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    Wyświetlanie 1-4 z 4
    Tytuł:
    Concepts of Chronic Lymphocytic Leukemia Pathogenesis: DNA Damage Response and Tumor Microenvironment.
    Autorzy:
    Frenzel LP; Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany.
    Reinhardt HC
    Pallasch CP
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    Źródło:
    Oncology research and treatment [Oncol Res Treat] 2016; Vol. 39 (1-2), pp. 9-16. Date of Electronic Publication: 2016 Jan 22.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't; Review
    MeSH Terms:
    DNA Damage/*genetics
    DNA Repair/*genetics
    Leukemia, Lymphocytic, Chronic, B-Cell/*pathology
    Leukemia, Lymphocytic, Chronic, B-Cell/*physiopathology
    Neoplasm Proteins/*genetics
    Tumor Microenvironment/*genetics
    Animals ; DNA, Neoplasm/genetics ; Gene Expression Regulation, Neoplastic/genetics ; Humans ; Models, Genetic ; Mutation/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Hypoxia-induced p38 MAPK activation reduces Mcl-1 expression and facilitates sensitivity towards BH3 mimetics in chronic lymphocytic leukemia.
    Autorzy:
    Huelsemann MF; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany [3] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany.
    Patz M; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany [3] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany.
    Beckmann L; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany [3] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany.
    Brinkmann K; 1] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany [2] Institute for Medical Microbiology, Immunology and Hygiene, University Hospital of Cologne, Cologne, Germany.
    Otto T; Institute of Physiology, University of Duisburg-Essen, Essen, Germany.
    Fandrey J; Institute of Physiology, University of Duisburg-Essen, Essen, Germany.
    Becker HJ; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany.
    Theurich S; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany.
    von Bergwelt-Baildon M; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany.
    Pallasch CP; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany [3] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany.
    Zahedi RP; Leibniz-Institute for Analytical Sciences-ISAS-e.V., Dortmund, Germany.
    Kashkar H; 1] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany [2] Institute for Medical Microbiology, Immunology and Hygiene, University Hospital of Cologne, Cologne, Germany.
    Reinhardt HC; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany [3] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany.
    Hallek M; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany [3] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany.
    Wendtner CM; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany [3] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany [4] Department I of Internal Medicine, Klinikum Schwabing, Munich, Germany.
    Frenzel LP; 1] Department I of Internal Medicine, University Hospital of Cologne, Cologne, Germany [2] Center of Integrated Oncology, University Hospital of Cologne, Cologne, Germany [3] Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University Hospital of Cologne, Cologne, Germany.
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    Źródło:
    Leukemia [Leukemia] 2015 Apr; Vol. 29 (4), pp. 981-4. Date of Electronic Publication: 2014 Nov 07.
    Typ publikacji:
    Letter; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Gene Expression Regulation, Leukemic*
    Antineoplastic Agents/*pharmacology
    B-Lymphocytes/*drug effects
    Biphenyl Compounds/*pharmacology
    Bridged Bicyclo Compounds, Heterocyclic/*pharmacology
    Myeloid Cell Leukemia Sequence 1 Protein/*antagonists & inhibitors
    Nitrophenols/*pharmacology
    Sulfonamides/*pharmacology
    p38 Mitogen-Activated Protein Kinases/*genetics
    B-Lymphocytes/metabolism ; B-Lymphocytes/pathology ; Bendamustine Hydrochloride ; CD40 Ligand/genetics ; CD40 Ligand/metabolism ; Cell Hypoxia ; Cells, Cultured ; Drug Resistance, Neoplasm ; Enzyme Activation ; Humans ; Leukemia, Lymphocytic, Chronic, B-Cell/genetics ; Leukemia, Lymphocytic, Chronic, B-Cell/metabolism ; Leukemia, Lymphocytic, Chronic, B-Cell/pathology ; Myeloid Cell Leukemia Sequence 1 Protein/genetics ; Myeloid Cell Leukemia Sequence 1 Protein/metabolism ; Nitrogen Mustard Compounds/pharmacology ; Oxygen/pharmacology ; Piperazines/pharmacology ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Signal Transduction ; Vidarabine/analogs & derivatives ; Vidarabine/pharmacology ; bcl-X Protein/genetics ; bcl-X Protein/metabolism ; p38 Mitogen-Activated Protein Kinases/metabolism
    Raport
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Rapid generation of human B-cell lymphomas via combined expression of Myc and Bcl2 and their use as a preclinical model for biological therapies.
    Autorzy:
    Leskov I; Koch Institute for Integrative Cancer Research and Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.
    Pallasch CP; Koch Institute for Integrative Cancer Research and Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.; Department I of Internal Medicine, Center of Integrated Oncology, University of Cologne, Cologne, Germany.; Cluster of Excellence for Cellular Stress Response in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
    Drake A; Koch Institute for Integrative Cancer Research and Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.
    Iliopoulou BP; Koch Institute for Integrative Cancer Research and Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.
    Souza A; Koch Institute for Integrative Cancer Research and Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.
    Shen CH; Koch Institute for Integrative Cancer Research and Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.
    Schweighofer CD; Department I of Internal Medicine, Center of Integrated Oncology, University of Cologne, Cologne, Germany.; Department of Hematopathology, UT M.D. Anderson Cancer Center Houston, TX 77030, USA.
    Abruzzo L; Cluster of Excellence for Cellular Stress Response in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
    Frenzel LP; Department I of Internal Medicine, Center of Integrated Oncology, University of Cologne, Cologne, Germany.; Cluster of Excellence for Cellular Stress Response in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
    Wendtner CM; Department I of Internal Medicine, Center of Integrated Oncology, University of Cologne, Cologne, Germany.; Cluster of Excellence for Cellular Stress Response in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
    Hemann MT; Koch Institute for Integrative Cancer Research and Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.
    Chen J; Koch Institute for Integrative Cancer Research and Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.
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    Źródło:
    Oncogene [Oncogene] 2013 Feb 21; Vol. 32 (8), pp. 1066-1072. Date of Electronic Publication: 2012 Apr 09.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Lymphoma, B-Cell/*metabolism
    Lymphoma, B-Cell/*pathology
    Proto-Oncogene Proteins c-bcl-2/*biosynthesis
    Proto-Oncogene Proteins c-myc/*biosynthesis
    Animals ; Disease Models, Animal ; Gene Expression Regulation, Neoplastic ; Humans ; Kaplan-Meier Estimate ; Lymphoma, B-Cell/genetics ; Mice ; Penetrance ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Proto-Oncogene Proteins c-myc/genetics ; Proto-Oncogene Proteins c-myc/metabolism ; Survival Analysis
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-4 z 4

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