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Tytuł:
Cutaneous expression of growth-associated protein 43 is not a compelling marker for human nerve regeneration in carpal tunnel syndrome.
Autorzy:
Carroll L; Nuffield Department of Clinical Neurosciences, University of Oxford, Oxford, United Kingdom.
Sandy-Hindmarch O; Nuffield Department of Clinical Neurosciences, University of Oxford, Oxford, United Kingdom.
Baskozos G; Nuffield Department of Clinical Neurosciences, University of Oxford, Oxford, United Kingdom.
Zhu GC; Nuffield Department of Clinical Neurosciences, University of Oxford, Oxford, United Kingdom.
McCarthy J; Nuffield Department of Clinical Neurosciences, University of Oxford, Oxford, United Kingdom.
Schmid A; Nuffield Department of Clinical Neurosciences, University of Oxford, Oxford, United Kingdom.
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Źródło:
PloS one [PLoS One] 2022 Nov 16; Vol. 17 (11), pp. e0277133. Date of Electronic Publication: 2022 Nov 16 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carpal Tunnel Syndrome*/surgery
Carpal Tunnel Syndrome*/pathology
GAP-43 Protein*/genetics
GAP-43 Protein*/metabolism
Peripheral Nervous System Diseases*/metabolism
Humans ; Biomarkers/metabolism ; Median Nerve/pathology ; Nerve Regeneration/physiology ; Skin/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Delayed CO 2 postconditioning promotes neurological recovery after cryogenic traumatic brain injury by downregulating IRF7 expression.
Autorzy:
Li Y; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Chen R; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Shen GP; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Yin J; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Li Y; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Zhao J; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Nan F; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Zhang SH; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Zhang HF; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Yang CH; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.
Wu MN; Key Laboratory of Cellular Physiology, Ministry of Education, Shanxi Medical University, Taiyuan, China.
Fan YY; Department of Pharmacology, Basic Medical Sciences Center, Shanxi Medical University, Taiyuan, China.; Key Laboratory of Cellular Physiology, Ministry of Education, Shanxi Medical University, Taiyuan, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2023 Nov; Vol. 29 (11), pp. 3378-3390. Date of Electronic Publication: 2023 May 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Brain Injuries, Traumatic*/metabolism
Carbon Dioxide*/metabolism
Carbon Dioxide*/therapeutic use
Interferon Regulatory Factor-7*/metabolism
Interferon Regulatory Factor-7*/therapeutic use
Animals ; Mice ; Disease Models, Animal ; GAP-43 Protein/metabolism ; Synaptophysin/metabolism ; Synaptophysin/therapeutic use
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Serum Level of Growth-Associated Protein 43 Is Associated with First-Episode Schizophrenia Patients without Antipsychotic Drugs Treatment.
Autorzy:
Xiao L; Department of Psychiatry, Nanjing Qinglongshan Mental Hospital, Nanjing 211123, Jiangsu, China.
Tang X; Affiliated WuTaiShan Hospital of Medical College of Yangzhou University, Yangzhou 225003, Jiangsu, China.
Hu X; The Second People's Hospital of Jiangning District, Nanjing 211103, Jiangsu, China.
Feng X; Department of Psychiatry, Nanjing Qinglongshan Mental Hospital, Nanjing 211123, Jiangsu, China.
Gong R; Department of Psychiatry, Nanjing Qinglongshan Mental Hospital, Nanjing 211123, Jiangsu, China.
Wang F; Department of Psychiatry, Nanjing Qinglongshan Mental Hospital, Nanjing 211123, Jiangsu, China.
Zhang X; Department of Geriatric Psychiatry, Affiliated Brain Hospital of Nanjing Medical University, Nanjing 210029, Jiangsu, China.
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Źródło:
Computational intelligence and neuroscience [Comput Intell Neurosci] 2022 May 16; Vol. 2022, pp. 4719271. Date of Electronic Publication: 2022 May 16 (Print Publication: 2022).
Typ publikacji:
Journal Article; Retracted Publication
MeSH Terms:
Antipsychotic Agents*/therapeutic use
GAP-43 Protein*/blood
Schizophrenia*/drug therapy
Schizophrenia*/metabolism
Humans ; Psychiatric Status Rating Scales
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
NEP1‑40 promotes myelin regeneration via upregulation of GAP‑43 and MAP‑2 expression after focal cerebral ischemia in rats.
Autorzy:
Zhao H; Department of Neurology, Dalian Municipal Central Hospital Affiliated to Dalian Medical University, Dalian, Liaoning 116033, P.R. China.
Liu ZD; Department of General Medicine, Central Hospital Affiliated to Shaoxing University, Shaoxing, Zhejiang 312000, P.R. China.
