Temat: "Inflammasomes" - OpacWWW

Skocz do pozycji: 1.

Tytuł:: The interaction of inflammasomes and gut microbiota: novel therapeutic insights.
Autorzy:: Manshouri S; Rajaei Cardiovascular Medical and Research Center, Iran University of Medical Sciences, Valiasr St, Niayesh Intersection, Tehran, 1995614331, Iran.
Seif F; Department of Photodynamic Therapy, Medical Laser Research Center, Academic Center for Education, Culture, and Research (ACECR), Tehran, Iran.; Department of Immunology and Allergy, Academic Center for Education, Culture, and Research (ACECR), Tehran, Iran.
Kamali M; Rajaei Cardiovascular Medical and Research Center, Iran University of Medical Sciences, Valiasr St, Niayesh Intersection, Tehran, 1995614331, Iran.
Bahar MA; Department of Immunology, Medical School, Iran University of Medical Sciences, Tehran, Iran.
Mashayekh A; Rajaei Cardiovascular Medical and Research Center, Iran University of Medical Sciences, Valiasr St, Niayesh Intersection, Tehran, 1995614331, Iran. .
Molatefi R; Cancer Immunology and Immunotherapy Research Center, Ardabil University of Medical Sciences, Ardabil, Iran. .; Pediatric Department of Bou Ali Hospital, Ardabil University of Medical Sciences, Ardabil, 56189-85991, Iran. .; Pokaż więcej
Źródło:: Cell communication and signaling : CCS [Cell Commun Signal] 2024 Apr 02; Vol. 22 (1), pp. 209. Date of Electronic Publication: 2024 Apr 02.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Inflammasomes*/metabolism
Gastrointestinal Microbiome*
Humans ; Cytokines/metabolism

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Skocz do pozycji: 2.

Tytuł:: Bif‑1 inhibits activation of inflammasome through autophagy regulatory mechanism.
Autorzy:: Zhang Y; Department of Critical Care Medicine, The Second Affiliated Hospital of Shenzhen University, Shenzhen, Guangdong 518101, P.R. China.
Su W; State Key Laboratory of Chemical Oncogenomics, Institute of Biopharmaceutical and Health Engineering, Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong 518055, P.R. China.
Niu Y; State Key Laboratory of Chemical Oncogenomics, Institute of Biopharmaceutical and Health Engineering, Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong 518055, P.R. China.
Zeng H; State Key Laboratory of Chemical Oncogenomics, Institute of Biopharmaceutical and Health Engineering, Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong 518055, P.R. China.
Liu L; Department of Critical Care Medicine, The Second Affiliated Hospital of Shenzhen University, Shenzhen, Guangdong 518101, P.R. China.
Wang L; Department of Critical Care Medicine, The Second Affiliated Hospital of Shenzhen University, Shenzhen, Guangdong 518101, P.R. China.
Xie W; State Key Laboratory of Chemical Oncogenomics, Institute of Biopharmaceutical and Health Engineering, Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong 518055, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2024 Apr; Vol. 29 (4). Date of Electronic Publication: 2024 Mar 08.
Typ publikacji:: Journal Article
MeSH Terms:: Inflammasomes*/metabolism
NLR Family, Pyrin Domain-Containing 3 Protein*/genetics
NLR Family, Pyrin Domain-Containing 3 Protein*/metabolism
NF-kappa B/metabolism ; Lipopolysaccharides/pharmacology ; Autophagy/genetics ; Anti-Inflammatory Agents/pharmacology ; Adenosine Triphosphate/pharmacology

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Skocz do pozycji: 3.

Tytuł:: The contribution of pattern recognition receptor signalling in the development of age related macular degeneration: the role of toll-like-receptors and the NLRP3-inflammasome.
Autorzy:: Brandli A; Department of Anatomy and Physiology, The University of Melbourne, Grattan St, Parkville, Victoria, 3010, Australia.; Roche Pharma Research and Early Development, F. Hoffmann-La Roche Ltd, Basel, Switzerland.
Vessey KA; Department of Anatomy and Physiology, The University of Melbourne, Grattan St, Parkville, Victoria, 3010, Australia.
Fletcher EL; Department of Anatomy and Physiology, The University of Melbourne, Grattan St, Parkville, Victoria, 3010, Australia. .; Pokaż więcej
Źródło:: Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 05; Vol. 21 (1), pp. 64. Date of Electronic Publication: 2024 Mar 05.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Inflammasomes*
Macular Degeneration*
Animals ; NLR Family, Pyrin Domain-Containing 3 Protein ; Toll-Like Receptors ; Inflammation

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Skocz do pozycji: 4.

