Informacja

Drogi użytkowniku, aplikacja do prawidłowego działania wymaga obsługi JavaScript. Proszę włącz obsługę JavaScript w Twojej przeglądarce.

English (EN)·Polski (PL)·Ukraiński (UK)
Przejdź do strony domowej biblioteki Uniwersytet Ekonomiczny we Wrocławiu Link otwiera się w nowym oknie Logo biblioteki Prolib Integro - strona głównaUniwersytet Ekonomiczny we Wrocławiu

Wyszukujesz frazę ""Fu, X"" wg kryterium: Autor

  Lista filtrów
Wyrównaj
Wyświetlanie 1-20 z 185
Tytuł:
In Vitro and In Vivo Photoprotective Effects of (-)-Loliode Isolated from the Brown Seaweed, Sargassum horneri .
Autorzy:
Wang L; College of Food Science and Engineering, Ocean University of China, Qingdao 266003, China.
Kim HS; National Marine Biodiversity Institute of Korea, 75, Jangsan-ro 101gil, Janghang-eup, Seocheon 33662, Korea.
Je JG; Department of Marine Life Sciences, Jeju National University, Jeju 63243, Korea.
Fu X; College of Food Science and Engineering, Ocean University of China, Qingdao 266003, China.
Huang C; Co-Innovation Center for Efficient Processing and Utilization of Forest Products, College of Chemical Engineering, Nanjing Forestry University, Nanjing 210037, China.
Ahn G; Department of Marine Bio Food Science, Chonnam National University, Yeosu 59626, Korea.
Oh JY; Food Safety and Processing Research Division, National Institute of Fisheries Science, Busan 46083, Korea.
Sanjeewa KKA; Department of Biosystem Technology, Faculty of Technology, University of Sri Jayewardenepura, Pitipana, Homagama 10206, Sri Lanka.
Xu J; College of Food Science and Engineering, Ocean University of China, Qingdao 266003, China.
Gao X; College of Food Science and Engineering, Ocean University of China, Qingdao 266003, China.
Yeo IK; Department of Marine Life Sciences, Jeju National University, Jeju 63243, Korea.
Jeon YJ; Department of Marine Life Sciences, Jeju National University, Jeju 63243, Korea.; Marine Science Institute, Jeju National University, Jeju 63333, Korea.
Pokaż więcej
Źródło:
Molecules (Basel, Switzerland) [Molecules] 2021 Nov 16; Vol. 26 (22). Date of Electronic Publication: 2021 Nov 16.
Typ publikacji:
Journal Article
MeSH Terms:
Benzofurans*/chemistry
Benzofurans*/isolation & purification
Benzofurans*/pharmacology
Sunscreening Agents*/chemistry
Sunscreening Agents*/isolation & purification
Sunscreening Agents*/pharmacology
Apoptosis/*drug effects
Dermis/*metabolism
Fibroblasts/*metabolism
Keratinocytes/*metabolism
Sargassum/*chemistry
Seaweed/*chemistry
Zebrafish/*metabolism
Animals ; Apoptosis/radiation effects ; Cell Line ; Humans ; Reactive Oxygen Species/metabolism ; Ultraviolet Rays/adverse effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Heat shock pretreatment improves mesenchymal stem cell viability by heat shock proteins and autophagy to prevent cisplatin-induced granulosa cell apoptosis.
Autorzy:
Wang Q
Li X; Department of Obstetrics and Gynecology, Zhujiang Hospital of Southern Medical University, Guangzhou, Guangdong, People's Republic of China.
Wang Q; Department of Obstetrics and Gynecology, Zhujiang Hospital of Southern Medical University, Guangzhou, Guangdong, People's Republic of China.
Xie J; Department of Obstetrics and Gynecology, Zhujiang Hospital of Southern Medical University, Guangzhou, Guangdong, People's Republic of China.
Xie C; Department of Orthopedics, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, Guangdong, People's Republic of China.
Fu X; Department of Obstetrics and Gynecology, Zhujiang Hospital of Southern Medical University, Guangzhou, Guangdong, People's Republic of China. .
Pokaż więcej
Źródło:
Stem cell research & therapy [Stem Cell Res Ther] 2019 Nov 26; Vol. 10 (1), pp. 348. Date of Electronic Publication: 2019 Nov 26.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Heat-Shock Response*
Apoptosis/*drug effects
Autophagic Cell Death/*drug effects
Cisplatin/*pharmacology
Granulosa Cells/*metabolism
Heat-Shock Proteins/*metabolism
Mesenchymal Stem Cells/*metabolism
Animals ; Cell Survival ; Female ; Rats ; Rats, Wistar
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CRISPR/Cas9 Knockout of Bak Mediates Bax Translocation to Mitochondria in response to TNF α /CHX-induced Apoptosis.
