Temat: "Insulin resistance" - OpacWWW

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Insulin resistance.

Insulin resistance is the body’s weakened ability to respond to or act on the insulin it produces. The condition, if untreated, leads to prediabetes or type 2 diabetes, which can go undiagnosed for years. Early diagnosis of insulin resistance allows patients to be proactive in order to delay or completely avoid the onset of type 2 diabetes.

Salem Press Encyclopedia, 2020. 1p.

Skocz do pozycji: 1 1.

Tytuł :: Triglyceride-Glucose Index Correlate With Telomere Length in Healthy Adults From the National Health and Nutrition Examination Survey.
Autorzy :: Hu L; Department of Cardiology, Peking University First Hospital, Beijing, China.
Zhang Q; Department of Occupational and Environmental Health Sciences, School of Public Health, Peking University, Beijing, China.
Bai Y; Department of Epidemiology and Health Statistics, Peking University, Beijing, China.
Hu G; School of Engineering Medicine, Beihang University, Beijing, China.; Beijing Advanced Innovation Center for Big Data-Based Precision Medicine, and Advanced Innovation Centre for Biomedical Engineering, Beihang University, Beijing, China.
Li J; Department of Cardiology, Peking University First Hospital, Beijing, China.; Pokaż więcej
Źródło :: Frontiers in endocrinology [Front Endocrinol (Lausanne)] 2022 Jun 02; Vol. 13, pp. 844073. Date of Electronic Publication: 2022 Jun 02 (Print Publication: 2022).
Typ publikacji :: Journal Article
MeSH Terms :: Insulin Resistance*/genetics
Adult ; Blood Glucose/analysis ; Glucose ; Humans ; Nutrition Surveys ; Telomere/chemistry ; Telomere/genetics ; Triglycerides
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 2 2.

Tytuł :: The Nonlinear Correlation Between a Novel Metabolic Score for Insulin Resistance and Subclinical Myocardial Injury in the General Population.
Autorzy :: Wang Z; Department of Cardiology, Zhongda Hospital, School of Medicine, Southeast University, Nanjing, China.
Li W; Department of Cardiology, Zhongda Hospital, School of Medicine, Southeast University, Nanjing, China.; Department of Cardiology, Affiliated Hospital of Yangzhou University, Yangzhou, China.
Li J; Department of Hematology and Oncology, Affiliated Xuchang People's Hospital of Xinxiang Medical College, Xuchang, China.
Liu N; Department of Cardiology, Zhongda Hospital, School of Medicine, Southeast University, Nanjing, China.; Pokaż więcej
Źródło :: Frontiers in endocrinology [Front Endocrinol (Lausanne)] 2022 May 24; Vol. 13, pp. 889379. Date of Electronic Publication: 2022 May 24 (Print Publication: 2022).
Typ publikacji :: Journal Article
MeSH Terms :: Cardiovascular Diseases*/epidemiology
Cardiovascular Diseases*/etiology
Insulin Resistance*
Aged ; Cross-Sectional Studies ; Female ; Heart Disease Risk Factors ; Humans ; Male ; Middle Aged ; Nutrition Surveys
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 3 3.

Tytuł :: Correlation between non-insulin-based insulin resistance indexes and the risk of prehypertension: A cross-sectional study.
Autorzy :: Zhang X; Cardiology Department, West China Hospital, Sichuan University, Chengdu, Sichuan, 610041, People's Republic of China.
Yu C; Cardiology Department, Pidu District People's Hospital & The 3rd Affiliated Hospital of Chengdu Medical College, Chengdu, Sichuan, 611700, People's Republic of China.
Ye R; Cardiology Department, West China Hospital, Sichuan University, Chengdu, Sichuan, 610041, People's Republic of China.
Liu T; Cardiology Department, Pidu District People's Hospital & The 3rd Affiliated Hospital of Chengdu Medical College, Chengdu, Sichuan, 611700, People's Republic of China.
Chen X; Cardiology Department, West China Hospital, Sichuan University, Chengdu, Sichuan, 610041, People's Republic of China.; Pokaż więcej
Źródło :: Journal of clinical hypertension (Greenwich, Conn.) [J Clin Hypertens (Greenwich)] 2022 May; Vol. 24 (5), pp. 573-581. Date of Electronic Publication: 2022 Apr 12.
Typ publikacji :: Journal Article
MeSH Terms :: Hypertension*/diagnosis
Hypertension*/epidemiology
Insulin Resistance*
Prehypertension*/epidemiology
Adult ; Biomarkers ; Blood Glucose/metabolism ; Cholesterol, HDL ; Cross-Sectional Studies ; Glucose ; Humans ; Triglycerides
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 4 4.