Tytuł:: Dictyophora indusiata polysaccharide mediates priming of the NLRP3 inflammasome activation via TLR4/ NF-κB signaling pathway to exert immunostimulatory effects.
Autorzy:: Liu Y; Jiangnan University, Wuxi School of Medicine, Wuxi, Jiangsu 214122, P.R. China.
Zhang H; Jiangnan University, Wuxi School of Medicine, Wuxi, Jiangsu 214122, P.R. China.
Li Y; Jiangnan University, Wuxi School of Medicine, Wuxi, Jiangsu 214122, P.R. China.
Zha H; Jiangnan University, Wuxi School of Medicine, Wuxi, Jiangsu 214122, P.R. China.
Gao Y; Jiangnan University, Wuxi School of Medicine, Wuxi, Jiangsu 214122, P.R. China.
Chen H; Yixing Hospital of Traditional Chinese Medicine, Wuxi, Jiangsu 214200, P.R. China.
Wang Y; Jiangnan University, Wuxi School of Medicine, Wuxi, Jiangsu 214122, P.R. China.
Zhou T; Yixing Hospital of Traditional Chinese Medicine, Wuxi, Jiangsu 214200, P.R. China.
Deng C; Jiangnan University, Wuxi School of Medicine, Wuxi, Jiangsu 214122, P.R. China.; Pokaż więcej
Źródło:: Journal of applied biomedicine [J Appl Biomed] 2024 Mar; Vol. 22 (1), pp. 23-32. Date of Electronic Publication: 2024 Mar 13.
Typ publikacji:: Journal Article
MeSH Terms:: NF-kappa B*/metabolism
Inflammasomes*/metabolism
Basidiomycota*
NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Toll-Like Receptor 4/metabolism ; NLR Proteins ; Signal Transduction ; Polysaccharides/pharmacology
SCR Organism:: Dictyophora indusiata

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Skocz do pozycji: 5.

Tytuł:: Tau induces inflammasome activation and microgliosis through acetylating NLRP3.
Autorzy:: Zhang L; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Department of Clinical Laboratory, Wuhan Fourth Hospital, Wuhan, China.
Gai Y; Department of Nuclear Medicine, Hubei Province Key Laboratory of Molecular Imaging, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Liu Y; Department of Biochemistry and Molecular Biology, School of Basic Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Meng D; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zeng Y; Department of Clinical Laboratory, The Central Hospital of Wuhan, Wuhan, China.
Luo Y; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhang H; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Wang Z; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Yang M; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Li Y; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Liu Y; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Lai Y; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Yang J; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Wu G; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Chen Y; Department of Pediatrics, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhu J; Britton Chance Center for Biomedical Photonics-MoE Key Laboratory for Biomedical Photonics, Advanced Biomedical Imaging Facility, Wuhan National Laboratory for Optoelectronics, Huazhong University of Science and Technology, Wuhan, China.
Liu S; Britton Chance Center for Biomedical Photonics-MoE Key Laboratory for Biomedical Photonics, Advanced Biomedical Imaging Facility, Wuhan National Laboratory for Optoelectronics, Huazhong University of Science and Technology, Wuhan, China.
Yu T; Britton Chance Center for Biomedical Photonics-MoE Key Laboratory for Biomedical Photonics, Advanced Biomedical Imaging Facility, Wuhan National Laboratory for Optoelectronics, Huazhong University of Science and Technology, Wuhan, China.
Zeng J; Department of Clinical Laboratory, Wuhan Fourth Hospital, Wuhan, China.
Wang J; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhu D; Britton Chance Center for Biomedical Photonics-MoE Key Laboratory for Biomedical Photonics, Advanced Biomedical Imaging Facility, Wuhan National Laboratory for Optoelectronics, Huazhong University of Science and Technology, Wuhan, China.
Wang X; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Shenzhen Huazhong University of Science and Technology Research Institute, Shenzhen, China.
Lan X; Department of Nuclear Medicine, Hubei Province Key Laboratory of Molecular Imaging, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Liu R; Department of Pathophysiology, School of Basic Medicine, Key Laboratory of Education Ministry of China/Hubei Province for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Department of Pediatrics, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.; Shenzhen Huazhong University of Science and Technology Research Institute, Shenzhen, China.; Pokaż więcej
Źródło:: Clinical and translational medicine [Clin Transl Med] 2024 Mar; Vol. 14 (3), pp. e1623.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Inflammasomes*/metabolism
Alzheimer Disease*/genetics
Alzheimer Disease*/pathology
Humans ; Mice ; Animals ; NLR Family, Pyrin Domain-Containing 3 Protein/genetics ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; tau Proteins/genetics ; tau Proteins/metabolism ; HEK293 Cells ; Molecular Docking Simulation ; Mice, Transgenic ; Acetyltransferases

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Skocz do pozycji: 6.