Zhang YB; Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing 100050, P.R. China.
Gao XY; Department of Neurology, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, Shandong 264000, P.R. China.
Wang C; Department of Neurology, Dalian Municipal Central Hospital Affiliated to Dalian Medical University, Dalian, Liaoning 116033, P.R. China.
Liu Y; Department of Neurology, Dalian Municipal Central Hospital Affiliated to Dalian Medical University, Dalian, Liaoning 116033, P.R. China.
Wang XF; Department of Neurology, Dalian Medical University, Dalian, Liaoning 116033, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Dec; Vol. 24 (6). Date of Electronic Publication: 2021 Oct 13.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Ischemia/*metabolism
Cerebral Infarction/*metabolism
GAP-43 Protein/*metabolism
Myelin Proteins/*metabolism
Myelin Sheath/*metabolism
Peptide Fragments/*metabolism
Animals ; Axons/metabolism ; Brain Ischemia/pathology ; Cerebral Infarction/pathology ; Disease Models, Animal ; GAP-43 Protein/genetics ; Male ; Myelin Proteins/genetics ; Myelin Sheath/genetics ; Nerve Regeneration ; Neurons/metabolism ; Nogo Proteins/metabolism ; Nogo Receptor 1/metabolism ; Oligodendroglia/metabolism ; Peptide Fragments/genetics ; Rats ; Rats, Sprague-Dawley ; Up-Regulation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Phosphorylation of GAP-43 T172 is a molecular marker of growing axons in a wide range of mammals including primates.
Autorzy:
Okada M; Department of Neurosurgery, Brain Research Institute, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, Japan.; Medical and Dental Hospital, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, Japan.; Departments of Neurochemistry and Molecular Cell Biology, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, 951-8510, Japan.
Kawagoe Y; Departments of Neurochemistry and Molecular Cell Biology, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, 951-8510, Japan.
Sato Y; Departments of Neurochemistry and Molecular Cell Biology, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, 951-8510, Japan.; Department of Chemistry, Faculty of Science, Hokkaido University, Sapporo, 060-0810, Japan.
Nozumi M; Departments of Neurochemistry and Molecular Cell Biology, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, 951-8510, Japan.
Ishikawa Y; Departments of Neurochemistry and Molecular Cell Biology, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, 951-8510, Japan.; Department of Orthopedic Surgery, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, Japan.
Tamada A; Departments of Neurochemistry and Molecular Cell Biology, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, 951-8510, Japan.; Department of iPS Cell Applied Medicine, Faculty of Medicine, Kansai Medical University, Hirakata, Osaka, 573-1010, Japan.
Yamazaki H; Department of Neurobiology and Behavior, Gunma University Graduate School of Medicine, Maebashi, Japan.
Sekino Y; Laboratory of Chemical Pharmacology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Tokyo, Japan.
Kanemura Y; Division of Regenerative Medicine, Department of Biomedical Research and Innovation, Institute for Clinical Research, National Hospital Organization Osaka National Hospital, Osaka, Japan.
Shinmyo Y; Department of Medical Neuroscience, Graduate School of Medical Sciences, Kanazawa University, Kanazawa, Japan.
Kawasaki H; Department of Medical Neuroscience, Graduate School of Medical Sciences, Kanazawa University, Kanazawa, Japan.
Kaneko N; Department of Developmental and Regenerative Neurobiology, Institute of Brain Science, Nagoya City University Graduate School of Medical Sciences, Nagoya, Japan.
Sawamoto K; Department of Developmental and Regenerative Neurobiology, Institute of Brain Science, Nagoya City University Graduate School of Medical Sciences, Nagoya, Japan.; Division of Neural Development and Regeneration, National Institute for Physiological Sciences, Okazaki, Japan.
Fujii Y; Department of Neurosurgery, Brain Research Institute, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, Japan.; Medical and Dental Hospital, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, Japan.
Igarashi M; Departments of Neurochemistry and Molecular Cell Biology, School of Medicine and Graduate School of Medical/Dental Sciences, Niigata University, Niigata, 951-8510, Japan. .
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Źródło:
Molecular brain [Mol Brain] 2021 Apr 08; Vol. 14 (1), pp. 66. Date of Electronic Publication: 2021 Apr 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Axons/*metabolism
Biomarkers/*metabolism
GAP-43 Protein/*metabolism
Mammals/*metabolism
Phosphothreonine/*metabolism
Amino Acid Sequence ; Animals ; Antibodies/metabolism ; Brain/embryology ; Callithrix ; Cells, Cultured ; Ferrets ; GAP-43 Protein/chemistry ; Growth Cones/metabolism ; HEK293 Cells ; Humans ; JNK Mitogen-Activated Protein Kinases/metabolism ; Mice, Inbred C57BL ; Nerve Regeneration ; Phosphorylation ; Primates ; Sciatic Nerve/injuries ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Neuromodulatory effects of repetitive transcranial magnetic stimulation on neural plasticity and motor functions in rats with an incomplete spinal cord injury: A preliminary study.
