Autor: "Haribabu, Bodduluri" - OpacWWW

Skocz do pozycji: 1.

Tytuł:: Type 3 secretion system induced leukotriene B4 synthesis by leukocytes is actively inhibited by Yersinia pestis to evade early immune recognition.
Autorzy:: Brady A; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Sheneman KR; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Pulsifer AR; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Price SL; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Garrison TM; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Maddipati KR; Department of Pathology, Lipidomics Core Facility, Wayne State University, Detroit, Michigan, United States of America.
Bodduluri SR; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Pan J; Biostatistics and Bioinformatics Facility, Brown Cancer Center, University of Louisville, Louisville, Kentucky, United States of America.
Boyd NL; Center for Cardiometabolic Science, Christina Lee Brown Environment Institute, Division of Environmental Medicine, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Zheng JJ; Center for Cardiometabolic Science, Christina Lee Brown Environment Institute, Division of Environmental Medicine, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Rai SN; Department of Pharmacology and Toxicology, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Hellmann J; Center for Cardiometabolic Science, Christina Lee Brown Environment Institute, Division of Environmental Medicine, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Haribabu B; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.
Uriarte SM; Deptartment of Oral Immunology & Infectious Diseases, University of Louisville, Louisville, Kentucky, United States of America.
Lawrenz MB; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, Kentucky, United States of America.; Center for Predictive Medicine for Biodefense and Emerging Infectious Diseases, Louisville, Kentucky, United States of America.; Pokaż więcej
Źródło:: PLoS pathogens [PLoS Pathog] 2024 Jan 25; Vol. 20 (1), pp. e1011280. Date of Electronic Publication: 2024 Jan 25 (Print Publication: 2024).
Typ publikacji:: Journal Article
MeSH Terms:: Yersinia pestis*/metabolism
Plague*/microbiology
Humans ; Animals ; Mice ; Type III Secretion Systems/metabolism ; Leukotriene B4/metabolism ; Leukocytes/metabolism ; Inflammation ; Bacterial Proteins/metabolism

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Skocz do pozycji: 2.

Tytuł:: Gut microbiota modulates lung fibrosis severity following acute lung injury in mice.
Autorzy:: Chioma OS; Division of Infectious Disease, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.
Mallott EK; Department of Biological Sciences, Vanderbilt University, Nashville, TN, USA.; Vanderbilt Microbiome Initiative, Vanderbilt University, Nashville, TN, USA.
Chapman A; Division of Infectious Disease, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.
Van Amburg JC; Division of Infectious Disease, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.
Wu H; Division of Infectious Disease, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.
Shah-Gandhi B; Division of Infectious Disease, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.
Dey N; Division of Infectious Disease, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.
Kirkland ME; Division of Infectious Disease, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.
Blanca Piazuelo M; Division of Gastroenterology, Hepatology and Nutrition Research, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.
Johnson J; Department of Pathology, Microbiology, and Immunology, Vanderbilt University School of Medicine, Nashville, TN, USA.
Bernard GR; Division of Pulmonary and Critical Care, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA.
Bodduluri SR; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, KY, USA.
Davison S; Comparative Medicine Research Unit, University of Louisville School of Medicine, Louisville, KY, USA.
Haribabu B; Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, KY, USA.
Bordenstein SR; Department of Biological Sciences, Vanderbilt University, Nashville, TN, USA.; Vanderbilt Microbiome Initiative, Vanderbilt University, Nashville, TN, USA.; Department of Pathology, Microbiology, and Immunology, Vanderbilt University School of Medicine, Nashville, TN, USA.
Drake WP; Division of Infectious Disease, Department of Medicine, Vanderbilt University School of Medicine, Nashville, TN, USA. .; Department of Pathology, Microbiology, and Immunology, Vanderbilt University School of Medicine, Nashville, TN, USA. .; Pokaż więcej
Źródło:: Communications biology [Commun Biol] 2022 Dec 22; Vol. 5 (1), pp. 1401. Date of Electronic Publication: 2022 Dec 22.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:: Acute Lung Injury*
Gastrointestinal Microbiome*
Pulmonary Fibrosis*/metabolism
Pulmonary Fibrosis*/pathology
Animals ; Humans ; Mice ; Disease Models, Animal ; Interleukin-17 ; Mice, Inbred C57BL ; Fibroblasts/metabolism ; Fibroblasts/microbiology

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Skocz do pozycji: 3.

