Informacja

Drogi użytkowniku, aplikacja do prawidłowego działania wymaga obsługi JavaScript. Proszę włącz obsługę JavaScript w Twojej przeglądarce.

Przeglądasz jako GOŚĆ

Wyszukujesz frazę ""Hepatocytes metabolism"" wg kryterium: Temat


Tytuł :
Iron and liver fibrosis:Mechanistic and clinical aspects
Autorzy :
Mehta, Kosha J
Farnaud, Sebastien Je
Sharp, Paul A
Pokaż więcej
Temat :
Carcinoma, Hepatocellular/pathology
Disease Progression
Hemochromatosis/metabolism
Hepatocytes/metabolism
Humans
Iron/metabolism
Iron Chelating Agents/therapeutic use
Liver/cytology
Liver Cirrhosis/diagnosis
Liver Diseases, Alcoholic/pathology
Liver Neoplasms/pathology
Non-alcoholic Fatty Liver Disease/pathology
Phlebotomy
Gastroenterology
Źródło :
Mehta, K J, Farnaud, S J & Sharp, P A 2019, ' Iron and liver fibrosis : Mechanistic and clinical aspects ', World Journal of Gastroenterology, vol. 25, no. 5, pp. 521-538 . https://doi.org/10.3748/wjg.v25.i5.521
Opis pliku :
application/pdf
Tytuł :
Necroptosis microenvironment directs lineage commitment in liver cancer
Autorzy :
Seehawer, Marco
Heinzmann, Florian
D’Artista, Luana
Harbig, Jule
Roux, Pierre-François
Hoenicke, Lisa
Dang, Hien
Klotz, Sabrina
Robinson, Lucas
Doré, Grégory
Rozenblum, Nir
Kang, Tae-Won
Chawla, Rishabh
Buch, Thorsten
Vucur, Mihael
Roth, Mareike
Zuber, Johannes
Luedde, Tom
Sipos, Bence
Longerich, Thomas
Heikenwalder, Mathias
Wang, Xin Wei
Bischof, Oliver
Zender, Lars
Pokaż więcej
Temat :
Cancer epigenetics
Cancer microenvironment
MESH: Animals
MESH: Apoptosis*/genetics
MESH: Cytokines/metabolism
MESH: DNA Transposable Elements/genetics
MESH: DNA-Binding Proteins/genetics
MESH: DNA-Binding Proteins/metabolism
MESH: Disease Models, Animal
MESH: Epigenesis, Genetic/genetics
MESH: Female
MESH: Gene Expression Profiling
MESH: Genes, myc
MESH: Genes, ras
MESH: Carcinogenesis/genetics
MESH: Hepatocytes/metabolism
MESH: Hepatocytes/pathology
MESH: Humans
MESH: Liver Neoplasms/genetics
MESH: Liver Neoplasms/pathology
MESH: Male
MESH: Mice
MESH: Mosaicism
MESH: Necrosis*/genetics
MESH: Proto-Oncogene Proteins c-akt/genetics
MESH: Carcinoma, Hepatocellular/genetics
MESH: T-Box Domain Proteins/genetics
MESH: T-Box Domain Proteins/metabolism
MESH: Transcription Factors/genetics
MESH: Transcription Factors/metabolism
MESH: Tumor Microenvironment
MESH: Carcinoma, Hepatocellular/pathology
MESH: Cell Differentiation
MESH: Cell Lineage*/genetics
MESH: Cholangiocarcinoma/genetics
MESH: Cholangiocarcinoma/pathology
MESH: Cyclin-Dependent Kinase Inhibitor p16/deficiency
[SDV.CAN]Life Sciences [q-bio]/Cancer
Źródło :
Nature, Nature Publishing Group, 2018, 562 (7725), pp.69-75. ⟨10.1038/s41586-018-0519-y⟩
Tytuł :
Necroptosis microenvironment directs lineage commitment in liver cancer
Autorzy :
Seehawer, Marco
Heinzmann, Florian
D’Artista, Luana
Harbig, Jule
Roux, Pierre-François
Hoenicke, Lisa
Dang, Hien
Klotz, Sabrina
Robinson, Lucas
Doré, Grégory
Rozenblum, Nir
Kang, Tae-Won
Chawla, Rishabh
Buch, Thorsten
Vucur, Mihael
Roth, Mareike
Zuber, Johannes
Luedde, Tom
Sipos, Bence
Longerich, Thomas
Heikenwalder, Mathias
Wang, Xin Wei
Bischof, Oliver
Zender, Lars
Pokaż więcej
Temat :
MESH: Animals
MESH: Male
MESH: Disease Models, Animal
610 Medicine & health
MESH: Epigenesis, Genetic/genetics
MESH: Hepatocytes/pathology
MESH: Transcription Factors/metabolism
MESH: Cholangiocarcinoma/pathology
Cancer epigenetics
MESH: DNA-Binding Proteins/metabolism
MESH: Transcription Factors/genetics
MESH: Carcinoma, Hepatocellular/genetics
