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Wyszukujesz frazę ""Inflammation"" wg kryterium: Temat


Tytuł :
LXW7 attenuates inflammation via suppressing Akt/nuclear factor kappa B and mitogen-activated protein kinases signaling pathways in lipopolysaccharide-stimulated BV2 microglial cells.
Autorzy :
Peng S; Department of Neurology, Peking University Shenzhen Hospital, Shenzhen, China.
Jia J; Department of Neurology, Peking University First Hospital, No.8 Xishiku Street, Xicheng District, Beijing 100034, China.
Sun J; Department of Neurology, Peking University Shenzhen Hospital, Shenzhen, China.
Xie Q; Department of Neurology, Peking University Shenzhen Hospital, Shenzhen, China.
Zhang X; Department of Neurology, Peking University Shenzhen Hospital, Shenzhen, China.
Deng Y; National Clinical Research Center for Geriatric Diseases Shenzhen Center, Peking University Shenzhen Hospital, Shenzhen, China. Electronic address: .
Yi L; Department of Neurology, Peking University Shenzhen Hospital, Shenzhen, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Dec; Vol. 77, pp. 105963. Date of Electronic Publication: 2019 Nov 13.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Inflammation/*drug therapy
Microglia/*drug effects
Mitogen-Activated Protein Kinases/*metabolism
NF-kappa B/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Signal Transduction/*drug effects
Animals ; Cell Line ; Cytokines/metabolism ; Inflammation/metabolism ; Inflammation Mediators/metabolism ; Lipopolysaccharides/pharmacology ; Mice ; Microglia/metabolism ; Protective Agents/pharmacology ; Up-Regulation/drug effects
Czasopismo naukowe
Tytuł :
The role of B regulatory (B10) cells in inflammatory disorders and their potential as therapeutic targets.
Autorzy :
Wu H; Department of Laboratory, People's Hospital of Jiangyin, Jiangsu 214400, China.
Su Z; International Genome Center, Jiangsu University, Zhenjiang 212013, China.
Barnie PA; International Genome Center, Jiangsu University, Zhenjiang 212013, China; Department of Biomedical Sciences, School of Allied Health Sciences, University of Cape Coast, Ghana. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2020 Jan; Vol. 78, pp. 106111. Date of Electronic Publication: 2019 Dec 24.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Autoimmune Diseases/*immunology
B-Lymphocytes, Regulatory/*immunology
Immunosuppressive Agents/*pharmacology
Inflammation/*immunology
Interleukin-10/*metabolism
Adoptive Transfer/methods ; Animals ; Antigen Presentation/drug effects ; Autoimmune Diseases/therapy ; B-Lymphocytes, Regulatory/drug effects ; B-Lymphocytes, Regulatory/metabolism ; B-Lymphocytes, Regulatory/transplantation ; Cell Communication/immunology ; Cytokines/metabolism ; Disease Models, Animal ; Humans ; Immunosuppressive Agents/therapeutic use ; Inflammation/therapy ; Inflammation Mediators/metabolism ; Interleukin-10/agonists ; Interleukin-10/immunology ; Th1 Cells/immunology ; Th2 Cells/immunology
Czasopismo naukowe
Tytuł :
RM, a novel resveratrol derivative, attenuates inflammatory responses induced by lipopolysaccharide via selectively increasing the Tollip protein in macrophages: A partial mechanism with therapeutic potential in an inflammatory setting.
Autorzy :
Kim WS; Advanced Radiation Technology Institute, Korea Atomic Energy Research Institute, Jeongeup, Jeonbuk 56212, Republic of Korea.
Kim K; Department of Food Science and Technology, Kongju National University, Yesan 32439, Republic of Korea.
Byun EB; Advanced Radiation Technology Institute, Korea Atomic Energy Research Institute, Jeongeup, Jeonbuk 56212, Republic of Korea.
Song HY; Advanced Radiation Technology Institute, Korea Atomic Energy Research Institute, Jeongeup, Jeonbuk 56212, Republic of Korea; Department of Biotechnology, College of Life Science and Biotechnology, Korea University, Seoul 136-701, Republic of Korea.
Han JM; Advanced Radiation Technology Institute, Korea Atomic Energy Research Institute, Jeongeup, Jeonbuk 56212, Republic of Korea.
