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Wyszukujesz frazę ""Inflammation pathology"" wg kryterium: Temat


Tytuł :
Regulatory T Cells Restrain Pathogenic T Helper Cells during Skin Inflammation.
Autorzy :
Tom Hartwig
Pascale Zwicky
Bettina Schreiner
Nikhil Yawalkar
Phil Cheng
Alexander Navarini
Reinhard Dummer
Lukas Flatz
Curdin Conrad
Christoph Schlapbach
Burkhard Becher
Pokaż więcej
Temat :
610 Medicine & health
Clinic for Neurology
Adult
Aged
Aged, 80 and over
Animals
Female
Forkhead Transcription Factors/metabolism
Granulocyte-Macrophage Colony-Stimulating Factor/biosynthesis
Humans
Imiquimod/adverse effects
Inflammation/chemically induced
Inflammation/immunology
Inflammation/pathology
Male
Mice, Inbred C57BL
Middle Aged
Neutralization Tests
Phagocytes/pathology
Psoriasis/immunology
Psoriasis/pathology
Receptors, Antigen, T-Cell, alpha-beta/metabolism
Skin/pathology
T-Lymphocytes, Helper-Inducer/immunology
T-Lymphocytes, Regulatory/immunology
Aldara
CD4 T cells
Foxp3
GM-CSF
Treg cells
ipilimumab
melanoma
psoriasis
skin inflammation
chemical and pharmacologic phenomena
Dermatology Clinic
hemic and immune systems
integumentary system
Biology (General)
QH301-705.5
Źródło :
Hartwig, Tom; Zwicky, Pascale; Schreiner, Bettina; Yawalkar, Nikhil; Cheng, Phil; Navarini, Alexander; Dummer, Reinhard; Flatz, Lukas; Conrad, Curdin; Schlapbach, Christoph; Becher, Burkhard (2018). Regulatory T Cells Restrain Pathogenic T Helper Cells during Skin Inflammation. Cell Reports, 25(13):3564-3572.e4.
Hartwig, Tom; Zwicky, Pascale; Schreiner, Bettina; Yawalkar, Nikhil; Cheng, Phil; Navarini, Alexander; Dummer, Reinhard; Flatz, Lukas; Conrad, Curdin; Schlapbach, Christoph; Becher, Burkhard (2018). Regulatory T Cells Restrain Pathogenic T Helper Cells during Skin Inflammation. Cell reports, 25(13), 3564-3572.e4. Cell Press 10.1016/j.celrep.2018.12.012
Cell Reports, Vol 25, Iss 13, Pp 3564-3572.e4 (2018)
Cell reports, vol. 25, no. 13, pp. 3564-3572.e4
Opis pliku :
application/pdf
Tytuł :
CARD14 gain-of-function mutation alone is sufficient to drive IL-23/IL-17-mediated psoriasiform skin inflammation in vivo
Autorzy :
Mellett, Mark
Meier, Barbara
Mohanan, Deepa
Schairer, Rebekka
Cheng, Phil
Satoh, Takashi K
Kiefer, Betina
Ospelt, Caroline
Nobbe, Stephan
Thome, Margot
Contassot, Emmanuel
French, Lars E
Pokaż więcej
Temat :
Dermatology Clinic
610 Medicine & health
Animals
CARD Signaling Adaptor Proteins/genetics
CARD Signaling Adaptor Proteins/metabolism
Cells, Cultured
Cytokines/metabolism
DNA/genetics
DNA Mutational Analysis
Disease Models, Animal
Female
Gain of Function Mutation
Guanylate Kinases/genetics
Guanylate Kinases/metabolism
Humans
Inflammation/genetics
Inflammation/metabolism
Inflammation/pathology
Interleukin-17/metabolism
Interleukin-23/metabolism
Keratinocytes/metabolism
Keratinocytes/pathology
Membrane Proteins
Mice
Psoriasis/genetics
Psoriasis/metabolism
Psoriasis/pathology
Cell Biology, Biochemistry, Molecular Biology, Dermatology
Rheumatology Clinic and Institute of Physical Medicine
Źródło :
Mellett, Mark; Meier, Barbara; Mohanan, Deepa; Schairer, Rebekka; Cheng, Phil; Satoh, Takashi K; Kiefer, Betina; Ospelt, Caroline; Nobbe, Stephan; Thome, Margot; Contassot, Emmanuel; French, Lars E (2018). CARD14 gain-of-function mutation alone is sufficient to drive IL-23/IL-17-mediated psoriasiform skin inflammation in vivo. Journal of Investigative Dermatology, 138(9):2010-2023.
The Journal of investigative dermatology, vol. 138, no. 9, pp. 2010-2023
Opis pliku :
application/pdf
Tytuł :
Erysipelatoid Carcinoma
Autorzy :
Thiago Sande Miguel
Daniel Almeida da Costa
Ana Paula Moura de Almeida
Livia Cristina de Melo Pino
Daniel Cohen Goldemberg
Bruna Sande Miguel
Sebastiao Célio Horta Coelho Filho
Pokaż więcej
Temat :
Neoplasias cutâneas
Neoplasias da mama
R5-920
Breast neoplasms
Inflammation/pathology
Medicine (General)
Erysipeloid
Inflamação/patologia
Skin neoplasms
Erisipeloide
Źródło :
Revista da Associação Médica Brasileira v.64 n.6 2018
Revista da Associação Médica Brasileira, Vol 64, Iss 6, Pp 492-497
Revista da Associação Médica Brasileira
Opis pliku :
text/html
Tytuł :
Myeloid differentiation primary response gene (MyD) 88 signalling is not essential for intestinal fibrosis development
Autorzy :
Beerenwinkel, N.
