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Wyszukujesz frazę ""Fusion: genetics"" wg kryterium: Temat


Tytuł :
Cytogenetic characterisation of childhood acute lymphoblastic leukemia in Nicaragua
Index Terms :
Adolescent; Aneuploidy; Child; Child, Preschool; Chromosome Aberrations; Chromosome Banding; Core Binding Factor Alpha 2 Subunit/genetics; Female; Fusion Proteins, bcr-abl/genetics; Hepatomegaly/epidemiology; Hepatomegaly/etiology; Humans; Immunophenotyping; In Situ Hybridization, Fluorescence; Male; Myeloid-Lymphoid Leukemia Protein/genetics; Nicaragua/epidemiology; Oncogene Proteins, Fusion/genetics; Precursor Cell Lymphoblastic Leukemia-Lymphoma/drug therapy; Precursor Cell Lymphoblastic Leukemia-Lymphoma/epidemiology; Prognosis; Prospective Studies; Risk; Splenomegaly/epidemiology; Splenomegaly/etiology
info:eu-repo/semantics/doctoralThesis
URL :
http://nbn-resolving.org/urn/resolver.pl?urn=urn:nbn:ch:serval-BIB_2844B9A913AE9">http://nbn-resolving.org/urn/resolver.pl?urn=urn:nbn:ch:serval-BIB_2844B9A913AE9
https://serval.unil.ch/notice/serval:BIB_2844B9A913AE">https://serval.unil.ch/notice/serval:BIB_2844B9A913AE
info:eu-repo/semantics/altIdentifier/urn/urn:nbn:ch:serval-BIB_2844B9A913AE9
Zasób elektroniczny
Tytuł :
Signatures of Selection in Fusion Transcripts Resulting from Chromosomal Translocations in Human Cancer
Index Terms :
Neoplasms/genetics
Oncogene Proteins
Fusion/genetics
Translocation, Genetic
RNA, Messenger/genetics
Línea de Investigación Especial PIUNA Genética
Article
URL :
http://hdl.handle.net/10171/7313">http://hdl.handle.net/10171/7313
http://bit.ly/Jcxu3a">http://bit.ly/Jcxu3a
http://bit.ly/Jcxu3a">http://bit.ly/Jcxu3a
Zasób elektroniczny
Tytuł :
Epigenetic features of human mesenchymal stem cells determine their permissiveness for induction of relevant transcriptional changes by SYT-SSX1.
Index Terms :
Adolescent; Alleles; Child; Chromatin/metabolism; CpG Islands; DNA/genetics; Epigenesis, Genetic; Gene Expression Profiling; Gene Expression Regulation; Humans; Mesenchymal Stem Cells/cytology; Oncogene Proteins, Fusion/genetics; Oncogene Proteins, Fusion/physiology; Sarcoma, Synovial/metabolism; Transcription, Genetic; Translocation, Genetic
info:eu-repo/semantics/article
article
URL :
http://nbn-resolving.org/urn/resolver.pl?urn=urn:nbn:ch:serval-BIB_35F298DAEEF27">http://nbn-resolving.org/urn/resolver.pl?urn=urn:nbn:ch:serval-BIB_35F298DAEEF27
Zasób elektroniczny
Tytuł :
Treatment of patients with the hypereosinophilic syndrome with mepolizumab.