Park WY; Department of Science in Korean Medicine, Graduate School, Kyung Hee University, Seoul, Republic of Korea.
Yuk JM; Department of Infection Biology, College of Medicine, Chungnam National University, Daejeon 301-747, Republic of Korea; Department of Medical Science, College of Medicine, Chungnam National University, Daejeon 301-747, Republic of Korea.
Byun EH; Department of Food Science and Technology, Kongju National University, Yesan 32439, Republic of Korea. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2020 Jan; Vol. 78, pp. 106072. Date of Electronic Publication: 2019 Dec 10.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Inflammation/*drug therapy
Intracellular Signaling Peptides and Proteins/*metabolism
Macrophages/*drug effects
Resveratrol/*pharmacology
Animals ; Anti-Inflammatory Agents/radiation effects ; Anti-Inflammatory Agents/therapeutic use ; Down-Regulation/drug effects ; Drug Evaluation, Preclinical ; Gamma Rays ; Gene Knockdown Techniques ; Inflammation/immunology ; Inflammation Mediators/metabolism ; Intracellular Signaling Peptides and Proteins/genetics ; Intracellular Signaling Peptides and Proteins/immunology ; Lipopolysaccharides/immunology ; Macrophage Activation/drug effects ; Macrophages/immunology ; Macrophages/metabolism ; Mice ; Primary Cell Culture ; RAW 264.7 Cells ; Resveratrol/analogs & derivatives ; Resveratrol/radiation effects ; Resveratrol/therapeutic use ; Signal Transduction/drug effects ; Signal Transduction/immunology ; Toll-Like Receptors/metabolism ; Up-Regulation
Czasopismo naukowe
Tytuł :
Inhibition of NLRP3 inflammasome-mediated pyroptosis in macrophage by cycloastragenol contributes to amelioration of imiquimod-induced psoriasis-like skin inflammation in mice.
Autorzy :
Deng G; State Key Laboratory of Pharmaceutical Biotechnology, Department of Biotechnology and Pharmaceutical Sciences, School of Life Sciences, Nanjing University, 163 Xianlin Avenue, Nanjing 210023, China.
Chen W; Affiliated Hospital of Nanjing University of Chinese Medicine, 155 Hanzhong Road, Nanjing 210029, China.
Wang P; State Key Laboratory of Pharmaceutical Biotechnology, Department of Biotechnology and Pharmaceutical Sciences, School of Life Sciences, Nanjing University, 163 Xianlin Avenue, Nanjing 210023, China.
Zhan T; School of Medicine, Shenzhen University, 1066 Xueyuan Avenue, Shenzhen 518055, China.
Zheng W; State Key Laboratory of Pharmaceutical Biotechnology, Department of Biotechnology and Pharmaceutical Sciences, School of Life Sciences, Nanjing University, 163 Xianlin Avenue, Nanjing 210023, China.
Gu Z; Jiangsu Yongjian Pharmaceutical Co., Ltd., 1 Yaocheng Avenue, China Medical City, Taizhou 225300, China.
Wang X; School of Medicine, Shenzhen University, 1066 Xueyuan Avenue, Shenzhen 518055, China. Electronic address: .
Ji X; State Key Laboratory of Pharmaceutical Biotechnology, Department of Biotechnology and Pharmaceutical Sciences, School of Life Sciences, Nanjing University, 163 Xianlin Avenue, Nanjing 210023, China. Electronic address: .
Sun Y; State Key Laboratory of Pharmaceutical Biotechnology, Department of Biotechnology and Pharmaceutical Sciences, School of Life Sciences, Nanjing University, 163 Xianlin Avenue, Nanjing 210023, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Sep; Vol. 74, pp. 105682. Date of Electronic Publication: 2019 Jun 13.
Typ publikacji :
Journal Article
MeSH Terms :
Inflammasomes/*metabolism
Inflammation/*immunology
Macrophages/*immunology
Psoriasis/*immunology
Sapogenins/*therapeutic use
Skin/*immunology
Animals ; Astragalus propinquus/immunology ; Cell Movement ; Cells, Cultured ; Cytokines/metabolism ; Disease Models, Animal ; Female ; Humans ; Imiquimod ; Inflammation/chemically induced ; Inflammation Mediators/metabolism ; Mice ; Mice, Inbred C57BL ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Psoriasis/chemically induced ; Pyroptosis
Czasopismo naukowe
Tytuł :
Melatonin improves the structure and function of autografted mice ovaries through reducing inflammation: A stereological and biochemical analysis.