Tytuł:: Gut Microbiota Conversion of Dietary Ellagic Acid into Bioactive Phytoceutical Urolithin A Inhibits Heme Peroxidases.
Autorzy:: Saha P; Department of Nutritional Sciences, The Pennsylvania State University, University Park, Pennsylvania, United States of America.
Yeoh BS; Department of Nutritional Sciences, The Pennsylvania State University, University Park, Pennsylvania, United States of America.
Singh R; Department of Microbiology and Immunology, James Graham Brown Cancer Center, University of Louisville, Louisville, Kentucky, United States of America.
Chandrasekar B; Institute for Stem Cell Biology and Regenerative Medicine (inStem), UAS-GKVK Campus, Bellary Road, Bangalore, Karnataka, India.
Vemula PK; Institute for Stem Cell Biology and Regenerative Medicine (inStem), UAS-GKVK Campus, Bellary Road, Bangalore, Karnataka, India.; Ramalingaswami ReEntry Fellow, Dept. of Biotechnology, Govt. of India.
Haribabu B; Department of Microbiology and Immunology, James Graham Brown Cancer Center, University of Louisville, Louisville, Kentucky, United States of America.
Vijay-Kumar M; Department of Nutritional Sciences, The Pennsylvania State University, University Park, Pennsylvania, United States of America.; Department of Medicine, The Pennsylvania State University Medical Center, Hershey, Pennsylvania, United States of America.
Jala VR; Department of Microbiology and Immunology, James Graham Brown Cancer Center, University of Louisville, Louisville, Kentucky, United States of America.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2016 Jun 02; Vol. 11 (6), pp. e0156811. Date of Electronic Publication: 2016 Jun 02 (Print Publication: 2016).
Typ publikacji:: Journal Article
MeSH Terms:: Diet*
Gastrointestinal Microbiome*/drug effects
Coumarins/*pharmacology
Ellagic Acid/*metabolism
Heme/*metabolism
Peroxidase/*antagonists & inhibitors
Animals ; Biocatalysis/drug effects ; Bone Marrow Cells/cytology ; Coumarins/chemical synthesis ; Coumarins/metabolism ; Edema/pathology ; Humans ; Immunity, Innate/drug effects ; Iron/pharmacology ; Iron Chelating Agents/pharmacology ; Lactoperoxidase/antagonists & inhibitors ; Lactoperoxidase/metabolism ; Lipocalin-2/metabolism ; Mice, Inbred C57BL ; Neutrophils/drug effects ; Neutrophils/metabolism ; Peroxidase/metabolism ; Reactive Oxygen Species/metabolism ; Tetradecanoylphorbol Acetate/pharmacology ; Time Factors

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Skocz do pozycji: 4.

Tytuł:: Renal Protection by Genetic Deletion of the Atypical Chemokine Receptor ACKR2 in Diabetic OVE Mice.
Autorzy:: Zheng S; Department of Pediatrics, University of Louisville, Louisville, KY 40202, USA.
Coventry S; Department of Pathology, University of Louisville, Louisville, KY 40202, USA.
Cai L; Department of Pediatrics, University of Louisville, Louisville, KY 40202, USA.
Powell DW; Department of Medicine, University of Louisville, Louisville, KY 40202, USA.
Jala VR; Department of Microbiology and Immunology, University of Louisville, Louisville, KY 40202, USA.
Haribabu B; Department of Microbiology and Immunology, University of Louisville, Louisville, KY 40202, USA.
Epstein PN; Department of Pediatrics, University of Louisville, Louisville, KY 40202, USA.; Pokaż więcej
Źródło:: Journal of diabetes research [J Diabetes Res] 2016; Vol. 2016, pp. 5362506. Date of Electronic Publication: 2015 Dec 20.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Gene Deletion*
Albuminuria/*prevention & control
Diabetic Nephropathies/*prevention & control
Kidney/*metabolism
Nephritis/*prevention & control
Receptors, Chemokine/*deficiency
Age Factors ; Albuminuria/genetics ; Albuminuria/metabolism ; Albuminuria/physiopathology ; Animals ; Diabetic Nephropathies/genetics ; Diabetic Nephropathies/metabolism ; Diabetic Nephropathies/physiopathology ; Disease Models, Animal ; Fibrosis ; Gene Expression Regulation ; Genotype ; Humans ; Kidney/pathology ; Kidney/physiopathology ; Mice, Inbred C57BL ; Mice, Knockout ; Nephritis/genetics ; Nephritis/metabolism ; Nephritis/physiopathology ; Phenotype ; Receptors, Chemokine/analysis ; Receptors, Chemokine/genetics

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Skocz do pozycji: 5.

Tytuł:: Enhanced expression of G-protein coupled estrogen receptor (GPER/GPR30) in lung cancer.
Autorzy:: Jala VR; James Graham Brown Cancer Center, Department of Microbiology and Immunology, 505 South Hancock Street, Room 323, CTR Building, Louisville, KY 40202, USA. />Radde BN
Haribabu B
Klinge CM; Pokaż więcej
Źródło:: BMC cancer [BMC Cancer] 2012 Dec 28; Vol. 12, pp. 624. Date of Electronic Publication: 2012 Dec 28.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:: Adenocarcinoma/*metabolism
Carcinoma, Non-Small-Cell Lung/*metabolism
Lung Neoplasms/*metabolism
Neoplasm Proteins/*metabolism
Receptors, Estrogen/*metabolism
Receptors, G-Protein-Coupled/*metabolism
Animals ; Blotting, Western ; Cell Line, Tumor ; Humans ; Immunohistochemistry ; Mice ; RNA, Messenger/metabolism ; Real-Time Polymerase Chain Reaction

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Skocz do pozycji: 6.

Tytuł:: Ligand-induced nuclear translocation of S1P(1) receptors mediates Cyr61 and CTGF transcription in endothelial cells.
Autorzy:: Estrada R; Department of Microbiology and Immunology, Gheens Center on Aging, University of Louisville Health Sciences Center, Louisville, KY 40202, USA.
Wang L
Jala VR
Lee JF
Lin CY
Gray RD
Haribabu B
Lee MJ; Pokaż więcej
Źródło:: Histochemistry and cell biology [Histochem Cell Biol] 2009 Feb; Vol. 131 (2), pp. 239-49. Date of Electronic Publication: 2008 Oct 21.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:: Active Transport, Cell Nucleus*
Transcription, Genetic*
Connective Tissue Growth Factor/*genetics
Cysteine-Rich Protein 61/*genetics
Endothelial Cells/*metabolism
Receptors, Lysosphingolipid/*metabolism
Cell Membrane ; Endothelium, Vascular ; Humans ; Ligands ; Lysophospholipids/metabolism ; Sphingosine/analogs & derivatives ; Sphingosine/metabolism

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