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Wyszukujesz frazę ""Inflammation"" wg kryterium: Temat


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Tytuł :
Models of Inflammation: Carrageenan Air Pouch.
Autorzy :
Fehrenbacher JC; Department of Pharmacology and Toxicology and Stark Neurosciences Research Institute, Indiana University School of Medicine, Indianapolis, Indiana.
McCarson KE; Kansas Intellectual and Developmental Disabilities Research Center (KIDDRC), Department of Pharmacology, Toxicology and Therapeutics, University of Kansas Medical Center, Kansas City, Kansas.
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Źródło :
Current protocols [Curr Protoc] 2021 Aug; Vol. 1 (8), pp. e183.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents*/therapeutic use
Inflammation*/drug therapy
Animals ; Carrageenan/therapeutic use ; Disease Models, Animal ; Inflammation Mediators/therapeutic use ; Mice ; Rats
Czasopismo naukowe
Tytuł :
μSPE followed by HPLC-MS/MS for the determination of series D and E resolvins in biological matrices.
Autorzy :
Fanti F; University of Teramo, Faculty of Bioscience and Technology for Food, Agriculture and Environment, 64100, TE, Italy.
Oliva E; University of Teramo, Faculty of Bioscience and Technology for Food, Agriculture and Environment, 64100, TE, Italy.
Tortolani D; European Center for Brain Research, IRCCS Santa Lucia Foundation, 00164, Rome, Italy.
Di Meo C; University of Teramo, Faculty of Bioscience and Technology for Food, Agriculture and Environment, 64100, TE, Italy.
Fava M; University of Teramo, Faculty of Bioscience and Technology for Food, Agriculture and Environment, 64100, TE, Italy; European Center for Brain Research, IRCCS Santa Lucia Foundation, 00164, Rome, Italy.
Leuti A; Department of Medicine, Campus Bio-Medico University of Rome, 00128, Rome, Italy.
Rapino C; Faculty of Veterinary Medicine, University of Teramo, 64100, Teramo, Italy.
Sergi M; University of Teramo, Faculty of Bioscience and Technology for Food, Agriculture and Environment, 64100, TE, Italy. Electronic address: .
Maccarrone M; European Center for Brain Research, IRCCS Santa Lucia Foundation, 00164, Rome, Italy; Department of Biotechnological and Applied Clinical Sciences, University of L'Aquila, 67100, L'Aquila, Italy.
Compagnone D; University of Teramo, Faculty of Bioscience and Technology for Food, Agriculture and Environment, 64100, TE, Italy.
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Źródło :
Journal of pharmaceutical and biomedical analysis [J Pharm Biomed Anal] 2021 Sep 05; Vol. 203, pp. 114181. Date of Electronic Publication: 2021 May 29.
Typ publikacji :
Journal Article
MeSH Terms :
Inflammation*
Tandem Mass Spectrometry*
Chromatography, High Pressure Liquid ; Humans ; Inflammation Mediators ; Solid Phase Extraction
Czasopismo naukowe
Tytuł :
Long-Term Elevated Inflammatory Protein Levels in Asymptomatic SARS-CoV-2 Infected Individuals.
Autorzy :
Tserel L; Molecular Pathology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.
Jõgi P; Children's Clinic of Tartu University Hospital, Tartu, Estonia.; Department of Pediatrics, Institute of Clinical Medicine, University of Tartu, Tartu, Estonia.
Naaber P; SYNLAB Estonia, Tallinn, Estonia.; Department of Microbiology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.
Maslovskaja J; Molecular Pathology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.
Häling A; Molecular Pathology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.
Salumets A; Molecular Pathology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.
Zusinaite E; Institute of Technology, University of Tartu, Tartu, Estonia.
Soeorg H; Department of Microbiology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.
Lättekivi F; Department of Pathophysiology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.; Clinical Research Centre, Tartu University Hospital, Tartu, Estonia.
Ingerainen D; Family Doctor Center Järveotsa, Tallinn, Estonia.
Soots M; Family Doctor Center Kuressaare, Kuressaare, Estonia.
Toompere K; Department of Epidemiology and Biostatistics, Institute of Family Medicine and Public Health, University of Tartu, Tartu, Estonia.
Kaarna K; Clinical Research Centre, Tartu University Hospital, Tartu, Estonia.; Clinical Research Centre, Institute of Clinical Medicine, University of Tartu, Tartu, Estonia.