Autorzy :
Noori Hassanvand M; Department of Biology, Faculty of Science, Arak University, Arak 381-5688138, Iran.
Soleimani Mehranjani M; Department of Biology, Faculty of Science, Arak University, Arak 381-5688138, Iran. Electronic address: .
Shojafar E; Department of Biology, Faculty of Science, Arak University, Arak 381-5688138, Iran.
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Sep; Vol. 74, pp. 105679. Date of Electronic Publication: 2019 Jun 12.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*metabolism
Inflammation/*immunology
Melatonin/*metabolism
Ovary/*pathology
Animals ; Apoptosis ; Cytokines/metabolism ; Estradiol/blood ; Female ; Inflammation Mediators/metabolism ; Malondialdehyde/blood ; Mice ; Mice, Inbred Strains ; Ovary/transplantation ; Progesterone/blood ; Transplantation, Autologous
Czasopismo naukowe
Tytuł :
Lipopolysaccharide-induced expression of astrocyte elevated gene-1 promotes degeneration and inflammation of chondrocytes via activation of nuclear factor-κB signaling.
Autorzy :
Qing Z; Center for Translational Medicine, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, China; The Department of Joint Surgery, Knee Word, Honghui Hospital, Xian Jiaotong University, Xi'an 710054, China.
Ye J; Department of Anesthesiology, Honghui Hospital, Xi'an Jiaotong University, Xi'an 710054, China.
Wu S; Center for Translational Medicine, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Jun; Vol. 71, pp. 84-92. Date of Electronic Publication: 2019 Mar 14.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Adhesion Molecules/*metabolism
Chondrocytes/*physiology
Inflammation/*immunology
Joints/*pathology
NF-kappa B/*metabolism
Osteoarthritis/*immunology
Apoptosis ; Cell Adhesion Molecules/genetics ; Cell Line ; Cytokines/metabolism ; Humans ; Inflammation Mediators/metabolism ; Lipopolysaccharides/immunology ; RNA, Small Interfering/genetics ; Signal Transduction ; Up-Regulation
Czasopismo naukowe
Tytuł :
Protective effects of ambroxol in psoriasis like skin inflammation: Exploration of possible mechanisms.
Autorzy :
Sunkari S; Department of Regulatory Toxicology, National Institute of Pharmaceutical Education and Research (NIPER), Balanagar, Hyderabad, Telangana 500037, India.
Thatikonda S; Department of Regulatory Toxicology, National Institute of Pharmaceutical Education and Research (NIPER), Balanagar, Hyderabad, Telangana 500037, India.
Pooladanda V; Department of Regulatory Toxicology, National Institute of Pharmaceutical Education and Research (NIPER), Balanagar, Hyderabad, Telangana 500037, India.
Challa VS; Department of Regulatory Toxicology, National Institute of Pharmaceutical Education and Research (NIPER), Balanagar, Hyderabad, Telangana 500037, India.
Godugu C; Department of Regulatory Toxicology, National Institute of Pharmaceutical Education and Research (NIPER), Balanagar, Hyderabad, Telangana 500037, India. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Jun; Vol. 71, pp. 301-312. Date of Electronic Publication: 2019 Mar 29.
Typ publikacji :
Journal Article
MeSH Terms :
Ambroxol/*therapeutic use
Anti-Inflammatory Agents/*therapeutic use
Inflammation/*drug therapy
Macrophages/*immunology
Psoriasis/*drug therapy
Skin/*pathology
Animals ; Cytokines/metabolism ; Disease Models, Animal ; Humans ; Hyperplasia ; Imiquimod ; Inflammation Mediators/metabolism ; Lipopolysaccharides/immunology ; Male ; Mice ; Mice, Inbred BALB C ; Oxidative Stress/drug effects ; RAW 264.7 Cells ; Signal Transduction
Czasopismo naukowe
Tytuł :
Lupeol inhibits LPS-induced neuroinflammation in cerebellar cultures and induces neuroprotection associated to the modulation of astrocyte response and expression of neurotrophic and inflammatory factors.
Autorzy :
Oliveira-Junior MS; Department of Biochemistry and Biophysics, Institute of Health Sciences, Federal University of Bahia, Brazil.
