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Wyszukujesz frazę ""Gene Expression Regulation, Neoplastic drug effects"" wg kryterium: Temat


Tytuł :
Long-Term Vemurafenib Exposure Induced Alterations of Cell Phenotypes in Melanoma:Increased Cell Migration and Its Association with EGFR Expression
Autorzy :
Molnár, Eszter
Garay, Tamás
Donia, Marco
Baranyi, Marcell
Rittler, Dominika
Berger, Walter
Tímár, József
Grusch, Michael
Hegedűs, Balázs
Pokaż więcej
Temat :
Adult
Aged
Cell Line, Tumor
Cell Movement/drug effects
Cell Proliferation/drug effects
Drug Resistance, Neoplasm/drug effects
Epithelial-Mesenchymal Transition/drug effects
ErbB Receptors/metabolism
Erlotinib Hydrochloride/pharmacology
Female
Gene Expression Regulation, Neoplastic/drug effects
Humans
Inhibitory Concentration 50
Male
Melanoma/drug therapy
Middle Aged
Mutation/genetics
Neoplasm Proteins/genetics
Phenotype
Proto-Oncogene Proteins B-raf/genetics
RNA, Messenger/genetics
Signal Transduction/drug effects
Time Factors
Vemurafenib/pharmacology
Journal Article
Źródło :
Molnár, E, Garay, T, Donia, M, Baranyi, M, Rittler, D, Berger, W, Tímár, J, Grusch, M & Hegedűs, B 2019, ' Long-Term Vemurafenib Exposure Induced Alterations of Cell Phenotypes in Melanoma : Increased Cell Migration and Its Association with EGFR Expression ', International Journal of Molecular Sciences, vol. 20, no. 18, 4484 . https://doi.org/10.3390/ijms20184484
Opis pliku :
application/pdf
Tytuł :
Predicting efficacy of epirubicin by a multigene assay in advanced breast cancer within a Danish Breast Cancer Cooperative Group (DBCG) cohort: a retrospective-prospective blinded study
Autorzy :
Buhl, Anna Sofie Kappel
Christensen, Troels Dreier
Christensen, Ib Jarle
Nelausen, Knud Mejer
Balslev, Eva
Knoop, Ann Søegaard
Brix, Eva Harder
Svensson, Else
Glavicic, Vesna
Luczak, Adam
Langkjer, Sven Tyge
Linnet, Søren
Jakobsen, Erik Hugger
Bogovic, Jurij
Ejlertsen, Bent
Rasmussen, Annie
Hansen, Anker
Knudsen, Steen
Nielsen, Dorte
Jensen, Peter Buhl
Pokaż więcej
Temat :
Retrospective Studies
Clinical Trial
Aged
RNA, Messenger/genetics
Epirubicin
Gene Expression Regulation, Neoplastic/drug effects
Breast Neoplasms/drug therapy
Disease Progression
Middle Aged
Epirubicin/administration & dosage
Biomarkers, Pharmacological
Disease-Free Survival
Prospective Studies
Precision medicine
Journal Article
Predictive biomarker
Advanced breast cancer
Adult
Female
Humans
Neoplasm Proteins/genetics
Proportional Hazards Models
Źródło :
Buhl, A S K, Christensen, T D, Christensen, I J, Nelausen, K M, Balslev, E, Knoop, A S, Brix, E H, Svensson, E, Glavicic, V, Luczak, A, Langkjer, S T, Linnet, S, Jakobsen, E H, Bogovic, J, Ejlertsen, B, Rasmussen, A, Hansen, A, Knudsen, S, Nielsen, D & Jensen, P B 2018, ' Predicting efficacy of epirubicin by a multigene assay in advanced breast cancer within a Danish Breast Cancer Cooperative Group (DBCG) cohort : a retrospective-prospective blinded study ', Breast Cancer Research and Treatment, vol. 172, no. 2, pp. 391–400 . https://doi.org/10.1007/s10549-018-4918-4