MESH: Cytokines/metabolism
[SDV.CAN]Life Sciences [q-bio]/Cancer
Institute of Laboratory Animal Science
MESH: Female
Cancer microenvironment
MESH: Liver Neoplasms/pathology
MESH: T-Box Domain Proteins/metabolism
MESH: DNA-Binding Proteins/genetics
MESH: Gene Expression Profiling
MESH: Genes, myc
MESH: Carcinogenesis/genetics
MESH: Apoptosis*/genetics
MESH: Necrosis*/genetics
MESH: Liver Neoplasms/genetics
MESH: Mosaicism
MESH: Hepatocytes/metabolism
MESH: Humans
MESH: Cholangiocarcinoma/genetics
digestive system diseases
MESH: Carcinoma, Hepatocellular/pathology
MESH: DNA Transposable Elements/genetics
MESH: Mice
MESH: Tumor Microenvironment
MESH: Proto-Oncogene Proteins c-akt/genetics
MESH: T-Box Domain Proteins/genetics
MESH: Cell Differentiation
570 Life sciences
biology
MESH: Cell Lineage*/genetics
MESH: Cyclin-Dependent Kinase Inhibitor p16/deficiency
MESH: Genes, ras
Źródło :
Nature, Nature Publishing Group, 2018, 562 (7725), pp.69-75. ⟨10.1038/s41586-018-0519-y⟩
Seehawer, Marco; Heinzmann, Florian; D’Artista, Luana; Harbig, Jule; Roux, Pierre-François; Hoenicke, Lisa; Dang, Hien; Klotz, Sabrina; Robinson, Lucas; Doré, Grégory; Rozenblum, Nir; Kang, Tae-Won; Chawla, Rishabh; Buch, Thorsten; Vucur, Mihael; Roth, Mareike; Zuber, Johannes; Luedde, Tom; Sipos, Bence; Longerich, Thomas; Heikenwälder, Mathias; Wang, Xin Wei; Bischof, Oliver; Zender, Lars (2018). Necroptosis microenvironment directs lineage commitment in liver cancer. Nature, 562(7725):69-75.
Opis pliku :
application/pdf
Tytuł :
Secretion of Hepatitis C Virus Replication Intermediates Reduces Activation of Toll-Like Receptor 3 in Hepatocytes
Autorzy :
Grünvogel, Oliver
Colasanti, Ombretta
Lee, Ji-Young
Klöss, Volker
Belouzard, Sandrine
Reustle, Anna
Esser-Nobis, Katharina
Hesebeck-Brinckmann, Jasper
Mutz, Pascal
Hoffmann, Katrin
Mehrabi, Arianeb
Koschny, Ronald
Vondran, Florian W.R.
Gotthardt, Daniel
Schnitzler, Paul
Neumann-Haefelin, Christoph
Thimme, Robert
Binder, Marco
Bartenschlager, Ralf
Dubuisson, Jean
Dalpke, Alexander
Lohmann, Volker
Pokaż więcej
Temat :
MESH: RNA, Viral/metabolism
Rab27a
MESH: Hepacivirus/physiology
MESH: Primary Cell Culture
MESH: Immunity, Innate
MESH: Interferons/metabolism
MESH: Hepatocytes/immunology
MESH: RNA, Viral/immunology
MESH: Hepatocytes/metabolism
MESH: RNA, Double-Stranded/isolation & purification
[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/Virology
Exosomes
MESH: Hepatitis C, Chronic/immunology
Escape
MESH: Cell Line
MESH: RNA, Double-Stranded/metabolism
MESH: Extracellular Vesicles/metabolism
MESH: Signal Transduction/immunology
MESH: Toll-Like Receptor 3/immunology
viruses
MESH: Hepatitis C, Chronic/virology
MESH: Virus Replication/immunology
[SDV.IMM.II]Life Sciences [q-bio]/Immunology/Innate immunity
MESH: Humans
MESH: Extracellular Vesicles/immunology
MESH: Hepatitis C, Chronic/blood
MESH: Toll-Like Receptor 3/metabolism
Virology
MESH: Interferons/immunology
[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology
MESH: RNA, Double-Stranded/immunology
MESH: Host-Pathogen Interactions/immunology
MESH: RNA, Viral/isolation & purification
Źródło :
Gastroenterology, WB Saunders, 2018, 154 (8), pp.2237-2251.e16. ⟨10.1053/j.gastro.2018.03.020⟩
Tytuł :
Farnesoid X receptor : A 'homeostat' for hepatic nutrient metabolism
Autorzy :
Massafra, Vittoria
van Mil, Saskia W C
Pokaż więcej
Temat :
Animals
Bile Acids and Salts/metabolism
Energy Metabolism/genetics
Food
Glucose/metabolism
Hepatocytes/metabolism
Homeostasis/genetics