Autorzy:
Zhang J; Edmond H. Fischer Signal Transduction Laboratory, College of Life Sciences, Jilin University, Changchun, 130012, China.
Niu H; Edmond H. Fischer Signal Transduction Laboratory, College of Life Sciences, Jilin University, Changchun, 130012, China.
Zhao ZJ; Edmond H. Fischer Signal Transduction Laboratory, College of Life Sciences, Jilin University, Changchun, 130012, China.; Department of Pathology, University of Oklahoma Health Sciences Center, Oklahoma City, OK, USA.
Fu X; Edmond H. Fischer Signal Transduction Laboratory, College of Life Sciences, Jilin University, Changchun, 130012, China.
Wang Y; Edmond H. Fischer Signal Transduction Laboratory, College of Life Sciences, Jilin University, Changchun, 130012, China.
Zhang X; Edmond H. Fischer Signal Transduction Laboratory, College of Life Sciences, Jilin University, Changchun, 130012, China.
Zhang F; Scientific Research Centre of China-Japan Union Hospital, Jilin University, Changchun, 130033, China.
Zeng L; Edmond H. Fischer Signal Transduction Laboratory, College of Life Sciences, Jilin University, Changchun, 130012, China.
Pokaż więcej
Źródło:
BioMed research international [Biomed Res Int] 2019 Sep 17; Vol. 2019, pp. 9071297. Date of Electronic Publication: 2019 Sep 17 (Print Publication: 2019).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*genetics
Caspases/*genetics
bcl-2 Homologous Antagonist-Killer Protein/*genetics
bcl-2-Associated X Protein/*genetics
Apoptosis/drug effects ; CRISPR-Cas Systems/genetics ; Cell Line, Tumor ; Cycloheximide/pharmacology ; Cytosol/drug effects ; Gene Expression Regulation, Neoplastic/drug effects ; Gene Knockout Techniques ; Humans ; Mitochondria/drug effects ; Mitochondria/genetics ; Proto-Oncogene Proteins c-bcl-2/genetics ; RNA, Small Interfering/genetics ; Tumor Necrosis Factor-alpha/genetics ; Tumor Necrosis Factor-alpha/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Regulatory effects of miRNA‑181a on FasL expression in bone marrow mesenchymal stem cells and its effect on CD4+T lymphocyte apoptosis in estrogen deficiency‑induced osteoporosis.
Autorzy:
Shao B; Department of Operative Dentistry and Endodontics, Affiliated Hospital of Stomatology, Chongqing Medical University, Chongqing 401147, P.R. China.
Fu X; Department of Orthodontics, Second Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou, Zhejiang 310003, P.R. China.
Yu Y; Department of Operative Dentistry and Endodontics, Affiliated Hospital of Stomatology, Chongqing Medical University, Chongqing 401147, P.R. China.
Yang D; Department of Operative Dentistry and Endodontics, Affiliated Hospital of Stomatology, Chongqing Medical University, Chongqing 401147, P.R. China.
Pokaż więcej
Źródło:
Molecular medicine reports [Mol Med Rep] 2018 Jul; Vol. 18 (1), pp. 920-930. Date of Electronic Publication: 2018 May 16.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*immunology
Bone Marrow Cells/*immunology
CD4-Positive T-Lymphocytes/*immunology
Estrogens/*deficiency
Fas Ligand Protein/*immunology
Gene Expression Regulation/*immunology
Mesenchymal Stem Cells/*immunology
MicroRNAs/*immunology
Osteoporosis/*immunology
Animals ; Bone Marrow Cells/pathology ; CD4-Positive T-Lymphocytes/pathology ; Fas Ligand Protein/genetics ; Female ; Gene Expression Regulation/genetics ; Mesenchymal Stem Cells/pathology ; Mice ; MicroRNAs/genetics ; Osteoporosis/genetics ; Osteoporosis/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Selenocystine inhibits JEG-3 cell growth in vitro and in vivo by triggering oxidative damage-mediated S-phase arrest and apoptosis.
Autorzy:
Zhao M; Department of Orthopaedics, Taishan Hospital Affiliated to Taishan Medical University, Taian, Shandong, China.
Hou Y; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Fu X; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Li D; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Sun J; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Fu X; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Wei Z; Department of Oncology, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, Shandong, China.