Tytuł:: Unraveling the Role of the NLRP3 Inflammasome in Lymphoma: Implications in Pathogenesis and Therapeutic Strategies.
Autorzy:: Stergiou IE; Department of Pathophysiology, School of Medicine, National and Kapodistrian University of Athens, 11527 Athens, Greece.
Tsironis C; Department of Pathophysiology, School of Medicine, National and Kapodistrian University of Athens, 11527 Athens, Greece.
Papadakos SP; First Department of Pathology, School of Medicine, National and Kapodistrian University of Athens, 10679 Athens, Greece.
Tsitsilonis OE; Flow Cytometry Unit, Department of Biology, School of Science, National and Kapodistrian University of Athens, 15784 Athens, Greece.
Dimopoulos MA; Department of Clinical Therapeutics, School of Medicine, National and Kapodistrian University of Athens, Alexandra Hospital, 11528 Athens, Greece.
Theocharis S; First Department of Pathology, School of Medicine, National and Kapodistrian University of Athens, 10679 Athens, Greece.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2024 Feb 17; Vol. 25 (4). Date of Electronic Publication: 2024 Feb 17.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Inflammasomes*/metabolism
Lymphoma*/etiology
Lymphoma*/therapy
Humans ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Inflammation/metabolism ; Tumor Microenvironment

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Skocz do pozycji: 7.

Tytuł:: The BK Channel Limits the Pro-Inflammatory Activity of Macrophages.
Autorzy:: Chen Y; Institute of Pharmacology, University of Bern, 3010 Bern, Switzerland.
Markov N; Institute of Pharmacology, University of Bern, 3010 Bern, Switzerland.
Gigon L; Institute of Pharmacology, University of Bern, 3010 Bern, Switzerland.
Hosseini A; Institute of Pharmacology, University of Bern, 3010 Bern, Switzerland.
Yousefi S; Institute of Pharmacology, University of Bern, 3010 Bern, Switzerland.
Stojkov D; Institute of Pharmacology, University of Bern, 3010 Bern, Switzerland.
Simon HU; Institute of Pharmacology, University of Bern, 3010 Bern, Switzerland.; Institute of Biochemistry, Brandenburg Medical School, 16816 Neuruppin, Germany.; Pokaż więcej
Źródło:: Cells [Cells] 2024 Feb 09; Vol. 13 (4). Date of Electronic Publication: 2024 Feb 09.
Typ publikacji:: Journal Article
MeSH Terms:: Inflammasomes*/metabolism
Large-Conductance Calcium-Activated Potassium Channels*/metabolism
Calcium/metabolism ; Macrophages/metabolism ; Immunity, Innate ; Calcium-Calmodulin-Dependent Protein Kinases/metabolism

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Skocz do pozycji: 8.

Tytuł:: Poly-(ADP-ribose) polymerases inhibition by olaparib attenuates activities of the NLRP3 inflammasome and of NF-κB in THP-1 monocytes.
Autorzy:: Mustafa K; Institute of Heart and Vessel Diseases, The Second Affiliated Hospital, Dalian Medical University, Dalian, China.
Han Y; Department of Cardiology, Jinqiu Hospital of Liaoning Province, Shenyang, China.
He D; Institute of Heart and Vessel Diseases, The Second Affiliated Hospital, Dalian Medical University, Dalian, China.
Wang Y; Institute of Heart and Vessel Diseases, The Second Affiliated Hospital, Dalian Medical University, Dalian, China.
Niu N; Department of Cardiology, The Second Affiliated Hospital, Dalian Medical University, Dalian, China.
Jose PA; Department of Medicine, The George Washington University School of Medicine & Health Sciences, Washington, District of Columbia, United States of America.; Department of Physiology/Pharmacology, The George Washington University School of Medicine & Health Sciences, Washington, District of Columbia, United States of America.
Jiang Y; Department of Cardiology, The First Affiliated Hospital, Dalian Medical University, Dalian, China.
Kopp JB; Kidney Disease Section, Kidney Diseases Branch, National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Bethesda, Maryland, United States of America.
Lee H; Institute of Heart and Vessel Diseases, The Second Affiliated Hospital, Dalian Medical University, Dalian, China.; Department of Medicine, The George Washington University School of Medicine & Health Sciences, Washington, District of Columbia, United States of America.; Kidney Disease Section, Kidney Diseases Branch, National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Bethesda, Maryland, United States of America.
Qu P; Institute of Heart and Vessel Diseases, The Second Affiliated Hospital, Dalian Medical University, Dalian, China.; Department of Cardiology, The Second Affiliated Hospital, Dalian Medical University, Dalian, China.; Faculty of Medicine, Dalian University of Technology, Dalian, China.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2024 Feb 09; Vol. 19 (2), pp. e0295837. Date of Electronic Publication: 2024 Feb 09 (Print Publication: 2024).
Typ publikacji:: Journal Article
MeSH Terms:: Inflammasomes*/metabolism
Atherosclerosis*/metabolism
Phthalazines*
Piperazines*
Humans ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Monocytes/metabolism ; NF-kappa B/metabolism ; NF-KappaB Inhibitor alpha/metabolism ; Poly(ADP-ribose) Polymerases/metabolism ; Poly(ADP-ribose) Polymerase Inhibitors/pharmacology ; Endothelial Cells/metabolism ; Adenosine Diphosphate Ribose/metabolism ; Interleukin-1beta/metabolism