Humans
Intestinal Mucosa/metabolism
Liver/metabolism
Receptors, Cytoplasmic and Nuclear/physiology
Journal Article
Research Support, Non-U.S. Gov't
Review
Źródło :
Biochimica et Biophysica Acta. Molecular Basis of Disease, 1864(1), 45. Elsevier
Opis pliku :
image/pdf
Tytuł :
HFE mRNA expression is responsive to intracellular and extracellular iron loading:short communication
Autorzy :
Mehta, Kosha J.
Farnaud, Sebastien
Patel, Vinood B.
Pokaż więcej
Temat :
Antigens, CD/metabolism
Cells, Cultured
Gene Expression Regulation
Hemochromatosis/genetics
Hemochromatosis Protein/genetics
Hep G2 Cells
Hepatocytes/metabolism
Hepcidins/genetics
Histocompatibility Antigens Class I/genetics
Homeostasis
Humans
Iron/metabolism
Liver/metabolism
RNA, Messenger/genetics
Real-Time Polymerase Chain Reaction
Receptors, Transferrin/metabolism
Transferrin/metabolism
Molecular Biology
Genetics
Źródło :
Mehta, K J, Farnaud, S & Patel, V B 2017, ' HFE mRNA expression is responsive to intracellular and extracellular iron loading : short communication ', Molecular Biology Reports, vol. 44, no. 5, pp. 399-403 . https://doi.org/10.1007/s11033-017-4123-2
Opis pliku :
application/pdf
Tytuł :
Retinol saturase coordinates liver metabolism by regulating ChREBP activity
Autorzy :
Birkenfeld, Andreas L.
Schupp, Michael
Heidenreich, Steffi
Witte, Nicole
Weber, Pamela
Goehring, Isabel
Tolkachov, Alexander
von Loeffelholz, Christian
Döcke, Stephanie
Bauer, Michael
Stockmann, Martin
Pfeiffer, Andreas F. H.
Pietzke, Matthias
Kempa, Stefan
Muenzner, Matthias
Pokaż więcej
Temat :
Hepatocytes/metabolism
Fatty Liver/metabolism
Glucose/metabolism
Lipid Metabolism
Mice
Cancer Research
Article
Basic Helix-Loop-Helix Leucine Zipper Transcription Factors/metabolism
Oxidoreductases Acting on CH-CH Group Donors/genetics
Liver/metabolism
Mice, Inbred C57BL
Cardiovascular and Metabolic Diseases
Triglycerides/blood
Science
Animals
Humans
Male
Źródło :
Heidenreich, S, Witte, N, Weber, P, Goehring, I, Tolkachov, A, von Loeffelholz, C, Döcke, S, Bauer, M, Stockmann, M, Pfeiffer, A F H, Birkenfeld, A L, Pietzke, M, Kempa, S, Muenzner, M & Schupp, M 2017, ' Retinol saturase coordinates liver metabolism by regulating ChREBP activity ', Nature Communications, vol. 8, no. 1, pp. 384 . https://doi.org/10.1038/s41467-017-00430-w
Nature Communications, Vol 8, Iss 1, Pp 1-10 (2017)
Opis pliku :
application/pdf
Tytuł :
Reply to: 'Arginase 1: a potential marker of a common pattern of liver steatosis in HCV and NAFLD children'
Autorzy :
Assenat, Eric
Akkari, Leila
Moreau, Marie
Hibner, Urszula
Pokaż więcej
Temat :
[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology
[SDV.BBM]Life Sciences [q-bio]/Biochemistry, Molecular Biology
Hepatology
Chronic/*metabolism Hepatocytes/*metabolism Humans Liver/*metabolism Male Viral Proteins/*metabolism
Animals Hepacivirus/*metabolism Hepatitis C
ComputingMilieux_MISCELLANEOUS
Źródło :
Journal of Hepatology, Elsevier, 2015, 62 (5), pp.1208-1209. ⟨10.1016/j.jhep.2015.02.002⟩
J Hepatol, 2015, 62 (5), pp.1208--9. ⟨10.1016/j.jhep.2015.02.002 S0168-8278(15)00076-8 [pii]⟩

Ta witryna wykorzystuje pliki cookies do przechowywania informacji na Twoim komputerze. Pliki cookies stosujemy w celu świadczenia usług na najwyższym poziomie, w tym w sposób dostosowany do indywidualnych potrzeb. Korzystanie z witryny bez zmiany ustawień dotyczących cookies oznacza, że będą one zamieszczane w Twoim komputerze. W każdym momencie możesz dokonać zmiany ustawień dotyczących cookies