Kisand K; Molecular Pathology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.
Lutsar I; Department of Microbiology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.
Peterson P; Molecular Pathology, Institute of Biomedicine and Translational Medicine, University of Tartu, Tartu, Estonia.
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Źródło :
Frontiers in immunology [Front Immunol] 2021 Sep 17; Vol. 12, pp. 709759. Date of Electronic Publication: 2021 Sep 17 (Print Publication: 2021).
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Asymptomatic Infections*
Antibodies, Viral/*blood
CD4-Positive T-Lymphocytes/*immunology
COVID-19/*pathology
Inflammation Mediators/*blood
SARS-CoV-2/*immunology
Antibodies, Neutralizing/immunology ; Antibodies, Viral/immunology ; CD4 Lymphocyte Count ; Coronavirus Nucleocapsid Proteins/immunology ; Humans ; Inflammation/immunology ; Interleukin-18/blood ; Macrophages/immunology ; Monocytes/immunology ; Oncostatin M/blood ; Phosphoproteins/immunology ; Protein Domains/immunology ; S100A12 Protein/blood ; Spike Glycoprotein, Coronavirus/immunology ; Transforming Growth Factor alpha/blood
Czasopismo naukowe
Tytuł :
Toxicological Effects of Artificial Fine Particulate Matter in Rats through Induction of Oxidative Stress and Inflammation.
Autorzy :
Hong Z; Department of Otolaryngology-Head and Neck Surgery, The First Affiliated Hospital, Medical College, Xiamen University.; Xiamen Key Laboratory of Otolaryngology Head and Neck Surgery.; The School of Clinical Medicine, Fujian Medical University.
Zeng P; The School of Clinical Medicine, Fujian Medical University.
Zhuang G; Center for Atmospheric Chemistry Study, Department of Environmental Science and Engineering, Fudan University.
Guo Q; Department of Otolaryngology-Head and Neck Surgery, The First Affiliated Hospital, Medical College, Xiamen University.; Xiamen Key Laboratory of Otolaryngology Head and Neck Surgery.; The School of Clinical Medicine, Fujian Medical University.
Cai C; The School of Clinical Medicine, Fujian Medical University.; Department of Otorhinolaryngology, Zhongshan Hospital of Xiamen University.; Department of Otolaryngology Head and Neck Surgery, School of Medicine, Xiamen University.
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Źródło :
The Tohoku journal of experimental medicine [Tohoku J Exp Med] 2021 Sep; Vol. 255 (1), pp. 19-25.
Typ publikacji :
Journal Article
MeSH Terms :
Inflammation/*etiology
Oxidative Stress/*drug effects
Particulate Matter/*toxicity
Air Pollutants/chemistry ; Air Pollutants/toxicity ; Animals ; Female ; Humans ; Inflammation/pathology ; Inflammation Mediators/metabolism ; Inhalation Exposure ; Malondialdehyde/metabolism ; Models, Animal ; Nasal Mucosa/drug effects ; Nasal Mucosa/metabolism ; Nasal Mucosa/pathology ; Particle Size ; Particulate Matter/chemistry ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł :
The cGAS-STING pathway as a therapeutic target in inflammatory diseases.
Autorzy :
Decout A; Global Health Institute, Swiss Federal Institute of Technology Lausanne (EPFL), Lausanne, Switzerland.
Katz JD; IFM Therapeutics, Boston, MA, USA.
Venkatraman S; IFM Therapeutics, Boston, MA, USA.
Ablasser A; Global Health Institute, Swiss Federal Institute of Technology Lausanne (EPFL), Lausanne, Switzerland. .
