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Wyszukujesz frazę ""Han, R"" wg kryterium: Autor


Tytuł:
Encephalomyocarditis Virus 2A Protein Inhibited Apoptosis by Interaction with Annexin A2 through JNK/c-Jun Pathway.
Autorzy:
Han R; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing 100193, China.; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Liang L; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing 100193, China.
Qin T; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing 100193, China.
Xiao S; College of Veterinary Medicine, Northwest A&F University, Yangling 712100, China.
Liang R; Institute of Animal Sciences, Chinese Academy of Agricultural Sciences, Beijing 100193, China.
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Źródło:
Viruses [Viruses] 2022 Feb 09; Vol. 14 (2). Date of Electronic Publication: 2022 Feb 09.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/genetics
Annexin A2/*metabolism
Encephalomyocarditis virus/*physiology
JNK Mitogen-Activated Protein Kinases/*metabolism
Proto-Oncogene Proteins c-jun/*metabolism
Viral Proteins/*metabolism
Animals ; Annexin A2/genetics ; Cell Line ; Cricetinae ; JNK Mitogen-Activated Protein Kinases/genetics ; MAP Kinase Signaling System ; Protein Binding ; Proto-Oncogene Proteins c-jun/genetics ; Recombinant Proteins/genetics ; Recombinant Proteins/metabolism ; Swine ; Viral Proteins/genetics ; Virus Replication
Czasopismo naukowe
Tytuł:
TaPCNA plays a role in programmed cell death after UV-B exposure in wheat (Triticum aestivum).
Autorzy:
Du M; Higher Education Key Laboratory of Plant Molecular and Environment Stress Response (Shanxi Normal University) in Shanxi Province, Linfen City, Shanxi Province, China.
Zhang Y; Higher Education Key Laboratory of Plant Molecular and Environment Stress Response (Shanxi Normal University) in Shanxi Province, Linfen City, Shanxi Province, China.
Chen H; Higher Education Key Laboratory of Plant Molecular and Environment Stress Response (Shanxi Normal University) in Shanxi Province, Linfen City, Shanxi Province, China; and Corresponding authors. Emails: ; .
Han R; Higher Education Key Laboratory of Plant Molecular and Environment Stress Response (Shanxi Normal University) in Shanxi Province, Linfen City, Shanxi Province, China; and Corresponding authors. Emails: ; .
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Źródło:
Functional plant biology : FPB [Funct Plant Biol] 2021 Sep; Vol. 48 (10), pp. 1029-1038.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Triticum*/genetics
Gene Silencing ; Mesophyll Cells ; Plant Leaves
Czasopismo naukowe
Tytuł:
Oridonin Promotes Apoptosis and Restrains the Viability and Migration of Bladder Cancer by Impeding TRPM7 Expression via the ERK and AKT Signaling Pathways.
Autorzy:
Che X; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Zhan J; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Zhao F; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Zhong Z; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Chen M; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Han R; Department of Urology, The Second Hospital of Tianjin Medical University, Tianjin Institute of Urology, 300211 Tianjin, China.
Wang Y; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
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Źródło:
BioMed research international [Biomed Res Int] 2021 Jul 05; Vol. 2021, pp. 4340950. Date of Electronic Publication: 2021 Jul 05 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*/drug effects
Apoptosis*/genetics
Cell Movement*/drug effects
Cell Movement*/genetics
MAP Kinase Signaling System*/drug effects
MAP Kinase Signaling System*/genetics
Diterpenes, Kaurane/*pharmacology
Protein Serine-Threonine Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
TRPM Cation Channels/*metabolism
Urinary Bladder Neoplasms/*pathology
Animals ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Proliferation/genetics ; Cell Survival/drug effects ; Cell Survival/genetics ; Female ; Humans ; Mice, Inbred C57BL ; Protein Serine-Threonine Kinases/genetics ; TRPM Cation Channels/genetics ; Tumor Stem Cell Assay ; Urinary Bladder Neoplasms/genetics ; Xenograft Model Antitumor Assays ; Mice
Czasopismo naukowe
Tytuł:
Astaxanthin protects against early burn-wound progression in rats by attenuating oxidative stress-induced inflammation and mitochondria-related apoptosis.