Pereira EP; Department of Biochemistry and Biophysics, Institute of Health Sciences, Federal University of Bahia, Brazil.
de Amorim VCM; Department of Biochemistry and Biophysics, Institute of Health Sciences, Federal University of Bahia, Brazil.
Reis LTC; Department of Biochemistry and Biophysics, Institute of Health Sciences, Federal University of Bahia, Brazil.
do Nascimento RP; Department of Biochemistry and Biophysics, Institute of Health Sciences, Federal University of Bahia, Brazil.
da Silva VDA; Department of Biochemistry and Biophysics, Institute of Health Sciences, Federal University of Bahia, Brazil.
Costa SL; Department of Biochemistry and Biophysics, Institute of Health Sciences, Federal University of Bahia, Brazil. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 May; Vol. 70, pp. 302-312. Date of Electronic Publication: 2019 Mar 07.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Astrocytes/*physiology
Cerebellum/*pathology
Neurogenic Inflammation/*drug therapy
Pentacyclic Triterpenes/*pharmacology
Animals ; Astrocytes/drug effects ; Cells, Cultured ; Disease Models, Animal ; Gene Expression Regulation ; Humans ; Inflammation Mediators/metabolism ; Interleukin-6/genetics ; Interleukin-6/metabolism ; Lipopolysaccharides/immunology ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Nerve Growth Factors/metabolism ; Neurogenic Inflammation/immunology ; Neuroprotection ; Nitric Oxide Synthase Type II/genetics ; Nitric Oxide Synthase Type II/metabolism ; Rats ; Rats, Wistar ; Tubulin/metabolism ; Tumor Necrosis Factor-alpha/genetics ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł :
Diosgenin ameliorates testicular damage in streptozotocin-diabetic rats through attenuation of apoptosis, oxidative stress, and inflammation.
Autorzy :
Khosravi Z; School of Medicine, Shahed University, Tehran, Iran.
Sedaghat R; Department of Anatomy and Pathology, School of Medicine, Shahed University, Tehran, Iran.
Baluchnejadmojarad T; Department of Physiology, School of Medicine, Iran University of Medical Sciences, Tehran, Iran.
Roghani M; Neurophysiology Research Center, Department of Physiology, Shahed University, Tehran, Iran. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 May; Vol. 70, pp. 37-46. Date of Electronic Publication: 2019 Feb 19.
Typ publikacji :
Journal Article
MeSH Terms :
Diabetes Mellitus/*drug therapy
Diabetes Mellitus, Experimental/*drug therapy
Diosgenin/*therapeutic use
Inflammation/*drug therapy
Spermatozoa/*drug effects
Testis/*drug effects
Animals ; Apoptosis ; Cytokines/metabolism ; Disease Models, Animal ; Humans ; Inflammation Mediators/metabolism ; Male ; Malondialdehyde/metabolism ; Oxidative Stress ; Rats ; Sperm Count ; Spermatozoa/physiology ; Testis/pathology ; Testosterone/blood
Czasopismo naukowe
Tytuł :
Indirubin-3'-monoxime prevents aberrant activation of GSK-3β/NF-κB and alleviates high fat-high fructose induced Aβ-aggregation, gliosis and apoptosis in mice brain.
Autorzy :
Sathiya Priya C; Department of Biochemistry and Biotechnology, Annamalai University, Annamalai Nagar, Tamil Nadu, India.
Vidhya R; Department of Biochemistry and Biotechnology, Annamalai University, Annamalai Nagar, Tamil Nadu, India.
Kalpana K; Department of Biochemistry and Biotechnology, Annamalai University, Annamalai Nagar, Tamil Nadu, India.
Anuradha CV; Department of Biochemistry and Biotechnology, Annamalai University, Annamalai Nagar, Tamil Nadu, India. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 May; Vol. 70, pp. 396-407. Date of Electronic Publication: 2019 Mar 08.