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Źródło :
Nature reviews. Immunology [Nat Rev Immunol] 2021 Sep; Vol. 21 (9), pp. 548-569. Date of Electronic Publication: 2021 Apr 08.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms :
Adaptor Proteins, Signal Transducing/*antagonists & inhibitors
Inflammation/*therapy
Membrane Proteins/*antagonists & inhibitors
Nucleotidyltransferases/*antagonists & inhibitors
Adaptor Proteins, Signal Transducing/genetics ; Adaptor Proteins, Signal Transducing/immunology ; Animals ; Autophagy ; Cell Death ; Cell Proliferation ; DNA, Mitochondrial/genetics ; DNA, Mitochondrial/metabolism ; Enzyme Inhibitors/chemistry ; Enzyme Inhibitors/therapeutic use ; Gain of Function Mutation ; Humans ; Inflammation/immunology ; Inflammation/metabolism ; Inflammation Mediators/antagonists & inhibitors ; Inflammation Mediators/immunology ; Inflammation Mediators/metabolism ; Membrane Proteins/genetics ; Membrane Proteins/immunology ; Models, Biological ; Models, Molecular ; Nucleotides, Cyclic/immunology ; Nucleotides, Cyclic/metabolism ; Nucleotidyltransferases/genetics ; Nucleotidyltransferases/immunology ; Signal Transduction/drug effects ; Signal Transduction/genetics ; Signal Transduction/immunology
Czasopismo naukowe
Tytuł :
Suppression of LPS-Induced Inflammation and Cell Migration by Azelastine through Inhibition of JNK/NF-κB Pathway in BV2 Microglial Cells.
Autorzy :
Nguyen PL; College of Pharmacy and Integrated Research Institute for Drug Development, Dongguk University-Seoul, Goyang 10326, Korea.
Bui BP; College of Pharmacy and Integrated Research Institute for Drug Development, Dongguk University-Seoul, Goyang 10326, Korea.
Duong MTH; College of Pharmacy and Integrated Research Institute for Drug Development, Dongguk University-Seoul, Goyang 10326, Korea.
Lee K; College of Pharmacy and Integrated Research Institute for Drug Development, Dongguk University-Seoul, Goyang 10326, Korea.
Ahn HC; College of Pharmacy and Integrated Research Institute for Drug Development, Dongguk University-Seoul, Goyang 10326, Korea.
Cho J; College of Pharmacy and Integrated Research Institute for Drug Development, Dongguk University-Seoul, Goyang 10326, Korea.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Aug 23; Vol. 22 (16). Date of Electronic Publication: 2021 Aug 23.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Cell Movement/*drug effects
Inflammation/*drug therapy
MAP Kinase Signaling System/*drug effects
Microglia/*drug effects
NF-kappa B/*metabolism
Phthalazines/*pharmacology
Animals ; Cell Line ; Inflammation/chemically induced ; Inflammation/metabolism ; Inflammation Mediators/metabolism ; Interleukin-6/metabolism ; JNK Mitogen-Activated Protein Kinases/metabolism ; Lipopolysaccharides/pharmacology ; Mice ; Microglia/metabolism ; Nitric Oxide/metabolism ; Nitric Oxide Synthase Type II/metabolism ; Signal Transduction/drug effects ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł :
DAMPening COVID-19 Severity by Attenuating Danger Signals.
Autorzy :
Silva-Lagos LA; Department of Pathology and Medical Biology, University of Groningen, University Medical Center Groningen (UMCG), Groningen, Netherlands.
Pillay J; Department of Intensive Care, University Medical Center Groningen (UMCG), University of Groningen, Groningen, Netherlands.
van Meurs M; Department of Intensive Care, University Medical Center Groningen (UMCG), University of Groningen, Groningen, Netherlands.
Smink A; Department of Pathology and Medical Biology, University of Groningen, University Medical Center Groningen (UMCG), Groningen, Netherlands.
van der Voort PHJ; Department of Intensive Care, University Medical Center Groningen (UMCG), University of Groningen, Groningen, Netherlands.
de Vos P; Department of Pathology and Medical Biology, University of Groningen, University Medical Center Groningen (UMCG), Groningen, Netherlands.
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Źródło :
Frontiers in immunology [Front Immunol] 2021 Aug 12; Vol. 12, pp. 720192. Date of Electronic Publication: 2021 Aug 12 (Print Publication: 2021).
Typ publikacji :
Journal Article; Review
MeSH Terms :
Alarmins/*immunology
COVID-19/*physiopathology
Inflammation Mediators/*immunology
Adenosine/metabolism ; Alarmins/antagonists & inhibitors ; Animals ; COVID-19/complications ; COVID-19/immunology ; COVID-19/therapy ; Humans ; Inflammation/prevention & control ; Inflammation Mediators/antagonists & inhibitors ; Multiple Organ Failure/etiology ; Multiple Organ Failure/prevention & control ; Patient Acuity ; Signal Transduction ; Toll-Like Receptors/antagonists & inhibitors ; Toll-Like Receptors/immunology
Czasopismo naukowe
Tytuł :
Neutralizing interferon-α blocks inflammation-mediated vascular injury via PI3K and AMPK in systemic lupus erythematosus.