Tytuł :: Chondroitin sulfate stimulates the secretion of H 2 S by Desulfovibrio to improve insulin sensitivity in NAFLD mice.
Autorzy :: Chen L; Institute of Animal Nutrition and Feed, Jilin Academy of Agricultural Sciences, Changchun 130033, PR China.
Gao Y; Institute of Agricultural Products Processing Technology, Jilin Academy of Agricultural Sciences/National R&D Center for Milk Processing, Changchun 130033, PR China.
Zhao Y; Institute of Agricultural Products Processing Technology, Jilin Academy of Agricultural Sciences/National R&D Center for Milk Processing, Changchun 130033, PR China.
Yang G; Institute of Agricultural Products Processing Technology, Jilin Academy of Agricultural Sciences/National R&D Center for Milk Processing, Changchun 130033, PR China.
Wang C; Institute of Agricultural Products Processing Technology, Jilin Academy of Agricultural Sciences/National R&D Center for Milk Processing, Changchun 130033, PR China.
Zhao Z; Institute of Agricultural Products Processing Technology, Jilin Academy of Agricultural Sciences/National R&D Center for Milk Processing, Changchun 130033, PR China. Electronic address: .
Li S; Institute of Agricultural Products Processing Technology, Jilin Academy of Agricultural Sciences/National R&D Center for Milk Processing, Changchun 130033, PR China. Electronic address: .; Pokaż więcej
Źródło :: International journal of biological macromolecules [Int J Biol Macromol] 2022 Jul 31; Vol. 213, pp. 631-638. Date of Electronic Publication: 2022 Jun 03.
Typ publikacji :: Journal Article
MeSH Terms :: Desulfovibrio*/metabolism
Hydrogen Sulfide*/metabolism
Hydrogen Sulfide*/pharmacology
Insulin Resistance*
Non-alcoholic Fatty Liver Disease*/metabolism
Animals ; Chondroitin Sulfates/metabolism ; Chondroitin Sulfates/pharmacology ; Diet, High-Fat ; Liver/metabolism ; Mice ; Mice, Inbred C57BL ; Proto-Oncogene Proteins c-akt/metabolism
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 5 5.

Tytuł :: Treatment with spexin mitigates diet-induced hepatic steatosis in vivo and in vitro through activation of galanin receptor 2.
Autorzy :: Wang M; Key Laboratory for Metabolic Diseases in Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China.
Zhu Z; Key Laboratory for Metabolic Diseases in Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China.
Kan Y; Key Laboratory for Metabolic Diseases in Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China.
Yu M; Key Laboratory for Metabolic Diseases in Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China.
Guo W; Department of Endocrinology, Clinical Medical College, Yangzhou University, Yangzhou, Jiangsu, 225001, China.
Ju M; Department of Endocrinology, Clinical Medical College, Yangzhou University, Yangzhou, Jiangsu, 225001, China.
Wang J; Department of Endocrinology, Clinical Medical College, Yangzhou University, Yangzhou, Jiangsu, 225001, China.
Yi S; Department of Endocrinology, Clinical Medical College, Yangzhou University, Yangzhou, Jiangsu, 225001, China.
Han S; Key Laboratory for Metabolic Diseases in Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China.
Shang W; Key Laboratory for Metabolic Diseases in Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China.
Zhang Z; Department of Endocrinology, Clinical Medical College, Yangzhou University, Yangzhou, Jiangsu, 225001, China. Electronic address: .
Zhang L; Hanlin College, Nanjing University of Chinese Medicine, Taizhou, 225300, China. Electronic address: .
Fang P; Key Laboratory for Metabolic Diseases in Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing, 210023, China; Hanlin College, Nanjing University of Chinese Medicine, Taizhou, 225300, China. Electronic address: .; Pokaż więcej
Źródło :: Molecular and cellular endocrinology [Mol Cell Endocrinol] 2022 Jul 15; Vol. 552, pp. 111688. Date of Electronic Publication: 2022 May 30.
Typ publikacji :: Journal Article
MeSH Terms :: Insulin Resistance*/physiology
Liver Cirrhosis*/metabolism
Non-alcoholic Fatty Liver Disease*/metabolism
Peptide Hormones/*pharmacology
Animal Feed ; Animals ; Diet, High-Fat ; Liver/metabolism ; Mice ; Mice, Inbred C57BL ; PPAR alpha/genetics ; PPAR alpha/metabolism ; Receptor, Galanin, Type 2/metabolism ; Sirtuin 1/metabolism ; Triglycerides/metabolism
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 6 6.