Pokaż więcej
Źródło:
Journal of cancer research and therapeutics [J Cancer Res Ther] 2018; Vol. 14 (7), pp. 1540-1548.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Cystine/*analogs & derivatives
Organoselenium Compounds/*pharmacology
Oxidative Stress/*drug effects
S Phase Cell Cycle Checkpoints/*drug effects
Animals ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Choriocarcinoma/metabolism ; Cystine/pharmacology ; Disease Models, Animal ; Female ; Humans ; Mice ; Reactive Oxygen Species/metabolism ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Hydroxytyrosol contributes to cell proliferation and inhibits apoptosis in pulsed electromagnetic fields treated human umbilical vein endothelial cells in vitro.
Autorzy:
Cheng Y; Department of Bone Surgery, The First Hospital of Harbin City, Harbin, Heilongjiang 150010, P.R. China.
Qu Z; Department of Bone Surgery, The First Hospital of Harbin City, Harbin, Heilongjiang 150010, P.R. China.
Fu X; Department of Plastic Surgery, The First Hospital of Harbin City, Harbin, Heilongjiang 150010, P.R. China.
Jiang Q; Department of Plastic Surgery, The First Hospital of Harbin City, Harbin, Heilongjiang 150010, P.R. China.
Fei J; Department of Plastic Surgery, The First Hospital of Harbin City, Harbin, Heilongjiang 150010, P.R. China.
Pokaż więcej
Źródło:
Molecular medicine reports [Mol Med Rep] 2017 Dec; Vol. 16 (6), pp. 8826-8832. Date of Electronic Publication: 2017 Oct 03.
Typ publikacji:
Journal Article
MeSH Terms:
Electromagnetic Fields*
Apoptosis/*drug effects
Apoptosis/*radiation effects
Human Umbilical Vein Endothelial Cells/*drug effects
Human Umbilical Vein Endothelial Cells/*metabolism
Phenylethyl Alcohol/*analogs & derivatives
Biomarkers ; Cell Movement ; Cell Proliferation/drug effects ; Humans ; Hydrogen Peroxide ; Mitochondria/drug effects ; Mitochondria/metabolism ; Phenylethyl Alcohol/pharmacology ; Proto-Oncogene Proteins c-akt ; TOR Serine-Threonine Kinases/metabolism ; Transforming Growth Factor beta/metabolism ; Tumor Suppressor Protein p53
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Stromal cell-derived factor 1 protects human periodontal ligament stem cells against hydrogen peroxide-induced apoptosis.
Autorzy:
Feng Y; Department of Orthodontics, Second Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Fu X; Department of Orthodontics, Second Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou, Zhejiang 310009, P.R. China.
Lou X; Department of Dentistry, Punan Hospital of Pudong, Shanghai 200125, P.R. China.
Fu B; Department of Prosthodontics, Affiliated Dental Hospital, Zhejiang University School of Medicine, Hangzhou, Zhejiang 310000, P.R. China.
Pokaż więcej
Źródło:
Molecular medicine reports [Mol Med Rep] 2017 Oct; Vol. 16 (4), pp. 5001-5006. Date of Electronic Publication: 2017 Aug 08.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Apoptosis/*genetics
Chemokine CXCL12/*genetics
Hydrogen Peroxide/*pharmacology
Periodontal Ligament/*cytology
Stem Cells/*drug effects
Stem Cells/*metabolism
Biomarkers ; Caspase 3/metabolism ; Caspase 9/metabolism ; Cell Differentiation ; Cell Survival/drug effects ; Cell Survival/genetics ; Cells, Cultured ; Chemokine CXCL12/metabolism ; Humans ; RNA Interference ; RNA, Small Interfering/genetics ; Stem Cells/cytology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Overexpression of miR-21 in stem cells improves ovarian structure and function in rats with chemotherapy-induced ovarian damage by targeting PDCD4 and PTEN to inhibit granulosa cell apoptosis.
Autorzy:
Fu X; Department of Obstetrics and Gynecology, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, People's Republic of China. .
He Y; Department of Obstetrics and Gynecology, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, People's Republic of China.
Wang X; Department of Obstetrics and Gynecology, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, People's Republic of China.
Peng D; Department of Obstetrics and Gynecology, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, People's Republic of China.
Chen X; Department of Obstetrics and Gynecology, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, People's Republic of China.
Li X; Department of Obstetrics and Gynecology, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, People's Republic of China.
Wang Q; Department of Obstetrics and Gynecology, Zhujiang Hospital, Southern Medical University, Guangzhou, Guangdong, People's Republic of China.