Autorzy:
Marufa SA; Graduate Institute of Biomedical Materials and Tissue Engineering, College of Biomedical Engineering, Taipei Medical University, Taipei, Taiwan.; Physical Therapy Department, Faculty of Health Science, University of Muhammadiyah Malang, Indonesia.
Hsieh TH; School of Physical Therapy and Graduate Institute of Rehabilitation Science, Chang Gung University, Taoyuan, Taiwan.; Neuroscience Research Center, Chang Gung Memorial Hospital, Taoyuan, Taiwan.; Healthy Aging Research Center, Chang Gung University, Taoyuan, Taiwan.
Liou JC; School of Biomedical Engineering, College of Biomedical Engineering, Taipei Medical University, Taipei, Taiwan.
Chen HY; Department of Occupational Therapy and Graduate Institute of Behavioral Sciences, College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Peng CW; Graduate Institute of Biomedical Materials and Tissue Engineering, College of Biomedical Engineering, Taipei Medical University, Taipei, Taiwan.; School of Biomedical Engineering, College of Biomedical Engineering, Taipei Medical University, Taipei, Taiwan.; International PhD Program in Biomedical Engineering, College of Biomedical Engineering, Taipei Medical University, Taipei, Taiwan.; School of Gerontology Health Management, College of Nursing, Taipei Medical University, Taipei, Taiwan.
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Źródło:
PloS one [PLoS One] 2021 Jun 04; Vol. 16 (6), pp. e0252965. Date of Electronic Publication: 2021 Jun 04 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Neuronal Plasticity*
Recovery of Function*
Evoked Potentials, Motor/*physiology
GAP-43 Protein/*metabolism
Motor Cortex/*physiology
Spinal Cord Injuries/*therapy
Transcranial Magnetic Stimulation/*methods
Animals ; GAP-43 Protein/genetics ; Male ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The effects of exercise training on Kinesin and GAP-43 expression in skeletal muscle fibers of STZ-induced diabetic rats.
Autorzy:
Rahmati M; Department of Physical Education and Sport Sciences, Faculty of Literature and Human Sciences, Lorestan University, Khoramabad, Iran. .
Taherabadi SJ; Department of Physical Education and Sport Sciences, Faculty of Literature and Human Sciences, Lorestan University, Khoramabad, Iran.
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Źródło:
Scientific reports [Sci Rep] 2021 May 05; Vol. 11 (1), pp. 9535. Date of Electronic Publication: 2021 May 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Diabetes Mellitus, Experimental/*pathology
GAP-43 Protein/*analysis
Kinesins/*analysis
Muscle Fibers, Skeletal/*pathology
Animals ; Diabetes Mellitus, Experimental/metabolism ; Diabetes Mellitus, Experimental/therapy ; Endurance Training ; GAP-43 Protein/metabolism ; Kinesins/metabolism ; Male ; Muscle Fibers, Skeletal/metabolism ; Muscle, Skeletal/metabolism ; Muscle, Skeletal/pathology ; Physical Conditioning, Animal ; Rats, Wistar ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Effect of prenatal stress and extremely low-frequency electromagnetic fields on anxiety-like behavior in female rats: With an emphasis on prefrontal cortex and hippocampus.
Autorzy:
Hosseini E; Faculty of Veterinary Medicine, Division of Physiology, Department of basic science, Urmia University, Urmia, Iran.
Kianifard D; Faculty of Veterinary Medicine, Department of Basic Sciences, University of Tabriz, Tabriz, Iran.
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Źródło:
Brain and behavior [Brain Behav] 2023 Apr; Vol. 13 (4), pp. e2949. Date of Electronic Publication: 2023 Mar 21.
Typ publikacji:
Journal Article
MeSH Terms:
Electromagnetic Fields*/adverse effects
Brain-Derived Neurotrophic Factor*/metabolism
Female ; Rats ; Animals ; GAP-43 Protein/metabolism ; Hippocampus/metabolism ; Prefrontal Cortex/metabolism ; Anxiety/etiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
An Enriched Environment Promotes Motor Function through Neuroprotection after Cerebral Ischemia.
Autorzy:
Shi LF; Department of Endocrine Medicine, Jinshan Hospital, Fudan University, Shanghai, China.
Wang CJ; Department of Rehabilitation Medicine, Jinshan Hospital, Fudan University, Shanghai, China.
