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    Wyświetlanie 1-8 z 8
    Tytuł:
    Ongoing evolution of the Mycobacterium tuberculosis lactate dehydrogenase reveals the pleiotropic effects of bacterial adaption to host pressure.
    Autorzy:
    Stanley S; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Wang X; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Liu Q; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Kwon YY; Department of Molecular Metabolism, Harvard T. H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Frey AM; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Hicks ND; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Vickers AJ; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Hui S; Department of Molecular Metabolism, Harvard T. H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Fortune SM; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.; Ragon Institute of MGH, MIT, and Harvard, Cambridge, Massachusetts, United States of America.; Broad Institute of MIT and Harvard, Cambridge, Massachusetts, United States of America.
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    Źródło:
    PLoS pathogens [PLoS Pathog] 2024 Feb 29; Vol. 20 (2), pp. e1012050. Date of Electronic Publication: 2024 Feb 29 (Print Publication: 2024).
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Mycobacterium tuberculosis*/metabolism
    Humans ; L-Lactate Dehydrogenase ; Lactic Acid/metabolism ; Pyruvates/metabolism ; Quinones/metabolism ; Phosphates/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Tuberculosis treatment failure associated with evolution of antibiotic resilience.
    Autorzy:
    Liu Q; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Zhu J; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Dulberger CL; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.; Department of Molecular and Cellular Biology, Harvard University, Boston, MA, USA.; Present address: BioNTech US, Cambridge, MA, USA.
    Stanley S; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Wilson S; Department of Molecular and Cellular Biology, Harvard University, Boston, MA, USA.
    Chung ES; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, Boston, MA 02111, USA.; Department of Biomedical Engineering, Tufts University School of Engineering, Medford, MA 02115, USA.
    Wang X; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Culviner P; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Liu YJ; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Hicks ND; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Babunovic GH; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Giffen SR; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Aldridge BB; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, Boston, MA 02111, USA.; Department of Biomedical Engineering, Tufts University School of Engineering, Medford, MA 02115, USA.
    Garner EC; Department of Molecular and Cellular Biology, Harvard University, Boston, MA, USA.
    Rubin EJ; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Chao MC; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.
    Fortune SM; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA 02115, USA.; Broad Institute of MIT and Harvard, Cambridge, Massachusetts, USA.
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    Źródło:
    Science (New York, N.Y.) [Science] 2022 Dec 09; Vol. 378 (6624), pp. 1111-1118. Date of Electronic Publication: 2022 Dec 08.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Tuberculosis*/drug therapy
    Tuberculosis*/microbiology
    Mycobacterium tuberculosis*/drug effects
    Mycobacterium tuberculosis*/genetics
    Mycobacterium tuberculosis*/isolation & purification
    Drug Resistance, Bacterial*/genetics
    Tuberculosis, Multidrug-Resistant*/genetics
    Evolution, Molecular*
    Antibiotics, Antitubercular*/pharmacology
    Antibiotics, Antitubercular*/therapeutic use
    Bacterial Proteins*/genetics
    Transcription Factors*/genetics
    Humans ; Genomics ; Treatment Failure ; Selection, Genetic
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Loss of RNase J leads to multi-drug tolerance and accumulation of highly structured mRNA fragments in Mycobacterium tuberculosis.
    Autorzy:
    Martini MC; Department of Biology and Biotechnology, Worcester Polytechnic Institute, Worcester, Massachusetts, United States of America.
    Hicks ND; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Xiao J; Program in Bioinformatics and Computational Biology, Worcester Polytechnic Institute, Worcester, Massachusetts, United States of America.
    Alonso MN; Department of Biology and Biotechnology, Worcester Polytechnic Institute, Worcester, Massachusetts, United States of America.
    Barbier T; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Sixsmith J; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Fortune SM; Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Shell SS; Department of Biology and Biotechnology, Worcester Polytechnic Institute, Worcester, Massachusetts, United States of America.; Program in Bioinformatics and Computational Biology, Worcester Polytechnic Institute, Worcester, Massachusetts, United States of America.
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    Źródło:
    PLoS pathogens [PLoS Pathog] 2022 Jul 13; Vol. 18 (7), pp. e1010705. Date of Electronic Publication: 2022 Jul 13 (Print Publication: 2022).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Mycobacterium tuberculosis*/genetics
    Mycobacterium tuberculosis*/metabolism
    Ribonucleases*/genetics
    Ribonucleases*/metabolism
    Drug Tolerance ; Endoribonucleases/genetics ; Endoribonucleases/metabolism ; RNA, Messenger/genetics ; RNA, Messenger/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Mutations in dnaA and a cryptic interaction site increase drug resistance in Mycobacterium tuberculosis.
    Autorzy:
    Hicks ND; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Giffen SR; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Culviner PH; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Chao MC; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Dulberger CL; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Liu Q; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Stanley S; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Brown J; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Sixsmith J; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Wolf ID; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Fortune SM; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.; Ragon Institute of MGH, MIT, and Harvard, Cambridge, Massachusetts, United States of America.; Broad Institute of MIT and Harvard, Cambridge, Massachusetts, United States of America.