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Skocz do pozycji: 9.

Tytuł:: FoxO6-Mediated TXNIP Induces Lipid Accumulation in the Liver through NLRP3 Inflammasome Activation.
Autorzy:: Kim ME; Department of Life Sciences, Chosun University College of Natural Science, Gwangju, Korea.
Lee JS; Department of Life Sciences, Chosun University College of Natural Science, Gwangju, Korea.
Kim TW; Department of Pharmacy, Kyungsung University College of Pharmacy, Busan, Korea.
Park MH; Department of Pharmacy, Kyungsung University College of Pharmacy, Busan, Korea.
Kim DH; Department of Food Science & Technology, Pusan National University College of Natural Resources and Life Science, Miryang, Korea.; Pokaż więcej
Źródło:: Endocrinology and metabolism (Seoul, Korea) [Endocrinol Metab (Seoul)] 2024 Feb; Vol. 39 (1), pp. 127-139. Date of Electronic Publication: 2024 Feb 22.
Typ publikacji:: Journal Article
MeSH Terms:: Inflammasomes*/metabolism
NLR Family, Pyrin Domain-Containing 3 Protein*/genetics
NLR Family, Pyrin Domain-Containing 3 Protein*/metabolism
Animals ; Mice ; Inflammation/metabolism ; Liver/metabolism ; Palmitates

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Skocz do pozycji: 10.

Tytuł:: NR3C2 mediates oxidised low-density lipoprotein-induced human coronary endothelial cells dysfunction via modulation of NLRP3 inflammasome activation.
Autorzy:: Chen X; Department of Cardiovascular Surgery, First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang, China.
Li W; Department of Cardiovascular Surgery, First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang, China.
Chang C; Department of Pathology, First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang, China.; Pokaż więcej
Źródło:: Autoimmunity [Autoimmunity] 2023 Dec; Vol. 56 (1), pp. 2189135.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Inflammasomes*/genetics
Inflammasomes*/metabolism
MicroRNAs*
Humans ; Apoptosis/genetics ; Endothelial Cells/metabolism ; Lipoproteins, LDL/pharmacology ; Lipoproteins, LDL/metabolism ; NLR Family, Pyrin Domain-Containing 3 Protein/genetics ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Receptors, Mineralocorticoid

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Skocz do pozycji: 11.

Tytuł:: NLRP3 inflammasome-a likely target for the treatment of immunologic conjunctivitis: A protocol for systematic review and meta-analysis.
Autorzy:: Liu R; Heilongjiang University of Chinese Medicine, Harbin, Heilongjiang, China.
Fang Y; Heilongjiang Mingshui Kangying Hospital, Suihua, Heilongjiang, China.
Yang F; The EYE Hospital of Daqing, Daqing, Heilongjiang, China.
Liu D; Senhai Hospital, Harbin, Heilongjiang, China.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2024 Jan 26; Vol. 19 (1), pp. e0296994. Date of Electronic Publication: 2024 Jan 26 (Print Publication: 2024).
Typ publikacji:: Journal Article
MeSH Terms:: Conjunctivitis*
Inflammasomes*
Animals ; Humans ; Meta-Analysis as Topic ; NLR Family, Pyrin Domain-Containing 3 Protein ; Randomized Controlled Trials as Topic ; Research Design ; Systematic Reviews as Topic

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Skocz do pozycji: 12.