Yu KW; Department of Rehabilitation Medicine, Huashan Hospital, Fudan University, Shanghai, China.
Wu JF; Department of Rehabilitation Medicine, Huashan Hospital, Fudan University, Shanghai, China.
Zhang QQ; Department of Rehabilitation Medicine, Huashan Hospital, Fudan University, Shanghai, China.
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Źródło:
BioMed research international [Biomed Res Int] 2023 Feb 08; Vol. 2023, pp. 4143633. Date of Electronic Publication: 2023 Feb 08 (Print Publication: 2023).
Typ publikacji:
Journal Article
MeSH Terms:
Brain Ischemia*
Stroke*
Ischemic Stroke*
Humans ; Mice ; Animals ; Neuroprotection ; bcl-2-Associated X Protein ; GAP-43 Protein ; Mice, Inbred C57BL ; Infarction, Middle Cerebral Artery ; Disease Models, Animal
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Impact of GAP-43, Cx43 and actin expression on the outcome and overall survival in diffuse and anaplastic gliomas.
Autorzy:
Krigers A; Department of Neurosurgery, Medical University of Innsbruck, Anichstrasse 35, 6020, Innsbruck, Austria.
Demetz M; Department of Neurosurgery, Medical University of Innsbruck, Anichstrasse 35, 6020, Innsbruck, Austria.
Moser P; Department of Neuropathology, University Hospital of Innsbruck, Tirol Kliniken, Austria.
Kerschbaumer J; Department of Neurosurgery, Medical University of Innsbruck, Anichstrasse 35, 6020, Innsbruck, Austria.
Brawanski KR; Department of Neurosurgery, Medical University of Innsbruck, Anichstrasse 35, 6020, Innsbruck, Austria.
Fritsch H; Department of Anatomy, Histology and Embryology, Medical University of Innsbruck, Innsbruck, Austria.
Thomé C; Department of Neurosurgery, Medical University of Innsbruck, Anichstrasse 35, 6020, Innsbruck, Austria.
Freyschlag CF; Department of Neurosurgery, Medical University of Innsbruck, Anichstrasse 35, 6020, Innsbruck, Austria. .
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Źródło:
Scientific reports [Sci Rep] 2023 Feb 04; Vol. 13 (1), pp. 2024. Date of Electronic Publication: 2023 Feb 04.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Neoplasms*/pathology
Glioma*/pathology
Adult ; Humans ; Middle Aged ; Actins/genetics ; Anaplasia ; Connexin 43/genetics ; Connexin 43/metabolism ; GAP-43 Protein ; Prognosis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Transcriptome profiling unveils GAP43 regulates ABC transporters and EIF2 signaling in colorectal cancer cells.
Autorzy:
Chen X; School of Life Science and Technology, Harbin Institute of Technology, Harbin, 150001, China.
Wu H; Department of Clinical Laboratory, Hangzhou Cancer Hospital, Hangzhou,, 320000, Zhejiang, China.; The NHC Key Laboratory of Drug Addition Medicine, The First Affiliated Hospital of Kunming Medical University, Kunming, 650000, China.
Feng J; School of Life Science and Technology, Harbin Institute of Technology, Harbin, 150001, China.
Li Y; School of Life Science and Technology, Harbin Institute of Technology, Harbin, 150001, China.
Lv J; School of Life Science and Technology, Harbin Institute of Technology, Harbin, 150001, China.
Shi W; School of Life Science and Technology, Harbin Institute of Technology, Harbin, 150001, China.
Fan W; Department of Infectious and Medicine, Heilongjiang Provincial Hospital, Harbin, 150036, China.
Xiao L; School of Life Science and Technology, Harbin Institute of Technology, Harbin, 150001, China.
Sun D; Department of Pediatrics, Data People's hospital, Shenmu, 719301, China.
Jiang M; Department of Clinical Laboratory, Hangzhou Cancer Hospital, Hangzhou,, 320000, Zhejiang, China. .
Shi M; School of Life Science and Technology, Harbin Institute of Technology, Harbin, 150001, China. .
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Źródło:
BMC cancer [BMC Cancer] 2021 Jan 05; Vol. 21 (1), pp. 24. Date of Electronic Publication: 2021 Jan 05.
Typ publikacji:
Journal Article
MeSH Terms:
DNA Methylation*
Gene Expression Regulation, Neoplastic*
ATP-Binding Cassette Transporters/*metabolism
Biomarkers, Tumor/*metabolism
Colorectal Neoplasms/*pathology
Eukaryotic Initiation Factor-2/*metabolism
GAP-43 Protein/*metabolism
ATP-Binding Cassette Transporters/genetics ; Apoptosis ; Biomarkers, Tumor/genetics ; Cell Proliferation ; Colorectal Neoplasms/genetics ; Colorectal Neoplasms/metabolism ; Eukaryotic Initiation Factor-2/genetics ; GAP-43 Protein/genetics ; Gene Regulatory Networks ; Humans ; Prognosis ; Promoter Regions, Genetic ; Transcriptome ; Tumor Cells, Cultured
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
GAP-43 is involved in the orientation of cell division by interacting with GΑI during neurogenesis.