Buhl, A S K, Christensen, T D, Christensen, I J, Nelausen, K M, Balslev, E, Knoop, A S, Brix, E H, Svensson, E, Glavicic, V, Luczak, A, Langkjer, S T, Linnet, S, Jakobsen, E H, Bogovic, J, Ejlertsen, B, Rasmussen, A, Hansen, A, Knudsen, S, Nielsen, D & Jensen, P B 2018, ' Predicting efficacy of epirubicin by a multigene assay in advanced breast cancer within a Danish Breast Cancer Cooperative Group (DBCG) cohort : a retrospective-prospective blinded study ' Breast Cancer Research and Treatment, vol. 172, no. 2, pp. 391–400 . https://doi.org/10.1007/s10549-018-4918-4
Buhl, A S K, Christensen, T D, Christensen, I J, Nelausen, K M, Balslev, E, Knoop, A S, Brix, E H, Svensson, E, Glavicic, V, Luczak, A, Langkjer, S T, Linnet, S, Jakobsen, E H, Bogovic, J, Ejlertsen, B, Rasmussen, A, Hansen, A, Knudsen, S, Nielsen, D & Jensen, P B 2018, ' Predicting efficacy of epirubicin by a multigene assay in advanced breast cancer within a Danish Breast Cancer Cooperative Group (DBCG) cohort : a retrospective-prospective blinded study ', Breast Cancer Research and Treatment, vol. 172, no. 2, pp. 391-400 . https://doi.org/10.1007/s10549-018-4918-4
Breast Cancer Research and Treatment
Opis pliku :
application/pdf
Tytuł :
A Quinoline-Based DNA Methyltransferase Inhibitor as a Possible Adjuvant in Osteosarcoma Therapy
Autorzy :
Manara, Maria Cristina
Valente, Sergio
Cristalli, Camilla
Nicoletti, Giordano
Landuzzi, Lorena
Zwergel, Clemens
Mazzone, Roberta
Stazi, Giulia
Arimondo, Paola
Pasello, Michela
Guerzoni, Clara
Picci, Piero
Nanni, Patrizia
Lollini, Pier-Luigi
Mai, Antonello
Scotlandi, Katia
Pokaż więcej
Temat :
MESH: Animals
MESH: Osteosarcoma/drug therapy
MESH: Osteosarcoma/metabolism
MESH: Quinolines/chemistry
MESH: Male
MESH: DNA (Cytosine-5-)-Methyltransferase 1/genetics
MESH: Antineoplastic Combined Chemotherapy Protocols/therapeutic use
DNA Methyltransferase Inhibitor
MESH: Osteosarcoma/genetics
MESH: Aminoquinolines/pharmacology
MESH: Tumor Burden/genetics
chemotherapy
epigenetics
MESH: Aminoquinolines/administration & dosage
MESH: Doxorubicin/administration & dosage
MESH: Enzyme Inhibitors/chemistry
[SDV.CAN]Life Sciences [q-bio]/Cancer
MESH: Bone Neoplasms/drug therapy
MESH: Enzyme Inhibitors/administration & dosage
MESH: Enzyme Inhibitors/pharmacology
MESH: Xenograft Model Antitumor Assays
MESH: Bone Neoplasms/genetics
MESH: Benzamides/pharmacology
MESH: Tumor Burden/drug effects
MESH: Cisplatin/administration & dosage
MESH: Humans
MESH: Bone Neoplasms/metabolism
DNA methylation
MESH: Gene Expression Regulation, Neoplastic/drug effects
patient-derived xenograft (PDX)
MESH: Mice, Knockout
MESH: DNA (Cytosine-5-)-Methyltransferase 1/antagonists & inhibitors
MESH: DNA (Cytosine-5-)-Methyltransferase 1/metabolism
MESH: Benzamides/administration & dosage
[CHIM.THER]Chemical Sciences/Medicinal Chemistry
osteosarcoma
MESH: Cell Line, Tumor
Źródło :
Molecular Cancer Therapeutics
Molecular Cancer Therapeutics, American Association for Cancer Research, 2018, 17 (9), pp.1881-1892. ⟨10.1158/1535-7163.MCT-17-0818⟩
Opis pliku :
STAMPA
Tytuł :
A Quinoline-Based DNA Methyltransferase Inhibitor as a Possible Adjuvant in Osteosarcoma Therapy
Autorzy :
Manara, Maria Cristina
Valente, Sergio
Cristalli, Camilla
Nicoletti, Giordano
Landuzzi, Lorena
Zwergel, Clemens
Mazzone, Roberta
Stazi, Giulia
Arimondo, Paola
Pasello, Michela