Tytuł:: SARS-CoV-2 S protein activates NLRP3 inflammasome and deregulates coagulation factors in endothelial and immune cells.
Autorzy:: Villacampa A; Department of Pharmacology, School of Medicine, Universidad Autónoma de Madrid, Madrid, Spain.
Alfaro E; Respiratory Diseases Group, Respiratory Service, La Paz University Hospital, IdiPAZ, Madrid, Spain.; Biomedical Research Networking Center on Respiratory Diseases (CIBERES), Madrid, Spain.
Morales C; Department of Pharmacology, School of Medicine, Universidad Autónoma de Madrid, Madrid, Spain.
Díaz-García E; Respiratory Diseases Group, Respiratory Service, La Paz University Hospital, IdiPAZ, Madrid, Spain.; Biomedical Research Networking Center on Respiratory Diseases (CIBERES), Madrid, Spain.
López-Fernández C; Respiratory Diseases Group, Respiratory Service, La Paz University Hospital, IdiPAZ, Madrid, Spain.
Bartha JL; Department of Obstetrics and Gynecology, School of Medicine, Universidad Autónoma de Madrid, Madrid, Spain.; Gynecology and Obstetrics Service, La Paz University Hospital, Madrid, Spain.
López-Sánchez F; Gynecology and Obstetrics Service, La Paz University Hospital, Madrid, Spain.
Lorenzo Ó; Laboratory of Diabetes and Vascular pathology, IIS-Fundación Jiménez Díaz, Madrid, Spain.; Biomedical Research Networking Centre on Diabetes and Associated Metabolic Disorders (CIBERDEM), Madrid, Spain.; Department of Medicine, School of Medicine, Universidad Autónoma de Madrid, Madrid, Spain.
Moncada S; Department of Pharmacology, School of Medicine, Universidad Autónoma de Madrid, Madrid, Spain.
Sánchez-Ferrer CF; Department of Pharmacology, School of Medicine, Universidad Autónoma de Madrid, Madrid, Spain.; Vascular Pharmacology and Metabolism (FARMAVASM) group, IdiPAZ, Madrid, Spain.
García-Río F; Respiratory Diseases Group, Respiratory Service, La Paz University Hospital, IdiPAZ, Madrid, Spain.; Biomedical Research Networking Center on Respiratory Diseases (CIBERES), Madrid, Spain.; Department of Medicine, School of Medicine, Universidad Autónoma de Madrid, Madrid, Spain.
Cubillos-Zapata C; Respiratory Diseases Group, Respiratory Service, La Paz University Hospital, IdiPAZ, Madrid, Spain. .; Biomedical Research Networking Center on Respiratory Diseases (CIBERES), Madrid, Spain. .
Peiró C; Department of Pharmacology, School of Medicine, Universidad Autónoma de Madrid, Madrid, Spain. .; Vascular Pharmacology and Metabolism (FARMAVASM) group, IdiPAZ, Madrid, Spain. .; Pokaż więcej
Źródło:: Cell communication and signaling : CCS [Cell Commun Signal] 2024 Jan 15; Vol. 22 (1), pp. 38. Date of Electronic Publication: 2024 Jan 15.
Typ publikacji:: Video-Audio Media; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Inflammasomes*/metabolism
COVID-19*
Spike Glycoprotein, Coronavirus*
Humans ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; COVID-19 Vaccines ; NF-kappa B/metabolism ; von Willebrand Factor ; SARS-CoV-2 ; Human Umbilical Vein Endothelial Cells/metabolism ; Interleukin-1beta/metabolism

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Skocz do pozycji: 13.

Tytuł:: cGAS-STING, inflammasomes and pyroptosis: an overview of crosstalk mechanism of activation and regulation.
Autorzy:: Liu J; Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, 610075, China.; TCM Regulating Metabolic Diseases Key Laboratory of Sichuan Province, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, 610075, China.
Zhou J; The Second Hospital of Ningbo, Ningbo, 315099, China.
Luan Y; Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, 201203, China.
Li X; Yueyang Hospital of Integrated Traditional Chinese and Western Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200080, China.
Meng X; Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, 610075, China.
Liao W; Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, 610075, China.
Tang J; Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, 610075, China. .; TCM Regulating Metabolic Diseases Key Laboratory of Sichuan Province, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, 610075, China. .
Wang Z; Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, 610075, China. .; TCM Regulating Metabolic Diseases Key Laboratory of Sichuan Province, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, 610075, China. .; Pokaż więcej
Źródło:: Cell communication and signaling : CCS [Cell Commun Signal] 2024 Jan 09; Vol. 22 (1), pp. 22. Date of Electronic Publication: 2024 Jan 09.
Typ publikacji:: Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:: Inflammasomes*
Pyroptosis*
Nucleotidyltransferases ; Immunity, Innate ; Caspase 1

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Skocz do pozycji: 14.