Autorzy:
Huang R; Institute of Central Laboratory, Capital Institute of Pediatrics, Beijing, China.; Department of Neurobiology, Beijing Institute for Brain Disorders, Beijing Center of Neural Regeneration and Repair, Key Laboratory for Neurodegenerative Diseases of the Ministry of Education, Capital Medical University, Beijing, China.
Xiao H; Institute of Central Laboratory, Capital Institute of Pediatrics, Beijing, China.; Graduate School of Peking Union Medical College, Chinese Academy of Medical Science & Peking Union Medical College, Beijing, China.
Zhao J; Department of Neurobiology, Beijing Institute for Brain Disorders, Beijing Center of Neural Regeneration and Repair, Key Laboratory for Neurodegenerative Diseases of the Ministry of Education, Capital Medical University, Beijing, China.
Ju L; Department of Neurobiology, Beijing Institute for Brain Disorders, Beijing Center of Neural Regeneration and Repair, Key Laboratory for Neurodegenerative Diseases of the Ministry of Education, Capital Medical University, Beijing, China.
Wen Y; Department of Neurobiology, Beijing Institute for Brain Disorders, Beijing Center of Neural Regeneration and Repair, Key Laboratory for Neurodegenerative Diseases of the Ministry of Education, Capital Medical University, Beijing, China.; Ningxia Key Laboratory of Cerebrocranial Diseases, Department of Anatomy, Histology and Embryology, School of Basic Medical Sciences, Ningxia Medical University, Yinchuan, China.
Xu Q; Department of Neurobiology, Beijing Institute for Brain Disorders, Beijing Center of Neural Regeneration and Repair, Key Laboratory for Neurodegenerative Diseases of the Ministry of Education, Capital Medical University, Beijing, China.
Cui X; Institute of Central Laboratory, Capital Institute of Pediatrics, Beijing, China.
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Źródło:
The International journal of neuroscience [Int J Neurosci] 2020 Feb; Vol. 130 (2), pp. 144-152. Date of Electronic Publication: 2019 Sep 26.
Typ publikacji:
Journal Article
MeSH Terms:
Brain/*growth & development
Cell Division/*physiology
GAP-43 Protein/*physiology
GTP-Binding Protein alpha Subunits/*metabolism
Neurogenesis/*physiology
Animals ; Brain/metabolism ; Embryo, Mammalian ; GAP-43 Protein/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Transgenic
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Evaluation of the concentration of growth associated protein-43 and glial cell-derived neurotrophic factor in degenerated intervertebral discs of the lumbosacral region of the spine.
Autorzy:
Staszkiewicz R; Department of Neurosurgery, 5th Military Clinical Hospital with the SP ZOZ Polyclinic in Krakow, Kraków, Poland.; Department of Histology, Cytophysiology, and Embryology, Faculty of Medicine in Zabrze, Academy of Silesia Zabrze, Poland.
Gralewski M; Department of Neurosurgery, 5th Military Clinical Hospital with the SP ZOZ Polyclinic in Krakow, Kraków, Poland.; Department of Histology, Cytophysiology, and Embryology, Faculty of Medicine in Zabrze, Academy of Silesia Zabrze, Poland.
Gładysz D; Department of Neurosurgery, 5th Military Clinical Hospital with the SP ZOZ Polyclinic in Krakow, Kraków, Poland.; Department of Histology, Cytophysiology, and Embryology, Faculty of Medicine in Zabrze, Academy of Silesia Zabrze, Poland.
Bryś K; Department of Histology, Cytophysiology, and Embryology, Faculty of Medicine in Zabrze, Academy of Silesia Zabrze, Poland.
Garczarek M; Department of Neurosurgery, 5th Military Clinical Hospital with the SP ZOZ Polyclinic in Krakow, Kraków, Poland.
Gadzieliński M; Department of Neurosurgery, 5th Military Clinical Hospital with the SP ZOZ Polyclinic in Krakow, Kraków, Poland.
Marcol W; Department of Physiology, School of Medicine in Katowice, 49613Medical University of Silesia, Katowice, Poland.; Department of Neurosurgery, Provincial Specialist Hospital No. 2 in Jastrzębie - Zdrój, Jastrzębie-Zdrój, Poland.