Guerzoni, Clara
Picci, Piero
Nanni, Patrizia
Lollini, Pier-Luigi
Mai, Antonello
Scotlandi, Katia
Pokaż więcej
Temat :
MESH: Aminoquinolines/administration & dosage
MESH: Aminoquinolines/pharmacology
MESH: Cisplatin/administration & dosage
MESH: DNA (Cytosine-5-)-Methyltransferase 1/antagonists & inhibitors
MESH: DNA (Cytosine-5-)-Methyltransferase 1/genetics
MESH: DNA (Cytosine-5-)-Methyltransferase 1/metabolism
MESH: Doxorubicin/administration & dosage
MESH: Enzyme Inhibitors/administration & dosage
MESH: Enzyme Inhibitors/chemistry
MESH: Enzyme Inhibitors/pharmacology
MESH: Gene Expression Regulation, Neoplastic/drug effects
MESH: Humans
MESH: Animals
MESH: Male
MESH: Mice, Knockout
MESH: Osteosarcoma/drug therapy
MESH: Osteosarcoma/genetics
MESH: Osteosarcoma/metabolism
MESH: Quinolines/chemistry
MESH: Tumor Burden/drug effects
MESH: Tumor Burden/genetics
MESH: Xenograft Model Antitumor Assays
MESH: Antineoplastic Combined Chemotherapy Protocols/therapeutic use
MESH: Benzamides/administration & dosage
MESH: Benzamides/pharmacology
MESH: Bone Neoplasms/drug therapy
MESH: Bone Neoplasms/genetics
MESH: Bone Neoplasms/metabolism
MESH: Cell Line, Tumor
[CHIM.THER]Chemical Sciences/Medicinal Chemistry
[SDV.CAN]Life Sciences [q-bio]/Cancer
Źródło :
Molecular Cancer Therapeutics
Molecular Cancer Therapeutics, American Association for Cancer Research, 2018, 17 (9), pp.1881-1892. ⟨10.1158/1535-7163.MCT-17-0818⟩
Tytuł :
Active-site mTOR inhibitors augment HSV1-dICP0 infection in cancer cells via dysregulated eIF4E/4E-BP axis
Autorzy :
Zakaria, Chadi
Sean, Polen
Hoang, Huy-Dung
Leroux, Louis-Phillipe
Watson, Margaret
Workenhe, Samuel Tekeste
Hearnden, Jaclyn
Pearl, Dana
Truong, Vinh Tai
Robichaud, Nathaniel
Yanagiya, Akiko
Tahmasebi, Soroush
Jafarnejad, Seyed Mehdi
Jia, Jian-Jun
Pelin, Adrian
Diallo, Jean-Simon
Le Boeuf, Fabrice
Bell, John Cameron
Mossman, Karen Louise
Graber, Tyson Ernst
Jaramillo, Maritza
Sonenberg, Nahum
Alain, Tommy
Pokaż więcej
Temat :
MESH: Adaptator Proteins, Signal Transducing/genetics
MESH: Adaptator Proteins, Signal Transducing/metabolism
MESH: Mice
MESH: Neoplasms/complications
MESH: Animals
MESH: Neoplasms/genetics
MESH: Neoplasms/pathology
MESH: Neoplasms/virology
MESH: Organisms, Genetically Modified
MESH: Phosphoproteins/genetics
MESH: Phosphoproteins/metabolims
MESH: Protein Kinase Inhibitors/pharmacology
MESH: Herpes Simplex/complications
MESH: Signal Transduction/genetics
MESH: TOR Serine-Threonine Kinases/antagonists & inhibitiors
MESH: TOR Serine-Threonine Kinases/chemistry
MESH: Catalytic Domain/drug effects
MESH: Ubiquitin-Protein Ligases/deficiency
MESH: Ubiquitin-Protein Ligases/genetics
MESH: Vero Cells
MESH: Cells, Cultured
MESH: Cercopithecus aethiops
MESH: Eukaryotic Initiation Factor-4E/genetics
MESH: Herpes Simplex/genetics
MESH: Eukaryotic Initiation Factor-4E/metabolism
MESH: Gene Expression Regulation, Neoplastic/drug effects
MESH: HEK293 Cells
MESH: Herpes Simplex/pathology
MESH: Herpesvirus 1, Human/drug effects
MESH: Herpesvirus 1, Human/genetics
MESH: Humans
MESH: Immediate-Early Proteins/deficiency
MESH: Immediate-Early Proteins/genetics
[SDV.CAN]Life Sciences [q-bio]/Cancer
Źródło :
