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Wyszukujesz frazę ""Huang, Jian"" wg kryterium: Autor


Tytuł:
LncRNA SNHG1 alleviated apoptosis and inflammation during ischemic stroke by targeting miR-376a and modulating CBS/H 2 S pathway.
Autorzy:
Lv L; Department of Neurosurgery, First Affiliated Hospital, University of South China, Hengyang, P.R. China.
Xi HP; Department of Neurosurgery, First Affiliated Hospital, University of South China, Hengyang, P.R. China.
Huang JC; Department of Neurosurgery, First Affiliated Hospital, University of South China, Hengyang, P.R. China.
Zhou XY; Department of Neurosurgery, First Affiliated Hospital, University of South China, Hengyang, P.R. China.
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Źródło:
The International journal of neuroscience [Int J Neurosci] 2021 Dec; Vol. 131 (12), pp. 1162-1172. Date of Electronic Publication: 2020 Jul 07.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*physiology
Cystathionine beta-Synthase/*metabolism
Cytokines/*metabolism
Hydrogen Sulfide/*metabolism
Inflammation/*metabolism
Ischemic Stroke/*metabolism
MicroRNAs/*metabolism
RNA, Long Noncoding/*metabolism
Cells, Cultured ; Down-Regulation ; Humans ; Hypoglycemia/metabolism ; Hypoxia/metabolism ; Signal Transduction/physiology
Czasopismo naukowe
Tytuł:
Nox4 has a crucial role in uric acid‑induced oxidative stress and apoptosis in renal tubular cells.
Autorzy:
Li Z; Department of Urology, Sun Yat‑sen Memorial Hospital, Sun Yat‑sen University, Guangzhou, Guangdong 510120, P.R. China.
Sheng Y; Department of Urology, Sun Yat‑sen Memorial Hospital, Sun Yat‑sen University, Guangzhou, Guangdong 510120, P.R. China.
Liu C; Department of Urology, Sun Yat‑sen Memorial Hospital, Sun Yat‑sen University, Guangzhou, Guangdong 510120, P.R. China.
Li K; Department of Urology, Sun Yat‑sen Memorial Hospital, Sun Yat‑sen University, Guangzhou, Guangdong 510120, P.R. China.
Huang X; Department of Urology, Sun Yat‑sen Memorial Hospital, Sun Yat‑sen University, Guangzhou, Guangdong 510120, P.R. China.
Huang J; Department of Urology, Sun Yat‑sen Memorial Hospital, Sun Yat‑sen University, Guangzhou, Guangdong 510120, P.R. China.
Xu K; Department of Urology, Sun Yat‑sen Memorial Hospital, Sun Yat‑sen University, Guangzhou, Guangdong 510120, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2016 May; Vol. 13 (5), pp. 4343-8. Date of Electronic Publication: 2016 Apr 04.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Gene Expression Regulation, Enzymologic/*drug effects
Kidney Tubules/*enzymology
NADPH Oxidases/*metabolism
Oxidative Stress/*drug effects
Uric Acid/*pharmacology
Cell Line ; Humans ; NADPH Oxidase 4 ; Phosphorylation/drug effects ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Reactive Oxygen Species/metabolism ; Up-Regulation/drug effects ; bcl-2-Associated X Protein/metabolism ; p38 Mitogen-Activated Protein Kinases/metabolism
Czasopismo naukowe
Tytuł:
Apoptotic and autophagic pathways with relevant small-molecule compounds, in cancer stem cells.
Autorzy:
Zhang L; State Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy, West China Hospital, Sichuan University, Chengdu, 610041, China.; School of Traditional Chinese Materia Medica, Shenyang Pharmaceutical University, Shenyang, 110016, China.
Tong X; State Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy, West China Hospital, Sichuan University, Chengdu, 610041, China.; School of Pharmacy, China Pharmaceutical University, Nanjing, 211198, China.
Li J; State Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy, West China Hospital, Sichuan University, Chengdu, 610041, China.
Huang Y; State Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy, West China Hospital, Sichuan University, Chengdu, 610041, China.
Hu X; State Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy, West China Hospital, Sichuan University, Chengdu, 610041, China.
Chen Y; Department of Gastrointestinal Surgery, West China Hospital, Sichuan University, Chengdu, 610041, China.
Huang J; School of Traditional Chinese Materia Medica, Shenyang Pharmaceutical University, Shenyang, 110016, China.
Wang J; School of Traditional Chinese Materia Medica, Shenyang Pharmaceutical University, Shenyang, 110016, China.
Liu B; State Key Laboratory of Biotherapy/Collaborative Innovation Center of Biotherapy, West China Hospital, Sichuan University, Chengdu, 610041, China.