Sobański D; Department of Histology, Cytophysiology, and Embryology, Faculty of Medicine in Zabrze, Academy of Silesia Zabrze, Poland.; Department of Neurosurgery, Szpital sw Rafala w Krakowie, Krakow, Poland.
Grabarek BO; Department of Neurosurgery, 5th Military Clinical Hospital with the SP ZOZ Polyclinic in Krakow, Kraków, Poland.; Department of Histology, Cytophysiology, and Embryology, Faculty of Medicine in Zabrze, Academy of Silesia Zabrze, Poland.
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Źródło:
Molecular pain [Mol Pain] 2023 Jan-Dec; Vol. 19, pp. 17448069231158287.
Typ publikacji:
Journal Article
MeSH Terms:
Intervertebral Disc*/metabolism
Intervertebral Disc Degeneration*/metabolism
Humans ; Glial Cell Line-Derived Neurotrophic Factor/metabolism ; GAP-43 Protein/metabolism ; Lumbosacral Region
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Association of CSF GAP-43 and APOE ε4 with Cognition in Mild Cognitive Impairment and Alzheimer's Disease.
Autorzy:
Zhu Y; Department of Geriatrics, The First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou 310003, China.
Guo X; Department of Neurosurgery, Tongde Hospital of Zhejiang Province, Hangzhou 310012, China.
Zhu F; Department of Geriatrics, The First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou 310003, China.
Zhang Q; Department of Geriatrics, The First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou 310003, China.; Key Laboratory of Diagnosis and Treatment of Aging and Physic-Chemical Injury Diseases of Zhejiang Province, The First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou 310003, China.
Yang Y; Department of Geriatrics, The First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou 310003, China.; Key Laboratory of Diagnosis and Treatment of Aging and Physic-Chemical Injury Diseases of Zhejiang Province, The First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou 310003, China.
For The Alzheimer's Disease Neuroimaging Initiative
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Źródło:
Cells [Cells] 2022 Dec 21; Vol. 12 (1). Date of Electronic Publication: 2022 Dec 21.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Alzheimer Disease*/etiology
Cognitive Dysfunction*/complications
Humans ; Apolipoprotein E4/genetics ; GAP-43 Protein ; Cognition
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CSF GAP-43 as a biomarker of synaptic dysfunction is associated with tau pathology in Alzheimer's disease.
Autorzy:
Qiang Q; Department of Neurology, Cognitive Disorders Center, Huadong Hospital, Fudan University, Shanghai, China.; Department of Neurology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, Japan.
Skudder-Hill L; Yuquan Hospital, Tsinghua University School of Clinical Medicine, Beijing, China.
Toyota T; Department of Neurology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, Japan.
Wei W; Department of Neurology, Cognitive Disorders Center, Huadong Hospital, Fudan University, Shanghai, China.
Adachi H; Department of Neurology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2022 Oct 17; Vol. 12 (1), pp. 17392. Date of Electronic Publication: 2022 Oct 17.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
MeSH Terms:
Alzheimer Disease*/pathology
Cognitive Dysfunction*
Amyloid beta-Peptides/cerebrospinal fluid ; Biomarkers ; Disease Progression ; GAP-43 Protein ; Humans ; Peptide Fragments/cerebrospinal fluid ; tau Proteins/cerebrospinal fluid
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Research on Polycaprolactone-Gelatin Composite Scaffolds Carrying Nerve Growth Factor for the Repair of Spinal Cord Injury.
Autorzy:
Yang S; Department of Orthopedics, General Hospital of Ningxia Medical University, Yinchuan 750004, China.
Zhang N; Department of Surgery, Yinchuan Second People's Hospital, Yinchuan 750011, China.
Dong Y; Department of Orthopedics, General Hospital of Ningxia Medical University, Yinchuan 750004, China.
Zhang X; Senior Department of Orthopedics, The Fourth Medical Center of PLA General Hospital, Beijing 100048, China.
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Źródło:
Disease markers [Dis Markers] 2022 Sep 29; Vol. 2022, pp. 3880687. Date of Electronic Publication: 2022 Sep 29 (Print Publication: 2022).
Typ publikacji:
Journal Article; Retracted Publication
MeSH Terms:
Gelatin*
Spinal Cord Injuries*/metabolism
Spinal Cord Injuries*/therapy
Animals ; Collagen ; Female ; GAP-43 Protein ; Nerve Growth Factor ; Neurofilament Proteins ; Polyesters ; Rats ; Rats, Sprague-Dawley ; Recovery of Function/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
GAP-43 Induces the Differentiation of Bone Marrow-Derived Mesenchymal Stem Cells into Retinal Ganglial-Like Cells.
Autorzy:
Wang X; Department of Neurosurgery, The Second Hospital of Jilin University, Changchun, 130000 Jilin, China.