Typ publikacji :
Journal Article
MeSH Terms :
Amyloid beta-Peptides/*metabolism
Brain/*metabolism
Indoles/*therapeutic use
Neurodegenerative Diseases/*drug therapy
Neurogenic Inflammation/*drug therapy
Oximes/*therapeutic use
Animals ; Apoptosis ; Brain/pathology ; Diet, High-Fat ; Disease Models, Animal ; Fructose/administration & dosage ; Gliosis ; Glycogen Synthase Kinase 3 beta/antagonists & inhibitors ; Glycogen Synthase Kinase 3 beta/metabolism ; Humans ; Indoles/pharmacology ; Inflammation Mediators/metabolism ; Male ; Mice ; NF-kappa B/metabolism ; Oximes/pharmacology ; Phosphorylation ; Protein Aggregation, Pathological ; Signal Transduction
Czasopismo naukowe
Tytuł :
Kaempferol protects chondrogenic ATDC5 cells against inflammatory injury triggered by lipopolysaccharide through down-regulating miR-146a.
Autorzy :
Jiang R; Department of Orthopaedics, China-Japan Union Hospital of Jilin University, Changchun 130033, Jilin, China.
Hao P; Department of Orthopaedics, Heze Municipal Hospital, Heze 274031, Shandong, China.
Yu G; Department of Orthopaedics, Heze Municipal Hospital, Heze 274031, Shandong, China.
Liu C; Department of Orthopaedics, Heze Municipal Hospital, Heze 274031, Shandong, China.
Yu C; Department of Orthopaedics, Heze Municipal Hospital, Heze 274031, Shandong, China.
Huang Y; Department of Orthopaedics, China-Japan Union Hospital of Jilin University, Changchun 130033, Jilin, China.
Wang Y; Department of Orthopaedics, China-Japan Union Hospital of Jilin University, Changchun 130033, Jilin, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Apr; Vol. 69, pp. 373-381. Date of Electronic Publication: 2019 Feb 15.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Chondrocytes/*physiology
Inflammation/*drug therapy
Kaempferols/*pharmacology
MicroRNAs/*genetics
Osteoarthritis/*drug therapy
Animals ; Apoptosis/drug effects ; Cell Line ; Cytokines/metabolism ; Decorin/genetics ; Decorin/metabolism ; Disease Models, Animal ; Humans ; Inflammation Mediators/metabolism ; Lipopolysaccharides/immunology ; Mice ; Phosphatidylinositol 3-Kinases/metabolism ; RNA, Small Interfering/genetics
Czasopismo naukowe
Tytuł :
Inulin alleviates inflammation of alcoholic liver disease via SCFAs-inducing suppression of M1 and facilitation of M2 macrophages in mice.
Autorzy :
Wang Z; Clinical Medical College, Ningxia Medical University, Yinchuan 750004, Ningxia, China; Department of Gastroenterology, General Hospital of Ningxia Medical University, Yinchuan, Ningxia 750004, China.
Zhang X; College of Traditional Chinese Medicine, Ningxia Medical University, Yinchuan, Ningxia 750004, China.
Zhu L; Department of Pathogenic Biology and Medical Immunology, School of Basic Medical Sciences, Ningxia Medical University, Yinchuan, Ningxia 750004, China.
Yang X; Clinical Medical College, Ningxia Medical University, Yinchuan 750004, Ningxia, China.
He F; Department of Gastroenterology, General Hospital of Ningxia Medical University, Yinchuan, Ningxia 750004, China.
Wang T; Department of Pathogenic Biology and Medical Immunology, School of Basic Medical Sciences, Ningxia Medical University, Yinchuan, Ningxia 750004, China.
Bao T; Clinical Medical College, Ningxia Medical University, Yinchuan 750004, Ningxia, China.
Lu H; Clinical Medical College, Ningxia Medical University, Yinchuan 750004, Ningxia, China.
Wang H; Department of Pathogenic Biology and Medical Immunology, School of Basic Medical Sciences, Ningxia Medical University, Yinchuan, Ningxia 750004, China. Electronic address: .
Yang S; Department of Gastroenterology, General Hospital of Ningxia Medical University, Yinchuan, Ningxia 750004, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2020 Jan; Vol. 78, pp. 106062. Date of Electronic Publication: 2019 Dec 09.