Autorzy:
Fang Q; Department of Burns, Second Affiliated Hospital, Zhejiang University, College of Medicine, 88 Jiefang Road, Hangzhou 310009, Zhejiang, China.; Department of Plastic Surgery, Binjiang Branch, Second Affiliated Hospital, Zhejiang University, College of Medicine, 1511 Jianghong Road, Hangzhou 310000, Zhejiang, China.
Guo S; Department of Burns, Second Affiliated Hospital, Zhejiang University, College of Medicine, 88 Jiefang Road, Hangzhou 310009, Zhejiang, China.
Zhou H; Department of Burns, Second Affiliated Hospital, Zhejiang University, College of Medicine, 88 Jiefang Road, Hangzhou 310009, Zhejiang, China.
Han R; Department of Dermatology, Sir Run Run Shaw Hospital, School of Medicine, Zhejiang University, 3 East Qingchun Road, Hangzhou 310020, Zhejiang, China.
Wu P; Department of Burns, Second Affiliated Hospital, Zhejiang University, College of Medicine, 88 Jiefang Road, Hangzhou 310009, Zhejiang, China.
Han C; Department of Burns, Second Affiliated Hospital, Zhejiang University, College of Medicine, 88 Jiefang Road, Hangzhou 310009, Zhejiang, China.
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Źródło:
Scientific reports [Sci Rep] 2017 Jan 27; Vol. 7, pp. 41440. Date of Electronic Publication: 2017 Jan 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/drug effects
Disease Progression*
Oxidative Stress*/drug effects
Burns/*drug therapy
Burns/*pathology
Inflammation/*pathology
Mitochondria/*metabolism
Animals ; Burns/genetics ; Chromones/pharmacology ; Inflammation Mediators/metabolism ; Male ; Mitochondria/drug effects ; Models, Biological ; Morpholines/pharmacology ; Phosphorylation/drug effects ; Protective Agents/pharmacology ; Protective Agents/therapeutic use ; Proto-Oncogene Proteins c-akt/metabolism ; Rats, Sprague-Dawley ; Up-Regulation/drug effects ; Up-Regulation/genetics ; Xanthophylls/administration & dosage ; Xanthophylls/pharmacology ; Xanthophylls/therapeutic use ; bcl-Associated Death Protein/metabolism
Czasopismo naukowe
Tytuł:
Clematichinenoside (AR) Attenuates Hypoxia/Reoxygenation-Induced H9c2 Cardiomyocyte Apoptosis via a Mitochondria-Mediated Signaling Pathway.
Autorzy:
Ding H; State Key Laboratory of Natural Medicines, Department of Physiology, China Pharmaceutical University, Nanjing 210009, China. .; College of Pharmacy, Xinjiang Medical University, Urumqi 830011, China. .
Han R; College of Pharmacy, Xinjiang Medical University, Urumqi 830011, China. .
Chen X; College of Pharmacy, Xinjiang Medical University, Urumqi 830011, China. .
Fang W; State Key Laboratory of Natural Medicines, Department of Physiology, China Pharmaceutical University, Nanjing 210009, China. .
Liu M; Cancer Hospital Affiliated to Xinjiang Medical University, Urumqi 830011, China. .
Wang X; Xinjiang Key Laboratory of Medical Animal Model Research, Clinical Medical Research Institute of the First Affiliated Hospital of Xinjiang Medical University, Urumqi 830011, China. .
Wei Q; Xinjiang Key Laboratory of Medical Animal Model Research, Clinical Medical Research Institute of the First Affiliated Hospital of Xinjiang Medical University, Urumqi 830011, China. .
Kodithuwakku ND; Institute of Indigenous Medicine, University of Colombo, Rajagiriya 11600, Sri Lanka. darshi_.
Li Y; State Key Laboratory of Natural Medicines, Department of Physiology, China Pharmaceutical University, Nanjing 210009, China. .