Tytuł:: Inhibition of the interaction between microglial adenosine 2A receptor and NLRP3 inflammasome attenuates neuroinflammation posttraumatic brain injury.
Autorzy:: Du H; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.; The General Hospital of Tibet Military Command, Tibet, China.
Li CH; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.
Gao RB; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.
Tan Y; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.; Department of Pathophysiology, College of High-Altitude Military Medicine, Army Medical University, Chongqing, China.
Wang B; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.
Peng Y; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.
Yang N; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.
Ning YL; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.; Institute of Brain and Intelligence, Army Medical University, Chongqing, China.
Li P; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.; Institute of Brain and Intelligence, Army Medical University, Chongqing, China.
Zhao Y; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.; Institute of Brain and Intelligence, Army Medical University, Chongqing, China.
Zhou YG; Department of Army Occupational Disease, State Key Laboratory of Trauma and Chemical Poisoning, Research Institute of Surgery and Daping Hospital, Army Medical University, Chongqing, China.; Institute of Brain and Intelligence, Army Medical University, Chongqing, China.; Pokaż więcej
Źródło:: CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Jan; Vol. 30 (1), pp. e14408. Date of Electronic Publication: 2023 Aug 11.
Typ publikacji:: Journal Article
MeSH Terms:: Brain Injuries, Traumatic*/metabolism
Inflammasomes*/metabolism
Animals ; Mice ; Adenosine/metabolism ; Mice, Knockout ; Microglia ; Neuroinflammatory Diseases ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism

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Skocz do pozycji: 15.

Tytuł:: NLRP1- A CINDERELLA STORY: a perspective of recent advances in NLRP1 and the questions they raise.
Autorzy:: Barry K; Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, VIC, Australia.; Department of Molecular and Translational Sciences, Monash University, Clayton, VIC, Australia.
Murphy C; 7 Warren St., Upton, MA, 01568, USA.
Mansell A; Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, VIC, Australia. .; Department of Molecular and Translational Sciences, Monash University, Clayton, VIC, Australia. .; Pokaż więcej
Źródło:: Communications biology [Commun Biol] 2023 Dec 16; Vol. 6 (1), pp. 1274. Date of Electronic Publication: 2023 Dec 16.
Typ publikacji:: Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:: NLR Family, Pyrin Domain-Containing 3 Protein*/genetics
NLR Family, Pyrin Domain-Containing 3 Protein*/metabolism
Inflammasomes*/metabolism
Humans ; Adaptor Proteins, Signal Transducing/genetics ; Adaptor Proteins, Signal Transducing/metabolism ; NLR Proteins/genetics ; NLR Proteins/metabolism ; Apoptosis Regulatory Proteins/genetics ; Apoptosis Regulatory Proteins/metabolism

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Skocz do pozycji: 16.

Tytuł:: Altered Ex Vivo NLRP3 Inflammasome Activation Is Associated with 28-Day Mortality in Septic Patients.
Autorzy:: Coudereau R; Hospices Civils de Lyon, Edouard Herriot Hospital, Immunology Laboratory, 69437 Lyon, France.; EA 7426 'Pathophysiology of Injury-Induced Immunosuppression' (Université Claude Bernard Lyon 1-Hospices Civils de Lyon-bioMérieux), Joint Research Unit HCL-Biomérieux, 69437 Lyon, France.
Monneret G; Hospices Civils de Lyon, Edouard Herriot Hospital, Immunology Laboratory, 69437 Lyon, France.; EA 7426 'Pathophysiology of Injury-Induced Immunosuppression' (Université Claude Bernard Lyon 1-Hospices Civils de Lyon-bioMérieux), Joint Research Unit HCL-Biomérieux, 69437 Lyon, France.
Lukaszewicz AC; EA 7426 'Pathophysiology of Injury-Induced Immunosuppression' (Université Claude Bernard Lyon 1-Hospices Civils de Lyon-bioMérieux), Joint Research Unit HCL-Biomérieux, 69437 Lyon, France.; Hospices Civils de Lyon, Edouard Herriot Hospital, Anesthesia and Critical Care Medicine Department, 69437 Lyon, France.
Py BF; CIRI, Centre International de Recherche en Infectiologie, Univ Lyon, Inserm U1111, Université Claude Bernard-Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.
Argaud L; Hospices Civils de Lyon, Edouard Herriot Hospital, Medical Intensive Care Department, 69002 Lyon, France.
Cour M; Hospices Civils de Lyon, Edouard Herriot Hospital, Medical Intensive Care Department, 69002 Lyon, France.
Bidar F; EA 7426 'Pathophysiology of Injury-Induced Immunosuppression' (Université Claude Bernard Lyon 1-Hospices Civils de Lyon-bioMérieux), Joint Research Unit HCL-Biomérieux, 69437 Lyon, France.; Hospices Civils de Lyon, Edouard Herriot Hospital, Anesthesia and Critical Care Medicine Department, 69437 Lyon, France.
Gossez M; Hospices Civils de Lyon, Edouard Herriot Hospital, Immunology Laboratory, 69437 Lyon, France.; CIRI, Centre International de Recherche en Infectiologie, Univ Lyon, Inserm U1111, Université Claude Bernard-Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.
Venet F; Hospices Civils de Lyon, Edouard Herriot Hospital, Immunology Laboratory, 69437 Lyon, France.; CIRI, Centre International de Recherche en Infectiologie, Univ Lyon, Inserm U1111, Université Claude Bernard-Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.; Pokaż więcej
Źródło:: Viruses [Viruses] 2023 Dec 13; Vol. 15 (12). Date of Electronic Publication: 2023 Dec 13.
Typ publikacji:: Journal Article
MeSH Terms:: Inflammasomes*/metabolism
Sepsis*/mortality
Humans ; Caspase 1/metabolism ; Longitudinal Studies ; Nigericin ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Prospective Studies

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Skocz do pozycji: 17.

