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Wyszukujesz frazę ""Han, Jing"" wg kryterium: Autor

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Wyświetlanie 1-20 z 76
Tytuł:
Role of RING-Type E3 Ubiquitin Ligases in Inflammatory Signalling and Inflammatory Bowel Disease.
Autorzy:
Zhu L; Division of Gastroenterology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Li Y; Division of Gastroenterology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Zhou L; Division of Gastroenterology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Yang G; Division of Gastroenterology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Wang Y; Division of Gastroenterology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Han J; Division of Gastroenterology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Li L; Division of Gastroenterology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Zhang S; Division of Gastroenterology, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
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Źródło:
Mediators of inflammation [Mediators Inflamm] 2020 Aug 10; Vol. 2020, pp. 5310180. Date of Electronic Publication: 2020 Aug 10 (Print Publication: 2020).
Typ publikacji:
Journal Article; Review
MeSH Terms:
Inflammation/*metabolism
Inflammatory Bowel Diseases/*metabolism
Ubiquitin-Protein Ligases/*metabolism
Animals ; Autophagy ; Biomarkers/metabolism ; Colitis/metabolism ; Humans ; Immune System ; MAP Kinase Signaling System ; Mice ; Phosphatidylinositol 3-Kinases/metabolism ; Protein Processing, Post-Translational ; RNA, Untranslated/metabolism ; Signal Transduction ; Transforming Growth Factor beta/metabolism ; Ubiquitin/chemistry ; Ubiquitination
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Metformin suppresses retinal angiogenesis and inflammation in vitro and in vivo.
Autorzy:
Han J; Department of Ophthalmology, Henry Ford Health System, Detroit, Michigan, United States of America.; Department of Ophthalmology, Tangdu Hospital, Fourth Military Medical University, Xi'an, Shaanxi, People's Republic of China.
Li Y; Department of Ophthalmology, Henry Ford Health System, Detroit, Michigan, United States of America.
Liu X; Department of Ophthalmology, Henry Ford Health System, Detroit, Michigan, United States of America.
Zhou T; Department of Ophthalmology, Henry Ford Health System, Detroit, Michigan, United States of America.
Sun H; Departments of Ophthalmology and Anatomy and Cell Biology, Wayne State University, School of Medicine, Detroit, Michigan, United States of America.
Edwards P; Department of Ophthalmology, Henry Ford Health System, Detroit, Michigan, United States of America.
Gao H; Department of Ophthalmology, Henry Ford Health System, Detroit, Michigan, United States of America.
Yu FS; Departments of Ophthalmology and Anatomy and Cell Biology, Wayne State University, School of Medicine, Detroit, Michigan, United States of America.
Qiao X; Department of Ophthalmology, Henry Ford Health System, Detroit, Michigan, United States of America.
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Źródło:
PloS one [PLoS One] 2018 Mar 07; Vol. 13 (3), pp. e0193031. Date of Electronic Publication: 2018 Mar 07 (Print Publication: 2018).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Inflammation/*prevention & control
Metformin/*pharmacology
Neovascularization, Pathologic/*prevention & control
Retina/*drug effects
Animals ; Apoptosis/drug effects ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Cells, Cultured ; Endothelial Cells/drug effects ; Endothelial Cells/metabolism ; Humans ; Hypoglycemic Agents/pharmacology ; Inflammation/genetics ; Inflammation/metabolism ; Inflammation Mediators/metabolism ; Mice, Inbred C57BL ; Mice, Knockout ; Neovascularization, Pathologic/genetics ; Neovascularization, Pathologic/metabolism ; Receptors, LDL/genetics ; Receptors, LDL/metabolism ; Retina/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Qishenyiqi protects ligation-induced left ventricular remodeling by attenuating inflammation and fibrosis via STAT3 and NF-κB signaling pathway.
Autorzy:
Li C; Modern Research Center for Traditional Chinese Medicine, Beijing University of Chinese Medicine, Beijing, China.
Wang Y; Basic Medical College, Beijing University of Chinese Medicine, Beijing, China.
Qiu Q; Anzhen Hospital of Capital Medical University, Beijing, China.
Shi T; Basic Medical College, Beijing University of Chinese Medicine, Beijing, China.
Wu Y; Basic Medical College, Beijing University of Chinese Medicine, Beijing, China.
Han J; Basic Medical College, Beijing University of Chinese Medicine, Beijing, China.
Chai X; Modern Research Center for Traditional Chinese Medicine, Beijing University of Chinese Medicine, Beijing, China.
Wang W; Basic Medical College, Beijing University of Chinese Medicine, Beijing, China.
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Źródło:
PloS one [PLoS One] 2014 Aug 14; Vol. 9 (8), pp. e104255. Date of Electronic Publication: 2014 Aug 14 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Drugs, Chinese Herbal/*pharmacology
Fibrosis/*drug therapy
Heart Ventricles/*drug effects
Inflammation/*drug therapy
NF-kappa B/*metabolism
STAT3 Transcription Factor/*metabolism
Ventricular Remodeling/*drug effects
Angiotensin II/metabolism ; Animals ; Captopril/pharmacology ; Fibrosis/metabolism ; Heart Failure/drug therapy ; Heart Failure/metabolism ; Heart Ventricles/metabolism ; Hemodynamics/drug effects ; Inflammation/metabolism ; Interleukin-6/metabolism ; Ligation/adverse effects ; Male ; Matrix Metalloproteinases/metabolism ; NADPH Oxidases/metabolism ; Oxidative Stress/drug effects ; Protective Agents/pharmacology ; Rats ; Rats, Sprague-Dawley ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Involvement of Advanced Glycation End Products in the Pathogenesis of Diabetic Retinopathy
Autorzy:
Jing Xu
Lin-Jiang Chen
Jian Yu
Han-Jing Wang
Fan Zhang
Qiong Liu
Jing Wu
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Temat:
Advanced glycation end products
Diabetic retinopathy
Endothelial dysfunction
Inflammation
Angiogenesis
Physiology
QP1-981
Biochemistry
QD415-436
Źródło:
Cellular Physiology and Biochemistry, Vol 48, Iss 2, Pp 705-717 (2018)
Opis pliku:
electronic resource
Relacje:
https://www.karger.com/Article/FullText/491897; https://doaj.org/toc/1015-8987; https://doaj.org/toc/1421-9778
Dostęp URL:
https://doaj.org/article/407191c05e5b4afca902405166924435  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Wyświetlanie 1-20 z 76

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