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Źródło:
Cell proliferation [Cell Prolif] 2015 Aug; Vol. 48 (4), pp. 385-97. Date of Electronic Publication: 2015 May 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Autophagy/*drug effects
Neoplasms/*drug therapy
Neoplastic Stem Cells/*drug effects
Neoplastic Stem Cells/*metabolism
Signal Transduction/*drug effects
Animals ; Antineoplastic Agents/chemistry ; Humans ; Molecular Targeted Therapy ; Neoplasms/metabolism ; Neoplasms/pathology ; Neoplastic Stem Cells/pathology
Czasopismo naukowe
Tytuł:
Antitumor activity of CDA-Ⅱ, a urinary preparation, on human multiple myeloma cell lines via the mitochondrial pathway.
Autorzy:
Yang M; Department of Hematology, Institute of Hematology, The First Affiliated Hospital of Zhejiang University, School of Medicine, Hangzhou, Zhejiang, P.R. China.
Huang J; Department of Hematology, Institute of Hematology, The First Affiliated Hospital of Zhejiang University, School of Medicine, Hangzhou, Zhejiang, P.R. China.
Ma QL; Department of Hematology, Institute of Hematology, The First Affiliated Hospital of Zhejiang University, School of Medicine, Hangzhou, Zhejiang, P.R. China.
Xu GX; Department of Hematology, Institute of Hematology, The First Affiliated Hospital of Zhejiang University, School of Medicine, Hangzhou, Zhejiang, P.R. China.
Jin J; Department of Hematology, Institute of Hematology, The First Affiliated Hospital of Zhejiang University, School of Medicine, Hangzhou, Zhejiang, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2014 Mar; Vol. 9 (3), pp. 1025-31. Date of Electronic Publication: 2014 Jan 22.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Mitochondria/*drug effects
Multiple Myeloma/*pathology
Peptides/*pharmacology
Phenylacetates/*pharmacology
Antineoplastic Agents/isolation & purification ; Antineoplastic Agents/urine ; Caspase 3/chemistry ; Caspase 3/metabolism ; Caspase 9/metabolism ; Caspase Inhibitors/pharmacology ; Cell Line, Tumor ; Humans ; Interleukin-6/metabolism ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/metabolism ; Multiple Myeloma/metabolism ; Myeloid Cell Leukemia Sequence 1 Protein/metabolism ; NF-kappa B/antagonists & inhibitors ; NF-kappa B/metabolism ; Oligopeptides/pharmacology ; Peptides/isolation & purification ; Peptides/urine ; Phenylacetates/isolation & purification ; Phenylacetates/urine ; Poly(ADP-ribose) Polymerases/metabolism ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Transcription Factor RelA/metabolism ; X-Linked Inhibitor of Apoptosis Protein/metabolism ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł:
Grape seed proanthocyanidins inhibit H2O2-induced osteoblastic MC3T3-E1 cell apoptosis via ameliorating H2O2-induced mitochondrial dysfunction.
Autorzy:
Zhang Z; Department of Orthopaedics, the Second Affiliated Hospital of Inner Mongolia Medical University, China.
Zheng L
Zhao Z
Shi J
Wang X
Huang J
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Źródło:
The Journal of toxicological sciences [J Toxicol Sci] 2014; Vol. 39 (5), pp. 803-13.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Grape Seed Extract/*pharmacology
Hydrogen Peroxide/*toxicity
Mitochondria/*metabolism
Osteoblasts/*pathology
Oxidative Stress/*drug effects
Proanthocyanidins/*pharmacology
Animals ; Bone Resorption/etiology ; Cells, Cultured ; Electron Transport Complex IV/metabolism ; Free Radicals/metabolism ; Grape Seed Extract/therapeutic use ; Membrane Potential, Mitochondrial/drug effects ; Mice ; Osteoporosis/drug therapy ; Proanthocyanidins/therapeutic use
Czasopismo naukowe
Tytuł:
Suppression of the long non-coding RNA LINC01279 triggers autophagy and apoptosis in lung cancer by regulating FAK and SIN3A.
Autorzy:
Wu, Jiancong
Huang, Xiaobi
Li, Xiaofang
Zhou, Honglian
Chen, Xiaorao
Chen, Yongyang
Guo, Yudong
Huang, Jian
Huang, Hanqing
Huang, Zhong
Chen, Guoan
Yang, Zhixiong
Zhang, Jian
Su, Wenmei
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Temat:
LINCRNA
LUNG cancer
FOCAL adhesion kinase
NON-small-cell lung carcinoma
AUTOPHAGY
CANCER cell differentiation
Źródło:
Discover Oncology; 1/2/2024, Vol. 15 Issue 1, p1-16, 16p
Czasopismo naukowe

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