Tytuł :: Renoprotective effect of empagliflozin in cafeteria diet-induced insulin resistance in rats: Modulation of HMGB-1/TLR-4/NF-κB axis.
Autorzy :: Al-Wakeel DE; Department of Pharmacology and Toxicology, Faculty of Pharmacy, Mansoura University, Mansoura, Egypt.
El-Kashef DH; Department of Pharmacology and Toxicology, Faculty of Pharmacy, Mansoura University, Mansoura, Egypt. Electronic address: dalia_.
Nader MA; Department of Pharmacology and Toxicology, Faculty of Pharmacy, Mansoura University, Mansoura, Egypt.; Pokaż więcej
Źródło :: Life sciences [Life Sci] 2022 Jul 15; Vol. 301, pp. 120633. Date of Electronic Publication: 2022 May 12.
Typ publikacji :: Journal Article
MeSH Terms :: Dyslipidemias*
Insulin Resistance*
Animals ; Benzhydryl Compounds ; Diet ; Glucosides ; HMGB Proteins ; Insulin ; NF-kappa B/metabolism ; Rats ; Toll-Like Receptor 4
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 7 7.

Tytuł :: A comparison of the ratios of C-reactive protein and triglycerides to high-density lipoprotein-cholesterol as biomarkers of metabolic syndrome in African Americans and non-Hispanic Whites.
Autorzy :: Jialal I; Veterans Affairs Medical Center, Mather, CA, United States of America. Electronic address: .
Adams-Huet B; UT Southwestern Medical Center, Dallas, TX, United States of America.
Remaley AT; Translational Vascular Medicine Branch, NHLBI, NIH, Bethesda, MD, United States of America.; Pokaż więcej
Źródło :: Journal of diabetes and its complications [J Diabetes Complications] 2022 Jul; Vol. 36 (7), pp. 108231. Date of Electronic Publication: 2022 Jun 15.
Typ publikacji :: Journal Article
MeSH Terms :: Insulin Resistance*
Metabolic Syndrome*/diagnosis
African Americans ; Biomarkers ; C-Reactive Protein ; Cholesterol, HDL ; Humans ; Nutrition Surveys ; Triglycerides
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 8 8.

Tytuł :: Normalization of maternal adiponectin in obese pregnant mice prevents programming of impaired glucose metabolism in adult offspring.
Autorzy :: Dumolt J; Division of Reproductive Sciences, Department of OB/GYN, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA.
Powell TL; Division of Reproductive Sciences, Department of OB/GYN, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA.; Section of Neonatology, Department of Pediatrics, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA.
Jansson T; Division of Reproductive Sciences, Department of OB/GYN, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA.
Rosario FJ; Division of Reproductive Sciences, Department of OB/GYN, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA.; Pokaż więcej
Źródło :: FASEB journal : official publication of the Federation of American Societies for Experimental Biology [FASEB J] 2022 Jul; Vol. 36 (7), pp. e22383.
Typ publikacji :: Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :: Diabetes, Gestational*/metabolism
Glucose Intolerance*/metabolism
Glucose Intolerance*/prevention & control
Insulin Resistance*
Adiponectin/metabolism ; Animals ; Female ; Fetal Macrosomia/metabolism ; Glucose/metabolism ; Insulin/metabolism ; Male ; Mice ; Mice, Obese ; Placenta/metabolism ; Pregnancy
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 9 9.