Autorzy :
Ding X; Institute of Pediatrics, The Second Xiangya Hospital, Central South University, Changsha, China.; Laboratory of Pediatrics Nephrology, Institute of Pediatrics, The Second Xiangya Hospital, Central South University, Changsha, China.
Xiang W; Hainan Maternal and Children's Medical Center, Haikou, China.
Yi R; Institute of Pediatrics, The Second Xiangya Hospital, Central South University, Changsha, China.; Institute of Pediatrics, HaiKou Hospital of the Maternal and Child Health, Haikou, China.
Huang X; Hainan Maternal and Children's Medical Center, Haikou, China.
Lin Q; Hainan Maternal and Children's Medical Center, Haikou, China.
He X; Institute of Pediatrics, The Second Xiangya Hospital, Central South University, Changsha, China.; Laboratory of Pediatrics Nephrology, Institute of Pediatrics, The Second Xiangya Hospital, Central South University, Changsha, China.
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Źródło :
Immunology [Immunology] 2021 Oct; Vol. 164 (2), pp. 372-385. Date of Electronic Publication: 2021 Jun 20.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
AMP-Activated Protein Kinases/*immunology
Antibodies, Neutralizing/*immunology
Inflammation/*immunology
Interferon-alpha/*immunology
Lupus Erythematosus, Systemic/*immunology
Phosphatidylinositol 3-Kinases/*immunology
Vascular System Injuries/*immunology
Animals ; Cells, Cultured ; Dendritic Cells/immunology ; Endothelial Cells/immunology ; Female ; Inflammation Mediators/immunology ; Mice ; Mice, Inbred C57BL ; Toll-Like Receptor 7/immunology
Czasopismo naukowe
Tytuł :
Lipopolysaccharide Binding Protein and CD14, Cofactors of Toll-like Receptors, Are Essential for Low-Grade Inflammation-Induced Exacerbation of Cartilage Damage in Mouse Models of Posttraumatic Osteoarthritis.
Autorzy :
Won Y; National Creative Research Initiatives Center for Osteoarthritis Pathogenesis and School of Life Sciences, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea.
Yang JI; National Creative Research Initiatives Center for Osteoarthritis Pathogenesis and School of Life Sciences, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea.
Park S; National Creative Research Initiatives Center for Osteoarthritis Pathogenesis and School of Life Sciences, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea.
Chun JS; National Creative Research Initiatives Center for Osteoarthritis Pathogenesis and School of Life Sciences, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea.
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Źródło :
Arthritis & rheumatology (Hoboken, N.J.) [Arthritis Rheumatol] 2021 Aug; Vol. 73 (8), pp. 1451-1460. Date of Electronic Publication: 2021 Jul 13.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Acute-Phase Proteins/*metabolism
Carrier Proteins/*metabolism
Inflammation Mediators/*metabolism
Lipopolysaccharide Receptors/*metabolism
Membrane Glycoproteins/*metabolism
Osteoarthritis/*genetics
Toll-Like Receptors/*metabolism
Animals ; Cartilage, Articular ; Chondrocytes/metabolism ; Diet, High-Fat/adverse effects ; Disease Models, Animal ; Endotoxemia/complications ; Inflammation ; Menisci, Tibial/metabolism ; Mice ; Mice, Knockout ; Osteoarthritis/etiology ; Signal Transduction/genetics
Czasopismo naukowe
Tytuł :
(R)-Ketamine ameliorates lethal inflammatory responses and multi-organ injury in mice induced by cecum ligation and puncture.
Autorzy :
Zhang J; Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba 260-8670, Japan; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, PR China.
Ma L; Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba 260-8670, Japan; Department of Anesthesiology, Renmin Hospital of Wuhan University, Wuhan 430022, PR China.
Hashimoto Y; Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba 260-8670, Japan; Department of Respirology, Chiba University Graduate School of Medicine, Chiba 260-8670, Japan.
Wan X; Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba 260-8670, Japan.
Shan J; Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba 260-8670, Japan.
Qu Y; Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba 260-8670, Japan.
Hashimoto K; Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba 260-8670, Japan. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2021 Nov 01; Vol. 284, pp. 119882. Date of Electronic Publication: 2021 Aug 10.