Index Terms :
Sciences bio-médicales et agricoles
Adult
Antibodies, Monoclonal -- adverse effects
Antibodies, Monoclonal -- therapeutic use
Double-Blind Method
Drug Therapy, Combination
Eosinophils -- drug effects
Female
Glucocorticoids -- administration & dosage
Humans
Hypereosinophilic Syndrome -- drug therapy
Hypereosinophilic Syndrome -- genetics
Interleukin-5 -- immunology
Leukocyte Count
Male
Middle Aged
Oncogene Proteins, Fusion -- genetics
Prednisone -- administration & dosage
Receptor, Platelet-Derived Growth Factor alpha -- genetics
Treatment Failure
mRNA Cleavage and Polyadenylation Factors -- genetics
info:eu-repo/semantics/article
info:ulb-repo/semantics/articlePeerReview
info:ulb-repo/semantics/openurl/article
URL :
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/51567">http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/51567
http://worldcat.org/search?q=on:EQY+http://difusion-oai.ulb.ac.be/oai/request+DCG_ENTIRE_REPOSITORY+CNTCOLL">http://worldcat.org/search?q=on:EQY+http://difusion-oai.ulb.ac.be/oai/request+DCG_ENTIRE_REPOSITORY+CNTCOLL
Zasób elektroniczny
Tytuł :
Role of the polycomb repressive complex 2 in acute promyelocytic leukemia.
Index Terms :
Sciences bio-médicales et agricoles
Carrier Proteins -- physiology
Cell Differentiation
DNA Methylation
Epigenesis, Genetic
Gene Silencing
Granulocytes -- physiology
Histones
Humans
Leukemia, Promyelocytic, Acute -- metabolism
Nuclear Proteins -- physiology
Oncogene Proteins, Fusion -- genetics
Oncogene Proteins, Fusion -- physiology
Repressor Proteins -- metabolism
Tretinoin -- pharmacology
Tumor Cells, Cultured
info:eu-repo/semantics/article
info:ulb-repo/semantics/articlePeerReview
info:ulb-repo/semantics/openurl/article
URL :
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/50489">http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/50489
http://worldcat.org/search?q=on:EQY+http://difusion-oai.ulb.ac.be/oai/request+DCG_ENTIRE_REPOSITORY+CNTCOLL">http://worldcat.org/search?q=on:EQY+http://difusion-oai.ulb.ac.be/oai/request+DCG_ENTIRE_REPOSITORY+CNTCOLL
Zasób elektroniczny
Tytuł :
Hypereosinophilic syndrome: lymphoproliferative and myeloproliferative variants
Index Terms :
Hématologie
Cytologie
Pathologies particulières
Médecine pathologie humaine
Antibodies, Monoclonal -- therapeutic use
Antigens, CD -- immunology
Antigens, Neoplasm -- immunology
Antineoplastic Agents -- therapeutic use
Glucocorticoids -- therapeutic use
Glycoproteins -- immunology
Humans
Hypereosinophilic Syndrome -- classification
Hypereosinophilic Syndrome -- diagnosis
Hypereosinophilic Syndrome -- drug therapy
Immunologic Factors -- therapeutic use
Interleukin-5 -- biosynthesis
Interleukin-5 -- immunology
Lymphoproliferative Disorders -- diagnosis
Lymphoproliferative Disorders -- drug therapy
Lymphoproliferative Disorders -- physiopathology
Myeloproliferative Disorders -- diagnosis
Myeloproliferative Disorders -- drug therapy
Myeloproliferative Disorders -- physiopathology
Oncogene Proteins, Fusion -- genetics
Piperazines -- therapeutic use
Prognosis
Pyrimidines -- therapeutic use
Receptor, Platelet-Derived Growth Factor alpha -- genetics
mRNA Cleavage and Polyadenylation Factors -- genetics
CD3- CD4+ T-cell
Eosinophil
FIP1L1-PDGFRα
IL-5
Lymphoma
info:eu-repo/semantics/article
info:ulb-repo/semantics/articlePeerReview
info:ulb-repo/semantics/openurl/article
URL :
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/20643">http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/20643
http://worldcat.org/search?q=on:EQY+http://difusion-oai.ulb.ac.be/oai/request+DCG_ENTIRE_REPOSITORY+CNTCOLL">http://worldcat.org/search?q=on:EQY+http://difusion-oai.ulb.ac.be/oai/request+DCG_ENTIRE_REPOSITORY+CNTCOLL
Zasób elektroniczny
Tytuł :
A direct binding site for Grb2 contributes to transformation and leukemogenesis by the Tel-Abl (ETV6-Abl) tyrosine kinase.