Typ publikacji :
Journal Article
MeSH Terms :
Fatty Acids, Volatile/*metabolism
Gastrointestinal Microbiome/*immunology
Inflammation/*diet therapy
Inulin/*administration & dosage
Liver Diseases, Alcoholic/*diet therapy
Macrophages/*immunology
Administration, Oral ; Animals ; Coculture Techniques ; Disease Models, Animal ; Ethanol/administration & dosage ; Ethanol/toxicity ; Fatty Acids, Volatile/analysis ; Fatty Acids, Volatile/immunology ; Feces/chemistry ; Inflammation/immunology ; Inflammation/pathology ; Intestinal Mucosa/microbiology ; Liver/cytology ; Liver/immunology ; Liver/pathology ; Liver Diseases, Alcoholic/immunology ; Liver Diseases, Alcoholic/pathology ; Macrophage Activation ; Macrophages/metabolism ; Male ; Mice ; Primary Cell Culture ; RAW 264.7 Cells
Czasopismo naukowe
Tytuł :
Endoplasmic reticulum stress is involved in ventilator-induced lung injury in mice via the IRE1α-TRAF2-NF-κB pathway.
Autorzy :
Ye L; Department of Anesthesiology, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China; Perioperative Medicine Research Center, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China.
Zeng Q; Department of Anesthesiology, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China; Perioperative Medicine Research Center, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China.
Dai H; Department of Anesthesiology, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China; Perioperative Medicine Research Center, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China.
Zhang W; Department of Anesthesiology, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China; Perioperative Medicine Research Center, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China.
Wang X; Department of Anesthesiology, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China; Perioperative Medicine Research Center, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China.
Ma R; Department of Anesthesiology, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China; Perioperative Medicine Research Center, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China.
Hong X; Department of Anesthesiology, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China; Perioperative Medicine Research Center, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China.
Zhao C; Department of Anesthesiology, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China; Perioperative Medicine Research Center, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China.
Pan L; Department of Anesthesiology, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China; Perioperative Medicine Research Center, Guangxi Medical University Cancer Hospital, Nanning 530021, Guangxi Zhuang Autonomous Region, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2020 Jan; Vol. 78, pp. 106069. Date of Electronic Publication: 2019 Dec 13.
Typ publikacji :
Journal Article
MeSH Terms :
Endoplasmic Reticulum Stress/*immunology
Inflammation/*immunology
Signal Transduction/*immunology
Ventilator-Induced Lung Injury/*immunology
Animals ; Bronchoalveolar Lavage Fluid/immunology ; Cytokines/immunology ; Cytokines/metabolism ; Disease Models, Animal ; Endoplasmic Reticulum Stress/drug effects ; Endoribonucleases/antagonists & inhibitors ; Endoribonucleases/metabolism ; Humans ; Inflammation/diagnosis ; Inflammation/drug therapy ; Inflammation/pathology ; Lung/immunology ; Lung/pathology ; Male ; Mice ; NF-kappa B/metabolism ; Protein-Serine-Threonine Kinases/antagonists & inhibitors ; Protein-Serine-Threonine Kinases/metabolism ; Severity of Illness Index ; Signal Transduction/drug effects ; TNF Receptor-Associated Factor 2/metabolism ; Up-Regulation/immunology ; Ventilator-Induced Lung Injury/diagnosis ; Ventilator-Induced Lung Injury/drug therapy ; Ventilator-Induced Lung Injury/pathology
Czasopismo naukowe
Tytuł :
Berberine suppresses IL-33-induced inflammatory responses in mast cells by inactivating NF-κB and p38 signaling.
Autorzy :
Li W; Department of Cardiology, Affiliated Liyuan Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430077, China.
Yin N; Department of Anatomy, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan 430065, China.
Tao W; School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan 430065, China.
Wang Q; Department of Pathogen Biology, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan 430065, China.
Fan H; Department of Pathogen Biology, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan 430065, China.
Wang Z; Department of Pathogen Biology, School of Basic Medical Sciences, Hubei University of Chinese Medicine, Wuhan 430065, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Jan; Vol. 66, pp. 82-90. Date of Electronic Publication: 2018 Nov 13.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*therapeutic use
Berberine/*therapeutic use
Hypersensitivity/*drug therapy
Inflammation/*drug therapy
Mast Cells/*drug effects
Animals ; Cells, Cultured ; Cytokines/metabolism ; Humans ; Immunoglobulin E/metabolism ; Inflammation Mediators/metabolism ; Interleukin-33/metabolism ; Male ; Mast Cells/immunology ; NF-kappa B/metabolism ; Phosphorylation ; Rats ; Rats, Sprague-Dawley ; Signal Transduction ; p38 Mitogen-Activated Protein Kinases/metabolism
Czasopismo naukowe
Tytuł :
The protective role of microRNA-133b in restricting hippocampal neurons apoptosis and inflammatory injury in rats with depression by suppressing CTGF.
