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Wyszukujesz frazę ""INTERLEUKIN-21"" wg kryterium: Temat

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Wyświetlanie 1-20 z 1973
Tytuł:
Expression of Interleukin-21 and Interleukin-21 receptor in lymphocytes derived from tumor-draining lymph nodes of breast cancer.
Autorzy:
Balouchi-Anaraki S; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Mohammadsadeghi S; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Norouzian M; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.; Department of Laboratory Sciences, School of Allied Medical Sciences, Hormozgan University of Medical Sciences, Bandar Abbas, Iran.
Rasolmali R; Department of Pathology, Shiraz Central Hospital, Shiraz, Iran.
Talei AR; Breast Diseases Research Center, Shiraz University of Medical Sciences, Shiraz, Iran.
Mehdipour F; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Ghaderi A; Shiraz Institute for Cancer Research, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.; Department of Immunology, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
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Źródło:
Breast disease [Breast Dis] 2022; Vol. 41 (1), pp. 373-382.
Typ publikacji:
Journal Article
MeSH Terms:
Breast Neoplasms*/pathology
Interleukin-21 Receptor alpha Subunit/*immunology
CD4-Positive T-Lymphocytes ; Female ; Humans ; Interleukins ; Lymph Nodes/pathology ; Receptors, Interleukin-21/genetics ; Receptors, Interleukin-21/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Altered Circulating Follicular T Helper Cell Subsets and Follicular T Regulatory Cells Are Indicators of a Derailed B Cell Response in Lupus, Which Could Be Modified by Targeting IL-21R.
Autorzy:
Szabó K; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Jámbor I; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Pázmándi K; Department of Immunology, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Nagy N; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Papp G; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
Tarr T; Division of Clinical Immunology, Institute of Internal Medicine, Faculty of Medicine, University of Debrecen, H-4032 Debrecen, Hungary.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Oct 13; Vol. 23 (20). Date of Electronic Publication: 2022 Oct 13.
Typ publikacji:
Journal Article
MeSH Terms:
Receptors, Interleukin-21*/metabolism
Lupus Erythematosus, Systemic*
Humans ; T-Lymphocytes, Helper-Inducer ; T-Lymphocytes, Regulatory ; Autoantibodies/metabolism ; Antibodies, Monoclonal/metabolism ; Complement C4/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin-21 Influences Glioblastoma Course: Biological Mechanisms and Therapeutic Potential.
Autorzy:
Repici, Alberto (AUTHOR)
Ardizzone, Alessio (AUTHOR)
Filippone, Alessia (AUTHOR)
Colarossi, Cristina (AUTHOR)
Mare, Marzia (AUTHOR)
Raciti, Gabriele (AUTHOR)
Mannino, Deborah (AUTHOR)
Cuzzocrea, Salvatore (AUTHOR)
Paterniti, Irene (AUTHOR)
Esposito, Emanuela (AUTHOR)
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Źródło:
Cells (2073-4409). Sep2023, Vol. 12 Issue 18, p2284. 17p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin 21 Receptor/Ligand Interaction Is Linked to Disease Progression in Pancreatic Cancer.
Autorzy:
Linnebacher A; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Mayer P; Clinic for Diagnostic and Interventional Radiology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Marnet N; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Bergmann F; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany.; Clinical Pathology, Klinikum Darmstadt GmbH, 64283 Darmstadt, Germany.
Herpel E; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Revia S; Helmholtz-University Group 'Cell Plasticity and Epigenetic Remodeling', German Cancer Research Center (DKFZ) and Institute of Pathology, University Hospital, 69120 Heidelberg, Germany.
Yin L; Molecular OncoSurgery Group, Department of General, Visceral, and Transplantation Surgery, University of Heidelberg and German Cancer Research Center, 69120 Heidelberg, Germany.
Liu L; Molecular OncoSurgery Group, Department of General, Visceral, and Transplantation Surgery, University of Heidelberg and German Cancer Research Center, 69120 Heidelberg, Germany.
Hackert T; Department of General, Visceral, and Transplantation Surgery, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Giese T; Institute of Immunology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
Herr I; Molecular OncoSurgery Group, Department of General, Visceral, and Transplantation Surgery, University of Heidelberg and German Cancer Research Center, 69120 Heidelberg, Germany.