Pokaż więcej
Źródło:
Stem cell research & therapy [Stem Cell Res Ther] 2017 Aug 14; Vol. 8 (1), pp. 187. Date of Electronic Publication: 2017 Aug 14.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Antineoplastic Agents/*adverse effects
Apoptosis Regulatory Proteins/*metabolism
Granulosa Cells/*metabolism
Ovary/*pathology
PTEN Phosphohydrolase/*metabolism
Stem Cells/*metabolism
Animals ; Cells, Cultured ; Female ; Genetic Vectors/metabolism ; Gonadal Steroid Hormones/metabolism ; Granulosa Cells/pathology ; Lentivirus/metabolism ; Mesenchymal Stem Cells/metabolism ; MicroRNAs/genetics ; MicroRNAs/metabolism ; Organ Size ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Rats, Wistar ; Transfection
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
PA-MSHA inhibits proliferation and induces apoptosis in human non-small cell lung cancer cell lines with different genotypes.
Autorzy:
Zhao X; Department of Medical Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, P.R. China.
Wu X; Department of Medical Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, P.R. China.
Yu W; Fudan University Shanghai Cancer Center, Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, P.R. China.
Cai X; Fudan University Shanghai Cancer Center, Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, P.R. China.
Liu Q; Fudan University Shanghai Cancer Center, Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, P.R. China.
Fu X; Fudan University Shanghai Cancer Center, Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, P.R. China.
Liu Z; Fudan University Shanghai Cancer Center, Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, P.R. China.
Hu D; Department of Medicine, Beijing Wonder Biopharmaceutical Co., Ltd., Beijing 101407, P.R. China.
Pan S; Department of Medicine, Beijing Wonder Biopharmaceutical Co., Ltd., Beijing 101407, P.R. China.
Huang Q; Department of Medicine, Beijing Wonder Biopharmaceutical Co., Ltd., Beijing 101407, P.R. China.
Pokaż więcej
Źródło:
Molecular medicine reports [Mol Med Rep] 2016 Dec; Vol. 14 (6), pp. 5369-5376. Date of Electronic Publication: 2016 Oct 20.
Typ publikacji:
Journal Article
MeSH Terms:
Genotype*
Apoptosis/*drug effects
Apoptosis/*genetics
Carcinoma, Non-Small-Cell Lung/*genetics
Fimbriae Proteins/*pharmacology
Lung Neoplasms/*genetics
Biomarkers ; Carcinoma, Non-Small-Cell Lung/metabolism ; Caspases/metabolism ; Cell Cycle/drug effects ; Cell Cycle/genetics ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Dose-Response Relationship, Drug ; Humans ; Inhibitory Concentration 50 ; Lung Neoplasms/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Glutaric acid-mediated apoptosis in primary striatal neurons.
Autorzy:
Tian F; Department of Pediatrics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China ; Department of Pediatrics, First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, China.
Fu X; Department of Pediatrics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China.
Gao J; Department of Pediatrics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China.
Ying Y; Department of Pediatrics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China.
Hou L; Department of Pediatrics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China.
Liang Y; Department of Pediatrics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China.
Ning Q; Laboratory of Infectious Immunology, Department of Infectious Disease, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China.
Luo X; Department of Pediatrics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China.
Pokaż więcej
Źródło:
BioMed research international [Biomed Res Int] 2014; Vol. 2014, pp. 484731. Date of Electronic Publication: 2014 May 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Corpus Striatum/*drug effects
Glutarates/*pharmacology
Neurons/*drug effects
6-Cyano-7-nitroquinoxaline-2,3-dione/pharmacology ; Animals ; Apoptosis/genetics ; Caspase 3/genetics ; Cells, Cultured ; RNA, Messenger/genetics ; Rats ; Rats, Sprague-Dawley ; Receptors, N-Methyl-D-Aspartate/genetics ; Up-Regulation/drug effects ; Up-Regulation/genetics ; alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Effects of targeted suppression of glutaryl-CoA dehydrogenase by lentivirus-mediated shRNA and excessive intake of lysine on apoptosis in rat striatal neurons.