Typ publikacji :
Journal Article
MeSH Terms :
Cecum/*pathology
Inflammation/*drug therapy
Inflammation/*pathology
Ketamine/*therapeutic use
Multiple Organ Failure/*drug therapy
Multiple Organ Failure/*pathology
Animals ; Biomarkers/metabolism ; Cytokines/blood ; Disease Models, Animal ; Inflammation/blood ; Inflammation Mediators/blood ; Ketamine/pharmacology ; Ligation ; Lung/metabolism ; Lung/pathology ; Male ; Mice, Inbred C57BL ; Multiple Organ Failure/blood ; Organ Size/drug effects ; Protective Agents/pharmacology ; Protective Agents/therapeutic use ; Punctures ; Sepsis/blood ; Sepsis/drug therapy
Czasopismo naukowe
Tytuł :
Palmitic acid induces inflammation in placental trophoblasts and impairs their migration toward smooth muscle cells through plasminogen activator inhibitor-1.
Autorzy :
Rampersaud AM; Department of Anatomy and Cell Biology, Schulich School of Medicine and Dentistry, University of Western Ontario, London, ON, Canada.
Dunk CE; Research Centre for Women's and Infants' Health, Lunenfeld-Tanenbaum Research Institute, Sinai Health System, Toronto, ON, Canada.
Lye SJ; Research Centre for Women's and Infants' Health, Lunenfeld-Tanenbaum Research Institute, Sinai Health System, Toronto, ON, Canada.; Department of Obstetrics and Gynecology, Faculty of Medicine, University of Toronto, Toronto, ON, Canada.
Renaud SJ; Department of Anatomy and Cell Biology, Schulich School of Medicine and Dentistry, University of Western Ontario, London, ON, Canada.; Children's Health Research Institute, Lawson Health Research Institute, London, ON, Canada.
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Źródło :
Molecular human reproduction [Mol Hum Reprod] 2020 Nov 01; Vol. 26 (11), pp. 850-865.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*/drug effects
Cell Movement*/genetics
Inflammation*/blood
Inflammation*/chemically induced
Inflammation*/pathology
Inflammation*/physiopathology
Palmitic Acid/*pharmacology
Plasminogen Activator Inhibitor 1/*physiology
Trophoblasts/*physiology
Adult ; Cells, Cultured ; Decidua/drug effects ; Decidua/physiology ; Female ; HEK293 Cells ; Humans ; Inflammation Mediators/blood ; Inflammation Mediators/pharmacology ; Myocytes, Smooth Muscle/drug effects ; Myocytes, Smooth Muscle/physiology ; Obesity/blood ; Obesity/complications ; Obesity/physiopathology ; Palmitic Acid/blood ; Placenta/cytology ; Placenta/drug effects ; Placentation/drug effects ; Placentation/physiology ; Plasminogen Activator Inhibitor 1/blood ; Pregnancy ; Pregnancy Complications/blood ; Pregnancy Complications/physiopathology ; Trophoblasts/cytology ; Trophoblasts/drug effects ; Young Adult
Czasopismo naukowe
Tytuł :
Neurovascular inflammation in the pathogenesis of brain arteriovenous malformations.
Autorzy :
Krithika S; Cardiovascular Diseases and Diabetes Biology, Rajiv Gandhi Centre for Biotechnology, Thiruvananthapuram, Kerala, India.
Sumi S; Cardiovascular Diseases and Diabetes Biology, Rajiv Gandhi Centre for Biotechnology, Thiruvananthapuram, Kerala, India.
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Źródło :
Journal of cellular physiology [J Cell Physiol] 2021 Jul; Vol. 236 (7), pp. 4841-4856. Date of Electronic Publication: 2020 Dec 20.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms :
Brain/*blood supply
Intracranial Arteriovenous Malformations/*pathology
Neurogenic Inflammation/*pathology
Cerebral Hemorrhage/pathology ; Epigenesis, Genetic/genetics ; Gene Expression/genetics ; Humans ; Inflammation/pathology ; Inflammation Mediators/metabolism ; Polymorphism, Genetic/genetics ; Risk Factors
Czasopismo naukowe
Tytuł :
Characterization of N -Octadecanoyl-5-hydroxytryptamide Anti-Inflammatory Effect.
Autorzy :
Giorno TBS; Laboratório de Farmacologia da Dor e da Inflamação, Instituto de Ciências Biomédicas, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21941-901, Brazil.