Gaida MM; Institute of Pathology, University Hospital Heidelberg, 69120 Heidelberg, Germany. .; Institute of Pathology, University Medical Center Mainz, 55131 Mainz, Germany. .
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Źródło:
Cells [Cells] 2019 Sep 18; Vol. 8 (9). Date of Electronic Publication: 2019 Sep 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Disease Progression*
Carcinoma, Pancreatic Ductal/*metabolism
Pancreatic Neoplasms/*metabolism
Receptors, Interleukin-21/*metabolism
Animals ; Carcinoma, Pancreatic Ductal/pathology ; Cell Proliferation ; Chickens ; Humans ; Interleukins/metabolism ; Ligands ; Pancreatic Neoplasms/pathology ; Receptors, Interleukin-21/genetics ; Tumor Cells, Cultured
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interleukin-21 receptor signalling is not critically required for imiquimod-induced psoriasiform dermatitis in mice.
Autorzy:
Kim HJ; Department of dermatology, Gachon University Gil Medical Center, Incheon, Korea.
Kim SH; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Kim TG; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Park JY; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Lee M; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Kim DS; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
Lee MG; Department of dermatology, Severance hospital, Cutaneous Biology Research Institute and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea.
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Źródło:
Experimental dermatology [Exp Dermatol] 2018 Feb; Vol. 27 (2), pp. 191-195. Date of Electronic Publication: 2018 Jan 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Dermatitis/*genetics
Interleukin-21 Receptor alpha Subunit/*metabolism
Psoriasis/*genetics
Receptors, Interleukin-21/*metabolism
Animals ; CD4-Positive T-Lymphocytes/cytology ; Dermatitis/metabolism ; Imiquimod ; Inflammation ; Interferon Inducers/pharmacology ; Interleukin-21 Receptor alpha Subunit/genetics ; Interleukin-23 Subunit p19/metabolism ; Intraepithelial Lymphocytes/cytology ; Mice ; Mice, Knockout ; Mice, Transgenic ; Psoriasis/chemically induced ; Psoriasis/metabolism ; Receptors, Interleukin-21/genetics ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
IL-21/IL-21R Regulates the Neutrophil-Mediated Pathologic Immune Response during Chlamydial Respiratory Infection.
Autorzy:
Zeng J; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Xu Y; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Tan L; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Zha X; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Yang S; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Zhang H; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Tuo Y; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Sun R; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Niu W; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Pang G; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Sun L; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
Bai H; Department of Immunology, Key Laboratory of Immune Microenvironment and Disease of the Educational Ministry of China, Tianjin Key Laboratory of Cellular and Molecular Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin, China.
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Źródło:
Mediators of inflammation [Mediators Inflamm] 2022 Jun 01; Vol. 2022, pp. 4322092. Date of Electronic Publication: 2022 Jun 01 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Chlamydia Infections*
Chlamydia muridarum*
Interleukin-21 Receptor alpha Subunit/*metabolism
Animals ; Immunity ; Inflammation/pathology ; Interleukins ; Lung/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Myeloid Differentiation Factor 88/metabolism ; Neutrophils/metabolism ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Combined immunodeficiency with marginal zone lymphoma due to a novel homozygous mutation in IL-21R gene and successful treatment with hematopoietic stem cell transplantation.
Autorzy:
Edeer Karaca N; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
Özek G; Faculty of Medicine, Department of Pediatric Hematology and Oncology, Ege University, İzmir, Turkey.
Ataseven E; Faculty of Medicine, Department of Pediatric Hematology and Oncology, Ege University, İzmir, Turkey.
Tökmeci N; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
Şenol HD; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
Kıran E; Faculty of Medicine, Department of Pediatric Gastroenterology Gastroenterology, Hepatology and Nutrition, Ege University, İzmir, Turkey.
Aykut A; Faculty of Medicine, Department of Medical Genetics, Ege University, İzmir, Turkey.
Durmaz A; Faculty of Medicine, Department of Medical Genetics, Ege University, İzmir, Turkey.