Tytuł :: Dietary patterns and the risks of metabolic syndrome and insulin resistance among young adults: Evidence from a longitudinal study.
Autorzy :: Ushula TW; School of Public Health, Faculty of Medicine, The University of Queensland, 266 Herston Rd, Herston QLD 4006, Australia; Nutrition and Dietetics Department, Faculty of Public Health, Jimma University, Jimma, Ethiopia. Electronic address: .
Mamun A; Institute for Social Science Research, The University of Queensland, 80 Meiers Road, Indooroopilly, QLD 4068, Australia; The ARC Centre of Excellence for Children and Families Over the Life Course, The University of Queensland, 80 Meiers Road, Indooroopilly, QLD 4068, Australia; Queensland Alliance for Environmental Health Sciences, The University of Queensland, 80 Meiers Road, Indooroopilly, QLD 4068, Australia.
Darssan D; School of Public Health, Faculty of Medicine, The University of Queensland, 266 Herston Rd, Herston QLD 4006, Australia.
Wang WYS; Faculty of Medicine, The University of Queensland, Princess Alexandra Hospital, Brisbane, Australia.
Williams GM; School of Public Health, Faculty of Medicine, The University of Queensland, 266 Herston Rd, Herston QLD 4006, Australia.
Whiting SJ; College of Pharmacy and Nutrition, University of Saskatchewan, 107 Wiggins Rd, Saskatoon, SK, Canada.
Najman JM; School of Public Health, Faculty of Medicine, The University of Queensland, 266 Herston Rd, Herston QLD 4006, Australia.; Pokaż więcej
Źródło :: Clinical nutrition (Edinburgh, Scotland) [Clin Nutr] 2022 Jul; Vol. 41 (7), pp. 1523-1531. Date of Electronic Publication: 2022 May 18.
Typ publikacji :: Journal Article
MeSH Terms :: Insulin Resistance*
Metabolic Syndrome*/epidemiology
Adult ; Diet ; Diet, Western/adverse effects ; Feeding Behavior ; Humans ; Insulin ; Longitudinal Studies ; Middle Aged ; Prospective Studies ; Risk Factors ; Vegetables ; Young Adult
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 10 10.

Tytuł :: Interplay of retinol binding protein 4 with obesity and associated chronic alterations (Review).
Autorzy :: Flores-Cortez YA; Laboratory of Molecular Biology and Genomic, Faculty of Biological Chemical Sciences, Autonomous University of Guerrero, Chilpancingo, Guerrero 39087, Mexico.
Barragán-Bonilla MI; Laboratory of Molecular Biology and Genomic, Faculty of Biological Chemical Sciences, Autonomous University of Guerrero, Chilpancingo, Guerrero 39087, Mexico.
Mendoza-Bello JM; Laboratory of Molecular Biology and Genomic, Faculty of Biological Chemical Sciences, Autonomous University of Guerrero, Chilpancingo, Guerrero 39087, Mexico.
González-Calixto C; Faculty of Nursing, Autonomous University of Guerrero, Acapulco, Guerrero 39610, Mexico.
Flores-Alfaro E; Laboratory of Clinical and Molecular Epidemiology, Faculty of Biological and Chemical Sciences, Autonomous University of Guerrero, Chilpancingo, Guerrero 39087, Mexico.
Espinoza-Rojo M; Laboratory of Molecular Biology and Genomic, Faculty of Biological Chemical Sciences, Autonomous University of Guerrero, Chilpancingo, Guerrero 39087, Mexico.; Pokaż więcej
Źródło :: Molecular medicine reports [Mol Med Rep] 2022 Jul; Vol. 26 (1). Date of Electronic Publication: 2022 Jun 03.
Typ publikacji :: Journal Article; Review
MeSH Terms :: Insulin Resistance*
Obesity*/genetics
Retinol-Binding Proteins, Plasma*/genetics
Diabetes Mellitus, Type 2/genetics ; Humans ; Insulin/metabolism ; Tumor Necrosis Factor-alpha/metabolism ; Vitamin A/metabolism
: Czasopismo naukowe

Szczegóły Pełny tekst Pełny tekst

Skocz do pozycji: 11 11.

Tytuł :: Endothelial Cell Insulin Signaling Regulates CXCR4 (C-X-C Motif Chemokine Receptor 4) and Limits Leukocyte Adhesion to Endothelium.
Autorzy :: Rathjen T; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).; Novo Nordisk A/S, Måløv, Denmark (T.R., H.W., G.S.O., G.K.P.).
Kunkemoeller B; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).
Cederquist CT; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).
Wang X; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).
Lockhart SM; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).
Patti JC; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).
Willenbrock H; Novo Nordisk A/S, Måløv, Denmark (T.R., H.W., G.S.O., G.K.P.).
Olsen GS; Novo Nordisk A/S, Måløv, Denmark (T.R., H.W., G.S.O., G.K.P.).
Povlsen GK; Novo Nordisk A/S, Måløv, Denmark (T.R., H.W., G.S.O., G.K.P.).
Beck HC; Odense University Hospital, University of Southern Denmark (H.C.B., L.M.R.).
Rasmussen LM; Odense University Hospital, University of Southern Denmark (H.C.B., L.M.R.).
Li Q; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).
Park K; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).
King GL; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).
Rask-Madsen C; Joslin Diabetes Center and Harvard Medical School, Boston, MA (T.R., B.K., C.T.C., X.W., S.M.L., J.C.P., Q.L., K.P., G.L.K., C.R.-M.).; Pokaż więcej
Źródło :: Arteriosclerosis, thrombosis, and vascular biology [Arterioscler Thromb Vasc Biol] 2022 Jul; Vol. 42 (7), pp. e217-e227. Date of Electronic Publication: 2022 Jun 02.
Typ publikacji :: Journal Article
MeSH Terms :: Diabetes Mellitus, Type 2*/genetics
Diabetes Mellitus, Type 2*/metabolism
Insulin Resistance*
Receptors, CXCR4/*metabolism
Animals ; Chemokine CXCL12/genetics ; Chemokine CXCL12/metabolism ; Endothelial Cells/metabolism ; Endothelium/metabolism ; Insulin ; Leukocytes/metabolism ; Mice ; Obesity/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Receptors, CXCR4/genetics
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 12 12.