Lutz, C.
Weder, B.
Hünerwadel, A.
Fagagnini, S.
Lang, B.
Rossel, J. B.
Rogler, G.
Misselwitz, B.
Hausmann, M.
Pokaż więcej
Temat :
Gastrointestinal models
Animals
Collagen/metabolism
Disease Models, Animal
Fibrosis/metabolism
Fibrosis/pathology
Inflammation/metabolism
Inflammation/pathology
Intestinal Mucosa/metabolism
Intestines/pathology
Matrix Metalloproteinase 9/metabolism
Mice
Mice, Inbred C57BL
Myeloid Differentiation Factor 88/metabolism
Neutrophils/metabolism
Neutrophils/pathology
Signal Transduction/physiology
Transforming Growth Factor beta1/metabolism
Intestinal diseases
610 Medicine & health
Science
Medicine
Clinic for Gastroenterology and Hepatology
Article
Źródło :
Scientific Reports, 7 (1)
Lutz, C; Weder, B; Hünerwadel, A; Fagagnini, S; Lang, B; Beerenwinkel, N; Rossel, J B; Rogler, G; Misselwitz, B; Hausmann, M (2017). Myeloid differentiation primary response gene (MyD) 88 signalling is not essential for intestinal fibrosis development. Scientific Reports, 7:17678.
Scientific Reports, Vol 7, Iss 1, Pp 1-10 (2017)
Scientific reports, vol. 7, no. 1, pp. 17678
Opis pliku :
application/pdf; application/application/pdf
Tytuł :
Loss of E-cadherin as Part of a Migratory Phenotype in Melanoma Is Associated With Ulceration
Autorzy :
Bønnelykke-Behrndtz, Marie Louise
Steiniche, Torben
Nørgaard, Peter
Danielsen, Allan V
Damsgaard, Tine E
Christensen, Ib J.
Bastholt, Lars
Møller, Holger J.
Schmidt, Henrik
Pokaż więcej
Temat :
E-cadherin
EMT
melanoma
ulceration
Melanoma/pathology
Humans
Skin Neoplasms/pathology
Phenotype
Cadherins/metabolism
Ulcer/pathology
Neutrophils/pathology
Inflammation/pathology
Antigens, CD
Cell Movement
Źródło :
Bønnelykke-Behrndtz, M L, Steiniche, T, Nørgaard, P, Danielsen, A V, Damsgaard, T E, Christensen, I J, Bastholt, L, Møller, H J & Schmidt, H 2017, ' Loss of E-cadherin as Part of a Migratory Phenotype in Melanoma Is Associated With Ulceration ', American Journal of Dermatopathology, vol. 39, no. 9, pp. 672–678 . https://doi.org/10.1097/DAD.0000000000000750
Tytuł :
Shaping mycolactone for therapeutic use against inflammatory disorders
Autorzy :
Guenin-Mace, Laure
Baron, Ludivine
Chany, Anne-Caroline
Tresse, Cédric
Saint-Auret, Sarah
Jönsson, Friederike
Le Chevalier, Fabien
Bruhns, Pierre
Bismuth, Georges
Hidalgo-Lucas, Sophie
Bisson, Jean-François
Blanchard, Nicolas
Demangel, Caroline
Pokaż więcej
Temat :
MESH: Animals
MESH : Pain/complications
MESH: Inflammation/drug therapy
[SDV.IMM]Life Sciences [q-bio]/Immunology
MESH : Animals
MESH: Jurkat Cells
[SDV] Life Sciences [q-bio]
MESH : HeLa Cells
MESH: Mycobacterium ulcerans/physiology
MESH : Protective Agents/therapeutic use
[SDV.BC]Life Sciences [q-bio]/Cellular Biology
MESH: Inflammation/pathology
[SDV.BC] Life Sciences [q-bio]/Cellular Biology
[SDV.BDD]Life Sciences [q-bio]/Development Biology
MESH : Pain/drug therapy
MESH: Protective Agents/therapeutic use
MESH: Immunomodulation
MESH: HeLa Cells
MESH : Jurkat Cells
MESH : Inflammation/drug therapy
[SDV.IMM] Life Sciences [q-bio]/Immunology
MESH : Mycobacterium ulcerans/physiology
MESH: Tetradecanoylphorbol Acetate/pharmacology
[SDV.BDD] Life Sciences [q-bio]/Development Biology
MESH: Pain/drug therapy
MESH: Humans
MESH : Tetradecanoylphorbol Acetate/pharmacology
MESH : Immunomodulation
MESH : Macrolides/therapeutic use
MESH: Pain/complications
MESH: Mice
MESH : Macrolides/chemistry
MESH : Chronic Disease
MESH : Inflammation/pathology
MESH : Humans
[SDV]Life Sciences [q-bio]
MESH: Chronic Disease
MESH: Macrolides/chemistry
MESH: Macrolides/therapeutic use
MESH : Mice
Źródło :
Science Translational Medicine
Science Translational Medicine, American Association for the Advancement of Science, 2015, 7 (289), pp.289ra85 - 289ra85. 〈10.1126/scitranslmed.aab0458〉
Science Translational Medicine, American Association for the Advancement of Science, 2015, 7 (289), pp.289ra85 - 289ra85. ⟨10.1126/scitranslmed.aab0458⟩

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