Ma C; Department of Neurosurgery, The First Hospital of Jilin University, Changchun, 130021 Jilin, China.
Nie L; Department of Ophthalmology, The Second Hospital of Jilin University, Changchun, 130000 Jilin, China.
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Źródło:
Computational and mathematical methods in medicine [Comput Math Methods Med] 2022 Apr 21; Vol. 2022, pp. 4949206. Date of Electronic Publication: 2022 Apr 21 (Print Publication: 2022).
Typ publikacji:
Journal Article; Retracted Publication
MeSH Terms:
Mesenchymal Stem Cells*
Retinal Ganglion Cells*
Animals ; Bone Marrow ; Cell Differentiation/physiology ; GAP-43 Protein/metabolism ; GAP-43 Protein/pharmacology ; Humans ; LIM-Homeodomain Proteins/metabolism ; LIM-Homeodomain Proteins/pharmacology ; Rats ; Transcription Factors/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Plasma neuregulin 1 as a synaptic biomarker in Alzheimer's disease: a discovery cohort study.
Autorzy:
Vrillon A; Université Paris Cité, Inserm U1144, Paris, France. .; Université Paris Cité, Center of Cognitive Neurology, Lariboisière Fernand-Widal Hospital, APHP, 200 rue du Faubourg Saint-Denis, 75010, Paris, France. .
Mouton-Liger F; Université Paris Cité, Inserm U1144, Paris, France.
Martinet M; Université Paris Cité, Inserm U1144, Paris, France.; Université Paris Cité, Center of Cognitive Neurology, Lariboisière Fernand-Widal Hospital, APHP, 200 rue du Faubourg Saint-Denis, 75010, Paris, France.
Cognat E; Université Paris Cité, Inserm U1144, Paris, France.; Université Paris Cité, Center of Cognitive Neurology, Lariboisière Fernand-Widal Hospital, APHP, 200 rue du Faubourg Saint-Denis, 75010, Paris, France.
Hourregue C; Université Paris Cité, Center of Cognitive Neurology, Lariboisière Fernand-Widal Hospital, APHP, 200 rue du Faubourg Saint-Denis, 75010, Paris, France.
Dumurgier J; Université Paris Cité, Center of Cognitive Neurology, Lariboisière Fernand-Widal Hospital, APHP, 200 rue du Faubourg Saint-Denis, 75010, Paris, France.
Bouaziz-Amar E; Université Paris Cité, Inserm U1144, Paris, France.; Université Paris Cité, Department of Biochemistry, APHP GHU Nord Lariboisière-Fernand Widal, Paris, France.
Brinkmalm A; Institute of Neuroscience and Physiology, The Salhgrenska Academy at the University of Gothenburg, Mölndal, Sweden.; Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.
Blennow K; Institute of Neuroscience and Physiology, The Salhgrenska Academy at the University of Gothenburg, Mölndal, Sweden.; Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.
Zetterberg H; Institute of Neuroscience and Physiology, The Salhgrenska Academy at the University of Gothenburg, Mölndal, Sweden.; Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.; UK Dementia Research Institute at UCL, London, UK.; Department of Neurodegenerative Disease, UCL Institute of Neurology, London, UK.; Hong Kong Center for Neurodegenerative Diseases, Hong Kong, China.
Hugon J; Université Paris Cité, Inserm U1144, Paris, France.; Université Paris Cité, Center of Cognitive Neurology, Lariboisière Fernand-Widal Hospital, APHP, 200 rue du Faubourg Saint-Denis, 75010, Paris, France.
Paquet C; Université Paris Cité, Inserm U1144, Paris, France.; Université Paris Cité, Center of Cognitive Neurology, Lariboisière Fernand-Widal Hospital, APHP, 200 rue du Faubourg Saint-Denis, 75010, Paris, France.
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Źródło:
Alzheimer's research & therapy [Alzheimers Res Ther] 2022 May 23; Vol. 14 (1), pp. 71. Date of Electronic Publication: 2022 May 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Alzheimer Disease*/cerebrospinal fluid
Cognitive Dysfunction*/diagnosis
Amyloid beta-Peptides/cerebrospinal fluid ; Biomarkers/cerebrospinal fluid ; Cohort Studies ; GAP-43 Protein ; Humans ; Neuregulin-1 ; Neurogranin/cerebrospinal fluid ; Peptide Fragments/cerebrospinal fluid ; Retrospective Studies ; tau Proteins/cerebrospinal fluid
Czasopismo naukowe
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Tytuł:
Mice lacking growth-associated protein 43 develop cardiac remodeling and hypertrophy.