Tytuł:: C3a/C3aR synergies with TGF-β to promote epithelial-mesenchymal transition of renal tubular epithelial cells via the activation of the NLRP3 inflammasome.
Autorzy:: You D; Department of Nephrology, Blood Purification Research Center, The First Affiliated Hospital of Fujian Medical University, Chazhong Road 20, Fuzhou, 350005, China.; Fujian Clinical Research Center for Metabolic Chronic Kidney Disease, The First Affiliated Hospital of Fujian Medical University, Fuzhou, 350005, China.; Department of Nephrology, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, 350212, China.
Nie K; Department of Nephrology, Blood Purification Research Center, The First Affiliated Hospital of Fujian Medical University, Chazhong Road 20, Fuzhou, 350005, China.; Fujian Clinical Research Center for Metabolic Chronic Kidney Disease, The First Affiliated Hospital of Fujian Medical University, Fuzhou, 350005, China.; Department of Nephrology, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, 350212, China.
Wu X; Department of Nephrology, Blood Purification Research Center, The First Affiliated Hospital of Fujian Medical University, Chazhong Road 20, Fuzhou, 350005, China.; Fujian Clinical Research Center for Metabolic Chronic Kidney Disease, The First Affiliated Hospital of Fujian Medical University, Fuzhou, 350005, China.; Department of Nephrology, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, 350212, China.
Weng M; Department of Nephrology, Blood Purification Research Center, The First Affiliated Hospital of Fujian Medical University, Chazhong Road 20, Fuzhou, 350005, China.; Fujian Clinical Research Center for Metabolic Chronic Kidney Disease, The First Affiliated Hospital of Fujian Medical University, Fuzhou, 350005, China.; Department of Nephrology, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, 350212, China.
Yang L; Department of Nephrology, Blood Purification Research Center, The First Affiliated Hospital of Fujian Medical University, Chazhong Road 20, Fuzhou, 350005, China.; Fujian Clinical Research Center for Metabolic Chronic Kidney Disease, The First Affiliated Hospital of Fujian Medical University, Fuzhou, 350005, China.; Department of Nephrology, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, 350212, China.
Chen Y; Department of Nephrology, Blood Purification Research Center, The First Affiliated Hospital of Fujian Medical University, Chazhong Road 20, Fuzhou, 350005, China.; Fujian Clinical Research Center for Metabolic Chronic Kidney Disease, The First Affiliated Hospital of Fujian Medical University, Fuzhou, 350005, China.; Department of Nephrology, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, 350212, China.
Cui J; Department of Nephrology, Blood Purification Research Center, The First Affiliated Hospital of Fujian Medical University, Chazhong Road 20, Fuzhou, 350005, China.; Fujian Clinical Research Center for Metabolic Chronic Kidney Disease, The First Affiliated Hospital of Fujian Medical University, Fuzhou, 350005, China.; Department of Nephrology, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, 350212, China.
Wan J; Department of Nephrology, Blood Purification Research Center, The First Affiliated Hospital of Fujian Medical University, Chazhong Road 20, Fuzhou, 350005, China. .; Fujian Clinical Research Center for Metabolic Chronic Kidney Disease, The First Affiliated Hospital of Fujian Medical University, Fuzhou, 350005, China. .; Department of Nephrology, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, 350212, China. .; Pokaż więcej
Źródło:: Journal of translational medicine [J Transl Med] 2023 Dec 11; Vol. 21 (1), pp. 904. Date of Electronic Publication: 2023 Dec 11.
Typ publikacji:: Journal Article
MeSH Terms:: Inflammasomes*/metabolism
NLR Family, Pyrin Domain-Containing 3 Protein*/metabolism
Male ; Animals ; Mice ; Transforming Growth Factor beta ; Mice, Inbred C57BL ; Epithelial-Mesenchymal Transition ; Epithelial Cells/metabolism ; Interleukin-1beta/metabolism ; Fibrosis

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Skocz do pozycji: 18.