Index Terms :
Medicine and Health Sciences
Adaptor Proteins, Signal Transducing
Animals
Binding Sites
Cell Transformation, Neoplastic: genetics; metabolism
GRB2 Adaptor Protein
Leukemia: etiology; metabolism
Mice
Mitogen-Activated Protein Kinases: metabolism
Oncogene Proteins, Fusion: genetics; metabolism
Phosphatidylinositol 3-Kinases: metabolism
Protein Binding
Protein Structure, Tertiary
Protein-Tyrosine Kinases
Proteins: metabolism
Tyrosine: metabolism
article
Źródło :
Molecular and cellular biology vol 24, no 11, 4685-4695; 0270-7306
URL :
http://www.escholarship.org/uc/item/8xm3z9x2">http://www.escholarship.org/uc/item/8xm3z9x2
Zasób elektroniczny
Tytuł :
A direct binding site for Grb2 contributes to transformation and leukemogenesis by the Tel-Abl (ETV6-Abl) tyrosine kinase.
Autorzy :
Million, Ryan P
Pokaż więcej
Index Terms :
Adaptor Proteins
Signal Transducing
Animals
Binding Sites
Cell Transformation
Neoplastic: genetics
metabolism
GRB2 Adaptor Protein
Leukemia: etiology
Mice
Mitogen-Activated Protein Kinases: metabolism
Oncogene Proteins
Fusion: genetics
Phosphatidylinositol 3-Kinases: metabolism
Protein Binding
Protein Structure
Tertiary
Protein-Tyrosine Kinases
Proteins: metabolism
Tyrosine: metabolism
article
Źródło :
Molecular and cellular biology vol 24, iss 11, 4685-4695 0270-7306
URL :
https://escholarship.org/uc/item/8xm3z9x2">https://escholarship.org/uc/item/8xm3z9x2
https://escholarship.org/">https://escholarship.org/
Zasób elektroniczny
Tytuł :
A murine model of CML blast crisis induced by cooperation between BCR/ABL and NUP98/HOXA9.
Index Terms :
Medicine and Health Sciences
Animals
Antineoplastic Agents: pharmacology
Benzamides
Blast Crisis: pathology
Bone Marrow Cells: pathology
Bone Marrow Transplantation
Cell Division
Cloning, Molecular
Disease Models, Animal
Disease Progression
Fusion Proteins, bcr-abl: genetics
Green Fluorescent Proteins
Homeodomain Proteins: genetics
Humans
Leukemia, Myelogenous, Chronic, BCR-ABL Positive: pathology
Luminescent Proteins: genetics
Mice
Nuclear Pore Complex Proteins: genetics
Oncogene Proteins, Fusion: genetics
Piperazines: pharmacology
Proviruses: genetics
Pyrimidines: pharmacology
Tumor Cells, Cultured
article
Źródło :
Dash, Ajeeta B; Williams, Ifor R; Kutok, Jeffery L; Tomasson, Michael H; Anastasiadou, Ema; Lindahl, Kathleen; et al.(2002). A murine model of CML blast crisis induced by cooperation between BCR/ABL and NUP98/HOXA9.. Proceedings of the National Academy of Sciences of the United States of America, 99(11), 7622 - 7627. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/9f89s0ps
URL :
http://www.escholarship.org/uc/item/9f89s0ps">http://www.escholarship.org/uc/item/9f89s0ps
Zasób elektroniczny
Tytuł :
A murine model of CML blast crisis induced by cooperation between BCR/ABL and NUP98/HOXA9.