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2016 May 30; Vol. 21 (6). Date of Electronic Publication: 2016 May 30.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Mitochondria/*drug effects
Mitochondria/*metabolism
Myocytes, Cardiac/*drug effects
Myocytes, Cardiac/*metabolism
Saponins/*pharmacology
Signal Transduction/*drug effects
Animals ; Biomarkers ; Caspase 3/metabolism ; Cell Line, Tumor ; Cell Survival/drug effects ; Cytochromes c/metabolism ; Hypoxia/metabolism ; Ion Channel Gating ; Membrane Potential, Mitochondrial/drug effects ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Myocardial Reperfusion Injury/metabolism ; Protective Agents/chemistry ; Protective Agents/pharmacology ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Saponins/chemistry ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł:
Cadmium telluride quantum dots (CdTe-QDs) and enhanced ultraviolet-B (UV-B) radiation trigger antioxidant enzyme metabolism and programmed cell death in wheat seedlings.
Autorzy:
Chen H; Higher Education Key Laboratory of Plant Molecular and Environmental Stress Response (Shanxi Normal University) in Shanxi Province, Linfen, China; School of Life Science, Shanxi Normal University, Linfen, China.
Gong Y; Higher Education Key Laboratory of Plant Molecular and Environmental Stress Response (Shanxi Normal University) in Shanxi Province, Linfen, China; School of Chemistry and Material Science, Shanxi Normal University, Linfen, China.
Han R; Higher Education Key Laboratory of Plant Molecular and Environmental Stress Response (Shanxi Normal University) in Shanxi Province, Linfen, China; School of Life Science, Shanxi Normal University, Linfen, China.
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Źródło:
PloS one [PLoS One] 2014 Oct 20; Vol. 9 (10), pp. e110400. Date of Electronic Publication: 2014 Oct 20 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/drug effects
Apoptosis*/radiation effects
Quantum Dots*
Antioxidants/*metabolism
Cadmium Compounds/*pharmacology
Oxidoreductases/*metabolism
Plant Proteins/*metabolism
Seedlings/*enzymology
Tellurium/*pharmacology
Triticum/*enzymology
Ultraviolet Rays/*adverse effects
Czasopismo naukowe
Tytuł:
Cathepsin L plays a role in quinolinic acid-induced NF-Κb activation and excitotoxicity in rat striatal neurons.
Autorzy:
Wang YR; Department of Pharmacology and Laboratory of Aging and Nervous Diseases (SZS0703), Soochow University School of Pharmaceutical Science, Wen Jing Road, Suzhou, China.
Qin S
Han R
Wu JC
Liang ZQ
Qin ZH
Wang Y
Pokaż więcej
Źródło:
PloS one [PLoS One] 2013 Sep 20; Vol. 8 (9), pp. e75702. Date of Electronic Publication: 2013 Sep 20 (Print Publication: 2013).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Cathepsin L/*physiology
Corpus Striatum/*drug effects
NF-kappa B/*metabolism
Neurons/*drug effects
Quinolinic Acid/*pharmacology
Receptors, N-Methyl-D-Aspartate/*metabolism
Animals ; Blotting, Western ; Cell Proliferation ; Cells, Cultured ; Corpus Striatum/metabolism ; Cysteine Proteinase Inhibitors/pharmacology ; Dipeptides/pharmacology ; Fluorescent Antibody Technique ; Glutamic Acid/metabolism ; Ketones/pharmacology ; Male ; Neurons/metabolism ; Proteolysis ; Rats ; Rats, Sprague-Dawley ; Receptors, N-Methyl-D-Aspartate/antagonists & inhibitors ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
Tytuł:
p53 induction contributes to excitotoxic neuronal death in rat striatum through apoptotic and autophagic mechanisms.
Autorzy:
Wang Y; Department of Pharmacology and Laboratory of Aging and Nervous Diseases (SZS0703), Soochow University School of Medicine, Suzhou, China.