Lima FA; Laboratório de Análise de Aromas, Instituto de Química, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21941-901, Brazil.
Brand ALM; Laboratório de Análise de Aromas, Instituto de Química, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21941-901, Brazil.
Oliveira CM; Laboratório de Análise de Aromas, Instituto de Química, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21941-901, Brazil.
Rezende CM; Laboratório de Análise de Aromas, Instituto de Química, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21941-901, Brazil.
Fernandes PD; Laboratório de Farmacologia da Dor e da Inflamação, Instituto de Ciências Biomédicas, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21941-901, Brazil.
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Źródło :
Molecules (Basel, Switzerland) [Molecules] 2021 Jun 18; Vol. 26 (12). Date of Electronic Publication: 2021 Jun 18.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Inflammation/*drug therapy
Serotonin/*pharmacology
Animals ; Carrageenan/pharmacology ; Cell Line ; Cell Movement/drug effects ; Cytokines/metabolism ; Disease Models, Animal ; Inflammation/chemically induced ; Inflammation/metabolism ; Inflammation Mediators/metabolism ; Interleukin-10/metabolism ; Leukocytes/drug effects ; Leukocytes/metabolism ; Macrophages/drug effects ; Macrophages/metabolism ; Male ; Mice ; Nitric Oxide/metabolism ; RAW 264.7 Cells
Czasopismo naukowe
Tytuł :
The NR4A agonist, Cytosporone B, attenuates pro-inflammatory mediators in human colorectal cancer tissue ex vivo.
Autorzy :
Ismaiel M; School of Medicine, University College Dublin, Dublin, Ireland; Department of Surgery, St. Vincent's University Hospital, Dublin, Ireland.
Murphy B; School of Medicine, University College Dublin, Dublin, Ireland; Department of Surgery, St. Vincent's University Hospital, Dublin, Ireland.
Aldhafiri S; School of Veterinary Medicine, University College Dublin, Dublin, Ireland.
Giffney HE; School of Veterinary Medicine, University College Dublin, Dublin, Ireland.
Thornton K; School of Veterinary Medicine, University College Dublin, Dublin, Ireland.
Mukhopadhya A; School of Veterinary Medicine, University College Dublin, Dublin, Ireland.
Keogh CE; Department of Anatomy, Physiology and Cell Biology, School of Veterinary Medicine, University of California Davis, Davis, CA, USA.
Fattah S; School of Pharmacy and Biomolecular Sciences, Royal College of Surgeons in Ireland, Dublin, Ireland.
Mohan HM; Department of Surgery, St. Vincent's University Hospital, Dublin, Ireland.
Cummins EP; School of Medicine, University College Dublin, Dublin, Ireland; Conway Institute of Biomolecular and Biomedical Research, University College Dublin, Dublin, Ireland.
Murphy EP; School of Medicine, University of Limerick, Limerick, Ireland.
Winter DC; School of Medicine, University College Dublin, Dublin, Ireland; Department of Surgery, St. Vincent's University Hospital, Dublin, Ireland. Electronic address: .
Crean D; School of Veterinary Medicine, University College Dublin, Dublin, Ireland; Conway Institute of Biomolecular and Biomedical Research, University College Dublin, Dublin, Ireland. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2021 May 21; Vol. 554, pp. 179-185. Date of Electronic Publication: 2021 Mar 30.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Colorectal Neoplasms/*drug therapy
Inflammation Mediators/*metabolism
Nuclear Receptor Subfamily 4, Group A, Member 1/*agonists
Phenylacetates/*pharmacology
Adult ; Aged ; Aged, 80 and over ; Chemokines/metabolism ; Colorectal Neoplasms/immunology ; Colorectal Neoplasms/metabolism ; Colorectal Neoplasms/pathology ; Cytokines/metabolism ; Female ; Humans ; Inflammation/immunology ; Inflammation/metabolism ; Inflammation/pathology ; Male ; Middle Aged
Czasopismo naukowe
Tytuł :
Ribes nigrum Leaf Extract Preferentially Inhibits IFN-γ-Mediated Inflammation in HaCaT Keratinocytes.
Autorzy :
Magnavacca A; Department of Pharmacological and Molecular Sciences, University of Milan, 20133 Milan, Italy.