PLoS Pathogens, Public Library of Science, 2018, 14 (8), pp.e1007264. ⟨10.1371/journal.ppat.1007264⟩
Tytuł :
Active-site mTOR inhibitors augment HSV1-dICP0 infection in cancer cells via dysregulated eIF4E/4E-BP axis
Autorzy :
Chadi Zakaria
Polen Sean
Huy-Dung Hoang
Louis-Phillipe Leroux
Margaret Watson
Samuel Tekeste Workenhe
Jaclyn Hearnden
Dana Pearl
Vinh Tai Truong
Nathaniel Robichaud
Akiko Yanagiya
Soroush Tahmasebi
Seyed Mehdi Jafarnejad
Jian-Jun Jia
Adrian Pelin
Jean-Simon Diallo
Fabrice Le Boeuf
John Cameron Bell
Karen Louise Mossman
Tyson Ernst Graber
Maritza Jaramillo
Nahum Sonenberg
Tommy Alain
Pokaż więcej
Temat :
MESH: Animals
RC581-607
MESH: Herpes Simplex/pathology
MESH: Catalytic Domain/drug effects
MESH: Adaptator Proteins, Signal Transducing/metabolism
MESH: Herpesvirus 1, Human/genetics
Immunologic diseases. Allergy
MESH: Signal Transduction/genetics
[SDV.CAN]Life Sciences [q-bio]/Cancer
MESH: Phosphoproteins/genetics
MESH: Herpesvirus 1, Human/drug effects
MESH: TOR Serine-Threonine Kinases/antagonists & inhibitiors
MESH: Eukaryotic Initiation Factor-4E/metabolism
MESH: Adaptator Proteins, Signal Transducing/genetics
MESH: Cercopithecus aethiops
MESH: Organisms, Genetically Modified
MESH: Neoplasms/pathology
MESH: Ubiquitin-Protein Ligases/deficiency
MESH: HEK293 Cells
MESH: Eukaryotic Initiation Factor-4E/genetics
MESH: Neoplasms/genetics
MESH: Neoplasms/virology
MESH: Cells, Cultured
MESH: Humans
MESH: Immediate-Early Proteins/deficiency
MESH: Neoplasms/complications
QH301-705.5
MESH: Mice
MESH: Protein Kinase Inhibitors/pharmacology
MESH: Gene Expression Regulation, Neoplastic/drug effects
MESH: Immediate-Early Proteins/genetics
MESH: Ubiquitin-Protein Ligases/genetics
MESH: Herpes Simplex/genetics
MESH: TOR Serine-Threonine Kinases/chemistry
Biology (General)
MESH: Phosphoproteins/metabolims
MESH: Herpes Simplex/complications
MESH: Vero Cells
Źródło :
Zakaria, C, Sean, P, Hoang, H-D, Leroux, L-P, Watson, M, Workenhe, S T, Hearnden, J, Pearl, D, Truong, V T, Robichaud, N, Yanagiya, A, Tahmasebi, S, Jafarnejad, S M, Jia, J-J, Pelin, A, Diallo, J-S, Le Boeuf, F, Bell, J C, Mossman, K L, Graber, T E, Jaramillo, M, Sonenberg, N & Alain, T 2018, ' Active-site mTOR inhibitors augment HSV1-dICP0 infection in cancer cells via dysregulated eIF4E/4E-BP axis ', PLoS Pathogens, vol. 14, no. 8, e1007264 . https://doi.org/10.1371/journal.ppat.1007264
PLoS Pathogens, Public Library of Science, 2018, 14 (8), pp.e1007264. ⟨10.1371/journal.ppat.1007264⟩
PLoS Pathogens, Vol 14, Iss 8, p e1007264 (2018)
Opis pliku :
application/pdf
Tytuł :
Targeting miR-9 in gastric cancer cells using locked nucleic acid oligonucleotides
Autorzy :
Lima, Joana Filipa
Carvalho, Joana
Pinto-Ribeiro, Inês
Almeida, Carina
Wengel, Jesper
Cerqueira, Laura
Figueiredo, Céu
Oliveira, Carla
Azevedo, Nuno Filipe
Pokaż więcej
Temat :
Locked nucleic acid
Cytology
E-cadherin
Research Article
Antigens, CD
LNA-AMOs
Stomach Neoplasms/drug therapy
MicroRNAs/antagonists & inhibitors
QH573-671
Gene Expression Regulation, Neoplastic/drug effects
Cell Line, Tumor
Oligonucleotides/pharmacology
QH426-470
Cell Survival/drug effects
MiR-9
Genetics
FISH
MicroRNA
Down-Regulation
Humans
Cadherins/genetics
Cell Proliferation/drug effects