Autorzy :
Pei G; Intervention Department, The Second Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450014, China.
Xu L; Intervention Department, The Second Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450014, China.
Huang W; Intervention Department, The Second Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450014, China.
Yin J; Health Examination Department, Qingdao Hiser Medica, Qingdao 266000, Shandong, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2020 Jan; Vol. 78, pp. 106076. Date of Electronic Publication: 2019 Dec 09.
Typ publikacji :
Journal Article
MeSH Terms :
Connective Tissue Growth Factor/*genetics
Depression/*immunology
Hippocampus/*pathology
Inflammation/*genetics
MicroRNAs/*metabolism
Animals ; Apoptosis/genetics ; Apoptosis/immunology ; Depression/genetics ; Depression/pathology ; Depression/psychology ; Disease Models, Animal ; Disease Resistance/genetics ; Hippocampus/cytology ; Hippocampus/immunology ; Humans ; Inflammation/immunology ; Inflammation/pathology ; Male ; Neurons/immunology ; Neurons/pathology ; Rats ; Stress, Psychological/complications ; Stress, Psychological/psychology
Czasopismo naukowe
Tytuł :
circAtp9b knockdown alleviates LPS-caused inflammation provided that microRNA-27a is upregulated.
Autorzy :
Sun J; Neonatal Intensive Care Unit, Henan Provincial People's Hospital, Zhengzhou 450003, Henan, China.
Wang X; Neonatal Intensive Care Unit, Henan Provincial People's Hospital, Zhengzhou 450003, Henan, China.
Wang D; Neonatal Intensive Care Unit, Henan Provincial People's Hospital, Zhengzhou 450003, Henan, China.
Zhao Z; Neonatal Intensive Care Unit, Henan Provincial People's Hospital, Zhengzhou 450003, Henan, China.
Zhang L; Neonatal Intensive Care Unit, Henan Provincial People's Hospital, Zhengzhou 450003, Henan, China.
Zhang J; Neonatal Intensive Care Unit, Henan Provincial People's Hospital, Zhengzhou 450003, Henan, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2020 Jan; Vol. 78, pp. 105925. Date of Electronic Publication: 2019 Nov 15.
Typ publikacji :
Journal Article
MeSH Terms :
Inflammation/*genetics
MicroRNAs/*genetics
RNA, Circular/*genetics
Apoptosis ; Cell Line ; Gene Silencing ; Humans ; Inflammation/chemically induced ; Inflammation/immunology ; Interleukin-6/immunology ; Lipopolysaccharides ; Reactive Oxygen Species/immunology ; Tumor Necrosis Factor-alpha/immunology ; Up-Regulation
Czasopismo naukowe
Tytuł :
Glucagon-like peptide-1 receptor agonist exendin-4 mitigates lipopolysaccharide-induced inflammatory responses in RAW264.7 macrophages.
Autorzy :
Lu C; Department of General Surgery, Chinese People's Liberation Army General Hospital, Beijing, China.
Xie T; Department of General Surgery, Chinese People's Liberation Army General Hospital, Beijing, China.
Guo X; Department of General Surgery, Chinese People's Liberation Army General Hospital, Beijing, China.
Wu D; Department of General Surgery, Chinese People's Liberation Army General Hospital, Beijing, China.
Li S; Department of General Surgery, Chinese People's Liberation Army General Hospital, Beijing, China.
Li X; Department of General Surgery, Chinese People's Liberation Army General Hospital, Beijing, China.
Lu Y; Department of General Surgery, Chinese People's Liberation Army General Hospital, Beijing, China.
Wang X; Department of General Surgery, Chinese People's Liberation Army General Hospital, Beijing, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Dec; Vol. 77, pp. 105969. Date of Electronic Publication: 2019 Nov 01.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Exenatide/*pharmacology
Inflammation/*drug therapy
Inflammation/*metabolism
Lipopolysaccharides/*pharmacology
Macrophages/*drug effects
Macrophages/*metabolism
Animals ; Cell Line ; Cyclooxygenase 2/metabolism ; Cytokines/metabolism ; Diabetes Mellitus, Type 2/metabolism ; Glucagon-Like Peptide 1/metabolism ; Inflammation/chemically induced ; Mice ; NF-kappa B/metabolism ; Nitric Oxide/metabolism ; RAW 264.7 Cells ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł :
Trichostatin A modulates the macrophage phenotype by enhancing autophagy to reduce inflammation during polymicrobial sepsis.