Dong XX
Cao Y
Liang ZQ
Han R
Wu JC
Gu ZL
Qin ZH
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Źródło:
The European journal of neuroscience [Eur J Neurosci] 2009 Dec; Vol. 30 (12), pp. 2258-70. Date of Electronic Publication: 2009 Dec 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Autophagy/*drug effects
Corpus Striatum/*drug effects
Neurotoxins/*toxicity
Quinolinic Acid/*toxicity
Tumor Suppressor Protein p53/*metabolism
Adenine/analogs & derivatives ; Adenine/toxicity ; Animals ; Apoptosis/physiology ; Autophagy/physiology ; Benzothiazoles/toxicity ; Cell Death/drug effects ; Cell Death/physiology ; Corpus Striatum/physiology ; Corpus Striatum/ultrastructure ; DNA Fragmentation/drug effects ; Genes, p53 ; NF-kappa B/antagonists & inhibitors ; Neurons/drug effects ; Neurons/physiology ; Neurons/ultrastructure ; Peptides/toxicity ; Rats ; Rats, Sprague-Dawley ; Receptors, N-Methyl-D-Aspartate/agonists ; Toluene/analogs & derivatives ; Toluene/toxicity ; Transcriptional Activation ; Tumor Suppressor Protein p53/antagonists & inhibitors
Czasopismo naukowe
Tytuł:
Resveratrol induces apoptosis and differentiation in acute promyelocytic leukemia (NB4) cells.
Autorzy:
Cao Y; Department of Pharmacology, Institute of Materia Medica, Peking Union Medical College and Chinese Academy of Medical Sciences, Beijing 100050, China. />Wang F
Liu HY
Fu ZD
Han R
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Źródło:
Journal of Asian natural products research [J Asian Nat Prod Res] 2005 Aug; Vol. 7 (4), pp. 633-41.
Typ publikacji:
Journal Article
MeSH Terms:
Anticarcinogenic Agents/*pharmacology
Apoptosis/*drug effects
Cell Differentiation/*drug effects
Leukemia, Promyelocytic, Acute/*pathology
Stilbenes/*pharmacology
Antineoplastic Agents/pharmacology ; Cell Proliferation/drug effects ; DNA Damage ; Humans ; Resveratrol ; Tretinoin/pharmacology ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł:
Role of lncRNA NR2F1-AS1 and lncRNA H19 Genes in Hepatocellular Carcinoma and Their Effects on Biological Function of Huh-7
Autorzy:
Ji WC
Bao GJ
Yang FW
Sun L
Han R
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Temat:
lncrna h19
lncrna nr2f1-as1
hepatocellular carcinoma (hcc)
trophoblast cells
invasion
apoptosis
Neoplasms. Tumors. Oncology. Including cancer and carcinogens
RC254-282
Źródło:
Cancer Management and Research, Vol Volume 13, Pp 941-951 (2021)
Opis pliku:
electronic resource
Relacje:
https://www.dovepress.com/role-of-lncrna-nr2f1-as1-and-lncrna-h19-genes-in-hepatocellular-carcin-peer-reviewed-article-CMAR; https://doaj.org/toc/1179-1322
Dostęp URL:
https://doaj.org/article/51989b38926547628aad7e01282838d3  Link otwiera się w nowym oknie
Czasopismo naukowe
Tytuł:
Salvianolic Acid Alleviated Blood–Brain Barrier Permeability in Spontaneously Hypertensive Rats by Inhibiting Apoptosis in Pericytes via P53 and the Ras/Raf/MEK/ERK Pathway
Autorzy:
Wu Q
Yuan X
Li B
Han R
Zhang H
Xiu R
Pokaż więcej
Temat:
salvianolic acid a
blood-brain barrier
hypertension
pericytes
apoptosis
Therapeutics. Pharmacology
RM1-950
Źródło:
Drug Design, Development and Therapy, Vol Volume 14, Pp 1523-1534 (2020)
Opis pliku:
electronic resource
Relacje:
https://www.dovepress.com/salvianolic-acid-alleviated-bloodndashbrain-barrier-permeability-in-sp-peer-reviewed-article-DDDT; https://doaj.org/toc/1177-8881
Dostęp URL:
https://doaj.org/article/4a9645d3001c4371b87edec92d7b55d1  Link otwiera się w nowym oknie
Czasopismo naukowe

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