Autorzy:
Gao J; Department of Pediatrics, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhang C
Fu X
Yi Q
Tian F
Ning Q
Luo X
Pokaż więcej
Źródło:
PloS one [PLoS One] 2013 May 02; Vol. 8 (5), pp. e63084. Date of Electronic Publication: 2013 May 02 (Print Publication: 2013).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Knockdown Techniques*
RNA, Small Interfering*/genetics
Apoptosis/*genetics
Glutaryl-CoA Dehydrogenase/*genetics
Lentivirus/*genetics
Lysine/*pharmacology
Neurons/*cytology
Amino Acid Metabolism, Inborn Errors/enzymology ; Amino Acid Metabolism, Inborn Errors/metabolism ; Amino Acid Metabolism, Inborn Errors/pathology ; Animals ; Apoptosis/drug effects ; Base Sequence ; Biological Transport/genetics ; Brain Diseases, Metabolic/enzymology ; Brain Diseases, Metabolic/metabolism ; Brain Diseases, Metabolic/pathology ; Caspase Inhibitors/pharmacology ; Cell Survival/drug effects ; Cell Survival/genetics ; Gene Expression Regulation/drug effects ; Gene Expression Regulation/genetics ; Glutaryl-CoA Dehydrogenase/deficiency ; Glutaryl-CoA Dehydrogenase/metabolism ; Lysine/metabolism ; Membrane Potential, Mitochondrial/drug effects ; Membrane Potential, Mitochondrial/genetics ; Neostriatum/cytology ; Neurons/drug effects ; Neurons/metabolism ; Rats ; Rats, Sprague-Dawley
SCR Disease Name:
Glutaric Acidemia I
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Anti-apoptotic effect and the mechanism of orientin on ischaemic/reperfused myocardium.
Autorzy:
Fu XC; Shenyang Pharmaceutical University, Department of Pharmacology, Shenyang, Liaonin 10016, China. />Wang MW
Li SP
Wang HL
Pokaż więcej
Źródło:
Journal of Asian natural products research [J Asian Nat Prod Res] 2006 Apr-May; Vol. 8 (3), pp. 265-72.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Flavonoids/*pharmacology
Glucosides/*pharmacology
Animals ; Caspase 3/genetics ; Caspase 3/metabolism ; Cytochromes c/genetics ; Cytochromes c/metabolism ; Gene Expression Regulation/drug effects ; Male ; Myocardial Reperfusion Injury ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Rats ; Rats, Wistar ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Specific amino acid dependency regulates invasiveness and viability of androgen-independent prostate cancer cells.
Autorzy:
Fu YM; Cancer Prevention & Research Center, Department of Pharmaceutical Sciences, College of Pharmacy, Washington State University, Pullman, WA 99164-6510, USA.
Yu ZX
Li YQ
Ge X
Sanchez PJ
Fu X
Meadows GG
Pokaż więcej
Źródło:
Nutrition and cancer [Nutr Cancer] 2003; Vol. 45 (1), pp. 60-73.
Typ publikacji:
Comparative Study; Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
MeSH Terms:
Amino Acids/*deficiency
Apoptosis/*physiology
Cell Cycle/*drug effects
Prostatic Neoplasms/*pathology
Amino Acids/metabolism ; Amino Acids/pharmacology ; Androgens/metabolism ; Animals ; Cell Adhesion ; Cell Division/drug effects ; Cell Line, Tumor ; DNA Fragmentation ; Flow Cytometry ; Focal Adhesion Kinase 1 ; Focal Adhesion Protein-Tyrosine Kinases ; Humans ; Male ; Mitogen-Activated Protein Kinases/metabolism ; Neoplasm Invasiveness ; Neoplasm Metastasis ; Neoplasm Proteins/biosynthesis ; Phenylalanine/deficiency ; Phosphorylation ; Prostatic Neoplasms/metabolism ; Protein-Tyrosine Kinases/metabolism ; Tyrosine/deficiency
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
From PTK-STAT signaling to caspase expression and apoptosis induction.
Autorzy:
Fu XY; Department of Pathology, Yale University School of Medicine, New Haven, Connecticut, CT 06520, USA. xin-yuan.fu@yale.edu
Pokaż więcej
Źródło:
Cell death and differentiation [Cell Death Differ] 1999 Dec; Vol. 6 (12), pp. 1201-8.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Apoptosis/*physiology
Caspases/*biosynthesis
DNA-Binding Proteins/*metabolism
Protein-Tyrosine Kinases/*metabolism
Transcription Factors/*metabolism
Gene Expression Regulation, Enzymologic ; Models, Biological ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Wyświetlanie 1-20 z 185

Ta witryna wykorzystuje pliki cookies do przechowywania informacji na Twoim komputerze. Pliki cookies stosujemy w celu świadczenia usług na najwyższym poziomie, w tym w sposób dostosowany do indywidualnych potrzeb. Korzystanie z witryny bez zmiany ustawień dotyczących cookies oznacza, że będą one zamieszczane w Twoim komputerze. W każdym momencie możesz dokonać zmiany ustawień dotyczących cookies