Autorzy:
Bevere M; Department of Neuroscience, Imaging and Clinical Sciences, University 'G. d'Annunzio' of Chieti-Pescara, 66100, Chieti, Italy.; Center for Advanced Studies and Technology (CAST), University 'G. d'Annunzio' of Chieti-Pescara, 66100, Chieti, Italy.
Morabito C; Department of Neuroscience, Imaging and Clinical Sciences, University 'G. d'Annunzio' of Chieti-Pescara, 66100, Chieti, Italy.; Center for Advanced Studies and Technology (CAST), University 'G. d'Annunzio' of Chieti-Pescara, 66100, Chieti, Italy.
Guarnieri S; Department of Neuroscience, Imaging and Clinical Sciences, University 'G. d'Annunzio' of Chieti-Pescara, 66100, Chieti, Italy. .; Center for Advanced Studies and Technology (CAST), University 'G. d'Annunzio' of Chieti-Pescara, 66100, Chieti, Italy. .
Mariggiò MA; Department of Neuroscience, Imaging and Clinical Sciences, University 'G. d'Annunzio' of Chieti-Pescara, 66100, Chieti, Italy.; Center for Advanced Studies and Technology (CAST), University 'G. d'Annunzio' of Chieti-Pescara, 66100, Chieti, Italy.
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Źródło:
Histochemistry and cell biology [Histochem Cell Biol] 2022 May; Vol. 157 (5), pp. 547-556. Date of Electronic Publication: 2022 Feb 24.
Typ publikacji:
Journal Article
MeSH Terms:
Calmodulin*/genetics
Calmodulin*/metabolism
Ventricular Remodeling*
Animals ; Calcium/metabolism ; GAP-43 Protein/metabolism ; Heart ; Hypertrophy/metabolism ; Mice ; Mice, Knockout ; Myocytes, Cardiac/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Generating Vegfr3 reporter transgenic mouse expressing membrane-tagged Venus for visualization of VEGFR3 expression in vascular and lymphatic endothelial cells.
Autorzy:
Watanabe C; Department of Stem Cells and Human Disease Models, Research Center for Animal Life Science, Shiga University of Medical Science, Otsu, Shiga, Japan.
Matsushita J; Department of Stem Cells and Human Disease Models, Research Center for Animal Life Science, Shiga University of Medical Science, Otsu, Shiga, Japan.
Azami T; Department of Stem Cells and Human Disease Models, Research Center for Animal Life Science, Shiga University of Medical Science, Otsu, Shiga, Japan.
Tsukiyama-Fujii S; Department of Stem Cells and Human Disease Models, Research Center for Animal Life Science, Shiga University of Medical Science, Otsu, Shiga, Japan.
Tsukiyama T; Department of Stem Cells and Human Disease Models, Research Center for Animal Life Science, Shiga University of Medical Science, Otsu, Shiga, Japan.
Mizuno S; Animal Resource Center, University of Tsukuba, Tsukuba, Ibaraki, Japan.
Takahashi S; Animal Resource Center, University of Tsukuba, Tsukuba, Ibaraki, Japan.; Department of Anatomy and Embryology, Faculty of Medicine, University of Tsukuba, Tsukuba, Ibaraki, Japan.; International Institute for Integrative Sleep Medicine, Life Science Center, University of Tsukuba, Tsukuba, Ibaraki, Japan.
Ema M; Department of Stem Cells and Human Disease Models, Research Center for Animal Life Science, Shiga University of Medical Science, Otsu, Shiga, Japan.
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Źródło:
PloS one [PLoS One] 2019 Jan 02; Vol. 14 (1), pp. e0210060. Date of Electronic Publication: 2019 Jan 02 (Print Publication: 2019).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Bacterial Proteins/*metabolism
Endothelial Cells/*metabolism
GAP-43 Protein/*metabolism
Luminescent Proteins/*metabolism
Vascular Endothelial Growth Factor Receptor-3/*metabolism
Animals ; Bacterial Proteins/genetics ; Blood Vessels/metabolism ; Cell Line, Tumor ; Cell Membrane/metabolism ; Embryo, Mammalian/blood supply ; Embryo, Mammalian/cytology ; Embryo, Mammalian/metabolism ; Female ; GAP-43 Protein/genetics ; Gene Expression ; Luminescent Proteins/genetics ; Lymphatic Vessels/cytology ; Lymphatic Vessels/metabolism ; Mice, Inbred C57BL ; Mice, Inbred ICR ; Mice, Transgenic ; Microscopy, Confocal ; Neoplasms, Experimental/blood supply ; Neoplasms, Experimental/genetics ; Neoplasms, Experimental/metabolism ; Neovascularization, Pathologic/genetics ; Neovascularization, Pathologic/metabolism ; Vascular Endothelial Growth Factor Receptor-3/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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