Tytuł :: Agomelatine, a structural analog of melatonin, improves kidney dysfunction through regulating the AMPK/mTOR signaling pathway to promote autophagy in obese rats.
Autorzy :: Promsan S; Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand.
Thongnak L; Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand.
Pengrattanachot N; Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand.
Phengpol N; Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand.
Sutthasupha P; Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand.
Lungkaphin A; Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand; Functional Food Research Center for Well-Being, Chiang Mai University, Chiang Mai, Thailand. Electronic address: .; Pokaż więcej
Źródło :: Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association [Food Chem Toxicol] 2022 Jul; Vol. 165, pp. 113190. Date of Electronic Publication: 2022 May 29.
Typ publikacji :: Journal Article
MeSH Terms :: Insulin Resistance*
Melatonin*/pharmacology
AMP-Activated Protein Kinases/metabolism ; Acetamides ; Animals ; Autophagy ; Kidney ; Male ; Naphthalenes ; Obesity/metabolism ; Rats ; Rats, Wistar ; Signal Transduction ; TOR Serine-Threonine Kinases/metabolism
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 13 13.

Tytuł :: Family history of diabetes moderates metabolic depression endophenotypes in overweight/obese adults.
Autorzy :: Ong SL; Stanford University School of Medicine, Stanford, CA, 94305, USA.
Abbasi F; Department of Medicine, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, CA, 94305, USA.
Watson K; Department of Psychiatry, Stanford University School of Medicine, Stanford, CA, 94305, USA.
Robakis T; Department of Psychiatry, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.
Myoraku A; Department of Psychiatry, Stanford University School of Medicine, Stanford, CA, 94305, USA.
Rasgon N; Department of Psychiatry, Stanford University School of Medicine, Stanford, CA, 94305, USA. Electronic address: .; Pokaż więcej
Źródło :: Journal of psychiatric research [J Psychiatr Res] 2022 Jul; Vol. 151, pp. 583-589. Date of Electronic Publication: 2022 May 23.
Typ publikacji :: Journal Article
MeSH Terms :: Depressive Disorder, Major*/complications
Depressive Disorder, Major*/epidemiology
Diabetes Mellitus, Type 2*/complications
Diabetes Mellitus, Type 2*/epidemiology
Insulin Resistance*
Adult ; Cross-Sectional Studies ; Depression/complications ; Depression/etiology ; Endophenotypes ; Humans ; Middle Aged ; Obesity ; Overweight/epidemiology
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 14 14.

Tytuł :: Insulin resistance and weight gain in women treated for early stage breast cancer.
Autorzy :: Makari-Judson G; Division of Hematology-Oncology, Baystate Medical Center, 376 Birnie Ave, Springfield, MA, 01107, USA. .; University of Massachusetts Medical School-Baystate, Springfield, MA, USA. .
Viskochil R; Department of Population Health Sciences, University of Utah, Salt Lake City, UT, USA.; Huntsman Cancer Institute, Salt Lake City, UT, USA.
Katz D; Division of Hematology-Oncology, Baystate Medical Center, 376 Birnie Ave, Springfield, MA, 01107, USA.; University of Massachusetts Medical School-Baystate, Springfield, MA, USA.
Barham R; Division of Hematology-Oncology, Baystate Medical Center, 376 Birnie Ave, Springfield, MA, 01107, USA.
Mertens WC; Division of Hematology-Oncology, Baystate Medical Center, 376 Birnie Ave, Springfield, MA, 01107, USA.; University of Massachusetts Medical School-Baystate, Springfield, MA, USA.; Pokaż więcej
Źródło :: Breast cancer research and treatment [Breast Cancer Res Treat] 2022 Jul; Vol. 194 (2), pp. 423-431. Date of Electronic Publication: 2022 May 21.
Typ publikacji :: Journal Article; Observational Study
MeSH Terms :: Breast Neoplasms*/complications
Breast Neoplasms*/drug therapy
Insulin Resistance*
Body Mass Index ; Female ; Humans ; Insulin ; Prospective Studies ; Weight Gain
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 15 15.