Aksu G; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
Aksoylar S; Faculty of Medicine, Department of Pediatric Hematology and Oncology, Ege University, İzmir, Turkey.
Aydoğdu S; Faculty of Medicine, Department of Pediatric Gastroenterology Gastroenterology, Hepatology and Nutrition, Ege University, İzmir, Turkey.
Çetingül N; Faculty of Medicine, Department of Pediatric Hematology and Oncology, Ege University, İzmir, Turkey.
Kütükçüler N; Faculty of Medicine, Department of Pediatric Immunology and Allergy, Ege University, İzmir, Turkey.
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Źródło:
Pediatric hematology and oncology [Pediatr Hematol Oncol] 2021 Nov; Vol. 38 (8), pp. 745-752. Date of Electronic Publication: 2021 May 08.
Typ publikacji:
Case Reports; Journal Article
MeSH Terms:
Hematopoietic Stem Cell Transplantation*
Lymphoma*/genetics
Lymphoma*/therapy
Primary Immunodeficiency Diseases*/complications
Primary Immunodeficiency Diseases*/genetics
Primary Immunodeficiency Diseases*/therapy
Child ; Cryptosporidiosis ; Cytomegalovirus Infections ; Diarrhea/etiology ; Diarrhea/therapy ; Exanthema/etiology ; Exanthema/therapy ; Female ; Humans ; Mutation ; Persistent Infection ; Pneumonia, Viral ; Receptors, Interleukin-21/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Morphology of the immune cells in the wall of the human uterine tube and their possible impact on reproduction—uterine tube as a possible immune privileged organ.
Autorzy:
Visnyaiová, Kristína
Varga, Ivan
Feitscherová, Claudia
Pavlíková, Lada
Záhumenský, Jozef
Mikušová, Renáta
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Temat:
FALLOPIAN tubes
CELL morphology
KILLER cells
IMMUNOCOMPETENT cells
SCIENTIFIC literature
INTERLEUKIN-21
Źródło:
Frontiers in Cell & Developmental Biology; 2024, p1-9, 9p
Czasopismo naukowe
Szczegóły
Tytuł:
Anti-CD20 treatment attenuates Th2 cell responses: implications for the role of lung follicular mature B cells in the asthmatic mice.
Autorzy:
He, Jilong
Li, Jingling
Lin, Qibin
Ni, Haiyang
Huang, Sisi
Cheng, Hong
Ding, Xuhong
Huang, Yi
Yu, Hongying
Xu, Yaqing
Nie, Hanxiang
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Temat:
B cells
TH2 cells
KILLER cells
REGULATORY T cells
INTERLEUKIN-21
HOUSE dust mites
Źródło:
Inflammation Research; Mar2024, Vol. 73 Issue 3, p433-446, 14p
Czasopismo naukowe
Szczegóły
Tytuł:
Metabolic alterations of peripheral blood immune cells and heterogeneity of neutrophil in intracranial aneurysms patients.
Autorzy:
Ya, Xiaolong
Ma, Long
Liu, Chenglong
Ge, Peicong
Xu, Yiqiao
Zheng, Zhiyao
Mou, Siqi
Wang, Rong
Zhang, Qian
Ye, Xun
Zhang, Dong
Zhang, Yan
Wang, Wenjing
Li, Hao
Zhao, Jizong
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Temat:
INTRACRANIAL aneurysms
MONONUCLEAR leukocytes
BLOOD cells
CEREBROVASCULAR disease
NEUTROPHILS
DISEASE progression
INTERLEUKIN-21
Źródło:
Clinical & Translational Medicine; Feb2024, Vol. 14 Issue 2, p1-23, 23p
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Myricetin ameliorates the IL-21-induced tumorigenic phenotype of adjuvant-induced arthritis FLS by modulating the choline kinase signaling cascade
Autorzy:
Jose, Ann Miriam
Rasool, MahaboobkhanAff1, IDs11626023008276_cor2
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Źródło:
In Vitro Cellular & Developmental Biology - Animal. 59(10):811-820
Czasopismo naukowe
Szczegóły
Wyświetlanie 1-20 z 1973

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