Piazza S; Department of Pharmacological and Molecular Sciences, University of Milan, 20133 Milan, Italy.
Cammisa A; Specialist in Dermatology and Venereology, Corso di Porta Romana 131, 20122 Milan, Italy.
Fumagalli M; Department of Pharmacological and Molecular Sciences, University of Milan, 20133 Milan, Italy.
Martinelli G; Department of Pharmacological and Molecular Sciences, University of Milan, 20133 Milan, Italy.
Giavarini F; Department of Pharmacological and Molecular Sciences, University of Milan, 20133 Milan, Italy.
Sangiovanni E; Department of Pharmacological and Molecular Sciences, University of Milan, 20133 Milan, Italy.
Dell'Agli M; Department of Pharmacological and Molecular Sciences, University of Milan, 20133 Milan, Italy.
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Źródło :
Molecules (Basel, Switzerland) [Molecules] 2021 May 20; Vol. 26 (10). Date of Electronic Publication: 2021 May 20.
Typ publikacji :
Journal Article
MeSH Terms :
Inflammation/*chemically induced
Interferon-gamma/*pharmacology
Keratinocytes/*drug effects
Plant Extracts/*pharmacology
Plant Leaves/*chemistry
Ribes/*chemistry
Cell Line ; Cytokines/administration & dosage ; Cytokines/metabolism ; Humans ; Inflammation Mediators/administration & dosage ; Inflammation Mediators/metabolism ; Intercellular Adhesion Molecule-1/metabolism ; Kaempferols/pharmacology ; Keratinocytes/metabolism ; NF-kappa B/metabolism ; Quercetin/pharmacology
Czasopismo naukowe
Tytuł :
Trefoil Factor Family (TFF) Peptides and Their Links to Inflammation: A Re-evaluation and New Medical Perspectives.
Autorzy :
Hoffmann W; Institute of Molecular Biology and Medicinal Chemistry, Otto-von-Guericke University Magdeburg, Leipziger Str. 44, 39120 Magdeburg, Germany.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 May 06; Vol. 22 (9). Date of Electronic Publication: 2021 May 06.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Inflammation/*metabolism
Inflammation/*pathology
Trefoil Factors/*metabolism
Animals ; Colon/pathology ; Humans ; Inflammation Mediators/metabolism ; Models, Biological ; Neoplasms/metabolism ; Neoplasms/pathology ; Trefoil Factors/chemistry
Czasopismo naukowe
Tytuł :
Increased Dysfunctional and Plastic Regulatory T Cells in Idiopathic Orbital Inflammation.
Autorzy :
Chen J; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Ye H; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Xiao W; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Mao Y; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Ai S; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Chen R; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Lian X; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Shi L; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Wang X; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Bi S; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Yang S; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Ji X; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Zhang T; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
Yang H; State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University, Guangzhou, China.
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Źródło :
Frontiers in immunology [Front Immunol] 2021 May 03; Vol. 12, pp. 634847. Date of Electronic Publication: 2021 May 03 (Print Publication: 2021).
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Plasticity*
Inflammation/*immunology
Orbital Diseases/*immunology
T-Lymphocytes, Regulatory/*immunology
Case-Control Studies ; Cells, Cultured ; Coculture Techniques ; Fibroblasts/immunology ; Fibroblasts/metabolism ; Fibroblasts/pathology ; Fibrosis ; Humans ; Inflammation/blood ; Inflammation/diagnosis ; Inflammation Mediators/blood ; Interferon-gamma/metabolism ; Interleukin-1 Receptor-Like 1 Protein/blood ; Interleukin-33/blood ; Interleukin-33/genetics ; Orbital Diseases/blood ; Orbital Diseases/diagnosis ; Phenotype ; T-Lymphocytes, Regulatory/metabolism
Czasopismo naukowe
Tytuł :
Cytokine-regulated Th17 plasticity in human health and diseases.
Autorzy :
Cerboni S; Translational Science and Experimental Medicine, Research and Early Development, Respiratory and Immunology (R&I, BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Sweden.
Gehrmann U; Translational Science and Experimental Medicine, Research and Early Development, Respiratory and Immunology (R&I, BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Sweden.
Preite S; Bioscience, In vivo, Research and Early Development, Respiratory & Immunology (R&I, BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Sweden.