Tytuł :: Increased plasma fatty acid clearance, not fatty acid concentration, is associated with muscle insulin resistance in people with obesity.
Autorzy :: Cao C; Center for Human Nutrition, Washington University School of Medicine, 660 S Euclid Ave, St Louis, MO 63110, United States of America.
Koh HE; Center for Human Nutrition, Washington University School of Medicine, 660 S Euclid Ave, St Louis, MO 63110, United States of America.
Van Vliet S; Center for Human Nutrition, Washington University School of Medicine, 660 S Euclid Ave, St Louis, MO 63110, United States of America.
Patterson BW; Center for Human Nutrition, Washington University School of Medicine, 660 S Euclid Ave, St Louis, MO 63110, United States of America.
Reeds DN; Center for Human Nutrition, Washington University School of Medicine, 660 S Euclid Ave, St Louis, MO 63110, United States of America.
Laforest R; Mallinckrodt Institute of Radiology, Washington University School of Medicine, 660 S Euclid Ave, St Louis, MO 63110, United States of America.
Gropler RJ; Mallinckrodt Institute of Radiology, Washington University School of Medicine, 660 S Euclid Ave, St Louis, MO 63110, United States of America.
Mittendorfer B; Center for Human Nutrition, Washington University School of Medicine, 660 S Euclid Ave, St Louis, MO 63110, United States of America. Electronic address: .; Pokaż więcej
Źródło :: Metabolism: clinical and experimental [Metabolism] 2022 Jul; Vol. 132, pp. 155216. Date of Electronic Publication: 2022 May 13.
Typ publikacji :: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
MeSH Terms :: Insulin Resistance*/physiology
Fatty Acids/metabolism ; Fatty Acids, Nonesterified ; Glucose/metabolism ; Humans ; Insulin/metabolism ; Kinetics ; Muscle, Skeletal/metabolism ; Obesity/metabolism
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 16 16.

Tytuł :: Procyanidins and its metabolites by gut microbiome improves insulin resistance in gestational diabetes mellitus mice model via regulating NF-κB and NLRP3 inflammasome pathway.
Autorzy :: Liu Y; Department of Clinical Nutrition, Union Shenzhen Hospitalof Huazhong University of Science and Technology, Shenzhen, China.
Sun R; Department of Clinical Nutrition, Union Shenzhen Hospitalof Huazhong University of Science and Technology, Shenzhen, China.
Lin X; Department of Nutrition and Food Hygiene, Guangdong Provincial Key Laboratory of Tropical Disease Research, School of Public Health, Southern Medical University, Guangzhou, China.
Wu L; Department of Clinical Nutrition, Union Shenzhen Hospitalof Huazhong University of Science and Technology, Shenzhen, China.
Chen H; Injury Prevention Research Center, Shantou University Medical College, Shantou, China.
Shen S; Department of Clinical Nutrition, Union Shenzhen Hospitalof Huazhong University of Science and Technology, Shenzhen, China.
Li Y; Department of Clinical Nutrition, Chengdu Shuangliu District Maternal and Child Health Hospital, Chengdu, China.
Wei Y; Department of Clinical Nutrition, Union Shenzhen Hospitalof Huazhong University of Science and Technology, Shenzhen, China.
Deng G; Department of Clinical Nutrition, Union Shenzhen Hospitalof Huazhong University of Science and Technology, Shenzhen, China. Electronic address: .; Pokaż więcej
Źródło :: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie [Biomed Pharmacother] 2022 Jul; Vol. 151, pp. 113078. Date of Electronic Publication: 2022 May 12.
Typ publikacji :: Journal Article
MeSH Terms :: Diabetes, Gestational*/metabolism
Gastrointestinal Microbiome*
Insulin Resistance*/physiology
Proanthocyanidins*/pharmacology
Proanthocyanidins*/therapeutic use
Animals ; Disease Models, Animal ; Female ; Humans ; Inflammasomes/metabolism ; Inflammation/drug therapy ; Inflammation/metabolism ; Insulin ; Mice ; NF-kappa B/metabolism ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Pregnancy
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 17 17.