Autorzy :
Dash, Ajeeta B
Pokaż więcej
Index Terms :
Animals
Antineoplastic Agents: pharmacology
Benzamides
Blast Crisis: pathology
Bone Marrow Cells: pathology
Bone Marrow Transplantation
Cell Division
Cloning
Molecular
Disease Models
Animal
Disease Progression
Fusion Proteins
bcr-abl: genetics
Green Fluorescent Proteins
Homeodomain Proteins: genetics
Humans
Leukemia
Myelogenous
Chronic
BCR-ABL Positive: pathology
Luminescent Proteins: genetics
Mice
Nuclear Pore Complex Proteins: genetics
Oncogene Proteins
Fusion: genetics
Piperazines: pharmacology
Proviruses: genetics
Pyrimidines: pharmacology
Tumor Cells
Cultured
article
Źródło :
Proceedings of the National Academy of Sciences of the United States of America vol 99, iss 11, 7622-7627 0027-8424
URL :
https://escholarship.org/uc/item/9f89s0ps">https://escholarship.org/uc/item/9f89s0ps
https://escholarship.org/">https://escholarship.org/
Zasób elektroniczny
Tytuł :
Membrane interactions of mutated forms of the influenza fusion peptide.
Index Terms :
Chimie
Alanine -- genetics
Amino Acid Sequence
Deuterium -- metabolism
Glutamic Acid -- genetics
Glycine -- metabolism
Hemagglutinin Glycoproteins, Influenza Virus -- genetics
Hemagglutinin Glycoproteins, Influenza Virus -- metabolism
Kinetics
Lipid Bilayers -- metabolism
Membrane Fusion -- genetics
Molecular Sequence Data
Nuclear Magnetic Resonance, Biomolecular
Phosphorus Isotopes
Protein Conformation
Protein Structure, Secondary -- genetics
Serine -- genetics
Spectroscopy, Fourier Transform Infrared
Valine -- genetics
Viral Fusion Proteins -- genetics
Viral Fusion Proteins -- metabolism
info:eu-repo/semantics/article
info:ulb-repo/semantics/articlePeerReview
info:ulb-repo/semantics/openurl/article
URL :
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/77604">http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/77604
http://worldcat.org/search?q=on:EQY+http://difusion-oai.ulb.ac.be/oai/request+DCG_ENTIRE_REPOSITORY+CNTCOLL">http://worldcat.org/search?q=on:EQY+http://difusion-oai.ulb.ac.be/oai/request+DCG_ENTIRE_REPOSITORY+CNTCOLL
Zasób elektroniczny
Tytuł :
Induction of myeloproliferative disease in mice by tyrosine kinase fusion oncogenes does not require granulocyte-macrophage colony-stimulating factor or interleukin-3.
Index Terms :
Medicine and Health Sciences
Animals
Bone Marrow Transplantation
Cell Transformation
Neoplastic: drug effects
genetics
Disease Models
Animal
Granulocyte-Macrophage Colony-Stimulating Factor: genetics
pharmacology
Interleukin-3: genetics
Mice
Knockout
Myeloproliferative Disorders: etiology
pathology
Neoplasm Proteins: genetics
Oncogene Proteins
Fusion: genetics
Paracrine Communication
Transduction
Genetic
cytokine
granulocyte macrophage colony stimulating factor
hybrid protein
interleukin 3
neurotrophin 3 receptor
platelet derived growth factor beta receptor
protein tyrosine kinase
animal tissue
apoptosis
article
cell proliferation
chromosome translocation
controlled study
leukemia cell
mouse
myelopoiesis
myeloproliferative disorder
nonhuman
oncogene
pathogenesis
priority journal
Neoplastic
Granulocyte-Macrophage Colony-Stimulating Factor
Interleukin-3
Myeloproliferative Disorders
Neoplasm Proteins
Fusion
article
Źródło :
Tomasson, M H; Williams, I R; Li, S; Kutok, J; Cain, D; Gillessen, S; et al.(2001). Induction of myeloproliferative disease in mice by tyrosine kinase fusion oncogenes does not require granulocyte-macrophage colony-stimulating factor or interleukin-3.. Blood, 97(5), 1435 - 1441. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/4cv2724w
URL :
http://www.escholarship.org/uc/item/4cv2724w">http://www.escholarship.org/uc/item/4cv2724w
Zasób elektroniczny

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