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    Źródło:
    PLoS pathogens [PLoS Pathog] 2020 Nov 30; Vol. 16 (11), pp. e1009063. Date of Electronic Publication: 2020 Nov 30 (Print Publication: 2020).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural
    MeSH Terms:
    Drug Resistance, Multiple, Bacterial*
    Antitubercular Agents/*pharmacology
    Bacterial Proteins/*genetics
    DNA-Binding Proteins/*genetics
    Isoniazid/*pharmacology
    Mycobacterium tuberculosis/*genetics
    Tuberculosis/*microbiology
    DNA Replication ; Genome-Wide Association Study ; Humans ; Mutation ; Mycobacterium tuberculosis/drug effects ; Tuberculosis/drug therapy
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    A natural polymorphism of Mycobacterium tuberculosis in the esxH gene disrupts immunodomination by the TB10.4-specific CD8 T cell response.
    Autorzy:
    Sutiwisesak R; Immunology and Microbiology Program, Graduate School of Biomedical Science, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.; Department of Microbiology and Physiological Systems, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.
    Hicks ND; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Boyce S; Department of Microbiology and Physiological Systems, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.
    Murphy KC; Immunology and Microbiology Program, Graduate School of Biomedical Science, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.; Department of Microbiology and Physiological Systems, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.
    Papavinasasundaram K; Department of Microbiology and Physiological Systems, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.
    Carpenter SM; Department of Microbiology and Physiological Systems, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.
    Boucau J; Ragon Institute of Massachusetts General Hospital, Massachusetts Institute of Technology and Harvard University, Cambridge, MA, United States of America.
    Joshi N; Ragon Institute of Massachusetts General Hospital, Massachusetts Institute of Technology and Harvard University, Cambridge, MA, United States of America.
    Le Gall S; Ragon Institute of Massachusetts General Hospital, Massachusetts Institute of Technology and Harvard University, Cambridge, MA, United States of America.
    Fortune SM; Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, United States of America.
    Sassetti CM; Immunology and Microbiology Program, Graduate School of Biomedical Science, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.; Department of Microbiology and Physiological Systems, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.
    Behar SM; Immunology and Microbiology Program, Graduate School of Biomedical Science, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.; Department of Microbiology and Physiological Systems, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.
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    Źródło:
    PLoS pathogens [PLoS Pathog] 2020 Oct 19; Vol. 16 (10), pp. e1009000. Date of Electronic Publication: 2020 Oct 19 (Print Publication: 2020).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural
    MeSH Terms:
    Antigens, Bacterial/*immunology
    Mycobacterium tuberculosis/*genetics
    Mycobacterium tuberculosis/*immunology
    Animals ; Antigens, Bacterial/genetics ; CD4-Positive T-Lymphocytes/immunology ; CD8-Positive T-Lymphocytes/immunology ; Epitopes, T-Lymphocyte/immunology ; Humans ; Mice ; Mice, Inbred C57BL ; Tuberculosis/immunology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Detection of Low-Level Mixed-Population Drug Resistance in Mycobacterium tuberculosis Using High Fidelity Amplicon Sequencing.
    Autorzy:
    Colman RE; Translational Genomics Research Institute, Flagstaff, AZ, United States of America.
    Schupp JM; Translational Genomics Research Institute, Flagstaff, AZ, United States of America.
    Hicks ND; Translational Genomics Research Institute, Flagstaff, AZ, United States of America.
    Smith DE; Translational Genomics Research Institute, Flagstaff, AZ, United States of America.
    Buchhagen JL; Translational Genomics Research Institute, Flagstaff, AZ, United States of America.
    Valafar F; San Diego State University, San Diego, CA, United States of America.
    Crudu V; Phthisiopneumology Institute (PPI), Chisinau, Republic of Moldova.
    Romancenco E; University of California San Diego, San Diego, CA, United States of America.
    Noroc E; Phthisiopneumology Institute (PPI), Chisinau, Republic of Moldova.
    Jackson L; University of California San Diego, San Diego, CA, United States of America.
    Catanzaro DG; University of Arkansas College of Education and Health Professions, Fayetteville, AR, United States of America.
    Rodwell TC; University of California San Diego, San Diego, CA, United States of America.
    Catanzaro A; University of California San Diego, San Diego, CA, United States of America.
    Keim P; Translational Genomics Research Institute, Flagstaff, AZ, United States of America; Center for Microbial Genetics & Genomics, Northern Arizona University, Flagstaff, AZ, United States of America.
    Engelthaler DM; Translational Genomics Research Institute, Flagstaff, AZ, United States of America.
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    Źródło:
    PloS one [PLoS One] 2015 May 13; Vol. 10 (5), pp. e0126626. Date of Electronic Publication: 2015 May 13 (Print Publication: 2015).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Antitubercular Agents/*pharmacology
    Drug Resistance, Multiple, Bacterial/*genetics
    Mycobacterium tuberculosis/*genetics
    Tuberculosis, Multidrug-Resistant/*microbiology
    Tuberculosis, Pulmonary/*microbiology
    Antitubercular Agents/therapeutic use ; DNA, Bacterial/genetics ; DNA, Bacterial/isolation & purification ; Gene Frequency ; Genetic Loci ; High-Throughput Nucleotide Sequencing ; Humans ; Microbial Sensitivity Tests ; Molecular Diagnostic Techniques ; Polymorphism, Single Nucleotide ; Sequence Analysis, DNA ; Sputum/microbiology ; Tuberculosis, Multidrug-Resistant/diagnosis ; Tuberculosis, Multidrug-Resistant/drug therapy ; Tuberculosis, Pulmonary/diagnosis ; Tuberculosis, Pulmonary/drug therapy
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-8 z 8

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