Autorzy :
Cui SN; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Chen ZY; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Yang XB; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Chen L; Department of Anesthesiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Yang YY; Department of Anesthesiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Pan SW; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Wang YX; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Xu JQ; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhou T; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Xiao HR; Department of Anesthesiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Qin L; Department of Anesthesiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Yuan SY; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China. Electronic address: .
Shang Y; Department of Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Institute of Anesthesiology and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Dec; Vol. 77, pp. 105973. Date of Electronic Publication: 2019 Oct 31.
Typ publikacji :
Journal Article
MeSH Terms :
Autophagy/*drug effects
Hydroxamic Acids/*pharmacology
Inflammation/*drug therapy
Macrophages, Peritoneal/*drug effects
Sepsis/*drug therapy
Animals ; Biomarkers/metabolism ; Cell Line ; Cytokines/metabolism ; Disease Models, Animal ; Histone Deacetylase Inhibitors/pharmacology ; Inflammation/metabolism ; Ligation/methods ; Lung/drug effects ; Lung/metabolism ; Macrophage Activation/drug effects ; Macrophages, Peritoneal/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Phenotype ; Punctures/methods ; RAW 264.7 Cells ; Sepsis/metabolism
Czasopismo naukowe
Tytuł :
Suppression of miR-21 and miR-155 of macrophage by cinnamaldehyde ameliorates ulcerative colitis.
Autorzy :
Qu S; Experiment Center for Science and Technology, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Shen Y; School of Pharmacy, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Wang M; Experiment Center for Science and Technology, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Wang X; Experiment Center for Science and Technology, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China. Electronic address: .
Yang Y; Experiment Center for Science and Technology, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Feb; Vol. 67, pp. 22-34. Date of Electronic Publication: 2018 Dec 06.
Typ publikacji :
Journal Article
MeSH Terms :
Acrolein/*analogs & derivatives
Anti-Inflammatory Agents/*therapeutic use
Colitis, Ulcerative/*drug therapy
Colon/*physiology
Inflammation/*drug therapy
Macrophages, Peritoneal/*physiology
MicroRNAs/*genetics
Acrolein/therapeutic use ; Animals ; Antigens, Differentiation/metabolism ; Colitis, Ulcerative/chemically induced ; Colon/pathology ; Dextran Sulfate ; Disease Models, Animal ; Humans ; Inflammasomes/metabolism ; Inflammation Mediators/metabolism ; Interleukin-10/metabolism ; Male ; Mice ; Mice, Inbred BALB C ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; RAW 264.7 Cells ; Reactive Oxygen Species/metabolism ; U937 Cells
Czasopismo naukowe
Tytuł :
NecroX-5 ameliorates inflammation by skewing macrophages to the M2 phenotype.
Autorzy :
Nam SY; Department of Plastic & Reconstructive Surgery, Seoul National University Bundang Hospital, Seongnam 13620, Republic of Korea.
Shin BH; Department of Biomedical Engineering, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.
Lee M; Department of Plastic & Reconstructive Surgery, Seoul National University Bundang Hospital, Seongnam 13620, Republic of Korea.
Lee S; Department of Plastic & Reconstructive Surgery, Seoul National University Bundang Hospital, Seongnam 13620, Republic of Korea.
Heo CY; Department of Plastic & Reconstructive Surgery, Seoul National University Bundang Hospital, Seongnam 13620, Republic of Korea. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2019 Jan; Vol. 66, pp. 139-145. Date of Electronic Publication: 2018 Nov 16.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*therapeutic use
Colitis/*drug therapy
Heterocyclic Compounds, 4 or More Rings/*therapeutic use
Inflammation/*drug therapy
Macrophages/*drug effects
Sulfones/*therapeutic use
Animals ; Cell Differentiation ; Cell Plasticity ; Colitis/chemically induced ; Cytokines/metabolism ; Dextran Sulfate ; Disease Models, Animal ; Humans ; Inflammation Mediators/metabolism ; Macrophages/physiology ; Male ; Mice ; Mice, Inbred C57BL ; RAW 264.7 Cells ; Th2 Cells/immunology
Czasopismo naukowe

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