Tytuł :: Mechanisms by which smoothelin-like protein 1 reverses insulin resistance in myotubules and mice.
Autorzy :: Tamas I; Department of Medical Chemistry, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Major E; Department of Medical Chemistry, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Horvath D; Department of Medical Chemistry, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Keller I; Department of Medical Chemistry, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Ungvari A; Department of Medical Chemistry, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Haystead TA; Duke University School of Medicine, Department of Pharmacology and Cancer Biology, Durham, NC, USA.
MacDonald JA; Department of Biochemistry and Molecular Biology, Cumming School of Medicine, University of Calgary, Calgary, AB, Canada.
Lontay B; Department of Medical Chemistry, Faculty of Medicine, University of Debrecen, Debrecen, Hungary. Electronic address: .; Pokaż więcej
Źródło :: Molecular and cellular endocrinology [Mol Cell Endocrinol] 2022 Jul 01; Vol. 551, pp. 111663. Date of Electronic Publication: 2022 May 01.
Typ publikacji :: Journal Article
MeSH Terms :: Insulin Resistance*/physiology
Muscle Proteins/*metabolism
Phosphoproteins/*metabolism
Animals ; Glucose/metabolism ; Insulin/metabolism ; Insulin Receptor Substrate Proteins/metabolism ; Mice ; Phosphatidylinositol 3-Kinases/metabolism ; Phosphorylation
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 18 18.

Tytuł :: Lactoferrin improves hepatic insulin resistance and pancreatic dysfunction in high-fat diet and streptozotocin-induced diabetic mice.
Autorzy :: Du Y; Department of Nutrition and Food Hygiene, School of Public Health, Soochow University, Suzhou, 215123, China; Department of Clinical Nutrition, Children's Hospital of Zhejiang University School of Medicine, Hangzhou, China.
Li D; Department of Nutrition and Food Hygiene, School of Public Health, Soochow University, Suzhou, 215123, China.
Chen J; Department of Nutrition and Food Hygiene, School of Public Health, Soochow University, Suzhou, 215123, China.
Li YH; Department of Nutrition and Food Hygiene, School of Public Health, Soochow University, Suzhou, 215123, China.
Zhang Z; Department of General Surgery, the First Affiliated Hospital of Soochow University, Suzhou, 215006, China; State Key Laboratory of Radiation Medicine and Protection, School of Radiation Medicine and Protection, Soochow University, Suzhou 215123, China.
Hidayat K; Department of Nutrition and Food Hygiene, School of Public Health, Soochow University, Suzhou, 215123, China.
Wan Z; Department of Nutrition and Food Hygiene, School of Public Health, Soochow University, Suzhou, 215123, China.
Xu JY; State Key Laboratory of Radiation Medicine and Protection, School of Radiation Medicine and Protection, Soochow University, Suzhou 215123, China.
Qin LQ; Department of Nutrition and Food Hygiene, School of Public Health, Soochow University, Suzhou, 215123, China.; Pokaż więcej
Źródło :: Nutrition research (New York, N.Y.) [Nutr Res] 2022 Jul; Vol. 103, pp. 47-58. Date of Electronic Publication: 2022 Mar 27.
Typ publikacji :: Journal Article
MeSH Terms :: Diabetes Mellitus, Experimental*/drug therapy
Diabetes Mellitus, Type 2*/drug therapy
Insulin Resistance*
Animals ; Diet, High-Fat/adverse effects ; Insulin ; Lactoferrin/adverse effects ; Lactoferrin/metabolism ; Liver/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Pancreas/metabolism ; Phosphatidylinositol 3-Kinase/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Streptozocin/adverse effects ; Streptozocin/metabolism
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 19 19.