Mitra S; CNRS, INSERM, CHU Lille, Institut pour la Recherche contre le Cancer de Lille, UMR9020 - UMR-S 1277 - CANTHER - Cancer Heterogeneity, Plasticity and Resistance to Therapies, Univ. Lille, Lille, France.
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Źródło :
Immunology [Immunology] 2021 May; Vol. 163 (1), pp. 3-18. Date of Electronic Publication: 2020 Nov 06.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms :
Cell Plasticity*
Cytokines/*metabolism
Inflammation Mediators/*metabolism
Th17 Cells/*metabolism
Animals ; Autoimmunity ; Cell Lineage ; Chromatin Assembly and Disassembly ; Cytokines/genetics ; Epigenesis, Genetic ; Humans ; Inflammation/immunology ; Inflammation/metabolism ; Neoplasms/immunology ; Neoplasms/metabolism ; Phenotype ; Signal Transduction ; Th17 Cells/immunology ; Transcription Factors/genetics ; Transcription Factors/metabolism ; Tumor Escape ; Tumor Microenvironment
Czasopismo naukowe
Tytuł :
Functional differentiation and regulation of follicular T helper cells in inflammation and autoimmunity.
Autorzy :
Dong L; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
He Y; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
Cao Y; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
Wang Y; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
Jia A; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
Wang Y; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
Yang Q; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
Li W; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
Bi Y; State Key Laboratory of Pathogen and Biosecurity, Beijing Institute of Microbiology and Epidemiology, Beijing, China.
Liu G; Key Laboratory of Cell Proliferation and Regulation Biology, Ministry of Education, Institute of Cell Biology, College of Life Sciences, Beijing Normal University, Beijing, China.
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Źródło :
Immunology [Immunology] 2021 May; Vol. 163 (1), pp. 19-32. Date of Electronic Publication: 2020 Nov 18.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms :
Autoimmunity*
Cell Differentiation*
Autoimmune Diseases/*immunology
Inflammation/*immunology
T Follicular Helper Cells/*immunology
Animals ; Autoimmune Diseases/metabolism ; Humans ; Inflammation/metabolism ; Inflammation Mediators/metabolism ; Phenotype ; Signal Transduction ; T Follicular Helper Cells/metabolism
Czasopismo naukowe
Tytuł :
Glutathionylation chemistry promotes interleukin-1 beta-mediated glycolytic reprogramming and pro-inflammatory signaling in lung epithelial cells.
Autorzy :
Aboushousha R; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
Elko E; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
Chia SB; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
Manuel AM; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
van de Wetering C; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
van der Velden J; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
MacPherson M; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
Erickson C; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
Reisz JA; Department of Biochemistry and Molecular Genetics, University of Colorado, Aurora, CO, USA.
D'Alessandro A; Department of Biochemistry and Molecular Genetics, University of Colorado, Aurora, CO, USA.
Wouters EFM; Department of Respiratory Medicine, NUTRIM School of Nutrition and Translational Research in Metabolism, Toxicology and Metabolism, Maastricht University Medical Center, Maastricht, the Netherlands.; Ludwig Boltzmann Institute for Lung Research, Vienna, Austria.
Reynaert NL; Department of Respiratory Medicine, NUTRIM School of Nutrition and Translational Research in Metabolism, Toxicology and Metabolism, Maastricht University Medical Center, Maastricht, the Netherlands.
Lam YW; Department of Biology, University of Vermont College of Medicine, Burlington, VT, USA.
Anathy V; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
van der Vliet A; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
Seward DJ; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
Janssen-Heininger YMW; Department of Pathology and Laboratory Medicine, University of Vermont College of Medicine, Burlington, VT, USA.
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Źródło :
FASEB journal : official publication of the Federation of American Societies for Experimental Biology [FASEB J] 2021 May; Vol. 35 (5), pp. e21525.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Cellular Reprogramming*
Glycolysis*
Glutaredoxins/*physiology
Glutathione/*metabolism
Inflammation/*immunology
Interleukin-1beta/*pharmacology
Lung/*immunology
Animals ; Cytokines/metabolism ; Epithelial Cells/cytology ; Epithelial Cells/drug effects ; Epithelial Cells/immunology ; Epithelial Cells/metabolism ; Inflammation/metabolism ; Inflammation/pathology ; Inflammation Mediators/metabolism ; Lung/cytology ; Lung/drug effects ; Lung/metabolism ; Metabolome ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Oxidation-Reduction
Czasopismo naukowe

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