Źródło :
BMC Molecular Biology, Vol 19, Iss 1, Pp 1-13 (2018)
Lima, J F, Carvalho, J, Pinto-Ribeiro, I, Almeida, C, Wengel, J, Cerqueira, L, Figueiredo, C, Oliveira, C & Azevedo, N F 2018, ' Targeting miR-9 in gastric cancer cells using locked nucleic acid oligonucleotides ', BMC Molecular Biology, vol. 19, 6 . https://doi.org/10.1186/s12867-018-0107-6
Opis pliku :
application/pdf
Tytuł :
Functional role of Kv1.1 and Kv1.3 channels in the neoplastic progression steps of three cancer cell lines, elucidated by scorpion peptides
Autorzy :
Aissaoui, Dorra
Mlayah-Bellalouna, Saoussen
Jebali, Jed
Abdelkafi-Koubaa, Zaineb
Souid, Soumaya
Moslah, Wassim
Othman, Houcemeddine
Luis, José
Elayeb, Mohamed
Marrakchi, Naziha
Essafi-Benkhadir, Khadija
Srairi-Abid, Najet
Pokaż więcej
Temat :
MESH: Animals
biological phenomena, cell phenomena, and immunity
MESH: Scorpion Venoms/pharmacology
MESH: Potassium Channel Blockers/pharmacology
[SDV.BC]Life Sciences [q-bio]/Cellular Biology
[SDV.CAN]Life Sciences [q-bio]/Cancer
Potassium channel subtypes
MESH: Neoplasms/drug therapy
MESH: Peptides/chemistry
MESH: Cell Proliferation/drug effects
Proliferation
MESH: Neoplasms/genetics
MESH: Shaker Superfamily of Potassium Channels/genetics
MESH: Humans
MESH: Amino Acid Sequence/genetics
complex mixtures
MESH: Potassium Channel Blockers/chemistry
MESH: Potassium/metabolism
MESH: Gene Expression Regulation, Neoplastic/drug effects
nervous system
Adhesion and migration
[SDV.BBM]Life Sciences [q-bio]/Biochemistry, Molecular Biology
natural sciences
urogenital system
Cancer
MESH: Kv1.3 Potassium Channel/genetics
[SDV]Life Sciences [q-bio]
MESH: Peptides/pharmacology
MESH: Scorpion Venoms/chemistry
MESH: Scorpions/chemistry
MESH: Carcinogenesis/drug effects
Scorpion peptide
MESH: Cell Line, Tumor
Źródło :
International Journal of Biological Macromolecules
International Journal of Biological Macromolecules, Elsevier, 2018, 111, pp.1146 - 1155. ⟨10.1016/j.ijbiomac.2018.01.144⟩
Tytuł :
Hedgehog inhibition mediates radiation sensitivity in mouse xenograft models of human esophageal adenocarcinoma.
Autorzy :
Teichman, Jennifer
Dodbiba, Lorin
Thai, Henry
Fleet, Andrew
Morey, Trevor
Liu, Lucy
McGregor, Madison
Cheng, Dangxiao
Chen, Zhuo
Darling, Gail
Brhane, Yonathan
Song, Yuyao
Espin-Garcia, Osvaldo
Xu, Wei
Girgis, Hala
Schwock, Joerg
MacKay, Helen
Bristow, Robert
Ailles, Laurie
Liu, Geoffrey
Pokaż więcej
Temat :
Cancer Treatment
Digestive System
Animal Models
Pharmaceutics
Radiation Tolerance/drug effects
Gene Expression Regulation, Neoplastic/drug effects
Chemotherapy
Oncology
Mice, SCID
Animals
Xenograft Model Antitumor Assays
Cell Proliferation/drug effects
Biology and Life Sciences
Signal Transduction
Chemoradiotherapy
Hedgehog Signaling
Cancer Chemotherapy
Radiation Therapy
Science
Epithelium
Biphenyl Compounds/pharmacology
Gene Expression
Mouse Models
Research Article
Gastrointestinal Tract
Anatomy
Hedgehog Proteins/antagonists & inhibitors
Mice
Apoptosis/drug effects
Clinical Medicine
Genetics
Biological Tissue
Adenocarcinoma/drug therapy
Esophageal Neoplasms/drug therapy
Pyridines/pharmacology
Clinical Oncology
Experimental Organism Systems
Drug Therapy
Combination Chemotherapy