Tytuł :: Endothelial HSP72 is not reduced in type 2 diabetes nor is it a key determinant of endothelial insulin sensitivity.
Autorzy :: Pettit-Mee RJ; Department of Nutrition and Exercise Physiology, grid.134936.aUniversity of Missouri, Columbia, Missouri.
Power G; Department of Nutrition and Exercise Physiology, grid.134936.aUniversity of Missouri, Columbia, Missouri.
Cabral-Amador FJ; Department of Medicine, University of Missouri, Columbia, Missouri.
Ramirez-Perez FI; Department of Medicine, University of Missouri, Columbia, Missouri.
Soares RN; Department of Medicine, University of Missouri, Columbia, Missouri.
Sharma N; Department of Medicine, University of Missouri, Columbia, Missouri.
Liu Y; Department of Nutrition and Exercise Physiology, grid.134936.aUniversity of Missouri, Columbia, Missouri.
Christou DD; Department of Applied Physiology and Kinesiology, University of Florida, Gainesville, Florida.
Kanaley JA; Department of Nutrition and Exercise Physiology, grid.134936.aUniversity of Missouri, Columbia, Missouri.
Martinez-Lemus LA; Department of Medicine, University of Missouri, Columbia, Missouri.; Department of Medical Pharmacology and Physiology, University of Missouri, Columbia, Missouri.; Dalton Cardiovascular Research Center, grid.134936.aUniversity of Missouri, Columbia, Missouri.
Manrique-Acevedo C; Dalton Cardiovascular Research Center, grid.134936.aUniversity of Missouri, Columbia, Missouri.; Division of Endocrinology, Diabetes and Metabolism, Department of Medicine, University of Missouri, Columbia, Missouri.; Research Services, Harry S. Truman Memorial MO Veterans Hospital, Columbia, Missouri.
Padilla J; Department of Nutrition and Exercise Physiology, grid.134936.aUniversity of Missouri, Columbia, Missouri.; Dalton Cardiovascular Research Center, grid.134936.aUniversity of Missouri, Columbia, Missouri.; Pokaż więcej
Źródło :: American journal of physiology. Regulatory, integrative and comparative physiology [Am J Physiol Regul Integr Comp Physiol] 2022 Jul 01; Vol. 323 (1), pp. R43-R58. Date of Electronic Publication: 2022 Apr 26.
Typ publikacji :: Journal Article
MeSH Terms :: Diabetes Mellitus, Type 2*/metabolism
HSP72 Heat-Shock Proteins*/genetics
HSP72 Heat-Shock Proteins*/metabolism
Insulin Resistance*
Animals ; Endothelial Cells/metabolism ; Glucose/metabolism ; Insulin/metabolism ; Mice
: Czasopismo naukowe

Szczegóły

Skocz do pozycji: 20 20.

Tytuł :: SERPINA3C ameliorates adipose tissue inflammation through the Cathepsin G/Integrin/AKT pathway.
Autorzy :: Li BY; Key Laboratory of Metabolism and Molecular Medicine of the Ministry of Education, Department of Biochemistry and Molecular Biology of School of Basic Medical Sciences and Department of Endocrinology and Metabolism of Zhongshan Hospital, Fudan University, Shanghai, 200032, China.
Guo YY; Key Laboratory of Metabolism and Molecular Medicine of the Ministry of Education, Department of Biochemistry and Molecular Biology of School of Basic Medical Sciences and Department of Endocrinology and Metabolism of Zhongshan Hospital, Fudan University, Shanghai, 200032, China.
Xiao G; Key Laboratory of Metabolism and Molecular Medicine of the Ministry of Education, Department of Biochemistry and Molecular Biology of School of Basic Medical Sciences and Department of Endocrinology and Metabolism of Zhongshan Hospital, Fudan University, Shanghai, 200032, China.
Guo L; School of Kinesiology, and Shanghai Frontiers Science Research Base of Exercise and Metabolic Health, Shanghai University of Sport, Shanghai, 200438, China. Electronic address: .
Tang QQ; Key Laboratory of Metabolism and Molecular Medicine of the Ministry of Education, Department of Biochemistry and Molecular Biology of School of Basic Medical Sciences and Department of Endocrinology and Metabolism of Zhongshan Hospital, Fudan University, Shanghai, 200032, China. Electronic address: .; Pokaż więcej
Źródło :: Molecular metabolism [Mol Metab] 2022 Jul; Vol. 61, pp. 101500. Date of Electronic Publication: 2022 Apr 15.
Typ publikacji :: Journal Article
MeSH Terms :: Adipose Tissue*/metabolism
Cathepsin G*/metabolism
Cathepsin G*/pharmacology
Insulin Resistance*/physiology
Integrin alpha5beta1*/metabolism
Obesity*/etiology
Obesity*/metabolism
Serpins*/deficiency
Serpins*/metabolism
Adipocytes/metabolism ; Animals ; Diet, High-Fat/adverse effects ; Inflammation/drug therapy ; Inflammation/metabolism ; Integrin beta1/metabolism ; Integrins/metabolism ; Male ; Mice ; Mice, Knockout ; Proto-Oncogene Proteins c-akt/metabolism
: Czasopismo naukowe

Szczegóły

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