT University, Bundoora, Victoria 3082, Australia.
Sekar NC; School of Health and Biomedical Sciences, RMIT University, Bundoora, Victoria 3082, Australia.
Thurgood P; Baker Heart and Diabetes Institute, Melbourne, Victoria 3004, Australia.; School of Engineering, RMIT University, Melbourne, Victoria 3000, Australia.
Needham S; Leading Technology Group, Kew, Victoria 3101, Australia.
Peter K; Baker Heart and Diabetes Institute, Melbourne, Victoria 3004, Australia.; Department of Cardiometabolic Health, The University of Melbourne, Parkville, Victoria 3010, Australia.
Khoshmanesh K; Baker Heart and Diabetes Institute, Melbourne, Victoria 3004, Australia.; School of Engineering, RMIT University, Melbourne, Victoria 3000, Australia.
Baratchi S; Baker Heart and Diabetes Institute, Melbourne, Victoria 3004, Australia.; School of Health and Biomedical Sciences, RMIT University, Bundoora, Victoria 3082, Australia.; Department of Cardiometabolic Health, The University of Melbourne, Parkville, Victoria 3010, Australia.
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Źródło:
ACS biomaterials science & engineering [ACS Biomater Sci Eng] 2023 Dec 11; Vol. 9 (12), pp. 6947-6955. Date of Electronic Publication: 2023 Nov 29.
Typ publikacji:
Journal Article
MeSH Terms:
Foam Cells*/metabolism
Foam Cells*/pathology
Endothelial Cells*
Leukocytes, Mononuclear ; Tumor Necrosis Factor-alpha/pharmacology ; Tumor Necrosis Factor-alpha/metabolism ; Monocytes/metabolism
Czasopismo naukowe
Tytuł:
TFEB SUMOylation in macrophages accelerates atherosclerosis by promoting the formation of foam cells through inhibiting lysosomal activity.
Autorzy:
Wang K; Department of Pathology, Xinhua Hospital, Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Zhou W; Department of Urology, Renji Hospital, Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Hu G; Department of Biochemistry and Molecular Cell Biology, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wang L; Department of Ophthalmology, Xinhua Hospital, Affiliated to Shanghai Jiao Tong University School of Medicine, No. 1665, Kongjiang Rd., Shanghai, China.
Cai R; Department of Biochemistry and Molecular Cell Biology, Shanghai Jiao Tong University School of Medicine, Shanghai, China. .
Tian T; Department of Ophthalmology, Xinhua Hospital, Affiliated to Shanghai Jiao Tong University School of Medicine, No. 1665, Kongjiang Rd., Shanghai, China. .
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Źródło:
Cellular and molecular life sciences : CMLS [Cell Mol Life Sci] 2023 Nov 11; Vol. 80 (12), pp. 358. Date of Electronic Publication: 2023 Nov 11.
Typ publikacji:
Journal Article
MeSH Terms:
Atherosclerosis*/genetics
Atherosclerosis*/metabolism
Foam Cells*/metabolism
Animals ; Mice ; Lipoproteins, LDL/pharmacology ; Lipoproteins, LDL/metabolism ; Lysosomes/metabolism ; Macrophages/metabolism ; Mice, Knockout ; Mice, Transgenic ; Sumoylation
Czasopismo naukowe
Tytuł:
Rac2 mediate foam cell formation and associated immune responses in THP-1 to promote the process of atherosclerotic plaques.
Autorzy:
Xu L; Department of clinical laboratory, Xinhua Hospital Affiliated to Dalian University, Dalian, Liaoning 116021, China.
Ren H; Department of Ultrasound, the Sixth Medical Center of Chinese PLA General Hospital, Beijing 100048, China.
Xie D; Department of Cardiology, The Second Hospital of Dalian Medical University, Dalian, Liaoning 116027, China.
Zhang F; Department of Cardiology, Peking University People's Hospital, Beijing 100044, China.
Hu X; Department of Cardiology, Peking University People's Hospital, Beijing 100044, China.
Fang S; Department of Cardiology, Peking University People's Hospital, Beijing 100044, China.
Wang H; Department of Cardiology, The Second Hospital of Dalian Medical University, Dalian, Liaoning 116027, China. Electronic address: .
He D; Department of Cardiology, Peking University People's Hospital, Beijing 100044, China. Electronic address: .
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Źródło:
Molecular immunology [Mol Immunol] 2023 Nov; Vol. 163, pp. 196-206. Date of Electronic Publication: 2023 Oct 13.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Atherosclerosis*/metabolism
Foam Cells*/immunology
Plaque, Atherosclerotic*/immunology
Animals ; Mice ; Immunity ; Inflammation/metabolism ; Lipoproteins, LDL/pharmacology ; Lipoproteins, LDL/metabolism
Czasopismo naukowe

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