Research and Analysis Methods
Tumor Cells, Cultured
Medicine
Mice, Inbred NOD
Cell Biology
Model Organisms
Cell Signaling
Medicine and Health Sciences
Humans
Esophagus
Źródło :
Teichman, J, Dodbiba, L, Thai, H, Fleet, A, Morey, T, Liu, L, McGregor, M, Cheng, D, Chen, Z, Darling, G, Brhane, Y, Song, Y, Espin-Garcia, O, Xu, W, Girgis, H, Schwock, J, MacKay, H, Bristow, R, Ailles, L & Liu, G 2018, ' Hedgehog inhibition mediates radiation sensitivity in mouse xenograft models of human esophageal adenocarcinoma ', PLoS ONE, vol. 13, no. 5, pp. e0194809 . https://doi.org/10.1371/journal.pone.0194809
PLoS ONE, Vol 13, Iss 5, p e0194809 (2018)
Tytuł :
Bortezomib prevents cytarabine resistance in MCL, which is characterized by down-regulation of dCK and up-regulation of SPIB resulting in high NF-κB activity
Autorzy :
Ek, Sara
Freiburghaus, Catja
Emruli, Venera Kuci
Johansson, Angelica
Eskelund, Christian Winther
Grønbæk, Kirsten
Olsson, Roger
Ek, Fredrik
Jerkeman, Mats
Pokaż więcej
Temat :
Cytarabine
Research Article
SPIB
Gene Expression Regulation, Neoplastic/drug effects
Lymphoma, Mantle-Cell/genetics
DCK
Research Support, Non-U.S. Gov't
Neoplasms. Tumors. Oncology. Including cancer and carcinogens
Histone Deacetylase Inhibitors/pharmacology
Signal Transduction/drug effects
Deoxycytidine Kinase/genetics
MCL
Transcription Factors/genetics
Bortezomib/pharmacology
Cytarabine/pharmacology
Dose-Response Relationship, Drug
Transcription, Genetic
NF-κB
Drug Resistance, Neoplasm/drug effects
Antineoplastic Agents/pharmacology
DNA-Binding Proteins/genetics
NF-kappa B/metabolism
Cell Line, Tumor
RC254-282
Journal Article
Gene Expression Profiling
Humans
Źródło :
BMC Cancer, Vol 18, Iss 1, Pp 1-17 (2018)
Freiburghaus, C, Emruli, V K, Johansson, A, Eskelund, C W, Grønbæk, K, Olsson, R, Ek, F, Jerkeman, M & Ek, S 2018, ' Bortezomib prevents cytarabine resistance in MCL, which is characterized by down-regulation of dCK and up-regulation of SPIB resulting in high NF-κB activity ', BMC Cancer, vol. 18, no. 1, 466 . https://doi.org/10.1186/s12885-018-4346-1
Opis pliku :
application/pdf
Tytuł :
Williams syndrome transcription factor (WSTF) acts as an activator of estrogen receptor signaling in breast cancer cells and the effect can be abrogated by 1α,25-dihydroxyvitamin D3
Autorzy :
Lundqvist, Johan
Kirkegaard, Tove
Laenkholm, Anne Vibeke
Duun-Henriksen, Anne Katrine
Bak, Martin
Feldman, David
Lykkesfeldt, Anne E
Pokaż więcej
Temat :
Aromatase/genetics
Breast Neoplasms/genetics
Down-Regulation/drug effects
Estrogen Receptor alpha/genetics
Female
Gene Expression Regulation, Neoplastic/drug effects
Humans
MCF-7 Cells
Promoter Regions, Genetic
Signal Transduction
Transcription Factors/genetics
Vitamin D/analogs & derivatives
Źródło :
Lundqvist, J, Kirkegaard, T, Laenkholm, A V, Duun-Henriksen, A K, Bak, M, Feldman, D & Lykkesfeldt, A E 2018, ' Williams syndrome transcription factor (WSTF) acts as an activator of estrogen receptor signaling in breast cancer cells and the effect can be abrogated by 1α,25-dihydroxyvitamin D 3 ', Journal of Steroid Biochemistry and Molecular Biology, vol. 177, pp. 171-178 . https://doi.org/10.1016/j.jsbmb.2017.06.003

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