Tytuł:: Characterization and inhibition of inflammasome responses in severe and non-severe asthma.
Autorzy:: Horvat JC; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia. .
Kim RY; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.; University of Technology Sydney, Faculty of Science, School of Life Sciences, Sydney, Australia.
Weaver N; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.
Augood C; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.; Centenary Institute, Centre for Inflammation, and University of Technology Sydney, Faculty of Science, School of Life Sciences, Sydney, Australia.
Brown AC; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.
Donovan C; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.; University of Technology Sydney, Faculty of Science, School of Life Sciences, Sydney, Australia.
Dupre P; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.; University of Montpellier, Montpellier Cancer Research Institute (IRCM), Montpellier, France.
Gunawardhana L; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.
Mayall JR; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.
Hansbro NG; Centenary Institute, Centre for Inflammation, and University of Technology Sydney, Faculty of Science, School of Life Sciences, Sydney, Australia.
Robertson AAB; The University of Queensland, School of Chemistry and Molecular Biosciences, Brisbane, Australia.
O'Neill LAJ; Trinity College Dublin, Trinity Biomedical Sciences Institute, School of Biochemistry and Immunology, Dublin, Ireland.
Cooper MA; Sitala Bio Ltd., Cambridge, UK.
Holliday EG; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.
Hansbro PM; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.; Centenary Institute, Centre for Inflammation, and University of Technology Sydney, Faculty of Science, School of Life Sciences, Sydney, Australia.
Gibson PG; University of Newcastle and Hunter Medical Research Institute, Newcastle, Australia.; Pokaż więcej
Źródło:: Respiratory research [Respir Res] 2023 Dec 04; Vol. 24 (1), pp. 303. Date of Electronic Publication: 2023 Dec 04.
Typ publikacji:: Journal Article
MeSH Terms:: Inflammasomes*
Asthma*/diagnosis
Asthma*/drug therapy
Humans ; Male ; Female ; NLR Family, Pyrin Domain-Containing 3 Protein ; Nigericin/pharmacology ; Lipopolysaccharides ; Leukocytes, Mononuclear ; Interleukin-1beta ; Sulfonamides

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Skocz do pozycji: 19.

Tytuł:: Qiqilian ameliorates vascular endothelial dysfunction by inhibiting NLRP3-ASC inflammasome activation in vivo and in vitro .
Autorzy:: Luo Y; The First Affiliated Hospital of Guangxi University of Chinese Medicine, Nanning, P.R. China.
Tan Z; Guangxi Medical University, Nanning, P.R. China.
Ye Y; No. 923 Hospital of the PLA Joint Logistics Support Force, Nanning, P.R. China.
Ma X; Guangxi University of Chinese Medicine, Nanning, P.R. China.
Yue G; Guangxi Internation Zhuang Medicine Hospital to Guangxi University of Chinese Medicine, Nanning, P.R. China.; Guangxi Internation Zhuang Medicine Hospital, Nanning, P.R. China.; Pokaż więcej
Źródło:: Pharmaceutical biology [Pharm Biol] 2023 Dec; Vol. 61 (1), pp. 815-824.
Typ publikacji:: Journal Article
MeSH Terms:: Hypertension*/drug therapy
Inflammasomes*
Animals ; Humans ; Rats ; Human Umbilical Vein Endothelial Cells ; Interleukin-18/metabolism ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Rats, Inbred SHR ; Rats, Inbred WKY

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Skocz do pozycji: 20.

Tytuł:: The pseudokinase MLKL contributes to host defense against Streptococcus pluranimalium infection by mediating NLRP3 inflammasome activation and extracellular trap formation.
Autorzy:: Lei YX; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.
Liu Y; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.; Animal Husbandry Institute, Agriculture and Animal Husbandry Academy of Inner Mongolia, Hohhot, China.
Xing LH; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.
Wu YJ; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.
Wang XY; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.
Meng FH; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.
Lou YN; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.
Ma ZG; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.
Yuan L; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.; Inner Mongolia Engineering Technology Research Center of Germplasm Resources Conservation and Utilization, College of Life Sciences, Inner Mongolia University, Hohhot, China.
Yu SX; State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, College of Life Sciences, Inner Mongolia University, Hohhot, China.; Inner Mongolia Engineering Technology Research Center of Germplasm Resources Conservation and Utilization, College of Life Sciences, Inner Mongolia University, Hohhot, China.; Pokaż więcej
Źródło:: Virulence [Virulence] 2023 Dec; Vol. 14 (1), pp. 2258057. Date of Electronic Publication: 2023 Sep 24.
Typ publikacji:: Journal Article
MeSH Terms:: Extracellular Traps*
Inflammasomes*/metabolism
Streptococcal Infections*/genetics
Streptococcal Infections*/metabolism
Animals ; Mice ; NLR Family, Pyrin Domain-Containing 3 Protein/genetics ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Protein Kinases/genetics
SCR Organism:: Streptococcus pluranimalium

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